Статті в журналах: "Maternal blood pressure"

1

Boyd, Heather A. "Maternal Blood Pressure During Pregnancy." Hypertension 76, no. 3 (September 2020): 670–71. http://dx.doi.org/10.1161/hypertensionaha.120.15458.

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2

de Swiet, Michael. "Maternal blood pressure and birthweight." Lancet 355, no. 9198 (January 2000): 81–82. http://dx.doi.org/10.1016/s0140-6736(99)00288-3.

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3

Gillman, Matthew W., Carol L. Link, Janet W. Rich-Edwards, Ellice S. Lieberman, and Steven E. Lipshultz. "Maternal age and newborn blood pressure." Circulation 103, suppl_1 (March 2001): 1347. http://dx.doi.org/10.1161/circ.103.suppl_1.9999-18.

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0018 Background. Blood pressure (bp) level after birth may reflect the fetal cardiovascular environment, but few data exist on newborn bp. Methods. We report findings from a new cohort of pregnant women and their offspring, Project Viva. Enrollment as of September, 2000, is 1338 pregnant women (of a target 6000), and 581 have delivered. Data collected include maternal dietary, sociodemographic, economic, psychological, lifestyle, and clinical variables; and infant bp, pulse rate, length, weight, and circumferences measured during the first 2 days of life. We averaged 5 systolic bp readings taken with a Dinamap automated bp recorder. This abstract is limited to the first 260 measured newborns (131 girls and 129 boys) and partial maternal data. Results. The highest newborn systolic bp was in children of the oldest mothers, and the lowest in the maternal age group 20-24 years ( Table). Multivariate adjustment for infant birth weight and sex and for maternal body mass index, pregnancy weight gain, 3 rd trimester bp, and number of pregnancies did not attenuate the differences. For example, adjusted mean newborn systolic bp was 10.8 (95% CI 2.4, 19.2) mmHg higher among children of 40-44 y.o. than 20-24 y.o. mothers. Variables minimally or not related to newborn systolic bp included birth weight and length, pulse rate, head, chest, and abdominal circumferences, gestational age, and maternal body mass index, weight gain, and systolic bp. Conclusions. Higher maternal age was associated with higher newborn systolic bp. Whereas bp later in childhood predicts adult hypertension and its consequences, newborn bp may represent different phenomena, such as pre- and peri-natal influences on cardiac structure and function. Studies of newborn bp may shed new light on effects of the fetal environment on lifelong cardiovascular health. Table 1.

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4

Gomez-Sanchez, Elise P., and Celso E. Gomez-Sanchez. "Maternal Hypertension and Progeny Blood Pressure." Hypertension 33, no. 6 (June 1999): 1369–73. http://dx.doi.org/10.1161/01.hyp.33.6.1369.

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5

Lim, Wai-Yee, Yung-Seng Lee, Fabian Kok-Peng Yap, Izzudin Mohd Aris, Ngee Lek, Michael Meaney, Peter D. Gluckman, et al. "Maternal Blood Pressure During Pregnancy and Early Childhood Blood Pressures in the Offspring." Medicine 94, no. 45 (November 2015): e1981. http://dx.doi.org/10.1097/md.0000000000001981.

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6

Lim, Wai-Yee, Kenneth Kwek, Yap-Seng Chong, Yung-Seng Lee, Fabian Yap, Yiong-Huak Chan, Keith M. Godfrey, Peter D. Gluckman, Seang-Mei Saw, and An Pan. "Maternal adiposity and blood pressure in pregnancy." Journal of Hypertension 32, no. 4 (April 2014): 857–64. http://dx.doi.org/10.1097/hjh.0000000000000096.

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7

Wells, Ellen M., Lynn R. Goldman, Jeffery M. Jarrett, Benjamin J. Apelberg, Julie B. Herbstman, Kathleen L. Caldwell, Rolf U. Halden, and Frank R. Witter. "Selenium and maternal blood pressure during childbirth." Journal of Exposure Science & Environmental Epidemiology 22, no. 2 (November 23, 2011): 191–97. http://dx.doi.org/10.1038/jes.2011.42.

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8

Gillman, Matthew W., Sheryl L. Rifas-Shiman, Ken P. Kleinman, Janet W. Rich-Edwards, and Steven E. Lipshultz. "Maternal Calcium Intake and Offspring Blood Pressure." Circulation 110, no. 14 (October 5, 2004): 1990–95. http://dx.doi.org/10.1161/01.cir.0000143199.93495.96.

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9

Law, C. M., D. J. Barker, A. R. Bull, and C. Osmond. "Maternal and fetal influences on blood pressure." Archives of Disease in Childhood 66, no. 11 (November 1, 1991): 1291–95. http://dx.doi.org/10.1136/adc.66.11.1291.

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10

Taylor, S. J. C., K. Hird, P. Whincup, and D. Cook. "Relation between birth weight and blood pressure is independent of maternal blood pressure." BMJ 317, no. 7159 (September 5, 1998): 680. http://dx.doi.org/10.1136/bmj.317.7159.680.

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11

Yesil, Gizem Dilan, Olta Gishti, Janine F. Felix, Irwin Reiss, Mohammad Kamran Ikram, Eric A. P. Steegers, Albert Hofman, Vincent W. V. Jaddoe, and Romy Gaillard. "Influence of Maternal Gestational Hypertensive Disorders on Microvasculature in School-Age Children." American Journal of Epidemiology 184, no. 9 (November 1, 2016): 605–15. http://dx.doi.org/10.1093/aje/kww059.

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Abstract Gestational hypertensive disorders may lead to vascular changes in the offspring. We examined the associations of maternal blood pressure development and hypertensive disorders during pregnancy with microvasculature adaptations in the offspring in childhood. This study was performed as part of the Generation R Study in Rotterdam, the Netherlands (2002–2012), among 3,748 pregnant mothers and their children for whom information was available on maternal blood pressure in different periods of pregnancy and gestational hypertensive disorders. Childhood retinal arteriolar and venular calibers were assessed at the age of 6 years. We found that higher maternal systolic and diastolic blood pressures in early pregnancy were associated with childhood retinal arteriolar narrowing (P < 0.05). Higher maternal systolic blood pressure in late pregnancy, but not in middle pregnancy, was associated with childhood narrower retinal venular caliber (standard deviation score per standardized residual increase in systolic blood pressure: −0.05; 95% confidence interval: −0.08, −0.01). Paternal blood pressure was not associated with childhood retinal vessel calibers. Children of mothers with gestational hypertensive disorders tended to have narrower retinal arteriolar caliber (standard deviation score: −0.13, 95% confidence interval: −0.27, 0.01). Our results suggest that higher maternal blood pressure during pregnancy is associated with persistent microvasculature adaptations in their children. Further studies are needed to replicate these observations.

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12

Gaillard, Romy, and Vincent W. V. Jaddoe. "Assessment of maternal blood pressure development during pregnancy." Journal of Hypertension 33, no. 1 (January 2015): 61–62. http://dx.doi.org/10.1097/hjh.0000000000000419.

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13

Cabral, Maria, Maria J. Fonseca, Camila González-Beiras, Ana C. Santos, Liane Correia-Costa, and Henrique Barros. "Maternal Smoking: A Life Course Blood Pressure Determinant?" Nicotine & Tobacco Research 20, no. 6 (May 31, 2017): 674–80. http://dx.doi.org/10.1093/ntr/ntx117.

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14

VYAS, S., S. CAMPBELL, S. BOWER, and K. N. NICOLAIDES. "Maternal Abdominal Pressure Alters Fetal Cerebral Blood Flow." Obstetrical & Gynecological Survey 46, no. 3 (March 1991): 138. http://dx.doi.org/10.1097/00006254-199103000-00003.

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15

Vyas, S., S. Campbell, S. Bower, and K. H. Nicolaides. "Maternal abdominal pressure alters fetal cerebral blood flow." BJOG: An International Journal of Obstetrics and Gynaecology 97, no. 8 (August 1990): 740–42. http://dx.doi.org/10.1111/j.1471-0528.1990.tb16250.x.

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16

Falkner, Bonita. "Maternal and gestational influences on childhood blood pressure." Pediatric Nephrology 35, no. 8 (February 21, 2019): 1409–18. http://dx.doi.org/10.1007/s00467-019-4201-x.

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17

Leary, S. D. "Maternal diet in pregnancy and offspring blood pressure." Archives of Disease in Childhood 90, no. 5 (May 1, 2005): 492–93. http://dx.doi.org/10.1136/adc.2004.070698.

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18

Wells, E., L. Goldman, K. Caldwell, R. Jones-L, B. Apelberg, and J. Herbstman. "Modeling Low Level Cord Blood Lead Exposure and Maternal Blood Pressure." Epidemiology 18, Suppl (September 2007): S172. http://dx.doi.org/10.1097/01.ede.0000276863.63298.23.

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19

Mitchell, Courtney J., Alan Tita, Sarah B. Anderson, Daniel N. Pasko, and Lorie M. Harper. "Adverse Outcomes with Maternal Blood Pressure Less than 140/90 in Pregnancy Complicated by Hypertension." American Journal of Perinatology 36, no. 13 (January 15, 2019): 1394–400. http://dx.doi.org/10.1055/s-0038-1677474.

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Objective We assessed the risk of small for gestational age and other outcomes in pregnancies complicated by chronic hypertension with blood pressure <140/90 mm Hg. Study Design Retrospective cohort of singletons with hypertension at a single institution from 2000 to 2014. Mean systolic blood pressure and mean diastolic blood pressure were analyzed as continuous and dichotomous variables (<120/80 and 120–139/80–89 mm Hg). The primary outcome was small for gestational age. Secondary outcomes included birth weight, preeclampsia, preterm birth <35 weeks, and a composite of adverse neonatal outcomes. Results Small for gestational age was not increased with a mean systolic blood pressure <120 mm Hg compared with a mean systolic blood pressure 120 to 129 mm Hg (adjusted odds ratio [AOR] 1.6; 95% confidence interval [CI] 0.92–2.79). Mean diastolic blood pressure <80 mm Hg was associated with a decrease in the risk preeclampsia (AOR 0.57; 95% CI 0.35–0.94), preterm birth <35 weeks (AOR 0.35; 95% CI 0.20–0.62), and the composite neonatal outcome (AOR 0.42; 95% CI 0.22–0.81). Conclusion Mean systolic blood pressure <120 mm Hg and mean diastolic blood pressure <80 mm Hg were not associated with increased risk of small for gestational age when compared with higher, normal mean systolic and diastolic blood pressures.

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20

Martins, Juliana de Oliveira, Maurício Isaac Panício, Marcos Paulo Suehiro Dantas, and Guiomar Nascimento Gomes. "Effect of maternal diabetes on female offspring." Einstein (São Paulo) 12, no. 4 (December 2014): 413–19. http://dx.doi.org/10.1590/s1679-45082014ao3200.

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Objective To evaluate the effect of maternal diabetes on the blood pressure and kidney function of female offspring, as well as if such changes exacerbate during pregnancy.MethodsDiabetes mellitus was induced in female rats with the administration of streptozotocin in a single dose, one week before mating. During pregnancy, blood pressure was measured through plethysmography. On the 20th day of pregnancy, the animals were placed for 24 hours in metabolic cages to obtain urine samples. After the animals were removed from the cages, blood samples were withdrawn. One month after pregnancy, new blood and urine sample were collected. Kidney function was evaluated through proteinuria, plasma urea, plasma creatinine, creatinine excretion rate, urinary flow, and creatinine clearance.Results The female offspring from diabetic mothers showed an increase in blood pressure, and a decrease in glomerular filtration rate in relation to the control group.Conclusion Hyperglycemia during pregnancy was capable of causing an increase in blood pressure and kidney dysfunction in the female offspring.

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21

van den Hil, Leontine C. L., H. Rob Taal, Layla L. de Jonge, Denise H. M. Heppe, Eric A. P. Steegers, Albert Hofman, Albert J. van der Heijden, and Vincent W. V. Jaddoe. "Maternal first-trimester dietary intake and childhood blood pressure: the Generation R Study." British Journal of Nutrition 110, no. 8 (March 26, 2013): 1454–64. http://dx.doi.org/10.1017/s0007114513000676.

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Suboptimal maternal dietary intake during pregnancy might lead to fetal cardiovascular adaptations and higher blood pressure in the offspring. The aim of the present study was to investigate the associations of maternal first-trimester dietary intake with blood pressure in children at the age of 6 years. We assessed first-trimester maternal daily dietary intake by a FFQ and measured folate, homocysteine and vitamin B12 concentrations in the blood, in a population-based prospective cohort study among 2863 mothers and children. Childhood systolic and diastolic blood pressure was measured using a validated automatic sphygmomanometer. First-trimester maternal daily intake of energy, fat, protein and carbohydrate was not associated with childhood blood pressure. Furthermore, maternal intake of micronutrients was not associated with childhood blood pressure. Also, higher maternal vitamin B12 concentrations were associated with a higher diastolic blood pressure (0·31 mmHg per standard deviation increase in vitamin B12 (95 % CI 0·06, 0·56)). After taking into account multiple testing, none of the associations was statistically significant. Maternal first-trimester folate and homocysteine concentrations were not associated with childhood blood pressure. The results from the present study suggest that maternal Fe intake and vitamin B12 concentrations during the first trimester of pregnancy might affect childhood blood pressure, although the effect estimates were small and were not significant after correction for multiple testing. Further studies are needed to replicate these findings, to elucidate the underlying mechanisms and to assess whether these differences in blood pressure persist in later life.

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22

Birukov, Anna, Florian Herse, Julie H. Nielsen, Henriette B. Kyhl, Michaela Golic, Kristin Kräker, Nadine Haase, et al. "Blood Pressure and Angiogenic Markers in Pregnancy." Hypertension 76, no. 3 (September 2020): 901–9. http://dx.doi.org/10.1161/hypertensionaha.119.13966.

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Pregnancy-induced hypertension is a severe pregnancy complication, increasing risk of long-term cardiovascular disease in mothers and offspring. We hypothesized that maternal blood pressure in pregnancy associated with offspring blood pressure; that the associations were sex-specific; and that maternal circulating placental angiogenic markers (PlGF [placental growth factor] and sFlt-1 [soluble fms-like tyrosine kinase-1]) mediated this relationship. We analyzed data from 2434 women and 2217 children from the Odense Child Cohort, a prospective Danish cohort study. Offspring blood pressure trajectory from 4 months to 5 years was highly associated to maternal first, second, and third trimester blood pressure, and mean blood pressure in pregnancy, independent of maternal and offspring covariates. There were offspring sex-specific associations: Girls from mothers in the highest quartile of first and third trimester blood pressure had significantly higher systolic blood pressure at 5 years than the rest of the cohort (mean difference±SEM: 1.81±0.59 and 2.11±0.59 mm Hg, respectively, all P <0.01); whereas boys had significantly higher diastolic blood pressure at 5 years (mean difference±SEM: 1.11±0.45 and 1.03±0.45, respectively, all P <0.05). Concentrations of PlGF at gestational week 28 correlated inversely to maternal gestational blood pressure trajectory, independent of the diagnosis of pregnancy-induced hypertension, adjusted β coefficients (95% CI) for predicting systolic blood pressure (SBP): −3.18 (−4.66 to −1.70) mm Hg, for predicting diastolic blood pressure (DBP): −2.48 (−3.57 to −1.40) mm Hg. In conclusion, maternal gestational blood pressure predicted offspring blood pressure trajectory until 5 years in a sex-differential manner. Furthermore, subtle alterations in blood pressure in early pregnancy preceded hypertension or preeclampsia, and PlGF was a mediator of cardiovascular health in pregnancy.

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23

Woods, Lori L., and Douglas A. Weeks. "Prenatal programming of adult blood pressure: role of maternal corticosteroids." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 289, no. 4 (October 2005): R955—R962. http://dx.doi.org/10.1152/ajpregu.00455.2004.

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Both maternal glucocorticoid administration and maternal dietary protein or food restriction in pregnancy cause fewer nephrons and hypertension in the adult offspring. The purpose of these studies was to determine the extent to which nutritional factors contribute to programming of offspring hypertension by maternal glucocorticoids. Pregnant rats were treated with dexamethasone (100 μg·kg−1·d−1sc) on days 1–10 (ED) or days 15–20 (LD) of pregnancy. Additional groups of pregnant animals were pair fed to the early (EDPF) and late (LDPF) dexamethasone-treated groups, and another group was untreated or given vehicle (C). The dams treated with dexamethasone reduced their food intake and lost or failed to gain a normal amount of weight during treatment; body weights of ED dams caught up to normal after the treatment period, whereas those of LD dams did not. In adulthood (∼21 wks), chronically instrumented male offspring of ED had normal blood pressures (125 ± 2 mmHg vs. 126 ± 1 mmHg in C), whereas LD offspring were hypertensive (136 ± 3 mmHg). However, LDPF offspring were equally hypertensive (134 ± 2 mmHg). Glomerular filtration rates normalized to body weight were not significantly different among groups. Qualitatively similar results were found in female offspring. Thus the long-term effects of maternal glucocorticoid administration at this dose on offspring’s blood pressure may, in large part, be accounted for by the reduction in maternal food intake. These data suggest that maternal glucocorticoids and maternal food or protein restriction may, at least in part, share a common mechanism in programming offspring for hypertension. The window of sensitivity of future offspring blood pressure to either maternal insult coincides with nephrogenesis in the rat, suggesting that impaired renal development could play an important role in this programming.

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24

Tanaka, Hiroaki, Junichi Hasegawa, Shinji Katsuragi, Kayo Tanaka, Tatsuya Arakaki, Masamitsu Nakamura, Eijiro Hayata, et al. "Are There Maternal Deaths Related to Hemorrhagic Stroke Due to Hypertensive Disorder of Pregnancy That Could Be Potentially Preventable by Tight Hypertension Management in Antepartum? A Retrospective Study from the Maternal Death Exploratory Committee in Japan." Journal of Clinical Medicine 12, no. 8 (April 17, 2023): 2908. http://dx.doi.org/10.3390/jcm12082908.

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Background: Unlike Europe and the United States, Japan has seen numerous maternal deaths from hemorrhagic strokes related to hypertensive disorders of pregnancy (HDP). This study retrospectively analyzed deaths associated with HDP-related hemorrhagic stroke in Japan to determine the number of deaths that may have been prevented with blood pressure control during pregnancy. Methods: This study included maternal deaths related to hemorrhagic stroke cases. The proportion of patients without proteinuria whose blood pressure exceeded 140/90 mmHg between 14+0 and 33+6 weeks of gestation were determined. Lastly, the application of tight antihypertensive management was evaluated. Results: Among 34 HDP-related maternal deaths, 4 cases involved patients without proteinuria whose blood pressures exceeded 140/90 mmHg between 14+0 and 33+6 weeks of gestation. These included two chronic hypertension and two gestational hypertension cases. None of the patients received antihypertensive agents, and their blood pressures were managed leniently. Conclusion: Among HDP-related hemorrhagic stroke deaths in Japan, only a few cases of maternal death could have been prevented with tight blood pressure management, as described in the CHIPS randomized controlled trial. Therefore, to prevent HDP-related hemorrhagic stroke in Japan, new preventive strategies during pregnancy should be established.

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25

Alexander, Barbara T. "Is Maternal Blood Pressure the Key to Vascular Dysfunction in Preterm Offspring With Elevated Blood Pressure?" Hypertension 56, no. 1 (July 2010): 34–35. http://dx.doi.org/10.1161/hypertensionaha.110.153338.

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26

Jensen, Eva Johanne Leknes, Ellen Aagaard Nohr, Thomas Scholbach, and Torbjørn Moe Eggebø. "Peripheral Blood Flow Intensity in Maternal Kidneys and Correlation with Blood Pressure." Maternal-Fetal Medicine 2, no. 3 (February 24, 2020): 141–47. http://dx.doi.org/10.1097/fm9.0000000000000039.

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27

Maggioni, C., G. Cornélissen, K. Otsuka, F. Halberg, D. Consonni, and U. Nicolini. "Circadian rhythm of maternal blood pressure and fetal growth." Biomedicine & Pharmacotherapy 59 (October 2005): S86—S91. http://dx.doi.org/10.1016/s0753-3322(05)80015-0.

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28

Gillman, Matthew W., Janet W. Rich-Edwards, Sheryl L. Rifas-Shiman, Ellice S. Lieberman, Ken P. Kleinman, and Steven E. Lipshultz. "Maternal age and other predictors of newborn blood pressure." Journal of Pediatrics 144, no. 2 (February 2004): 240–45. http://dx.doi.org/10.1016/j.jpeds.2003.10.064.

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Barrett, Helen L., Karin Lust, Narelle Fagermo, Leonie K. Callaway, and Lee Minuzzo. "Moyamoya disease in pregnancy: maintenance of maternal blood pressure." Obstetric Medicine 5, no. 1 (December 8, 2011): 32–34. http://dx.doi.org/10.1258/om.2011.110046.

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Moyamoya disease is a rare cerebrovascular occlusive disorder characterized by stenosis in the circle of Willis with the development of a compensatory circulation. It has been associated with significant morbidity in pregnancy including intracranial haemorrhage, ischaemic stroke and epilepsy. We present the case of a 26-year-old woman with a previous diagnosis of moyamoya vasculopathy with bilateral superficial temporal to middle cerebral artery bypass grafting. During the second trimester, she developed significant neurological symptoms related to postural hypotension in the presence of a stenosis of the right-sided graft. The hypotension was treated with fludrocortisone therapy with improvement in blood pressure and symptoms. Moyamoya vasculopathy poses unique challenges to obstetric care. This is the first report of use of fludrocortisone for maintenance of blood pressure during pregnancy in this condition.

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30

Friedman, Ernest H. "Neurobiology of prenatal maternal blood pressure response to stress." American Journal of Obstetrics and Gynecology 176, no. 3 (March 1997): 733. http://dx.doi.org/10.1016/s0002-9378(97)70592-1.

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31

Lafeber, H. N. "Maternal Age and Other Predictors of Newborn Blood Pressure." Yearbook of Obstetrics, Gynecology and Women's Health 2006 (January 2006): 162. http://dx.doi.org/10.1016/s1090-798x(08)70410-7.

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32

Barker, D., C. Osmond, S. Grant, K. L. Thornburg, C. Cooper, S. Ring, and G. Davey-Smith. "Maternal cotyledons at birth predict blood pressure in childhood." Placenta 34, no. 8 (August 2013): 672–75. http://dx.doi.org/10.1016/j.placenta.2013.04.019.

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33

Hu, Jie, Yuanyuan Li, Bin Zhang, Tongzhang Zheng, Jun Li, Yang Peng, Aifen Zhou, et al. "Impact of the 2017 ACC/AHA Guideline for High Blood Pressure on Evaluating Gestational Hypertension–Associated Risks for Newborns and Mothers." Circulation Research 125, no. 2 (July 5, 2019): 184–94. http://dx.doi.org/10.1161/circresaha.119.314682.

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Rationale: In 2017, the American College of Cardiology (ACC)/American Heart Association (AHA) released a new hypertension guideline for nonpregnant adults, using lower blood pressure values to identify hypertension. However, the impact of this new guideline on the diagnosis of gestational hypertension and the associated maternal and neonatal risks are unknown. Objective: To estimate the impact of adopting the 2017 ACC/AHA guideline on detecting gestational blood pressure elevations and the relationship with maternal and neonatal risk in the perinatal period using a retrospective cohort design. Methods and Results: This study included 16 345 women from China. Systolic and diastolic blood pressures of each woman were measured at up to 22 prenatal care visits across different stages of pregnancy. Logistic and linear regressions were used to estimate associations of blood pressure categories with the risk of preterm delivery, early-term delivery, and small for gestational age, and indicators of maternal liver, renal, and coagulation functions during pregnancy. We identified 4100 (25.1%) women with gestational hypertension using the 2017 ACC/AHA guideline, compared with 4.2% using the former definition. Gestational hypertension, but not elevated blood pressure (subclinical blood pressure elevation), was significantly associated with altered indicators of liver, renal, and coagulation functions during pregnancy for mothers and increased risk of adverse birth outcomes for newborns; adjusted odds ratios (95% CIs) for gestational hypertension stage 2 were 2.23 (1.18–4.24) for preterm delivery, 2.05 (1.67–2.53) for early-term delivery, and 1.43 (1.13–1.81) for small for gestational age. Conclusions: Adopting the 2017 ACC/AHA guideline would result in a substantial increase in the prevalence of gestational hypertension; subclinical blood pressure elevations during late pregnancy were not associated with increased maternal and neonatal risk in this cohort. Therefore, the 2017 ACC/AHA guideline may improve the detection of high blood pressure during pregnancy and the efforts to reduce maternal and neonatal risk. Replications in other populations are required.

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Deng, Wenfeng, Lanlan Guo, Miaoting Zhou, and Huishu Liu. "P-091. Ambulatory blood pressure parameters in maternal hypotension: Smooth subtype of short-term blood pressure rhythms." Pregnancy Hypertension 25 (September 2021): e58-e59. http://dx.doi.org/10.1016/j.preghy.2021.07.126.

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35

Tenhola, Sirpa, Eero Rahiala, Pirjo Halonen, Esko Vanninen, and Raimo Voutilainen. "Maternal Preeclampsia Predicts Elevated Blood Pressure in 12-Year-Old Children: Evaluation by Ambulatory Blood Pressure Monitoring." Pediatric Research 59, no. 2 (February 2006): 320–24. http://dx.doi.org/10.1203/01.pdr.0000196734.54473.e3.

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36

Rusydiana, Hamidah Aula, Mira Dewi, Hadi Riyadi, and Lilik Kustiyah. "Maternal Fasting Blood Glucose, Blood Pressure, and Amniotic Fluid Volume with Baby Birth Length and Weight." Jurnal Ilmiah Kesehatan (JIKA) 4, no. 2 (August 31, 2022): 277–87. http://dx.doi.org/10.36590/jika.v4i2.311.

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Maternal health during pregnancy is affecting fetal health in the womb and after birth. This study was conducted to see how maternal fasting blood glucose, blood pressure, and amniotic fluid volume in third semester correlate with baby birth weight and length. The design of this research was observasional in cohort program from third trimester pregnancy until delivery. Result showed that the rising of maternal fasting blood glucose in third trimester was predicted to significantly increasing the baby’s birth weight (b= 7,24, p= 0,025). Inversely, the increase in blood pressure during pregnancy had the potential to lower the weight of the baby (b= -13,94, p= 0,058) although not significanty proven in this study. The correlation between maternal amniotic fluid volume had not shown significant results with neither birth length nor weight of the offspring in this study. It is recommended that pregnant women could maintain their health and also conducted regular health checks of blood glucose, blood pressure, and amniotic fluid volume to avoid pregnancy adverse outcomes if detected.

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37

Daniel, S. S., R. I. Stark, M. M. Myers, P. J. Tropper, and Y. I. Kim. "Blood pressure and HR in the fetal lamb: relationship to hypoglycemia, hypoxemia, and growth restriction." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 271, no. 5 (November 1, 1996): R1415—R1421. http://dx.doi.org/10.1152/ajpregu.1996.271.5.r1415.

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We examined blood pressure and heart rate (HR) in relation to glucose and arterial PO2 (PaO2) at approximately 121 days (early) and at approximately 140 days (late) gestation in 12 growth-restricted and 10 control fetal lambs. Mild growth restriction (relative to maternal weight) was produced by withdrawal of 25 ml/day of maternal blood during the second half of pregnancy (P < 0.05). Fetuses from this model are hypoglycemic during early and late gestation but hypoxemic only during late study. Mean systolic and diastolic pressures in the experimental group were approximately 8.0 mmHg lower than the corresponding values in controls at both studies (P < 0.05). Fetal HR (FHR) was 15.4 beats/min lower (P < 0.05) in 10 but was higher than control in 2 experimental fetuses that were also not growth restricted. There were significant correlations between late systolic pressure and HR and PaO2 (r = 0.54, P = 0.046 and r = 0.50, P = 0.049, respectively) and between FHR and blood pressure and birth weight/maternal weight (P < 0.05). We conclude that, in this model, fetal blood pressure and HR may serve as good indicators of hypoxemia and growth restriction.

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38

Baranova, E., and O. Bolshakova. "Prognostic values of high blood pressure duringpregnancy." "Arterial’naya Gipertenziya" ("Arterial Hypertension") 14, no. 1 (February 28, 2008): 22–26. http://dx.doi.org/10.18705/1607-419x-2008-14-1-22-26.

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Arterial hypertension in pregnancy is now believed to be a risk factor for future maternal cardiovascular diseases. Despite the low immediate cardiovascular risk in a population of young women, a pregnancy complicated with hypertension carries a significant additional risk of future disease.

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Bukar, I. A., S. A. Ibrahim, D. G. Mahdi, and I. U. Yarube. "Relationship between Fetal Heart Rate, Cardiac Index and Maternal Blood Pressure in Second Trimester of Pregnancy between 14 to 20 Weeks of Gestation in Yobe State Specialist Hospital Damaturu." Journal of Advances in Medicine and Medical Research 35, no. 22 (October 12, 2023): 118–28. http://dx.doi.org/10.9734/jammr/2023/v35i225253.

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Aims: The aim of this study is to assess the relationship between fetal heart rate, fetal cardiac index and maternal blood pressure at 14-20 weeks of gestation among antenatal clients attending Yobe state specialist hospital, Damaturu. Study Design: The study is a cross-sectional study. Place and Duration of Study: The study was conducted in Yobe State specialist hospital Damaturu between June 2021 and November 2021. Methodology: The samples were selected using a purposive sampling method where 150 pregnant women were selected, and group into three categories (hypertensive, hypotensive and normotensive). Subjects undergo ultrasound scan imaging. The data was analyzed using software program SPSS statistical package (IBM Corp. IBM SPSS Statistics for Macintosh, Version 25.0. Armonk, NY, USA). Results: A statistically significant relationship was observed between maternal blood pressure and fetal heart rate (p=0.000), but no significant relationship observed between maternal blood pressure and fetal cardiac index (p=0.476), and also between maternal age and the fetal heart rate (p=0.533) or between maternal age and fetal cardiac index (p=0.528). Conclusion: Fetal heart rate has shown a significant correlation with maternal blood pressure. Thus maternal blood pressure changes can serve as a highly valuable parameter in providing vital information on fetal condition during pregnancy

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Rabe, Heike, Varsha Bhatt-Mehta, Stephen A. Bremner, Aisling Ahluwalia, Renske Mcfarlane, Simin Baygani, Beau Batton, et al. "Antenatal and perinatal factors influencing neonatal blood pressure: a systematic review." Journal of Perinatology 41, no. 9 (August 7, 2021): 2317–29. http://dx.doi.org/10.1038/s41372-021-01169-5.

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Abstract Objective A comprehensive understanding of the factors contributing to perinatal blood pressure is vital to ensure optimal postnatal hemodynamic support. The objective of this study was to review existing literature on maternal and perinatal factors influencing blood pressure in neonates up to 3 months corrected age. Methods A systematic search of published literature in OVID Medline, OVID Embase and the COCHRANE library identified publications relating to maternal factors affecting blood pressure of neonates up to corrected age of 3 months. Summary data were extracted and compared (PROSPERO CRD42018092886). Results Of the 3683 non-duplicate publications identified, 44 were eligible for inclusion in this review. Topics elicited were sociodemographic factors, maternal health status, medications, smoking during pregnancy, and cord management at birth. Limited data were available for each factor. Results regarding the impact of these factors on neonatal blood pressure were inconsistent across studies. Conclusions There is insufficient evidence to draw definitive conclusions regarding the impact of various maternal and perinatal factors on neonatal blood pressure. Future investigations of neonatal cardiovascular therapies should account for these factors in their study design. Similarly, studies on maternal diseases and perinatal interventions should include neonatal blood pressure as part of their primary or secondary analyses.

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Dunietz, Galit Levi, Wei Hao, Kerby Shedden, Claudia Holzman, Ronald Chervin, Lynda Lisabeth, Marjorie Treadwell, and Louise O’Brien. "470 Maternal Habitual Snoring and Blood Pressure Trajectories in Pregnancy." Sleep 44, Supplement_2 (May 1, 2021): A185. http://dx.doi.org/10.1093/sleep/zsab072.469.

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Abstract Introduction Habitual snoring has been associated with hypertensive disorders of pregnancy. However, exactly when blood pressure (BP) trajectories diverge between pregnant women with and without habitual snoring is unknown. Moreover, the potentially differential impact of chronic versus pregnancy-onset habitual snoring on maternal BP trajectories during pregnancy has not been examined. Methods In a cohort study of 1,305 pregnant women from a large Midwestern medical center, participants were asked about habitual snoring (≥3 nights/week) and whether their symptoms began prior to or during pregnancy. Demographic and BP data throughout pregnancy, systolic (SBP) and diastolic (DBP) were abstracted from medical charts. Linear mixed models were used to examine associations between habitual snoring-onset and pregnancy BP trajectories. Results Thirty percent of women reported snoring before pregnancy (chronic snoring) and an additional 23% reported pregnancy-onset snoring. Overall, women with pregnancy-onset snoring had higher mean SBP and DBP compared to those with chronic habitual snoring or controls (non-habitual snoring). In gestational week-specific comparisons with controls, SBP became significantly higher around 20 weeks’ gestation among women with pregnancy-onset snoring and in the third trimester among women with chronic snoring. Pairwise mean differences in DBP were significant only among women with pregnancy-onset snoring relative to controls, after 15 weeks’ gestation. Conclusion In a large cohort of pregnant women, those with pregnancy-onset or chronic habitual snoring had significantly elevated systolic BP in comparison to non-habitual snoring controls, in the second and third trimester, respectively. The findings of divergent BP trajectories suggest the two groups of women with habitual snoring in pregnancy should be considered separately when evaluating gestational ‘windows’ for increased BP monitoring and provide insight into pathophysiologic changes. Support (if any) Dr. Dunietz was supported by an F32 National Research Service Award from the National Institute of Child Health and Development (NIH/NICHD F32 HD091938); Dr. O’Brien was supported by the following during the course of this study: the Gene and Tubie Gilmore Fund for Sleep Research, the University of Michigan Institute for Clinical and Health Research (MICHR) grants UL1RR024986 and UL1TR000433, MICHR seed pilot grant F021024, the National Heart, Lung, and Blood Institute (R21 HL089918 and K23 HL095739) and in part by R21 HL087819.

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Godfrey, K. M., T. Forrester, D. J. P. Barker, A. A. Jackson, J. P. Landman, J. St E. Hall, V. Cox, and C. Osmond. "Maternal nutritional status in pregnancy and blood pressure in childhood." BJOG: An International Journal of Obstetrics and Gynaecology 101, no. 5 (May 1994): 398–403. http://dx.doi.org/10.1111/j.1471-0528.1994.tb11911.x.

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Adamczak, Joanna, and Edward Wolf. "Maternal Blood Pressure Adaptation in the First Trimester of Pregnancy." American Journal of Perinatology 27, no. 04 (December 10, 2009): 339–42. http://dx.doi.org/10.1055/s-0029-1243305.

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Harder, Thomas, Kerstin Franke, Andreas Plagemann, and Rainer Kohlhoff. "Early nutrition and later blood pressure: Effect of maternal diabetes." Journal of Pediatrics 139, no. 6 (December 2001): 0905–6. http://dx.doi.org/10.1067/mpd.2001.119175.

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Thilaganathan, Baskaran. "Association of Higher Maternal Blood Pressure With Lower Infant Birthweight." Hypertension 67, no. 3 (March 2016): 499–500. http://dx.doi.org/10.1161/hypertensionaha.115.06880.

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Brans, Yves W., Thomas J. Kuehl, Robert H. Hayashi, and Patty Reyes. "Maternal Blood Pressure and Fetal Ultrasonography in Normal Baboon Pregnancies." Journal of Medical Primatology 19, no. 7 (September 1990): 641–49. http://dx.doi.org/10.1111/j.1600-0684.1990.tb00471.x.

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Roseboom, Tessa J., Jan H. P. van der Meulen, Gert A. van Montfrans, Anita C. J. Ravelli, Clive Osmond, David J. P. Barker, and Otto P. Bleker. "Maternal nutrition during gestation and blood pressure in later life." Journal of Hypertension 19, no. 1 (January 2001): 29–34. http://dx.doi.org/10.1097/00004872-200101000-00004.

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Lupton, Samantha J., Christine L. Chiu, Sanja Lujic, Annemarie Hennessy, and Joanne M. Lind. "Association between parity and breastfeeding with maternal high blood pressure." American Journal of Obstetrics and Gynecology 208, no. 6 (June 2013): 454.e1–454.e7. http://dx.doi.org/10.1016/j.ajog.2013.02.014.

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Dalton, Kevin J., Keith Manning, Philip J. Robarts, James H. Dripps, and Janet R. Currie. "Computerized home telemetry of maternal blood pressure in hypertensive pregnancy." International Journal of Bio-Medical Computing 21, no. 3-4 (November 1987): 175–87. http://dx.doi.org/10.1016/0020-7101(87)90085-7.

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McGarvey, S. T., S. H. Zinner, W. C. Willett, and B. Rosner. "Maternal prenatal dietary potassium, calcium, magnesium, and infant blood pressure." Hypertension 17, no. 2 (February 1991): 218–24. http://dx.doi.org/10.1161/01.hyp.17.2.218.

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