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Статті в журналах з теми "Lung welding"

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Автор: Grafiati

Опубліковано: 30 травня 2022

Оновлено: 31 травня 2022

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1

Oh, Jung-Hwa, Mi-Jin Yang, Jeong-Doo Heo, Young-Su Yang, Han-Jin Park, Se-Myo Park, Myung-Sang Kwon, Chang-Woo Song, Seokjoo Yoon, and Il Je Yu. "Inflammatory response in rat lungs with recurrent exposure to welding fumes." Toxicology and Industrial Health 28, no. 3 (July 5, 2011): 203–15. http://dx.doi.org/10.1177/0748233711410906.

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Анотація:

As chronic exposure to welding fumes causes pulmonary diseases, such as pneumoconiosis, public concern has increased regarding continued exposure to these hazardous gases in the workplace. In a previous study, the inflammatory response to welding fume exposure was analysed in rat lungs in the case of recurrent exposure and recovery periods. Thus using lung samples, well-annotated by histological observation and biochemical analysis, this study examines the gene expression profiles to identify phenotype-anchored genes corresponding to lung inflammation and the repair phenomenon after recurrent welding fume exposure. Seven genes ( Mmp12, Cd5l, LOC50101, LOC69183, Spp1, and Slc26a4) were found to be significantly up-regulated according to the severity of the lung injury. In addition, the transcription and translation of Trem2, which was up-regulated in response to the repair process, were validated using a real-time polymerase chain reaction, Western blotting, and immunohistochemistry. The differentially expressed genes in the exposure and recovery groups were also classified using k-means and hierarchical clustering, plus their toxicological function and canonical pathways were further analysed using Ingenuity Pathways Analysis Software. As a result, this comprehensive and integrative analysis of the transcriptional changes that occur during repeated exposure provides important information on the inflammation and repair processes after welding-fume-induced lung injury.

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2

Desy, Riesa, and Lilis Sulistyorini. "The Analysis of Exposure Welding Fumes with Impaired Lung Faal Workers Welding PT. PAL Indonesia (Persero)." JURNAL KESEHATAN LINGKUNGAN 9, no. 2 (July 27, 2018): 154. http://dx.doi.org/10.20473/jkl.v9i2.2017.154-162.

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Welding is an activity that has a major risk for lung function disorders resulting from gas and fumes generated. The general objective of this study was to analyze the overview of fumes exposure, individual characteristics with impaired lung function welding workers in PT. PAL INDONESIA (Persero). The type of this research is an observational study and is descriptive with cross sectional approach. The sample used the total population of the labor of welding PT. PAL INDONESIA (Persero). Measuring levels of fumes using HVDS, measurements were performed on a 4 point welding workshop. Examining of lung function is done by using a spirometer. And the risk factor data obtained from questionnaires and interviews. The results of this study showed levels of fumes that exceed 2 NAB contained in welding workshop From the results of pulmonary function measurement, there are three workers with impaired lung function by the category of mild obstruction and restriction. Characteristics of individuals who have an influence on the occurrence of pulmonary function disorders worker is of masks used by workers. Welding workers should wear masks that have been provided by PT. PAL INDONESIA. PT. PAL INDONESIA should undertake an obligation to conduct periodic health for workers.

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3

Meo, Sultan A., M. Abdul Azeem, and M. M. F. Subhan. "Lung Function in Pakistani Welding Workers." Journal of Occupational and Environmental Medicine 45, no. 10 (October 2003): 1068–73. http://dx.doi.org/10.1097/01.jom.0000085889.16029.6b.

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4

Pesch, Beate, Benjamin Kendzia, Hermann Pohlabeln, Wolfgang Ahrens, Heinz-Erich Wichmann, Jack Siemiatycki, Dirk Taeger, et al. "Exposure to Welding Fumes, Hexavalent Chromium, or Nickel and Risk of Lung Cancer." American Journal of Epidemiology 188, no. 11 (September 5, 2019): 1984–93. http://dx.doi.org/10.1093/aje/kwz187.

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Abstract To investigate the risk of lung cancer after exposure to welding fumes, hexavalent chromium (Cr(VI)), and nickel, we analyzed 3,418 lung cancer cases and 3,488 controls among men from 2 German case-control studies (1988–1996). We developed a welding-process exposure matrix from measurements of these agents, and this was linked with welding histories from a job-specific questionnaire to calculate cumulative exposure variables. Logistic regression models were fitted to estimate odds ratios with confidence intervals conditional on study, and they adjusted for age, smoking, and working in other at-risk occupations. Additionally, we mutually adjusted for the other exposure variables under study. Overall, 800 cases and 645 controls ever worked as regular or occasional welders. Odds ratios for lung cancer with high exposure were 1.55 (95% confidence interval (CI): 1.17, 2.05; median, 1.8 mg/m3 × years) for welding fumes, 1.85 (95% CI: 1.35, 2.54; median, 1.4 μg/m3 × years) for Cr(VI), and 1.60 (95% CI: 1.21, 2.12; median, 9 μg/m3 × years) for nickel. Risk estimates increased with increasing cumulative exposure to welding fumes and with increasing exposure duration for Cr(VI) and nickel. Our results showed that welding fumes, Cr(VI), and nickel might contribute independently to the excess lung cancer risk associated with welding. However, quantitative exposure assessment remains challenging.

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5

Riccelli, Maria Grazia, Matteo Goldoni, Diana Poli, Paola Mozzoni, Delia Cavallo, and Massimo Corradi. "Welding Fumes, a Risk Factor for Lung Diseases." International Journal of Environmental Research and Public Health 17, no. 7 (April 8, 2020): 2552. http://dx.doi.org/10.3390/ijerph17072552.

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Анотація:

(1) Background: Welding fumes (WFs) are composed of fine and ultrafine particles, which may reach the distal airways and represent a risk factor for respiratory diseases. (2) Methods: In vitro and in vivo studies to understand WFs pathogenesis were selected. Epidemiological studies, original articles, review, and meta-analysis to examine solely respiratory disease in welders were included. A systematic literature search, using PubMed, National Institute for Occupational Safety and Health Technical Information Center (NIOSHTIC), and Web of Science databases, was performed. (3) Results: Dose, time of exposure, and composition of WFs affect lung injury. Inflammation, lung defense suppression, oxidative stress, DNA damage, and genotoxic effects were observed after exposure both to mild and stainless steel WFs. (4) Conclusions: The detection of lung diseases associated with specific occupational exposure is crucial as complete avoidance or reduction of the exposure is difficult to achieve. Further studies in the area of particle research may aid the understanding of mechanisms involved in welding-related lung disease and to expand knowledge in welding-related cardiovascular diseases.

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6

Zhao, Jianan, Yu Feng, Marcio Bezerra, Jun Wang, and Ted Sperry. "Numerical simulation of welding fume lung dosimetry." Journal of Aerosol Science 135 (September 2019): 113–29. http://dx.doi.org/10.1016/j.jaerosci.2019.05.006.

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7

Honaryar, Manoj Kumar, Ruth M. Lunn, Danièle Luce, Wolfgang Ahrens, Andrea ’t Mannetje, Johnni Hansen, Liacine Bouaoun, et al. "Welding fumes and lung cancer: a meta-analysis of case-control and cohort studies." Occupational and Environmental Medicine 76, no. 6 (April 4, 2019): 422–31. http://dx.doi.org/10.1136/oemed-2018-105447.

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BackgroundAn estimated 110 million workers are exposed to welding fumes worldwide. Welding fumes are classified by the International Agency for Research on Cancer as carcinogenic to humans (group 1), based on sufficient evidence of lung cancer from epidemiological studies.ObjectiveTo conduct a meta-analysis of case-control and cohort studies on welding or exposure to welding fumes and risk of lung cancer, accounting for confounding by exposure to asbestos and tobacco smoking.MethodsThe literature was searched comprehensively in PubMed, reference lists of relevant publications and additional databases. Overlapping populations were removed. Meta-relative risks (mRRs) were calculated using random effects models. Publication bias was assessed using funnel plot, Eggers’s test and Begg’s test.ResultsForty-five studies met the inclusion criteria (20 case-control, 25 cohort/nested case-control), which reduced to 37 when overlapping study populations were removed. For ‘ever’ compared with ‘never’ being a welder or exposed to welding fumes, mRRs and 95% CIs were 1.29 (1.20 to 1.39; I2=26.4%; 22 studies) for cohort studies, 1.87 (1.53 to 2.29; I2=44.1%; 15 studies) for case-control studies and 1.17 (1.04 to 1.38; I2=41.2%) for 8 case-control studies that adjusted for smoking and asbestos exposure. The mRRs were 1.32 (95% CI 1.20 to 1.45; I2=6.3%; 15 studies) among ‘shipyard welders’, 1.44 (95% CI 1.07 to 1.95; I2=35.8%; 3 studies) for ‘mild steel welders’ and 1.38 (95% CI 0.89 to 2.13; I2=68.1%; 5 studies) among ‘stainless steel welders’. Increased risks persisted regardless of time period, geographic location, study design, occupational setting, exposure assessment method and histological subtype.ConclusionsThese results support the conclusion that exposure to welding fumes increases the risk of lung cancer, regardless of the type of steel welded, the welding method (arc vs gas welding) and independent of exposure to asbestos or tobacco smoking.

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8

Peters, Susan, Jerome Lavoue, Marissa Baker, and Hans Kromhout. "O2E.4 Evaluation of exposure assessment methods in epidemiological studies: the welding example." Occupational and Environmental Medicine 76, Suppl 1 (April 2019): A21.1—A21. http://dx.doi.org/10.1136/oem-2019-epi.55.

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Exposure assessment quality is a fundamental consideration in the design and evaluation of observational studies. High quality exposure assessment is particularly relevant for outcomes with long latency, such as cancer, where detailed information on past exposures are often missing and must therefore be estimated.For the IARC Monograph on welding, the exposure group provided an overview of assessment methods used in the key epidemiological studies. Strengths and weaknesses of each study were assessed, along with their potential effects on interpretation of risk estimates.For the association between lung cancer and welding fume exposure, 9 cohort and 10 case-control studies were reviewed. For ocular melanoma and ultraviolet radiation (UVR) from welding, 7 case-control studies were reviewed. Quality criteria were: full occupational histories, and standardized, blinded and quantitative exposure assessment. Additional criteria for lung cancer: specifically assessing welding fumes and using information on welding tasks. For ocular melanoma: assessing artificial and solar radiation separately, taking into account eye burns, eye protection and welding type.Exposure assessment of welding fumes by applying a ‘welding-exposure matrix’ (n=2) or welding-specific questionnaires (n=3) were considered highest quality, followed by case-by-case expert assessment (n=5) or general job-exposure matrices (JEMs, n=4). Job title alone was considered less informative (n=5). For exposure to UVR, JEMs were most informative (n=2), followed by self-reported eye burns and self-reported exposure from specific welding types (n=2), although caution is advised regarding recall bias. Assessing welding fume exposure or ever exposure to welding arcs as proxy for UVR was considered less informative. For both exposures, ever versus never welder, or assessments based on data collected from proxies, were considered least informative.The overall evaluation was that there is sufficient evidence in humans for the carcinogenicity of welding fumes and ultraviolet radiation from welding.

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9

Bleier, Benjamin S., Neri M. Cohen, Jason D. Bloom, James N. Palmer, and Noam A. Cohen. "Laser Tissue Welding in Lung and Tracheobronchial Repair." Chest 138, no. 2 (August 2010): 345–49. http://dx.doi.org/10.1378/chest.09-2721.

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10

Mulyana, Mulyana, Nuri Purwito Purwito Adi, Meily L. Kurniawidjaja, Andi Wijaya, and Irawan Yusuf. "Lung Function Status of Workers Exposed to Welding Fume: A Preliminary Study." Indonesian Biomedical Journal 8, no. 1 (April 1, 2016): 37. http://dx.doi.org/10.18585/inabj.v8i1.196.

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BACKGROUND: Exposure to welding fume in the workplace was associated with lung function disorders and occupational asthma. In this study, we determined lung function parameters in men workers exposed to welding fumes from heavy equipment manufacturer. This study is a preliminary study of biomonitoring program in worker exposed to welding fume as our main study. METHODS: A study with case-control design, random study, was conducted among welder (59 subjects) and non-welder (34 subjects) with more than one year experience in the same job task in a heavy equipment manufacturer. All subjects completed physical examination, informed consent, questionnaire and lung function status. Lung function status was measured by spirometer with vital capacity (VC), forced vital capacity (FCV), forced expiratory volume in one second (FEV1) and ratio of FEV1/FVC as test parameters. Linear regression model was developed to identify the risk factor of lung function parameter status using age, working period and smoking status as variables. RESULTS: This study showed that there were significant lower VC, FVC and FEV1 in welder than non-welder, but not difference in ratio of FEV1/FVC. However, there was no significant difference among welder from foundry and fabrication plan. By multivariate analysis, working period was found as a risk factor for lower parameters in lung function among welder. CONCLUSION: Lung function parameters status were significantly lower in welder than non-welder, and working period was the most important indicator for lung function status evaluation among welder. KEYWORDS: vital capacity, VC, forced vital capacity, FCV, forced expiratory volume in one second, FEV1, lung function, ratio of FEV1/FVC, working period

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11

Dauter, U. M., A. Alhamdow, A. Cediel-Ulloa, A. Gudmundsson, A. R. Gliga, M. Albin, and K. Broberg. "Occupational welding exposure and lung cancer-related epigenetic modifications." Toxicology Letters 350 (September 2021): S75. http://dx.doi.org/10.1016/s0378-4274(21)00422-7.

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12

Wahyudi, Bambang Adi, Duwi Leksono Edy, and Wiyono Wiyono. "Automation of Welding Fumes based on MQ-2 Sensor in Welding Laboratory." Jurnal Temapela 3, no. 1 (November 9, 2020): 1–9. http://dx.doi.org/10.25077/temapela.3.1.1-9.2020.

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In practical welding activities, there are potential hazards that must be addressed. These potential hazards include exposure to toxic fumes and dust. Fumes in the welding process contain a lot of metal oxides and can cause chronic effects in the form of eye irritation, sensory irritation, and impaired lung physiological function. Therefore, an air intake sistem is needed to minimize the volume of fumes in the welding chamber . This study aims to design an automation of exhaust smoke using the sensor module, microcontroller and actuator. The resulting tool is equipped with a display of detected air quality conditions, and an alarm indicating that the air quality is in a dangerous level. The method used is experimental development with 3 main stages, namely: designing hardware and software, testing the tools and analyzing their work functions. The results obtained are that the MQ-2 sensor is able to detect changes in the concentration of welding smoke and can be used as an analog input to control the work of the indicator lights, alarms and exhaust fans. The average ability of the exhaust fan to absorb the smoke concentration in the welding chamber is 26.85 seconds for 33 ppm at 40 mm long welding and 29.51 seconds for 37 ppm at 80 mm long welding process. With this capability, this tool can be used as a technical solution related to Occupational Health and Safety in welding laboratories.

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13

Ambroise, Denis, Pascal Wild, and Jean-Jacques Moulin. "Update of a meta-analysis on lung cancer and welding." Scandinavian Journal of Work, Environment & Health 32, no. 1 (February 2006): 22–31. http://dx.doi.org/10.5271/sjweh.973.

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14

Brammertz, Astrid. "Welders and Operators of Welding Automatons - Prevention of Lung Disease." Chest 126, no. 4 (October 2004): 767S. http://dx.doi.org/10.1378/chest.126.4_meetingabstracts.767s.

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15

Matrat, Mireille, Florence Guida, Francesca Mattei, Sylvie Cénée, Diane Cyr, Joëlle Févotte, Marie Sanchez, et al. "Welding, a risk factor of lung cancer: the ICARE study." Occupational and Environmental Medicine 73, no. 4 (February 10, 2016): 254–61. http://dx.doi.org/10.1136/oemed-2015-102964.

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16

Linchevskyy, Oleksandr, Anatoliy Makarov, and Vadym Getman. "Lung sealing using the tissue-welding technology in spontaneous pneumothorax☆☆☆." European Journal of Cardio-Thoracic Surgery 37, no. 5 (May 2010): 1126–28. http://dx.doi.org/10.1016/j.ejcts.2009.11.017.

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17

McCarrick, Sarah, Hanna L. Karlsson, and Ulrika Carlander. "Modelled lung deposition and retention of welding fume particles in occupational scenarios: a comparison to doses used in vitro." Archives of Toxicology 96, no. 4 (February 21, 2022): 969–85. http://dx.doi.org/10.1007/s00204-022-03247-9.

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AbstractTranslating particle dose from in vitro systems to relevant human exposure remains a major challenge for the use of in vitro studies in assessing occupational hazard and risk of particle exposure. This study aimed to model the lung deposition and retention of welding fume particles following occupational scenarios and subsequently compare the lung doses to those used in vitro. We reviewed published welding fume concentrations and size distributions to identify input values simulating real-life exposure scenarios in the multiple path particle dosimetry (MPPD) model. The majority of the particles were reported to be below 0.1 μm and mass concentrations ranged between 0.05 and 45 mg/m3. Following 6-h exposure to 5 mg/m3 with a count median diameter of 50 nm, the tracheobronchial lung dose (0.89 µg/cm2) was found to exceed the in vitro cytotoxic cell dose (0.125 µg/cm2) previously assessed by us in human bronchial epithelial cells (HBEC-3kt). However, the tracheobronchial retention decreased rapidly when no exposure occurred, in contrast to the alveolar retention which builds-up over time and exceeded the in vitro cytotoxic cell dose after 1.5 working week. After 1 year, the tracheobronchial and alveolar retention was estimated to be 1.15 and 2.85 µg/cm2, respectively. Exposure to low-end aerosol concentrations resulted in alveolar retention comparable to cytotoxic in vitro dose in HBEC-3kt after 15–20 years of welding. This study demonstrates the potential of combining real-life exposure data with particle deposition modelling to improve the understanding of in vitro concentrations in the context of human occupational exposure.

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18

Sørensen, Anita Rath, Ane Marie Thulstrup, Johnni Hansen, Cecilia Høst Ramlau-Hansen, Andrea Meersohn, Axel Skytthe, and Jens Peter Bonde. "Risk of lung cancer according to mild steel and stainless steel welding." Scandinavian Journal of Work, Environment & Health 33, no. 5 (October 2007): 379–86. http://dx.doi.org/10.5271/sjweh.1157.

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19

Siew, Sie Sie, Timo Kauppinen, Pentti Kyyrönen, Pirjo Heikkilä, and Eero Pukkala. "Exposure to iron and welding fumes and the risk of lung cancer." Scandinavian Journal of Work, Environment & Health 34, no. 6 (December 2008): 444–50. http://dx.doi.org/10.5271/sjweh.1296.

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20

Thaon, Isabelle, Valérie Demange, Fabrice Herin, Annie Touranchet, and Christophe Paris. "Increased Lung Function Decline in Blue-collar Workers Exposed to Welding Fumes." Chest 142, no. 1 (July 2012): 192–99. http://dx.doi.org/10.1378/chest.11-0647.

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21

Kendzia, Benjamin, Thomas Behrens, Karl-Heinz Jöckel, Jack Siemiatycki, Hans Kromhout, Roel Vermeulen, Susan Peters, et al. "Welding and Lung Cancer in a Pooled Analysis of Case-Control Studies." American Journal of Epidemiology 178, no. 10 (September 19, 2013): 1513–25. http://dx.doi.org/10.1093/aje/kwt201.

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22

Sjogren, B., K. S. Hansen, H. Kjuus, and P. G. Persson. "Exposure to stainless steel welding fumes and lung cancer: a meta-analysis." Occupational and Environmental Medicine 51, no. 5 (May 1, 1994): 335–36. http://dx.doi.org/10.1136/oem.51.5.335.

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23

Dev, Manish, and Arvind Bhardwaj. "Respiratory symptoms and spirometric abnormalities among welders in the welding workplace of the Indian unorganized sector." Work 69, no. 3 (July 16, 2021): 885–94. http://dx.doi.org/10.3233/wor-213521.

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BACKGROUND: Welding is one of the most hazardous professions across the globe. Several risk factors are associated with Indian unorganized welding units such as welding gases, fumes and dust particles resulting in various respiratory health problems. OBJECTIVE: The objective of the present study is to examine the prevalence of respiratory symptoms, their associated factors and lung function impairment amongst the welders of the unorganized sector in India. METHODS: A cross-sectional study was conducted across 283 welders of age group 18–55 years from Punjab, India who responded to the interviewer-administrated respiratory symptoms questionnaire. Also, the lung function parameters of 50 male welders (exposed group) and 50 male non-welders (control group) working in the same environment were evaluated. RESULTS: The prevalence of respiratory symptoms such as chronic cough (38.86%), phlegm (38.86%), shortness of breath (33.56%), wheezing (32.15%), chest tightness (36.40%,) and sputum (34.27%) was reported by the welders. The observed mean values of forced vital capacity (FVC), forced expiratory volume in 1s (FEV1) and FEV1/FVC ratio of welders were (3.37±0.175), (2.59±0.16) and (76.63±6.16) respectively and that of non-welders were (3.70±0.15), (3.05±0.25) and (82.49±7.62) respectively. CONCLUSIONS: The welders were found to be at significant risk of developing pulmonary diseases. Long working hours, low level of education, hazardous working conditions, lack of implementation of safety laws make welders more vulnerable to health risks. The welders of Indian unorganized sector work without any technical training related to welding and safety. Proper orientation sessions about the workplace hazards and to maintain hygiene at their workplace should be organized. Welders must be provided with Personal Protective Equipment (PPE). Welders should take proper health check-up and medication to sustain healthiness

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24

Brand, Peter, Monika Gube, Kerstin Gerards, Jens Bertram, Heinz Kaminski, Astrid Christiane John, Thomas Kuhlbusch, et al. "Internal Exposure, Effect Monitoring, and Lung Function in Welders After Acute Short-Term Exposure to Welding Fumes From Different Welding Processes." Journal of Occupational and Environmental Medicine 52, no. 9 (September 2010): 887–92. http://dx.doi.org/10.1097/jom.0b013e3181f09077.

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25

Khmel, O. V., I. A. Kalabukha, Y. N. Maetnyi, V. E. Ivaschenko, Y. M. Voloshin, and R. A. Veremeenko. "USE OF LIVE TISSUE WELDING TECHNOLOGY IN LUNG RESECTION IN PATIENTS WITH TUBERCULOSIS." Ukrainian Pulmonology Journal 103, no. 1 (2019): 41–43. http://dx.doi.org/10.31215/2306-4927-2019-103-1-41-43.

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26

J�ckel, Karl-Heinz, Wolfgang Ahrens, Hermann Pohlabeln, Ulrich Bolm-Audorff, and Klaus Michael M�ller. "Lung cancer risk and welding: Results from a case-control study in Germany." American Journal of Industrial Medicine 33, no. 4 (April 1998): 313–20. http://dx.doi.org/10.1002/(sici)1097-0274(199804)33:4<313::aid-ajim1>3.0.co;2-v.

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27

't Mannetje, A., P. Brennan, D. Zaridze, N. Szeszenia-Dabrowska, P. Rudnai, J. Lissowska, E. Fabianova, et al. "Welding and Lung Cancer in Central and Eastern Europe and the United Kingdom." American Journal of Epidemiology 175, no. 7 (February 16, 2012): 706–14. http://dx.doi.org/10.1093/aje/kwr358.

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28

James M. Antonini, Nicholas J. Lawr. "EFFECT OF WELDING FUME SOLUBILITY ON LUNG MACROPHAGE VIABILITY AND FUNCTION IN VITRO." Journal of Toxicology and Environmental Health, Part A 58, no. 6 (November 1999): 343–63. http://dx.doi.org/10.1080/009841099157205.

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29

Zeidler-Erdely, Patti C., Terence G. Meighan, Aaron Erdely, Lori A. Battelli, Michael L. Kashon, Michael Keane, and James M. Antonini. "Lung tumor promotion by chromium-containing welding particulate matter in a mouse model." Particle and Fibre Toxicology 10, no. 1 (2013): 45. http://dx.doi.org/10.1186/1743-8977-10-45.

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30

Christensen, Sigve W., Jens Bonde, and Øyvind Omland. "A prospective study of decline in lung function in relation to welding emissions." Journal of Occupational Medicine and Toxicology 3, no. 1 (2008): 6. http://dx.doi.org/10.1186/1745-6673-3-6.

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31

Szram, J., S. Schofield, M. Cosgrove, and P. Cullinan. "Welding, lung function decline and respiratory symptoms: a systematic review of cohort studies." Occupational and Environmental Medicine 68, Suppl_1 (September 1, 2011): A7. http://dx.doi.org/10.1136/oemed-2011-100382.21.

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Jöckel, Karl-Heinz, Wolfgang Ahrens, and Ulrich Bolm-Audorff. "Lung Cancer Risk and Welding—Preliminary Results From an Ongoing Case-Control Study." American Journal of Industrial Medicine 25, no. 6 (June 1994): 805–12. http://dx.doi.org/10.1002/ajim.4700250605.

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Heo, Jeong-Doo, Jung-Hwa Oh, Kyuhong Lee, Chang-Woo Song, Seokjoo Yoon, and Jin Soo Han. "Gene expression profiling in the lung from cynomolgus monkeys repeatedly exposed to welding fume." Toxicology Letters 189 (September 2009): S93—S94. http://dx.doi.org/10.1016/j.toxlet.2009.06.282.

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Gliga, Anda R., Tahir Taj, Maria Hedmer, Eva Assarsson, Lars Rylander, Maria Albin, and Karin Broberg. "Mild steel welding is associated with alterations in circulating levels of cancer-related proteins." Archives of Toxicology 93, no. 12 (October 22, 2019): 3535–47. http://dx.doi.org/10.1007/s00204-019-02594-4.

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Abstract Welding fumes were recently classified as carcinogenic to humans and worldwide millions work as welders or perform welding operations. The purpose of this study was to identify new biomarkers of welding-induced carcinogenesis. We evaluated a panel of 91 putative cancer-related proteins in serum in a cohort of welders working with mild steel (n = 77) and controls (n = 94) from southern Sweden sampled on two occasions 6-year apart using a longitudinal analysis (linear mixed models). The significant results from the longitudinal analysis were tested for reproducibility in welders (n = 88) and controls (n = 69) sampled once during the same sampling period as timepoint 1 or timepoint 2 (linear regression models), i.e., in a cross-sectional setting. The models were adjusted for age, body-mass index, and use of snus. All study participants were non-smokers at recruitment. Exposure to welding fumes was assessed using questionnaires and respirable dust measurement in the breathing zone that was adjusted for personal respiratory protection equipment. The median respirable dust in welders was 0.7 (0.2–4.2) and 0.5 (0.1–1.9) mg/m3 at the first and second timepoints, respectively. We identified 14 cancer-related proteins that were differentially expressed in welders versus controls in the longitudinal analysis, out of which three were also differentially expressed in the cross-sectional analysis (cross-sectional group). Namely, syndecan 1 (SDC1), folate receptor 1 (FOLR1), and secreted protein acidic and cysteine rich (SPARC) were downregulated, in welders compared with controls. In addition, FOLR1 was negatively associated with years welding. Disease and function analysis indicated that the top proteins are related to lung cancer as well as cell invasion and migration. Our study indicates that moderate exposure to welding fumes is associated with changes in circulating levels of putative cancer-related proteins, out of which FOLR1 showed a clear dose–response relationship. It is, however, unclear to which extent these changes are adaptive or potential early biomarkers of cancer.

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35

ANTONINI, J. "Suppression in lung defense responses after bacterial infection in rats pretreated with different welding fumes." Toxicology and Applied Pharmacology 200, no. 3 (November 2004): 206–18. http://dx.doi.org/10.1016/j.taap.2004.04.022.

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36

Szram, Joanna, Susie J. Schofield, Martin P. Cosgrove, and Paul Cullinan. "Welding, longitudinal lung function decline and chronic respiratory symptoms: a systematic review of cohort studies." European Respiratory Journal 42, no. 5 (December 20, 2012): 1186–93. http://dx.doi.org/10.1183/09031936.00206011.

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Antonini, James M., and Jenny R. Roberts. "Chromium in Stainless Steel Welding Fume Suppresses Lung Defense Responses Against Bacterial Infection in Rats." Journal of Immunotoxicology 4, no. 2 (January 2007): 117–27. http://dx.doi.org/10.1080/15476910701336953.

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38

Anttila, S. "Dissolution of stainless steel welding fumes in the rat lung: an x ray microanalytical study." Occupational and Environmental Medicine 43, no. 9 (September 1, 1986): 592–96. http://dx.doi.org/10.1136/oem.43.9.592.

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39

Babanov, SA A., and DS S. Budash. "CYTOKINE PROFILE AND IMPORTANCE OF ITS ASSESSMENT IN FORECASTING THE COURSE OF DUST DISEASES OF THE LUNGS." Science and Innovations in Medicine 2, no. 1 (March 15, 2017): 61–64. http://dx.doi.org/10.35693/2500-1388-2017-0-1-61-64.

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Aim - study of cellular immunity and cytokine profile in dust lung diseases caused by exposure to aerosols of various degrees of fibrogenity; assessment of the role of immunological changes in the pathogenesis of occupational lung diseases of dust aetiology. Materials and methods. 161 patients from the contact group with chronic dust bronchitis, silicosis and pneumoconiosis due to exposure to welding aerosols were examined. The control group included 60 people who did not have contact with occupational hazards at work and who were declared healthy according to complete physical examination. The diagnosis was made on the basis of professional anamnesis, sanitary-hygienic characteristics of working conditions (work in contact with aerosols of various degrees of fibrogenity), and a complete clinical and instrumental examination of patients. The levels of cytokines IL-1a, IL-ф, IL-4, IL-8, TNF-a, IFN-y and fibronectin in the blood serum were determined using enzyme-linked immunosorbent assay. Results. The specific features of the course of dust diseases of the lungs were revealed (identified features of the cytokine profile, hypercytokinaemia of a significant number of cytokines), which allows us to characterize the occurrence and the progression of dust diseases of the lungs. Conclusion. Determination of the cytokine profile allows us not only to improve the quality of early detection, but also to optimize the strategy of primary and secondary prophylaxis of this disease, to predict the course of the disease, and to reduce the number of disabling forms.

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Zeidler-Erdely, Patti C., Lori A. Battelli, Sam Stone, Bean T. Chen, David G. Frazer, Shih-Houng Young, Aaron Erdely, Michael L. Kashon, Ronnee Andrews, and James M. Antonini. "Short-term inhalation of stainless steel welding fume causes sustained lung toxicity but no tumorigenesis in lung tumor susceptible A/J mice." Inhalation Toxicology 23, no. 2 (February 2011): 112–20. http://dx.doi.org/10.3109/08958378.2010.548838.

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41

Tagurum, Yetunde O., Martin D. Gwomson, Pankyes M. Yakubu, James A. Igbita, Moses P. Chingle, and Oluwabunmi O. Chirdan. "Awareness of occupational hazards and utilization of PPE amongst welders in Jos metropolis, Nigeria." International Journal of Research in Medical Sciences 6, no. 7 (June 25, 2018): 2227. http://dx.doi.org/10.18203/2320-6012.ijrms20182808.

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Background: Welding poses a range of both well-known and subtle hazards to health and safety. These hazards can act quickly or may show up only in the long term. They can be rapidly fatal (electric shock or exposure to cadmium fumes) or have delayed effects (lung changes over time). This study aimed to assess the awareness of occupational hazards and utilization of PPE amongst welders in Jos metropolis.Methods: This was a descriptive, cross-sectional study involving 295 welders in Jos metropolis. An interviewer-administered questionnaire was used to collect data which was entered and analyzed using Epi-info version 3.5.4 statistical software. A probability value of p≤0.05 was considered statistically significant.Results: All the respondents were males with a mean age of 24.6±7.7 years. The study revealed that 293 (99.3%) were aware of occupational hazards in welding. In this study, goggles were the most frequently used PPE 98%, then gloves 65.4%, boots 58%, overalls 36.3%, facemask 30.6% and earplugs 12.9%. A statistically significant (p≤0.05) relationship was found between employment pattern as well as working hours per day and the use of safety devices.Conclusions: The study showed that most of the welders had fair knowledge of welding related health problems, hazards and safety devices, and utilization of safety devices was less than optimal. An educational campaign on workplace hazards, types and proper use of different personal protective devices should be instituted for welders on a regular basis by the welders association, local and state government.

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Gomes, J., C. Guerreiro, D. Lavrador, P. A. Carvalho, and R. M. Miranda. "TEM analysis as a tool for toxicological assessment of occupational exposure to airborne nanoparticles from welding." Microscopy and Microanalysis 19, S4 (August 2013): 153–54. http://dx.doi.org/10.1017/s1431927613001384.

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Welding is the principal industrial process used for joining metals. However, it can produce dangerous fumes that may be hazardous to the welder’s health and it is estimated that, presently, 1-2% of workers from different professional backgrounds (which accounts for more than 3 million persons) are subjected to welding fume and gas action. With the advent of new types of welding procedures and consumables, the number of welders exposed to welding fumes is growing constantly in spite of the mechanization and automation of the processes. Simultaneously, the number of publications on epidemiologic studies and the devices for welders’ protection is also increasing. Apart from that, the influence of very ultrafine particulate, lying in the nanoparticles range, on human health has been pointed to be of much concern as airborne nanoparticles are resulting both from nanotechnologies processes and also from macroscopic common industrial processes such as welding. In fact, nanotoxicological research is still in its infancy and the issuing and implementation of standards for appropriate safety control systems can still take several years. Yet, the advanced understanding of toxicological phenomena on the nanometre scale is largely dependent on technological innovations and scientific results stemming from enhanced R&D. Meanwhile, the industry has to adopt proactive risk management strategies in order to provide a safe working environment for their staff, clients and customers, and obtain products without posing health threats at any point of their lifecycle. Understanding the relationship of airborne nano sized particulate and human health, under different environmental conditions is of great importance for improving exposure estimates and for developing efficient control strategies to reduce human exposure and health risk and for establishing, evaluating and improving regulations and legislation both on air quality, airborne emissions and the incorporation of nano sized materials in other products and commodities.Mass measurement methods are not sufficiently sensitive for airborne nanoparticles and are not sensitive toward the specific health relevant properties of nanoparticles. The most sensitive concentration measured in this particle range (> 100 nm diameter) is the number concentration. In fact, surface area is a relevant metric for nanoparticles, as most of the processes in the human body environment take place via the particle surface, which is increasing significantly with decreasing particle size in the nanometer size range for the same amount of mass. In order to perform the toxicological assessment of welding processes a Nanoparticle Surface Area Monitor, TSI, Model 3550, based on diffusion charging (measuring the electrostatic charge on a sampled aerosol, mainly composed of nanoparticles) was used for monitoring the emission of nanoparticles resulting from several welding processes. This equipment indicates the human lung-deposited surface area of particles expressed as square micrometers per cubic centimeter of air (µm2/cm3). Although this instrument is very precise and its use has been validated for this purpose, the definite presence of nanoparticles in welding fume has to be complemented by microscopy techniques such as transmission electron microscopy (TEM), which has proved very helpful in order to establish the size, shape and aggregation habit of sampled aerosols, as well as energy dispersive X-ray spectroscopy (EDS) for performing the chemical analysis of collected nanoparticles. Figure 1 shows the aspect of aggregates of particles, in the nano size range, collected from welding fume during shielded metal arc welding (SMAW) of carbon steel and friction stir welding (FSW) of aluminium alloys, respectively. As expected in arc welding aggregates of larger dimensions are produced in more roundish shaped particles while in FSW these are finer. Chemical analysis determined by EDS showed that the emitted nanoparticles of SMAW are mostly resulting from burning of electrode coating and less from its core. Therefore, in order to reduce particulate emissions, special care has to be paid to the chemical composition of the electrodes coating.The work was supported by the Portuguese Science Foundation through PEst-OE/EQB/LA0023/2011, PEst-OE/EME/UI0667/2011 and PEst-OE/CTM-UI0084/2011 and grants.

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43

Sung, J. H. "Recovery from Welding-Fume-Exposure-Induced Lung Fibrosis and Pulmonary Function Changes in Sprague Dawley Rats." Toxicological Sciences 82, no. 2 (December 1, 2004): 608–13. http://dx.doi.org/10.1093/toxsci/kfh289.

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Falcone, L. M., A. Erdely, V. Kodali, R. Salmen, L. A. Battelli, T. Dodd, W. McKinney, et al. "Inhalation of iron-abundant gas metal arc welding-mild steel fume promotes lung tumors in mice." Toxicology 409 (November 2018): 24–32. http://dx.doi.org/10.1016/j.tox.2018.07.007.

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45

Zeidler-Erdely, Patti C., Lauryn M. Falcone, and James M. Antonini. "Influence of welding fume metal composition on lung toxicity and tumor formation in experimental animal models." Journal of Occupational and Environmental Hygiene 16, no. 6 (April 1, 2019): 372–77. http://dx.doi.org/10.1080/15459624.2019.1587172.

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46

Falcone, Lauryn M., Aaron Erdely, Terence G. Meighan, Lori A. Battelli, Rebecca Salmen, Walter McKinney, Samuel Stone, et al. "Inhalation of gas metal arc–stainless steel welding fume promotes lung tumorigenesis in A/J mice." Archives of Toxicology 91, no. 8 (January 4, 2017): 2953–62. http://dx.doi.org/10.1007/s00204-016-1909-2.

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47

Karataş, M., TN Öziş, M. Büyükşekerci, M. Gündüzöz, OG Özakıncı, G. Gök, S. Neşelioğlu, and Ö. Erel. "Thiol-disulfide homeostasis and ischemia-modified albumin levels as indicators of oxidative stress in welders’ lung disease." Human & Experimental Toxicology 38, no. 11 (August 26, 2019): 1227–34. http://dx.doi.org/10.1177/0960327119871093.

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Welders’ lung disease refers to mixed exposure to different kinds of metals and chemicals from welding fumes, which affect all parts of the respiratory tract including airways and parenchyma together. This study aimed to investigate the oxidative status in patients with welders’ lung (PWL) by means of thiol-disulfide homeostasis and ischemia-modified albumin (IMA) levels. The male welder workers diagnosed with welders’ lung disease and healthy individuals were recruited in the study. Plasma levels of disulfide, disulfide/native thiol ratio, disulfide/total thiol ratio, IMA, and catalase (CAT) were determined. Pulmonary function test parameters of both groups were compared. The thiol-disulfide homeostasis parameters of PWL and control group were as follows: disulfide (20.5 ± 6.3 vs. 16.2 ± 3.9 μmol L−1, p < 0.001), disulfide/native thiol (4.36 (1.59) vs. 4.0 (1.64), p = 0.024), and disulfide/total thiol (4.01 (1.34) vs. 3.71 (1.41), p = 0.024). IMA levels in PWL were significantly higher than the control group (1.37 (0.27) mg dL−1 vs. 0.49 (0.61) mg dL−1, p < 0.001), whereas CAT activities were significantly higher in the control group (106.6 (54.5) kU L−1 vs. 78.3 (67.8) kU L−1, p = 0.003). The findings of the present study revealed that oxidative stress plays a key role in the pathogenesis of welders’ lung disease. Plasma thiol-disulfide homeostasis and IMA levels might be indicators of oxidative stress in PWL.

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Bilokon, Oleg V., Elen V. Shaida, Petro P. Sokur, and Borys O. Kravchuk. "New Approaches to the Surgical Treatment of Lung and Bronchial Tumors in Children." International Journal of Biology and Biomedical Engineering 15 (July 30, 2021): 311–17. http://dx.doi.org/10.46300/91011.2021.15.37.

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Today, it is relevant to search for new, better methods of surgical treatment of tumors and organ-preserving and video-assisted surgery is gaining more and more popularity. The aim of the study was to improve the results of surgical treatment of children with primary and secondary (metastatic) lung tumors using high-frequency live tissue welding (HF LTW) in order to increase the treatment effectiveness. Novelty of the study lies in the expanding the knowledge of the new researched method, for enhancement of survivability relatively to chemotherapy and radiation methods, that are studied in the previous works. Advantages of the introduced method are bloodless, fast, low traumatic operations 103 case reports in children with primary and metastatic lung tumors were analyzed, including 34 patients with benign and malignant lung tumors and 69 children with metastatic bronchial and pulmonary lesions undergoing treatment from 2002 to 2018 were examined. Benign lung tumors were diagnosed in 17 patients. Malignant tumors were observed in 17 patients, including 11 lung carcinoid tumors. Metastatic bronchial and pulmonary lesions most often occurred with osteosarcoma (28) and nephroblastoma (17). In 34 children with primary tumors, 37 were undergone surgical interventions. Most often, atypical resection was used – 16 (43.3%), in particular, with carcinoid – in 7 (43.8%) patients and with hamartoma – in 4 (25%) patients. In metastatic lesions, in most cases, the same resection was performed. From 58 resections 25 (43%) were performed for osteosarcoma and 15 (26%) for nephroblastoma. The HF LTW method was used in 29 patients for benign and malignant primary tumors and in 62 (73%) surgical procedures for lung metastases to remove a tumor, vascular coagulation and sealing of the lung tissue. Using HF LTW surgical operations of different directions and volumes can be performed effectively in benign, malignant and metastatic bronchial and pulmonary lesions in children.

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Yu, Il Je, Kyung Seuk Song, Hee Kyung Chang, Jeong Hee Han, Yong Hyun Chung, Kuy Tae Han, Kyu Hyuck Chung, and Ho Keun Chung. "Recovery from manual metal arc-stainless steel welding-fume exposure induced lung fibrosis in Sprague–Dawley rats." Toxicology Letters 143, no. 3 (August 2003): 247–59. http://dx.doi.org/10.1016/s0378-4274(03)00154-1.

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Antonini, James M., Robert W. Clarke, G. G. Krishna Murthy, Pradeep Sreekanthan, Neil Jenkins, Thomas W. Eagar, and Joseph D. Brain. "Freshly generated stainless steel welding fume induces greater lung inflammation in rats as compared to aged fume." Toxicology Letters 98, no. 1-2 (September 1998): 77–86. http://dx.doi.org/10.1016/s0378-4274(98)00103-9.

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