Дисертації з теми "Lsm Domain"
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Tuladhar, Kapil. "Lim-only domain proteins in developmental haematopoiesis." Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:d6b73e89-7095-402f-9d9f-4d7837a4db00.
Повний текст джерелаPinto, Belinda Sophia Geyer Pamela Kent. "Understanding the role of LEM domain proteins in Drosophila development." [Iowa City, Iowa] : University of Iowa, 2009. http://ir.uiowa.edu/etd/421.
Повний текст джерелаJurata, Linda Wagner. "Identification and analysis of the nuclear LIM domain interactor NLI /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 1998. http://wwwlib.umi.com/cr/ucsd/fullcit?p9904815.
Повний текст джерелаPinto, Belinda Sophia. "Understanding the role of LEM domain proteins in Drosophila development." Diss., University of Iowa, 2009. https://ir.uiowa.edu/etd/421.
Повний текст джерелаDiefenbacher, Markus Elmar. "The transcriptional co-activator function of the LIM-domain protein nTrip6." Eggenstein-Leopoldshafen Forschungszentrum Karlsruhe GmbH, 2010. http://d-nb.info/1002907535/34.
Повний текст джерелаNisevic, Ivan. "Detection and analysis of LIM domain-mediated interactions between transcription factors." Thesis, The University of Sydney, 2015. http://hdl.handle.net/2123/15711.
Повний текст джерелаGu, Wenchao. "Exploring the roles of LIM domain binding proteins in zebrafish development." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:54f520f6-170a-480a-a195-1a0739055031.
Повний текст джерелаKlaavuniemi, T. (Tuula). "PDZ-LIM domain proteins and α-actinin at the muscle Z-disk". Doctoral thesis, University of Oulu, 2006. http://urn.fi/urn:isbn:9514282647.
Повний текст джерелаDuan, Jie. "Active Control of Vehicle Powertrain Noise Applying Frequency Domain Filtered-x LMS Algorithm." University of Cincinnati / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1243614246.
Повний текст джерелаKhurana, Bharat. "Characterization of DLIM1, a novel cytoskeleton-associated LIM domain containing protein of Dictyostelium discoideum." [S.l. : s.n.], 2000. http://deposit.ddb.de/cgi-bin/dokserv?idn=961945737.
Повний текст джерелаDiefenbacher, Markus Elmar [Verfasser]. "The transcriptional co-activator function of the LIM-domain protein nTrip6 / Markus Elmar Diefenbacher." Eggenstein-Leopoldshafen : Forschungszentrum Karlsruhe GmbH, 2010. http://d-nb.info/1002907535/34.
Повний текст джерелаProeschel, Christoph Johann Wolfgang. "The cloning and characterisation of Lnk-1 : a novel LIM-domain containing protein kinase." Thesis, University College London (University of London), 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.294778.
Повний текст джерелаRobertson, Neil. "Development and application of simple FRET-based methods for aggregation-prone LIM domain interactions." Thesis, The University of Sydney, 2017. http://hdl.handle.net/2123/16912.
Повний текст джерелаBajic, Vladan. "DESIGN AND IMPLEMENTATION OF AN ADAPTIVE NOISE CANCELING SYSTEM IN WAVELET TRANSFORM DOMAIN." University of Akron / OhioLINK, 2005. http://rave.ohiolink.edu/etdc/view?acc_num=akron1132784671.
Повний текст джерелаEssawy, Nada. "Characterization of emerin LEM-domain missense mutations present in patients with exclusive atrial cardiac defects." Electronic Thesis or Diss., Sorbonne université, 2018. http://www.theses.fr/2018SORUS299.
Повний текст джерелаEmery-Dreifuss Muscular Dystrophy (EDMD) is among the most widely common human genetic muscular dystrophies. The cardiac involvement in the disease is the most life-threatening symptom and the major cause of mortality. The majority of cases of its X-linked type are due to mutations in a gene encoding for the nuclear envelope protein, emerin. Despite the considerable advances that have been achieved in terms of the characterization of emerin structure, various binding partners, and functions in the human body, the picture is still rather incomplete. Fifty years now after EDMD had been first documented, researchers still fall short of understanding the pathophysiology of the disease. Thereby, it comes as no surprise that, to date, there is no described treatment of EDMD. Accordingly, this thesis is an initial attempt to characterize three emerin LEM-domain missense mutations (P22L, ΔK37, and T43I) present in patients with exclusive cardiac defects. The main objective of this thesis is to investigate the effect of the three mutations on: emerin structure, its self-assembly, and interactions with some of its well-described binding partners. The presented work highlights that albeit the localization of the three mutations in the only folded region of emerin, the variants show no global defect in their structure, except for the destabilization of the LEM-domain of the variant ΔK37. Importantly, the mutants are able to self-assemble, yet with astonishing fast polymerization kinetics. In addition, the investigations have illustrated that the three variants, despite the presence of the mutations in the BAF-binding domain, are surprisingly capable of binding BAF. The analysis did not reveal any differences in the mutants binding to Ig-fold domain of lamin A/C. Further, there is no defect in ΔK37 phosphorylation by Src kinase. Also, preliminary characterization of the ΔK37 mutation in immortalized human fibroblasts has featured no overt defects in mechanobiology, and in the expression of nuclear envelope or cytoskeletal proteins. Taken all together, the presented work outlines that the three emerin missense mutations display no defects in several prominent emerin properties, which are questioned in this thesis. On the basis of the results of the conducted research, considerable insight has been gained with regard to the consequences of the mutations of interest. In other words, the presented work lends support to following investigations in order to explore other unquestioned properties or functions of emerin that might be associated with the pathophysiology of EDMD
Salpingidou, Georgia. "The role of emerin and LEM domain proteins in nuclear envelope assembly and cytoskeleton organisation." Thesis, Durham University, 2005. http://etheses.dur.ac.uk/2203/.
Повний текст джерелаBarton, Lacy Jo. "Defining the role of the nuclear lamina LEM Domain protein Otefin in germline stem cells." Diss., University of Iowa, 2014. https://ir.uiowa.edu/etd/2043.
Повний текст джерелаGuruswamy, Aarumugam Bhupathi Rajan. "Independent Domain of Symmetric Encryption using Least SignificantBit : Computer Vision, Steganography and Cryptography Techniques." Thesis, Högskolan Dalarna, Datateknik, 2011. http://urn.kb.se/resolve?urn=urn:nbn:se:du-10063.
Повний текст джерелаAnantharaman, B. "Compressed Domain Processing of MPEG Audio." Thesis, Indian Institute of Science, 2001. https://etd.iisc.ac.in/handle/2005/3914.
Повний текст джерелаAnantharaman, B. "Compressed Domain Processing of MPEG Audio." Thesis, Indian Institute of Science, 2001. http://hdl.handle.net/2005/68.
Повний текст джерелаDiefenbacher, Markus Elmar [Verfasser], and A. [Akademischer Betreuer] Cato. "The transcriptional co-activator function of the LIM-domain protein nTrip6 / Markus Elmar Diefenbacher. Betreuer: A. Cato." Karlsruhe : KIT-Bibliothek, 2009. http://d-nb.info/1014222877/34.
Повний текст джерелаEssawy, Nada [Verfasser]. "Characterization of emerin LEM-domain missense mutations present in patients with exclusive atrial cardiac defects / Nada Essawy." Berlin : Freie Universität Berlin, 2019. http://d-nb.info/1179277864/34.
Повний текст джерелаArnaud, Dominique. "Identification et caractérisation de la famille de protéines à domaine LIM chez le peuplier." Orléans, 2007. http://www.theses.fr/2007ORLE2035.
Повний текст джерелаBossenz, Michael. "In-vivo-Analyse des LIM-Domäne-bindenden Kofaktors RLIM in Vertebraten." [S.l. : s.n.], 2004. http://deposit.ddb.de/cgi-bin/dokserv?idn=971998973.
Повний текст джерелаWang, Hui. "The Roles of a LIM Domain Protein, Hic-5/ARA55, in TGF-β Signaling in Prostate Cancer Cells". Case Western Reserve University School of Graduate Studies / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=case1220931692.
Повний текст джерелаBanthien, Nils [Verfasser]. "The four-and-a-half-LIM-domain Protein FHL2 is a novel regulator of pulmonary fibrosis / Nils Banthien." Gießen : Universitätsbibliothek, 2020. http://d-nb.info/1216142955/34.
Повний текст джерелаEmond, Julien. "Contribution à l'étude des structures passives verre-silicium dans le domaine millimétrique." Phd thesis, Université Paris-Est, 2010. http://tel.archives-ouvertes.fr/tel-00600678.
Повний текст джерелаNahar, Taslima [Verfasser], and Markus [Akademischer Betreuer] Hecker. "Role of the lim-domain proteins LPP and ZYXIN in hypertension-induced cardiovascular remodeling / Taslima Nahar ; Betreuer: Markus Hecker." Heidelberg : Universitätsbibliothek Heidelberg, 2017. http://d-nb.info/1178010651/34.
Повний текст джерелаTerano, Tomo. "Transcriptional control of fetal liver hematopoiesis : dominant negative effect of the overexpression of the LIM domain mutants of LMO2." Kyoto University, 2005. http://hdl.handle.net/2433/144782.
Повний текст джерелаRaskin, Anna Maria. "Four and half LIM Domain-1 protein and its role in passive mechanics and hypertrophic signaling of the heart." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC campuses, 2008. http://wwwlib.umi.com/cr/ucsd/fullcit?p3320727.
Повний текст джерелаTitle from first page of PDF file (viewed October 3, 2008). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
Nguyen, Ngoc Hung, Majid Dowlatnia, and Azhar Sarfraz. "Implementation of the LMS and NLMS algorithms for Acoustic Echo Cancellationin teleconference systemusing MATLAB." Thesis, Växjö University, School of Mathematics and Systems Engineering, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:vxu:diva-6368.
Повний текст джерелаIn hands-free telephony and in teleconference systems, the main aim is to provide agood free voice quality when two or more people communicate from different places.The problem often arises during the conversation is the creation of acoustic echo. Thisproblem will cause the bad quality of voice signal and thus talkers could not hearclearly the content of the conversation, even thought lost the important information.This acoustic echo is actually the noise which is created by the reflection of soundwaves by the wall of the room and the other things exist in the room. The mainobjective for engineers is the cancellation of this acoustic echo and provides an echofree environment for speakers during conversation. For this purpose, scientists designdifferent adaptive filter algorithms. Our thesis is also to study and simulate theacoustics echo cancellation by using different adaptive algorithms.
König, Katharina [Verfasser]. "The role of Four-and-a-half LIM domain protein 2 in dendritic cell migration / Katharina König. Mathematisch-Naturwissenschaftliche Fakultät." Bonn : Universitäts- und Landesbibliothek Bonn, 2011. http://d-nb.info/1018829962/34.
Повний текст джерелаMUNDEL, CHRISTOPHE. "Etude de wlim-1, une proteine a domaines lim de tournesol (helianthus annuus l. )." Université Louis Pasteur (Strasbourg) (1971-2008), 1999. http://www.theses.fr/1999STR13024.
Повний текст джерелаBaron, Kyla Doreen. "The Role of LMO4 in the Regulation of SLK Localization & Activation within Migrating Cells and in Murine Mammary Tumorigenesis." Thesis, Université d'Ottawa / University of Ottawa, 2016. http://hdl.handle.net/10393/34195.
Повний текст джерелаBridge, Katherine S. "Regulation of HIF-l and the hypoxic response by the tumour suppressor LIMDl and LIM domains-containing proteins." Thesis, University of Nottingham, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.606229.
Повний текст джерелаMedves, Sandrine. "Effets de Tes (Testine), une protéine à domaine LIM, sur la structure et la dynamique du cytosquelette d'actine." Université Louis Pasteur (Strasbourg) (1971-2008), 2008. http://www.theses.fr/2008STR13174.
Повний текст джерелаTränka, Christopher [Verfasser], Elke [Gutachter] Butt, and Ralf [Gutachter] Bargou. "Untersuchungen zur Expression und Funktion des „LIM and SH3 Domain Proteins“ (LASP-1) in Medulloblastomen / Christopher Tränka ; Gutachter: Elke Butt, Ralf Bargou." Würzburg : Universität Würzburg, 2016. http://d-nb.info/1116363542/34.
Повний текст джерелаTränka, Christopher [Verfasser], Elke Gutachter] Butt, and Ralf C. [Gutachter] [Bargou. "Untersuchungen zur Expression und Funktion des „LIM and SH3 Domain Proteins“ (LASP-1) in Medulloblastomen / Christopher Tränka ; Gutachter: Elke Butt, Ralf Bargou." Würzburg : Universität Würzburg, 2016. http://nbn-resolving.de/urn:nbn:de:bvb:20-opus-139539.
Повний текст джерелаNeghabian, Dariusch [Verfasser]. "Rolle des four and-a-half LIM domain 1 (FHL-1) Proteins bei der Entstehung der Rechtsherzhypertrophie im Mausmodell des pulmonalarteriellen bandings / Dariusch Neghabian." Gießen : Universitätsbibliothek, 2019. http://d-nb.info/120035253X/34.
Повний текст джерелаBordel, Anne-Claire. "Les protéines à domaines LIM chez le protoplaste de tournesol (Helianthus annuus L.) : expression des gènes et cytolocalisation." Phd thesis, Institut National Polytechnique de Toulouse - INPT, 2000. http://tel.archives-ouvertes.fr/tel-00012190.
Повний текст джерелаNous avons étudié l'expression des gènes LIM précédemment décrits chez le tournesol dans les protoplastes par RT-PCR. Nous avons détecté un transcrit pour le gène HaWLIM-1, mais pas pour les deux autres gènes HaPLIM-1 et HaPLIM-2. Des anticorps polyclonaux spécifiques de la protéine HaWLIM-1 reconnaissent en immunoblot deux polypeptides distincts, dont les masses moléculaires sont respectivement de 52 kDa et 78 kDa. Ces masses moléculaires sont nettement supérieures à la taille attendue pour la protéine HaWLIM-1. Ces résultats indiquent que la protéine n'est pas présente sous forme de monomères dans les protoplastes, mais qu'elle participe à la formation de complexes protéiques stables.
L'étude par immunocytologie de la localisation intracellulaire de la protéine HaWLIM-1 révèle que cette protéine est présente simultanément dans deux compartiments distincts : le noyau et le cytoplasme. Dans le noyau, elle s'accumule préférentiellement dans le nucléole, et pourrait jouer un rôle dans la régulation de la transcription des gènes des ARNr, ou dans l'assemblage des ribosomes. Dans le cytoplasme, différentes approches, incluant des expériences de double-marquage et de déstructuration de composants du cytosquelette, ont permis de mettre en évidence une très forte colocalisation de la protéine HaWLIM-1 avec les microtubules, ce qui suggère un rôle pour cette protéine dans l'organisation du cytosquelette.
Lors de la culture des protoplastes, le gène HaWLIM-1 s'exprime constamment, avec cependant des variations dans le niveau d'expression. Des expériences d'immunoblot utilisant les anticorps spécifiques de la protéine HaWLIM-1 indiquent que de nouveaux polypeptides, de masses moléculaires égales à 35 kDa, 42 kDa et 64 kDa, apparaissent au cours de la culture. Cette observation suggère que la protéine HaWLIM-1 possède la capacité de s'associer et de se dissocier avec de nouveaux complexes protéiques au cours du développement. La protéine HaWLIM-1 est associée aux microtubules pendant tous les stades de la division : elle est présente au niveau de la bande préprophasique à la fin de l'interphase, au niveau du fuseau mitotique pendant la mitose, et au niveau du phragmoplaste pendant la cytokinèse. Ces observations semblent indiquer que la protéine HaWLIM-1 occupe une fonction importante au niveau des microtubules.
GASS, NATHALIE. "Etude de la fonction physiologique et moleculaire d'une proteine a domaines lim, haplim1, specifiquement exprimee dans le pollen." Université Louis Pasteur (Strasbourg) (1971-2008), 2000. http://www.theses.fr/2000STR13134.
Повний текст джерелаDietz, Carola. "Expression der Tyrosinkinase Focal Adhesion Kinase (FAK) und des Four and a Half LIM Domain 2 Proteins (FHL2) im Zervixkarzinom und deren Korrelation mit klinischen Parametern /." Freiburg i.Br, 2008. http://opac.nebis.ch/cgi-bin/showAbstract.pl?sys=000254224.
Повний текст джерелаPašalić, Zlatana. "The four and a half LIM domain protein 2 (FHL2) interacts with CALM and is highly expressed in acute myeloid leukemia (AML) with complex aberrant karyotypes." kostenfrei, 2008. http://edoc.ub.uni-muenchen.de/10582/.
Повний текст джерелаPasalic, Zlatana. "The four and a half LIM domain protein 2 (FHL2) interacts with CALM and is highly expressed in acute myeloid leukemia (AML) with complex aberrant karyotypes." Diss., lmu, 2008. http://nbn-resolving.de/urn:nbn:de:bvb:19-105825.
Повний текст джерелаSchnittger, Josef [Verfasser]. "Characterization of a novel interaction between four-and-a-half-LIM domains 2 and cardiomyopathy-associated protein 5 in cardiac myocytes / Josef Schnittger." Hamburg : Staats- und Universitätsbibliothek Hamburg Carl von Ossietzky, 2019. http://nbn-resolving.de/urn:nbn:de:gbv:18-ediss-87981.
Повний текст джерелаTholl, Stéphane. "Etude biochimique comparative des "Actin Depolymerizing Factors"(ADFs) d'Arabidopsis : activité inattendue de pontage des filaments d'actine pour les ADFs appartenant à la sous-classe III." Thesis, Strasbourg, 2012. http://www.theses.fr/2012STRAJ002.
Повний текст джерелаActin cytoskeleton organization and dynamics are tightly regulated by many actin-binding proteins (ABPs). Among ABPs, the actin-depolymerizing factors (ADFs) play a major role in actin filament turnover by promoting actin filament severing and facilitating pointed end depolymerization. Arabidopsis thaliana has 11 functional proteins that can be classified into four subclasses according to their expression profile and phylogenetic relationships. We provide evidence that subclass III ADF5 and ADF9 are unconventional ADFs since they do not display typical actin filament depolymerizing activities. Instead, they exhibit opposite activities with a surprisingly high ability to stabilize and crosslink actin filaments into long and thick actin bundles both in vitro and in live cells. Competition experiments with ADF1 support that ADF9 antagonizes the depolymerizing activity of conventional ADFs. We report the characterization of a not yet described knockout Arabidopsis mutant. Data strongly suggests a role for ADF9 in cell elongation. Indeed, hypocotyls are significantly longer in adf9 mutant than in wild- type seedlings, and this phenotype is enhanced in dark growth conditions in which the ADF9 gene is normally preferentially expressed. The analysis of hypocotyl epidermal cells indicates that this phenotype is essentially due to an increase of cell expansion. Surprisingly, adf9 seedlings exhibit shorter roots than control plants, suggesting a complex link between actin cytoskeleton organization and cell elongation. Finally, the reduced ability of adf9- derived calli to proliferate supports a role for ADF9 in cell division as well
Gupta, Shuchi [Verfasser], and Bernhard [Akademischer Betreuer] Nieswandt. "The role of the Canonical transient receptor potential 6 (TRPC6) channel and the C terminal LIM domain protein of 36 kDa (CLP36) for platelet function / Shuchi Gupta. Betreuer: Bernhard Nieswandt." Würzburg : Universitätsbibliothek der Universität Würzburg, 2013. http://d-nb.info/1043906622/34.
Повний текст джерелаBALTZ, RACHEL. "Etude structurale et fonctionnelle d'une proteine specifique de pollen a domaines lim et de son gene chez le tournesol (helianthus annuus l. )." Université Louis Pasteur (Strasbourg) (1971-2008), 1994. http://www.theses.fr/1994STR13178.
Повний текст джерелаSklar, Alexander Gabriel. "Channel Modeling Applied to Robust Automatic Speech Recognition." Scholarly Repository, 2007. http://scholarlyrepository.miami.edu/oa_theses/87.
Повний текст джерелаGole, Shirish Gajanan. "Understanding the Role of Lsm Domain in Translation Repression Activity of RGG-motif Containing Protein Scd6." Thesis, 2017. http://etd.iisc.ac.in/handle/2005/4286.
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