Готові списки джерел за темами / Lachnospiraceae

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Добірка наукової літератури з теми "Lachnospiraceae"

Автор: Grafiati

Опубліковано: 23 вересня 2022

Оновлено: 28 січня 2023

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Статті в журналах з теми "Lachnospiraceae"

Vacca, Mirco, Giuseppe Celano, Francesco Maria Calabrese, Piero Portincasa, Marco Gobbetti, and Maria De Angelis. "The Controversial Role of Human Gut Lachnospiraceae." Microorganisms 8, no. 4 (April 15, 2020): 573. http://dx.doi.org/10.3390/microorganisms8040573.

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The complex polymicrobial composition of human gut microbiota plays a key role in health and disease. Lachnospiraceae belong to the core of gut microbiota, colonizing the intestinal lumen from birth and increasing, in terms of species richness and their relative abundances during the host’s life. Although, members of Lachnospiraceae are among the main producers of short-chain fatty acids, different taxa of Lachnospiraceae are also associated with different intra- and extraintestinal diseases. Their impact on the host physiology is often inconsistent across different studies. Here, we discuss changes in Lachnospiraceae abundances according to health and disease. With the aim of harnessing Lachnospiraceae to promote human health, we also analyze how nutrients from the host diet can influence their growth and how their metabolites can, in turn, influence host physiology.

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Kitagami, Y., N. Kanzaki, and Y. Matsuda. "First report of segmented filamentous bacteria associated with Rhigonema sp. (Nematoda: Rhigonematidae) dwelling in hindgut of Riukiaria sp. (Diplopoda: Xystodesmidae)." Helminthologia 56, no. 3 (September 1, 2019): 219–28. http://dx.doi.org/10.2478/helm-2019-0018.

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SummaryWe morphologically and molecularly characterized segmented filamentous bacteria (SFB) associated with Rhigonema sp. nematodes in millipede hindguts. Seventy-three Riukiaria sp. millipedes were collected from a broad-leaf forest in Japan, and nematodes were excised from the millipede’s hindguts. The occurrence rate of SFB associated with nematodes was 24 % (10/41) for males, 47 % (14/30) for females, and 100 % (2/2) for juveniles. Genomic DNA was extracted from four SFB-rich nematode heads, and we obtained 40 bacterial clones via analysis of nearly full-length 16S rDNA gene sequences. At the phylum level, Firmicutes, Proteobacteria, and Verrucomicrobia accounted for 55 %, 40 %, and 5 % of SFB, respectively. In Firmicutes, Clostridiaceae (28 %) and Lachnospiraceae (15 %) were the dominant groups. Our sequences were divided into seven and three subclades between Firmicutes and Proteobacteria in the phylogenetic tree. In the Firmicutes clade, eight sequences were classified as Lachnospiraceae with a bootstrap value >83 %. A phylogenetic tree involving known uncultured Lachnospiraceae sequences characterized the phylogenetic position of SFB associated with nematodes. Our results suggest that the association of SFB with nematode bodies was probably incidental and that SFB are not always present in millipede hindguts. Our bacterial groups corresponded to those of arthropod hindgut, and SFB associated with nematodes were inferred to belong to Lachnospiraceae. Because the Lachnospiraceae sequences obtained in this study showed specific lineages that differed from all the known deposited sequence data, these groups may be unique to Riukiaria sp.

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Guo, Yawei, Xiaohui Zhu, Sha Zeng, Mingyi He, Xiurong Xing, and Changyuan Wang. "miRNA-10a-5p Alleviates Insulin Resistance and Maintains Diurnal Patterns of Triglycerides and Gut Microbiota in High-Fat Diet-Fed Mice." Mediators of Inflammation 2020 (August 17, 2020): 1–8. http://dx.doi.org/10.1155/2020/8192187.

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miRNA-10a is rhythmically expressed and regulates genes involved in lipid and glucose metabolism. However, the effects of miRNA-10a on obesity and glucose intolerance, as well as on the diurnal pattern of expression of circadian clock genes, remain unknown. We explored the effects of miRNA-10a-5p on insulin resistance and on the diurnal patterns of serum triglycerides and gut microbiota in high-fat diet- (HFD-) fed mice. The results showed that oral administration of miRNA-10a-5p significantly prevented body weight gain and improved glucose tolerance and insulin sensitivity in HFD-fed mice. Administration of miRNA-10a-5p also maintained the diurnal rhythm of Clock, Per2, and Cry1 expression, as well as serum glucose and triglyceride levels. Surprisingly, the diurnal oscillations of three genera of microbes, Oscillospira, Ruminococcus, and Lachnospiraceae, disrupted by HFD feeding, maintained by administration of miRNA-10a-5p. Moreover, a strong positive correlation was found between hepatic Clock expression and relative abundance of Lachnospiraceae, both in control mice (r=0.877) and in mice administered miRNA-10a-5p (r=0.853). Furthermore, we found that along with changes in Lachnospiraceae abundance, butyrate content in the feces maintained a diurnal rhythm after miRNA-10a-5p administration in HFD-fed mice. In conclusion, we suggest that miRNA-10a-5p may improve HFD-induced glucose intolerance and insulin resistance through the modulation of the diurnal rhythm of Lachnospiraceae and its metabolite butyrate. Therefore, miRNA-10a-5p may have preventative properties in subjects with metabolic disorders.

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Tran, Mai H., Hajeung Park, Christopher L. Nobles, Pabalu Karunadharma, Li Pan, Guocai Zhong, Haimin Wang, et al. "A more efficient CRISPR-Cas12a variant derived from Lachnospiraceae bacterium MA2020." Molecular Therapy - Nucleic Acids 24 (June 2021): 40–53. http://dx.doi.org/10.1016/j.omtn.2021.02.012.

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Juhász, János, Balázs Ligeti, Márió Gajdács, Nóra Makra, Eszter Ostorházi, Ferenc Balázs Farkas, Balázs Stercz, et al. "Colonization Dynamics of Multidrug-Resistant Klebsiella pneumoniae Are Dictated by Microbiota-Cluster Group Behavior over Individual Antibiotic Susceptibility: A Metataxonomic Analysis." Antibiotics 10, no. 3 (March 7, 2021): 268. http://dx.doi.org/10.3390/antibiotics10030268.

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Gastrointestinal carriage of multidrug-resistant (MDR) bacteria is one of the main risk factors for developing serious, difficult-to-treat infections. Given that there is currently no all-round solution to eliminate colonization with MDR bacteria, it is particularly important to understand the dynamic process of colonization to aid the development of novel decolonization strategies. The aim of our present study was to perform metataxonomic analyses of gut microbiota dynamics during colonization with an extended-spectrum β-lactamase (ESBL)- and carbapenemase-producing Klebsiella pneumoniae (ECKP) strain in mice; additionally, to ascertain the effects of antibiotic administration (ampicillin, ceftazidime, and ciprofloxacin) on the establishment and elimination of ECKP intestinal colonization. We have found that the phyla Bacteroidetes and Firmicutes were most dominant in all of the treatment groups; however, Bacteroidetes was more common in the groups treated with antibiotics compared to the control group. Significant differences were observed among the different antibiotic-treated groups in beta but not alpha diversity, implying that the difference is the relative abundance of some bacterial community members. Bacteria from the Lachnospiraceae family (including Agathobacter, Anaerostipes, Lachnoclostridium 11308, Lachnospiraceae UCG-004, Lachnospiraceae NK3A20 group 11318, Lachnospiraceae NK4A136 group 11319, Roseburia, and Tyzzerella) showed an inverse relationship with the carriage rate of the ECKP strain, whereas members of Enterobacteriaceae and the ECKP strain have shown a correlational relationship. Our results suggest that the composition of the microbial community plays a primary role in the MDR-colonization rate, whereas the antibiotic susceptibility of individual MDR strains affects this process to a lesser extent. Distinct bacterial families have associated into microbial clusters, collecting taxonomically close species to produce survival benefits in the gut. These associations do not develop at random, as they may be attributed to the presence of specific metabolomic networks. A new concept should be introduced in designing future endeavors for MDR decolonization, supplemented by knowledge of the composition of the host bacterial community and the identification of bacterial clusters capable of suppressing or enhancing the invader species.

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Carlier, Jean-Philippe, Guylène K'ouas, Isabelle Bonne, Alain Lozniewski, and Francine Mory. "Oribacterium sinus gen. nov., sp. nov., within the family ‘Lachnospiraceae’ (phylum Firmicutes)." International Journal of Systematic and Evolutionary Microbiology 54, no. 5 (September 1, 2004): 1611–15. http://dx.doi.org/10.1099/ijs.0.63060-0.

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A hitherto unknown anaerobic bacillus isolated from sinus pus in a young child (strain AIP 354.02T) was characterized by using phenotypic and genotypic methods. 16S rRNA gene sequence analysis indicated that this strain was phylogenetically affiliated with several sequences of cloned 16S rRNA gene inserts previously deposited in the public databases. According to their 16S rRNA gene sequence similarities, these uncultivated bacteria, together with strain AIP 354.02T, formed a separate subgroup belonging to the family ‘Lachnospiraceae’ within the phylum Firmicutes. Oribacterium gen. nov. is proposed for this group of organisms and Oribacterium sinus gen. nov. sp. nov. for strain AIP 354.02T (=CIP 107991T=CCUG 48084T).

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Park, Miey, Jihee Choi, and Hae-Jeung Lee. "Flavonoid-Rich Orange Juice Intake and Altered Gut Microbiome in Young Adults with Depressive Symptom: A Randomized Controlled Study." Nutrients 12, no. 6 (June 18, 2020): 1815. http://dx.doi.org/10.3390/nu12061815.

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Depression is not just a general mental health problem but a serious medical illness that can worsen without treatment. The gut microbiome plays a major role in the two-way communication system between the intestines and brain. The current study examined the effects of flavonoids on depression by observing the changes in the gut microbiome and depressive symptoms of young participants consuming flavonoid-rich orange juice. The depressive symptom was assessed using the Center for Epidemiological Studies Depression Scale (CES-D), a psychiatric screening tool used to detect preexisting mental disorders. The study population was randomly divided into two groups: the flavonoid-rich orange juice (FR) and an equicaloric flavonoid-low orange cordial (FL) group. For 8 weeks, participants consumed FR (serving a daily 380 mL, 600 ± 5.4 mg flavonoids) or FL (serving a daily 380 mL, 108 ± 2.6 mg flavonoids). In total, 80 fecal samples from 40 participants (mean age, 21.83 years) were sequenced. Regarding depression, we observed positive correlations between brain-derived neurotrophic factor (BDNF) and the Lachnospiraceae family (Lachnospiraceae_uc and Murimonas) before flavonoid orange juice treatment. Most notably, the abundance of the Lachnospiraceae family (Lachnospiraceae_uc, Eubacterium_g4, Roseburia_uc, Coprococcus_g2_uc, Agathobacter_uc) increased after FR treatment compared to that after FL treatment. We also validated the presence of unclassified Lachnospiraceae through sensitive real-time quantitative polymerase chain reaction using stool samples from participants before and after flavonoid treatment. Our results provide novel interventional evidence that alteration in the microbiome due to flavonoid treatment is related to a potential improvement in depression in young adults.

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Dou, Xiujing, Nan Gao, Di Yan, and Anshan Shan. "Sodium Butyrate Alleviates Mouse Colitis by Regulating Gut Microbiota Dysbiosis." Animals 10, no. 7 (July 7, 2020): 1154. http://dx.doi.org/10.3390/ani10071154.

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Inflammatory bowel disease (IBD) develops as a result of complicated interactions between genetic susceptibility, excessive innate immunity, and environmental factors, which are mainly related to the gut microbiota. The present study aimed to elucidate the protective effects and underlying mechanisms of a short-chain fatty acid salt, sodium butyrate, on colonic inflammation induced by dextran sulfate sodium (DSS) in mice. Pretreatment with sodium butyrate attenuated colitis, as demonstrated by the decreased disease activity index (DAI), colon length shortening, spleen tumidness, and histopathology scores, while maintaining intestinal barrier integrity, as observed by H&E staining and electron microscopy. 16S rRNA sequence analysis revealed that sodium butyrate caused a remarkable alteration of the gut microbiota. Bacteroides, Lachnospiraceae, the Lachnospiraceae NK4A136 group, and Ruminiclostridium 6 presented dramatic differences after sodium butyrate supplementation. This work verifies that sodium butyrate can improve mouse colitis via microbe–host interactions by regulating the microbial community. Taken together, the findings demonstrate that sodium butyrate shows great potential as a probiotic agent for ameliorating colitis.

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Companys, Judit, Maria José Gosalbes, Laura Pla-Pagà, Lorena Calderón-Pérez, Elisabet Llauradó, Anna Pedret, Rosa Maria Valls, et al. "Gut Microbiota Profile and Its Association with Clinical Variables and Dietary Intake in Overweight/Obese and Lean Subjects: A Cross-Sectional Study." Nutrients 13, no. 6 (June 13, 2021): 2032. http://dx.doi.org/10.3390/nu13062032.

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We aimed to differentiate gut microbiota composition of overweight/obese and lean subjects and to determine its association with clinical variables and dietary intake. A cross-sectional study was performed with 96 overweight/obese subjects and 32 lean subjects. Anthropometric parameters were positively associated with Collinsella aerofaciens, Dorea formicigenerans and Dorea longicatena, which had higher abundance the overweight/obese subjects. Moreover, different genera of Lachnospiraceae were negatively associated with body fat, LDL and total cholesterol. Saturated fatty acids (SFAs) were negatively associated with the genus Intestinimonas, a biomarker of the overweight/obese group, whereas SFAs were positively associated with Roseburia, a biomarker for the lean group. In conclusion, Dorea formicigenerans, Dorea longicatena and Collinsella aerofaciens could be considered obesity biomarkers, Lachnospiraceae is associated with lipid cardiovascular risk factors. SFAs exhibited opposite association profiles with butyrate-producing bacteria depending on the BMI. Thus, the relationship between diet and microbiota opens new tools for the management of obesity.

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Amir, Itay, Philippe Bouvet, Christine Legeay, Uri Gophna, and Abraham Weinberger. "Eisenbergiella tayi gen. nov., sp. nov., isolated from human blood." International Journal of Systematic and Evolutionary Microbiology 64, Pt_3 (March 1, 2014): 907–14. http://dx.doi.org/10.1099/ijs.0.057331-0.

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A catalase-positive, rod-shaped, non-proteolytic, non-motile, anaerobic bacterial strain, designated B086562T, was isolated from a blood culture of an 84-year-old male patient in Israel. According to 16S rRNA gene sequence phylogeny, this strain has no known close relatives among recognized bacteria but should be placed within the family Lachnospiraceae . The most closely related recognized bacteria were from the ‘ Clostridium clostridioforme group’: C. clostridioforme (92.4 %) and Clostridium bolteae (92.3 %). The isolate produced butyrate, lactate, acetate and succinate as major metabolic end products. The major fatty acids were C16 : 0 and C18 : 1 cis 9 DMA and the DNA G+C content was 46.0 mol%. On the basis of the phenotypic properties and phylogenetic distinctiveness, the blood isolate represents a novel species of a new genus in the family Lachnospiraceae , for which the name Eisenbergiella tayi gen. nov., sp. nov. is proposed. The type strain of Eisenbergiella tayi is B086562T ( = LMG 27400T = DSM 26961T = ATCC BAA-2558T).

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Більше джерел

Дисертації з теми "Lachnospiraceae"

Wang, Yazhou. "Influence of Dectin-1 and Dectin-2 receptors on the susceptibility to gut inflammation." Thesis, Sorbonne université, 2021. http://www.theses.fr/2021SORUS523.

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Une dysbiose du microbiote intestinal a été identifiée comme impliquée dans la pathogenèse des maladies inflammatoires chroniques de l'intestin (MICI). Il a été démontré que les interactions hôte-bactéries affectent le développement des MICI, tandis que le rôle des interactions hôte-champignons dans les MICI sont peu décrites. Les lectines de type C sont des récepteurs impliqués dans la reconnaissance de motifs du mycobiote et font les réponses immunitaires face aux agents pathogènes fongiques. Nous avons évalué l'impact de la déficience des récepteurs Dectin-1 (D-1KO), Dectin-2 (D-2KO) et de la double déficience (D-1/2KO) dans l'inflammation intestinale. L’absence de D-1 ou D-2 n’a pas modifié la gravité de l'inflammation intestinale, tandis que les souris D-1/2KO étaient résistantes à la colite. Le rôle protecteur de D-1/2KO a été confirmé chez des souris WT ayant reçu le microbiote intestinal desD-1/2KO, suggérant que la protection était largement due au microbiote. L'analyse du microbiote des souris D-1/2KO montre que le microbiote bactérien, et en particulier la famille des Lachnospiraceae, mais pas le mycobiote, présentait une forte modification par rapport aux souris WT. Une supplémentation en Blautia hansenii a pu également assurer la protection, appuyant que cette protection était largement médiée par le microbiote bactérien. Ces résultats montrent que la déficience D-1/2 protége les souris de la colite via une modulation du microbiote intestinal bactérien
Gut microbiota dysbiosis has been identified as being involved in the pathogenesis of inflammatory bowel disease (IBD). Host-bacterial interactions have been shown to affect the development of IBD, while the role of host-fungal interactions in IBD is poorly described. C-type lectins are receptors involved in the recognition of patterns of the mycobiota and shape the immune responses to fungal pathogens. We evaluated the impact of the deficiency of the receptors Dectin-1 (D-1KO), Dectin-2 (D-2KO) and dual deficiency (D-1/2KO) in intestinal inflammation. The absence of D-1 or D-2 did not alter the severity of intestinal inflammation, whereas D-1/2KO mice were resistant to colitis. The protective role of D-1/2KO was confirmed in WT mice receiving the gut microbiota of D-1/2KO by fecal transfer, suggesting that protection was largely due to the gut microbiota. Analysis of the microbiota of D-1/2KO mice shows that the bacterial microbiota, and in particular the Lachnospiraceae family, but not the mycobiota, showed a strong change compared to the microbiota of the WT mice. Blautia hansenii supplementation was also able to provide protection, supporting that this protection was largely mediated by the bacterial microbiota and not the fungal microbiota. These results show that D-1/2KO deficiency protect mice from DSS-induced colitis via modulation of the bacterial gut microbiota

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Meehan, Conor J., and R. G. Beiko. "A phylogenomic view of ecological specialization in the Lachnospiraceae, a family of digestive tract-associated bacteria." 2014. http://hdl.handle.net/10454/17256.

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Yes
Several bacterial families are known to be highly abundant within the human microbiome, but their ecological roles and evolutionary histories have yet to be investigated in depth. One such family, Lachnospiraceae (phylum Firmicutes, class Clostridia) is abundant in the digestive tracts of many mammals and relatively rare elsewhere. Members of this family have been linked to obesity and protection from colon cancer in humans, mainly due to the association of many species within the group with the production of butyric acid, a substance that is important for both microbial and host epithelial cell growth. We examined the genomes of 30 Lachnospiraceae isolates to better understand the origin of butyric acid capabilities and other ecological adaptations within this group. Butyric acid production-related genes were detected in fewer than half of the examined genomes with the distribution of this function likely arising in part from lateral gene transfer (LGT). An investigation of environment-specific functional signatures indicated that human gut-associated Lachnospiraceae possess genes for endospore formation, whereas other members of this family lack key sporulation-associated genes, an observation supported by analysis of metagenomes from the human gut, oral cavity, and bovine rumen. Our analysis demonstrates that adaptation to an ecological niche and acquisition of defining functional roles within a microbiome can arise through a combination of both habitat-specific gene loss and LGT.
Canadian Institute for Health Research (grant number CMF-108026), Genome Atlantic and the Canada Research Chairs program to R.G.B.

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Частини книг з теми "Lachnospiraceae"

Stackebrandt, Erko. "The Family Lachnospiraceae." In The Prokaryotes, 197–201. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-30120-9_363.

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Cotta, Michael, and Robert Forster. "The Family Lachnospiraceae, Including the Genera Butyrivibrio, Lachnospira and Roseburia." In The Prokaryotes, 1002–21. New York, NY: Springer US, 2006. http://dx.doi.org/10.1007/0-387-30744-3_35.

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Hu, Li, Shaoping Zhu, Xiaoping Peng, Kanglan Li, Wanjuan Peng, Yu Zhong, Chenyao Kang, Xingxing Cao, Zhou Liu, and Bin Zhao. "High Salt Elicits Brain Inflammation and Cognitive Dysfunction, Accompanied by Alternations in the Gut Microbiota and Decreased SCFA Production." In Advances in Alzheimer’s Disease. IOS Press, 2022. http://dx.doi.org/10.3233/aiad220029.

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Background: Excessive salt intake is considered as an important risk factor for cognitive impairment, which might be the consequence of imbalanced intestinal homeostasis. Objective: To investigate the effects of dietary salt on the gut microbiota and cognitive performance and the underlying mechanisms. Methods: Adult female C57BL/6 mice were maintained on either normal chow (control group, CON) or sodium-rich chow containing 8% NaCl (high-salt diet, HSD) for 8 weeks. Spatial learning and memory ability, short-chain fatty acids (SCFAs) concentrations, gut bacterial flora composition, blood-brain barrier permeability, and proinflammatory cytokine levels and apoptosis in the brain were evaluated. Results: The mice fed a HSD for 8 weeks displayed impaired learning and memory abilities. HSD significantly reduced the proportions of Bacteroidetes (S24-7 and Alloprevotella) and Proteobacteria and increased that of Firmicutes (Lachnospiraceae and Ruminococcaceae). SCFA concentrations decreased in the absolute concentrations of acetate, propionate, and butyrate in the fecal samples from the HSD-fed mice. The HSD induced both BBB dysfunction and microglial activation in the mouse brain, and increased the IL-1β, IL-6, and TNF-α expression levels in the cortex. More importantly, the degree of apoptosis was higher in the cortex and hippocampus region of mice fed the HSD, and this effect was accompanied by significantly higher expression of cleaved caspase-3, caspase-3, and caspase-1. Conclusion: The HSD directly causes cognitive dysfunction in mice by eliciting an inflammatory environment and triggering apoptosis in the brain, and these effects are accompanied by gut dysbiosis, particularly reduced SCFA production.

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Тези доповідей конференцій з теми "Lachnospiraceae"

Silverman, Gregg J., Doua F. Azzouz, Brad H. Rovin, Roberto Caricchio, Jill P. Buyon, and Alexander Alekseyenko. "AI-06 Lupus nephritis is linked to dysbiosis, increased gut leakiness and immunity to an intestinal commensal lachnospiracaea species." In LUPUS 21ST CENTURY 2018 CONFERENCE, Abstracts of the Fourth Biannual Scientific Meeting of the North and South American and Caribbean Lupus Community, Armonk, New York, USA, September 13 – 15, 2018. Lupus Foundation of America, 2018. http://dx.doi.org/10.1136/lupus-2018-lsm.6.

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