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Статті в журналах з теми "Intrauterine programming"
Lister, Rolanda. "Intrauterine Programming of Diabetes Induced Cardiac Embryopathy." Diabetes & Obesity International Journal 4, no. 3 (2019): 1–14. http://dx.doi.org/10.23880/doij-16000202.
Повний текст джерелаFernandez‐Capetillo, Oscar. "Intrauterine programming of ageing." EMBO reports 11, no. 1 (December 11, 2009): 32–36. http://dx.doi.org/10.1038/embor.2009.262.
Повний текст джерелаFowden, A. L., and A. J. Forhead. "Endocrine mechanisms of intrauterine programming." Reproduction 127, no. 5 (May 2004): 515–26. http://dx.doi.org/10.1530/rep.1.00033.
Повний текст джерелаIge, S. F., R. E. Akhigbe, and O. O. Akinsemola. "Intrauterine Programming and Postnatal Hypertension." Research Journal of Obstetrics and Gynecology 4, no. 1 (January 1, 2011): 1–27. http://dx.doi.org/10.3923/rjog.2011.1.27.
Повний текст джерелаBarker, David J. P. "Intrauterine programming of adult disease." Molecular Medicine Today 1, no. 9 (December 1995): 418–23. http://dx.doi.org/10.1016/s1357-4310(95)90793-9.
Повний текст джерелаFowden, A. L., O. A. Valenzuela, O. R. Vaughan, J. K. Jellyman, and A. J. Forhead. "Glucocorticoid programming of intrauterine development." Domestic Animal Endocrinology 56 (July 2016): S121—S132. http://dx.doi.org/10.1016/j.domaniend.2016.02.014.
Повний текст джерелаFowden, A. L., A. J. Forhead, P. M. Coan, and G. J. Burton. "The Placenta and Intrauterine Programming." Journal of Neuroendocrinology 20, no. 4 (April 2008): 439–50. http://dx.doi.org/10.1111/j.1365-2826.2008.01663.x.
Повний текст джерелаRemacle, C., O. Dumortier, V. Bol, K. Goosse, P. Romanus, N. Theys, T. Bouckenooghe, and B. Reusens. "Intrauterine programming of the endocrine pancreas." Diabetes, Obesity and Metabolism 9, s2 (November 2007): 196–209. http://dx.doi.org/10.1111/j.1463-1326.2007.00790.x.
Повний текст джерелаGale, C. R. "Intrauterine Programming of Adult Body Composition." Journal of Clinical Endocrinology & Metabolism 86, no. 1 (January 1, 2001): 267–72. http://dx.doi.org/10.1210/jc.86.1.267.
Повний текст джерелаGale, Catharine R., Christopher N. Martyn, Samantha Kellingray, Richard Eastell, and Cyrus Cooper. "Intrauterine Programming of Adult Body Composition1." Journal of Clinical Endocrinology & Metabolism 86, no. 1 (January 2001): 267–72. http://dx.doi.org/10.1210/jcem.86.1.7155.
Повний текст джерелаДисертації з теми "Intrauterine programming"
Franko, Kathryn Lynette. "Regulation and intrauterine programming of glucogenic capacity." Thesis, University of Cambridge, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.613228.
Повний текст джерелаCrispi, Brillas Fàtima. "Fetal programming of cardiovascular dysfunction in intrauterine growth restriction." Doctoral thesis, Universitat de Barcelona, 2009. http://hdl.handle.net/10803/2276.
Повний текст джерелаFetal growth restriction (FGR), with a prevalence of 5-10% in newborns, is associated with increased cardiovascular mortality in adulthood, but the pathophysiological links of this relationship are only partially understood. The main hypothesis of this thesis was that FGR induces primary cardiac dysfunction and remodelling in utero that persists postnatally and leads to increased cardiovascular risk in adulthood.
METHODS
Cardiovascular function was assessed in a cohort of FGR fetuses and correlated to the severity stages of FGR, presence of preeclampsia and also perinatal data in order to evaluate its potential utility in the clinical management of these fetuses. Finally, cardiac and vascular function was also assessed in childhood.
RESULTS
In utero, FGR fetuses showed signs of subclinical cardiac dysfunction measured by echocardiography (increased E/A ratios and isovolumic times with normal cardiac output) from early stages. Cardiac dysfunction deteriorated further with the progression of fetal compromise, together with the appearance of biochemical signs of cell damage (increased heart-fatty acid binding protein concentrations in cord blood). Preeclampsia per se was not associated to cardiac function in FGR fetuses. Cardiac function parameters, such as ductus venosus and myocardial performance index, were independently associated with perinatal death in preterm FGR. Therefore, a combination cardiac parameters may be useful in the clinical management of preterm FGR by stratifying the estimated probability of death. Children with FGR showed changes in cardiac shape (more globular morphology), subclinical cardiac dysfunction (increased heart rate and reduced stroke volume and myocardial peak velocities) and vascular remodelling (increased blood pressure and carotid intima-media thickness).
CONCLUSIONS
FGR present cardiovascular dysfunction in utero that persists postnatlly. These findings suggest that fetal growth restriction induces primary cardiac changes which could explain the increased predisposition to cardiovascular disease in adult life. Given its high prevalence in the general population, this might have to be taken into account in assessing cardiovascular risk factors and treatment.
INTRODUCCIÓN
El retaso de crecimiento intrauterino (CIR), con una prevalencia del 5-10% de los recién nacidos, se asocial a un aumento de la mortalidad cardiovascular en vida adulta, pero la fisiopatología de esta correlación aun es incierta. La principal hipótesis de esta tesis fue que el CIR induce una disfunción y remodelado cardiovascular primario in útero que después persiste en vida postnatal y condiciona un aumento del riesgo cardiovascular en vida adulta.
MÉTODOS
En una cohorte de fetos CIR se evaluó la función cardiovascular y se correlacionó con el grado de severidad del CIR, la presencia de preeclampsia y también los resultados perinatales, para poder evaluar su potencial utilidad en el manejo clínico de estos fetos. Finalmente, la función cardiovascular también fue evaluada postnatalmente.
RESULTADOS
En útero, los fetos CIR mostraron signos de disfunción cardiaca subclínica medida mediante eco cardiografía (aumento de los ratios E/A i tiempos isovolumétricos, con gasto cardíaco normal) des de estadios iniciales de severidad. La función cardiaca mostró un deterioro progresivo con el aumento de severidad del CIR, a la vez que aparecieron signos bioquímicos de daño miocárdico en estadios finales de deterioro (aumento de los niveles de heart-fatty acid binding proteins en sangre de cordón). La preeclampsia per se no mostró asociación con el grado de disfunción cardiaca. Algunos parámetros de función cardiaca fetal, como el ductus venoso y el índice de rendimiento miocárdico, mostraron una asociación independiente con la mortalidad perinatal en los casos de CIR pretérmino. A nivel postnatal, los niños con antecedente de CIR mostraron cambios en la morfología cardiaca (más globular), disfunción cardiaca subclínica (aumento de la frecuencia cardiaca, y reducción del volumen de eyección y velocidades miocárdicas) y remodelación vascular (aumento de la presión arterial y grosor de las carótidas).
CONCLUSIONES
Los casos de CIR presentan una disfunción cardiovascular in útero que persiste postnatalmente. Los resultados sugieren que el CIR induce cambios cardiacos primarios que podría explicar la mayor predisposición a patología cardiovascular en vida adulta.
Tegethoff, Marion. "Fetal origins of pediatric disease fetoplacental plasticity and intrauterine programming by stress and glucocorticoids." Göttingen Cuvillier, 2009. http://d-nb.info/999629417/04.
Повний текст джерелаKelly, Amy, and Amy Kelly. "Adaptations in the Pancreatic Islet Transcriptome of Intrauterine Growth Restricted Fetuses." Diss., The University of Arizona, 2017. http://hdl.handle.net/10150/624586.
Повний текст джерелаHuber, Hillary. "Aggressive Behavioral Phenotype in Intrauterine Growth Restricted (IUGR) Baboons Exposed to Moderate Nutrient Restriction Early in Development." OpenSIUC, 2014. https://opensiuc.lib.siu.edu/dissertations/824.
Повний текст джерелаAlves, Márcio Bonesso. "Flexibilidade cognitiva em roedores adultos expostos à restrição de crescimento intrauterino : investigação do papel do cuidado materno e da resposta neuroquímica à recompensa." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2014. http://hdl.handle.net/10183/109720.
Повний текст джерелаIntrauterine growth restriction (IUGR) is associated with altered food preferences as well as increased risk for obesity in adult life. However, while the adverse metabolic outcomes have been better characterized in this condition, the neurobehavioral disabilities and particular abnormalities in specific areas of the brain have received less attention. Moreover, the role of maternal care in the stablishment of these outcomes should be evaluated. The aim of this study was to investigate the effects of IUGR on the cognitive flexibility of adult animals, exploring the putative involvement of maternal care and the neurochemical response to reward – through the measurement of tyrosine hydroxylase (TH) content in the orbitofrontal (OF) cortex and nucleus accumbens (nacc) - on this behavioral outcome. Materials and methods: From day 10 of gestation and through lactation, Sprague-Dawley rats received either an ad libitum (AdLib), or a 50% food restricted (FR) diet. At birth, pups were cross-fostered, generating AdLib/AdLib and FR/AdLib groups (pregnancy/lactation). From day 2 to 7 postpartum maternal care was recorded in a five daily sessions. Cognitive flexibility was measured using the Attentional Set-Shifting Task (ASST) with a sweet pellet as a reward. Animals were also submitted to 4 hours of fasting and then exposed to a sweet food for 1 hour to verify the palatable food consumption. TH content in the OF cortex and Nacc was measured at baseline or in response to palatable food intake. Results: Dams of FR group showed less care toward her pups. More precisely, these FR dams showed less licking/grooming than AdLib dams (p<0.01), without difference in the nursing postures and time spent out of the nest. Pups of FR dams had reduced birth weight (p<0.01). At 90 days of age, FR/AdLib males showed increased intake of sweet food in the acute exposure (p=0.04), while females did not differ in the amount consumed (p=0.37). When compared to Controls, IUGR females needed fewer trials to reach criterion in the ASST(p=0.04) and had an increase in TH in response to sweet food intake in the OFC (p=0.04), but not at baseline. No differences were seen in males (p=0.51). In the nacc, there was an increase in TH at baseline in IUGR males (p=0.03) and females (p=0.02). Discussion: The protocol used was efficient in inducing IUGR in the pups. To characterize maternal care in different protocols is crucial to better understand the relationships between early adverse environment and their coincident adverse outcomes in adult life. The improvement in performance of FR females in ASST and concomitant increase in OFC TH in respone to sweet food consumption suggests that palatable food cues are stronger for intrauterine restricted females. Besides, alterations found in the Nacc in both sexes suggest that IUGR induces modifications in the central response to palatable food cues and its intake, affecting dopamine release in select structures of the brain reward system.
Ekert, Jason Elliot. "Intrauterine programming of leptin / Jason Elliot Ekert." 2000. http://hdl.handle.net/2440/19853.
Повний текст джерелаxxiii, 1 v. (various pagings) : ill. (some col.) ; 30 cm.
Title page, contents and abstract only. The complete thesis in print form is available from the University Library.
The guinea pig and pig were evaluated as experimental animal models in which to investigate mechanisms of in utero leptin programming in humans.
Thesis (Ph.D.)--Adelaide University, Dept. of Obstetrics and Gynaecology, 2001
Rueda-Clausen, Christian Federico. "Long-term cardiovascular and metabolic effects of hypoxia-induced intrauterine growth restriction." Phd thesis, 2011. http://hdl.handle.net/10048/1768.
Повний текст джерелаMaftei, Oana. "Intrauterine influences on obesity and insulin resistance in pre-pubertal children." Thesis, 2012. http://hdl.handle.net/2440/75507.
Повний текст джерелаThesis (Ph.D.) -- University of Adelaide, School of Population Health and Clinical Practice and School of Paediatrics and Reproductive Health, 2012
Книги з теми "Intrauterine programming"
Seckl, Jonathan R., and Yves Christen, eds. Hormones, Intrauterine Health and Programming. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-02591-9.
Повний текст джерелаHormones, Intrauterine Health and Programming. Springer, 2014.
Знайти повний текст джерелаSeckl, Jonathan R., and Yves Christen. Hormones, Intrauterine Health and Programming. Springer, 2014.
Знайти повний текст джерелаЧастини книг з теми "Intrauterine programming"
Cooper, Cyrus, Muhammad K. Javaid, Karen Walker-Bone, Elaine M. Dennison, and Nigel K. Arden. "The Intrauterine Programming of Osteoporosis." In Medical Science Symposia Series, 43–50. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4615-1061-1_5.
Повний текст джерелаJiao, Zhexiao, Hao Kou, Dan Xu, Hanwen Luo, and Hui Wang. "Intrauterine Programming and Effects of Caffeine." In Diet, Nutrition, and Fetal Programming, 339–53. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-60289-9_25.
Повний текст джерелаShin, Bo-Chul, and Sherin U. Devaskar. "Regulation of Skeletal Muscle GLUT4 in Intrauterine Growth Restriction Offspring." In Fetal and Early Postnatal Programming and Its Influence on Adult Health, 103–24. Boca Raton : Taylor & Francis, 2017. | Series: Oxidative stress and disease: CRC Press, 2017. http://dx.doi.org/10.1201/9781315154312-6.
Повний текст джерелаOrtega-Márquez, Ana Laura, Angélica Morales-Miranda, and Sumiko Morimoto. "Impact of Epigenetic Mechanisms on the Regulation of Gene Expression During Intrauterine Programming of the Endocrine Pancreas." In Handbook of Nutrition, Diet, and Epigenetics, 777–92. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-55530-0_69.
Повний текст джерелаOrtega-Márquez, Ana Laura, Angélica Morales-Miranda, and Sumiko Morimoto. "Impact of Epigenetic Mechanisms on the Regulation of Gene Expression During Intrauterine Programming of the Endocrine Pancreas." In Handbook of Nutrition, Diet, and Epigenetics, 1–16. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-31143-2_69-1.
Повний текст джерелаSchäffer, Leonhard, Tilo Burkhardt, Maren Tomaske, Manfred Rauh, and Ernst Beinder. "6 Intrauterine corticosteroids for lung maturation: Observations of HPA axis function and cardiac autonomic balance in the neonate." In Perinatal Programming, edited by Andreas Plagemann. Berlin, Boston: DE GRUYTER, 2011. http://dx.doi.org/10.1515/9783110249453.51.
Повний текст джерелаGlezerman, Marek. "Intrauterine Development of Sex Differences—Fetal Programming * *For easier reading, I will be using throughout this chapter the term “fetus” whenever I refer to the embryo or the fetus and I will use the term “mother” for the pregnant woman. Moreover, whenever I use throughout this chapter the terms “increased risk” for a certain type of pathology, I mean just that, namely that a preexisting risk may increase, but still leave the majority of children unaffected." In Principles of Gender-Specific Medicine, 237–49. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-12-803506-1.00039-5.
Повний текст джерела