Готові списки джерел за темами / Intracerebral monitoring

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Добірка наукової літератури з теми "Intracerebral monitoring"

Автор: Grafiati
Опубліковано: 11 січня 2025
Оновлено: 5 липня 2025

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Статті в журналах з теми "Intracerebral monitoring"

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Lee, Kevin R., Ivo Drury, Elizabeth Vitarbo, and Julian T. Hoff. "Seizures induced by intracerebral injection of thrombin: a model of intracerebral hemorrhage." Journal of Neurosurgery 87, no. 1 (1997): 73–78. http://dx.doi.org/10.3171/jns.1997.87.1.0073.

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✓ The coagulation cascade plays an important role in brain edema formation caused by intracerebral blood. In particular, thrombin produces brain injury via direct brain cell toxicity. Seizures and increased cerebral electrical activity are commonly associated with intracerebral blood and are possible effects of thrombin leading to cell injury in the brain. In this study, artificial clots containing concentrations of thrombin found in hematomas were infused intracerebrally in rats. The animals were observed clinically for seizure activity, behavior, and neurological deficits. Several animals underwent video electroencephalographic (EEG) monitoring during intracerebral infusion and for 30 minutes postinfusion. All animals were killed 24 hours after injection, and brain water and ion contents were measured to determine the amount of brain edema. Clinically, thrombin produced focal motor seizures in all animals. None of the control animals or those receiving Nα-(2-Naphthalenesulfonyl-glycyl)-4-amidino-DL-phenylalanine-piperidide (α-NAPAP), a thrombin inhibitor added to the thrombin, showed clinical evidence of seizures. Of the rats undergoing EEG monitoring, all animals receiving thrombin showed electrical evidence of seizure activity, whereas none of the control animals exhibited seizure activity. There was no evidence of seizure activity on EEG monitoring when α-NAPAP was injected along with the thrombin. In addition, the artificial clots containing thrombin produced agitation and a circling tendency in the rats, along with brain edema. These results indicate that the coagulation cascade is involved in seizure production and increased brain electrical activity, which contribute to the neurological deficits and brain edema formation that are seen with intracerebral hemorrhage.
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2

Hemphill, J. Claude, Diane Morabito, Mary Farrant, and Geoffrey T. Manley. "Brain Tissue Oxygen Monitoring in Intracerebral Hemorrhage." Neurocritical Care 3, no. 3 (2005): 260–70. http://dx.doi.org/10.1385/ncc:3:3:260.

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Demierre, Bertrand, Falko A. Stichnoth, Akira Hori, and Otto Spoerri. "Intracerebral ganglioglioma." Journal of Neurosurgery 65, no. 2 (1986): 177–82. http://dx.doi.org/10.3171/jns.1986.65.2.0177.

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✓ Ganglioglioma in the temporal lobe was encountered in 0.6% of a series of 998 patients undergoing biopsy for intracranial tumors. It was more frequent in young adults and children. Seizures were the main symptom and had been present over a long period of time in some patients. Clinical examination also revealed intracranial pressure, focal signs, and behavioral problems, but these symptoms were not always evident. Electroencephalography was not specific. Calcifications were visible on computerized tomography (CT) in five cases. The cystic and well-circumscribed aspects of these tumors were apparent on CT scans; these features were confirmed at operation and on magnetic resonance imaging (MRI), when it was also found that the tumors were partially solid. Therefore, MRI seems to be a useful diagnostic method, particularly when a cystic tumor is revealed on CT, and is also valuable for postoperative monitoring. Surgical treatment for ganglioglioma is recommended, with total extirpation if possible.
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Santamarina Pérez, Estevo, Raquel Delgado-Mederos, Marta Rubiera, et al. "Transcranial Duplex Sonography for Monitoring Hyperacute Intracerebral Hemorrhage." Stroke 40, no. 3 (2009): 987–90. http://dx.doi.org/10.1161/strokeaha.108.524249.

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Kapinos, G., and J. C. Hemphill. "Clinicoradiologic acute monitoring after intracerebral hemorrhage: Toward standards?" Neurology 81, no. 2 (2013): 102–3. http://dx.doi.org/10.1212/wnl.0b013e31829a3564.

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Mathru, M. "Transcranial Duplex Sonography for Monitoring Hyperacute Intracerebral Hemorrhage." Yearbook of Anesthesiology and Pain Management 2010 (January 2010): 171–73. http://dx.doi.org/10.1016/s1073-5437(09)79362-3.

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Verlooy, J., L. Heytens, G. Veeckmans, and P. Selosse. "Intracerebral temperature monitoring in severely head injured patients." Acta Neurochirurgica 134, no. 1-2 (1995): 76–78. http://dx.doi.org/10.1007/bf01428508.

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Helbok, Raimund, Ravi Chandra Madineni, Michael J. Schmidt, et al. "Intracerebral Monitoring of Silent Infarcts After Subarachnoid Hemorrhage." Neurocritical Care 14, no. 2 (2010): 162–67. http://dx.doi.org/10.1007/s12028-010-9472-9.

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Bauer, Richard, Michael Gabl, Alois Obwegeser, Klaus Galiano, Josef Barbach, and Iradj Mohsenipour. "Neurochemical monitoring using intracerebral microdialysis during cardiac resuscitation." Intensive Care Medicine 30, no. 1 (2004): 159–61. http://dx.doi.org/10.1007/s00134-003-2015-5.

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Meybohm, P., E. Cavus, B. Bein, et al. "Neurochemical monitoring using intracerebral microdialysis during systemic haemorrhage." Acta Neurochirurgica 149, no. 7 (2007): 691–98. http://dx.doi.org/10.1007/s00701-007-1231-0.

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Більше джерел

Дисертації з теми "Intracerebral monitoring"

1

Antonsson, Johan. "On optical methods for intracerebral measurements during stereotactic and functional neurosurgery : Experimental studies." Doctoral thesis, Linköping : Linköping University, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-8081.

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Patriota, Gustavo Cartaxo. "Avaliação da autorregulação cerebral dinâmica através da reatividade cerebrovascular em suíno com volume expansivo por balão simulando aumento de hematoma intracerebral." Universidade de São Paulo, 2017. http://www.teses.usp.br/teses/disponiveis/5/5138/tde-18122017-093809/.

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INTRODUÇÃO: A autorregulação cerebral representa um dos mecanismos fisiopatológicos incertos na hemorragia intracerebral espontânea, cujo comprometimento pode influenciar no resultado prognóstico e terapêutico. O objetivo deste trabalho é avaliar a autorregulação cerebral dinâmica em modelo suíno de hemorragia intracerebral espontânea através do índice de reatividade pressórica cerebrovascular e determinar a eficácia das intervenções clínicas e cirúrgicas. MÉTODOS: Foram estudados 21 suínos híbridos machos com idade de 3 meses. O modelo experimental simulou o efeito expansivo de uma hemorragia intracerebral espontânea de grande volume quando comparado ao cérebro humano. Foram avaliados volumes de expansão diferentes, distribuídos em três grupos com sete suínos cada. O protocolo anestésico incluiu uma monitoração hemodinâmica invasiva associada a preservação da autorregulação cerebral. Os experimentos foram submetidos a monitoração neurológica multimodal e divididos em 5 fases. O índice de reatividade pressórica cerebrovascular estimou a autorregulacão cerebral durante todas as fases, sendo as três primeiras sem intervenções terapêuticas e as duas últimas para avaliar a eficácia das intervenções salina hipertônica e cirurgia. RESULTADOS: Os grupos avaliados foram homogêneos e sem diferença estatística quanto ao comprometimento da autorregulação cerebral comparando os diferentes volumes e tempos de compressão durante as duas primeiras horas da expansão do volume intracraniano. O comprometimento do índice de reatividade pressórica cerebrovascular ocorreu em alguns experimentos influenciando nas fases de tratamento subsequentes, salina hipertônica e cirurgia. CONCLUSÕES: Volumes expansivos elevados podem comprometer a autorregulação cerebral dinâmica e apresentar desfecho terapêutico desfavorável. A intervenção clínica e cirúrgica tem benefício nos experimentos com preservação do índice de reatividade pressórica cerebrovascular<br>INTRODUCTION: Cerebral autoregulation represents one of the uncertain pathophysiological mechanisms in spontaneous intracerebral hemorrhage, whose impairment may influence prognostic and therapeutic outcome. The aim of this study was to evaluate the dynamic cerebral autoregulation in the swine model of spontaneous intracerebral hemorrhage through the cerebrovascular reactivity index and to determine the efficacy of clinical and surgical interventions. METHODS: Twenty-one male hybrid pigs aged 3 months were studied. The experimental model simulated the expansive effect of a large intracerebral hemorrhage when compared to the human brain. Different volumes were evaluated, distributed in three groups with seven pigs each. Each experiment was divided in five phases. The anesthetic protocol included invasive hemodynamic monitoring associated with the preservation of cerebral autoregulation. Multimodallity monitoring was realised in all experiments. The cerebrovascular reactivity index estimated the cerebral autoregulation during all phases. The first three phases were without therapeutic interventions, and the last two phases were with therapeutic intervention of hypertonic saline solution and neurosurgery respectively. RESULTS: The evaluated groups were homogeneous and without statistical difference regarding the impairment of the cerebral autoregulation comparing different volumes and compression times during the first two hours of the intracranial volume expansion. CONCLUSIONS: Elevated expansive volumes may compromise dynamic cerebral autoregulation and have unfavorable therapeutic outcome. Clinical and surgical intervention had benefit in the experiments with preservation of cerebrovascular reactivity index
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Chen, Shuting. "Évaluation en temps réel du recrutement des réserves énergétiques cérébrales en glycogène pendant une dépolarization corticale envahissante ou la potentialisation à long terme hippocampique." Electronic Thesis or Diss., Lyon 1, 2024. http://www.theses.fr/2024LYO10203.

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Le glycogène astrocytaire est la seule forme de réserve énergétique dans le cerveau, généralement considéré comme mobilisé via la navette astrocyte-neurone dans des conditions telles que l'ischémie ou l'activité neuronale intense. La dépolarisation corticale envahissante (DCE) est une vague de dépolarisation quasi-complète des neurones et des cellules gliales, posant une augmentation de besoins métaboliques au tissu cérébral. La potentialisation à long terme (LTP) est reconnue comme le mécanisme moléculaire de la formation de la mémoire, suite à une stimulation à haute fréquence. L'hypothèse était que le glycogène serait mobilisé lors des SD et de l'induction de la LTP, fournissant ainsi un modèle pour étudier le métabolisme du glycogène et sa fonction dans un cerveau normal. Les réserves de glycogène ont été bloquées par l'administration locale d'un inhibiteur de la glycogène phosphorylase, 1,4-didésoxy-1,4-imino-D-arabinitol (DAB). Les SDs ont été enregistrées à l'aide de l'électrocorticographie (ECoG). Les concentrations extracellulaires de lactate, de glucose et d'oxygène dans le cortex ont été enregistrées pendant les SDs seconde par seconde à l'aide de biocapteurs enzymatiques à microélectrodes en présence ou en absence de DAB. Le blocage des réserves de glycogène a prolongé la durée de la repolarisation tissulaire après la dépolarisation. Les SDs ont induit une augmentation transitoire de la concentration extracellulaire de lactate dans le cortex, accompagnée d'une diminution du glucose et d'une consommation d'oxygène. En présence de DAB, la diminution du glucose et l'augmentation du taux métabolique de l'oxygène n'ont pas été réduites mais ont duré plus longtemps, tandis que la libération de lactate a été diminuée avec le traitement DAB, indiquant que la glycogénolyse libère principalement du lactate. De plus, en présence de DAB, l'administration supplémentaire de lactate par voie intraveineuse a permis de rétablir une durée normale de dépolarisation, ce qui suggère que le lactate libéré par les réserves de glycogène fournit un supplément d'énergie nécessaire à la repolarisation tissulaire. Ensuite, lors de l'induction et du maintien de la LTP, le lactate est libéré pendant la stimulation et probablement en continu pendant 75 minutes après la stimulation. Cependant, le traitement au DAB a bloqué la potentialisation tout au long de l'enregistrement de la réponse postsynaptique d'une heure. Par conséquent, la glycogénolyse se passe en quelques secondes pour alimenter le métabolisme énergétique en libérant du lactate dans l'espace extracellulaire, qui est ensuite métabolisé de manière anaérobie comme source d'énergie supplémentaire<br>Astrocytic glycogen is the only form of brain energy stores, which is general considered to be mobilized through astrocyte-neuron lactate shuttle in conditions like ischemia or intense neuronal activity. Cortical spreading depolarization (SD) is a propagating wave of near-complete depolarization of neurons and glial cells, posing a metabolic challenge on brain tissue. Long-term potentiation (LTP) is recognized as the molecular mechanism of memory formation, following high frequency stimulation. We hypothesized that glycogen would be mobilized during CSDs and LTP induction, providing a model to study glycogen metabolism and its function in a normal brain. Glycogen stores were blocked by local administration of glycogen phosphorylase inhibitor 1,4-dideoxy-1,4-imino-D-arabinitol (DAB). SDs were recorded using eletrocorticalgraphy (ECoG) recordings. Extracellular lactate, glucose, and oxygen concentrations in cortex were monitored during SDs second by second using enzymatic microelectrode biosensors in presence or absence of DAB. Blocking glycogen stores increased the duration of tissue repolarization after depolarization. SDs induced a transient increase in cortical extracellular lactate concentration accompanied by glucose decrease and oxygen consumption. In the presence of DAB, the glucose decrease and elevated oxygen metabolic rate was not reduced but lasted longer, while lactate release was diminished with DAB treatment, indicating that glycogenolysis predominantly release lactate. Furthermore, in the presence of DAB, supplemental intravenous lactate administration rescued a normal duration of depolarization, suggesting that lactate released from glycogen stores provide a boost in energy supply required for tissue repolarization. Next, in the induction and maintenance of LTP, lactate is released during the stimulation and is probably released continuously during 75min after stimulation, however, DAB treatment blocked the potentiation throughout 1h recording of postsynaptic response. Therefore, glycogenolysis occurs within seconds to fuel energy metabolism through releasing lactate into extracellular space, which is then anaerobically metabolized as a supplemental energy source
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Keli, Barcelos Gleicy. "Valeur pronostique du « monitoring » du métabolisme énergétique cérébral chez les patients victimes d’une hémorragie sous-arachnoïdienne grave." Thesis, Lyon 1, 2012. http://www.theses.fr/2012LYO10300.

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Le ratio métabolique (MR) est un marqueur du métabolisme cérébral. Dans notre travail, nous avons démontré sa valeur pronostique chez 68 patients victimes d’une hémorragie sous-arachnoïdienne anévrysmale grave. En effet, une diminution du MR sous le seuil de 3,35 traduit un phénomène d’hyperglycolyse relative, dont le nombre d’événement est prédictive d’un pronostic défavorable avec une excellente sensibilité et spécificité. L’obtention de ces résultats est rendue possible, notamment après une phase de validation dans un modèle animal de procédures permettant de limiter les effets de facteurs pré-analytiques critiques. Ces résultats permettent d’envisager une étude pour savoir si l’intégration de ce marqueur dans la stratégie de prise en charge du patient, permet de modifier son devenir fonctionnel. Après avoir validé analytiquement les mesures de pyruvate, glucose et lactate impliquant la technique de microdialyse, nous avons étudié sur une cohorte de patients graves aSAH, modeste (n=18 patients) s’il existait des phénomènes d’hyperglycolyse et leur corrélation avec le pronostic. Dans notre série, à la différence de l’approche globale (cathétérisme de la veine jugulaire), un phénomène d’hyperglycolyse conduirait vers un bon pronostic. En fait, l’approche par microdialyse donne une information sur le métabolisme énergétique localisé à l’implantation de la sonde, alors que le MR donne une information globale, ce qui est probablement le facteur le plus important expliquant la différence d’interprétation entre les 2 approches. En l’absence d’outils de traitement de données et d’algorithmes de décision clinique validés, la microdialyse ne donne pas à l’heure actuelle, une valeur individuelle diagnostique ou pronostique. Un des résultats très prometteurs de ce travail, est la mise en évidence d’un phénomène d’hyperglycolyse relative globale lors du vasospasme, rapidement réversible chez les patients ayant bien évolué, alors qu’il perdure de nombreuses heures après le vasospasme chez les patients ayant évolué de manière péjorative. Ces résultats nécessitent d’être reproduits sur un nombre plus significatifs de patients, ce qui permettrait une confirmation radiologique du vasospasme de manière plus précoce afin de confirmer son importance, sa localisation et l’éventualité de le traiter rapidement<br>The metabolic ratio (MR) is an index of the brain energetic metabolism. In our study, we have demonstrated its prognostic value for 68 poor grade patients aneurysmal subarachnoid hemorrhage (aSAH): a MR below the threshold value of 3.35 reflects a phenomenon of global cerebral hyperglycolysis which, if repeated, is predictive of a bad outcome. These results were made possible after validation step in an animal mode which allowed to control the critical pre-analytical factors. Our results pave the way for a clinical study aiming to determine if taking into account the MR will help to improve the functional outcome of the aSAH patients. In another approach, based on the use of cerebral microdialysis, we have studied, in an 18 patients cohort, and after an analytical validation of a new biochemical analysis, if such cerebral hyperglycolysis phenomenon was a encountered in this cohort, if these was a correlation with the patients’ outcome. In contrast with the previous 68 aSAH patients, this hyperglycolysis phenomenon appears linked to a good outcome. This apparent discrepancy may be due the difference in the anatomical giving a more localized information on the brain metabolism than the jugular approach used for the MR determination. The most interesting of our results is the correlation found between hyperglycolysis and cerebral vasospasm. If conformed with a larger cohort of aSAH patients, the use of MR could allow an earlier detection and treatment of cerebral vasospasm
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Bonini, Francesca. "Le rôle du cortex frontal médian dans la supervision de l'action chez l'homme : études électrophysiologiques." Thesis, Aix-Marseille, 2016. http://www.theses.fr/2016AIXM5023/document.

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La capacité à évaluer les résultats nos actions est fondamentale pour adapter et optimiser notre comportement et dépend d’un système superviseur chargé d’évaluer l’action, détecter les erreurs, déclencher des corrections.Le réseau neuronal sous-jacent la supervision de l’action n’a pas été complètement caractérisé chez l’homme.Dans une première étude nous avons enregistré dans l’Aire Motrice Supplémentaire (AMS) des LFP évoqués par les réponses et modulés par la performance. Des LFP évoqués exclusivement par les erreurs ont été enregistrés plus tardivement dans le cortex préfrontal médian.Dans la deuxième étude, nous avons observé que les activités de hautes-fréquences gamma sont, elles aussi, modulées par la performance des sujets, mais dans un vaste réseau frontal et extra-frontal.Dans une troisième étude, utilisant des enregistrements simultanés électroencéphalographiques (EEG) et magnétoencéphalographiques (MEG), nous observé une activité évoquée par un feedback interne sur l’EEG (mais pas en MEG), alors qu'une activité évoquée par le feedback externe était bien visible sur les enregistrements MEG, indiquant que les générateurs de ces deux activités cérébrales, sont différents. Nos résultats montrent une implication de l’AMSp dans la supervision de l’action chez l’homme, bien plus importante que ce que l’on soupçonnait auparavant. L’AMS évalue précocement, et de façon continue, l’action en cours et elle engage vraisemblablement des structures préfrontales en cas d’erreur seulement. Le traitement de l’erreur d’action, selon qu'il se fonde sur des informations internes ou externes est certainement sous-tendu par des réseaux corticaux différents<br>The capacity to evaluate the outcome of our actions is fundamental for adapting and optimizing behaviour. This capability depends on an action monitoring system in charge of assessing ongoing actions, detecting errors, and evaluating outcomes.Electrical brain activity evoked by negative outcomes is thought to originate within the medial part of the frontal cortex. Nonetheless, the underlying neuronal network is incompletely characterised in humans.In the two first studies, we investigated the anatomical substrates of action monitoring in humans using intracerebral local field potential (LFP) recordings of cerebral cortex from epileptic patients. Response evoked LFPs sensitive to outcome were recorded from the Supplementary Motor Area proper (SMA), while LFPs evoked exclusively by errors were recorded later in the medial prefrontal cortex. High-gamma-frequency activity (60-180 Hz) was modulated as a function of action outcome in a vast frontal and extra-frontal network.In a third study using simultaneous recording of electroencephalography (EEG) and magnetoencephalography (MEG), we found that error related activity was detected by EEG (but not by MEG), while feedback-related activity was detected by MEG, indicating that the sources of these two forms of outcome-modulated brain activity are different.To conclude the SMA is much more involved in action monitoring than previously thought. SMA rapidly and continuously assesses ongoing actions and likely engages more rostral prefrontal structures in the case of error. Processing of action errors and of negative externally delivered feedback therefore appears to be supported by distinct cortical networks
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Hübner, Felix. "Die intraoperative Messung des intracerebralen Sauerstoffpartialdrucks als adjuvanter Monitoring-Parameter der cerebralen Durchblutung bei der operativen Versorgung cerebraler Aneurysmen." [S.l.] : [s.n.], 2000. http://deposit.ddb.de/cgi-bin/dokserv?idn=962118842.

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Hübner, Felix [Verfasser]. "Die intraoperative Messung des intracerebralen Sauerstoffpartialdrucks als adjuvanter Monitoring-Parameter der cerebralen Durchblutung bei der operativen Versorgung cerebraler Aneurysmen / vorgelegt von Felix Hübner." 2000. http://d-nb.info/962118842/34.

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Книги з теми "Intracerebral monitoring"

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McDonald, Vickie, and Marie Scully. Anticoagulants and antithrombotics in critical illness. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0051.

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Coagulation is best thought of using the cell-based model of coagulation. Patients commenced on heparin therapy should have their platelet count monitored early because of the risk of heparin-induced thrombocytopenia, which can occur on any type or dose of heparin. Emergency reversal of warfarin should be with prothrombin complex concentrate (containing factors II, VII, IX, and X) and not fresh frozen plasma. New oral anticoagulants have the advantage of predictable pharmacokinetics and do not require routine monitoring, but optimal reversal strategies for these agents are not clear. Thrombolytic agents lead to variable degrees of systemic lysis, which may cause haemorrhage, including intracerebral haemorrhage
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Prout, Jeremy, Tanya Jones, and Daniel Martin. Nervous and musculoskeletal systems. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199609956.003.0006.

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This chapter outlines the basic science related to the nervous and musculoskeletal systems which particularly apply to the conduct of anaesthesia. Consciousness, sleep and anaesthetic depth are discussed with the measurement of anaesthetic depth using bispectral index and evoked potentials. Factors which influence cerebral blood flow and intracerebral pressure are detailed, allowing understanding of neuroanaesthesia techniques. Pharmacological and anaesthetic management of seizures is also described. Diseases affecting the autonomic nervous system, testing for these disorders and the implication for anaesthesia are included. Physiological consequences of spinal cord injury with patterns of injury are described. Neuromuscular blocking agents, monitoring of blockade and implications for patients with neuromuscular disease is also explained.
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Reich, David L., Stephan A. Mayer, and Suzan Uysal, eds. Neuroprotection in Critical Care and Perioperative Medicine. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.001.0001.

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Clinicians caring for patients are challenged by the task of protecting the brain and spinal cord in high-risk situations. These include following cardiac arrest, in critical care settings, and during complex procedural and surgical care. This book provides a comprehensive overview of various types of neural injury commonly encountered in critical care and perioperative contexts and the neuroprotective strategies used to optimize clinical outcomes. In addition to introductory chapters on the physiologic modulators of neural injury and pharmacologic neuroprotectants, the topics covered include: imaging assessment; tissue biomarker identification; monitoring; assessment of functional outcomes and postoperative cognitive decline; traumatic brain injury; cardiac arrest and heart-related issues such as valvular and coronary artery bypass surgery, aortic surgery and stenting, and vascular and endovascular surgery; stroke; intracerebral hemorrhage; mechanical circulatory support; sepsis and acute respiratory distress syndrome; neonatal issues; spinal cord injury and spinal surgery; and issues related to general, orthopedic, peripheral vascular, and ear, nose and throat surgeries.
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Частини книг з теми "Intracerebral monitoring"

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Xi, Guohua, Y. Hua, R. F. Keep, J. G. Younger, and J. T. Hoff. "Brain Edema after Intracerebral Hemorrhage: The Effects of Systemic Complement Depletion." In Intracranial Pressure and Brain Biochemical Monitoring. Springer Vienna, 2002. http://dx.doi.org/10.1007/978-3-7091-6738-0_66.

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Wu, G., and F. P. Huang. "Effects of venom defibrase on brain edema after intracerebral hemorrhage in rats." In Intracranial Pressure and Brain Monitoring XII. Springer Vienna, 2005. http://dx.doi.org/10.1007/3-211-32318-x_78.

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Kawai, Nobuyuki, T. Nakamura, and S. Nagao. "Effects of Brain Hypothermia on Brain Edema Formation after Intracerebral Hemorrhage in Rats." In Intracranial Pressure and Brain Biochemical Monitoring. Springer Vienna, 2002. http://dx.doi.org/10.1007/978-3-7091-6738-0_60.

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4

Bhasin, R. R., Gouhua Xi, Y. Hua, R. F. Keep, and J. T. Hoff. "Experimental Intracerebral Hemorrhage Effect of Lysed Erythrocytes on Brain Edema and Blood-Brain Barrier Permeability." In Intracranial Pressure and Brain Biochemical Monitoring. Springer Vienna, 2002. http://dx.doi.org/10.1007/978-3-7091-6738-0_65.

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Unterberg, A., A. von Helden, G. H. Schneider, and W. L. Lanksch. "Monitoring of Jugular Venous Oxygen Saturation in Patients with Intracerebral Hematomas." In Advances in Neurosurgery. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77997-8_6.

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Francis, Richard, Barbara A. Gregson, and A. David Mendelow. "Attitudes in 2013 to Monitoring Intracranial Pressure for Traumatic Intracerebral Haemorrhage." In Acta Neurochirurgica Supplement. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-22533-3_3.

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7

Yang, Dixon, Marialaura Simonetto, Nelly Campo, Digna Cabral, and Tatjana Rundek. "Intracerebral Venous System: Monitoring by Transcranial Color-Coded Duplex Sonography (TCCS)." In Neurosonology in Critical Care. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81419-9_28.

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Ferreira, Ana V., Isabel Maia, and Celeste Dias. "Monitoring of Cerebrovascular Reactivity in Intracerebral Hemorrhage and Its Relation with Survival." In Acta Neurochirurgica Supplement. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-59436-7_37.

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Camps-Renom, Pol. "Intracerebral Hemorrhage in ICU: Dynamic Monitoring by Transcranial Color-Coded Duplex Sonography (TCCS)." In Neurosonology in Critical Care. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-81419-9_40.

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Helden, A., G. H. Schneider, A. Unterberg, and W. R. Lanksch. "Monitoring of Jugular Venous Oxygen Saturation in Comatose Patients with Subarachnoid Haemorrhage and Intracerebral Haematomas." In Monitoring of Cerebral Blood Flow and Metabolism in Intensive Care. Springer Vienna, 1993. http://dx.doi.org/10.1007/978-3-7091-9302-0_18.

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Тези доповідей конференцій з теми "Intracerebral monitoring"

1

Rejmstad, P., G. Akesson, J. Hillman, and K. Wardell. "A laser Doppler system for monitoring of intracerebral microcirculation." In 2012 34th Annual International Conference of the IEEE Engineering in Medicine and Biology Society (EMBC). IEEE, 2012. http://dx.doi.org/10.1109/embc.2012.6346346.

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Salam, Muhammad Tariqus, Dang Khoa Nguyen, and Mohamad Sawan. "A multichannel intracerebral EEG monitoring system for epilepsy presurgical evaluation." In 2011 24th IEEE Canadian Conference on Electrical and Computer Engineering (CCECE). IEEE, 2011. http://dx.doi.org/10.1109/ccece.2011.6030465.

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3

Martínez-García, Jacqueline, Ana Aguilera-Simón, Pol Camps-Renom, et al. "Monitoring cerebral autoregulation and hemodynamics over the evolution of cerebral edema in patients with intracerebral hemorrhage." In Clinical and Translational Biophotonics. Optica Publishing Group, 2024. http://dx.doi.org/10.1364/translational.2024.tm3b.7.

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4

Chen, Yue, Isuru S. Godage, Saikat Sengupta, et al. "An MRI-Compatible Robot for Intracerebral Hemorrhage Removal." In 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3451.

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Анотація:
Intracerebral Hemorrhage (ICH) is the deadliest form of stroke and occurs when blood, leaked from a ruptured vessel pools in the brain forming a pool of semi-coagulated blood called a hematoma. 1 in 50 people will have an ICH in their lifetime [1] and the 30-day mortality rate is 43% with half of the deaths occurring in the acute phase, which motivates the need for safe and rapid treatment. However, literature reviews show no significant benefit of surgical removal vs. “watchful waiting”, despite the potential value of decompressing the brain. It has been hypothesized that this is due to the significant disruption of healthy brain tissue required to reach the hemorrhagic site in open brain surgery. Recent studies conducted on phantom models have shown that a robotic needle made from curved, concentric, elastic tubes can reach a hemorrhagic site through a needle-sized path to successfully aspirate the hematoma. This approach has the potential to decompress the brain with far less disruption to surrounding brain tissue [4]. Those initial experiments were conducted under guidance from periodic (low rate) CT [2]. The need for intraoperative imaging was motivated by the fact that the brain shifts during aspiration, collapsing to fill the cavity left by voided blood. This approach has the potential advantage of “one stop shopping”, since ICH is typically diagnosed in the CT scanner. It is appealing to treat ICH immediately after diagnosis, while the patient is still in the scanner. However, CT also has the drawback of requiring ionizing radiation, as well as providing only intermittent images rather than real-time information. In this paper, we consider a Magnetic Resonance Imaging (MRI) guided approach, which provides the converse in terms of both benefits and drawbacks. MRI is not typically used to diagnose ICH, but it can provide detailed soft-tissue and hematoma contrast [3], and fast image updates, enabling real-time monitoring of brain deformation during the aspiration process. Toward performing ICH aspiration with a concentric tube robot in an MRI environment, this paper presents accuracy and MR-compatibility tests for a new MR-compatible robot designed for ICH removal.
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Aponte-Becerra, Laura, Rodrigo Quispe, Laura Mendez-Pino, Vera Novak, Magdy Selim, and Vasileios-Arsenios Lioutas. "Continuous glucose monitoring in acute stroke." In the 8th International Workshop on Innovative Simulation for Healthcare. CAL-TEK srl, 2019. http://dx.doi.org/10.46354/i3m.2019.iwish.016.

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"Hyperglycaemia upon admission is a pathophysiological response to acute brain ischemia that has been independently associated with high mortality rate and poor prognosis. Glycaemic variability (GV) has also shown association with poor clinical outcomes among stroke patients. GV is best assessed by continuous glucose monitoring (CGM), which enables consecutives glucose measurements every 5 minutes. This pilot study aimed: 1) To describe safety, feasibility and tolerability of CGM in the acute stroke setting; and 2) To compare CGM and conventional FS glucose-based monitoring regimen in terms of their relationship with GUA and the accuracy of hypoglycaemic episodes detection. Safety, feasibility and tolerability of CGM was excellent in our cohort of 23 patients with acute stroke (61% ischemic and 39% intracerebral haemorrhage) and there were no adverse events. CGM recorded ten hypoglycaemic episodes that were not detected by conventional FS monitoring. GUA was associated with coefficient of variation (CV) of CGM (p=0.03), CV of FS (p=0.01), standard deviation (SD) of CGM (p-value=0.01) and mean amplitude of glucose excursions (MAGE) (pvalue= 0.001)."
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Kohl-Bareis, Matthias, Hellmuth Obrig, Jens Steinbrink, Jasmin Malak, Kamil Uludag, and Arno Villringer. "Noninvasive cerebral blood flow monitoring by a dye bolus method:separation of extra- and intracerebral absorption changes by frequency-domain spectroscopy." In BiOS 2001 The International Symposium on Biomedical Optics, edited by Britton Chance, Robert R. Alfano, Bruce J. Tromberg, Mamoru Tamura, and Eva M. Sevick-Muraca. SPIE, 2001. http://dx.doi.org/10.1117/12.434501.

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