Добірка наукової літератури з теми "INTERFERONOPATIA"
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Статті в журналах з теми "INTERFERONOPATIA"
Souza, Victória Cabral de, Roberto de Barros Silva, and Caique Antunis. "INTERFERON-I COMO ALVO TERAPÊUTICO NO TRATAMENTO DO LÚPUS ERITEMATOSO SISTÊMICO: USO DOS ANTICORPOS MONOCLONAIS ANIFROLUMABE, RONTALIZUMABE E SIFALIMUMABE." Revista Ibero-Americana de Humanidades, Ciências e Educação 9, no. 1 (January 31, 2023): 717–31. http://dx.doi.org/10.51891/rease.v9i1.8303.
Повний текст джерелаTommasini, Alberto, Irene Bruno, Maria Elisa Morelli, and Loredana Lepore. "Le interferonopatie di tipo I." Medico e Bambino 40, no. 8 (September 23, 2021): 509–14. http://dx.doi.org/10.53126/meb40509.
Повний текст джерелаPayet, Cloé A., Axel You, Odessa-Maud Fayet, Nadine Dragin, Sonia Berrih-Aknin, and Rozen Le Panse. "Myasthenia Gravis: An Acquired Interferonopathy?" Cells 11, no. 7 (April 4, 2022): 1218. http://dx.doi.org/10.3390/cells11071218.
Повний текст джерелаBriggs, Tracy A., Anindita Paul, Gillian Rice, and Ariane L. Herrick. "RNASEH2B Related Adult-Onset Interferonopathy." Journal of Clinical Immunology 39, no. 6 (July 31, 2019): 620–22. http://dx.doi.org/10.1007/s10875-019-00673-w.
Повний текст джерелаRodero, Mathieu P., Marie-Louise Frémond, Gillian I. Rice, Bénédicte Neven, and Yanick J. Crow. "JAK inhibition in STING-associated interferonopathy." Annals of the Rheumatic Diseases 75, no. 12 (October 12, 2016): e75-e75. http://dx.doi.org/10.1136/annrheumdis-2016-210504.
Повний текст джерелаManoussakis, Menelaos N., Clio P. Mavragani, Adrianos Nezos, Evangelia Zampeli, Anastassios Germenis, and Haralampos M. Moutsopoulos. "Type I interferonopathy in a young adult." Rheumatology 56, no. 12 (August 14, 2017): 2241–43. http://dx.doi.org/10.1093/rheumatology/kex316.
Повний текст джерелаKrieger, Ben Zion, Theresa Calvelli, Anna Kadish, and Arye Rubinstein. "1042 INTERFERONOPATHY IN THE BARE LYMPHOCYTE SYNDROME." Pediatric Research 19, no. 4 (April 1985): 284A. http://dx.doi.org/10.1203/00006450-198504000-01072.
Повний текст джерелаPiotto, Daniela Gerent Petry, Kátia Tomie Kozu, and Sandro Félix Perazzio. "Quando suspeitar de doenças autoinflamatórias na infância?" Reumatologia Pediátrica, no. 2021 jan-mar;20(1) (March 31, 2021): 16–26. http://dx.doi.org/10.46833/reumatologiasp.2021.20.1.16-26.
Повний текст джерелаCrow, Yanick, Nandaki Keshavan, Jacques Patrick Barbet, Geanina Bercu, Vincent Bondet, Charlotte Boussard, Nathalie Dedieu, et al. "Cardiac valve involvement in ADAR-related type I interferonopathy." Journal of Medical Genetics 57, no. 7 (November 26, 2019): 475–78. http://dx.doi.org/10.1136/jmedgenet-2019-106457.
Повний текст джерелаKozlova, A. L., E. S. Romanenko, V. I. Burlakov, E. V. Deripapa, Sv P. Khomyakova, A. N. Remizov, G. V. Tereshenko, and A. Yu Shcherbina. "CLINICAL CASE OF TYPE I INTERFERONOPATHY: AICARDI–GOUTIERES SYNDROME." Pediatria. Journal named after G.N. Speransky 98, no. 3 (June 10, 2019): 257–65. http://dx.doi.org/10.24110/0031-403x-2019-98-3-257-265.
Повний текст джерелаДисертації з теми "INTERFERONOPATIA"
König, Nadja. "Identifizierung und Charakterisierung neuer Typ-1-Interferonopathie-assoziierter Gene." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2017. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-225727.
Повний текст джерелаViengkhou, Barney. "Delineating the pathogenetic cellular response in type I interferonopathic brain disease." Thesis, University of Sydney, 2020. https://hdl.handle.net/2123/24137.
Повний текст джерелаLucas, Nadja [Verfasser], Min Ae [Akademischer Betreuer] [Gutachter] Lee-Kirsch, and Axel [Gutachter] Roers. "Identifizierung und Charakterisierung neuer Typ-1-Interferonopathie-assoziierter Gene / Nadja König ; Gutachter: Min Ae Lee-Kirsch, Axel Roers ; Betreuer: Min Ae Lee-Kirsch." Dresden : Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2017. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-225727.
Повний текст джерелаKönig, Nadja [Verfasser], Min Ae [Akademischer Betreuer] [Gutachter] Lee-Kirsch, and Axel [Gutachter] Roers. "Identifizierung und Charakterisierung neuer Typ-1-Interferonopathie-assoziierter Gene / Nadja König ; Gutachter: Min Ae Lee-Kirsch, Axel Roers ; Betreuer: Min Ae Lee-Kirsch." Dresden : Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2017. http://d-nb.info/1135907390/34.
Повний текст джерелаAhmed, S. A. Abu Tayeh. "Constitutive RIG-I activation causes skin lesion resembling psoriasis in transgenic mice." Doctoral thesis, Kyoto University, 2021. http://hdl.handle.net/2433/263796.
Повний текст джерелаPayet, Cloé. "Study of Interferon type I in Myasthenia Gravis." Thesis, Sorbonne université, 2021. http://www.theses.fr/2021SORUS517.
Повний текст джерелаMyasthenia gravis (MG) is an autoimmune disease mediated by autoantibodies against the acetylcholine receptor (AChR). The thymus of patients is the effector organ and is characterized by chronic overexpression of interferon (IFN)-β. My PhD aimed to understand the implication of IFN-I in MG. First, I demonstrated that no IFN-I signature is detected in serum and PBMC of patients. IFN-I signature is specific to the thymus and I investigated the cause of this overexpression. IFN-I is produced in response to pathogen infection but also in response to endogenous nucleic acids (eNA) in diseases, such as interferonopathies. I demonstrated that molecules mimicking eNA, such as double-stranded DNA or RNA induced the overexpression of IFN-β and of AChR in human thymic epithelial cells (TEC) or in the thymus of mice. As I was suspecting eNA to be released by necrotic cells, I induced thymocyte necrosis in in vitro or in vivo models. I then showed an increased IFN-I signature and the expression of AChR in TEC and in the thymus of mice treated with dexamethasone. In addition, I observed in the MG thymus a decrease in thymic macrophages, cells responsible for the clearance of apoptotic cells. In mice, depletion of thymic macrophages led to an increase in necrotic thymocytes associated with IFN-I and AChR expression. Consequently, I hypothesize that in the MG thymus, a decrease in the number of macrophages may alter the processing of apoptotic cells leading to the release of eNA from necrotic thymocytes. These eNA would activate innate immunity signaling pathways leading to the IFN-β signature which would induce thymic changes self-sensitization against AChR
Тези доповідей конференцій з теми "INTERFERONOPATIA"
Schorling, D. C., R. Kaur, S. Donkervoort, C. G. Bönnemann, A. Lee-Kirsch, J. Kirschner, M. Eckenweiler, and W. G. Janzarik. "Phenotypic Spectrum of PNPT1: Interferonopathy or Not?" In Abstracts of the 46th Annual Meeting of the Society for Neuropediatrics. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1739628.
Повний текст джерела