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Статті в журналах з теми "Inflammation de bas grade":
Yoo, Ji Youn, Sarah Sniffen, Kyle Craig McGill Percy, Veera Bramhachari Pallaval, and Bojjibabu Chidipi. "Gut Dysbiosis and Immune System in Atherosclerotic Cardiovascular Disease (ACVD)." Microorganisms 10, no. 1 (January 5, 2022): 108. http://dx.doi.org/10.3390/microorganisms10010108.
Remmel, Paul, and Valérie Freiche. "Entéropathies chroniques du chat, quelles spécificités ?" Le Nouveau Praticien Vétérinaire canine & féline 19 (December 2022): 60–67. http://dx.doi.org/10.1051/npvcafe/2023010.
Cooper, J., T. Dang, M. Reeson, J. Dang, L. Dieleman, K. Kroeker, D. Baumgart, et al. "A113 DIAGNOSTIC ACCURACY OF COMPUTED TOMOGRAPHY ENTEROGRAPHY COMPARED TO BALLOON ASSISTED ENDOSCOPY IN CROHN’S DISEASE." Journal of the Canadian Association of Gastroenterology 6, Supplement_1 (March 1, 2023): 61–62. http://dx.doi.org/10.1093/jcag/gwac036.113.
Divella, Rosa, Graziella Marino, Stefania Infusino, Laura Lanotte, Gaia Gadaleta-Caldarola, and Gennaro Gadaleta-Caldarola. "The Mediterranean Lifestyle to Contrast Low-Grade Inflammation Behavior in Cancer." Nutrients 15, no. 7 (March 29, 2023): 1667. http://dx.doi.org/10.3390/nu15071667.
Rutsch, Frank, and Robert Terkeltaub. "Inflammation de bas grade et déficit en inhibiteurs physiologiques de la calcification dans les calcifications des artères: mécanismes permettant de comprendre les calcifications du cartilage." Revue du Rhumatisme 72, no. 3 (March 2005): 186–94. http://dx.doi.org/10.1016/j.rhum.2004.05.021.
Hébert, Marine, Tristan Méric, and Juan Hernandez. "Entéropathies inflammatoires chroniques chez le chien : actualités." Le Nouveau Praticien Vétérinaire canine & féline 19 (December 2022): 50–58. http://dx.doi.org/10.1051/npvcafe/2023009.
Valadez-Bustos, Escamilla-Silva, García-Vázquez, Gallegos-Corona, Amaya-Llano, and Ramos-Gómez. "Oral Administration of Microencapsulated B. Longum BAA-999 and Lycopene Modulates IGF-1/IGF-1R/IGFBP3 Protein Expressions in a Colorectal Murine Model." International Journal of Molecular Sciences 20, no. 17 (August 31, 2019): 4275. http://dx.doi.org/10.3390/ijms20174275.
Raucci, Angela, Federica Macrì, Stefania Castiglione, Ileana Badi, Maria Cristina Vinci, and Estella Zuccolo. "MicroRNA-34a: the bad guy in age-related vascular diseases." Cellular and Molecular Life Sciences 78, no. 23 (October 26, 2021): 7355–78. http://dx.doi.org/10.1007/s00018-021-03979-4.
Hirano, F., D. Van der Heijde, F. A. Van Gaalen, R. B. M. Landewé, C. Gaujoux-Viala, and S. Ramiro. "SAT0375 DETERMINANTS OF PATIENT’S GLOBAL ASSESSMENT OF WELL-BEING IN EARLY AXIAL SPONDYLOARTHRITIS; 5-YEAR LONGITUDINAL DATA FROM THE DESIR COHORT." Annals of the Rheumatic Diseases 79, Suppl 1 (June 2020): 1135–36. http://dx.doi.org/10.1136/annrheumdis-2020-eular.816.
Desjardins, Clément, Khê Hoang-Xuan, and Caroline Houillier. "Lymphome de bas grade." Revue Neurologique 178 (April 2022): S143. http://dx.doi.org/10.1016/j.neurol.2022.02.009.
Дисертації з теми "Inflammation de bas grade":
Alentorn, Agusti. "Caractérisation génomique et génétique des gliomes diffus de bas grade de l’adulte." Thesis, Paris 11, 2014. http://www.theses.fr/2014PA11T011.
Multildimensional molecular characterization of tumors and more specifically of gliomas is of pivotal importance to identify: (i) new biomarkers (i.e. diagnostic, prognostic, theranostic or predisposing), (ii) new therapeutic targets and (iii) to improve our understanding of molecular oncogenesis.Our work has confirmed and consolidated previous data published in the literature, for example that: (i) 1p/19q co-deletion is associated with better prognosis, (ii) IDH mutation is associated with better prognosis, (iii) TP53 mutations and 1p/19q codeletion are mutually exclusive and (iv) PDGFRA is rarely altered, at genomic level, in low-grade gliomas (LGG).More originally, we have identified several genomic groups, with clinical and biological relevances, in LGG and more specifically in LGG without 1p/19q co-deletion: (i) 19q-deleted, (ii) 11p-deleted, (iii) 7-gained, (iv) 19-gained and (v) unclassified. Interestingly, 19q deletion abrogates the positive prognostic value of IDH mutation in LGG without 1p/19q codeletion.We have also identified new recurrent somatic gene mutations in LGG (i.e. TEP1 and RNF40 mutations), supporting the critical role of telomeres and chromatin remodelling in LGG.Finally, we have characterized further 11p-deleted LGG that exhibit mostly astrocytic phenotype and poor prognosis. This subgroup includes LGG overexpressing genes of inflammatory/immune cells (GIM -Glioma infiltrating microglia-, M1 macrophages and M2 macrophages) and infiltrated by macrophagic/microglial cells. This peculiar microenvironment detected in 11p-deleted LGG might be used as a therapeutic target. In conclusion, our work participates to characterize clinico-biological portrait of LGG and to describe a singular genomic subgroup of LGG characterized by 11p loss
Allouche, Rania. "Effet anti-inflammatoire d’hydrolysats de protéines de surface ou intracellulaires de Streptococcus thermophilus obtenus après action de protéases digestives." Electronic Thesis or Diss., Université de Lorraine, 2022. http://www.theses.fr/2022LORR0344.
Inflammation is a mechanism that provides protection against injury, trauma, or infection caused by damaged cells, irritants, or pathogens. This process removes harmful agents and damaged tissue components. Nevertheless, chronic low-grade inflammation is often associated with various pathologies. Diet could be a promising way of action. Indeed, bioactive peptides derived from the hydrolysis of dietary proteins could modulate key inflammatory factors and consequently delay the onset of these chronic diseases. Furthermore, lactic acid bacteria, components of fermented milk products, exhibit anti-inflammatory properties both in vitro and in vivo studies. Among them, Streptococcus thermophilus (ST) is regularly consumed by a significant part of the population. Studies have shown that some strains of ST display anti inflammatory activity in vitro with an unknown mechanism of action. In this work, it was hypothesized that peptides released after hydrolysis by digestive proteases of the surface or intracellular proteins of this bacterium could be involved at least partially in this activity. Firstly, hydrolysates were obtained by shaving surface proteins with trypsin or pepsin followed or not by trypsinolysis. The tandem mass spectrometry analysis indicated that the majority of the identified peptides belonged to the surface proteins of this bacterium. Secondly, the anti inflammatory activity of the hydrolysates was evaluated in two inflamed cell models. The hydrolysate obtained after tryptic shaving and trypsinolysis of surface proteins of ST LMD 9 significantly decreased the secretion of the pro-inflammatory cytokine IL 8 in lipopolysaccharide (LPS) stimulated HT 29 cells. The same hydrolysate also reduced production of IL 8 and of the pro-inflammatory cytokine IL 1β as well as protein expression levels of Pro IL 1β and COX 2 in LPS-stimulated THP 1 macrophages. It was proposed that the surface protease PrtS could be a source of active peptides during gastrointestinal digestion. To verify this hypothesis, hydrolysates were prepared by shaving with pepsin followed or not by trypsinolysis of the surface proteins of two phenotypically distinct strains of ST: LMD 9 (PrtS+) and CNRZ 21N (PrtS-). Modulation of pro-inflammatory mediators IL 8, IL 1β, Pro IL 1β and COX 2 was assessed in LPS-stimulated THP 1 macrophages and IL 8 in LPS stimulated HT 29 cells. The hydrolysates from the two strains showed an anti inflammatory action but modulation of all these inflammatory mediators was strain, hydrolysate, and concentration dependent. Interestingly, the strain lacking PrtS also showed anti-inflammatory activity. Therefore, peptides released from surface proteins of ST strains by proteases of the gastrointestinal tract during digestion of a product containing this bacterium could exert anti-inflammatory effects and thus could reduce the risk of inflammation related chronic diseases. Finally, the intracellular proteins of the LMD 9 and CNRZ 21N strains were recovered by sonication and hydrolysed with Corolase PP, a mixture of pancreatic proteases. Hydrolysates generated from a fraction of these proteins of both strains demonstrated anti inflammatory action by modulating some of the pro-inflammatory mediators in LPS stimulated THP 1 macrophages. To our knowledge, this is the first study demonstrating the anti inflammatory activity of peptides derived from surface proteins and a fraction of the intracellular proteins of ST strains. These paraprobiotics or postbiotics, likely to be released in the digestive tract of the consumer, could participate in the overall anti inflammatory effect of S. thermophilus which had been demonstrated with certain strains. They could display beneficial effects on human health and therefore could be a promising bioactive ingredient for the development of novel functional foods for the prevention of low grade inflammation
Lasselin, Julie. "L’inflammation chronique à bas bruit et ses relations avec la fatigue et les altérations cognitives chez les patients souffrant de troubles métaboliques." Thesis, Bordeaux 2, 2012. http://www.theses.fr/2012BOR22001/document.
Cytokines produced during the activation of the immune system have the ability to act within the central nervous system and to induce a large number of behavioral alterations. When the activation of immune system becomes chronic and unregulated, these behavioral alterations may lead to the development of neuropsychiatric symptoms. The pathophysiology of neuropsychiatric symptoms that develop in conditions of chronic low-grade inflammation context (i.e., characterized by a chronic but low activation of inflammatory processes), remains unknown. The main aim of this thesis was to investigate the involvement of low-grade inflammation in the development of fatigue symptoms and cognitive alterations in patients with metabolic disorders including obesity and type 2 diabetes. These conditions are characterized by a chronic low-grade inflammatory state, manifesting by higher blood concentrations of inflammatory factors. This inflammatory state would originate, at least partially, from the adipose tissue. Moreover, fatigue symptoms and cognitive alterations are common in metabolic disorders. Given the role of inflammation in the physiopathology of these symptoms, their development could also rely on chronic low-grade inflammatory processes. Several objectives were defined to test this hypothesis: 1) to characterize fatigue symptoms and cognitive alterations in obese and diabetic patients; 2) to assess the relationship of systemic inflammation with the inflammatory state of the adipose tissue; and 3) to investigate the association of low-grade inflammation with fatigue symptoms and cognitive alterations in patients with metabolic disorders. Fatigue symptoms and cognitive function were respectively assessed using the multidimensional fatigue inventory (MFI) and the neuropsychological tests automated battery CANTAB in diabetic patients (type 1 and type 2) and in obese patients before and after bariatric surgery. A control group was included for each model (obesity and type 2 diabetes). Circulating concentrations of inflammatory markers, as well as expression of inflammatory markers in the visceral adipose tissue of obese patients, were measured. Our results indicate that fatigue symptoms, especially in the dimensions of general and physical fatigue, represent fundamental characteristics of patients suffering from metabolic disorders. In addition, cognitive alterations (psychomotor slowing and alterations in spatial planning performance) were measured in type 2 diabetic patients, more particularly those under insulin treatment, and in obese patients. Slight alterations in the test of backward spatial span were measured in obese patients. With respect to biological data, our results indicate significant relationships between systemic inflammation and inflammatory markers (inflammatory cytokines, including MCP1, and T-cell markers) in the visceral adipose tissue of obese patients. Interestingly, chronic low-grade inflammation was associated with fatigue symptoms (general fatigue, mental fatigue, reduced activity and motivation) and performance alterations in tests assessing executive functions. Altogether, these data support the hypothesis of the involvement of the adipose macrophages and T lymphocytes in the systemic inflammatory state associated with obesity. Moreover, these results suggest that systemic low-grade inflammation associated with metabolic disorders may contribute to the physiopathology of fatigue and cognitive alterations in these conditions. In conclusion, these studies provide a precise characterization of fatigue symptoms and cognitive alterations associated with metabolic disorders, such as obesity or type 2 diabetes. In addition, this thesis work gives interesting information about the relationships of chronic low-grade inflammation and fatigue and cognitive symptoms, and refines hypotheses regarding the involvement of inflammatory processes in the physiopathology of these symptoms in patients with diabetes or obesity
Pépin, Léopold. "Astrocytomes de bas-grade : apport de l'imageri moderne." Montpellier 1, 1992. http://www.theses.fr/1992MON11055.
Rorive, Sandrine. "Les astrocytomes de bas-grade: caractérisation moléculaire et implications cliniques." Doctoral thesis, Universite Libre de Bruxelles, 2010. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210181.
Le but des études entreprises au cours de ce travail de thèse est d’apporter une contribution à la compréhension des mécanismes de tumorigenèse qui différencient l’astrocytome de grade I des astrocytomes diffus (grade II-IV), de manière à identifier des voies biologiques qui permettraient, au moins en partie, d’expliquer ces différences de comportement.
Au cours de la première partie de ce travail, nous avons caractérisé les profils d’expression génomique des astrocytomes de grade I et de grade II, en comparant les données d’expression de gènes (évaluées par des technologies de micropuces d’ADN) de travaux publiés entre 2000 et 2005. L’expression des gènes identifiés a été validée par des analyses de RT-PCR quantitative sur une série indépendante d’astrocytomes de grade I, II et IV. Les fonctions biologiques des protéines codées par chacun de ces gènes ont fait l’objet de recherches bibliographiques détaillées afin de proposer un modèle permettant d’approcher les différences de comportement de ces tumeurs. Cette analyse nous a permis d’identifier TIMP4 (tissue inhibitor of metalloproteinases 4) et IGFBP2 (insulin-like growth factor binding protein 2) comme gènes candidats pour améliorer la caractérisation biologique et clinique des astrocytomes de grade I par rapport aux astrocytomes diffus. TIMP4 et IGFBP2 codent respectivement pour un inhibiteur endogène des métalloprotéinases matricielles (MMPs) et une protéine de liaison capable d’inhiber l’action des « insulin-like growth factors » (IGFs, dont IGFI et IGFII), des facteurs impliqués dans la croissance et la migration des astrocytes normaux et tumoraux.
Sur base de la surexpression de TIMP4 et d’IGFBP2 dans les astrocytomes de grade I, en comparaison aux astrocytomes diffus de grade II, nous avons posé l’hypothèse suivante :« L’absence d’agressivité des astrocytomes de grade I, en comparaison aux astrocytomes diffus (grade II-IV) pourrait en partie être liée à l’inhibition par TIMP-4 de la protéolyse des complexes IGFBP2-IGFII au sein de ces tumeurs ». Cette protéolyse, qui diminue l’affinité d’IGFBP2 pour IGFII, pourrait contribuer à libérer IGFII dans la matrice extracellulaire (MEC), favoriser la liaison d’IGFII à son récepteur IGF-IR et stimuler la croissance et la migration des cellules astrocytaires tumorales. Pour tester cette hypothèse, nous avons réalisé différentes analyses biochimiques afin i) de caractériser les actions protéolytiques de MMP-2, MMP-9 et MT1-MMP sur le complexe IGFBP2-IGFII, ii) d’identifier la libération d’IGFII lors du clivage de ce complexe, et iii) d’étudier l’action inhibitrice de TIMP-4. A l’aide d’un modèle cellulaire in vitro (lignée astrocytaire tumorale LN229), nous avons ensuite observé l’influence de la protéolyse du complexe IGFBP2-IGFII sur la croissance et la motilité cellulaire. Cette étude a montré :(1) la protéolyse du complexe IGFBP2-IGFII par MMP-9, (2) l’inhibition partielle de cette protéolyse par TIMP-4, (3) la libération d’IGFII résultant de cette protéolyse et (4) les effets stimulants de la libération d’IGFII sur la croissance et la motilité des cellules LN229. Cette étude souligne le rôle important de la protéolyse des complexes IGFBP2-IGFII dans l’agressivité des astrocytomes diffus. Elle confirme les effets stimulants propres d’IGFII, d’IGFBP2 et de MMP-9 sur la motilité et/ou la croissance des cellules astrocytaires tumorales. Enfin, elle identifie un rôle inhibiteur potentiel de TIMP-4 sur la protéolyse du complexe IGFBP2-IGFII, qui pourrait contribuer à expliquer le caractère plus indolent des astrocytomes de grade I en comparaison aux astrocytomes diffus.
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Doctorat en Sciences médicales
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Gerin, Chloé. "Modélisation et étude histologique de gliomes diffus de bas grade." Phd thesis, Université Paris-Diderot - Paris VII, 2012. http://tel.archives-ouvertes.fr/tel-00820353.
Gerin, Chloé. "Modélisation et études histologiques de gliomes diffus de bas grade." Paris 7, 2012. http://www.theses.fr/2012PA077066.
Diffuse low-grade gliomas (LGG) are primary brain tumors. After a slow growth, they evolve to high-grade gliomas, resulting into death. These tumors are very diffuse, thus diffîcult to treat. A better knownledge of them could allow to cure them or, failing that, to optimize treatments. We studied the growth of LGG with a simple mathematical model, which led us to speculate (i) that they arise in adolescence, (ii) that the age of the tumor at diagnosis can be calculated easily, and (iii) that the growth rate is an important prognostic factor. This last prediction is consistent with clinical observations. To test this spatial model, we have quantitatively characterized biopsy tissues of human LGG, particularly the presence of edema. The microscopic analysis of these data underpins the idea that edema is the cause of the abnormality seen on T2-weighted MR imaging. To take this new result into account, we have incorporated edema into the initial model as a consequence of the presence of tumor cells. This model helps explain the long decay of the tumor radius for tens of months after radiation therapy: as tumor cells become less numerous, drainage of the edema becomes predominant. This model, which has only three free parameters, has been validated thanks to clinical data from twenty patients
Darlix, Amélie. "Gliomes diffus de bas grade : données épidémiologiques et hypothèses étiologiques." Thesis, Montpellier, 2016. http://www.theses.fr/2016MONTT011/document.
The epidemiology and risks factors of diffuse low-grade gliomas (DLGG, or WHO grade II diffuse gliomas) are yet poorly known. This thesis aimed at describing the epidemiology (incidence rates, demographic data) and at looking for arguments in favor of environmental, functional and molecular risk factors, in the literature and by our works. Descriptive epidemiology: The analysis of an exhaustive series of incident cases of DLGG diagnosed between 2006 and 2011 allowed the determination of DLGG incidence (incidence rate standardized on the French population: 0,775/105 person-years) as well as that of each histological subtype described by the 2007 WHO classification. Environmental risk factors: We were able to demonstrate significant differences in the geographical distribution of WHO grade II and III diffuse gliomas in metropolitan France, with higher incidence rates in the North-East and Center regions. This heterogeneity stands in favor of environmental risk factors, even though there is to date no proven environmental risk factor for DLGG. Biological risk factors: Our work demonstrated the existence of a clear dichotomy, regarding molecular biology, between frontal DLGG, more frequently IDH-mutated and 1p19q codeleted, and temporo-insular tumors, less frequently IDH-mutated and 1p19q codeleted, suggesting different gliomagenesis pathways for these two patterns of tumors. Functional risk factors: Finally, data from the literature provide two main arguments in favor of the existence of functional risk factors in DLGG. First, the intra-cerebral location of these tumors is specific and distinct from that of other gliomas, with a preferential implication of “functional” areas. Second, macroscopic intra-cerebral changes have been reported following training on specific tasks, or in relation with a specific expertise. The microscopic mechanisms that underlie these modifications are uncertain but an implication (direct or indirect) of glial cells seems probable, and could favor gliomagenesis. To date, only few studies have investigated the correlation between the subject’s activity and the risk of DLGG. We thus propose, following this thesis, a case-control study to further investigate this issue. In conclusion, even though there is no demonstrated risk factor for DLGG, data from the literature, and conclusions from the present work, suggest the implication of environmental, functional and biological factors in DLGG genesis
Lopes, Manuel. "Chirurgie des gliomes de bas grade en zone fonctionnelle : réflexions éthiques." Paris 5, 2002. http://www.theses.fr/2002PA05N116.
Therapeutic controversies abouy low-grade gliomas arise both from the difficulties in clearly defining them from a histological point of view and from the subséquent imperfect knowledge of the natural history of the disease. The surgical attitude is thus heterogeneous ( from abstention to maximal possible resection involving in some cases functional areas), resulting from the difficulty in determining the best indication for each individual patient ; this lack of consensus generates several ethical issues. This fact has been assessed both practically
Claessens, Yann-Erick. "Etude de la dysérythropoièse au cours des syndromes myélodysplasiques de bas grade." Paris 5, 2004. http://www.theses.fr/2004PA05P632.
Anemia is a common feature in low grade myelodysplastic syndromes (MDS). However mechanisms responsible for this are unresolved. We describe a model of liquid cultures allowing the in vitro development of the erythroid lineage from CD34+ cells from MDS and control patients. We found an increased apoptosis and a decrease in expansion properties in MDS cultures. The Fas-Fas ligand system was responsible for the impairment of the erythroid development. We modulate this apoptotic signalling via inhibition of caspase-8 using a dominant negative of the adapter FADD, thus openening new therapeutic pathways. Significance of the enhanced apoptotic pathway mediated by Fas remains unresolved so far
Книги з теми "Inflammation de bas grade":
Capacchione, Vito. Infiammazione Silente (low-Grade Inflammation). Independently Published, 2018.
Tunjic, Luka. Mechanical Stimulation Low-Grade Inflammation Weight Gain: Muscles Upward Lifting Activity Weight Loss. Independently Published, 2021.
music theory in practice grade 1. abrms, 1991.
Taylor, Eric. Music Theory In Practice Grade 1. Associated Board of the Royal School of Music, 1990.
Carrero, Juan Jesús, and Peter Stenvinkel. The role of inflammation in chronic kidney disease. Edited by David J. Goldsmith. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0110.
Lafeber, Floris P. J. G., Nick J. Besselink, and Simon C. Mastbergen. Synovium and capsule. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199668847.003.0006.
Schiller, Adalbert, Adrian Covic, and Liviu Segall. Chronic tubulointerstitial nephritis. Edited by Adrian Covic. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0086_update_001.
Sahetya, Sarina. Acute Uncomplicated Bronchitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0029.
Tobar, Ximena, and Shannon B. Putman. Viral Gastroenteritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0030.
Частини книг з теми "Inflammation de bas grade":
Wang, Lihui, Xianhua Yu, and Jun-Ping Liu. "Telomere Damage Response and Low-Grade Inflammation." In Regulation of Inflammatory Signaling in Health and Disease, 213–24. Singapore: Springer Singapore, 2017. http://dx.doi.org/10.1007/978-981-10-5987-2_10.
Das, Undurti N. "Metabolic Syndrome X and low-grade systemic inflammation." In A Perinatal Strategy For Preventing Adult Disease: The Role Of Long-Chain Polyunsaturated Fatty Acids, 61–93. Boston, MA: Springer US, 2002. http://dx.doi.org/10.1007/978-1-4419-8564-4_6.
Das, Undurti N. "Low-grade Systemic Inflammation is Present in Common Diseases/Disorders." In Molecular Basis of Health and Disease, 175–79. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0495-4_6.
Piuri, Gabriele. "Individual Food Clusters Excess and Low-Grade Inflammation in Pregnancy." In Metabolic Syndrome and Complications of Pregnancy, 23–33. Cham: Springer International Publishing, 2015. http://dx.doi.org/10.1007/978-3-319-16853-1_2.
Ortega, Eduardo, Isabel Gálvez, and Leticia Martín-Cordero. "Extracellular Hsp70 and Low-Grade Inflammation- and Stress-Related Pathologies." In Heat Shock Proteins and Stress, 13–38. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-90725-3_2.
Fioranelli, Massimo, Marco Del Prete, Jahaira Carolina Aracena, Maria Grazia Roccia, Carlo Dal Lin, and Claudio Tomella. "Low-Dose Therapy for the Treatment of Low-Grade Chronic Inflammation." In Integrative Cardiology, 27–38. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-40010-5_3.
Guarner, Verónica, and Maria Esther Rubio-Ruiz. "Low-Grade Systemic Inflammation Connects Aging, Metabolic Syndrome and Cardiovascular Disease." In Interdisciplinary Topics in Gerontology, 99–106. Basel: S. KARGER AG, 2014. http://dx.doi.org/10.1159/000364934.
Daly, Robin M. "Dietary Factors and Chronic Low-Grade Systemic Inflammation in Relation to Bone Health." In Nutrition and Bone Health, 659–80. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-2001-3_39.
Daly, Robin M. "Nutrition, Aging, and Chronic Low-Grade Systemic Inflammation in Relation to Osteoporosis and Sarcopenia." In Nutritional Influences on Bone Health, 1–18. London: Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-2769-7_1.
Das, Undurti N. "Adult Diseases and Low-Grade Systemic Inflammation Have Their Origins in the Perinatal Period." In Molecular Basis of Health and Disease, 513–49. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0495-4_16.
Тези доповідей конференцій з теми "Inflammation de bas grade":
Campana, F., U. Ugo Ordioni, J. Del Grande, and N. Fakhry. "Adénocarcinome polymorphe de bas grade : A propos d’une localisation labiale." In 62ème Congrès de la SFCO. Les Ulis, France: EDP Sciences, 2014. http://dx.doi.org/10.1051/sfco/20146203008.
Lautenbach, Anne, Fabian Stoll, Oliver Mann, Philipp Busch, Tobias B. Huber, Heike Kielstein, Ina Bähr, and Jens Aberle. "65 Langfristige Verbesserung der chronischen Low-Grade-Inflammation nach bariatrischer Chirurgie." In Adipositas-Kongress 2021 – 37. Jahrestagung der Deutschen Adipositas-Gesellschaft e.V. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1735687.
Lautenbach, Anne, Fabian Stoll, Oliver Mann, Philipp Busch, Tobias B. Huber, Heike Kielstein, Ina Bähr, and Jens Aberle. "65 Langfristige Verbesserung der chronischen Low-Grade-Inflammation nach bariatrischer Chirurgie." In Adipositas-Kongress 2021 – 37. Jahrestagung der Deutschen Adipositas-Gesellschaft e.V. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1735687.
van den Borst, Bram, Harry R. Gosker, Geertjan Wesseling, Wilco de Jager, Valery A. C. V. Hellwig, Frank J. Snepvangers, and Annemie M. W. J. Schols. "Low-Grade Adipose Tissue Inflammation In Patients With Mild-To-Moderate COPD." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4124.
Wopereis, Suzan, Willem J. van den Brink, Tim J. van den Broek, Wilrike J. Pasman, and Femke P. M. Hoevenaars. "Postprandial Composite Biomarkers of Low-Grade Inflammation to Evaluate Nutritional Intervention Effects." In European Nutrition Conference. Basel Switzerland: MDPI, 2024. http://dx.doi.org/10.3390/proceedings2023091141.
Tabei, S., L. Scheffler, R. Chakaroun, S. Ziesche, A. Crane, M. Stumvoll, A. Dietrich, M. Blüher, M. Gericke, and P. Kovacs. "Low-grade inflammation in insulin resistance associates with bacterial load in adipose tissue." In Diabetes Kongress 2019 – 54. Jahrestagung der DDG. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1688193.
Leipe, Jan, Anna-Isabelle Kaelsch, Marcus Kleber, Hubert Scharnagl, Wolfgang Sattler, Winfried März, Ernst Malle, and Bernhard Kraemer. "FRI0660 LONG- AND SHORT-TERM ASSOCIATION OF LOW-GRADE SYSTEMIC INFLAMMATION WITH CARDIOVASCULAR MORTALITY." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.7919.
Horishnyi, I. M., and O. V. Chernyshov. "Low-grade inflammation in patients with asthma and risks of SARS-CoV-2 infection." In MODERN MEDICINE: THE USE OF CREATIVE INDUSTRIES IN THE HEALTHCARE SYSTEM. Baltija Publishing, 2021. http://dx.doi.org/10.30525/978-9934-26-182-4-7.
Lo Tam Loi, Adèle T., Susan Hoonhorst, nick hacken ten, J. W. J. Lammers, and Leo Koenderman. "Low Body Mass Index Is Associated With Low Grade Systemic Inflammation In Stable COPD Patients." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2553.
Fornasiero, Massimiliano, Iain Pierce, Matthew Webber, Kathryn E. Keenan, Karl F. Stupic, Rüdiger Bruehl, Bernd Ittermann, et al. "11 A medical device-grade T2 phantom for quality assurance of inflammation imaging by CMR." In British Society of Cardiovascular Magnetic Resonance 2021 Annual Meeting. BMJ Publishing Group Ltd and British Cardiovascular Society, 2021. http://dx.doi.org/10.1136/heartjnl-2021-bscmr.11.
Звіти організацій з теми "Inflammation de bas grade":
Akkus, Ozan. Regeneration of Musculoskeletal Tissues by Prolonged Low-Grade Inflammation. Fort Belvoir, VA: Defense Technical Information Center, October 2011. http://dx.doi.org/10.21236/ada562974.
Yang, Chao, Tao-junjin Lu, Jie Wang, Qing Zhang, Ze-Fen Wang, and Zhi-Qiang Li. Association of systemic immune-inflammation index with grade and prognosis in glioma patients: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0072.
Mateș, Letiția, Marius Emil Rusu, Ionel Fizeșan, Daniela-Saveta Popa, and Daniel Leucuța. Walnut intake interventions targeting biomarkers of metabolic syndrome and inflammation in middle-aged and older adults: a systematic review and meta-analysis of randomized controlled trials research protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, June 2022. http://dx.doi.org/10.37766/inplasy2022.6.0058.
Platz, Elizabeth A. Prospective Evaluation of Intraprostatic Inflammation and Focal Atrophy as a Predictor of Risk of High-Grade Prostate Cancer and Recurrence after Prostatectomy. Fort Belvoir, VA: Defense Technical Information Center, July 2014. http://dx.doi.org/10.21236/ada610016.
Platz, Elizabeth A. Prospective Evaluation of Intraprostatic Inflammation and Focal Atrophy as a Predictor of Risk of High-Grade Prostate Cancer and Recurrence after Prostatectomy. Fort Belvoir, VA: Defense Technical Information Center, July 2013. http://dx.doi.org/10.21236/ada593262.
Schreiber, Martin A. The Effect of Hypotensive Resuscitation and Fluid Type on Mortality, Bleeding, Coagulation and Dysfunctional Inflammation in a Swine Grade V Liver Injury Model. Fort Belvoir, VA: Defense Technical Information Center, January 2008. http://dx.doi.org/10.21236/ada490734.
Schreiber, Martin. The Effect of Hypotensive Resuscitation and Fluid Type on Mortality, Bleeding, Coagulation, & Dysfunctional Inflammation in a Swine Grade V Liver Injury Model. Fort Belvoir, VA: Defense Technical Information Center, January 2006. http://dx.doi.org/10.21236/ada446801.
Schreiber, Martin A. The Effect Of Hypotensive Resuscitation And Fluid Type On Mortality, Bleeding,Coagulation And Dysfunctional Inflammation In A Swine Grade V Liver Injury Model. Fort Belvoir, VA: Defense Technical Information Center, January 2012. http://dx.doi.org/10.21236/ada559947.
Liu, Yangjun, Wei Xie, Zbigniew Ossowski, Juan Li, Juan Yang, Yiming Luo, Xia Wu, and Liying Liu. Physical activity, abdominal obesity and inflammatory response in the elderly: a systematic review and meta-analysis of randomized-controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, March 2023. http://dx.doi.org/10.37766/inplasy2023.3.0051.
Czerwaty, Katarzyna, Karolina Dżaman, Krystyna Maria Sobczyk, and Katarzyna Irmina Sikrorska. The Overlap Syndrome of Obstructive Sleep Apnea and Chronic Obstructive Pulmonary Disease: A Systematic Review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2022. http://dx.doi.org/10.37766/inplasy2022.11.0077.