Добірка наукової літератури з теми "IL-10 deficiency"
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Статті в журналах з теми "IL-10 deficiency"
Asadullah, K., K. Stephanek, M. Leupold, D. Jasulaitis, H. Audring, H. D. Volk, W. D. Döcke, and W. Sterry. "Relative IL-10 deficiency and effects of IL-10 therapy in psoriasis." Journal of Dermatological Science 16 (March 1998): S30. http://dx.doi.org/10.1016/s0923-1811(98)83173-3.
Повний текст джерелаWan, Xin, Wen Juan Huang, Wen Chen, Hong-Guang Xie, Pan Wei, Xin Chen, and Chang-Chun Cao. "IL-10 Deficiency Increases Renal Ischemia-Reperfusion Injury." Nephron Experimental Nephrology 128, no. 1-2 (October 31, 2014): 37–45. http://dx.doi.org/10.1159/000366130.
Повний текст джерелаHennessy, A., H. L. Pilmore, L. A. Simmons, and D. M. Painter. "A Deficiency of Placental IL-10 in Preeclampsia." Journal of Immunology 163, no. 6 (September 15, 1999): 3491–95. http://dx.doi.org/10.4049/jimmunol.163.6.3491.
Повний текст джерелаZigmond, Ehud, Biana Bernshtein, Gilgi Friedlander, Catherine R. Walker, Simon Yona, Ki-Wook Kim, Ori Brenner, et al. "Macrophage-Restricted Interleukin-10 Receptor Deficiency, but Not IL-10 Deficiency, Causes Severe Spontaneous Colitis." Immunity 40, no. 5 (May 2014): 720–33. http://dx.doi.org/10.1016/j.immuni.2014.03.012.
Повний текст джерелаJones, Steven P., Steven D. Trocha, and David J. Lefer. "Cardioprotective actions of endogenous IL-10 are independent of iNOS." American Journal of Physiology-Heart and Circulatory Physiology 281, no. 1 (July 1, 2001): H48—H52. http://dx.doi.org/10.1152/ajpheart.2001.281.1.h48.
Повний текст джерелаGunnett, Carol A., Donald D. Heistad, Daniel J. Berg, and Frank M. Faraci. "IL-10 deficiency increases superoxide and endothelial dysfunction during inflammation." American Journal of Physiology-Heart and Circulatory Physiology 279, no. 4 (October 1, 2000): H1555—H1562. http://dx.doi.org/10.1152/ajpheart.2000.279.4.h1555.
Повний текст джерелаWang, B., L. Zhuang, H. Fujisawa, and D. N. Sauder. "Effect of IL-10 deficiency on the migration of epidermal Langerhans cells in IL-10 null mice." Journal of Dermatological Science 16 (March 1998): S90. http://dx.doi.org/10.1016/s0923-1811(98)83537-8.
Повний текст джерелаZhang, Guag-Xian, Fang Zhou, Bogoljub Ciric, Hongmei Li, Yaping Yan, Ke Li, Melissa Cullimore, et al. "Ability of LPS to regulate expression of tolerance-related molecules on dendritic cells is blocked by IL-10 deficiency (48.18)." Journal of Immunology 188, no. 1_Supplement (May 1, 2012): 48.18. http://dx.doi.org/10.4049/jimmunol.188.supp.48.18.
Повний текст джерелаPérez-de Puig, Isabel, Francesc Miró, Angélica Salas-Perdomo, Ester Bonfill-Teixidor, Maura Ferrer-Ferrer, Leonardo Márquez-Kisinousky, and Anna M. Planas. "IL-10 Deficiency Exacerbates the Brain Inflammatory Response to Permanent Ischemia without Preventing Resolution of the Lesion." Journal of Cerebral Blood Flow & Metabolism 33, no. 12 (September 11, 2013): 1955–66. http://dx.doi.org/10.1038/jcbfm.2013.155.
Повний текст джерелаMarco, Roberto Di, Ming Xiang, Paola Zaccone, Camilla Leonardi, Sabrina Franco, Pierluigi Meroni, and Ferdinando Nicolettl. "Concanavalin A-induced Hepatitis in Mice is Prevented by Interleukin (IL)-10 and Exacerbated by Endogenous IL-10 Deficiency." Autoimmunity 31, no. 2 (January 1999): 75–83. http://dx.doi.org/10.3109/08916939908994050.
Повний текст джерелаДисертації з теми "IL-10 deficiency"
Riquelme, Neira Roberto Andrés. "Influencia de la deficiencia de Il-10 sobre el fenotipo mastocitario, la microbiota y la respuesta inmune intestinal en un modelo murino." Doctoral thesis, Universitat Autònoma de Barcelona, 2018. http://hdl.handle.net/10803/664251.
Повний текст джерелаMast cells (MC) can participate in the response to microorganisms by various pattern recognition receptors (PRRs). After their activation through these receptors, MC can orchestrate a response by secreting immunological mediators such as cytokines. Among these, interleukin 10 (IL-10) is an important cytokine due to its immunomodulatory characteristics, as well as its ability to regulate the expression of MC proteases. Additionally, thanks to the existence of genetically modified murine models, such as IL-10 deficient (IL-10-/-) mice that develop colitis spontaneously, it is possible to investigate the potential role of IL-10 in MC response to activation with antigens of different microorganisms. On the other hand, the use of this animal model allows investigating the influence of this cytokine on the composition of the intestinal microbiota. This work has explored the functional role of IL-10 in differentiated MC in vitro, as well as the effects of IL-10 deficiency on the composition of the microbiota and the expression of factors related to the immune response, before (6 weeks) and at the onset (20 weeks) of colitis. For this purpose, the effect of IL-10 deficiency has been characterized on MC of different phenotype and after its activation via PRRs. Additionally, the effect produced by the lack of IL-10 on the microbiota composition, the expression of TLRs and proinflammatory cytokines, as well as the production of luminal IgA, in the same stages and after antibiotics treatment was evaluated. The results obtained indicated that the IL-10 deficiency produced different effects depending on the MC phenotype, age and type of PRR ligand. Thus, in the absence of IL-10, mucosal-like MC (MLMC) showed lower expression of TLR4 and NOD2 at week 6 and TLR7 at week 20. In addition, both MC phenotypes (mucosa and connective), showed a lower secretion of IL-6 and TNFα after TLR2 activation. The TLR4 and TLR7 activation in MLMC generated a lower secretion of IL-6 at week 6, while MLMC secreted less TNFα at week 20. Finally, after NOD2 stimulation, no cytokine secretion was observed in any of the MC phenotypes. On the other hand, it was observed that in IL-10-/- animals there are factors that potentially favor the development of colitis. Thus, IL-10-/- mice at week 6 showed representatives of Verrucomicrobia phylum and a lower relative abundance of Rikenellaceae and Lachnospiraceae taxa. Meanwhile at week 20 in IL-10-/- mice, microorganisms of the phylum TM7 were observed, as well as, a lower expression of IL-1β, IL-6, TLR6, -7 and -8, and an increase of TNFα and IgA. Additionally, the use of antibiotics before the development of colitis induced a decrease in diversity and a restructuring of the microbiota, together with a decrease in TLRs and cytokines expression, and a lower production of luminal IgA. In summary, these findings provide new insights on the role of MC and IL-10 in the host-microorganism interaction. They show how the IL-10 deficiency can affect the microbiota composition and the expression of factors associated with the immune response. And they suggest that early modification of the microbiota through the use of antibiotics in genetically susceptible individuals could alter the colitis progression.
Arrieta, Mendez Marie Claire. "The role of small intestinal permeability in the pathogenesis of colitis in the interleukin-10 gene deficient mouse." Phd thesis, 2011. http://hdl.handle.net/10048/1848.
Повний текст джерелаExperimental Medicine
Tso, Victor Key. "Nuclear Magnetic Resonance metabolomic fingerprint of the Interleukin 10 gene deficient mouse model of Inflammatory Bowel Disease." Master's thesis, 2010. http://hdl.handle.net/10048/1450.
Повний текст джерелаExperimental Medicine
Частини книг з теми "IL-10 deficiency"
Jyonouchi, Harumi, and Lee Geng. "Associations between Monocyte Cytokine Profiles and Co-Morbid Conditions in Autism Spectrum Disorders." In Autism Spectrum Disorder - Profile, Heterogeneity, Neurobiology and Intervention. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95548.
Повний текст джерелаТези доповідей конференцій з теми "IL-10 deficiency"
Zhang, Wanying, Jie Liao, Haonan Li, Hua Dong, Han Bai, Allison Liao Yang, Bruce D. Hammack, and Guang-Yu Yang. "Abstract 1006: Soluble epoxide hydrolase gene deficiency attenuates colitis-induced carcinogenesis in IL-10 knockout mice." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-1006.
Повний текст джерелаLendermon, E., X. Wang, I. Popescu, A. Salaney, M. Helfers, M. Brown, K. Noda, J. Yu, and J. F. McDyer. "IL-10 Deficiency in Mice Results in Accelerated Airway Rejection but Is Not Required for Costimulation Blockade-Induced Lung Allograft Acceptance." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a1005.
Повний текст джерелаMcGuire, John K., Cliff Rims, and Xiaodong Zhu. "Delayed Recovery After Acute P. Aeruginosa Pneumonia In CD103-Null Mice Is Associated With Deficiency Of IL-10 Producing T Cells In BAL And Lung." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3694.
Повний текст джерелаSantos, Daniel Oliveira, CHRISTIAN WALLACE SANTOS MENESES, MELISSA VIEIRA GOMES, and LARISSA EMILY OGANDO DE JESUS SENA. "AS RESPOSTAS DA IMUNIDADE INATA NO PACIENTE COM CANDIDÍASE MUCOCUTÂNEA CRÔNICA." In II Congresso Brasileiro de Imunologia On-line. Revista Multidisciplinar em Saúde, 2022. http://dx.doi.org/10.51161/ii-conbrai/6752.
Повний текст джерелаЗвіти організацій з теми "IL-10 deficiency"
Ficht, Thomas, Gary Splitter, Menachem Banai, and Menachem Davidson. Characterization of B. Melinensis REV 1 Attenuated Mutants. United States Department of Agriculture, December 2000. http://dx.doi.org/10.32747/2000.7580667.bard.
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