Добірка наукової літератури з теми "Id2 inhibiteur de E2A"
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Статті в журналах з теми "Id2 inhibiteur de E2A"
Nigten, Jeannet, Gorica Nikoloski, Theo De Witte, Bert A. Van der Reijden, and Joop H. Jansen. "Id1 and Id2 Are Retinoic Acid Responsive Genes and Induce a G0/G1 Arrest in Acute Promyelocytic Leukemia Cells." Blood 104, no. 11 (November 16, 2004): 2029. http://dx.doi.org/10.1182/blood.v104.11.2029.2029.
Повний текст джерелаGonda, Hiroyuki, Manabu Sugai, Yukiko Nambu, Tomoya Katakai, Yasutoshi Agata, Kazuhiro J. Mori, Yoshifumi Yokota, and Akira Shimizu. "The Balance Between Pax5 and Id2 Activities Is the Key to AID Gene Expression." Journal of Experimental Medicine 198, no. 9 (October 27, 2003): 1427–37. http://dx.doi.org/10.1084/jem.20030802.
Повний текст джерелаKim, Wonil, Kimberly D. Klarmann, and Jonathan R. Keller. "Impaired Erythropoiesis Of Gfi-1 Null Hematopoietic Progenitor Cells Is Rescued By Reducing Id2 Levels." Blood 122, no. 21 (November 15, 2013): 737. http://dx.doi.org/10.1182/blood.v122.21.737.737.
Повний текст джерелаPortis, Toni, and Richard Longnecker. "Epstein-Barr Virus LMP2A Interferes with Global Transcription Factor Regulation When Expressed during B-Lymphocyte Development." Journal of Virology 77, no. 1 (January 1, 2003): 105–14. http://dx.doi.org/10.1128/jvi.77.1.105-114.2003.
Повний текст джерелаYamamoto, Hideyuki, Fumihiko Hayakawa, Takahiko Yasuda, Yuka Minamikawa, Naoyuki Tange, Daiki Hirano, Yuki Kojima, et al. "ZNF384-Fusion Proteins Have High Affinity to EP300, Which Increases Their Transcriptional Activities." Blood 132, Supplement 1 (November 29, 2018): 1554. http://dx.doi.org/10.1182/blood-2018-99-114414.
Повний текст джерелаCondorelli, G., L. Vitelli, M. Valtieri, I. Marta, E. Montesoro, V. Lulli, R. Baer, and C. Peschle. "Coordinate expression and developmental role of Id2 protein and TAL1/E2A heterodimer in erythroid progenitor differentiation." Blood 86, no. 1 (July 1, 1995): 164–75. http://dx.doi.org/10.1182/blood.v86.1.164.bloodjournal861164.
Повний текст джерелаBoos, Markus D., Yoshifumi Yokota, Gerard Eberl, and Barbara L. Kee. "Mature natural killer cell and lymphoid tissue–inducing cell development requires Id2-mediated suppression of E protein activity." Journal of Experimental Medicine 204, no. 5 (April 23, 2007): 1119–30. http://dx.doi.org/10.1084/jem.20061959.
Повний текст джерелаJi, Ming, Huajie Li, Hyung Chan Suh, Kimberly D. Klarmann, Yoshifumi Yokota, and Jonathan R. Keller. "Id2 intrinsically regulates lymphoid and erythroid development via interaction with different target proteins." Blood 112, no. 4 (August 15, 2008): 1068–77. http://dx.doi.org/10.1182/blood-2008-01-133504.
Повний текст джерелаYan, W., A. Z. Young, V. C. Soares, R. Kelley, R. Benezra, and Y. Zhuang. "High incidence of T-cell tumors in E2A-null mice and E2A/Id1 double-knockout mice." Molecular and Cellular Biology 17, no. 12 (December 1997): 7317–27. http://dx.doi.org/10.1128/mcb.17.12.7317.
Повний текст джерелаMasson, Frederick, Margherita Ghisi, Joanna R. Groom, Axel Kallies, Cyril Seillet, Ricky W. Johnstone, Stephen L. Nutt, and Gabrielle T. Belz. "Id2 represses E2A-mediated activation of IL-10 expression in T cells." Blood 123, no. 22 (May 29, 2014): 3420–28. http://dx.doi.org/10.1182/blood-2014-03-561456.
Повний текст джерелаДисертації з теми "Id2 inhibiteur de E2A"
Lacroix, Aurélie. "Etude de la localisation de l’herpèsvirus humain de type 6 (HHV-6) dans les tissus hodgkiniens : recherche de l’expression de l’oncogène viral DR7 et de ses conséquences pour la cellule." Limoges, 2008. https://aurore.unilim.fr/theses/nxfile/default/45243d16-ed38-4721-8146-f2c5dd68586b/blobholder:0/2008LIMO4001.pdf.
Повний текст джерелаThere are two variants, A and B, of human lymphotropic herpesvirus type 6 (HHV-6). The B variant is associated with Hodgkin’s lymphoma (HL). However this association is contreversial in the literature because it only exists in rare cases and not in Reed-Sternberg cells (RS), the characteristic cells of HL. In the first part of our study quantitative PCR demonstrated the existence of HHV-6 alone or concommitantly with Epstein-Barr virus, known to be associated with HL, in a cohort of HL patients. We then used an anti-HHV-6 DR7B antibody produced in our laboratory to determine the cellular localization of HHV-6 and to detect its presence in large quantities in RS cells from 73. 7% of HL patients positive only for HHV-6. Furthermore, double labeling studies in patients from the cohort positive for both herpesviruses demonstrated the colocalization, notably in RS cells, of DR7B from HHV-6 and LMP1 from EBV oncoproteins, indicating a probable viral interaction. Finally, a stable transfection system was realised to adjust DR7B expression in mature B cells which are close to RS cells. In the presence of DR7B, a transcriptionnal and translational expression of Id2, an inhibitor of E2A wich is a transcription factor implicated in phenotypical reprogramming of RS cells, similar to RS cells in HL, was observed