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Автор: Grafiati
Опубліковано: 4 червня 2021
Оновлено: 28 липня 2024

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1

Jamali, Ghulam Mustafa, Anwar Ali Jamali, and Habibullah Shaikh. "HEPATITIS C VIRUS;." Professional Medical Journal 24, no. 11 (November 3, 2017): 1621–29. http://dx.doi.org/10.29309/tpmj/2017.24.11.646.

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Objectives: The plan of this current research was in the direction for towards theassessment of the existing ELISA (Enzyme Linked Immunosorbant Assay) method throughantibodies testing for identification of hepatitis C virus disease by comparing their outcome withthe Real Time polymerase chain reaction analysis. Setting: Peoples Medical College HospitalNawabshah. Period: December 2015 to December 2016. Methods: In this current research 100blood samples were analyzed due to the presence of anti-HCV antibodies by 3rd-generationenzyme-linked immunosorbent assay testing. All the specimens were 100% positive. Polymerasechain reaction test was performed according to the laboratory directions in anti- hepatitis C virusantibodies positive patients to validate the diagnosis of hepatitis C virus infectivity. Results: Thisresearch shows that, the entire results were positive by Enzyme Linked Immunosorbant Assaytesting. As compared with polymerase chain reaction the of Enzyme Linked ImmunosorbantAssay in this research the screening test for anti hepatitis C virus - antibodies is about 2%false positive. Out of the 100 samples 98 cases are positive by Real Time polymerase chainreaction analysis while only 02 cases report are negative (2%). Conclusion: The proportion ofhepatitis C virus infectivity was 100% by 3rd-generation enzyme-linked immunosorbent assaytesting, 98% by Real Time polymerase chain reaction analysis. As in our research the hepatitisC virus –Ribonucleic acid is present in 98% cases who are the Anti- hepatitis C virus antibodiespositive patients, it can be suggested that Anti-HCV antibodies detection by third generationELISA technique in routine procedure is sufficient to determine HCV infection.
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2

AYGEN, Bilgehan, Mustafa Kemal ÇELEN, İftihar KÖKSAL, Selma TOSUN, Oğuz KARABAY, Tansu YAMAZHAN, Orhan YILDIZ, Celal AYAZ, and Fehmi TABAK. "The Prevalence and Epidemiological Characteristics of Hepatitis B Virus and Hepatitis C Virus Coinfection in Turkey." Turkiye Klinikleri Journal of Medical Sciences 33, no. 5 (2013): 1245–49. http://dx.doi.org/10.5336/medsci.2012-32319.

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3

MD, Ian R. Mackay. "The new hepatitis virus: hepatitis C virus." Medical Journal of Australia 153, no. 5 (September 1990): 247–49. http://dx.doi.org/10.5694/j.1326-5377.1990.tb136892.x.

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4

Kim, Arthur. "Hepatitis C Virus." Annals of Internal Medicine 165, no. 5 (September 6, 2016): ITC33. http://dx.doi.org/10.7326/aitc201609060.

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5

Kaplan, David E. "Hepatitis C Virus." Annals of Internal Medicine 173, no. 5 (September 1, 2020): ITC33—ITC48. http://dx.doi.org/10.7326/aitc202009010.

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6

NAQVI, S. M. ABBAS, Muhammad Shiraz Khan, QURBAN ALI KHASKHELI, Muhammad Saeed Talpur, and SHAHID HABIB ANSARI. "HEPATITIS C VIRUS." Professional Medical Journal 13, no. 04 (December 16, 2006): 604–7. http://dx.doi.org/10.29309/tpmj/2006.13.04.4935.

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`To find out the prevalence of antibody to HCV in serum of blood donorpopulation in our Community (Karachi). To estimate level of serum ALT in blood donors as possible marker of liverinfectivity and hence increasing awareness among the people about the prevention and spread of HCV in Communityand to give suggestions in the formulation of blood transfusion policies. Setting: At Microbiology Department, BasicMedical Sciences Institute, Jinnah Postgraduate Medical Centre Karachi. Period: From September 2001 to January2002. Material and Methods: 150 subjects, consisting of volunteer blood donors and 50 subjects selected from healthypopulation who had never received or donated blood. Results: Among 150 blood donors, 07 subjects (4.66%) werefound to be anti HCV positive. Mean age of anti HCV positive donors was 32.85±7.35 years with male predominance.Conclusion: It is concluded that HCV is notorious for its infectivity, chronocity and complications. Hence HCV spreadshould be controlled by screening blood donors for anti HCV antibodies and observing Universal rules in medicalpractice.
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7

Dana, Franklin, Paul R. Becherer, and Bruce R. Bacon. "Hepatitis C virus." Postgraduate Medicine 95, no. 6 (June 1994): 121–30. http://dx.doi.org/10.1080/00325481.1994.11945847.

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8

Klevens, R. Monina, and Anne C. Moorman. "Hepatitis C virus." Journal of the American Dental Association 144, no. 12 (December 2013): 1340–47. http://dx.doi.org/10.14219/jada.archive.2013.0069.

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9

Ye, Jin. "Hepatitis C Virus." Arteriosclerosis, Thrombosis, and Vascular Biology 32, no. 5 (May 2012): 1099–103. http://dx.doi.org/10.1161/atvbaha.111.241448.

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10

Jadoul, Michel, Alberto Frosi, MariaClotilde Ragni, Leonardo Salvaggio, Silvia Vezzoli, Francesco Vezzoli, Medhat Darwish, et al. "Hepatitis C virus." Lancet 345, no. 8943 (January 1995): 189–91. http://dx.doi.org/10.1016/s0140-6736(95)90192-2.

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11

di Bisceglie, Adrian M. "Hepatitis C Virus." Gastroenterology 116, no. 1 (January 1999): 224–25. http://dx.doi.org/10.1016/s0016-5085(99)70263-5.

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12

DONNELLY, K. "Hepatitis C virus." Journal of WOCN 26, no. 3 (May 1999): 111–12. http://dx.doi.org/10.1016/s1071-5754(99)90026-8.

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13

Smith, C. I. "Hepatitis C Virus." BMJ 310, no. 6974 (January 28, 1995): 268–69. http://dx.doi.org/10.1136/bmj.310.6974.268a.

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14

A-KADER, HASSAN HESHAM, and WILLIAM F. BALISTRERI. "Hepatitis C virus." Pediatric Infectious Disease Journal 12, no. 10 (October 1993): 853–67. http://dx.doi.org/10.1097/00006454-199310000-00011.

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15

Wright, Teresa L., and Brian J. G. Pereira. "Hepatitis C Virus." Seminars in Dialysis 10, no. 5 (October 1, 2007): 241–44. http://dx.doi.org/10.1111/j.1525-139x.1997.tb00502.x.

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16

Donnelly, Kevin. "Hepatitis C Virus." Journal of Wound, Ostomy and Continence Nursing 26, no. 3 (May 1999): 111. http://dx.doi.org/10.1097/00152192-199905000-00005.

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17

Laique, Sobia N., and Hugo E. Vargas. "Hepatitis C Virus." American Journal of Gastroenterology 114, no. 2 (February 2019): 207–8. http://dx.doi.org/10.14309/ajg.0000000000000095.

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18

Farci, Patrizia. "HEPATITIS C VIRUS." Clinics in Liver Disease 5, no. 4 (November 2001): 895–916. http://dx.doi.org/10.1016/s1089-3261(05)70200-2.

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19

Pietschmann, Thomas, and Richard J. P. Brown. "Hepatitis C Virus." Trends in Microbiology 27, no. 4 (April 2019): 379–80. http://dx.doi.org/10.1016/j.tim.2019.01.001.

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20

Keeffe, Emmet B. "Hepatitis C Virus." Clinics in Liver Disease 9, no. 3 (August 2005): xi—xiii. http://dx.doi.org/10.1016/j.cld.2005.05.001.

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21

&NA;. "Hepatitis C Virus." Pediatric Infectious Disease Journal 32 (November 2013): L—1—L—14. http://dx.doi.org/10.1097/01.inf.0000437868.34196.30.

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22

Dultz, Georg, and Stefan Zeuzem. "Hepatitis C Virus." Gastroenterology Clinics of North America 44, no. 4 (December 2015): 807–24. http://dx.doi.org/10.1016/j.gtc.2015.07.008.

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23

Ampuero, Javier, and Manuel Romero-Gómez. "Hepatitis C Virus." Gastroenterology Clinics of North America 44, no. 4 (December 2015): 845–57. http://dx.doi.org/10.1016/j.gtc.2015.07.009.

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24

Asselah, Tarik, and Marc Bourlière. "Hepatitis C Virus." Gastroenterology Clinics of North America 44, no. 4 (December 2015): 859–70. http://dx.doi.org/10.1016/j.gtc.2015.07.013.

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25

Falk, Gary W. "Hepatitis C Virus." Gastroenterology Clinics of North America 44, no. 4 (December 2015): xiii. http://dx.doi.org/10.1016/j.gtc.2015.09.002.

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26

Roggendorf, M. "Hepatitis-C-Virus." DMW - Deutsche Medizinische Wochenschrift 115, no. 09 (August 18, 2009): 352–54. http://dx.doi.org/10.1055/s-0029-1235578.

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27

Ho, Monto. "Hepatitis C Virus." New England Journal of Medicine 325, no. 7 (August 15, 1991): 507–9. http://dx.doi.org/10.1056/nejm199108153250710.

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28

Pereira, C., C. A. Lee, and G. Dusheiko. "Hepatitis C virus." BMJ 303, no. 6805 (September 28, 1991): 783. http://dx.doi.org/10.1136/bmj.303.6805.783.

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29

Tedder, R. S., and E. M. Briggs. "Hepatitis C virus." BMJ 303, no. 6813 (November 23, 1991): 1331. http://dx.doi.org/10.1136/bmj.303.6813.1331.

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30

Jarrett, Monica, and Paula Cox. "Hepatitis C virus." Nursing Clinics of North America 39, no. 1 (March 2004): 219–29. http://dx.doi.org/10.1016/j.cnur.2003.11.014.

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31

Sherlock, Sheila. "Hepatitis C virus." Digestive Diseases and Sciences 41, S12 (December 1996): 3S—5S. http://dx.doi.org/10.1007/bf02087871.

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32

Bréchot, Christian. "Hepatitis C virus." Digestive Diseases and Sciences 41, S12 (December 1996): 6S—21S. http://dx.doi.org/10.1007/bf02087872.

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33

Plagemann, P. G. W. "Hepatitis C virus." Archives of Virology 120, no. 3-4 (September 1991): 165–80. http://dx.doi.org/10.1007/bf01310473.

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34

Sajjad, Aiza, Shahnawaz Gardezi, Fatima Mukhtar, Amna Anjum, Qiarush Saeed, and Noor Dawood. "HEPATITIS C VIRUS (HCV) INFECTION." Professional Medical Journal 22, no. 11 (November 10, 2015): 1403–8. http://dx.doi.org/10.29309/tpmj/2015.22.11.871.

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Анотація:
Background: With a high magnitude of hepatitis C in the country and theburden still rising it was imperative to assess the knowledge of HCV infected individuals, whichwould determine the further spread of the disease or otherwise based on the adoption of goodpractices by these patients. Objectives: To evaluate the knowledge and practice regardingHCV in Hepatitis C patients presenting at Ghurki Trust Teaching Hospital, Lahore and toformulate recommendations based on study results to improve knowledge about hepatitisC. Study Design: Descriptive cross-sectional study. Setting: Ghurki Trust Teaching Hospital(GTTH), Lahore. Period: January to May 2015. Methods: The patients of hepatitis C registeredat GTTH for treatment were included in the study after obtaining voluntary informed consentfrom the respondents and approval of the study from the institutional ethical review board. Theconvenience non-probability sampling technique was used to recruit 169 study participants.A pre-tested structured questionnaire was used to collect information, which was recordedand analyzed using the statistical package for social sciences version 21.0. Data is describedin the form of frequencies and percentages for categorical variables and mean and standarddeviation for continuous variable. Results: Of the 169 HCV patients, 110(65%) had heard ofhepatitis C before acquiring it, the popular source of information regarding HCV was identifiedas relatives by 67(39.8%) of the patients. 70(41.4%) of the respondents were aware of a virusbeing the cause of hepatitis C, 140(82.8%) knew that HCV can be spread through sharinginjecting equipment, nearly half the respondents 87(51.5%) had asked their family membersto get tested for HCV and 68(40.2%) patients practiced safe sex. Conclusion: Majority of therespondents had heard of HCV before acquiring the disease. A large proportion of patients wereaware of the disease being spread through sharing injecting equipment. Half of the patients hadtheir family members tested for HCV. But less than half practiced safe sex.
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35

Yuki, Nobukazu, Norio Hayashi, Akinori Kasahara, Hideki Hagiwara, Hideyuki Fusamoto, and Takenobu Kamada. "Antibodies to hepatitis C virus and hepatitis C virus infection." Gastroenterologia Japonica 28, S5 (May 1993): 76–79. http://dx.doi.org/10.1007/bf02989211.

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36

Mazzaro, C., G. Panarello, F. Tesio, G. Santini, M. Crovatto, G. Mazzi, F. Zorat, et al. "Hepatitis C virus risk: a hepatitis C virus related syndrome." Journal of Internal Medicine 247, no. 5 (May 2000): 535–45. http://dx.doi.org/10.1046/j.1365-2796.2000.00627.x.

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37

Andrade, Luís Jesuino, Paulo Melo, Isabel Lins, Raymundo Paraná, and Augusto Lins. "HEPATITIS C VIRUS AND HEPATITIS C-INFECTION." Brazilian Journal of Medicine and Human Health 3, no. 1 (May 31, 2015): 19–28. http://dx.doi.org/10.17267/2317-3386bjmhh.v3i1.453.

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38

Nouman, Muhammad Khuram, Bushra Zaidi, Ghulam Mohiuddin, Faryal Asif, and Muhammad Khan Malik. "HEPATITIS C." Professional Medical Journal 25, no. 03 (March 10, 2018): 387–91. http://dx.doi.org/10.29309/tpmj/2018.25.03.381.

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Background: Hepatitis C virus (HCV) is the most communal source of non-A,non-B viral hepatitis in the world. The disease is illusory, and the majority of patients do notacquire jaundice at its onset. Treatment of hepatitis C with interferon attained a sustainedvirological response (SVR) in almost 50% of the patients with HCV infection. Viral genotype isimportant to determine the response. The present study aims to provide the incidence of relapseof HCV in patients taking interferon therapy and to identify the predictors for relapse. StudyDesign: Retrospective observational study. Setting: Department of Medicine, DHQ TeachingHospital, Sargodha. Period: Two years. Methods: A total of 60 patients were retrospectivelyevaluated for this study. The exclusion criteria include the patients co- infected with hepatitisB virus or HIV. All the patients were monitored 2, 4, 8, 12, 16, and 24 weeks after the endof treatment with interferon alpha. Results: We observed that the patients with relapse weresignificantly older and heavier (P value < 0.05). At the start of treatment, viral load was higherin relapsed patients (P value < 0.04). Conclusion: On the bases of our study findings, we canconclude that low incidence of relapse occurred with interferon therapy. High ALT level, viralloads, older age and obesity were some of strong predictors of relapse among HCV patients.
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39

Calmet Schwartzmann, Fernando H., and Fernando H. Calmet Bruhn. "Hepatitis C." Diagnóstico 56, no. 1 (December 13, 2018): 24–30. http://dx.doi.org/10.33734/diagnostico.v56i1.119.

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El virus de la hepatitis C (VHC) es un virus monocatenario ARN perteneciente al género Hepacivirus de la familia Flaviviridae. En la década de 1970, tras el descubrimiento de hepatitis Ay B, se hizo claro que había casos de hepatitis post-transfusión que no podían ser explicados por estos virus, llevando a la descripción de hepatitis "no A, no B". No fue hasta 1989 que se aisló el VHC por primera vez y entre 1990 y 1992 se desarrolló y refinó una prueba de anticuerpos anti-VHC que fue implementado en los bancos de sangre, llevando a una gran reducción de la transmisión de este virus a nivel mundial. Desde entonces ha habido una proliferación de nuevos antivirales directos que han revolucionado el manejo de la hepatitis C, asociados a una eficacia que se aproxima a 100% y con efectos adversos mínimos.
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40

Calmet Schwartzmamn, Fernando H., and Fernando H. Calmet Bruhn. "Hepatitis C." Diagnóstico 56, no. 1 (January 30, 2020): 24–30. http://dx.doi.org/10.33734/diagnostico.v56i1.166.

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Анотація:
El virus de hepatitis C (VHC) es un virus monocatenario ARN perteneciente al género Hepacivirus de la familia Flaviviridae. En la década de 1970, tras el descubrimiento de hepatitis A y B, se hizo claro que había casos de hepatitis post-transfusión que no podían ser explicados por estos virus, llevando a la descripción de hepatitis ''no A, no B''. No fue hasta 1989 que se aisló el VHC por primera vez y emtre 1990 y 1992 se desarrolló y refinó una prueba de anticuerpos anti-VHC que fue implementado en los bancos de sangre, llevando a una gran reducción de la transmisión de este virus a nivel mundial. Desde entonces ha habido una proliferación de nuevos antivirales directos que han revolucionado el manejo de la hepatitis C, asociados a una eficacia que se aproxima a 100% y con efectos afversos mínimos.
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41

Belli, Luca S., Enrico Silini, Alberto Alberti, Giorgio Bellati, Claudio Vai, Ernesto Minola, Gianfranco Rondinara, et al. "Hepatitis C virus genotypes, hepatitis, and hepatitis C virus recurrence after liver transplantation." Liver Transplantation and Surgery 2, no. 3 (May 1996): 200–205. http://dx.doi.org/10.1002/lt.500020305.

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42

TANAKA, Yasuhito, and Masashi MIZOKAMI. "Genotype of hepatitis virus. 3. Hepatitis C virus." Kanzo 46, no. 8 (2005): 480–85. http://dx.doi.org/10.2957/kanzo.46.480.

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43

Anjum, Muhammad Usman, Muhammad Safdar Khan, Nazar Muhamamd Afridi, and Syed Humayun Shah. "HEPATITIS B AND C VIRUS INFECTIONS." Professional Medical Journal 22, no. 10 (October 10, 2015): 1284–88. http://dx.doi.org/10.29309/tpmj/2015.22.10.981.

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Анотація:
Patients with end stage renal disease require haemodialysis as a part of theirtreatment. The incidence of hepatitis B and C virus infection is quite high in patients undergoingmaintenance haemodialysis than in general population. This risk is specifically associatedwith use of blood and its products as well as repeated intravascular access in these patients.Objectives: To determine the seropositivity of hepatitis B and C virus infection in patientsreceiving haemodialysis. Design: Descriptive cross sectional study. Setting: NephrologyDepartment, Ayub Teaching Hospital, Abbottabad, Pakistan. Period: From October 2014 toApril 2015. Methods: Five hundred patients were included in the study based on inclusionand exclusion criteria. Demographic data was recorded and detailed history was taken fromeach patient specifically about the no of blood transfusions received, the frequency of dialysisand the dialysis done in other centers. All patients were checked for the presence of hepatitisB surface antigen (HbsAg) and antibodies to HCV using third generation enzyme linkedimmunoassay (ELISA). Results: Mean age of study sample was 46±5 years with 60.8 % males.Incidence of hepatitis positive cases was 164 (32.8 %), out of which 66 (13.2 %) patients wereHBV positive and 98 (19.08 %) patients were HCV positive. The hepatitis B and C infectionswere more common in males than females. Seropositivity of HBV and HCV was higher (HBV18.1 % and HCV 22.2 %) among haemodialysis patients who have received more than threeblood transfusions. The frequency of HBV and HCV infections increases significantly with theincrease in frequency of dialysis, with 49 (17.11 %) patients were HBV positive and 70 (24.5%) patients were HCV positive cases, who have received haemodialysis for more than fivetimes. There were 48 (15.7 %) HBV positive cases as well as 68 (22.3 %) HCV positive cases inpatients who have received their treatment from a single center. Conclusion: Hepatitis B andC infection is quite common in patients undergoing haemodialysis. The risk of these infectionscan be reduced by following infection control guidelines, proper training of the staff and strictscreening of blood and blood products specifically for hepatitis C virus.
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44

Miyamura, Tathuo, Tethuro Suzuki, and Yosiharu Mathuura. "Special issue: Hepatitis C virus. Structural proteins of hepatitis C virus." Uirusu 43, no. 2 (1993): 275–83. http://dx.doi.org/10.2222/jsv.43.2_275.

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45

Soni, Dr Payal, Dr Dipa Kinariwala, Dr Neeta Khandelwal, Dr Mehul Patel, Dr P. K. Shah Dr. P. K. Shah, and Dr M. M. Vegad Dr. M. M. Vegad. "Co-Infection of Hepatitis C Virus and Hepatitis B Virus in Human Immuno Deficiency Virus Positive Population in Ahmedabad, Gujarat." International Journal of Scientific Research 2, no. 9 (June 1, 2012): 17–18. http://dx.doi.org/10.15373/22778179/sep2013/158.

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46

GUPTA, NEHA, SIMA K. BHATT, and AMI M. SHAH. "Seroprevalence of Hepatitis-B Virus, Human Immunodeficiency Virus & Hepatitis-C Virus in Corneal Donors in Tertiary Care Centre, Ahmedabad." International Journal of Scientific Research 3, no. 2 (June 1, 2012): 455–56. http://dx.doi.org/10.15373/22778179/feb2014/150.

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47

Ohkawa, Kazuyoshi, Norio Hayashi, Nobukazu Yuki, Hideki Hagiwara, Michio Kato, Keiji Yamamoto, Hiroshi Eguchi, Hideyuki Fusamoto, Manabu Masuzawa, and Takenobu Kamada. "Hepatitis C virus antibody and hepatitis C virus replication in chronic hepatitis B patients." Journal of Hepatology 21, no. 4 (1994): 509–14. http://dx.doi.org/10.1016/s0168-8278(94)80094-4.

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Mosley, James W. "Hepatitis C Virus and Fulminant Hepatitis." Annals of Internal Medicine 115, no. 12 (December 15, 1991): 983. http://dx.doi.org/10.7326/0003-4819-115-12-983_2.

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Bhamidimarri, Kalyan Ram, James Park, and Douglas Dieterich. "Management of Hepatitis B Virus Coinfection: HIV, Hepatitis C Virus, Hepatitis D Virus." Current Hepatitis Reports 10, no. 4 (November 4, 2011): 262–68. http://dx.doi.org/10.1007/s11901-011-0115-1.

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Dubuisson, Jean. "Hepatitis C virus proteins." World Journal of Gastroenterology 13, no. 17 (2007): 2406. http://dx.doi.org/10.3748/wjg.v13.i17.2406.

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