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Статті в журналах з теми "Glioma Cell Lines"
Chen, Yi-Hsuan, Dueng-Yuan Hueng, and Wen-Chiuan Tsai. "Proteolipid Protein 2 Overexpression Indicates Aggressive Tumor Behavior and Adverse Prognosis in Human Gliomas." International Journal of Molecular Sciences 19, no. 11 (October 26, 2018): 3353. http://dx.doi.org/10.3390/ijms19113353.
Повний текст джерелаTuma, Rabiya. "Repopulating glioma cell lines." Journal of Cell Biology 164, no. 3 (January 26, 2004): 334–35. http://dx.doi.org/10.1083/jcb1643rr2.
Повний текст джерелаShi, Fei, Jie Hu, Ping Zheng, Yisong Lv, Hongyu Liu, Guiyun Zhang, and Hongyu Jiang. "LncRNA PANTR1 is Associated with Poor Prognostic and Suppresses Apoptosis in Glioma." Journal of Oncology 2023 (February 20, 2023): 1–14. http://dx.doi.org/10.1155/2023/8537036.
Повний текст джерелаBota, Daniela A., Daniela Alexandru, Stephen T. Keir, Darell Bigner, James Vredenburgh, and Henry S. Friedman. "Proteasome inhibition with bortezomib induces cell death in GBM stem-like cells and temozolomide-resistant glioma cell lines, but stimulates GBM stem-like cells' VEGF production and angiogenesis." Journal of Neurosurgery 119, no. 6 (December 2013): 1415–23. http://dx.doi.org/10.3171/2013.7.jns1323.
Повний текст джерелаHan, Jizhong, Yu Xiong, Huajiang Deng, Jie Zhou, Lilei Peng, Wei Xiang, Yang Ming, and Ligang Chen. "MiR-455-3p regulates glioma cell proliferation by targeting PAX6." Tropical Journal of Pharmaceutical Research 18, no. 4 (May 17, 2021): 689–95. http://dx.doi.org/10.4314/tjpr.v18i4.2.
Повний текст джерелаMatsumoto, Tsuyoshi, Eiichi Tani, Keizo Kaba, Hideki Shindo, and Katsuya Miyaji. "Expression of P-glycoprotein in human glioma cell lines and surgical glioma specimens." Journal of Neurosurgery 74, no. 3 (March 1991): 460–66. http://dx.doi.org/10.3171/jns.1991.74.3.0460.
Повний текст джерелаNakano, Atsuhisa, Eiichi Tani, Kaoru Miyazaki, Yoshihiro Yamamoto, and Jun-ichi Furuyama. "Matrix metalloproteinases and tissue inhibitors of metalloproteinases in human gliomas." Journal of Neurosurgery 83, no. 2 (August 1995): 298–307. http://dx.doi.org/10.3171/jns.1995.83.2.0298.
Повний текст джерелаZhou, You-xin, San-song Chen, Ting-feng Wu, Da-dong Ding, Xiong-hui Chen, Jin-ming Chen, Zuo-peng Su, et al. "A Novel Gene RNF138 Expressed in Human Gliomas and Its Function in the Glioma Cell Line U251." Analytical Cellular Pathology 35, no. 3 (2012): 167–78. http://dx.doi.org/10.1155/2012/519037.
Повний текст джерелаAlexiou, George A., Xanthi Xourgia, Evrysthenis Vartholomatos, Spyridon Tsiouris, John A. Kalef-Ezra, Andreas D. Fotopoulos, and Athanasios P. Kyritsis. "Comparison of 99mTc-Tetrofosmin and 99mTc-Sestamibi Uptake in Glioma Cell Lines: The Role of P-Glycoprotein Expression." International Journal of Molecular Imaging 2014 (November 10, 2014): 1–5. http://dx.doi.org/10.1155/2014/471032.
Повний текст джерелаJung, Tae-Young, Shin Jung, Hyang-Hwa Ryu, Young-Il Jeong, Yong-Hao Jin, Shu-Guang Jin, In-Young Kim, Sam-Suk Kang, and Hyung-Seok Kim. "Role of galectin-1 in migration and invasion of human glioblastoma multiforme cell lines." Journal of Neurosurgery 109, no. 2 (August 2008): 273–84. http://dx.doi.org/10.3171/jns/2008/109/8/0273.
Повний текст джерелаДисертації з теми "Glioma Cell Lines"
Gee, Abigail Louise. "Proton beam irradiation of glioma cell lines in vitro." Thesis, University of Liverpool, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.439489.
Повний текст джерелаFlorian, Catarina Ligia. "Proton nuclear magnetic resonance studies of human glioma cell lines." Thesis, University College London (University of London), 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309218.
Повний текст джерелаFerreira, Matthew Thomas. "Analysis of how the production and activity of PGD2 affects glioma cell lines." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/42/42134/tde-10042015-120110/.
Повний текст джерелаA Organização Mundial de Saúde classifica glioblastoma (GBM) como um astrocitoma tipo IV, fazendo uns dos tumores mais fatais que existe. A pesar dos avanços em quimioterapia, cirurgia e radioterapia que melhoram a longevidade de sobrevivência, a trajetória geral da doença permanece imutável. Tem sido demonstrado que células de GBM produzem níveis significativos de prostaglandinas, incluindo prostaglandina D2 (PGD2). PGD2 possui propriedades pro- e anti-tumorigenicos. Então, um entendimento mais completo da atividade de PGD2 em GBM pode gerar tratamentos mais efetivos. Através de técnicas como RT-PCR, imunohistoquimicas e HPLC espectrometria de massa em tandem, conseguimos confirmar a presença da síntese de PGD2 em linhagens de GBM. Tratamos linhagens de GBM com concentrações variáveis de PGD2 exógeno durante 72 horas e observamos seus efeitos na contagem de células, apoptose, mitose e viabilidade. Nossos resultados sugerem que PGD2 possui funções opostas em GBM dependendo em concentração (mM PGD2 vs. nM PGD2) e ativação de receptores.
Giegerich, Anna [Verfasser], Eric Thomas [Gutachter] Hahnen, and Thorsten [Gutachter] Simon. "Identification of CD133-positive cell populations within glioma cell lines / Anna Giegerich ; Gutachter: Eric Thomas Hahnen, Thorsten Simon." Köln : Deutsche Zentralbibliothek für Medizin, 2021. http://d-nb.info/1236928091/34.
Повний текст джерелаMoore, Casey Benjamin. "Cell cycle affects accumulation of β-D-5-o-Carboranyl-2'-Deoxyuridine(D-CDU) in human glioma cell line". Thesis, Georgia Institute of Technology, 2002. http://hdl.handle.net/1853/16348.
Повний текст джерелаCILIBRASI, CHIARA. "CHROMOSOMAL INSTABILITY IN GLIOMA STEM CELL LINES FROM GLIOBLASTOMA MULTIFORME: IMPLICATIONS FOR NEW THERAPEUTIC STRATEGIES." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2017. http://hdl.handle.net/10281/158150.
Повний текст джерелаGlioblastoma is the most common primary malignant brain tumour in the adult population. Despite multimodality treatment with surgery, radiotherapy and chemotherapy, outcomes are very poor, with less than 15% of patients alive after two years. Increasing evidence suggests that Glioma stem cells (GSCs) are likely to play an important role in the biology of this disease and are involved in treatment resistance and tumour recurrence following standard therapy. GSCs are characterized by enhanced self-renewal, highlighted by the expression of stem cell markers, such as CD133 and Nestin, elevated invasive behaviour, chemo and radiotherapy resistance, and the ability to generate multi-lineage progenities. A typical feature of GSCs is also the elevated chromosomal instability (CIN): they are characterized by various numerical and structural aberrations, deletions, amplification and loss of heterozygosity. A variety of alterations have been proposed as being responsible for CIN, including defects in genes involved in the regulation of the mitotic machinery, such as the Aurora Kinases, making them a promising therapeutic target for GSCs depletion. My thesis address two main aspects of this research area, aiming at the identification of new GSCs-targeted therapeutic strategies for GBM complete eradication. In the first part of my project I investigated the effect of Danusertib, a pan-Aurora kinases inhibitor on 5 GSC lines isolated from glioblastoma patients, previously characterized in our laboratory from a cytogenomic and epigenomic point of view. Results showed that response to Danusertib exposure was heterogeneous among GSC lines. Some of them were more sensitive to subtle changes in Aurora kinases activity, which result in huge morphological alterations, a rapid increase in polyploidy and subsequently in senescence, with a consistent reduction in clonogenic survival and proliferation. Interestingly I also observed that the more resistant cell lines showed an increase in ploidy and senescence after repeated rounds of Danusertib exposure, suggesting that there could be the presence of an intolerable ploidy threshold that leads cells to senescence. In the second part of my thesis I presented some preliminary results I achieved in Dr Hochegger’s lab (Genome Damage and Stability Center, University of Sussex, Brighton, UK), where I took part in a project aimed on setting up CrispR/Cas9 mediated GFP or RFP-tagged CD133 (PROM1 gene) and Nestin (NES gene) glioma stem cell lines in order to look, with live cell imaging techniques, for signs for asymmetric cell division, by which a single GSC would be able to both maintain a pool of self-renewing stem cells and produce differential progeny, using live cell imaging. The biological significance of asymmetric or symmetric division modes is not yet fully understood, but improved understanding of this phenomenon may lead to the development of preventative treatments or improved therapeutic options for brain tumour patients through the identification of novel targets that are involved in the control of asymmetric cell division in human brain tissue.
Stutzman, Alan. "The effect of Neuregen nutrient medium on the growth of rat glioma cell lines F98 and 9L /." Available to subscribers only, 2007. http://proquest.umi.com/pqdweb?did=1456287431&sid=9&Fmt=2&clientId=1509&RQT=309&VName=PQD.
Повний текст джерела"Department of Molecular Biology, Microbiology and Biochemistry." Includes bibliographical references (leaves 109-126). Also available online.
Tassone, Evelyne. "Extracellular matrix-degrading enzymes and control of fibroblast growth factor-2 (FGF-2) signaling in pediatric glioma cell lines." Doctoral thesis, Università degli studi di Padova, 2012. http://hdl.handle.net/11577/3422194.
Повний текст джерелаL’obiettivo principale del mio progetto di ricerca è stato analizzare il ruolo di due enzimi che degradano la matrice extracellulare, l’“heparanase” (HPSE) e la “membrane-type 1 matrix metalloproteinase” (MT1-MMP), nei gliomi pediatrici. Ho trascorso i primi due anni di Dottorato nel laboratorio del Dott. Maurizio Onisto (Università di Padova). Ho poi continuato il mio lavoro presso la New York University School of Medicine, sotto la supervisione del Prof. Paolo Mignatti, il cui lavoro sperimentale è focalizzato sull’approfondimento dei meccanismi molecolari alla base dell’attivazione del segnale intracellulare da parte di MT1-MMP e del suo inibitore fisiologico, il “tissue inhibitor of metalloproteinases-2” (TIMP-2). I gliomi, i più comuni tumori cerebrali primari, comprendono un gruppo eterogeneo di neoplasie che originano dalle cellule gliali. Nonostante i recenti progressi raggiunti nel trattamento e nel controllo di tali tumori, la prognosi dei bambini affetti da glioma, ed in particolare dalle sue forme più aggressive, rimane tuttora infausta. Pur essendo confinati nell’organo nel quale originano, i gliomi possono invadere tutte le aree del cervello. Uno degli eventi più importanti che caratterizzano l’invasività dei gliomi è costituito dalla degradazione della matrice extracellulare, un complesso meccanismo che coinvolge enzimi sia glicosidici sia proteolitici. HPSE è una endo-β-D-glucuronidasi secreta nella matrice extracellulare, nella quale taglia le catene di eparan solfato dei proteoglicani solubili e legati alla membrana. MT1-MMP, una proteasi legata alla membrana e composta da un dominio catalitico extracellulare e da una piccola coda citoplasmatica, è coinvolta nella degradazione proteolitica di proteine extracellulari e di membrana. Elevati livelli di HPSE e MT1-MMP sono stati riscontrati in numerosi tipi di tumore e tale evidenza sottolinea il ruolo chiave che essi svolgono nell’invasività tumorale e nella formazione di metastasi. In questo studio sono state caratterizzate cinque linee cellulari di glioma pediatrico derivanti da diversi tipi di glioma: due glioblastomi multiformi, un astrocitoma anaplastico, un astrocitoma diffuso ed un astrocitoma pilocitico. Con lo scopo iniziale di esaminare il ruolo di MT1-MMP nell’attivazione del segnale indotto dall’FGF-2, è stata inoltre utilizzata una linea cellulare di carcinoma mammario, la quale non esprime MT1-MMP e perciò rappresenta un modello ideale per studiare la regolazione della sua espressione. I dati riportati mostrano che, nelle cellule di carcinoma mammario, MT1-MMP regola l’attivazione del segnale intracellulare da parte del “fibroblast growth factor-2” (FGF-2) e controlla il legame di questo fattore di crescita alla superficie delle cellule. Nelle cellule di astrocitoma pediatrico non è stata identificata alcuna chiara correlazione tra espressione di HPSE, MT1-MMP o FGF-2 ed aggressività tumorale. I risultati inoltre dimostrano che il silenziamento genico di HPSE in una linea cellulare di glioblastoma pediatrico non influenza l’espressione del “vascular endothelial growth factor” (VEGF) o la proliferazione cellulare, ma determina la sovraespressione della “matrix metalloproteinase-2” (MMP-2) e di MT1-MMP. Inoltre, nelle cellule di glioma, l’attivazione di ERK1/2 da parte di FGF-2 non correla con l’espressione di MT1-MMP e risulta modificata dal trattamento con un inibitore di MMP. Infine, in tutte le cellule di glioma, anche TIMP-2 regola l’attivazione del segnale intracellulare. In conclusione, i risultati ottenuti mostrano che MT1-MMP non ha gli effetti nelle cellule di carcinoma mammario e di glioma pediatrico, indicando l’esistenza di un differente e più complesso meccanismo di controllo del segnale intracellulare. La caratterizzazione delle linee cellulari di astrocitoma pediatrico presentata in questa tesi offre una più completa conoscenza di questo gruppo di tumori ancora poco studiati.
BARONCHELLI, SIMONA. "Cytogenetic, genimic, epigenomic and drug sensitivity landscapes to unravel the complexity of glioma stem cell lines: a multi-level approach." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2011. http://hdl.handle.net/10281/27138.
Повний текст джерелаHammerbacher, Katharina [Verfasser], and Ralf [Akademischer Betreuer] Kinscherf. "Effects of novelly synthesized nucleolipides on different tumor cell lines (HT29, HepG2, Panc-1, RenCa) with special respect to glioma cell lines (BT4Ca, GOS3, G28, G112, U251, U87) of human or other species / Katharina Hammerbacher ; Betreuer: Ralf Kinscherf." Marburg : Philipps-Universität Marburg, 2020. http://d-nb.info/120746967X/34.
Повний текст джерелаЧастини книг з теми "Glioma Cell Lines"
Ohlfest, John R., and Stacy A. Decker. "Glioma Cell Lines: Role of Cancer Stem Cells." In Tumors of the Central Nervous System, Volume 1, 205–12. Dordrecht: Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-0344-5_21.
Повний текст джерелаWhittle, Ian R., W. L. Kimber, M. Li, H. S. Bell, and J. W. Ironside. "Glioma Cells Transduced with Selection Transgenes May Not Form Gliomas in vivo and Can Also Inhibit Glioma Formation by Admixed Wild Glioma Cell Lines." In Advances in Stereotactic and Functional Neurosurgery 12, 139–43. Vienna: Springer Vienna, 1997. http://dx.doi.org/10.1007/978-3-7091-6513-3_26.
Повний текст джерелаShinoura, Nobusada, Tatsuya Kondou, and Masumi Yoshioka. "Measuring the Effect of PDGF on Fibroblasts Using Glioma Cell Lines." In Biological Aspects of Brain Tumors, 235–43. Tokyo: Springer Japan, 1991. http://dx.doi.org/10.1007/978-4-431-68150-2_30.
Повний текст джерелаDing, G. R., X. W. Wang, K. C. Li, J. Miyakoshi, and G. Z. Guo. "Hsps expression in three human glioma cell lines after radio-frequency field exposure." In 7th Asian-Pacific Conference on Medical and Biological Engineering, 458–60. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-79039-6_114.
Повний текст джерелаWu, David J., Linda Reynolds, Dennis A. Carson, and Tsutomu Nobori. "Molecular Genetic Analysis of Chromosome 9p in Methylthioadenosine Phosphorylase Deficient Glioma Cell Lines." In Advances in Experimental Medicine and Biology, 207–11. New York, NY: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4615-7703-4_47.
Повний текст джерелаWikstrand, Carol J., Friedrich C. Grahmann, Rodney D. McComb, and Darell D. Bigner. "Antigenic heterogeneity of human glioma tissue and cell lines (HGL) defined by monoclonal antibodies (MAs)." In Biology of Brain Tumour, 495–99. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2297-9_69.
Повний текст джерелаSmith, B. H., M. A. Greenwood, C. J. Cummins, J. Ellis, C. Gibson, and P. L. Kornblith. "Non-nuclear cytotoxic actions of DNA cross-linking and/or alkylating agents in glioma-derived cell lines." In Biology of Brain Tumour, 91–96. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2297-9_13.
Повний текст джерелаBenabid, Alim L., Chantal Remy, and Christiane Chauvin. "Experimental models of rat brain tumors by stereotactic injection of C6 glioma and HTC hepatoma cell lines." In Biology of Brain Tumour, 221–26. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2297-9_30.
Повний текст джерелаColombatti, M., M. Bisconti, P. Lorenzi, G. Stevanoni, B. Dipasquale, M. Gerosa, and G. Tridente. "Human Glioma Cell Lines: Tumour Associated Antigens Distribution and Sensitivity to Antibody-Toxin or Ligand-Toxin Conjugates. A Preliminary Report." In Proceedings of the 8th European Congress of Neurosurgery, Barcelona, September 6–11, 1987, 121–25. Vienna: Springer Vienna, 1988. http://dx.doi.org/10.1007/978-3-7091-8978-8_26.
Повний текст джерелаZeltzer, Paul M., Sandra L. Schneider, Paul J. Marangos, and Mark H. Zweig. "Differential expression of neuron specific enolase and creatine phosphokinase-BB by human medulloblastoma and glioma tumors and neuroectodermal cell lines." In Biology of Brain Tumour, 51–59. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2297-9_7.
Повний текст джерелаТези доповідей конференцій з теми "Glioma Cell Lines"
Kofman, Alexander, Fadila Guessous, Charles G. diPierro, Evan Dupart, Yongde Bao, Susan Dhamala, Christopher Letson, et al. "Abstract 3163: Exploration of microRNA-34a effects in human glioma cell lines." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-3163.
Повний текст джерелаZlatnik, Elena, Anastasia Sitkovskaya, Sergey Kolpakov, Elena Kolpakova, Svetlana Filippova, and Irina Mezhevova. "EFFECT OF ROTAVIRUS STRAINS OF A NEW GROUP ON GLIOMA CELL LINES IN VITRO." In XVII INTERNATIONAL INTERDISCIPLINARY CONGRESS NEUROSCIENCE FOR MEDICINE AND PSYCHOLOGY. LCC MAKS Press, 2021. http://dx.doi.org/10.29003/m2131.sudak.ns2021-17/155-156.
Повний текст джерелаKhatami, S., A. Rendon, M. Yoshimitsu, J. Medin, and L. Lilge. "Effect of GFP expression on the sensitivity of glioma cell lines to photodynamic therapy." In Photonics North 2005, edited by Warren C. W. Chan, Kui Yu, Ulrich J. Krull, Richard I. Hornsey, Brian C. Wilson, and Robert A. Weersink. SPIE, 2005. http://dx.doi.org/10.1117/12.628706.
Повний текст джерелаRossi, Alex P., Eric C. Woolf, Kenneth S. Brooks, Marshall J. Fairres та Adrienne C. Scheck. "Abstract 3346: The ketone body β-hydroxybutyrate increases radiosensitivity in glioma cell lines in vitro". У Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3346.
Повний текст джерелаQuintavalle, Cristina, Michela Garofalo, Giulia Romano, Margherita Iaboni, Ciro Zanca, Monica Brenca, Gerald Nuovo, Carlo Maria Croce та Gerolama Condorelli. "Abstract LB-351: miR-221&222 regulate cell motility in glioma cell lines targeting the protein phosphates PTPμ". У Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-lb-351.
Повний текст джерелаHedegaard, Chris J., Charles N. Pegram, Darell D. Bigner, and Hans S. Poulsen. "Abstract 2732: Internalization of the dual-specific immunotoxin D2C7-(scdsFv)-PE38KDEL in malignant glioma cell lines." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2732.
Повний текст джерелаDikshit, Bhawana, Kunzang Chosdol, and Subrata Sinha. "Abstract A38: Overexpression of FAT1 in human GBM (glioblastoma multiforme) and high-grade glioma cell lines." In Abstracts: Second AACR International Conference on Frontiers in Basic Cancer Research--Sep 14-18, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.fbcr11-a38.
Повний текст джерелаLespinats, Sylvain, Katja Pinker-Domenig, Georg Wengert, Ivo Houben, Marc Lobbes, Andreas Stadlbauer, and Anke Meyer-Bäse. "Proteomic data analysis of glioma cancer stem-cell lines based on novel nonlinear dimensional data reduction techniques." In SPIE Commercial + Scientific Sensing and Imaging, edited by Liyi Dai, Yufeng Zheng, Henry Chu, and Anke D. Meyer-Bäse. SPIE, 2016. http://dx.doi.org/10.1117/12.2229133.
Повний текст джерелаGarcia-Claver, Ainoha, Gema Perez-Diaz, Carmen Granda, Yolanda Campos-Martin, Yolanda Ruano, Elisa Perez-Magan, Manuela Mollejo, and Barbara Melendez. "Abstract 732: Characterization of gene expression changes associated with Erlotinib and Temsirolimus treatment in glioma cell lines." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-732.
Повний текст джерелаBuckingham, John, Reyda Gonzalez-Nieves, and Mary L. Cutler. "Abstract 5140: Rsu1 and the IPP adhesion complex can regulate adhesion and migration in glioma cell lines." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5140.
Повний текст джерела