Статті в журналах з теми "Giuseppe Ferrari"

Este artículo analiza las fuentes e intertextos de la definición de eclecticismo que fray Juan Benito Díaz de Gamarra presenta en su libro Elementa recentioris philosophiae (San Miguel el Grande, 1774), quien recurre al pasaje “enseñan justicia junto con saber pío” (docent justitiam cum pia scientia) de los Stromata de Clemente de Alejandría para defender la utilidad de la filosofía ecléctica, porque valora a los autores antiguos y modernos por su utilidad moral y filosófica. Esta definición se inserta en el contexto de la transición del pensamiento escolástico hacia la filosofía moderna (recentior philosophia) y remite a un entramado interdiscursivo (con tratadistas como Giuseppe Antonio Ferrari y Andrés Piquer) en el que Díaz de Gamarra defiende la filosofía ecléctica moderna para la educación de la juventud americana al mismo tiempo que cuestiona la utilidad de la formación basada fundamentalmente en Aristóteles, como lo es el método escolástico.

Il saggio tenta di mettere in luce i legami profondi e meno evidenti che sembrano intercorrere tra la storia generale del Risorgimento italiano (che fece di Genova, grazie anche a protagonisti quali Nino Bixio, Goffredo Mameli e Giuseppe Mazzini, una delle "patrie ideali") e alcune delle molte tracce fisiche che essa ha lasciato nel tessuto costruito della cittŕ. L'Autore propone la rilettura di alcuni episodi di trasformazione e formazione della cittŕ negli anni delle vicende risorgimentali con riferimento al momento in cui queste conobbero una prima conclusione in coincidenza con la proclamazione del regno d'Italia: il caso di via Garibaldi, ad esempio, che fu aperta demolendo chirurgicamente parte dei circostanti quartieri medievali, per collegare al nuovo centro della cittŕ, organizzato intorno a piazza De Ferrari, la cinquecentesca via Aurea, sede dell'antica classe oligarchica, attraverso piazza Fontane Marose. Oppure con la costituzione di ambiti celebrativi, come nel caso emblematico del nuovo snodo urbano di piazza Corvetto con le statue raffiguranti Giuseppe Mazzini e Vittorio Emanuele II. La rilettura di alcuni episodi noti e ormai oggetto di numerose analisi, anche recenti, si estende poi agli anni e decenni successivi a quella data (almeno fino ai primi anni del Novecento) che ebbe certo un forte impatto simbolico ma non altrettanto evidenti e immediate ricadute sulla vita e sulla struttura della cittŕ. Tutto ciň determinň anche diffuse e dolorose demolizioni, mutilazioni o spostamenti di antichi monumenti.

Reprinted in 1801, Vico's New Science (originally published 1744) had a profound impact on Bourbon Restoration culture, particularly in Italy and France, where it touched post-revolutionary readers profoundly. The reasons for Vico's revival in the early nineteenth century relate closely to the trauma of the political and social changes of that era. Vico's readership seems to have had significant peaks during periods of rapid social transformation: nineteenth-century readers reread the New Science in an attempt to find the reasons for revolutionary failure, and to relate the terror, the sense of displacement, failure and trauma to recognizable laws, promising that, after a crisis, a period of renaissance must necessarily follow. This article analyses the hermeneutic practices of some post-revolutionary readers of Vico (Carlo Cattaneo, Vincenzo Cuoco, Giuseppe Ferrari, Ugo Foscolo, Francesco Lomonaco) and suggests a comparison with the practices of readers during the Second World War (Eric Auerbach, Carlo Levi, Mario Fubini). By doing so, I propose an interpretation of Vico's New Science as a ‘posthumous’ book, acquiring special shades of significance when its readers experience the feeling that nothing will ever be like before, and meditate upon it in isolation, in fear, in exile, upon return from the front, and in prison.

U radu se donose nepoznati arhivski zapisi te nekoliko novih atribucija kojima se proširuje opus padovanskog kipara Angela De Puttija (Padova, oko 1681. – Žminj, 1731.). Majstor je tijekom svoje karijere bio aktivan u Vicenzi, Ferrari, Goriziji i Ljubljani, odnosno na širem području Veneta, Furlanije, Kranjske, Koruške, Istre i Kvarnera. Iako kipar Angelo De Putti ima pozamašnu fortunu criticu i opus, dosad nije bio poznat podatak o njegovoj smrti. Iz novopronađenoga arhivskog zapisa saznajemo kako je Angelo De Putti umro u Žminju 14. ožujka 1731. godine u dobi od otprilike 50 godina te je bio pokopan u mjesnoj župnoj crkvi. Također, donosi se i podatak kako je u Žminju 31. prosinca 1743. godine umro i Angelov sin, kipar Giuseppe. U opus Angela De Puttija prije dolaska u Istru uvrštavaju se kipovi svetog Šimuna Stocka i Terezije Avilske s pročelja crkve Gospe Karmelske u Esteu. Isto tako pripisuju mu se i dva tugujuća anđela iz crkve Madonna del Pilastro u Esteu. Kao majstorov rad prepoznati su i kameni kipovi svetog Antuna Opata, Antuna Padovanskog, Antonina i dva anđela s pročelja župne crkve svetog Antonina u Salvaterri. Uz De Puttijeve kipove, na pročelju u Salvaterri prepoznati su i kipovi Giovannija i Antonija Bonazze. Tako su uz dva anđela s portala crkve u Salvaterri u opus Giovannija Bonazze još uvršteni mramorna figura Gospe Karmelske sa stipesa glavnog oltara crkve Gospe Karmelske u Esteu. S druge strane kao djela Antonija Bonazze prepoznati su kameni kipovi svete Eurozije i svete Ilarije iz Salvaterre te kip Uskrslog Krista s dražbe aukcijske kuće San Felice iz Pistoije.

Abstracttitle SUMMARY /title Two letters of Gianfrancesco Malfatti (1731-1807) are published. The first one is addressed to Cassini de Thury (1784), the second one to Vincenzo Monti (1806). These letters also regard other important figures of the eighteenth century in Ferrara and of the Napoleonic Period, such as the physician Giuseppe Antonio Testa (1756-1814) and Giovanni Battista Costabili Containi (1756-1841).

En 1928, Giuseppe Pardi publicó la primera de tres crónicas del siglo XV sobre Ferrara. Su edición de uno de estas tres crónicas, el Diario ferrarese dall’anno 1409 sino al 1502, sustituyó versión del siglo XVIII publicada por Ludovico Muratori. Pardi mostró una comprensión de los retos que presenta la transcripción de documentos antiguos, rehusando alterar este material y enriqueciendo las crónicas con un aparato crítico completo. A pesar de la disponibilidad y fácil acceso a materiales inimaginables hasta hace poco, las transcripciones de finales del siglo xx no llegan a los estándares establecidos por Pardi. Este artículo explora las transcripciones de las crónicas, en particular las de Pardi, ilustrando como los inicios del siglo xx fueron una época dorada sin igual antes o después.

The intersubjective paradigm advocated by Habermas laid the foundation for a new way of reaching rationally predicated consensus. His work indicates that what keeps people in an attitude of communicative action is rational basis and motivation. Upon analysing the intersubjective dimension of the practice of the community of philosophical inquiry, we realized that it possible to observe the transition from a subject-centred autonomy of treason (thinking for oneself), to the practice of a communicative rationality (focused on thinking in community). The inevitable assumptions of communication ground and sustain communities and allow for an environment of trust and care that enables community members to think freely and let themselves be driven by the power of “we”, “nos-otros”. It is not only in the context of communities of philosophical inquiry that the potential of Habermas’ intersubjective paradigm can be observed, but also in the discussion among specialists. To an extent, this is what we found when we studied the various approaches of Ann Sharp, David Kennedy, and Giuseppe Ferraro to the concept of the community of philosophical inquiry. The dialogue between these philosophers and the concept of the community of philosophical inquiry, their arguments, and the ways of achieving philosophical dialogue in community offer rich evidence of the productiveness of communicative rationality. We are aware that Ann Sharp’s concept of community of philosophical inquiry is different from David Kennedy’s, and that both are distinct from Giuseppe Ferraro’s circle of thinking. Nevertheless, we place these different approaches into dialogue, due largely to what is permitted by the movement of communicative rationality itself. These possibilities for dialogue are also grounded in the assumption of an intersubjective relationship between concepts, experiences and thoughts about concepts and experiences. By understanding the community of philosophical inquiry as a space of intersubjectivity (Sharp, 1987), opening the philosophical community to other non-discursive dimensions of communication (Kennedy, 1994), and analysing the circle of thinking that fills space with time and turns it into a place (Ferraro, 2018), we reveal that the community of philosophical inquiry is an open and fruitful concept and experience: a concept in motion.

Abstract Over the past decade, infectious agents, including bacteria, have come under scrutiny for contributing to almost 16% of all tumors. Indeed, emerging data report a microbial presence in many cancer types, with breast cancer accumulating the most diverse bacterial population. Pathogen defense mechanisms, such as CHI3L1, contribute to bacterial elimination; however, CHI3L1 is overexpressed in several cancer types and correlates with poor prognosis and shorter survival. However, the connection between intra-tumoral bacteria and CHI3L1 and how they contribute to cancer growth has not been fully characterized.To investigate if microbiota and CHI3L1 are connected, female BALB/c mice harboring 4T1-Luc cells were exposed to a broad-spectrum antibiotic mix to deplete the microbiota and to evaluate tumor growth and circulating CHI3L1 levels. 9 and 14 days after tumor inoculation, feces were collected and bacterial DNA isolated for 16S rRNA sequencing, and tumors were plated to identify intratumoral bacteria. To assess whether a specific bacterial strain could modulate CHI3L1 expression, we incubated 4T1 cells with supernatants from isolated bacteria, which contain metabolites and extracellular factors. Isolated bacteria that could upregulate CHI3L1 expression were then used to infect 4T1 cells prior to subcutaneous injection in the mammary fat pad of BALB/c mice to evaluate if bacterial infection changes tumor growth rate.In the 4T1 mouse breast cancer model, a broad-spectrum antibiotic treatment was able to: deplete host microbiota, abrogate tumor growth, and reduce CHI3L1 levels in the sera and in CD45+ immune infiltrating cells in tumor and colon. When 4T1 cells were exposed to bacterial supernatants in vitro, we observed that CHI3L1 was upregulated by a specific strain of E. coli. To simulate the effect of intra-tumoral microbiota, we infected 4T1 cells with either the isolated E. coli or S. aureus (which did not induce any CHI3L1 modulation in vitro) prior to subcutaneous injection. Strikingly, E. coli infection increased tumor progression with respect to the control S. aureus infection (p<0.01).Moreover, we observed an increase in CHI3L1 produced by tumor immune infiltrates in E. coli-infected 4T1, highlighting a key role for microbiota and CHI3L1 in tumor progression. Overall, our findings shed light on the important link between tumor associated bacteria and CHI3L1 in cancer progression. Identifying the mechanism of action through which microbiota and CHI3L1 influence cancer growth will open new possibilities for improved screening, prognosis, and survival for patients with breast cancer. Citation Format: Alessandro Mauro Mozzarelli, Sara Carloni, Valentina Ferrrari, Silvia Giugliano, Luca Tiraboschi, Antonino Lo Cascio, Giulia Fornasa, Daniele Braga, Davide Golzato, Nicola Segata, Giuseppe Penna, Maria Rescigno. Microbiota mediated CHI3L1 expression as a novel candidate biomarker for breast cancer progression [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2022; 2022 Apr 8-13. Philadelphia (PA): AACR; Cancer Res 2022;82(12_Suppl):Abstract nr 3059.

Abstract Background:The introduction of anti-HER2 therapies, trastuzumab and pertuzumab (HP) to the treatment of metastatic HER2-positive breast cancer has had a transformational impact on the outcomes for this disease. Nevertheless, disease progression is ultimately observed in most patients within 3 years carrying the potential for morbidity and potentially more toxic therapies. To identify selective strategies to prevent disease progression, we sought to elucidate mechanisms of resistance to anti-HER2 therapies by analyzing a large cohort of genomically and clinically annotated HER2+ breast cancers. Methods: Patients with advanced HER2+ breast cancer who underwent prospective clinical tumor sequencing utilizing MSK-IMPACT assay between April 2014 and February 2021 were included in the analysis. Clinical HER2 positivity was defined as per ASCO/CAP guidelines. Cox proportional hazard models were used to determine the association between genomic alterations and progression-free survival (PFS) on 1st line HP and taxane-based therapy (THP). Only patients with a sequenced pre-treatment tumor sample were included in the survival analysis. Recurrent mutations identified were modeled in HER2+ breast cancer cell lines using short hairpin RNAs and CRISPR/Cas9, and the sensitivity of these isogenic pairs to HER2-targeted therapies was evaluated via metabolic and colony formation assays of cell proliferation. Results: We identified 733 ERBB2-amplified primary (n=385) and metastatic (n=348) that underwent sequencing. Concurrent PIK3CA mutations were identified in 30% of the tumors. Pathogenic activating alterations involving the MAPK pathway were observed in 12.8% of tumors with the most frequent alterations being NF1 loss, ERBB2 and RAS activating mutations. MAPK alterations were significantly enriched in the metastatic tumors (16.6%) compared to the treatment-naïve primaries (9.8%, p=0.020). The outcome analysis included 145 patients with advanced clinically HER2+ breast cancer whose tumors were sequenced prior to starting 1st line THP. Twenty percent (29/145) of tumors did not show genomic ERBB2 amplification as detected by NGS and had a significantly worse outcome (median PFS of 9.4 months [95% CI 5.5-19] and 23 months [95% CI 17-30], respectively; p=0.015). PIK3CA mutations were also associated with a shorter PFS (mutant: 13 months [95%CI: 7.7-18] vs wild type: 23 months [95%CI 17-16], p=0.0013). We further found reduced PFS in MAPK altered tumors (median PFS 9.9 months; 95% CI: 5.5-17) compared to the rest of the population (median PFS 21 months; 95% CI: 17-30; p=0.01). On multivariable analysis adjusted for estrogen receptor status, and presence of PIK3CA/AKT1/PTEN mutations and genomic ERBB2 amplification, MAPK pathway alterations were independently associated with worse outcome (HR: 2.25; 95% CI: 1.29, 3.93; multivariate p = 0.0043). To establish a causal role for MAPK alterations in reducing efficacy of anti-HER2 therapy, we depleted NF1 expression or expressed mutant KRAS or BRAF in a panel of HER2+ breast cancer cell lines. Consistently, MAPK-altered cell lines exhibited resistance to FDA approved HER2 inhibitors in vitro and in vivo. Conclusions: This clinicogenomic analysis of mechanisms of resistance to anti-HER2 therapy demonstrated that PIK3CA activating mutations and lack of genomic ERBB2 amplification as detected by tumor sequencing are associated with shortened PFS on HP-based therapy. Our analysis uniquely identified MAPK pathway alterations as additional potential drivers of resistance to anti-HER2 therapy. Inhibition of the PI3K or MAPK pathway in such tumors may represent a new therapeutic strategy to extend H/P benefit. Citation Format: Emanuela Ferraro, Alison Smith, Anton Safonov, Paulino Tallon De Lara, Cristina Bernado', Enrique J. Arenas Lahuerta, Joaquín Arribas, David Solit, Jorge S. Reis-Filho, Neal Rosen, Larry Norton, Shanu Modi, Mark E. Robson, Chau T. Dang, Giuseppe Curigliano, Sarat Chandarlapaty, Pedram Razavi. Genomic analysis of 733 HER2+ breast cancers identifies recurrent pathways alterations associated with anti-HER2 resistance and new therapeutic vulnerabilities [abstract]. In: Proceedings of the 2021 San Antonio Breast Cancer Symposium; 2021 Dec 7-10; San Antonio, TX. Philadelphia (PA): AACR; Cancer Res 2022;82(4 Suppl):Abstract nr GS3-03.

Abstract Background: Varying levels of HER2 expression without ERBB2 gene amplification can be detected by immunohistochemistry (IHC) in approximately 60% of all invasive breast cancers (BCs). HER2-low-expressing BCs have recently been shown to respond to novel anti-HER2 antibody drug conjugates. Several studies have demonstrated that HER2-low BCs do not seem to constitute a distinct clinical and transcriptomic subtype as compared to HER2-0 BCs. Here we sought to define the clinicopathologic features and repertoire of somatic genetic alterations in HER2-low BCs. Methods: We retrieved clinical, pathological, and genomic data of BCs that were subjected to targeted sequencing using the FDA-cleared MSK-IMPACT assay from April 2014 to December 2021. After removing cases where any available biopsy had HER2 3+ and/or positive ERBB2 fluorescence in situ hybridization (FISH), 3608 samples (primary=1347; post-treatment/metastatic=2261) were included. Tumors were classified as HER2-low if they had an HER2 IHC score of 1+ or 2+ with a non-amplified FISH assay and HER2-0 if they had an HER2 IHC score of 0. Somatic mutations and DNA copy number alterations from MSK-IMPACT were analyzed. Multiple testing correction using the Benjamini-Hochberg method was applied to control for the false discovery rate (q). Q values < 0.1 were considered significant. Results: Among 3608 HER2- BCs, 1460 (40%) and 2148 (60%) were HER2-0 and HER2-low, respectively. Hormone receptor (HR) expression was significantly higher in HER2-low than HER2-0 tumors in both primary (781 [68.3%] vs 362 [31.7%]; p< 0.001) and metastatic (1031 [60.5%] vs 673 [39.5%]; p< 0.001) samples. A higher proportion of HER2-low tumors was found in metastatic than primary samples (59% vs 41%; p< 0.001) in this cohort. No difference in histology subtype, tumor grade, disease stage (among primary tumors), mutational signatures, and tumor mutational burden was found overall and when cases were stratified by HR expression. In HR-positive BCs, HER2-0 BCs harbored higher frequency of TP53 (33% vs 25%; odds ratio [OR] 1.49, 95% confidence interval [CI] 1.25-1.78, q< 0.001) and CDKN1A (1% vs 0%; OR 17.47, 95% CI 2.48-756.37, q=0.02) alterations than HER2-low BCs. Similar findings were observed in metastatic but not in primary HR-positive BCs. No differences were detected in HR-negative BCs stratified into HER2-low and HER2-0. Given the potential misclassification that exists between IHC HER2-0 and HER2-1+, we then conducted an exploratory analysis splitting the HER2-low group into HER2 1+ and 2+. TP53 alterations remained significantly enriched in HER2-0 compared to HER2-1+ HR-positive tumors (33% vs 24%; OR 1.55, 95% CI 1.28-1.87, q< 0.001). In HR-positive BCs, HER2-2+ displayed a higher frequency of genetic alterations in genes encoding for transcription factors, such as MYC (14.2% vs 7.3%; OR 2.09, 95% CI 1.44-3.04, q=0.02) and YAP1 (2% vs 0.3%; OR 6.86, 95% CI 1.7-39.57, q=0.1), and DNA damage response, such as FAM175A (1.6% vs 0%; OR 18.23, 95% CI 2.43-807.73, q=0.03) and BRCA2 (4% vs 1%; OR 3.09, 95% CI 1.49-6.55, q=0.1), than HER2-0 tumors. In HR-negative HER2-2+ tumors, a higher frequency of PIK3CA mutations was observed in comparison to HER2-0 (36.9% vs 19.5%; OR 2.41, 1.4-4.1, q=0.1), overall and in the metastatic setting. Conclusions: HER2-low BCs seem not to represent a distinct pathologic subtype. At the genomic level, however, some differences were identified and these became more conspicuous upon subclassification of HER2 IHC expression into 1+ and 2+. Further investigation into methods that more accurately detect and quantify low levels of HER2 expression in BC samples as well as better characterize the biology behind the HER2-low/ultralow expression is warranted. Citation Format: Antonio Marra, Anton Safonov, Joshua Drago, Emanuela Ferraro, Pier Selenica, Andrea Gazzo, Giuseppe Curigliano, Shanu Modi, Pedram Razavi, Jorge Reis-Filho, Sarat Chandarlapaty. HER2-07 Genomic Characterization of Primary and Metastatic HER2-low Breast Cancers [abstract]. In: Proceedings of the 2022 San Antonio Breast Cancer Symposium; 2022 Dec 6-10; San Antonio, TX. Philadelphia (PA): AACR; Cancer Res 2023;83(5 Suppl):Abstract nr HER2-07.

e4436026This text presents some reflections on the possibilities of a philosophical education in prisons in the current scenario of the actual pandemic in Brazil. To do so, we first consider, in "Pandemic times: are we worse than covid-19, the critical state of the so-called "civilization” that the pandemic has helped to highlight; in a second moment, "Times of prisons: disappearance by the power of hate" we consider the current state of education in prisons in Brazil, the effects on them of the pandemic and the way the Bolsonaro government responded to it; finally, in "Times of school: reappearance by the wisdom of love" we consider the actual state of education in the schools at prisons and what a philosophical education could be in pandemic times: not only a love of wisdom, but a wisdom of love with and for otherness, to, who knows, bring forth politics of life, and with them, freedom, justice and social peace.ResumoO artigo apresenta reflexões sobre as possibilidades de se pensar uma educação filosófica nas escolas no cárcere no atual cenário de pandemia no Brasil. Para isso consideramos, num primeiro momento, em “Tempos de pandemia: nós somos piores que o covid-19?”, o estado crítico da chamada civilização que a pandemia contribuiu a evidenciar; num segundo momento, “Tempos de cárcere: o desaparecimento pela força do ódio” nos focamos no estado atual do cárcere no Brasil, assim como nos efeitos nele da pandemia e da forma do governo Bolsonaro responder a ela; finalmente, em “Tempos de escola: o reaparecimento pela sabedoria do amor” consideramos o estado atual da educação nas escolas no cárcere no Brasil e o que poderia uma educação filosófica em tempos de pandemia: consideramos a filosofia não apenas um amor à sabedoria, mas uma sabedoria do amor com, pela e para as outridades, para, quem sabe, fazer brotar políticas de vida e, com elas, liberdade, justiça e paz social.ResumenEste texto presenta reflexiones sobre las posibilidades de pensar una educación filosófica en escuelas presiónales en el actual escenario de pandemia en Brasil. Para ello, primero consideramos, en "Tiempos de pandemia: ¿somos peores que el covid-19?" el estado crítico de la llamada "civilización" que la pandemia ha ayudado a resaltar; en un segundo momento “Tiempos de cárcel: la desaparición por la fuerza del odio" consideramos el estado actual de la educación en las prisiones en Brasil y los efectos en ella de la pandemia y la respuesta dada por el gobierno de Bolsonaro; por último, en "Tiempos de escuela: la reaparición por la sabiduría del amor" consideramos el estado actual de la educación en las escuelas carcelarias de Brasil y lo que podría ser una educación filosófica en tiempos de pandemia: no sólo un amor a la sabiduría, sino una sabiduría de amor con, para y por las otredades, para, quién sabe, hacer brotar políticas da vida y, con ellas, libertad, justicia y paz social.Palavras-chave: Cárcere, Ensino de filosofia, Outridade.Keywords: Prison, Teaching philosophy, Otherness.Palabras clave: Cárcel, Enseñanza de la filosofía, Otredades.ReferencesADORNO, Theodor; HORKHEIMER, Max. 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Nije dovoljno poznato koji su doprinos dramskom stvaralaštvu u Zadru dali domaći pisci, kojih je književni izraz bio talijanski, i koliki je odjek imala njihova riječ u kazalištu, u gradu i izvan njega. Ovaj rad ima za cilj da otkrije i vrednuje taj njihov doprinos i odjek. Krajem XVIII i početkom XIX stoljeća Ivan Kreljanović-Albinoni, zanijet antitiranskim raspoloženjem V. Alfierija, oslanjajući se na poetiku Arcadije, najprije sastavlja Orazia, gdje evocira legendu o borbi Horacija i Kuriacija, zatim Manlija Capitolina, s razrađenom idejom o slobodi, te niiz drama i melodrama s istim ili sličnim temama. Kreljainovićeva je zasluga što je, i izvan granica Zadra, posvjedočio o rađanju kulturne i nacionalne svijesti svojih zemljaka. U vrijeme kada je polemički živ romantizatn presjekao put Arcadiji, javlja se Giuseppe Ferrari-Cupdlli, koji koristi prigodne i dramske sastave — kao Il serto conteso — da istakne slavnu tradiciju i hrabrost slavenskih junaka u borbi protiv Turaka i ostalih osvajača. Legende i mitovi, pomiješani s povijesnim zbivanjima u dalmatinskoj prošlosti, osnovna su književna vrela ovog pisca. Značajan je prilog promicanju kazališne djelatnosti Vitaliana Brunellija, a još više Luigija Ficherta, romantičara i pobornika ideje da svaki narod ima pravo na svoju domovinu. Iz njegove drame Bielka di Bosnia zrači želja za bratskim življenjem različitih naroda i o pravu na vlastiti identitet. Plativši danak kazališnoj tradiciji, ali zadojeni novostvorenom kulturnom i političkom klimom, u drugoj polovici XIX stoljeća zadarski dramski autori išli su u potragu za suvremenijim motivima i novim izražajnim sredstvima. Tako se Giuseppe Sabalich trudi da se oslobodi stega tradicije, pribjegavajući miješanju tema i stilova, mijenjanju oblika i jezika. Uporan i plodonosan okreće se narodnim izvorima gdje nalazi obilje zgoda i priča. Antonio de’Bersa i Arturo Colautti posvećuju LeaLru kratki dio svog stvaralačkog razdoblja. De’Bersa se pojavio bez većih umjetničkih pretenzija, a Colautti s namjerom da ostavi traga u teatru. Njegovo nastojanje da rastereti djelo od romantičkih primjesa i verističkog sadržaja i da ih zamijeni dramskom konstrukcijom i virtuoznim dijalogom nije urodio osobitim plodom, ali iako njegovi tekstovi nisu mogli živjeti na pozornici, oni ipak pokazuju da je on bio majstor riječi i da, kao i Sabalichevi, pobuđuju čitaočevu znatiželju. Zadar je u XIX stoljeću imao svoje priznate dramske autore, koji su nastojali uhvatiti korak s općim dostignućima dramskog stvaralaštva pa, iako se nisu probili do estetskih polja, ima ostvarenja gdje je umjetnička riječ oživjela scenu. Početkom novog vijeka talijanska riječ u kazališnom stvaranju ustuknut će pred poletnim hrvatskim književnim izrazom, nadahnutim vlastitim vitalizmom.

Abstract The Society gratefully acknowledges the time and effort of the following individuals who served as reviewers of abstracts for this meeting: ASH ABSTRACTS COORDINATING REVIEWERS Blanche P. Alter Stephen M. Ansell Ralph B. Arlinghaus Scott Armstrong Asad Bashey Philip Bierman Neil Blumberg Chiara Bonini Dominique Bonnet Jacqueline Boultwood Rena Buckstein John C. Byrd Marc Carrier Lucio H. Castilla Selina Chen-Kiang Nicholas Chiorazzi Jorge Cortes-Franco Claire E. Dearden Mary C. Dinauer Harry Paul Erba Carolyn A. Felix Pierre Fenaux Debra L. Friedman Irene M. Ghobrial Jason R. Gotlib Brandon Hayes-Lattin Cheryl A. Hillery Achille Iolascon Jean-Pierre J. Issa Sundar Jagannath Diane F. Jelinek H. Phillip Koeffler John Koreth Robert J. Kreitman Robert B. Levy David Lillicrap Richard Lottenberg John D. McMannis Mark D. Minden Charles G. Mullighan Arnon Nagler Peter J. Newman Robert Z. Orlowski Antonio Palumbo Julie A. Panepinto Warren S. Pear Sibrand Poppema Barbara Pro Ching-Hon Pui A. Koneti Rao Aaron P. Rapoport Pieter H. Reitsma Douglas D. Ross J. Eric Russell Barbara Savoldo Kirk R. Schultz Radek C. Skoda Marilyn L. Slovak Susan Smyth Hugo ten Cate Herve Tilly John M. Timmerman Ivo Touw Amy J. Wagers Russell E. Ware Catherine J. Wu Virginia M. Zaleskas ASH ABSTRACTS REVIEWERS Camille Abboud Omar Abdel-Wahab Jeremy Abramson Suneet Agarwal Sikander Ailawadhi Onder Alpdogan Andrew Aprikyan Mary Armanios Aneel Ashrani Norio Asou Aglaia Athanassiadou Eyal Attar Mohammad Azam Maria Baer Jorg Baesecke Sarah Ball Karen Ballen Frederic Baron Shannon Bates Minoo Battiwalla Marie Bene Charles Bennett James Berenson Steven Bernstein Francesco Bertoni Monica Bessler Wolfgang Bethge Kapil Bhalla Deepa Bhojwani James Bieker Bruce R. Blazar Annemarie Block David Bodine Catherine Bollard Antonio Bonati Eric Bouhassira Benjamin Braun Christopher Bredeson Patrick Brown Ross Brown Jan Burger Dario Campana Jose Cancelas Paul Carpenter Andrew Carroll James Casella Rebecca Chan Roy Chemaly Benny Chen Jerry Cheng Linzhao Cheng Bruce Cheson Mark Chiang Athar Chishti Hearn Cho Magdalena Chrzanowska-Wodnicka Richard E. Clark Joseph Connors Kenneth Cooke Miguel Cruz Adam Cuker Sandeep Dave Janice Davis Sproul Lucia De Franceschi Philip De Groot Rodney DeKoter Richard Delarue Stephen Devereux Steven Devine Paola Jorge Di Don Diamond Meletios Dimopoulos John DiPersio Angela Dispenzieri Benjamin Djulbegovic Jing-fei Dong James Downing William Drobyski Rafael Duarte Charles Dumontet Kieron Dunleavy Brian Durie Dimitar Efremov Elizabeth Eklund Jonas Emsley Patricia Ernst Andrew Evens Chris Fegan Andrew Feldman Giuliana Ferrari Willem Fibbe Adele Fielding Thoas Fioretos Robert Flaumenhaft Rafael Fonseca James Foran Joseph Frank Janet Franklin Paul Frenette Alan Friedman Terry Fry Saghi Gaffari Naomi Galili Patrick Gallagher Anne Galy David Garcia Randy Gascoyne Cristina Gasparetto Norbert Gattermann Tobias Gedde-Dahl Alan Gewirtz Francis Giles Robert Godal Lucy Godley Ivana Gojo Norbert Gorin Andre Goy Eric Grabowski Steven Grant Timothy Graubert Elizabeth Griffiths H. Leighton Grimes Claudia Haferlach Corinne Haioun Parameswaran Hari Christine Harrison Robert Hasserjian Nyla Heerema Shelly Heimfeld Roland Herzog Elizabeth Hexner Teru Hideshima William H. Hildebrand Gerhard Hildebrandt Devendra Hiwase Karin Hoffmeister Donna Hogge Scott Howard Brian Huntly Hiroto Inaba Baba Inusa Shai Izraeli Suresh Jhanwar Amy Johnson Craig Jordan Joseph Jurcic Nina Kadan-Lottick Lawrence Kaplan Jonathan Kaufman Neil Kay Michelle Kelliher Craig Kessler H. Jean Khoury Allison King Joseph Kiss Issay Kitabayashi Robert Klaassen Christoph Klein Yoshihisa Kodera Alexander Kohlmann Barbara Konkle Michael Kovacs Robert Kralovics Amrita Krishnan Nicolaus Kroger Ashish Kumar Ralf Küppers Jeffery Kutok Ann LaCasce Raymond Lai David Lane Peter Lane Richard Larson Michelle Le Beau Gregoire Le Gal Ollivier Legrand Suzanne Lentzsch John Leonard John Levine Ross Levine Linheng Li Renhao Li Zhenyu Li Wendy Lim Charles Linker Jeffrey Lipton Per Ljungman John Lollar Philip Low David Lucas Selina Luger Leo Luznik Gary Lyman Jaroslaw Maciejewski Elizabeth MacIntyre Nigel Mackman Luca Malcovati Guido Marcucci Tomer Mark Susan Maroney Giovanni Martinelli Peter Maslak Alan Mast Grant McArthur Philip McCarthy Michael McDevitt Peter McLaughlin Bruno Medeiros Jules P.P. Meijerink Junia Melo Thomas Mercher Bradley Messmer Marco Mielcarek Ken Mills Shin Mineishi Arturo Molina Silvia Montoto Marie Joelle Mozziconacci Auayporn Nademanee Vesna Najfeld Eneida Nemecek Ellis Neufeld Peter Newburger Heyu Ni Charlotte Marie Niemeyer Yago Nieto Anne Novak Paul O\'Donnell Vivian Oehler Fritz Offner Johannes Oldenburg Rebecca Olin Richard J. O'Reilly Thomas Ortel Keiya Ozawa Rose Ann Padua Sung-Yun Pai James Palis Derwood Pamphilon Animesh Pardanani Farzana Pashankar Andrea Pellagatti Catherine Pellat-Deceunynck Louis Pelus Chris Pepper Melanie Percy Andrew Perkins Luke Peterson Andrew Pettitt Javier Pinilla-Ibarz Kimmo Porkka David Porter Amy Powers Claude Preudhomme Frederick Racke Margaret Ragni Thomas Raife Alessandro Rambaldi Mariusz Ratajczak Pavan Reddy Mary Relling Tannishtha Reya Lisa Rimsza Stefano Rivella Isabelle Riviere Pamela Robey Gail Roboz Aldo Roccaro Maria Alma Rodriguez Frank Rosenbauer Laura Rosinol Alan Rosmarin Giuseppe Saglio Jonathan Said Valeria Santini Ravindra Sarode Yogenthiran Saunthararajah Bipin Savani Alan Schechter Charles Schiffer Robert Schlossman Laurie Sehn Rita Selby Orhan Sezer Sadhna Shankar John Shaughnessy Jordan Shavit Kevin Sheehan Shalini Shenoy Colin Sieff Paul Simmons Seema Singhal Sonali Smith Gerard Socie Pieter Sonneveld Simona Soverini David Spaner Steven Spitalnik Kostas Stamatopoulos David Steensma Richard Stone Toshio Suda Perumal Thiagarajan Courtney Thornburg Rodger Tiedemann David Traver Guido Tricot Darrell Triulzi Suzanne Trudel Christel Van Geet Karin Vanderkerken David Varon Amit Verma Srdan Verstovsek Ravi Vij Dan Vogl Loren Walensky Edmund Waller George Weiner Daniel Weisdorf Karl Welte Peter Westervelt Adrian Wiestner P.W. Wijermans John Wingard Anne Woolfrey Mingjiang Xu Qing Yi Anas Younes Ryan Zarychanski Arthur Zelent Clive Zent Dong-Er Zhang Xianzheng Zhou James Zimring