Добірка наукової літератури з теми "Genotoxic stress response"
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Статті в журналах з теми "Genotoxic stress response"
Antoch, Marina P., and Roman V. Kondratov. "Circadian Proteins and Genotoxic Stress Response." Circulation Research 106, no. 1 (January 8, 2010): 68–78. http://dx.doi.org/10.1161/circresaha.109.207076.
Повний текст джерелаChen, Ting-Yu, Bu-Miin Huang, Tang K. Tang, Yu-Ying Chao, Xiao-Yi Xiao, Pei-Rong Lee, Li-Yun Yang, and Chia-Yih Wang. "Genotoxic stress-activated DNA-PK-p53 cascade and autophagy cooperatively induce ciliogenesis to maintain the DNA damage response." Cell Death & Differentiation 28, no. 6 (January 18, 2021): 1865–79. http://dx.doi.org/10.1038/s41418-020-00713-8.
Повний текст джерелаDbaibo, G. S., M. Y. Pushkareva, R. A. Rachid, N. Alter, M. J. Smyth, L. M. Obeid, and Y. A. Hannun. "p53-dependent ceramide response to genotoxic stress." Journal of Clinical Investigation 102, no. 2 (July 15, 1998): 329–39. http://dx.doi.org/10.1172/jci1180.
Повний текст джерелаShi, Qing-Mei, Yan-Ming Wang, Xin-De Zheng, Raymond Teck Ho Lee, and Yue Wang. "Critical Role of DNA Checkpoints in Mediating Genotoxic-Stress–induced Filamentous Growth inCandida albicans." Molecular Biology of the Cell 18, no. 3 (March 2007): 815–26. http://dx.doi.org/10.1091/mbc.e06-05-0442.
Повний текст джерелаSun, X., H. Shimizu, and K. Yamamoto. "Identification of a novel p53 promoter element involved in genotoxic stress-inducible p53 gene expression." Molecular and Cellular Biology 15, no. 8 (August 1995): 4489–96. http://dx.doi.org/10.1128/mcb.15.8.4489.
Повний текст джерелаLiu, Li, Jiri Veis, Wolfgang Reiter, Edwin Motari, Catherine E. Costello, John C. Samuelson, Gustav Ammerer, and David E. Levin. "Regulation of Pkc1 Hyper-Phosphorylation by Genotoxic Stress." Journal of Fungi 7, no. 10 (October 17, 2021): 874. http://dx.doi.org/10.3390/jof7100874.
Повний текст джерелаSuh, Yousin, Kang-Ae Lee, Woo-Ho Kim, Bok-Ghee Han, Jan Vijg, and Sang Chul Park. "Aging alters the apoptotic response to genotoxic stress." Nature Medicine 8, no. 1 (January 2002): 3–4. http://dx.doi.org/10.1038/nm0102-3.
Повний текст джерелаDutertre, Martin, Gabriel Sanchez, Marie-Cécile De Cian, Jérôme Barbier, Etienne Dardenne, Lise Gratadou, Gwendal Dujardin, Catherine Le Jossic-Corcos, Laurent Corcos, and Didier Auboeuf. "Cotranscriptional exon skipping in the genotoxic stress response." Nature Structural & Molecular Biology 17, no. 11 (October 24, 2010): 1358–66. http://dx.doi.org/10.1038/nsmb.1912.
Повний текст джерелаFORNACE, ALBERT J., JOANY JACKMAN, M. CHRISTINE HOLLANDER, BARBARA HOFFMAN-LIEBERMANN, and DAN A. LIEBERMANN. "Genotoxic-Stress-Response Genes and Growth-Arrest Genes." Annals of the New York Academy of Sciences 663, no. 1 Aging and Cel (November 1992): 139–53. http://dx.doi.org/10.1111/j.1749-6632.1992.tb38657.x.
Повний текст джерелаKumari, Nidhi, M. Abul Hassan, Xiangdong Lu, Robert G. Roeder, and Debabrata Biswas. "AFF1 acetylation by p300 temporally inhibits transcription during genotoxic stress response." Proceedings of the National Academy of Sciences 116, no. 44 (October 14, 2019): 22140–51. http://dx.doi.org/10.1073/pnas.1907097116.
Повний текст джерелаДисертації з теми "Genotoxic stress response"
Konstantinidou, C. "Stage-specific response of NC lineages to genotoxic stress." Thesis, University College London (University of London), 2015. http://discovery.ucl.ac.uk/1458881/.
Повний текст джерелаPurmessur, Nadia Sheree. "The regulation of p53-dependent microRNA expression in response to genotoxic stress." Thesis, University of Leicester, 2014. http://hdl.handle.net/2381/28637.
Повний текст джерелаVickridge, Elise. "Management of E. coli sister chromatid cohesion in response to genotoxic stress." Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLS172/document.
Повний текст джерелаMaintaining genome integrity through replication is an essential process for the cell cycle. However, many factors can compromise this replication and thus the genome integrity. Mitomycin C is a genotoxic agent that creates a covalent link between the two DNA strands. When the replication fork encounters the DNA crosslink, it breaks and creates a DNA double strand break (DSB). Escherichia coli (E.coli) is a widely used model for studying complex DNA mechanisms. When facing a DNA DSB, E. coli activates the SOS response pathway. The SOS response comprises over 50 genes that are under the control of a LexA-repressed promoter. Upon a DSB induction, RecA, a central protein of the SOS response will trigger the degradation of LexA and all the SOS genes will be expressed.We have developed a novel molecular biology tool that reveals contacts between sister chromatids that are cohesive. It has been shown in the lab (Lesterlin et al. 2012) that during a regular cell cycle, the two newly replicated sister chromatids stay in close contact for 10 to 20 min before segregating to separate cell halves thanks to the action of Topoisomerase IV. This step is called sister chromatid cohesion. We have used this molecular biology tool to study sister chromatid cohesion upon a genotoxic stress induced by mitomycin C (MMC). We have shown that sister chromatid cohesion is maintained and prolonged when the cell is facing a DSB. Moreover, this sister chromatid cohesion is dependent on RecN, an SOS induced structural maintenance of chromosome-like (SMC-like) protein. In the absence of RecN, the proximity between both sister chromatids is lost and this has a deleterious effect on cell viability. By tagging the chromosome with fluorescent proteins, we have revealed that RecN can also mediated a progressive regression of two previously segregated sister chromatids and this is coordinated with a whole nucleoid compaction. Further studies showed that this genome compaction is orderly and is not the result of a random compaction in response to DNA damage.Interestingly, inhibiting TopoIV in a recN mutant fully restores viability and sister chromatid cohesion suggesting that RecN’s action is mainly structural. Preserving cohesion through precatenanes is sufficient to favor repair and cell viability even in the absence of RecN.An RNA-seq experiment in a WT strain and a recN mutant revealed that the whole SOS response is downregulated in a recN mutant. This suggests that RecN may have an effect on the induction of the SOS response and thus RecA filament formation. This is in good agreement with the change in RecA-mcherry foci formation we observed. In the WT strain, the RecA-mcherry foci are defined as described in previous work. However, in the recN, the RecA-mcherry foci seemed to form bundle like structures. These RecA bundles were previsously described by Lesterlin et al. in the particular case of a DSB occurring on a chromatid that has already been segregated from its homolog. This could mean that in the absence of recN, the sister chromatids segregate and RecA forms bundle like structures in order to perform a search for the intact homologous sister chromatid.Altogether, these results reveal that RecN is an essential protein for sister chromatid cohesion upon a genotoxic stress. RecN favors sister chromatid cohesion by preventing their segregation. Through a whole nucleoid rearrangement, RecN mediates sister chromatid regression, favoring DNA repair and cell viability
Davidson, Adam. "Investigating the Role of Interferon Regulatory Factor 3 in Response to Genotoxic Stress." Thèse, Université d'Ottawa / University of Ottawa, 2013. http://hdl.handle.net/10393/24928.
Повний текст джерелаLiu, Jia, and 劉佳. "Role of FBXO31 in regulating MAPK-mediated genotoxic stress response and cancer cell survival." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/205657.
Повний текст джерелаpublished_or_final_version
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Al-Asadi, Mazin Gh. "Investigation of dormancy in acute myeloid leukaemia cells and the induction of dormancy in their response to genotoxic stress." Thesis, University of Nottingham, 2017. http://eprints.nottingham.ac.uk/43320/.
Повний текст джерелаStockwell, Simon Reidar. "The role of threonine 286 phosphorylation on cyclin D1 in sub-cellular localisation and proteolysis in response to genotoxic stress." Thesis, Institute of Cancer Research (University Of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406688.
Повний текст джерелаDuarte, Alexandra. "The interplay between MYCN and the DNA damage response : modulation of MYCN expression, its interactions with components of the DNA damage response and cellular responses to N-myc following genotoxic stress." Thesis, Imperial College London, 2012. http://hdl.handle.net/10044/1/9832.
Повний текст джерелаCook, Peter Joseph. "Eya a dual function nuclear factor crucial for regulation of developmental gene expression and prevention of apoptosis in response to genotoxic stress /." Diss., [La Jolla, Calif.] : University of California, San Diego, 2009. http://wwwlib.umi.com/cr/ucsd/fullcit?p3344509.
Повний текст джерелаTitle from first page of PDF file (viewed March 13, 2009). Available via ProQuest Digital Dissertations. Vita. Includes bibliographical references.
Tomkins, C. E. "Cellular responses to genotoxic stress." Thesis, University of Oxford, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.362104.
Повний текст джерелаКниги з теми "Genotoxic stress response"
Grewal, Mandeep Kaur. ARAP2 is induced in response to genotoxic stress and B cell stimulation. 2005.
Знайти повний текст джерелаLaposa, Rebecca Rachelle. The role of DNA repair and genotoxic stress responses in xenobiotic-initiated teratogenesis. 2001.
Знайти повний текст джерелаLaposa, Rebecca Rachelle. The role of DNA repair and genotoxic stress responses in xenobiotic-initiated teratogenesis. 2001.
Знайти повний текст джерелаЧастини книг з теми "Genotoxic stress response"
Holbrook, N. J., Y. Liu, and A. J. Fornace. "Signaling events controlling the molecular response to genotoxic stress." In Stress-Inducible Cellular Responses, 273–88. Basel: Birkhäuser Basel, 1996. http://dx.doi.org/10.1007/978-3-0348-9088-5_18.
Повний текст джерелаMacovei, Anca, Mattia Donà, Daniela Carbonera, and Alma Balestrazzi. "Plant Response to Genotoxic Stress: A Crucial Role in the Context of Global Climate Change." In Abiotic Stress Response in Plants, 13–26. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2016. http://dx.doi.org/10.1002/9783527694570.ch2.
Повний текст джерелаSibon, Ody C. M., and William E. Theurkauf. "Centrosome Regulation in Response to Environmental and Genotoxic Stress." In Centrosomes in Development and Disease, 211–23. Weinheim, FRG: Wiley-VCH Verlag GmbH & Co. KGaA, 2005. http://dx.doi.org/10.1002/3527603808.ch11.
Повний текст джерелаFatima, Uzma, Mohd Farhan Khan, Jamal e Fatima, Uzma Shahab, Saheem Ahmad, and Mohd Aslam Yusuf. "DNA Damage, Response, and Repair in Plants Under Genotoxic Stress." In Stress Signaling in Plants: Genomics and Proteomics Perspective, Volume 2, 151–71. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-42183-4_7.
Повний текст джерелаLiu, Yusen, Myriam Gorospe, Nikki J. Holbrook, and Carl W. Anderson. "Posttranslational Mechanisms Leading to Mammalian Gene Activation in Response to Genotoxic Stress." In DNA Damage and Repair, 263–98. Totowa, NJ: Humana Press, 1998. http://dx.doi.org/10.1007/978-1-59259-455-9_15.
Повний текст джерелаLücke-Huhle, Christine. "Review: Gene Amplification—A Cellular Response to Genotoxic Stress." In Induced Effects of Genotoxic Agents in Eukaryotic Cells, 81–96. CRC Press, 2020. http://dx.doi.org/10.1201/9781003075387-6.
Повний текст джерелаHolbrook, Nikki J., Jennifer D. Leuthy, Jong Sung Park, and Joseph Fargnoli. "gadd153, a Growth Arrest and DNA Damage Inducible Gene: Expression in Response to Genotoxic Stress." In Induced Effects of Genotoxic Agents in Eukaryotic Cells, 125–40. CRC Press, 2020. http://dx.doi.org/10.1201/9781003075387-9.
Повний текст джерелаPietrucha, Barbara. "Ataxia Telangiectasia." In Ataxia - Practice Essentials and Interventions [Working Title]. IntechOpen, 2023. http://dx.doi.org/10.5772/intechopen.112005.
Повний текст джерелаKondratov, Roman V., Victoria Y. Gorbacheva, and Marina P. Antoch. "The Role of Mammalian Circadian Proteins in Normal Physiology and Genotoxic Stress Responses." In Current Topics in Developmental Biology, 173–216. Elsevier, 2007. http://dx.doi.org/10.1016/s0070-2153(06)78005-x.
Повний текст джерелаТези доповідей конференцій з теми "Genotoxic stress response"
Nickens, Kristen P., Ying Han, Harini Shandilya, Gary F. Gerard, Eric P. Kaldjian, Jakob Wikstrom, Orian Shirihai, Steven Patierno, and Susan Ceryak. "Abstract 122: Mitochondrial dysregulation and cellular death resistance in response to genotoxic stress." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-122.
Повний текст джерелаTessari, Anna, Kareesma Parbhoo, Meghan Pawlikowski, Matteo Fassan, Eliana Rulli, Claudia Foray, Alessandra Fabbri, et al. "Abstract A112: RanBP9 protects cells from genotoxic stress and increased expression is predictive of worse response to platinum in NSCLC patients." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics; October 26-30, 2017; Philadelphia, PA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1535-7163.targ-17-a112.
Повний текст джерелаMao, Pingping, Mary Jardine, Lynn Niemaszyk, Jessica Haghkerdar, Eric Yang, Esty G. yanco, Damayanti Desai, et al. "Abstract 2958: The novel protein kinase STK17A is a direct p53 target gene that mediates response to genotoxic and nutritional stress in a cancer cell context-dependent manner." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2958.
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