Добірка наукової літератури з теми "Fetal adiposity"
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Статті в журналах з теми "Fetal adiposity"
Farah, Nadine, Jennifer Hogan, Vicky O'Dwyer, Bernard Stuart, Mairead Kennelly, and Michael J. Turner. "Influence of Maternal Glycemia on Intrauterine Fetal Adiposity Distribution after a Normal Oral Glucose Tolerance Test at 28 Weeks Gestation." Experimental Diabetes Research 2011 (2011): 1–6. http://dx.doi.org/10.1155/2011/951203.
Повний текст джерелаFranca Neto, Antonio H., Melania M. Amorim, Adriana S. Melo, Maria do Carmo P. Lima, Aline Sena, and Lívia Dantas. "Accumulation of Adiposity Fetal During Pregnancy." Obstetrics & Gynecology 127 (May 2016): 132S—133S. http://dx.doi.org/10.1097/01.aog.0000483535.48712.39.
Повний текст джерелаFranca Neto, Antonio H., Melania M. Amorim, Adriana S. Melo, Aline Sena, Maria do Carmo P. Lima, and Jamila V. Silva. "Thigh Measurement and Adiposity Fetal Accumulation." Obstetrics & Gynecology 127 (May 2016): 149S. http://dx.doi.org/10.1097/01.aog.0000483595.55443.9d.
Повний текст джерелаVega-Sanchez, Rodrigo, Hector A. Barajas-Vega, Guadalupe Rozada, Aurora Espejel-Nuñez, Jorge Beltran-Montoya, and Felipe Vadillo-Ortega. "Association between adiposity and inflammatory markers in maternal and fetal blood in a group of Mexican pregnant women." British Journal of Nutrition 104, no. 12 (July 23, 2010): 1735–39. http://dx.doi.org/10.1017/s0007114510002825.
Повний текст джерелаSena, Aline Silva Santos, Alex Sandro Rolland de Souza, Vivianne de Oliveira Barros, Maria do Carmo Pinto Lima, Adriana Suely Oliveira Melo, and Melania Maria Ramos de Amorim. "Prenatal factors associated with fetal visceral adiposity." Jornal de Pediatria 96, no. 3 (May 2020): 341–49. http://dx.doi.org/10.1016/j.jped.2018.11.013.
Повний текст джерелаSena, Aline Silva Santos, Alex Sandro Rolland de Souza, Vivianne de Oliveira Barros, Maria do Carmo Pinto Lima, Adriana Suely Oliveira Melo, and Melania Maria Ramos de Amorim. "Prenatal factors associated with fetal visceral adiposity." Jornal de Pediatria (Versão em Português) 96, no. 3 (May 2020): 341–49. http://dx.doi.org/10.1016/j.jpedp.2018.11.025.
Повний текст джерелаJarvie, Eleanor M., Frances M. Stewart, Jane E. Ramsay, E. Ann Brown, Barbara J. Meyer, Gunilla Olivecrona, Bruce A. Griffin, and Dilys J. Freeman. "Maternal Adipose Tissue Expansion, A Missing Link in the Prediction of Birth Weight Centile." Journal of Clinical Endocrinology & Metabolism 105, no. 3 (December 13, 2019): e814-e825. http://dx.doi.org/10.1210/clinem/dgz248.
Повний текст джерелаLuo, Z.-C., A.-M. Nuyt, E. Delvin, F. Audibert, I. Girard, B. Shatenstein, E. Levy, P. Julien, and W. D. Fraser. "Understanding parental anthropometrical, maternal, and fetal metabolic determinants of fetal adiposity." Canadian Journal of Diabetes 35, no. 2 (January 2011): 177. http://dx.doi.org/10.1016/s1499-2671(11)52143-4.
Повний текст джерелаEdwards, L. J., J. R. McFarlane, K. G. Kauter, and I. C. McMillen. "Impact of periconceptional nutrition on maternal and fetal leptin and fetal adiposity in singleton and twin pregnancies." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 288, no. 1 (January 2005): R39—R45. http://dx.doi.org/10.1152/ajpregu.00127.2004.
Повний текст джерелаWallace, Jacqueline M., John S. Milne, Clare L. Adam, and Raymond P. Aitken. "Impact of donor and recipient adiposity on placental and fetal growth in adolescent sheep." Reproduction 153, no. 4 (April 2017): 381–94. http://dx.doi.org/10.1530/rep-16-0590.
Повний текст джерелаДисертації з теми "Fetal adiposity"
Solé, Navais Pol. "Prenatal one carbon metabolism and in utero programming of growth and adiposity in the offspringPrenatal one carbon metabolism and in utero programming of growth and adiposity in the offspring." Doctoral thesis, Universitat Rovira i Virgili, 2016. http://hdl.handle.net/10803/401551.
Повний текст джерелаEl metabolismo monocarbonado influye positivamente la salud en estadios tempranos y tardíos de la vida (e.g. reducción en la prevalencia de defectos del tubo neural y la muerte por accidente vascular cerebral después de la fortificación con ácido fólico). Se desconocen los efectos del metabolismo monocarbonado in utero en el crecimiento postnatal. Se han atribuido efectos adversos a un estado elevado en folato. El objetivo fue evaluar las interacciones entre el folato y cobalamina prenatales i sus efectos durante el embarazo y sobre el crecimiento y adiposidad en la progenie. Se estudiaron 563 mujeres del Reus-Tarragona Birth Cohort durante el embarazo y 162 descendientes hasta los 7.5 años de edad. Un estado elevado en folatos no modificó la asociación entre concentraciones inadecuadas de cobalamina y sus marcadores metabólicos o hematológicos durante el embarazo. Un estado inadecuado al inicio del embarazo en folato eritrocitario, cobalamina o tHcy al inicio del embarazo se asoció con menor estatura en la descendencia. Un estado inadecuado en holoTC se asoció con mayor IMC en los descendientes. Los polimorfismos maternos MTHFR 677C>T y TCN2 776C>T se asociaron con mayor y menor IMC y estatura desde el nacimiento hasta la infancia respectivamente. El polimorfismo MTRR 66A>G del niño se relacionaba con menor talla y peso. Estos resultados avalan la seguridad de concentraciones elevadas en folato plasmático y sostienen el papel del metabolismo monocarbonado prenatal en el crecimiento y adiposidad postnatales.
One carbon metabolism positively influences health in early and late stages of life (e.g. folic acid fortification reduced neural tube defect prevalence, homocysteine in adults and possiblystroke mortality). Whether the effects of in utero one carbon metabolism affect postnatal growth is unknown. Harmful effects of elevated folate status have been reported. We evaluated the interactions between prenatal folate and cobalamin and their effects during pregnancy and on offspring growth and adiposity at mid-childhood. 563 women from the Reus-Tarragona Birth Cohort were followed-up throughout pregnancy and 162 of their offspring until mid-childhood, when growth and adiposity were measured. Folate status did not modify the association between inadequate cobalamin and its metabolic or haematological indicators. Early pregnancy inadequate RBC folate, cobalamin or tHcy were associated with lower offspring height. Inadequate holoTC was also associated with higher offspring BMI. Maternal MTHFR 677C>T and TCN2 776C>T genetic variants were associated with higher and lower offspring BMI and height from birth to mid-childhood, respectively. Child MTRR 66A>G was associated with lower linear and ponderal growth. This endorses the safety of elevated folate status, and supports a role for prenatal one carbon metabolism on postnatal growth and adiposity.
Ferraro, Zachary Michael. "An Examination of Maternal Contributors and Potential Modifiers of Fetal Growth in Pregnancy." Thèse, Université d'Ottawa / University of Ottawa, 2012. http://hdl.handle.net/10393/22817.
Повний текст джерелаNasr, Elizabeth Maria Bismarck. "Excesso de peso corporal na adolescência segundo períodos críticos para a gênese da obesidade durante a infância." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/6/6138/tde-31012013-085539/.
Повний текст джерелаIntroduction: Considering that obesity is a major risk for many diseases as well as the difficulties and elevated costs for its treatment, its prevention and the identification of early avoidable health risks, such as nutritional status in critical periods of life, represent important aspects for scientific investigation. Objective: Verify the relationship between excessive body weight during adolescence according to birth nutrition status and excessive body weight during the first year of life and at the period of adiposity rebound. Method: This study was conducted with schoolchildren of both sexes, enrolled in Public Schools in São José dos Campos - SP (SJC-SP). The data was collected in 3 phases, the first consisted of collecting anthropometric information during adolescence, considering body mass index (BMI), arm and abdominal circumferences and the sum of skin fold thickness (triceps and sub scapular). Information about mother´s education, breastfeeding and birth nutritional status was collected in the second phase through a survey which the parents answered. The children were classified according to birth weight for gestational age, ponderal index and birth weight. In the last phase, information about nutritional status during infancy was obtained from medical registers of primary health units in the city. Logistical regression analyses were made to investigate the association between excessive body weight at 10 years of age, considered as dependent variable and, as independent variables, the nutritional status at time of birth, the first year of age and during the period of adiposity rebound (between 5 and 7 years of age). The analyses were adjusted by other variables. Results: The students presented a mean of 131.3 months of age (10.99). An elevated correlation was observed between parents information about birth weight and length and the information registered at the medical documents of primary health units in the city (Correlation coefficient: 0.95 and 0.98, for weight and for length, respectively). An association between elevated birth weight and birth weight small for gestational age and excessive body weight during adolescence was observed. A weak association between excessive body weight during the first year of life and at 10 years of age was identified. It was also verified association in relation to adiposity during the period of adiposity rebound and excessive weight at 10 years of age. Conclusion: These results show consistency in the hypothesis that the critical period for growth development could predispose to future obesity, identified in the present study during early adolescence
Tavares, Neuziane Kloos Amorim. "Avaliação das consequências da limitação do tamanho da prole de ratos Wistar ao nascimento sobre seu desenvolvimento ponderal e características morfofuncionais na idade adulta." Universidade de São Paulo, 2013. http://www.teses.usp.br/teses/disponiveis/5/5148/tde-27092013-155436/.
Повний текст джерелаDuring intrauterine life, the developing fetus is susceptible to environmental changes that can alter the phenotype of the individual in postnatal life. This phenomenon is called fetal programming. Events that occur during critical periods of rapid cell division may alter the structure and function of organ systems, resulting in consequences both early (low birth weight) and late (diseases in adulthood) in life. The experimental protocols of most of the studies on fetal programming involve a reduction in litter size soon after birth. This approach hampers the interpretation and reproducibility of the observed results. The purpose of this investigation was to determine if blood pressure, carbohydrate metabolism and energy expenditure in adult offspring are influenced by litter size. Female Wistar rats were fed standard rat chow ad libitum and were mated with male rats at 12 weeks of age. After birth, the offspring were divided into three groups: unchanged litter size (GU), culled to eight neonates (G8) and culled to four neonates (G4). At 12 weeks of age, the body weight; blood pressure; food intake; glucose, insulin, cholesterol and triacylglycerol levels; brown adipose tissue mass; adiposity index; renal mass; and cardiac mass were determined. The body weight, adiposity index, glucose level, insulin level and HOMA index were higher in males and females in the G4 group than in the G8 and GU groups. However, food consumption was lower in G4 males. The blood pressure was higher in the GU group. In summary, a small litter size is related to obesity, possible alterations in energy expenditure and insulin resistance in adult offspring
Ballesteros, Pérez Mònica. "Multímeros de la adiponectina en la diabetes gestacional: relación con parámetros maternos y repercusión en el crecimiento fetal." Doctoral thesis, Universitat Rovira i Virgili, 2011. http://hdl.handle.net/10803/63591.
Повний текст джерелаAdipose tissue plays an active role in metabolic homeostasis. Its functions are mediated by factors secreted by adipocytes known as adipocytokines. Adiponectin is an adipocytokine produced in the adult fat tissue and fetuses in various fetal tissues. Present in the bloodstream in the form of multimers of different molecular weights with different functional implications. Is inversely related to levels of insulin resistance, which increased during pregnancy, gestational diabetes to worsen in the (DMG), an entity associated with fetal macrosomia, we postulate that the quantity and distribution of adiponectin multimers both maternal and fetal, can be one of the factors involved in regulating fetal growth and neonatal adiposity. We postulate a correlation of levels of maternal and fetal adiponectin. We also evaluated new methods of weight estimation in three-dimensional ultrasound.
Grothe, Jon-Frederic [Verfasser], Jörg [Gutachter] Dötsch, and Christhardt [Gutachter] Köhler. "Plazentare Dysfunktion bei maternaler Adipositas : Auswirkungen auf die fetale und plazentare Entwicklung / Jon-Frederic Grothe ; Gutachter: Jörg Dötsch, Christhardt Köhler." Köln : Deutsche Zentralbibliothek für Medizin, 2020. http://d-nb.info/1223658457/34.
Повний текст джерелаPoprzeczny, Amanda Josephine. "Maternal Overweight and Obesity: Effect on Fetal Growth and Adiposity." Thesis, 2022. https://hdl.handle.net/2440/136348.
Повний текст джерелаThesis (Ph.D.) -- University of Adelaide, Adelaide Medical School, 2022
Krafczyk, Bianca [Verfasser]. "Maternale und fetale, neonatale Risiken der Adipositas in der Schwangerschaft : Analyse eines Schwangerenkollektivs mit 508.926 Einlingsgeburten der Jahre 1998 - 2000 der Bundesrepublik Deutschland / vorgelegt von Bianca Krafczyk." 2008. http://d-nb.info/995099642/34.
Повний текст джерелаКниги з теми "Fetal adiposity"
Gluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Macronutrients and fibre requirements during pregnancy. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0004.
Повний текст джерелаGluckman, Sir Peter, Mark Hanson, Chong Yap Seng, and Anne Bardsley. Polyunsaturated fatty acids in pregnancy and breastfeeding. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198722700.003.0005.
Повний текст джерелаЧастини книг з теми "Fetal adiposity"
Khanal, Prabhat, and Mette Olaf Nielsen. "Maternal Undernutrition and Visceral Adiposity." In Diet, Nutrition, and Fetal Programming, 91–105. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-60289-9_8.
Повний текст джерелаSchäfer-Graf, Ute. "Abnormer Glukosemetabolismus und Adipositas: fetale und neonatale Kurz- und Langzeitrisiken und ihr klinisches Management." In Praxisbuch Adipositas in der Geburtshilfe, 145–58. Berlin, Heidelberg: Springer Berlin Heidelberg, 2022. http://dx.doi.org/10.1007/978-3-662-61906-3_7.
Повний текст джерелаLow, Felicia, Peter Gluckman, and Mark Hanson. "Fetal and Early Postnatal Life Determinants of Adiposity." In Handbook of Obesity, 127–36. CRC Press, 2014. http://dx.doi.org/10.1201/b16473-14.
Повний текст джерелаТези доповідей конференцій з теми "Fetal adiposity"
Bukowski, Michael, Brij Singh, James Roemmich, and Kate Larson. "Lipidomic analysis of TRPC1 Ca2+-permeable channel-knock out mouse demonstrates a vital role in placental tissue sphingolipid and triacylglycerol homeostasis under high-fat diet." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/tjdt4839.
Повний текст джерела