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Статті в журналах з теми "Eye"

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Автор: Grafiati

Опубліковано: 4 червня 2021

Оновлено: 5 березня 2023

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1

Ishikawa, Hiroto, Kazutaka Uchida, Yoshio Takesue, Junya Mori, Takamasa Kinoshita, Shohei Morikawa, Fumiki Okamoto, et al. "Clinical Characteristics and Outcomes in 314 Japanese Patients with Bacterial Endophthalmitis: A Multicenter Cohort Study from J-CREST." Pathogens 10, no. 4 (March 24, 2021): 390. http://dx.doi.org/10.3390/pathogens10040390.

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Bacterial endophthalmitis is an intraocular infection that causes rapid vison loss. Pathogens can infect the intraocular space directly (exogenous endophthalmitis (ExE)) or indirectly (endogenous endophthalmitis (EnE)). To identify predictive factors for the visual prognosis of Japanese patients with bacterial endophthalmitis, we retrospectively examined the bacterial endophthalmitis characteristics of 314 Japanese patients and performed statistics using these clinical data. Older patients, with significantly more severe clinical symptoms, were prevalent in the ExE group compared with the EnE group. However, the final best-corrected visual acuity (BCVA) was not significantly different between the ExE and EnE groups. Bacteria isolated from patients were not associated with age, sex, or presence of eye symptoms. Genus Streptococcus, Streptococcus pneumoniae, and Enterococcus were more prevalent in ExE patients than EnE patients and contributed to poor final BCVA. The presence of eye pain, bacterial identification, and poor BCVA at baseline were risk factors for final visual impairment.

2

Bonini, N. M., Q. T. Bui, G. L. Gray-Board, and J. M. Warrick. "The Drosophila eyes absent gene directs ectopic eye formation in a pathway conserved between flies and vertebrates." Development 124, no. 23 (December 1, 1997): 4819–26. http://dx.doi.org/10.1242/dev.124.23.4819.

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The fly eyes absent (eya) gene which is essential for compound eye development in Drosophila, was shown to be functionally replaceable in eye development by a vertebrate Eya homolog. The relationship between eya and that of the eyeless gene, a Pax-6 homolog, critical for eye formation in both flies and man, was defined: eya was found to be essential for eye formation by eyeless. Moreover, eya could itself direct ectopic eye formation, indicating that eya has the capacity to function as a master control gene for eye formation. Finally, we show that eya and eyeless together were more effective in eye formation than either gene alone. These data indicate conservation of the pathway of eya function between flies and vertebrates; they suggest a model whereby eya/Eya gene function is essential for eye formation by eyeless/Pax-6, and that eya/Eya can in turn mediate, via a regulatory loop, the activity of eyeless/Pax-6 in eye formation.

3

Sami, A. R. "Eye, eye." BMJ 298, no. 6686 (June 3, 1989): 1523. http://dx.doi.org/10.1136/bmj.298.6686.1523-b.

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4

Krishnamoorthy, Gurumoorthy, and Hartmut Wekerle. "EAE: An immunologist's magic eye." European Journal of Immunology 39, no. 8 (August 2009): 2031–35. http://dx.doi.org/10.1002/eji.200939568.

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5

Rodman, David. "Eye to eye." Israel Affairs 23, no. 2 (March 4, 2017): 450–51. http://dx.doi.org/10.1080/13537121.2017.1302176.

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6

Durden, Mark. "Eye-to-Eye." Art History 23, no. 1 (March 2000): 124–29. http://dx.doi.org/10.1111/1467-8365.00199.

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7

Mathieson, Kay. "Eye to eye." Nursery World 2015, no. 19 (September 21, 2015): 20. http://dx.doi.org/10.12968/nuwa.2015.19.20.

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8

Goddard, Charlotte. "Eye to eye." Nursery World 2018, no. 21 (October 15, 2018): 16–17. http://dx.doi.org/10.12968/nuwa.2018.21.16.

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9

Ye, Yvaine. "Eye to eye." New Scientist 239, no. 3194 (September 2018): 24–25. http://dx.doi.org/10.1016/s0262-4079(18)31616-6.

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10

Whatley, Kirsten. "Eye to Eye." River Teeth: A Journal of Nonfiction Narrative 8, no. 1 (2006): 136–38. http://dx.doi.org/10.1353/rvt.2006.0027.

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11

Zimmerman, John E., Quang T. Bui, Haixi Liu, and Nancy M. Bonini. "Molecular Genetic Analysis of Drosophila eyes absent Mutants Reveals an Eye Enhancer Element." Genetics 154, no. 1 (January 1, 2000): 237–46. http://dx.doi.org/10.1093/genetics/154.1.237.

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Abstract The eyes absent (eya) gene is critical for normal eye development in Drosophila and is highly conserved to vertebrates. To define regions of the gene critical for eye function, we have defined the mutations in the four viable eya alleles. Two of these mutations are eye specific and undergo transvection with other mutations in the gene. These were found to be deletion mutations that remove regulatory sequence critical for eye cell expression of the gene. Two other viable alleles cause a reduced eye phenotype and affect the function of the gene in additional tissues, such as the ocelli. These mutations were found to be insertion mutations of different transposable elements within the 5′ UTR of the transcript. Detailed analysis of one of these revealed that the transposable element has become subject to regulation by eye enhancer sequences of the eya gene, disrupting normal expression of EYA in the eye. More extended analysis of the deletion region in the eye-specific alleles indicated that the deleted region defines an enhancer that activates gene expression in eye progenitor cells. This enhancer is responsive to ectopic expression of the eyeless gene. This analysis has defined a critical regulatory region required for proper eye expression of the eya gene.

12

Selvapriya, Ms R., and Raghul D. "The Invisible Eye." International Journal of Trend in Scientific Research and Development Volume-3, Issue-3 (April 30, 2019): 66–67. http://dx.doi.org/10.31142/ijtsrd21627.

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13

Miller, Julie Ann. "Eye to (Third) Eye." Science News 128, no. 19 (November 9, 1985): 298. http://dx.doi.org/10.2307/3970164.

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14

Wolpaw, Daniel R. "Seeing Eye to Eye." New England Journal of Medicine 365, no. 22 (December 2011): 2052–53. http://dx.doi.org/10.1056/nejmp1108469.

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15

Fagenson‐Eland, Ellen A., S. Gayle Baugh, and Melenie J. Lankau. "Seeing eye to eye." Career Development International 10, no. 6/7 (October 2005): 460–77. http://dx.doi.org/10.1108/13620430510620557.

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16

Vidal, David. "Eye for an eye." European Journal of Marketing 48, no. 1/2 (February 4, 2014): 47–67. http://dx.doi.org/10.1108/ejm-03-2011-0173.

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Purpose – The purpose of this research is to examine why a buying firm in a marketing channel may retaliate against its supplier. The objective of this paper is thus to understand the individual and organizational variables that may prompt a buying company to retaliate against its supplier following negative critical incidents (NCI). Design/methodology/approach – Data was collected from 171 retailers associated with one focal manufacturer and analyzed through PLS path modeling procedures. Findings – Results demonstrate that retaliation is the outcome of individual factors related to a buyer's cognitive (causal attributions) and emotional (anger) processes triggered by NCI as well as organizational forces (trust and dependence) related to more stable characteristics of the interfirm relationship. Originality/value – Compared with existing contributions, the proposed model adopts a multilevel approach and considers retaliation as the outcome of individual as well as organizational forces. On the individual level, echoing the rapidly growing idea that emotions, not just cognitions, are a relevant object of study within interorganizational relationships, this paper empirically investigates the effect of one negative emotion (anger) and of causal attributions in buyer-seller partnerships. On the organizational level, this research examines the influence of trust and dependence, two central variables in business-to-business marketing theory, which seem closely related to retaliation.

17

Seery, Mary Ellen, Pegi M. Davis, and Lawrence J. Johnson. "Seeing Eye-to-Eye." Remedial and Special Education 21, no. 5 (September 2000): 268–319. http://dx.doi.org/10.1177/074193250002100504.

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18

DORNHORST, A. C. "Seeing eye to eye." Nature 329, no. 6142 (October 1987): 758. http://dx.doi.org/10.1038/329758b0.

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19

Rosser, Sylvia L. "The Eye Book: Eye and Eye Problems Explained." Optometry and Vision Science 78, no. 12 (December 2001): 865. http://dx.doi.org/10.1097/00006324-200112000-00007.

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20

Curtiss, J., and M. Mlodzik. "Morphogenetic furrow initiation and progression during eye development in Drosophila: the roles of decapentaplegic, hedgehog and eyes absent." Development 127, no. 6 (March 15, 2000): 1325–36. http://dx.doi.org/10.1242/dev.127.6.1325.

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The Drosophila signaling factor decapentaplegic (dpp) mediates the effects of hedgehog (hh) in tissue patterning by regulating the expression of tissue-specific genes. In the eye disc, the transcription factors eyeless (ey), eyes absent (eya), sine oculis (so) and dachshund (dac) participate with these signaling molecules in a complex regulatory network that results in the initiation of eye development. Our analysis of functional relationships in the early eye disc indicates that hh and dpp play no role in regulating ey, but are required for eya, so and dac expression. We show that restoring expression of eya in loss-of-function dpp mutant backgrounds is sufficient to induce so and dac expression and to rescue eye development. Thus, once expressed, eya can carry out its functions in the absence of dpp. These experiments indicate that dpp functions downstream of or in parallel with ey, but upstream of eya, so and dac. Additional control is provided by a feedback loop that maintains expression of eya and so and includes dpp. The fact that exogenous overexpression of ey, eya, so and dac interferes with wild-type eye development demonstrates the importance of such a complicated mechanism for maintaining proper levels of these factors during early eye development. Whereas initiation of eye development fails in either Hh or Dpp signaling mutants, the subsequent progression of the morphogenetic furrow is only slowed down. However, we find that clones that are simultaneously mutant for Hh and Dpp signaling components completely block furrow progression and eye differentiation, suggesting that Hh and Dpp serve partially redundant functions in this process. Interestingly, furrow-associated expression of eya, so and dac is not affected by double mutant tissue, suggesting that some other factor(s) regulates their expression during furrow progression.

21

Qin, Dan-Yi, and Ying-Ping Deng. "Transgenic dry eye mouse models: powerful tools to study dry eye disease." International Journal of Ophthalmology 15, no. 4 (April 18, 2022): 635–45. http://dx.doi.org/10.18240/ijo.2022.04.18.

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Dry eye disease (DED) is one of the most common chronic multifactorial ocular surface diseases with high prevalence and complex pathogenesis. DED results in several ocular discomforts, vision fluctuation, and even potential damage of the ocular surface, bringing heavy burdens both on individuals and the society. The pathology of DED consists of tear film hyperosmolarity and immune responses on the ocular surface. Mice are widely used for developing models that simulate human DED features for investigating its pathogenesis and treatment. DED can be classified into aqueous-deficiency dry eye (ADDE) and evaporative dry eye (EDE). ADDE can be further divided into Sjögren syndrome dry eye (SSDE) and non-Sjögren syndrome dry eye (NSSDE). SSDE mouse models include natural strains, typified by non-obese diabetic (NOD) mice, and genetically engineered ones, like Aire-/- and Id3 knockout mice. Intrinsic EDE mainly refers to meibomian gland dysfunction (MGD). Eda-/- Tabby, Sod1-/-, Elovl1-/- are the most common transgenic MGD mouse models. Transgenic mouse models provide useful tools for studying the pathogenesis of DED and evaluating its novel therapies. This review compares the major transgenic dry eye mouse models and discusses their applications in DED research.

22

Carpenter, R. H. S. "Supplementary Eye Field: Keeping an Eye on Eye Movement." Current Biology 14, no. 11 (June 2004): R416—R418. http://dx.doi.org/10.1016/j.cub.2004.05.032.

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23

Bui, Quang T., John E. Zimmerman, Haixi Liu, and Nancy M. Bonini. "Molecular Analysis of Drosophilaeyes absentMutants Reveals Features of the Conserved Eya Domain." Genetics 155, no. 2 (June 1, 2000): 709–20. http://dx.doi.org/10.1093/genetics/155.2.709.

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AbstractThe eyes absent (eya) gene is critical to eye formation in Drosophila; upon loss of eya function, eye progenitor cells die by programmed cell death. Moreover, ectopic eya expression directs eye formation, and eya functionally synergizes in vivo and physically interacts in vitro with two other genes of eye development, sine oculis and dachshund. The Eya protein sequence, while highly conserved to vertebrates, is novel. To define amino acids critical to the function of the Eya protein, we have sequenced eya alleles. These mutations have revealed that loss of the entire Eya Domain is null for eya activity, but that alleles with truncations within the Eya Domain display partial function. We then extended the molecular genetic analysis to interactions within the Eya Domain. This analysis has revealed regions of special importance to interaction with Sine Oculis or Dachshund. Select eya missense mutations within the Eya Domain diminished the interactions with Sine Oculis or Dachshund. Taken together, these data suggest that the conserved Eya Domain is critical for eya activity and may have functional subregions within it.

24

Majumder, ParthopratimDutta, Kowsigan Magesan, and Vikas Khetan. "An eye within an eye." Indian Journal of Ophthalmology 67, no. 1 (2019): 139. http://dx.doi.org/10.4103/ijo.ijo_1135_18.

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25

Shekhar, Madhu, R. Sankarananthan, and RSenthil Prasad. "An eye inside an eye." Indian Journal of Ophthalmology - Case Reports 1, no. 2 (2021): 180. http://dx.doi.org/10.4103/ijo.ijo_2779_20.

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26

Honavar, SantoshG, and Raju Kumar. "An eye for an eye." Oman Journal of Ophthalmology 7, no. 3 (2014): 109. http://dx.doi.org/10.4103/0974-620x.142590.

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27

Hayashi, Mitsuko. "An eye for an eye." TRENDS IN THE SCIENCES 4, no. 4 (1999): 53–55. http://dx.doi.org/10.5363/tits.4.4_53.

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28

Seltzer, David. "An Eye for an Eye." International Studies in Philosophy 39, no. 1 (2007): 59–77. http://dx.doi.org/10.5840/intstudphil200739126.

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29

King, Bruce, and Bandula Chandraratna. "An Eye for an Eye." World Literature Today 76, no. 1 (2002): 135. http://dx.doi.org/10.2307/40157066.

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30

P.Y. "An Eye for an Eye." Scientific American 282, no. 6 (June 2000): 34. http://dx.doi.org/10.1038/scientificamerican0600-34b.

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31

Sinn, Rebecca, and Joachim Wittbrodt. "An eye on eye development." Mechanisms of Development 130, no. 6-8 (June 2013): 347–58. http://dx.doi.org/10.1016/j.mod.2013.05.001.

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32

Cavonius, C. R. "Not seeing eye to eye." Nature 370, no. 6487 (July 1994): 259–60. http://dx.doi.org/10.1038/370259a0.

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33

Khawaja, Irfan. "An Eye for an Eye?" Teaching Philosophy 26, no. 2 (2003): 199–204. http://dx.doi.org/10.5840/teachphil200326221.

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34

Dolman, Constance. "Eye to eye by proxy." Kind en Adolescent Praktijk 5, no. 2 (June 2006): 73–74. http://dx.doi.org/10.1007/bf03059585.

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35

Jerrold, Laurance. "An eye for an eye." American Journal of Orthodontics and Dentofacial Orthopedics 135, no. 2 (February 2009): 260–61. http://dx.doi.org/10.1016/j.ajodo.2008.09.011.

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36

Dodaro, Nicholas R. "An Eye for an Eye." Archives of Ophthalmology 113, no. 6 (June 1, 1995): 824. http://dx.doi.org/10.1001/archopht.1995.01100060150051.

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37

Leiserson, W. M., N. M. Bonini, and S. Benzer. "Transvection at the eyes absent gene of Drosophila." Genetics 138, no. 4 (December 1, 1994): 1171–79. http://dx.doi.org/10.1093/genetics/138.4.1171.

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Abstract The Drosophila eyes absent (eya) gene is required for survival and differentiation of eye progenitor cells. Loss of gene function in the eye results in reduction or absence of the adult compound eye. Certain combinations of eya alleles undergo partial complementation, with dramatic restoration of eye size. This interaction is sensitive to the relative positions of the two alleles in the genome; rearrangements predicted to disrupt pairing of chromosomal homologs in the eya region disrupt complementation. Ten X-ray-induced rearrangements that suppress the interaction obey the same general rules as those that disrupt transvection at the bithorax complex and the decapentaplegic gene. Moreover, like transvection in those cases, the interaction at eya depends on the presence of normal zeste function. The discovery of transvection at eya suggests that transvection interactions of this type may be more prevalent than generally thought.

38

Patro, Manisha Kumari. "Counseling in Eye Care." Journal of Multidisciplinary Research in Healthcare 1, no. 2 (April 6, 2015): 133–36. http://dx.doi.org/10.15415/jmrh.2015.12009.

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39

Carey, David P. "Eye–hand coordination: Eye to hand or hand to eye?" Current Biology 10, no. 11 (June 2000): R416—R419. http://dx.doi.org/10.1016/s0960-9822(00)00508-x.

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40

Schoenemann, Brigitte. "Trilobite Eyes and a New Type of Neural Superposition Eye in an Ancient System." Palaeontographica Abteilung A 281, no. 1-3 (November 7, 2007): 63–91. http://dx.doi.org/10.1127/pala/281/2007/63.

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41

Khatib, T., A. D. Singh, and H. S. Dua. "One-eyed Odin: an eye for wisdom." British Journal of Ophthalmology 95, no. 12 (November 16, 2011): 1651. http://dx.doi.org/10.1136/bjophthalmol-2011-301147.

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42

Jenkins, Philip. "From "Eye-Witness" to "Private Eye"." Chesterton Review 18, no. 2 (1992): 225–43. http://dx.doi.org/10.5840/chesterton199218211.

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43

Fukuda, Masahiko, and Hsiao-Fu Wang. "Dry Eye and Closed Eye Tears." Cornea 19, Supplement 1 (May 2000): S44—S48. http://dx.doi.org/10.1097/00003226-200005001-00009.

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44

Potter, Polyxeni. "Eye to Eye in the Village." Emerging Infectious Diseases 14, no. 12 (December 2008): 1978–80. http://dx.doi.org/10.3201/eid1412.ac1412.

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45

Hibberd, Patricia L., and Ann Sullivan Baker. "Dangers of "Eye-to-Eye" Contact." Infection Control and Hospital Epidemiology 10, no. 3 (March 1989): 99–101. http://dx.doi.org/10.2307/30105106.

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46

Kestelyn, P. G. "AN EYE ON INFLAMMATORY EYE DISEASE." Acta Clinica Belgica 60, no. 5 (October 2005): 270–75. http://dx.doi.org/10.1179/acb.2005.044.

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47

Hibberd, Patricia L., and Ann Sullivan Baker. "Dangers of "Eye-to-Eye" Contact." Infection Control and Hospital Epidemiology 10, no. 3 (March 1989): 99–101. http://dx.doi.org/10.1086/645974.

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48

Hill, Robert E., and Duncan R. Davidson. "Comparative Development: Seeing eye to eye." Current Biology 4, no. 12 (December 1994): 1155–57. http://dx.doi.org/10.1016/s0960-9822(00)00262-1.

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49

SCHWAB, I. R. "From eye spots to eye shine." British Journal of Ophthalmology 84, no. 11 (November 1, 2000): 1214–16. http://dx.doi.org/10.1136/bjo.84.11.1214.

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50

MacGILLNRAY, R. G., and J. A. ODELL. "Eye to eye with Murphy's law." Anaesthesia 41, no. 3 (March 1986): 334. http://dx.doi.org/10.1111/j.1365-2044.1986.tb12819.x.

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