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Автор: Grafiati
Опубліковано: 15 червня 2024

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  1. Статті в журналах
  2. Дисертації
  3. Книги
  4. Частини книг
  5. Тези доповідей конференцій

Статті в журналах з теми "EMF exposure map":

1

Wang, Shanshan, and Joe Wiart. "Sensor-Aided EMF Exposure Assessments in an Urban Environment Using Artificial Neural Networks." International Journal of Environmental Research and Public Health 17, no. 9 (April 28, 2020): 3052. http://dx.doi.org/10.3390/ijerph17093052.

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This paper studies the time and space mapping of the electromagnetic field (EMF) exposure induced by cellular base station antennas (BSA) using artificial neural networks (ANN). The reconstructed EMF exposure map (EEM) in urban environment is obtained by using data from EMF sensor networks, drive testing and information accessible in a public database, e.g., locations and orientations of BSA. The performance of EEM is compared with Exposure Reference Map (ERM) based on simulations, in which parametric path loss models are used to reflect the complexity of urban cities. Then, a new hybrid ANN, which has the advantage of sorting and utilizing inputs from simulations efficiently, is proposed. Using both hybrid ANN and conventional regression ANN, the EEM is reconstructed and compared to the ERM first by the reconstruction approach considering only EMF exposure assessed from sensor networks, where the required number of sensors towards good reconstruction is explored; then, a new reconstruction approach using the sensors information combined with EMF along few streets from drive testing. Both reconstruction approaches use simulations to mimic measurements. The influence of city architecture on EMF exposure reconstruction is analyzed and the addition of noise is considered to test the robustness of ANN as well.
2

Seo, Myung-Soon, Jae-Wook Choi, Kyung-Hee Kim, and Hyung-Do Choi. "The Relationship between Risk Perception of Cell Phones and Objective Knowledge of EMF in Korea." International Journal of Environmental Research and Public Health 17, no. 19 (October 1, 2020): 7207. http://dx.doi.org/10.3390/ijerph17197207.

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This study examines differences between the level of objective knowledge regarding radio-frequency electromagnetic fields (RF-EMF) and risk perception of cell phones in Korea. We also investigate the extent to which socio-demographic factors, perceived EMF exposure, objective knowledge regarding EMF, and psychological factors influence the risk perception of cell phones using hierarchical multiple regression. All 3393 study subjects completed a survey measuring the degree of risk perception of EMF. They were sampled in accordance with representative proportions of sex, age group, and region of residence as shown in the 2019 Resident Registration Population Statistics reported by Korea. The variables that have the most influence on risk perception of cell phones can be induced from the beta values for each variable: The subjective factor, perceived level of exposure to EMF (β = 0.253), was more strongly related to risk perception of cell phones than level of knowledge regarding EMF, an objective factor in this study. Of the psychological factors, Dreadfulness (β = 0.331), Personal knowledge (β = −174), and Familiarity (β = −089) influenced risk perceptions of cell phones; Controllability did not. On the risk cognition map, people though that it was easy to control risk related to Cell phone use in daily life, while risk related to High technology was uncontrollable.
3

Gonzalez-Rubio, Jesus, Alberto Najera, and Enrique Arribas. "Comprehensive personal RF-EMF exposure map and its potential use in epidemiological studies." Environmental Research 149 (August 2016): 105–12. http://dx.doi.org/10.1016/j.envres.2016.05.010.

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4

Pang, L., S. J. Decker, and A. R. Saltiel. "Bombesin and epidermal growth factor stimulate the mitogen-activated protein kinase through different pathways in Swiss 3T3 cells." Biochemical Journal 289, no. 1 (January 1, 1993): 283–87. http://dx.doi.org/10.1042/bj2890283.

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Both bombesin and epidermal growth factor (EGF) are potent mitogens in Swiss 3T3 cells that nonetheless have dissimilar receptor structures. To explore possible common intracellular events involved in the stimulation of cellular growth by these two peptides, we have evaluated the regulation of the mitogen-activated protein (MAP) kinase. Exposure of Swiss 3T3 cells to bombesin, EGF or the protein kinase C activator phorbol 12-myristate 13-acetate (PMA) causes the rapid and transient stimulation of the enzyme activity. Pretreatment of cells with the protein kinase inhibitor H-7, or down-regulation of cellular protein kinase C by prolonged exposure to PMA, causes a decrease of over 90% in the activation of MAP kinase by bombesin. In contrast, these treatments have no effect on the stimulation of MAP kinase by EGF. The stimulation of MAP kinase activity by bombesin is dose-dependent, occurring over a narrow concentration range of the peptide. Both EGF and bombesin stimulate the phosphorylation of an immunoprecipitable MAP kinase protein migrating at 42 kDa on SDS/PAGE. Phosphoamino acid analysis of this phosphorylated protein reveals that EGF and bombesin stimulate phosphorylation on tyrosine, threonine and serine residues. Tyrosine phosphorylation of the enzyme, as evaluated by antiphosphotyrosine blotting of the immunoprecipitated protein, reveals that the time course of phosphorylation by both mitogens correlates with stimulation of enzyme activity. These results provide further evidence for the convergence of discrete pathways emanating from tyrosine kinase and G-protein-linked receptors in the regulation of MAP kinase.
5

Georgiou, Christos D., Electra Kalaitzopoulou, Marianna Skipitari, Polyxeni Papadea, Athina Varemmenou, Vassilios Gavriil, Evangelia Sarantopoulou, Zoe Kollia, and Alkiviadis-Constantinos Cefalas. "Physical Differences between Man-Made and Cosmic Microwave Electromagnetic Radiation and Their Exposure Limits, and Radiofrequencies as Generators of Biotoxic Free Radicals." Radiation 2, no. 4 (September 22, 2022): 285–302. http://dx.doi.org/10.3390/radiation2040022.

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The critical arguments for radiofrequency radiation exposure limits are currently based on the principle that radio frequencies (RF) and electromagnetic fields (EMFs) are non-ionising, and their exposure limits are even 100-fold lower than those emitted from the Sun in the whole RF-EMF spectrum. Nonetheless, this argument has been challenged by numerous experimental and theoretical studies on the diverse biological effects of RF-EMF at much lower power density (W/m2) levels than today’s exposing limits. On the other hand, less attention has been given to counterarguments based on the differences in the physics concepts underlying man-made versus natural electromagnetic radiation (EMR) and on the fact that man’s biology has been adapted to the natural EMR levels reaching Earth’s surface at single EMF wavelengths, which are the natural limits of man’s exposure to EMFs. The article highlights the main points of interaction of natural and man-made radiation with biomatter and reveals the physical theoretical background that explains the effects of man-made microwave radiation on biological matter. Moreover, the article extends its analysis on experimental quantum effects, establishing the “ionising-like” effects of man-made microwave radiation on biological matter.
6

Kelleher, M. D., M. K. Abe, T. S. Chao, M. Jain, J. M. Green, J. Solway, M. R. Rosner, and M. B. Hershenson. "Role of MAP kinase activation in bovine tracheal smooth muscle mitogenesis." American Journal of Physiology-Lung Cellular and Molecular Physiology 268, no. 6 (June 1, 1995): L894—L901. http://dx.doi.org/10.1152/ajplung.1995.268.6.l894.

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Abnormal growth of airway smooth muscle may play an important role in the pathogenesis of human airway diseases. Little is known about the proliferative responses of cultured airway smooth muscle cells, nor of the precise pathways responsible for mitogenesis in these cells. We assessed DNA synthesis, cell proliferation, and mitogen-activated protein (MAP) kinase activation in bovine tracheal myocytes after exposure to four potential mitogens: platelet-derived growth factor (PDGF), epidermal growth factor (EGF), insulin-like growth factor-1 (IGF-1), and 5-hydroxytryptamine (5-HT). Stimulation with either PDGF or IGF-1 induced substantial increases in DNA synthesis and cell number, as reflected by [3H]thymidine incorporation, flow cytometry, and methylene blue staining. Treatment with EGF or 5-HT, on the other hand, induced only modest DNA synthesis and no increase in cell number. Immunoblots and kinase renaturation assays of cell extracts demonstrated activation of both the 42- and 44-kDa MAP kinases within minutes of either PDGF, IGF-1, EGF, or 5-HT exposure. However, relative to EGF and 5-HT stimulation, late-phase MAP kinase activation was significantly greater after treatment with the mitogens PDGF and IGF-1. We conclude that in cultured bovine tracheal myocytes 1) PDGF and IGF-1 are potent mitogens; 2) MAP kinase may be activated subsequent to stimulation of either receptor tyrosine kinases (PDGF, EGF, IGF-1) or G protein-linked receptors lacking in known tyrosine kinase activity (5-HT); and 3) unsustained MAP kinase activation is insufficient for mitogenesis. Finally, the finding that mitogenicity correlates with the late phase of MAP kinase activation is consistent with the notion that sustained MAP kinase activation is important for bovine tracheal myocyte proliferation.
7

Gökmen, Uğur, Zübeyde Özkan, and Sema Bilge Ocak. "Impact of the gamma and neutron attenuation behaviors on the functionally graded composite materials." Physica Scripta 96, no. 12 (December 1, 2021): 125326. http://dx.doi.org/10.1088/1402-4896/ac41ef.

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Abstract Gamma-ray and neutron shielding properties of the AA6082 + TiO2 (0–50 wt%) functionally graded composite materials (FGCMs) were investigated using the PSD software. The values of the mean free path (MFP), half-value layer (HVL), linear attenuation coefficients (LAC), mass attenuation coefficient (MAC), tenth-value layer (TVL), exposure buildup factors (EBF), effective atomic number (Z eff ), effective conductivity (C eff ), and fast neutron removal cross-sections (FNRC) were found for the energy range between 0.015–15 MeV. The increase in the TiO2 content in the AA6082 composite material has raised the values of MAC and LAC. The calculations for the EBFs were carried out using the G-P fitting method for the energy range between 0.015–15 MeV and penetration depth of up to 40 mfp. The results revealed that HVL values ranged between 0.01–0.116 cm, TVL values ranged between 0.01–0.385 cm, FNRC values ranged between 7.918–10.017 cm−1, and C eff values ranged between 5.67 × 1010 and 9.85 × 1010 S m−1. The AA6082 + TiO2 (50%) composite material was observed to provide the maximum photon and neutron shielding capacity since it offered the highest Z eff , MAC, and FNRC values, and the lowest HVL value. In terms of several aspects, the research is considered original. Besides contributing to several technologies including nanotechnology and space technologies, present research’s results may contribute to nuclear technology.
8

Heasley, L. E., S. I. Senkfor, S. Winitz, A. Strasheim, I. Teitelbaum, and T. Berl. "Hormonal regulation of MAP kinase in cultured rat inner medullary collecting tubule cells." American Journal of Physiology-Renal Physiology 267, no. 3 (September 1, 1994): F366—F373. http://dx.doi.org/10.1152/ajprenal.1994.267.3.f366.

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Mitogen-activated protein (MAP) kinase is a widely expressed protein serine/threonine kinase that serves as a convergence point for many signaling pathways including receptor tyrosine kinases, G protein-coupled receptors, and protein kinase C (PKC). The hormonal regulation of MAP kinase was studied in cultured established rat inner medullary collecting tubule (RIMCT) cells. Neither vasopressin nor beta-adrenergic agonists stimulated MAP kinase, despite clear stimulation of adenosine 3',5'-cyclic monophosphate (cAMP)-dependent protein kinase. In contrast, carbachol, ATP, and epidermal growth factor (EGF), which are known to antagonize vasopressin action in the RIMCT, stimulated the MAP kinase pathway. This stimulation was mimicked by the phorbol ester, 12-O-tetradecanoylphorbol-13-acetate, which directly activates PKC. The potency with which EGF and carbachol activated MAP kinase was similar to the potency with which they inhibited vasopressin-stimulated cAMP accumulation. To assess the role of Gi proteins in these stimulatory events, RIMCT cells were pretreated with pertussis toxin to inhibit Gi-mediated signaling. Pertussis toxin did not influence ATP- or EGF-stimulated MAP kinase, but completely inhibited carbachol stimulation, suggesting that Gi proteins mediate muscarinic stimulation. Prolonged exposure of RIMCT cells to high phorbol ester concentrations to downregulate PKC ablated carbachol- and ATP-stimulated MAP kinase, but not EGF-stimulated MAP kinase, suggesting that PKC is a component of the network involved in MAP kinase activation by purinergic and muscarinic agonists. Investigation of the sidedness of the hormonal stimulations indicated that EGF-stimulated MAP kinase was highly polarized, occurring exclusively from the basolateral surface, whereas carbachol stimulated MAP kinase similarly from either cell surfaces.(ABSTRACT TRUNCATED AT 250 WORDS)
9

Bevington, Michael. "The Prevalence of People With Restricted Access to Work in Man-Made Electromagnetic Environments." Journal of Environment and Health Science 5, no. 1 (January 18, 2019): 1–12. http://dx.doi.org/10.15436/2378-6841.19.2402.

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Some surveys have identified people who have restricted access to work in environments with man-made electromagnetic exposures. This study attempts to determine their prevalence, an aspect not previously investigated in its own right. It is based on analyses of the two different types of surveys of people with Idiopathic Environmental Intolerance attributed to Electromagnetic Fields (IEI-EMF), or Electromagnetic Hyper-Sensitivity (EHS), either of the general population or of people with IEI-EMF/EHS. In addition, there are different definitions of IEI-EMF/EHS, with a range of subconscious, mild, moderate or severe symptoms, potentially leading in three stages to hyper-sensitivity. The current evidence is assessed as indicating that, in addition to subconscious sensitivity, the prevalence of IEI-EMF/EHS is between about 5.0 and 30 per cent of the general population for mild cases, 1.5 and 5.0 per cent for moderate cases and < 1.5 per cent for severe cases. The prevalence of people restricted in their access to work in a man-made electromagnetic environment is estimated at 0.65 per cent of the general population, at about 18% of the general population with moderate IEI-EMF/EHS. The estimate of 0.65% equates to 435,500 people in the UK’s population of 67 million. Some reasons for possible under-reporting are discussed. Adjustments can enable some people with this disability to remain in employment, suggesting that rates of restriction in access to work may fall as employers become aware of what adjustments are needed.
10

Lee, M., C. Hwang, J. Lee, H. Slavkin, and D. Warburton. "TGF-beta isoforms differentially attenuate EGF mitogenicity and receptor activity in fetal lung mesenchymal cells." American Journal of Physiology-Lung Cellular and Molecular Physiology 273, no. 2 (August 1, 1997): L374—L381. http://dx.doi.org/10.1152/ajplung.1997.273.2.l374.

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To evaluate signaling interactions, combinations of epidermal growth factor (EGF) and transforming growth factor-beta (TGF-beta) isoforms were applied to primary fetal mouse lung mesenchymal cells isolated at 16 days of gestation. The three isoforms of TGF-beta had similar mitogenic potentials, as assessed by thymidine incorporation (half-maximal effective concentration approximately 2 ng/ml). However, combined exposure to EGF and TGF-beta yielded an isoform-dependent attenuation of EGF-induced mitogenesis. Combinations of 20 ng/ml EGF and 2 ng TGF-beta 1, TGF-beta 2, or TGF-beta 3 resulted in thymidine incorporation values 0.76, 0.74, and 0.86 times that of EGF alone, respectively; attenuation of EGF mitogenicity, interactions between EGF and TGF-beta isoforms, and differences between isoforms were all statistically significant by analysis of variance. Treatment with TGF-beta isoforms significantly reduced EGF-induced receptor angiotensin II substrate phosphorylation. TGF-beta isoform-specific signaling also significantly attenuated EGF-induced phosphorylation of the mitogen-activated protein (MAP) kinase extracellular signal-regulated kinase 2. These results suggest that isoform-specific TGF-beta signaling modulates the EGF signal transduction pathway upstream of MAP kinase.
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Статті в журналах

Дисертації з теми "EMF exposure map":

1

Mallik, Mohammed Tariqul Hassan. "Electromagnetic Field Exposure Reconstruction by Artificial Intelligence." Electronic Thesis or Diss., Université de Lille (2022-....), 2023. https://pepite-depot.univ-lille.fr/ToutIDP/EDENGSYS/2023/2023ULILN052.pdf.

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Le sujet de l'exposition aux champs électromagnétiques a fait l'objetd'une grande attention à la lumière du déploiement actuel du réseaucellulaire de cinquième génération (5G). Malgré cela, il reste difficilede reconstituer avec précision le champ électromagnétique dans unerégion donnée, faute de données suffisantes. Les mesures in situ sontd'un grand intérêt, mais leur viabilité est limitée, ce qui renddifficile la compréhension complète de la dynamique du champ. Malgré legrand intérêt des mesures localisées, il existe encore des régions nontestées qui les empêchent de fournir une carte d'exposition complète. Larecherche a exploré des stratégies de reconstruction à partird'observations provenant de certains sites localisés ou de capteursdistribués dans l'espace, en utilisant des techniques basées sur lagéostatistique et les processus gaussiens. En particulier, desinitiatives récentes se sont concentrées sur l'utilisation del'apprentissage automatique et de l'intelligence artificielle à cettefin. Pour surmonter ces problèmes, ce travail propose de nouvellesméthodologies pour reconstruire les cartes d'exposition aux CEM dans unezone urbaine spécifique en France. L'objectif principal est dereconstruire des cartes d'exposition aux ondes électromagnétiques àpartir de données provenant de capteurs répartis dans l'espace. Nousavons proposé deux méthodologies basées sur l'apprentissage automatiquepour estimer l'exposition aux ondes électromagnétiques. Pour la premièreméthode, le problème de reconstruction de l'exposition est défini commeune tâche de traduction d'image à image. Tout d'abord, les données ducapteur sont converties en une image et l'image de référencecorrespondante est générée à l'aide d'un simulateur basé sur le tracédes rayons. Nous avons proposé un réseau adversarial cGAN conditionnépar la topologie de l'environnement pour estimer les cartes d'expositionà l'aide de ces images. Le modèle est entraîné sur des images de cartesde capteurs tandis qu'un environnement est donné comme entréeconditionnelle au modèle cGAN. En outre, la cartographie du champélectromagnétique basée sur le Generative Adversarial Network estcomparée au simple Krigeage. Les résultats montrent que la méthodeproposée produit des estimations précises et constitue une solutionprometteuse pour la reconstruction des cartes d'exposition. Cependant,la production de données de référence est une tâche complexe car elleimplique la prise en compte du nombre de stations de base actives dedifférentes technologies et opérateurs, dont la configuration du réseauest inconnue, par exemple les puissances et les faisceaux utilisés parles stations de base. En outre, l'évaluation de ces cartes nécessite dutemps et de l'expertise. Pour répondre à ces questions, nous avonsdéfini le problème comme une tâche d'imputation de données manquantes.La méthode que nous proposons prend en compte l'entraînement d'un réseauneuronal infini pour estimer l'exposition aux champs électromagnétiques.Il s'agit d'une solution prometteuse pour la reconstruction des cartesd'exposition, qui ne nécessite pas de grands ensembles d'apprentissage.La méthode proposée est comparée à d'autres approches d'apprentissageautomatique basées sur les réseaux UNet et les réseaux adversairesgénératifs conditionnels, avec des résultats compétitifs
The topic of exposure to electromagnetic fields has received muchattention in light of the current deployment of the fifth generation(5G) cellular network. Despite this, accurately reconstructing theelectromagnetic field across a region remains difficult due to a lack ofsufficient data. In situ measurements are of great interest, but theirviability is limited, making it difficult to fully understand the fielddynamics. Despite the great interest in localized measurements, thereare still untested regions that prevent them from providing a completeexposure map. The research explored reconstruction strategies fromobservations from certain localized sites or sensors distributed inspace, using techniques based on geostatistics and Gaussian processes.In particular, recent initiatives have focused on the use of machinelearning and artificial intelligence for this purpose. To overcome theseproblems, this work proposes new methodologies to reconstruct EMFexposure maps in a specific urban area in France. The main objective isto reconstruct exposure maps to electromagnetic waves from some datafrom sensors distributed in space. We proposed two methodologies basedon machine learning to estimate exposure to electromagnetic waves. Forthe first method, the exposure reconstruction problem is defined as animage-to-image translation task. First, the sensor data is convertedinto an image and the corresponding reference image is generated using aray tracing-based simulator. We proposed an adversarial network cGANconditioned by the environment topology to estimate exposure maps usingthese images. The model is trained on sensor map images while anenvironment is given as conditional input to the cGAN model.Furthermore, electromagnetic field mapping based on the GenerativeAdversarial Network is compared to simple Kriging. The results show thatthe proposed method produces accurate estimates and is a promisingsolution for exposure map reconstruction. However, producing referencedata is a complex task as it involves taking into account the number ofactive base stations of different technologies and operators, whosenetwork configuration is unknown, e.g. powers and beams used by basestations. Additionally, evaluating these maps requires time andexpertise. To answer these questions, we defined the problem as amissing data imputation task. The method we propose takes into accountthe training of an infinite neural network to estimate exposure toelectromagnetic fields. This is a promising solution for exposure mapreconstruction, which does not require large training sets. The proposedmethod is compared with other machine learning approaches based on UNetnetworks and conditional generative adversarial networks withcompetitive results

Книги з теми "EMF exposure map":

1

Cohen, Jeffrey A., Justin J. Mowchun, Victoria H. Lawson, and Nathaniel M. Robbins. A 48-Year-Old with Progressive Weakness and Pain. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190491901.003.0005.

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Guillain-Barré syndrome may present in several ways, although predominant proximal weakness is a common feature of the disease to recognize. The differential diagnosis may be extensive and can include infection, vasculitis, toxin exposure, and malignancy. A lumbar puncture must be done with minimal delay to evaluate for cerebrospinal fluid (CSF) albuminocytological dissociation, however results may be normal early in the course of the disease. EMG/NCS are helpful to support the diagnosis, and early treatment with intravenous immunoglobulin (IVIG) is essential. This chapter discusses the clinical features and diagnostic considerations of this important condition.

Частини книг з теми "EMF exposure map":

1

Grandolfo, Martino, and Paolo Vecchia. "Natural and Man-Made Environmental Exposures to Static and ELF Electromagnetic Fields." In Biological Effects and Dosimetry of Static and ELF Electromagnetic Fields, 49–70. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4613-2099-9_4.

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2

Selvin, Steve. "Matched Analysis (One Case and Two Controls)." In Epidemiologic Analysis, 108–28. Oxford University PressNew York, NY, 2001. http://dx.doi.org/10.1093/oso/9780195144895.003.0008.

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Abstract To determine if exposure to electromagnetic radiation is associated with the risk of a birth defect. More specifically, does a newborn infant whose mother lives near high-voltage power lines have an increased risk of a congenital anomaly? DATA Of the infants born in the east-central region of France (1988-1991) and observed during the first year of life, 118 had major congenital anomalies. Each of these 118 infants were matched to two randomly selected controls who were of the same gender and born approximately on the same date in the same municipality. For all 354 study subjects, the distance from their mother’s residence (home/apartment during pregnancy) was measured to the nearest 225 or 400 kilovolt high-voltage power line. These data were derived entirely from birth records and detailed maps. To reflect risk, two measures of maternal/infant exposure to electromagnetic field (emf) radiation were used-the distance as measured directly in meters and the distance dichot­ omized into less than or equal to 100 meters and more than 100 meters. The motivation for the matched design was to equalize, at least partially, com-

Тези доповідей конференцій з теми "EMF exposure map":

1

Mallik, Mohammed, Sofiane Kharbech, Taghrid Mazloum, Shanshan Wang, Joe Wiart, Davy P. Gaillot, and Laurent Clavier. "EME-Net: A U-net-based Indoor EMF Exposure Map Reconstruction Method." In 2022 16th European Conference on Antennas and Propagation (EuCAP). IEEE, 2022. http://dx.doi.org/10.23919/eucap53622.2022.9769645.

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2

Sheikh, Abdul Q., Andrei Kogan, and Daria A. Narmoneva. "Electromagnetic Field Mediates Capillary-Like Network Formation via MAPK/ERK Signaling Cascade." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206710.

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Abnormal angiogenesis (formation of capillaries) plays an important role in the impaired diabetic wound healing and has emerged as a new target area for therapeutic interventions. Pulsed magnetic field therapy, which was initially used for healing of bone fractures, has been recently introduced as a potential therapy to treat diabetic and chronic wounds [1], although the mechanisms responsible for improved healing are still unclear. Electromagnetic fields (EMF) have been shown to act as a directional cues in cellular responses such as migration and activations of several signal transduction cascades [2]. Recent literature delineates an important role of GHz EMF (i.e., with an oscillation period of a fraction of a nanosecond) in inducing rapid and sustained phosphorylation of mitrogen-activated kinase and extracellular-signal-regulated kinase (MAPK/ERK) [3]. Recent studies have also implicated MAP kinase in mediating the phosphorylation of Connexin-43 (Cx43) that accompanies regulation of cell-cell communication via connexin gap junctions [4]. Importantly, both MAPK/ERK pathway and Cx43 signaling are involved in the process of angiogenesis [5,6]. Therefore, the goal of this study was to test the hypothesis that high-frequency (7.5GHz) EMFs promote angiogenesis in vitro via MAPK/ERK and/or Cx43 signaling. We used a custom-built EMF exposure setup and a self-assembling peptide nanoscaffold as a controlled angiogenic microenvironment [7] to quantify the effect of EMF on capillary formation and underlying cellular responses.
3

Gobba, Fabriziomaria. "1650 Magnetic resonance imaging (mri) workers: emf exposure, occupational risk and prevention. an update." In 32nd Triennial Congress of the International Commission on Occupational Health (ICOH), Dublin, Ireland, 29th April to 4th May 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/oemed-2018-icohabstracts.1206.

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4

Bedini, A., C. Giliberti, R. Palomba, E. D'Emilia, and L. Giuliani. "Architecture of Exposure Systems in the Range of the Microwaves for Biological Applications." In 2007 International Symposium on Microwave, Antenna, Propagation and EMC Technologies for Wireless Communications. IEEE, 2007. http://dx.doi.org/10.1109/mape.2007.4393503.

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Rumeng, Tan, Wu Tong, Kun Zhu, Chen Shaochuan, and Luo Senwen. "Method of data acquisition and map generation for radio frequency electromagnetic fields exposure level." In 2018 IEEE International Symposium on Electromagnetic Compatibility and 2018 IEEE Asia-Pacific Symposium on Electromagnetic Compatibility (EMC/APEMC). IEEE, 2018. http://dx.doi.org/10.1109/isemc.2018.8393860.

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Niu, S. "1649a Ilo activities for the prevention of the risk related to occupational exposure to emf in workers." In 32nd Triennial Congress of the International Commission on Occupational Health (ICOH), Dublin, Ireland, 29th April to 4th May 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/oemed-2018-icohabstracts.1202.

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Filosto, Simone, Cathleen Becker, and Tzipora Goldkorn. "A Novel Post-Translational Mechanism Underlies The EGF Receptor Resistance To Tyrosine Kinase Inhibition Upon CS Exposure Of Airway Epithelial Cells." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6294.

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