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Статті в журналах з теми "EDN"

Автор: Grafiati
Опубліковано: 21 вересня 2024

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1

Apata, Ibironke W., John Hanfelt, James L. Bailey, and Vandana Dua Niyyar. "Chlorhexidine-impregnated transparent dressings decrease catheter-related infections in hemodialysis patients: a quality improvement project." Journal of Vascular Access 18, no. 2 (March 2017): 103–8. http://dx.doi.org/10.5301/jva.5000658.

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Purpose Central venous catheters (CVC) are associated with increased infection rates, morbidity and mortality compared to other hemodialysis vascular access. Chlorhexidine-impregnated transparent (CHG-transparent) dressings allow for continuous antimicrobial exposure and easy visibility of the CVC insertion site. We conducted a quality improvement project to compare catheter-related infection (CRI) rates in two dressing regimens – CHG-transparent dressings and adhesive dry gauze dressing in hemodialysis patients with tunneled CVCs. Methods The study was conducted in two phases. In phase 1, CHG-transparent dressing was introduced to EDC hemodialysis unit, while EDG and EDN hemodialysis units, served as the control sites and maintained adhesive dry gauze dressing. Phase 2 of the study involved replacing the adhesive dry gauze dressing with CHG-transparent dressing at EDG and EDN and maintaining CHG-transparent dressing at EDC. CRI rates at each hemodialysis unit during the 12-month intervention were compared to CRI rates for the 12-month pre-intervention period for each study phase. CRI rates were also compared between all three hemodialysis units. Results In phase 1, CRI rates (per 1000 days) in EDC (intervention site) decreased by 52% (1.69 vs. 0.82, p<0.05) and increased by 12% (1.80 vs. 2.02, p = 0.75) at EDG, and 35% (0.91 vs. 1.23, p = 0.40) at EDN. In phase 2, CRI rates at EDG and EDN (intervention sites) decreased by 86% (1.86 vs. 0.26 p<0.05), and 53% (1.89 vs. 0.88, p<0.05), respectively, and decreased by 20% at EDC (0.73 vs. 0.58, p = 0.65). Conclusions Replacing adhesive dry gauze dressing with CHG-transparent dressing for hemodialysis patients with tunneled CVC was associated with decreased CRI rates.
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2

Heris Anita, Sita, Fenny Clara Ardiati, Kharisma Panji Ramadhan, Raden Permana Budi Laksana, Fahriya Puspita Sari, Oktan Dwi Nurhayat, and Dede Heri Yuli Yanto. "Decolorization of Synthetic Dyes by Tropical Fungi Isolated from Taman Eden 100, Toba Samosir, North Sumatra, Indonesia." HAYATI Journal of Biosciences 29, no. 4 (March 29, 2022): 417–27. http://dx.doi.org/10.4308/hjb.29.4.417-427.

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Taman Eden 100 is one of the tourist parks located in Toba Samosir, North Sumatra, Indonesia that provides a wide range of biodiversity. The objective of this research was to obtain the potential fungal isolate with high laccase activity to decolorize synthetic dyes. The results show that six isolates of Perenniporia subtephropora EDN 050, Trametes hirsuta EDN 082, T. hirsuta EDN 084, T. hirsuta EDN 085, Deconica coprophila EDN 114, and T. pavonia EDN 134, which were confirmed by molecular identification using 5.8 rDNA/ITS analysis, exhibited decolorization activity. Six potential isolates showed the ability to decolorize textile dyes of Acid Blue 129, RBBR,Orange II, and Reactive Black 5. The highest decolorization rate of 100 ppm Acid Blue 129, RBBR, Orange II, and Reactive Black 5 dyes by using T. pavonia EDN 134 were 98.87%, 98.26%, 100%, and 98.11%, respectively after 96 h incubation. Almost all positive isolates also decolorized dyes at higher concentrations (1,000 ppm). This study offers the potential of Indonesian tropical fungal isolates for synthetic dyes waste treatment.
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3

Bourke, J. F. "EDF White Book: Skin Diseases in Europe, 2nd Edn." British Journal of Dermatology 154, no. 4 (February 20, 2006): 804–5. http://dx.doi.org/10.1111/j.1365-2133.2006.07172.x.

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4

Rahmadi, Mahardian, Zamrotul Izzah, Ahmad Dzulfikri Nurhan, and Suharjono Suharjono. "Chronic intake of energy drinks affects changes in kidney function biomarkers in a diabetes mellitus animal model." Pharmacy Education 24, no. 3 (May 1, 2024): 25–31. http://dx.doi.org/10.46542/pe.2024.243.2531.

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Background: Energy drinks are a food supplement consisting of multivitamins, macronutrients, taurine, and caffeine. Their excessive consumption is suspected to be a risk factor for chronic kidney failure. Objective: This study aimed to determine the effect of energy drinks on kidney function in rat models with diabetes mellitus (DM). Method: Thirty-two experimental male Wistar rats were injected with alloxan 150 mg/kg intraperitoneally to induce DM and then randomly divided into four groups. The positive control group was treated with drinking water. ED1, ED2, and ED3 groups were respectively treated with energy drinks coded EDK, EDH, and EDE, with a dose equivalent to caffeine 25 mg/kg twice a day for 15 days. All energy drinks used are distributed in the Asian region, especially in Southeast Asia. Urinalysis, haematology, and kidney histopathology evaluations were carried out. Result: Energy drinks affect BUN, serum creatinine, sodium, and potassium. In the histopathological observation of kidney tissue, there was a significant difference in the value of damage between the control group and the ED3 group. Conclusion: Energy drinks significantly increase the risk of impaired kidney function in diabetes mellitus rat models.
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5

Shih, Ming-Kuei, You-Lin Tain, Chiu-Min Cheng, Chien-Ning Hsu, Yu-Wei Chen, Hung-Tse Huang, Chi-I. Chang, and Chih-Yao Hou. "Separation and Identification of Resveratrol Butyrate Ester Complexes and Their Bioactivity in HepG2 Cell Models." International Journal of Molecular Sciences 22, no. 24 (December 17, 2021): 13539. http://dx.doi.org/10.3390/ijms222413539.

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Resveratrol butyrate ester (RBE) complexes have demonstrated higher antioxidant capacity and anti-fat accumulation activity in previous studies. In this study, silica gel, high-performance liquid chromatography, and 1H nuclear magnetic resonance were used for separation and identification of RBE complex components. With the exception of resveratrol, five different structures of ester derivatives were separated from silica gel: 3,4′-di-O-butanoylresveratrol (ED2, 18.8%), 3-O-butanoylresveratrol (ED4, 35.7%), 4′-O-butanoylresveratrol (ED5, 4.4%), 3,5,4′-tri-O-butanoylresveratrol (ED6, 1.5%), and 3,5-di-O-butanoylresveratrol (ED7, 0.7%). Among the ester derivatives obtained, ED2 and ED4 were the main ester derivatives in the RBE complex. Thus, the cellular antioxidant activities of the RBE mixture, ED2, and ED4 were evaluated. Results showed that the antioxidant capacity of ED2 and ED4 was higher than that of the RBE mixture, demonstrating that the number and position of butyrate esterification sites are related to cell survival rate and antioxidant capacity. This study is the first to report the successful isolation, structural identification, and cellular biological antioxidant activity of RBE complex derivatives, which are key characteristics for the potential practical application of RBE complexes.
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6

McLean, Graham. "Pharmacology, 5th edn." British Journal of Clinical Pharmacology 57, no. 1 (January 2004): 112. http://dx.doi.org/10.1111/j.1365-2125.2004.01916.x.

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7

Ramelet, Albert-Adrien, and Howard Maibach. "Sclerotherapy, 3rd edn." Journal of Cosmetic Dermatology 1, no. 4 (December 2002): 222. http://dx.doi.org/10.1111/j.1473-2165.2002.00004.x.

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8

Jellinger, K. A. "Neuroglia, 2nd edn." European Journal of Neurology 18, no. 12 (July 28, 2011): e154-e154. http://dx.doi.org/10.1111/j.1468-1331.2011.03492.x.

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9

Wood, Debra A. "Psychiatry, 2nd Edn." Australian & New Zealand Journal of Psychiatry 39, no. 9 (September 2005): 844. http://dx.doi.org/10.1080/j.1440-1614.2005.01692_1.x.

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10

Francis, Dr R. "‘Corrosion’, 3rd edn;." British Corrosion Journal 30, no. 3 (January 1995): 189–90. http://dx.doi.org/10.1179/bcj.1995.30.3.189.

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11

van Hunen, Jeroen. "Geodynamics, 3rd edn." Geophysical Journal International 200, no. 2 (January 6, 2015): 1236. http://dx.doi.org/10.1093/gji/ggu465.

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12

Mayhew, Peter J. "Evolution (2nd edn)." Heredity 82, no. 4 (April 1999): 471. http://dx.doi.org/10.1038/sj.hdy.6885362.

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13

du Boulay, C. "Pathology, 3rd edn." Histopathology 44, no. 1 (January 2004): 81. http://dx.doi.org/10.1111/j.1365-2559.2004.01764.x.

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14

Robinson, Gillian. "Gynaecology (4th edn)." Journal of Family Planning and Reproductive Health Care 38, no. 2 (March 27, 2012): 101. http://dx.doi.org/10.1136/jfprhc-2011-100272.

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15

Beattie, Elizabeth. "Dementia (4th edn)." Australasian Journal on Ageing 31, no. 1 (March 2012): 64–65. http://dx.doi.org/10.1111/j.1741-6612.2011.00594.x.

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16

Direkze, S. "Oncology (2nd edn)." British Journal of Cancer 96, no. 1 (January 2007): 182. http://dx.doi.org/10.1038/sj.bjc.6603485.

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17

Martin, Claude R. "Retailing (2nd edn)." Journal of Retailing and Consumer Services 3, no. 1 (January 1996): 58–59. http://dx.doi.org/10.1016/s0969-6989(99)80010-1.

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18

Campbell, Peter N. "Biochemistry (2nd edn)." Trends in Biochemical Sciences 20, no. 8 (August 1995): 330. http://dx.doi.org/10.1016/s0968-0004(00)89062-3.

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19

Latchman, David S. "Biochemistry (4th edn)." Trends in Biochemical Sciences 20, no. 11 (November 1995): 488. http://dx.doi.org/10.1016/s0968-0004(00)89112-4.

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20

Brock, Jeremy H. "Immunology (8th edn)." Immunology Today 19, no. 7 (July 1998): 335–36. http://dx.doi.org/10.1016/s0167-5699(98)01274-2.

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21

Harriman, Denis. "Muskelerkrankungen, 2nd edn." Neuromuscular Disorders 2, no. 2 (January 1992): 147. http://dx.doi.org/10.1016/0960-8966(92)90049-c.

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22

Meyer, Peter. "Biotechnology (3rd edn)." Trends in Genetics 12, no. 8 (August 1996): 326–27. http://dx.doi.org/10.1016/0168-9525(96)81469-7.

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23

Oxford, Geoff. "Evolution (2nd edn)." Trends in Ecology & Evolution 11, no. 7 (July 1996): 307–8. http://dx.doi.org/10.1016/0169-5347(96)81129-3.

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24

Elliott, Keith R. F. "Biochemistry, 3rd edn." Trends in Biochemical Sciences 15, no. 1 (January 1990): 35–36. http://dx.doi.org/10.1016/0968-0004(90)90132-u.

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25

Janković, Branislav D. "Neuroimmunoendocrinology (2nd edn)." Immunology Today 13, no. 7 (January 1992): 281–82. http://dx.doi.org/10.1016/0167-5699(92)90015-y.

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26

Guy, Keith. "Immunology (3rd edn)." Immunology Today 15, no. 8 (August 1994): 390–91. http://dx.doi.org/10.1016/0167-5699(94)90182-1.

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27

Gee, John H. R. "Ecology (3rd edn)." Trends in Ecology & Evolution 6, no. 1 (January 1991): 34–35. http://dx.doi.org/10.1016/0169-5347(91)90152-n.

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28

Townshend, A. "Atomabsorptionsspektrometrie, 3rd edn." Analytica Chimica Acta 183 (1986): 324–25. http://dx.doi.org/10.1016/0003-2670(86)80111-8.

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29

Lockwood, D. N. J. "Leprosy. 2nd Edn." Journal of Hospital Infection 30, no. 2 (June 1995): 165–66. http://dx.doi.org/10.1016/0195-6701(95)90161-2.

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30

Julian, Margaret. "Africa review 1987, 11th edn; Europe review 1987, 2nd edn; Latin America & Caribbean review 1987, 8th edn; Middle East review 1987, 13th edn." International Affairs 63, no. 3 (1987): 545. http://dx.doi.org/10.2307/2619352.

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31

Slifman, N. R., D. A. Loegering, D. J. McKean, and G. J. Gleich. "Ribonuclease activity associated with human eosinophil-derived neurotoxin and eosinophil cationic protein." Journal of Immunology 137, no. 9 (November 1, 1986): 2913–17. http://dx.doi.org/10.4049/jimmunol.137.9.2913.

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Abstract The eosinophil granule contains a series of basic proteins, including major basic protein, eosinophil peroxidase, eosinophil-derived neurotoxin (EDN), and eosinophil cationic protein (ECP). Both EDN and ECP are neurotoxins and helminthotoxins. Comparison of the partial N-terminal amino acid sequences of EDN and ECP showed 67% identity; surprisingly, they also showed structural homology to pancreatic ribonuclease (RNase). Therefore, we determined whether EDN and ECP possess RNase enzymatic activity. By spectrophotometric assay of acid soluble nucleotides formed from yeast RNA, purified EDN showed RNase activity similar to bovine pancreatic RNase, whereas ECP was 50 to 100 times less active. The RNase activity associated with ECP was not significantly inhibited after exposure of ECP to polyclonal or monoclonal antibody to EDN. These results indicate that EDN and ECP both possess RNase activity, the RNase activity of EDN and ECP is specific, and EDN and ECP have maintained not only structural but also functional homology to pancreatic RNase.
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32

Yang, De, Qian Chen, Shao Bo Su, Ping Zhang, Kahori Kurosaka, Rachel R. Caspi, Suzanne M. Michalek, Helene F. Rosenberg, Ning Zhang, and Joost J. Oppenheim. "Eosinophil-derived neurotoxin acts as an alarmin to activate the TLR2–MyD88 signal pathway in dendritic cells and enhances Th2 immune responses." Journal of Experimental Medicine 205, no. 1 (January 14, 2008): 79–90. http://dx.doi.org/10.1084/jem.20062027.

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Eosinophil-derived neurotoxin (EDN) is an eosinophil granule–derived secretory protein with ribonuclease and antiviral activity. We have previously shown that EDN can induce the migration and maturation of dendritic cells (DCs). Here, we report that EDN can activate myeloid DCs by triggering the Toll-like receptor (TLR)2–myeloid differentiation factor 88 signaling pathway, thus establishing EDN as an endogenous ligand of TLR2. EDN activates TLR2 independently of TLR1 or TLR6. When mice were immunized with ovalbumin (OVA) together with EDN or with EDN-treated OVA-loaded DCs, EDN enhanced OVA-specific T helper (Th)2-biased immune responses as indicated by predominant production of OVA-specific interleukin (IL)-5, IL-6, IL-10, and IL-13, as well as higher levels of immunoglobulin (Ig)G1 than IgG2a. Based on its ability to serve as a chemoattractant and activator of DCs, as well as the capacity to enhance antigen-specific immune responses, we consider EDN to have the properties of an endogenous alarmin that alerts the adaptive immune system for preferential enhancement of antigen-specific Th2 immune responses.
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33

Kuai, Shougang, and Peipei Zhao. "Association of EDN levels in patients with asthma and correlation with FEV1%: A meta-analysis." Allergy and Asthma Proceedings 44, no. 4 (July 1, 2023): 244–51. http://dx.doi.org/10.2500/aap.2023.44.230016.

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Background: Eosinophil-derived neurotoxin (EDN), an eosinophil degranulation product, is a good biomarker for eosinophilic inflammation of the airway. Several articles have shown that EDN levels are higher in patients with asthma than in controls, and EDN levels are correlated with the percentage of predicted forced expiratory volume in the first second (FEV1%) in patients with asthma. Their results were inconclusive. Methods: A comprehensive meta-analysis was performed to assess EDN levels between patients with asthma and controls, and the correlations between EDN and FEV1% in the patients with asthma. Fourteen relevant articles were identified from electronic data bases. Pooled standardized mean difference (SMD) with a 95% confidence interval (CI) for the difference of EDN levels between the patients with asthma and controls, and pooled coefficient (r) values with 95% CI for the correlations between EDN and FEV1%, respectively, were calculated. Results: A total of 14 articles were selected. Among the included reports, six articles related to the difference and eight essays on the correlation. Pooled effect size showed that EDN levels were higher in patients with asthma than in controls (SMD 2.85 [95% CI, 1.92‐3.78]). Furthermore, the pooled effect size showed that EDN levels were negatively correlated with FEV1% in patients with asthma (r ‐0.21 [95% CI, ‐0.28 to ‐0.14]). Conclusion: EDN levels increased in the patients with asthma compared with in the controls. They were correlated with FEV1% in the patients with asthma, which indicated that EDN could be a reliable marker to monitor asthma's therapeutic effects.
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34

Watkinson, A. R., and J. H. Zar. "Biostatistical Analysis, 3rd edn." Journal of Applied Ecology 33, no. 5 (October 1996): 1230. http://dx.doi.org/10.2307/2404706.

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35

Hill, Mark, and M. J. Crawley. "Plant Ecology, 2nd edn." Journal of Ecology 85, no. 4 (August 1997): 548. http://dx.doi.org/10.2307/2960578.

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36

Robinson, Clare H., S. E. Smith, and D. J. Read. "Mycorrhizal Symbiosis, 2nd edn." Journal of Ecology 85, no. 6 (December 1997): 925. http://dx.doi.org/10.2307/2960617.

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37

Alexandrou, Alex. "School leadership (2nd edn)." Professional Development in Education 35, no. 3 (September 2009): 495–96. http://dx.doi.org/10.1080/19415250902843473.

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38

Kumar, Kuldeep. "Robust Statistics, 2nd edn." Journal of the Royal Statistical Society: Series A (Statistics in Society) 174, no. 1 (January 2011): 241–42. http://dx.doi.org/10.1111/j.1467-985x.2010.00676_4.x.

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39

O'Loughlin, E. V. "Adolescent medicine. 2nd edn." Medical Journal of Australia 152, no. 2 (January 1990): 104. http://dx.doi.org/10.5694/j.1326-5377.1990.tb124484.x.

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40

Steiger, M. J. "Clinical Neurology, 2nd edn." Journal of the Royal Society of Medicine 91, no. 12 (December 1998): 656–57. http://dx.doi.org/10.1177/014107689809101221.

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41

Sheldon, Eric. "Neutrino Physics, 2nd edn." Contemporary Physics 53, no. 1 (January 2012): 53–54. http://dx.doi.org/10.1080/00107514.2011.633711.

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42

van der Vennet, Renee. "Art Psychotherapy, second edn." Arts & Health 4, no. 2 (June 2012): 185–86. http://dx.doi.org/10.1080/17533015.2011.598539.

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43

Cullen, Breda. "Clinical neuropsychology (5th Edn.)." Neuropsychological Rehabilitation 22, no. 6 (December 2012): 944–47. http://dx.doi.org/10.1080/09602011.2012.701865.

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44

Khan, K. "Football medicine, revised edn." British Journal of Sports Medicine 40, no. 10 (October 1, 2006): 881. http://dx.doi.org/10.1136/bjsm.2005.025478.

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45

Husain, Viqar. "Quantum Gravity (2nd edn)." Classical and Quantum Gravity 25, no. 12 (June 3, 2008): 129002. http://dx.doi.org/10.1088/0264-9381/25/12/129002.

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46

Kenyon, I. R. "Classical Mechanics (3rd edn)." Physics Bulletin 36, no. 9 (September 1985): 390. http://dx.doi.org/10.1088/0031-9112/36/9/025.

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47

Grace, M. A. "Elementary Particles (2nd edn)." Physics Bulletin 37, no. 5 (May 1986): 226. http://dx.doi.org/10.1088/0031-9112/37/5/035.

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48

Bhathena, R. K. "Contraception Today, 6th edn." Journal of Obstetrics and Gynaecology 28, no. 1 (January 2008): 143. http://dx.doi.org/10.1080/01443610701840935.

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49

&NA;. "Clinical anesthesia, 7th edn." European Journal of Anaesthesiology 31, no. 1 (January 2014): 63–64. http://dx.doi.org/10.1097/eja.0000000000000022.

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50

Jellinger, K. A. "Merritt's Neurology, 11th edn." European Journal of Neurology 13, no. 7 (July 2006): e15-e16. http://dx.doi.org/10.1111/j.1468-1331.2006.01275.x.

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