Дисертації з теми "Disorder system"
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Makvandian, Negar. "Seasonal Affective Disorder Monitoring System." Thesis, Uppsala universitet, Institutionen för informationsteknologi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-199586.
Повний текст джерелаQuansah, Amissah Richard. "The reward system and binge eating disorder." Doctoral thesis, Université Laval, 2020. http://hdl.handle.net/20.500.11794/66331.
Повний текст джерелаHamiter, Amelia. ""It's Not a Real Disorder": Attention Deficit/Hyperactivity Disorder and Paradigms of Childhood Harm." Scholarship @ Claremont, 2016. http://scholarship.claremont.edu/scripps_theses/849.
Повний текст джерелаSakaida, Masaru. "Disorder-induced quantum phenomena in inhomogeneous optical lattices." 京都大学 (Kyoto University), 2016. http://hdl.handle.net/2433/215289.
Повний текст джерелаMeade, Oonagh. "Members' experiences of a neuromuscular disorder online support group." Thesis, University of Nottingham, 2013. http://eprints.nottingham.ac.uk/14158/.
Повний текст джерелаJeans, Aruna. "The peripheral immune system of glycosphingolipid storage disorder mouse models." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.432342.
Повний текст джерелаCounts, Margaret S. E. "A profile of Georgia caregivers to Alzheimer's disease & related disorder victims." DigitalCommons@Robert W. Woodruff Library, Atlanta University Center, 1986. http://digitalcommons.auctr.edu/dissertations/749.
Повний текст джерелаMoakes, Hannah. "Dominance motivation, goal pursuit and mania in bipolar disorder." Thesis, University of Exeter, 2016. http://hdl.handle.net/10871/23445.
Повний текст джерелаWestlund, Kristina. "The Mirror Neuron System and Its Role in Autism Spectrum Disorder." Thesis, University of Skövde, School of Humanities and Informatics, 2009. http://urn.kb.se/resolve?urn=urn:nbn:se:his:diva-3128.
Повний текст джерелаThe discovery of mirror neurons and the mirror neuron system is one of the most interesting breakthroughs in the field of neuroscience in recent years. The topic stretches over a wide spectrum of research fields but one of the more prominent areas is concerned with the role of mirror neurons in autism spectrum disorder. It is hypothesized that an impaired mirror neuron system may be one of the main causes underlying the deficits seen in autistic individuals. Parallel to the broken mirror theory of autism there are critical voices claiming there is not enough empirical evidence to support such a theory. Research carried out in the area seems to offer support for both contradictory approaches making it hard to conclude the definite role of mirror neurons in this developmental disorder. Future research may offer conclusive answers concerning the role of the mirror neuron system in autism spectrum disorder as well as other important questions regarding the functional properties of the brain areas under question.
Minton, Gareth Owain. "Corticosteroid modulation of the mesocorticolimbic dopamine system : implications for bipolar disorder." Thesis, University of Newcastle Upon Tyne, 2008. http://hdl.handle.net/10443/745.
Повний текст джерелаFarber, Robert H. "Ocular motor system functioning in obsessive-compulsive disorder and Tourette syndrome /." Diss., Connect to a 24 p. preview or request complete full text in PDF format. Access restricted to UC IP addresses, 1998. http://wwwlib.umi.com/cr/ucsd/fullcit?p9917950.
Повний текст джерелаChou, I.-Jun. "Comparative epidemiology and quantitative neuroimaging of neuromyelitis optica spectrum disorder and multiple sclerosis." Thesis, University of Nottingham, 2017. http://eprints.nottingham.ac.uk/47717/.
Повний текст джерелаFleming, Hunter. "Approach to a Performance-Related Musculoskeletal Disorder in a Drummer." Digital Commons @ East Tennessee State University, 2020. https://dc.etsu.edu/asrf/2020/presentations/23.
Повний текст джерелаZaboli, Ghazal. "Genes of the serotonergic system & susceptibility to psychiatric disorders : a gene-based haplotype analysis approach /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-729-4/.
Повний текст джерелаSpiller, Sydney. "SPEECH-LANGUAGE PATHOLOGISTS’ PERSPECTIVE ON LANGUAGE DISORDER IN THE JUVENILE JUSTICE SYSTEM." OpenSIUC, 2020. https://opensiuc.lib.siu.edu/theses/2700.
Повний текст джерелаMaston, Kaitlyn L. "The Effects of In Situ Behavioral Skills Training on Parent Implementation of the PEAK Relational Training System." OpenSIUC, 2018. https://opensiuc.lib.siu.edu/theses/2294.
Повний текст джерелаQuist, Jennifer F. "Molecular genetic study of serotonin system genes in attention deficit hyperactivity disorder (ADHD)." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0017/MQ54146.pdf.
Повний текст джерелаGil, Tony Jr. "Development of an Expert System Prototype od Dyslexia and Attention Deficit Hyperactivity Disorder." NSUWorks, 1996. http://nsuworks.nova.edu/gscis_etd/538.
Повний текст джерелаHang, Zhihong. "Experimental investigation on the effect of disorder in metallo-photonic band gap system /." View abstract or full-text, 2004. http://library.ust.hk/cgi/db/thesis.pl?PHYS%202004%20HANG.
Повний текст джерелаCline, Jessie Irit. "Autonomic Nervous System Functioning and Callous-Unemotional Traits in Childhood-Onset Conduct Disorder." Diss., Temple University Libraries, 2013. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/216568.
Повний текст джерелаPh.D.
Although the current literature demonstrates relations between autonomic nervous system (ANS) functioning and conduct disorder (CD), there are inconsistencies across studies in the magnitude and direction of these associations, some of which may stem from heterogeneity within the CD diagnostic category. Considering callous-unemotional (CU) traits in research examining ANS functioning and CD relations could help to clarify these inconsistencies, given that CU traits identify a subgroup of youth with CD who exhibit a more severe and persistent course, as well as more negative correlates and sequelae than youth with CD without CU traits. However, there is a dearth of literature considering ANS processes among youth with CD with and without CU traits. Examining these relations, particularly during middle childhood when these processes may be amenable to intervention, has important implications for etiological, prevention, and intervention models. The present study examined relations among CD, CU, and ANS functioning among a sample of ethnic minority, urban children (N= 99, M= 9.87± 1.19 years old; 48.5% male; 94.9% African-American, 3% Latino/a). Specifically, I examined whether CU traits moderated the relations between CD and (a) parasympathetic nervous system (PNS) functioning and (b) sympathetic nervous system (SNS) functioning. In addition, I examined whether parenting behaviors (i.e., harsh parental discipline and parental warmth/involvement) influenced the relations between (a) CD and ANS functioning, and (b) CU and ANS functioning. Findings demonstrated that PNS functioning differed among children with high and low levels of CD symptoms depending on levels of CU traits. Within the current sample, among children with higher levels of CD symptoms, those with (a) higher CU symptom severity exhibited lower baseline respiratory sinus arrhythmia (RSA) and lower RSA reactivity (PNS withdrawal), compared to those with (b) lower CU symptom severity who demonstrated higher baseline RSA and higher RSA reactivity (PNS activation). Among children with lower CD symptom severity, those with (a) higher CU symptom severity exhibited higher baseline RSA and higher RSA reactivity, compared to those with (b) lower CU symptom severity who evidenced lower baseline RSA and lower RSA reactivity. Neither harsh parental discipline nor parental warmth/involvement moderated the relations between (a) CD and ANS functioning and (b) CU and ANS functioning. However, there were marginally significant associations between baseline RSA and (a) harsh parental discipline and (b) parental warmth/involvement, as well as between RSA reactivity and parental warmth/involvement in analyses examining CD, parenting, and ANS functioning. Furthermore, parental warmth/involvement tended to be associated with RSA reactivity in the analyses examining CU, parenting, and ANS functioning. Results have implications for facilitating the identification of children at risk for developing more pernicious subtypes of behavior problems, and contribute important information for the development of more individualized and potentially effective interventions for youth behavior problems, particularly among high-risk youth.
Temple University--Theses
Henje, Blom Eva. "Anxiety and depression in adolescent females autonomic regulation and differentiation /." Stockholm, 2010. http://diss.kib.ki.se/2010/978-91-7409-807-5/.
Повний текст джерелаBressler, Markus Michael. "A Case Study of Forensic Interviewing of Antisocial Personality Disorder Diagnosed Inmates." ScholarWorks, 2019. https://scholarworks.waldenu.edu/dissertations/7533.
Повний текст джерелаKeary, Therese A. "Autonomic Nervous System Functioning in Posttraumatic Stress Disorder at Rest and During Stress: The Role of the Parasympathetic Nervous System." [Kent, Ohio] : Kent State University, 2008. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=kent1219447142.
Повний текст джерелаTitle from PDF t.p. (viewed Oct. 15, 2009). Advisor: Joel Hughes. Keywords: PTSD, autonomic nervous system, parasympathetic nervous system, heart rate variability. Includes bibliographical references (p. 45-57).
Dratcu, Luiz. "An inquiry into the role of the serotonergic system of neurotransmission in panic disorder." Thesis, King's College London (University of London), 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.309168.
Повний текст джерелаHalwani, Gus F. (Gus Fizt). "Structural and functional adaptations of the Auditory-Motor System : insights from expertise & disorder." Thesis, Massachusetts Institute of Technology, 2012. http://hdl.handle.net/1721.1/78150.
Повний текст джерелаCataloged from PDF version of thesis. Vita.
Includes bibliographical references (p. 78-85).
While evidence from clinical and functional neuroimaging domains converges on a notion that auditory-motor networks can be remodeled functionally and structurally in response to experiences, studies that seek to evaluate these hypotheses by combining behavioral, functional, and structural measures are rare. Given relatively recent advances in neuroimaging, e.g. diffusion-tensor imaging (DTI) and functional neuroimaging methods (fMRI), it is now possible to structurally and functionally analyze these networks, as well as make inferences about them in situations where the networks are either functionally compromised by an auditory-motor feedback disorder, or structurally enhanced by an intense long-term auditory-motor training regimen. To this end, a three-fold course of study has been undertaken: (1) a between-group comparison of the structural aspects of the arcuate fasciculus (a prominent white-matter fiber tract that reciprocally connects the temporal and inferior frontal lobes and is thought to be important for auditory-motor interactions) of singers and those of matched nonsinging musicians, in order to evaluate the hypothesis that singers will exhibit structural differences specifically for aspects of vocal output that require rapid temporal processing and precise sound-motor matching. (2) a within-subject fMRI comparison of responses of young adults (non-musicians) to auditory feedback that is either unperturbed or shifted in pitch while they perform a pitch-matching task, to ascertain a functional network related to perceiving and perhaps compensating for mismatched auditory feedback. (3) a within-subject pilot study of the network ascertained in (2), now in a smaller group of young adults with an auditory-motor disorder/disconnection syndrome commonly referred to as tonedeafness (TD) or congential amusia (a conditioned marked by a high pitch discrimination threshold as well as readily apparent difficulty in matching pitches), in order to provide insight into how this network might behave in a state of long-term disorder. While this work corroborates previous work in clinical, behavioral, and neuroimaging domains, and sheds light on the organization of these auditory-motor networks (structurally and functionally) in the normal population, it also aids in understanding how these networks may be remodeled and optimized (structurally) in response to intense long-term training, how they adapt to an acutely compromised state (i.e. when input to the network is compromised or perturbed), as well as how they may adapt functionally in a chronically compromised state (i.e. tonedeafness). Taken together, these observations help to explain the functioning of the auditory-motor network in normal individuals and those with communication disorders, as well as well as shedding light on possible mechanisms of recovery as they participate in an intensive long-term auditory-motor therapy program.
by Gus F. Halwani.
Ph.D.in Speech and Hearing Bioscience and Technology
Carey, Paul D. (Paul Dermot). "Obsessive-compulsive disorder : serotonergic and dopaminergic system involvement in symptom generation and treatment response." Thesis, Stellenbosch : Stellenbosch University, 2008. http://hdl.handle.net/10019.1/21602.
Повний текст джерелаENGLISH ABSTRACT: Investigations into the neurobiology of obsessive-compulsive disorder (OCD) have provided useful insights into this prevalent and disabling disorder in recent decades. Encouraging advances have also been made in the pharmacological treatment of OCD. This has improved the quality of life for many who typically endure chronic unremitting symptoms. Despite the widespread use of first-line agents selective for the serotonergic system in OCD, relatively little is known about the neurobiology of treatment response, the specific components of the serotonin system involved in symptom modulation, and the overlapping and distinct brain regions impacted by alternative treatment options. Despite the advance that selective serotonin re-uptake inhibitors have been, a significant proportion of patients still fail to respond adequately to these agents, and alternative pharmacological interventions are required. The use of dopamine antagonists, a strategy which until recently has had only limited supporting data, presents one such alternative. Little however, is known about which subsets of patients are most likely to respond to these agents. In this thesis, I will present a series of six studies that use pharmacological treatments and single photon emission computed tomography (SPECT) to make contributions to three primary areas in OCD namely; neurobiology, treatment and the intersection of the two. First, I address OCD neurobiology by examining the impact of OCD on resting brain function. I then examine the effects of pharmacological challenge of the serotonin 1B receptor using sumatriptan on regional cerebral blood flow (rCBF) and clinical symptomatology. Second, I examine the intersection of neurobiology and treatment as I explore the changes in rCBF in response to treatment with inositol, a precursor of the phosphoinositol second messenger system. I then examine the distinct and overlapping effects on rCBF of treatment for 12 weeks with the selective serotonin re-uptake inhibitor (SSRI) citalopram across anxiety disorders. Third, I address treatment of OCD by examining the efficacy of controlled augmentation of serotonin re-uptake inhibitors with quetiapine, a dopamine antagonist, in treatment refractory OCD. I then combine this data with a second similar dataset to derive a predictive model for treatment outcome with quetiapine augmentation of SRIs. I demonstrate that rCBF in OCD differs significantly from normal controls, is correlated with severity in frontal brain regions, and remains an important line of investigation for OCD pathophysiology that has yet to fully delineated. Pharmacological challenge of the 5HT1B autoreceptor with the selective agonist sumatriptan results in heterogeneous behavioural and regional brain perfusion changes in OCD. Attenuation of pre-frontal perfusion following 5HT1B agonist administration is in line with the effects of SRIs. This work suggests that direct or indirect effects of SRIs on the 5HT1B receptor may be involved in mediating a clinical response in OCD. In the section exploring the intersection of neurobiology and treatment, I show that changes in rCBF partially parallel treatment response to SSRIs across a range of anxiety disorders. These data suggest that a degree of overlap exists in the neurobiology of treatment response or indeed core neurobiology across different anxiety disorders. I then show that effective treatment with inositol in OCD results in rCBF changes that are partially in line with the effects of SRIs on brain perfusion. These data support suggestions that second messengers may form part of the common pathway of action for effective anti-obsessional compounds. In the study in which we augmented SRIs with quetiapine, no advantage over placebo was found. This data has, however, recently been combined with similar data in meta-analyses and demonstrated a benefit over placebo. Finally, we found that patients who have failed fewer SRI trials, have more severe illness, and clinical dimensions with a putative dopaminergic underpinning, may derive preferential benefit from serotonin/dopamine antagonist augmentation of SRIs. Through this series of clinical treatment and functional brain imaging studies in OCD, I have contributed to the neurobiological understanding of OCD, and its treatment in refractory populations. In addition I have explored the intersection of these two domains using novel as well as conventional treatment across other anxiety disorders. Treatment and pharmacological challenges used, either directly or indirectly impacted the monoamine systems serotonin and dopamine and advanced our understanding of their involvement in symptom generation. Future work should focus on the functional intersection of brain function, treatment response, and functional genetic polymorphisms within the monoamine systems of the brain.
AFRIKAANSE OPSOMMING: Ondersoek na die neurobiologie van obsessief-kompulsiewe steuring (OKS) het in die afgelope dekades sinvolle bydraes gelewer tot die begrip van hierdie algemene en verminkende steuring. Bemoedigende vordering is ook in die farmakologiese behandeling van OKS gemaak. Dit het tot ’n verbetering in kwalitiet van lewe van meeste pasiënte gelei wat normaalweg kronies en onophoudelike simptome moet verduur. Ten spyte van die uiteenlopende gebruik van eerste-linie behandeling wat spesifiek inwerk op die serotonien sisteem in OKS, is relatief min bekend oor die neurobiologie van respons op behandeling. So ook is min bekend oor; eerstens die spesifieke komponente van die serotonien sisteem wat betrokke is by simptoom modulasie, en tweedens die gedeeltelik samevallende en afsonderlike brein streke wat deur alternatiewe farmakologiese behandelings beïnvloed word. Ten spyte van die vooruitgang wat die selektiewe serotonien heropname inhibeerders tot gevolg gehad het, is daar nog altyd ‘n betekenisvolle proporsie van pasiënte wat nie voldoende respondeer op hierdie behandelings opsie nie. Dus word alternatiewe opsies benodig. Een so ‘n opsie is die klas dopamien reseptor blokkeerders wat tot onlangs min ondersteunende data gehad het. So ook, is min bekend oor die subgroepe van pasiënte wat die meeste voordeel uit hierdie alternatief sal trek. In hierdie proefskrif sal ek ‘n reeks van ses studies wat farmakologiese middels en enkel foton emissie rekenaar tomografie (EFERT) gebruik om ‘n bydra tot kennis in drie primêre areas van OKS te maak. By name; neurobiologie, behandeling, en die kruispunt van die twee. Eerstens spreek ek neurobiologie aan deur middel van ’n studie wat rustende brein bloed vloei (rBBV) in OKS ondersoek. Hierna ondersoek ek veranderings op rBBV en simptome na eenmalige toediening van ‘n serotonien 1B reseptor agonis, sumatriptan. Tweedens ondersoek ek die kruispunt van neurobiologie en behandeling deur die effek van behandeling met inositol, ‘n voorloper van die fosfoinositol tweedeboodskapper sisteem, op rBBV. Ek ondersoek dan die rBBV patroon van veranderinge in brein streke wat deur twaalf weke van behandeling met die selektiewe serotonien heropname inhibeerder citalopram in verskeie angversteurings bewerkstellig word. Laastens, spreek ek behandeling van OKS aan deur middel van ‘n gekontroleerde studie wat ondersoek instel na die effektiwiteit van die byvoeging van quetiapien, ‘n dopamien reseptor antagonis, tot serotonien heropname inhibeerders in behandelingsweerstandige OKS. Ek kombineer dan hierdie data met ’n soortgelyke datastel om ‘n model af te lei wat kliniese uitkoms vir hierdie behandelings opsie voorspel. Ek het gedemonstreer dat rBBV in OKS betekenisvol verskil van gesonde vergelykbare kontroles. Hierdie verskille het gekorreleer met ernstigheid van OKS in frontale brein streke. Dus bly hierdie tipe studies ’n belangrike rigting van ondersoek in OKS patofisiologie wat tot op hede nie tenvolle uitgewerk is nie. Eenmalige toediening van sumatriptan, het heterogene gedrags en rBBV veranderings in OKS tot gevolg gehad. Pre-frontale verhogings in rBBV voor behandeling is met 5HT1B sumatriptan toediening verminder, ’n effek wat in lyn staan met die effek van selektiewe serotonien heropname inhibeerders. Hierdie werk stel voor dat direkte of indirekte effekte van selektiewe serotonien heropname inhibeerders op die 5HT1B reseptore betrokke mag wees by die meganisme van behandelingsrespons in OKS. In die afdeling waarin ek die kruispunt van neurobiologie en behandeling ondersoek, demonstreer ek dat rBBV veranderings gedeeltelik oorvleuel met dié wat deur selektiewe serotonien heropname inhibeerders veroorsaak word in verskeie angsversteurings. Hierdie data stel voor dat oorvleueling in die neurbiologie van beide behandelingsrespons en kern neurobiologie van hierdie angversteurings ’n waarskynlikheid is. Ek wys ook dat effektiewe behandeling met inositol in OKS ook veranderings in rBBV bewerkstellig wat gedeeltelik in lyn staan met dié van die selektiewe serotonien heropname inhibeerders. Hierdie data ondersteun dus hipoteses van ‘n gemeenskaplike meganisme, wat tweede boodskapper sisteme insluit, wat in die behandelings respons van effektiewe anti-obsessionale middels betrokke is. Die finale deel van hierdie proefskrif handel oor behandeling van OKS. Ten spyte van die onvermoë om ‘n verskil tussen quetiapien en plasebo te demonstreer, het ons onlangs met hierdie data in ‘n reeks meta-analises wel ‘n voordeel vir hierdie intervensie getoon. Ten slote, het ons gevind dat (1) pasiënte wat minder kursusse selektiewe serotonien heropname inhibeerders gefaal het; (2) voor behandeling ‘n erger vorm van OKS gehad het, en (3) ook voordoen met simptoom dimensies wat oënskynlik ‘n dopaminerge basis het, die grootste waarskynlikheid toon om met quetiapien byvoeging tot selektiewe serotonien heropname inhibeerders te respondeer. Met hierdie reeks behandelings en funksionele breinbeeldings ondersoeke, lewer ek ‘n bydra tot die begrip van OKS. Spesifiek dra ek by tot die begrip van die neurobiologie, hantering van behandelingsweerstandige OKS asook die kruispunt van die twee. Farmakologiese middels wat ons óf eenmalig óf vir ‘n volle behandelingskursus toegedien het, het direkte of indirekte uitwerkings op die serotonien and dopamien sisteme gehad, en dus dra hierdie werk ook by tot kennis oor dié se betrokkenheid al dan nie in simptoom modulasie in OKS. Toekomstige werk in die area sal in die breë fokus op die kruispunt van breinfunksie, behandelingsrespons en funksionele genetiese polimorfismes van die monoamien sisteem.
Gittins, Rebecca. "The cytoarchitecture of the human anterior cingulate cortex and its involvement in mood disorder." Thesis, University of Oxford, 2003. http://ora.ox.ac.uk/objects/uuid:8ca079f0-6133-4f6d-a470-3a590789d8bb.
Повний текст джерелаShaikh, Aamir. "Levels of PARP1-immunoreactivity in the Human Brain in Major Depressive Disorder." Digital Commons @ East Tennessee State University, 2020. https://dc.etsu.edu/honors/547.
Повний текст джерелаMiller, Erin M. "Dopamine and Glutamate Dysfunction in a Rodent Model of Attention-Deficit/Hyperactivity Disorder: Implications for Future Neuropharmacology." UKnowledge, 2014. http://uknowledge.uky.edu/neurobio_etds/10.
Повний текст джерелаDornbach-Bender, Allison. "Facets of Positive Affect and Risk for Bipolar Disorder: Role of the Behavioral Activation System." Thesis, University of North Texas, 2017. https://digital.library.unt.edu/ark:/67531/metadc1062834/.
Повний текст джерелаHuggins, Catherine. "EVALUATING THE PEAK: RELATIONAL TRAINING SYSTEM IN TEACHING REFLEXIVITY TO CHILDREN WITH AUTISM SPECTRUM DISORDER." OpenSIUC, 2017. https://opensiuc.lib.siu.edu/theses/2108.
Повний текст джерелаBanskota, Alisha. "Recommending Games to Adults with Autism Spectrum Disorder(ASD) for Skill Enhancement Using Minecraft." BYU ScholarsArchive, 2019. https://scholarsarchive.byu.edu/etd/7734.
Повний текст джерелаGerstein, Rachel. "THE LONG-TERM COURSE OF BIPOLAR SPECTRUM DISORDER: APPLICATIONS OF THE BEHAVIORAL APPROACH SYSTEM (BAS) MODEL." Diss., Temple University Libraries, 2011. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/73971.
Повний текст джерелаPh.D.
In this study, I tested predictions of the Behavioral Approach System (BAS) model as applied to the course of bipolar spectrum disorders. In this model, when a vulnerable individual experiences a BAS activation-relevant event, the weak regulatory strength of the BAS interacts with pre-event BAS state and is likely to lead to hypomania/mania. In contrast, when a vulnerable individual experiences a BAS deactivation-relevant event, the weak regulatory strength of the BAS interacts with pre- event BAS state and is likely to lead to depression. A secondary goal of this study involved comparing the BAS model to the cognitive-vulnerability stress model of bipolar disorder. Toward this end, data from a sample of 217 individuals (112 individuals with a diagnosis in the bipolar spectrum and 105 demographically similar, normal controls) participating in the Longitudinal Investigation of Bipolar Spectrum Disorders (LIBS) Project, a two-site prospective examination of the role of BAS, cognitive styles, and life events in the course of bipolar disorders among college students, were analyzed. The results of this study suggest that there is some support for both the BAS model and the cognitive-vulnerability stress model. Specifically, BAS-relevant cognitive styles, in interaction with congruent positive life events, predicted hypomanic episodes. There was less support for either model in the prediction of depression. There was some support for BAS sensitivity and BAS-relevant events each predicting the course of bipolar disorder. However, there was no support for the interaction of BAS sensitivity and BAS-relevant events predicting the type and number of mood episodes. As such, this study found more support for a BAS-related cognitive vulnerability-stress model, as compared to the "pure" BAS model, as applied to bipolar spectrum disorders. Following a review of the results, strengths and limitations, as well as clinical implications and potential future research directions are discussed.
Temple University--Theses
Fagergren, Pernilla. "Cocaine effects on striatal dynorphin and CART neuropeptides : association to mood disorder /." Stockholm, 2003. http://diss.kib.ki.se/2003/91-7349-515-8.
Повний текст джерелаGruber, David [Verfasser]. "The Role of the Hippocampal GABAergic System in the Development of Posttraumatic Stress Disorder / David Gruber." Berlin : Freie Universität Berlin, 2015. http://d-nb.info/1073868974/34.
Повний текст джерелаHora, Manpreet Kaur. "Feasibility Study in Development of a Wearable Device to Enable Emotion Regulation in Children with Autism Spectrum Disorder." Thesis, Virginia Tech, 2014. http://hdl.handle.net/10919/50518.
Повний текст джерелаMaster of Science
Alvarenga, Marlies. "An analysis of autonomic nervous system fuctioning in panic disorder and its relation to negative affect / Marles E. Alvarenga." Thesis, The Author [Mt. Helen, Vic.] :, 2002. http://researchonline.federation.edu.au/vital/access/HandleResolver/1959.17/45141.
Повний текст джерелаDoctor of Clinical Psychology
Richards, Joanne. "Improving the preliminary stages of the criminal justice system to accommodate people with Autism Spectrum Disorder (ASD)." Thesis, University of Portsmouth, 2015. https://researchportal.port.ac.uk/portal/en/theses/improving-the-preliminary-stages-of-the-criminal-justice-system-to-accommodate-people-with-autism-spectrum-disorder-asd(b3d136f3-6f7e-4430-adda-ef8cb69a4a13).html.
Повний текст джерелаAkpan, Emmanuel. "Intensive Outpatient Treatment Program for Patients with Depressive Disorder: A Parental Perspective." ScholarWorks, 2018. https://scholarworks.waldenu.edu/dissertations/4772.
Повний текст джерелаLowenstein, Joseph Aaron Socrates. "An investigation into the effects of different types of exercise on the maintenance of approach motivation levels using a population analogous to individuals with bipolar disorder." Thesis, University of Exeter, 2013. http://hdl.handle.net/10871/13561.
Повний текст джерелаRichmond, Julia R. "Testing emotion regulation and parasympathetic nervous system deficits as a mechanism for the transmission of borderline personality disorder." Cleveland State University / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=csu1496669065329167.
Повний текст джерелаScruggs, Kent, Tiffany Carrasco, and Michelle Chandley. "Analysis of Neuroinflammatory Markers in the BTBR T+tf/J Mouse Model of Autism Spectrum Disorder." Digital Commons @ East Tennessee State University, 2018. https://dc.etsu.edu/asrf/2018/schedule/50.
Повний текст джерелаGrahn, Robert. "Treatment repeaters : re-entry in care for clients with substance use disorder within the Swedish addiction treatment system." Doctoral thesis, Umeå universitet, Institutionen för socialt arbete, 2017. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-141601.
Повний текст джерелаVrouva, I. "The Achenbach System of Empirically Based Assessment as a measure of personality disorder traits in children and adolescents." Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1370570/.
Повний текст джерелаMusser, Erica. "A Multi-Method Investigation of Intrinsic and Extrinsic Emotion Regulation in Attention-Deficit/Hyperactivity Disorder." Thesis, University of Oregon, 2013. http://hdl.handle.net/1794/13403.
Повний текст джерелаGauffin, Per. "Underlättar medicinering av barn med ADHD barnets pedagogiska situation i skolan?" Thesis, Karlstad University, Faculty of Arts and Education, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:kau:diva-1514.
Повний текст джерелаPersons suffering from Attention deficit/hyperactivity disorder (ADHD) struggle with complications within the functions that regulate and control the brain activities, due to deficiencies in these functions within the affected nerve-paths. ADHD is a cognitive function impairment characterised by inattention, impulsiveness and over activity. According to Diagnostic and Statistic Manual of Mental Disorders, American Psychiatric Association (DSM-IV), certain diagnostic criteria of ADHD must be fulfilled in order for a person to be diagnosed with ADHD. The everyday problems caused by ADHD are individual and medication can have positive effects relieving the person’s impairing behaviour. The study is based on scientific literature, three quantitative scientific articles and preview material from the last study by Johnson, Fransson, Kadesjö & Gillberg, presently being scrutinised. Swedish as well as English literature has been used. The purpose of this study is to shed some light upon whether medication facilitates the child’s school situation. The result deals with the ADHD diagnosis and pharmacological therapy involving drugs that stimulate the central nervous system, as well as naturopathic medicine like Omega-3/6. The pedagogical aspect for children with ADHD in school has been observed and evaluated. In this matter it is important for the pedagogue to encourage the child by letting it find out that it can manage more than it thinks.
CISANI, FRANCESCA. "ROLE OF AMPA RECEPTORS IN PHYSIOPATHOLOGICAL CONDITIONS IN THE CENTRAL NERVOUS SYSTEM." Doctoral thesis, Università degli studi di Genova, 2021. http://hdl.handle.net/11567/1048422.
Повний текст джерелаCHANG, SHU-TING, and 張淑婷. "The Treatment System of Mental Disorder Criminals." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/sh4ryr.
Повний текст джерела國立臺北大學
法律學系一般生組
105
In order to solve the criminal problems of mental disorders, we should value the treatment system of mental disorder criminals. As a result, in this thesis, I intend to review and analyze the treatment system of mental disorder criminals in Taiwan. I hope that the research result can be beneficial to the development of the treatment system of mental disorder criminals. The methodologies adopted in this thesis are literature review, case study and comparative research method. The Chapter 1, Introduction. It describes the research motivation, the methods, and the scope of the study. The Chapter 2, Commitment. It describes the legislative purpose of the commitment, the elements the commitment, and how to enforce the commitment. In my opinion, there are some problems of commitment. In Chapter 3, it discusses that is there any possible way to help mental disorder criminals receive treatment. In our country, deferred prosecution, probation, compulsory hospitalization and compulsory community treatment may the way to help mental disorder criminals receive treatment. In Chapter 4, how to help mental disorder criminals receive treatment after commitment will be discussed. I suggest that conditions of supervision in Germany is worth our reference. The Chapter 5, Collaborative treatment system. It describes that the importance of the collaboration between criminal system and mental health system in treatment enforcement. In our country, the enforcement of deferred prosecution with psychiatric treatment, probation with psychiatric treatment , after care service shown the collaboration between criminal system and mental health system. Furthermore, in my point of view, to plan the treatment system of mental disorder criminals which the collaboration between criminal system and mental health system be valued, the Sequential Intercept Model can be used.The Chapter 6, Conclusion.
LUO, GUANG-CUN, and 羅廣村. "Intelligent Recognition System for Vocal Folds Disorder." Thesis, 2009. http://ndltd.ncl.edu.tw/handle/10599482937880025712.
Повний текст джерела國立臺灣科技大學
高分子系
97
The purpose of this study was to develop an intelligent recognition system for vocal cord disorders. The image processing technology and neural networks were applied to identify different disease conditions of vocal cords, including the presence of paralysis, tumor, malignant cancer, or no diseases. We first changed three random sets of original obtained images into a grayscale format, followed by applying histogram equalization to obtain a good contrast. We then used statistical threshold to segment the processed images. The binary images were lastly computed by labeling, dilation and erosion to obtain the position of the glottis. The features of the three sets of images would be the input of the neural network. After testing 95 samples, the experimental results reveal that the third set had the best recognition rate reaching 93.6%. The results of this experiment support that the intelligent recognition system has the ability to identify vocal cord disorder. Thereby the problems obtained from misdiagnosis and subjective knowledge in the medical field could be reduced effectively.
Boyle, Lia. "A Precision Medicine Approach to Understanding KIF1A Associated Neurological Disorder." Thesis, 2021. https://doi.org/10.7916/d8-0nef-s787.
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