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Добірка наукової літератури з теми "D-2KO"

Автор: Grafiati

Опубліковано: 23 вересня 2022

Оновлено: 28 січня 2023

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Статті в журналах з теми "D-2KO"

Nakamura, Yuri, Ko Sato, Hideki Yamamoto, Kana Matsumura, Ikumi Matsumoto, Toshiki Nomura, Tomomitsu Miyasaka, et al. "Dectin-2 Deficiency Promotes Th2 Response and Mucin Production in the Lungs after Pulmonary Infection with Cryptococcus neoformans." Infection and Immunity 83, no. 2 (November 24, 2014): 671–81. http://dx.doi.org/10.1128/iai.02835-14.

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Dectin-2 is a C-type lectin receptor that recognizes high mannose polysaccharides.Cryptococcus neoformans, a yeast-form fungal pathogen, is rich in polysaccharides in its cell wall and capsule. In the present study, we analyzed the role of Dectin-2 in the host defense againstC. neoformansinfection. In Dectin-2 gene-disrupted (knockout) (Dectin-2KO) mice, the clearance of this fungus and the inflammatory response, as shown by histological analysis and accumulation of leukocytes in infected lungs, were comparable to those in wild-type (WT) mice. The production of type 2 helper T (Th2) cytokines in lungs was higher in Dectin-2KO mice than in WT mice after infection, whereas there was no difference in the levels of production of Th1, Th17, and proinflammatory cytokines between these mice. Mucin production was significantly increased in Dectin-2KO mice, and this increase was reversed by administration of anti-interleukin 4 (IL-4) monoclonal antibody (MAb). The levels of expression of β1-defensin, cathelicidin, surfactant protein A (Sp-A), and Sp-D in infected lungs were comparable between these mice. Inin vitroexperiments, IL-12p40 and tumor necrosis factor alpha (TNF-α) production and expression of CD86 and major histocompatibility complex (MHC) class II by bone marrow-derived dendritic cells and alveolar macrophages were completely abrogated in Dectin-2KO mice. Finally, the disrupted lysates ofC. neoformans, but not of whole yeast cells, activated Dectin-2-triggered signaling in an assay with nuclear factor of activated T cells (NFAT)-green fluorescent protein (GFP) reporter cells expressing this receptor. These results suggest that Dectin-2 may oppose the Th2 response and IL-4-dependent mucin production in the lungs after infection withC. neoformans, and it may not be required for the production of Th1, Th17, and proinflammatory cytokines or for clearance of this fungal pathogen.

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Watts, S., J. M. Vogel, W. D. Harriman, T. Itoh, H. J. Stauss, and R. S. Goodenow. "DNA sequence analysis of the C3H H-2Kk and H-2Dk loci. Evolutionary relationships to H-2 genes from four other mouse strains." Journal of Immunology 139, no. 11 (December 1, 1987): 3878–85. http://dx.doi.org/10.4049/jimmunol.139.11.3878.

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Abstract We generated nucleotide sequences for H-2Kk and H-2Dk from the C3H mouse, as well as for a genomic clone of H-2Db, in order to conduct an evolutionary analysis of the H-2 genes from three haplotypes, k, d, and b. H-2Kk from both the C3H and AKR strains, H-2Kd, H-2Kb, H-2Dk, H-2Ld, H-2Dd, H-2Db, and H-2Dp DNA sequences were aligned, and the alignments used to construct phylogenetic trees inferring the evolutionary relationships among the nine genes by two independent methods. Both approaches yielded trees with similar topologies. In addition, the sequence alignments revealed patterns of nucleotide substitutions which implicate both point mutation and recombination in the divergence of the H-2 genes. Future considerations for evolutionary analysis of class I genes are discussed.

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KANG, DONG YEAP. "Sparse Highly Connected Spanning Subgraphs in Dense Directed Graphs." Combinatorics, Probability and Computing 28, no. 3 (November 5, 2018): 423–64. http://dx.doi.org/10.1017/s0963548318000469.

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Mader proved that every strongly k-connected n-vertex digraph contains a strongly k-connected spanning subgraph with at most 2kn - 2k2 edges, where equality holds for the complete bipartite digraph DKk,n-k. For dense strongly k-connected digraphs, this upper bound can be significantly improved. More precisely, we prove that every strongly k-connected n-vertex digraph D contains a strongly k-connected spanning subgraph with at most kn + 800k(k + Δ(D)) edges, where Δ(D) denotes the maximum degree of the complement of the underlying undirected graph of a digraph D. Here, the additional term 800k(k + Δ(D)) is tight up to multiplicative and additive constants. As a corollary, this implies that every strongly k-connected n-vertex semicomplete digraph contains a strongly k-connected spanning subgraph with at most kn + 800k2 edges, which is essentially optimal since 800k2 cannot be reduced to the number less than k(k - 1)/2.We also prove an analogous result for strongly k-arc-connected directed multigraphs. Both proofs yield polynomial-time algorithms.

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Béghin, Cyril. "d video 2koi ?" Vertigo 40, no. 2 (2011): 35. http://dx.doi.org/10.3917/ver.040.0035.

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Yum, Do-Young, Bong-Yong Lee, Dae-Hyum Hahm, and Jae-Gu Pan. "The yiaE Gene, Located at 80.1 Minutes on the Escherichia coli Chromosome, Encodes a 2-Ketoaldonate Reductase." Journal of Bacteriology 180, no. 22 (November 15, 1998): 5984–88. http://dx.doi.org/10.1128/jb.180.22.5984-5988.1998.

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ABSTRACT An open reading frame located in the bisC-cspAintergenic region, or at 80.1 min on the Escherichia colichromosome, encodes a hypothetical 2-hydroxyacid dehydrogenase, which was identified as a result of the E. coli Genome Sequencing Project. We report here that the product of the gene (yiaE) is a 2-ketoaldonate reductase (2KR). The gene was cloned and expressed with a C-terminal His tag in E. coli, and the protein was purified by metal-chelate affinity chromatography. The determination of the NH2-terminal amino acid sequence of the protein defined the translational start site of this gene. The enzyme was found to be a 2KR catalyzing the reduction of 2,5-diketo-d-gluconate to 5-keto-d-gluconate, 2-keto-d-gluconate (2KDG) to d-gluconate, 2-keto-l-gulonate tol-idonate. The reductase was optimally active at pH 7.5, with NADPH as a preferred electron donor. The deduced amino acid sequence showed 69.4% identity with that of 2KR from Erwinia herbicola. Disruption of this gene on the chromosome resulted in the loss of 2KR activity in E. coli. E. coli W3110 was found to grow on 2KDG, whereas the mutant deficient in 2KR activity was unable to grow on 2KDG as the carbon source, suggesting that 2KR is responsible for the catabolism of 2KDG in E. coli and the diminishment of produced 2KDG from d-gluconate in the cultivation of E. coli harboring a cloned gluconate dehydrogenase gene.

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Stelljes, Matthias, Stella Robert, Karin Frebel, Corinna Opitz, Jennifer Urh, Claudia Dahrenmöller, Christine Baumgart, Jörn C. Albring, Carsten Müller-Tidow, and Wolfgang E. Berdel. "Ubiquitary Expressed Major Histocompatibility (MHC) Class I and Minor Histocompatibility Alloantigens (mHAg) Are No Relevant Targets For Graft-Versus-Tumor (GvT) Reactions In Mice After Allogeneic Bone Marrow Transplantation." Blood 122, no. 21 (November 15, 2013): 3249. http://dx.doi.org/10.1182/blood.v122.21.3249.3249.

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Abstract Introduction Cell mediated GvT effects after allogeneic stem cell transplantation are heterogeneous with respect to effector cell populations, target antigens and their interrelation with graft-versus-host disease (GvHD). Mutated tumor-associated antigens (TAAs), as well as selectively or aberrantly expressed nonmutated antigens, represent potential targets for T cell-mediated GvT effects that are in principle separable from the generalized graft-versus-host reaction targeting widely expressed MHC alloantigens and mHAg. So far, it remains a matter of debate whether alloantigens ubiquitary expressed on tumor cells and normal host tissue are relevant targets of GvT responses. Methods To evaluate the role of MHC class I molecules and mHAg as targets for GvT reactions, we employed three different allogeneic parent-into-F1 murine transplant models (BALB/c [H-2d] or C57BL/6 [H-2b] → F1[BALB/c x C57BL/6] [H-2b/d] and BALB/c → F1[BALB/c x CBA/J] [H-2d/k]). As previously shown, these transplant models allow for separate variation in the histocompatibility between donor cells and normal host tissue on the one hand and between donor cells and tumor tissue on the other hand. Test tumors were the donor derived myeloma / leukemia cell lines MPC11 [H-2d] and C1498 [H-2b], which were stably transfected with MHC class I molecules H-2Kb, H-2Kd, H-2Kk or with the mHAg UTY. Results Compared with non-GvHD controls (F1[H-2b/d] mice transplanted with allogeneic bone marrow cells from BALB/c [H-2d] alone), significant GvT effects against the BALB/c derived tumor MPC11 occurred in F1[H-2b/d] recipients with severe GvHD (transplanted with bone marrow and additional splenic lymphocytes from BALB/c). Identical histocompatibility antigens of donor and tumor cells precluded allorecognition of tumor cells, leaving TAAs the only possible target antigens in this particular setting, with GvHD as a driving force for augmentation of tumor specific immune responses. The MPC11 myeloma [H-2d], stably transfected with the C57BL/6 derived MHC class I molecule H-2Kb (MPC11-Kb), showed similar tumor growth compared to the wild type MPC11 in F1[H-2b/d] mice transplanted from BALB/c donors. Similar results could be observed with the C57BL/6 derived C1498 leukemia [H-2b], transfected with the BALB/c MHC class I alloantigen H2Kd, in F1[H-2b/d] mice transplanted from C57BL/6 donors. In both experimental settings, the artificially expressed alloantigens H2Kb and H2Kd were also expressed ubiquitarily on tissues of F1[H-2b/d] recipients. In sharp contrast, MPC11 cells transfected with the CBA/J derived H2Kk (MPC11-Kk) alloantigen showed a significantly reduced tumor growth in F1[H-2b/d] recipients transplanted from BALB/c donors, demonstrating that MHC class I alloantigens are relevant targets, when expressed on tumors but not on healthy recipient tissue. Inoculation of MPC11-Kk in F1[H-2d/k] mice 3 or 7 days after transplantation from BALB/c donors showed a trend towards a reduced tumor growth compared to wild type MPC11 in recipients with severe GvHD. Later application of tumor cells (14 days after transplantation) showed similar growth pattern of both tumors, suggesting that MHC class I antigens might be GvT targets in the early phase after allogeneic transplantation. To evaluate the impact of ubiquitarily expressed mHAg, we established a C1498 cell line, which stably expressed UTY (C1498-UTY), a known immunogenic antigen of the male HY gene. In comparison with the wild type control, C1498-UTY showed similar tumor growth in male or female F1[H-2b/d] recipients, transplanted from naive female C57BL/6 donors. Immunization of female C57BL/6 donors with male splenocytes resulted in a significantly reduced tumor growth of the C1498-UTY in female F1[H-2b/d] recipients. In contrast, tumor growth of the UTY expressing and wild type C1498 was similar in male F1[H-2b/d] mice transplanted from immunized female C57BL/6 donors. Conclusion Our experimental data indicate that a biologically relevant GvT effect targeting MHC class I or mHAgs mainly occurs when these alloantigens are not ubiquitarily expressed, even in the context of severe GvHD. Consequently, TAAs and tissue- /hematopoietic specific antigens are most likely the main targets of GvT effects observed after allogeneic stem cell transplantation. Disclosures: No relevant conflicts of interest to declare.

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Zhou, Ruiqing, Xiaojun Xu, Huiqing He, Ziwen Guo, Xiaomin Niu, Weihua Li, Shuhua Lin, and Qifa Liu. "Preventative Effect of Glucolysis Inhibitor on Acute Graft Versus Host Disease in Mice after Allogenic Bone Marrow Transplantation." Blood 124, no. 21 (December 6, 2014): 2424. http://dx.doi.org/10.1182/blood.v124.21.2424.2424.

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Abstract Background: T-cell activation plays a critical role in the pathogenesis of acute graft-versus-host disease (aGVHD). Quiescent T cells utilize oxidative phosphorylation to generate ATP, whereas activated T cells utilize glycolysis, so using glycolysis inhibitor may be a metabolically regulator needed to control T cells inducing GVHD. Glucolysis inhibitor 3-Bromopyruvic acid (3-BrPA), a glucolysis inhibitor, can effectively induce multi-drug resistance leukemia cell lines apoptosis and enhance chemotherapy-induced cytotoxity to leukemia cells. Objective: This study aims to investigate the effect of glucolysis 3-BrPA inhibitor on aGVHD in mice after allogenic bone marrow transplantation (allo-BMT) and its mechanism. Methods: aGVHD model after allo-BMT was established by use of C57BL/6(H-2kb) mice as donors and BALB/c(H-2kd) mice as receptors. Bone marrow and/or spleen cells from donor mice were injected within 4h after total body irradiation (TBI) to receptor mice via tail vein. Drugs were administrated 1 h after the cells were injected as follows: (1) control: the TBI group without cells injection; (2) the BMT group with bone marrow cells injection; (3) the GVHD group with bone marrow and spleen cells injection; (4) the rap amycin group (RAPA, 20 nM/d×7d) with bone marrow and spleen cells injection; (5) the 3-BrPA group (3-BrPA, 50 nM/d×7d) with bone marrow and spleen cells injection; (6) the 3-BrPA(25 nM/d×7d) and rapamycin(10 nM/d) combination group with bone marrow and spleen cells injection. The transplanted mice were observed for symptoms of GVHD, survival time, survival rate and Thomas GVHD pathologic grade. H-2kb was determined in BALB/c(H-2kd) mice by flow cytometry (FCM) to confirm allogeneic chimeric rate, and serum level of cytokine was detected by protein microarray. Results: Allogeneic chimeric rates of survived mice determined at day 21 after transplantation ranged from 95% to100%, confirming complete donor mouse implantation. All mice in TBI group died within 14 d. Median survival time of mice was respectively 9.1, 20, 17.1 and 24.5 days in GVHD group , rapamycin group, 3-BrPA group and the combination of 3-BrPA and rapamycin group. Rapamycin group, 3-BrPA group and the combination of 3-BrPA and rapamycin group had increased median survival time than GVHD group, and mice received the combination of rapamycin and 3-BrPA had better survival than rapamycin or 3-BrPA group(n=10 for each group, P<0.01). GVHD-related symptoms scores of all the drug treated groups on 7-day and 14-day after transplantation were decreased compared to GVHD group (F=15.006, P<0.001). The cytokine arrays showed that the levels of Th1-associated cytokines IFN-γ increased and the levels of Th2-associated cytokines IL-4 deceased in the groups with drug treated compared to the GVHD group. Conclusions: In this study, the glycolysis inhibitor 3-BrPA has a significant inhibitory effect on GVHD, especially in combination with rapamycin. This effect might be achieved by regulating immune cells bias from Th1 cells towards Th2 cells. Disclosures Liu: National Natural Science Foundation of China (81270647, 81300445, 81200388): Research Funding; National High Technology Research and Development Program of China (863 Program) (2011AA020105): Research Funding; National Public Health Grand Research Foundation (201202017): Research Funding; Natural Science Foundation of Guangdong Province (S2012010009299): Research Funding; the project of health collaborative innovation of Guangzhou city (201400000003-4, 201400000003-1): Research Funding; the Technology Plan of Guangdong Province of China (2012B031800403): Research Funding; the project of the Zhujiang Science & Technology Star of Guangzhou city (2013027): Research Funding.

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Edera, L., та M. R. Pennington. "Estimating theI=3/2Kπ interaction in D decay". Physics Letters B 623, № 1-2 (вересень 2005): 55–64. http://dx.doi.org/10.1016/j.physletb.2005.07.030.

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Umezawa, Kiwamu, Kouta Takeda, Takuya Ishida, Naoki Sunagawa, Akiko Makabe, Kazuo Isobe, Keisuke Koba, et al. "A Novel Pyrroloquinoline Quinone-Dependent 2-Keto-d-Glucose Dehydrogenase from Pseudomonas aureofaciens." Journal of Bacteriology 197, no. 8 (February 2, 2015): 1322–29. http://dx.doi.org/10.1128/jb.02376-14.

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A gene encoding an enzyme similar to a pyrroloquinoline quinone (PQQ)-dependent sugar dehydrogenase from filamentous fungi, which belongs to new auxiliary activities (AA) family 12 in the CAZy database, was cloned fromPseudomonas aureofaciens. The deduced amino acid sequence of the cloned enzyme showed only low homology to previously characterized PQQ-dependent enzymes, and multiple-sequence alignment analysis showed that the enzyme lacks one of the three conserved arginine residues that function as PQQ-binding residues in known PQQ-dependent enzymes. The recombinant enzyme was heterologously expressed in anEscherichia coliexpression system for further characterization. The UV-visible (UV-Vis) absorption spectrum of the oxidized form of the holoenzyme, prepared by incubating the apoenzyme with PQQ and CaCl2, revealed a broad peak at approximately 350 nm, indicating that the enzyme binds PQQ. With the addition of 2-keto-d-glucose (2KG) to the holoenzyme solution, a sharp peak appeared at 331 nm, attributed to the reduction of PQQ bound to the enzyme, whereas no effect was observed upon 2KG addition to authentic PQQ. Enzymatic assay showed that the recombinant enzyme specifically reacted with 2KG in the presence of an appropriate electron acceptor, such as 2,6-dichlorophenol indophenol, when PQQ and CaCl2were added.1H nuclear magnetic resonance (1H-NMR) analysis of reaction products revealed 2-keto-d-gluconic acid (2KGA) as the main product, clearly indicating that the recombinant enzyme oxidizes the C-1 position of 2KG. Therefore, the enzyme was identified as a PQQ-dependent 2KG dehydrogenase (Pa2KGDH). Considering the high substrate specificity, the physiological function ofPa2KGDH may be for production of 2KGA.

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Kataoka, Naoya, Minenosuke Matsutani, Toshiharu Yakushi, and Kazunobu Matsushita. "Efficient Production of 2,5-Diketo-d-Gluconate via Heterologous Expression of 2-Ketogluconate Dehydrogenase in Gluconobacter japonicus." Applied and Environmental Microbiology 81, no. 10 (March 13, 2015): 3552–60. http://dx.doi.org/10.1128/aem.04176-14.

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ABSTRACT2,5-Diketo-d-gluconate (2,5DKG) is a compound that can be the intermediate ford-tartrate and also vitamin C production. AlthoughGluconobacter oxydansNBRC3293 produces 2,5DKG fromd-glucose viad-gluconate and 2-keto-d-gluconate (2KG), with accumulation of the product in the culture medium, the efficiency of 2,5DKG production is unsatisfactory because there is a large amount of residuald-gluconate at the end of the biotransformation process. Oxidation of 2KG to 2,5DKG is catalyzed by a membrane-bound flavoprotein-cytochromeccomplex: 2-keto-gluconate dehydrogenase (2KGDH). Here, we studied thekgdSLCgenes encoding 2KGDH inG. oxydansNBRC3293 to improve 2,5DKG production byGluconobacterspp. ThekgdS,kgdL, andkgdCgenes correspond to the small, large, and cytochrome subunits of 2KGDH, respectively. ThekgdSLCgenes were cloned into a broad-host-range vector carrying a DNA fragment of the putative promoter region of the membrane-bound alcohol dehydrogenase gene ofG. oxydansfor expression inGluconobacterspp. According to our results, 2KGDH that was purified from the recombinantGluconobactercells showed characteristics nearly the same as those reported previously. We also expressed thekgdSLCgenes in a mutant strain ofGluconobacter japonicusNBRC3271 (formerlyGluconobacter dioxyacetonicusIFO3271) engineered to produce 2KG efficiently from a mixture ofd-glucose andd-gluconate. This mutant strain consumed almost all of the starting materials (d-glucose andd-gluconate) to produce 2,5DKG quantitatively as a seemingly unique metabolite. To our knowledge, this is the first report of aGluconobacterstrain that produces 2,5DKG efficiently and homogeneously.

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Більше джерел

Дисертації з теми "D-2KO"

Wang, Yazhou. "Influence of Dectin-1 and Dectin-2 receptors on the susceptibility to gut inflammation." Thesis, Sorbonne université, 2021. http://www.theses.fr/2021SORUS523.

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Une dysbiose du microbiote intestinal a été identifiée comme impliquée dans la pathogenèse des maladies inflammatoires chroniques de l'intestin (MICI). Il a été démontré que les interactions hôte-bactéries affectent le développement des MICI, tandis que le rôle des interactions hôte-champignons dans les MICI sont peu décrites. Les lectines de type C sont des récepteurs impliqués dans la reconnaissance de motifs du mycobiote et font les réponses immunitaires face aux agents pathogènes fongiques. Nous avons évalué l'impact de la déficience des récepteurs Dectin-1 (D-1KO), Dectin-2 (D-2KO) et de la double déficience (D-1/2KO) dans l'inflammation intestinale. L’absence de D-1 ou D-2 n’a pas modifié la gravité de l'inflammation intestinale, tandis que les souris D-1/2KO étaient résistantes à la colite. Le rôle protecteur de D-1/2KO a été confirmé chez des souris WT ayant reçu le microbiote intestinal desD-1/2KO, suggérant que la protection était largement due au microbiote. L'analyse du microbiote des souris D-1/2KO montre que le microbiote bactérien, et en particulier la famille des Lachnospiraceae, mais pas le mycobiote, présentait une forte modification par rapport aux souris WT. Une supplémentation en Blautia hansenii a pu également assurer la protection, appuyant que cette protection était largement médiée par le microbiote bactérien. Ces résultats montrent que la déficience D-1/2 protége les souris de la colite via une modulation du microbiote intestinal bactérien
Gut microbiota dysbiosis has been identified as being involved in the pathogenesis of inflammatory bowel disease (IBD). Host-bacterial interactions have been shown to affect the development of IBD, while the role of host-fungal interactions in IBD is poorly described. C-type lectins are receptors involved in the recognition of patterns of the mycobiota and shape the immune responses to fungal pathogens. We evaluated the impact of the deficiency of the receptors Dectin-1 (D-1KO), Dectin-2 (D-2KO) and dual deficiency (D-1/2KO) in intestinal inflammation. The absence of D-1 or D-2 did not alter the severity of intestinal inflammation, whereas D-1/2KO mice were resistant to colitis. The protective role of D-1/2KO was confirmed in WT mice receiving the gut microbiota of D-1/2KO by fecal transfer, suggesting that protection was largely due to the gut microbiota. Analysis of the microbiota of D-1/2KO mice shows that the bacterial microbiota, and in particular the Lachnospiraceae family, but not the mycobiota, showed a strong change compared to the microbiota of the WT mice. Blautia hansenii supplementation was also able to provide protection, supporting that this protection was largely mediated by the bacterial microbiota and not the fungal microbiota. These results show that D-1/2KO deficiency protect mice from DSS-induced colitis via modulation of the bacterial gut microbiota

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Driouchi, Driss. "Contribution à la construction des plans factoriels fractionnaires D(2k-p)AM Rmax et applications." Paris 6, 2005. http://www.theses.fr/2005PA066086.

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Книги з теми "D-2KO"

Otruba, Kathy. COM20020I Rev. d 5Mbps ARCNET (ANSI 878. 1) Controller with 2K X 8 on-Chip RAM - Data Sheet. Microchip Technology Incorporated, 2020.

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Otruba, Kathy. COM20020I Rev. d 5Mbps ARCNET (ANSI 878. 1) Controller with 2K X 8 on-Chip RAM - Data Sheet. Microchip Technology Incorporated, 2020.

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Частини книг з теми "D-2KO"

Bettayeb, Said, Zevi Miller, Tony Peng, and Hal Sudborough. "Embedding k-D meshes into optimum hypercubes with dilation 2k-1 extended abstract." In Lecture Notes in Computer Science, 73–80. Berlin, Heidelberg: Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/3-540-58078-6_7.

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Pease, L. R., J. K. Pullen, Z. Cai, and R. M. Horton. "Contributions of Interlocus Exchange to the Structural Diversity of the H-2K, D, and L Alleles." In Molecular Evolution of the Major Histocompatibility Complex, 163–70. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-84622-9_13.

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Hay, J. H., J. P. Ballad, R. W. Crookes, D. C. Edwards, D. Robinson, and A. W. Morris. "DESIGN AND DEVELOPMENT OF A ±2kV ±10kA HIGH SPEED GTO SWITCHING AMPLIFIER FOR FAST EQUILIBRIUM CONTROL ON COMPASS-D." In Fusion Technology 1992, 867–71. Elsevier, 1993. http://dx.doi.org/10.1016/b978-0-444-89995-8.50167-3.

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"Results for (ii) and (iii) are shown in Table III, which records the range of odour potential for samples collected over a one year period. Mean values for odour potential are also shown, these having been calculated from approximately 15 measurements. From Table II the seasonal variation in odour problems is clearly apparent. This effect is most marked with hunus sludge as the proportion of viable bacteria in the sludge increases markedly during the spring, and coupled with the rising sludge temperature greatly increases the kinetics of the biochemical reduction of sulphur and nitrogen compounds. Table II. Variations with sample date of odour potential of humus sludge Odour strength (D) (Dilutions to Threshold) Sample Range Mean Monthly dates mean 22.1 5k-31.6k 15.8k ) 5.21.3k-20k 15.8k ) 15.8k 12.2 10k-20k 15.8k ) 19.2 12.6k-20k 15.8k ) 26.2 31.6k-40k 31.6k ) 25k 12.3 5k-20k 15.8k ) 26.3 20k-80k 50k ) 2.4 80k-200k 126k ) 126k 9.4 200k->250k 200k ) 14.5 250k->250k >250k 21.5 250k->250k >250k j >250k Table III. Inter-works variation in sludge odour potential Odour strength (D) (Dilutions to threshold) Fresh sludge 3-week old sludge Sludge and source (up to 24 hours old) Range Mean Range Mean Esholt 6.3k - 8k 8k 6.3k 8k primary Esholt 4k-250k 63k 630-6.3k 2k humus Knostrop 5k-15.8k 10k 6.3k-12.6k 10k primary/htmus mix Owlwood 320-5k 2.5k 12.6k-63k 40k surplus activated Owlwood not available 32k-80k 63k primary/surplus activated mix." In Odour Prevention and Control of Organic Sludge and Livestock Farming, 150. CRC Press, 1986. http://dx.doi.org/10.1201/9781482286311-62.

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Тези доповідей конференцій з теми "D-2KO"

Bose, Partha. "In-Line-Inspection (ILI) of a Pipeline in Constrained Condition and Establishing Integrity of the Pipeline." In ASME 2021 India Oil and Gas Pipeline Conference. American Society of Mechanical Engineers, 2021. http://dx.doi.org/10.1115/iogpc2021-64379.

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Abstract One 24” Diameter and around 40 km length Natural Gas pipeline caters as energy life line for Mumbai city; supplying app 4.5 MMSCMD Gas for Auto Sectors (CNG), House Hold (PNG), Power, Fertiliser, Petrochemical Sectors. Though line is equipped with Launcher & Receiver; but onward became challenging one for executing pigging for many constraints: - Presence of One SR bend - Presence of 1.5 D bends - Presence of 1.5 D bends with Back to Back configuration - Three (3) no Thermal Expansion Loops, in 2 Km stretch passing as above ground pipeline through bridge (above Creek) section The pipe line is passing through High Consequence areas, including its interim stretch of 2km passing as above ground section through bridge structure. Intelligent pigging is an obvious first preference for online precise integrity assessment for any pipeline. Site Specific Assessment, Detail Engineering & Committed approach resulted in Feasibility & Development of ILI Tool, Practical Testing in fore hand before actual pigging & onward Integrity Assessment of the pipeline conducted by accomplishing Successful ILI run.

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Guzman Moreno, R. A., L. G. Piñeros, J. Garcia, L. M. Pombo, A. Teheran, J. Bustillo, K. A. Guzman Moreno, V. Cadavid, and M. C. Mejia. "AB0618 Prevalence of hypovitaminosis d in adults with systemic lupus erythematosus and the relationship with sledai 2k in patients treated in two rheumatology services, bogota 2017." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.3990.

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