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Статті в журналах з теми "Cerebrovascular Accident"

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Автор: Grafiati

Опубліковано: 4 червня 2021

Оновлено: 27 січня 2023

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1

&NA;. "CEREBROVASCULAR ACCIDENT." Nursing 20, no. 1 (January 1990): 94–98. http://dx.doi.org/10.1097/00152193-199001000-00033.

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2

Ohki, Shin-ichi, Insu Kubota, Kei Aizawa, and Yoshio Misawa. "Aortogenic cerebrovascular accident." Interactive CardioVascular and Thoracic Surgery 9, no. 5 (November 2009): 899–900. http://dx.doi.org/10.1510/icvts.2009.212241.

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3

LOESCH, JUDITH A. "ABOUT CEREBROVASCULAR ACCIDENT." Nursing 20, no. 11 (November 1990): 26–29. http://dx.doi.org/10.1097/00152193-199011000-00011.

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4

Wilkinson, Graham, Melinda Parcell, and Avis Macdonald. "Cerebrovascular accident clinical pathway." Journal of Quality In Clinical Practice 20, no. 2-3 (June 2000): 109–12. http://dx.doi.org/10.1046/j.1440-1762.2000.00362.x.

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5

Bhuvaneshwari G and Somiya C. "Effectiveness of dysphagia exercises on swallowing ability among patients with Cerebrovascular Accidents." International Journal of Research in Pharmaceutical Sciences 11, no. 2 (April 4, 2020): 1515–18. http://dx.doi.org/10.26452/ijrps.v11i2.2027.

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Анотація:

Cerebrovascular accident (CVA) is one of the foremost reasons leading to mortality and morbidity throughout the world. It is the third biggest killer in India after a heart attack and cancer. It is like a chronic health condition which negatively impacts on quality of life. Dysphagia is one of the most successive side effects in patients with a stroke which is a loss of motion of throat muscles. This condition can disturb the gulping procedure and make eating, drinking, taking prescription and breathing trouble. Dysphagiaexercises are designed toenhancing muscles and coordinatingthe nervesandmuscles involved in swallowing.The pre-experimental design was used with 60 samples who matched the inclusion criteria who were selected by purposive sampling technique.The study aims is to assess the swallowing ability before and after dysphagia exercisesamong patients with cerebrovascular accidents. Demographical variables were collected by using self- structured questionnaires and Modified Mann Assessment of Swallowing Ability Scale.The study uncovered that the pre-test mean swallowing ability score was 42.45 with a standard deviation of 8.63 among the cerebrovascular accident patients. The post-test swallowing ability mean score of 50.54% with a standard deviation of 8.23 among the cerebrovascular accident patients. The study findings concluded thatDysphagia practices which was an effective, inexpensive, simple measure for improving swallowing ability among patients with cerebrovascular accident.

6

Kumar, GNanda, KV Ragi, and PradeepS Nair. "Pseudoxanthoma elasticum with cerebrovascular accident." Indian Journal of Dermatology, Venereology and Leprology 73, no. 3 (2007): 191. http://dx.doi.org/10.4103/0378-6323.32746.

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7

Young, B., and L. Yao. "M003 OMALIZUMAB ASSOCIATED CEREBROVASCULAR ACCIDENT." Annals of Allergy, Asthma & Immunology 127, no. 5 (November 2021): S58. http://dx.doi.org/10.1016/j.anai.2021.08.176.

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8

Cho, Ki Hyun. "The Management of Cerebrovascular Accident." Journal of the Korean Medical Association 45, no. 12 (2002): 1450. http://dx.doi.org/10.5124/jkma.2002.45.12.1450.

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9

Trevett, Andrew J., Nneka C. Nwokolo, Isi H. Kevau, and R. Andrew Seaton. "Cerebrovascular accident after taipan bite." Medical Journal of Australia 160, no. 2 (January 1994): 94. http://dx.doi.org/10.5694/j.1326-5377.1994.tb126533.x.

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10

Sutherland, Struan K. "Cerebrovascular accident after taipan bite." Medical Journal of Australia 160, no. 2 (January 1994): 94. http://dx.doi.org/10.5694/j.1326-5377.1994.tb126534.x.

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11

Özelçi Kavas, G., M. C. Akbostanci, P. Aribal Kocatürk, C. Isikay, A. Öcal, D. Tuncer, and N. Mutluer. "Antioxidant status in cerebrovascular accident." Pathophysiology 5 (June 1998): 210. http://dx.doi.org/10.1016/s0928-4680(98)81110-3.

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12

Weekes, E., and M. Elia. "Energy expenditure following cerebrovascular accident." Clinical Nutrition 10 (January 1991): 60. http://dx.doi.org/10.1016/0261-5614(91)90302-s.

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13

Eastwood, M. R., S. L. Rifat, H. Nobbs, and J. Ruderman. "Mood Disorder Following Cerebrovascular Accident." British Journal of Psychiatry 154, no. 2 (February 1989): 195–200. http://dx.doi.org/10.1192/bjp.154.2.195.

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Depression appears to be common after stroke, and therefore may have a bearing upon rehabilitation. To examine whether the depression is due to a specific brain lesion, or is reactive to the consequent disability, this study looked at the frequency and associations of depressed mood in a stroke rehabilitation unit in-patient population, unselected for site of lesion. Depression affected 50% of the patients; history of previous psychiatric disorder and cerebrovascular accident appeared to be important risk factors. There were hemispheric differences in the relationships between measures, with both the site-of-lesion and reactive viewpoints being upheld.

14

Kocatürk, Pelin Aribal, M. Cenk Akbostanci, Canan Işikay, Aylin Öcal, Deniz Tuncel, Güzin Özelçi Kavas, and Nermin Mutluer. "Antioxidant Status in Cerebrovascular Accident." Biological Trace Element Research 80, no. 2 (2001): 115–24. http://dx.doi.org/10.1385/bter:80:2:115.

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15

Kohler, Myrta, Hanna Mayer, Juerg Kesselring, and Susi Saxer. "Post-Cerebrovascular Accident Unpredictable Incontinence." Rehabilitation Nursing 44, no. 2 (2019): 69–77. http://dx.doi.org/10.1097/rnj.0000000000000097.

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16

Muntan, C. D., and V. Tuckler. "Cerebrovascular accident following MDMA ingestion." Journal of Medical Toxicology 2, no. 1 (March 2006): 16–18. http://dx.doi.org/10.1007/bf03161008.

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17

Gupta, Gaurav, Saurabh Kishor, and Aditya Kumar. "Serum uric acid, lipid profile and alkaline phosphatase levels in ischemic cerebrovascular accident patients." International Journal of Advances in Medicine 8, no. 8 (July 23, 2021): 1171. http://dx.doi.org/10.18203/2349-3933.ijam20212862.

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Background: Stroke or cerebrovascular accident (CVA) is noted as the second cause of mortality, especially in the elderly population. Recent studies indicated that higher concentrations of uric acid are involved in various vascular diseases. The findings of previous investigations suggest that, elevated serum alkaline phosphatase (ALP) levels may have a pathophysiological character in the occurrence of atherosclerotic vascular disease (AVD) of the heart and brain. This study evaluated the association between serum uric acid (SUA) levels, serum lipid levels, serum alkaline phosphatase (ALP) levels, and changes in ischemic cerebrovascular accident patients.Methods: All patients with Ischemic cerebrovascular accident age >50 years were included based on their clinical, laboratory, and radiological findings (including computed tomography (CT)/magnetic resonance imaging (MRI)) those admitted in our hospital. As control group 200 healthy individuals matched for sex and age were recruited from the same demographic area.Result: Multiple logistic regression analysis findings proposed four components as significant predictors in ischemic cerebrovascular accident (serum uric acid, serum ALP, LDL and HDL. In this study, it was found, that patients with ischemic cerebrovascular accident had significant difference (p<0.001) in serum uric acid and serum ALP than normal patients (non-ischemic cerebrovascular accident patients).Conclusions: Patients with ischemic cerebrovascular accident had significant difference (p<0.001) in SUA and serum ALP than normal patients (non-ischemic cerebrovascular accident patients). High SUA levels were observed to be associated significantly with ischemic stroke. On the basis of our study design, we cannot clarify that the elevated levels are the risk of ischemic stroke and it requires further studies.

18

Drapkina, O. M., I. I. Almazova, A. V. Smirnova, S. A. Berns, and R. N. Shepel. "Cerebrovascular Accident in a Patient with Polycythemia: a Case Report." Rational Pharmacotherapy in Cardiology 18, no. 1 (March 5, 2022): 79–84. http://dx.doi.org/10.20996/1819-6446-2022-02-10.

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Polycythemia vera is not only a clonal disease that causes hematopoietic stem cells, but also a pathology that often leads to thrombotic complications. Thrombosis can have different localization and is clinically manifested by stroke, myocardial infarction, deep vein thrombosis of the lower extremities, pulmonary embolism, thrombosis of the veins of internal organs and other conditions. One of the most formidable thrombotic complications is acute cerebrovascular accident. The heterogeneity of the possible causes of acute cerebrovascular accident requires a careful approach to differential diagnosis for timely diagnosis and individual, pathogenetically grounded selection of means of long-term antithrombotic therapy. The presented clinical case of the development of cerebrovascular accident in a patient with polycythemia vera demonstrates the importance of an informal approach to diagnosis, as well as interdisciplinary interaction for finding the true cause of the development of acute cerebrovascular accident and the appointment of pathogenetically based treatment, aimed, among other things, at the prevention of repeated episodes of acute cerebrovascular accident and others. thrombotic complications.

19

MARUYAMA, Hitoshi. "Physical Fitness of Cerebrovascular Accident Patients." Rigakuryoho kagaku 16, no. 1 (2001): 31–34. http://dx.doi.org/10.1589/rika.16.31.

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20

Olsen-Vetland, Pamela. "Urinary continence after a cerebrovascular accident." Nursing Standard 17, no. 39 (June 11, 2003): 37–41. http://dx.doi.org/10.7748/ns2003.06.17.39.37.c3405.

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21

Olsen-Vetland, Pamela. "Urinary continence after a cerebrovascular accident." Nursing Standard 17, no. 39 (June 11, 2003): 37–41. http://dx.doi.org/10.7748/ns.17.39.37.s46.

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22

Onder, Halil, and Sahin Hanalioglu. "QT dispersion and acute cerebrovascular accident." Journal of Neurosciences in Rural Practice 7, no. 04 (April 2016): 612–13. http://dx.doi.org/10.4103/0976-3147.185503.

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23

Manners, Jody, Alexis Steinberg, and Lori Shutter. "Early management of acute cerebrovascular accident." Current Opinion in Critical Care 23, no. 6 (December 2017): 556–60. http://dx.doi.org/10.1097/mcc.0000000000000462.

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24

Monica, R., B. Zahara, V. Ester, M. Valentin, and C. Jaume. "Risk factors for postoperative cerebrovascular accident." European Journal of Anaesthesiology 25, Sup 44 (May 2008): 56. http://dx.doi.org/10.1097/00003643-200805001-00174.

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25

Weightman, Margaret M. "Motor Unit Behavior Following Cerebrovascular Accident." Neurology Report 18, no. 1 (1994): 26–28. http://dx.doi.org/10.1097/01253086-199418010-00020.

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26

Trauner, Doris A., and Frank L. Mannino. "Neurodevelopmental outcome after neonatal cerebrovascular accident." Journal of Pediatrics 108, no. 3 (March 1986): 459–61. http://dx.doi.org/10.1016/s0022-3476(86)80897-6.

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27

Burney, Tracy L., Mukti Senapati, Samir Desai, S. T. Choudhary, and Gopal H. Badlani. "EFFECTS OF CEREBROVASCULAR ACCIDENT ON MICTURITION." Urologic Clinics of North America 23, no. 3 (August 1996): 483–90. http://dx.doi.org/10.1016/s0094-0143(05)70327-x.

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28

Urschel, John D., Doris F. Vretenar, William J. Dickout, and Someshwar S. Nakai. "Cerebrovascular accident complicating extended cervical mediastinoscopy." Annals of Thoracic Surgery 57, no. 3 (March 1994): 740–41. http://dx.doi.org/10.1016/0003-4975(94)90579-7.

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29

BUZZO (PRIHOI), Elena Lăcrămioara. "Speech therapy in stroke (cerebrovascular accident- CVA)." Revista Română de Terapia Tulburărilor de Limbaj şi Comunicare 1, no. 1 (October 15, 2015): 41–47. http://dx.doi.org/10.26744/rrttlc.2015.1.1.06.

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30

Chouhan, Mahendra, and Rakesh K. Sisodia. "The study of serum uric acid in cerebrovascular accident patients and correlation with hypertension." International Journal of Advances in Medicine 7, no. 3 (February 24, 2020): 418. http://dx.doi.org/10.18203/2349-3933.ijam20200517.

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Background: Cerebrovascular accident (CVA) is also called Stroke. These is a well-recognized epidemiological link between elevated serum uric acid and increased cerebrovascular risk. Several studies have identified as elevated serum uric acid concentration as a predictor of cerebrovascular events. The aim of this study was to correlate serum uric acid in cerebrovascular accident (CVA) patients with Hypertension.Methods: A prospective study was conducted on 100 patients aged >18 years of admitted with new onset focal/global neurological deficit/event with cerebrovascular accident (CVA). Brain imaging (CT/MRI) was performed on the patients within 24-48 hrs of admission. Data was collected with regards to patient’s demography, medical history, risk factors for stroke or vascular disease. Serum uric acid was measured as part of fasting biochemical profile taken within 24-48 hrs of admission by standard analytical methods in Biochemistry department.Results: Out of 100 patients included in this study 74 of them were male and 26 were female. Among 100 patients 61 patients had hemorrhage, out of which 45 (73.77%) were male and 16 (26.23%) were female, followed by 39 patients had infract, out of which 29 (74.35%) were male and 10 (25.65%) were female. Mean value of serum uric acid levels in hypertensive patients was significantly high in comparison to normotensive patients.Conclusions: Concluded that correlation of serum uric acid in cerebrovascular accident (CVA) patients with Hypertension was found statistically significant.

31

Parimala, P., V. Kalpana, and R. Maheshwari. "Effectiveness of Swallow Therapy on Swallowing Ability among Patients with Cerebrovascular Accident." CARDIOMETRY, no. 23 (August 20, 2022): 148–53. http://dx.doi.org/10.18137/cardiometry.2022.23.148153.

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Cerebrovascular accident occurs when the blood supply to the brain is disturbed in some way. As a result the brain cells are deprived of oxygen. It is also known as stroke. It is being observed as a rapidly growing problem and an important cause of illness and death in Saudi arabia. The major problem of cerebrovascular accident is paralysis of swallowing muscles leading to swallowing difficulty which is known as dysphagia. It involves the mouth, throat and esophagus. The goal of this study was to see how swallow treatment affected patients with cerebrovascular accidents at Saveetha Medical College and Hospital in Chennai. The study used a quasi-experimental one-group pre- and post-test design. A total of 30 samples were chosen with care. The Mann Assessment of Swallowing Skills (MASA) was used to examine swallowing ability on regular basis. The results reveal that there is a significant difference in swallowing capacity before and after therapy when using descriptive and interferential statistical approaches. The average score of swallowing ability before swallow therapy was 155.0±16.34, while the average score after swallow therapy was 170.87±11.12. At p<0.001 level, the estimated paired ‘t’ test value of t = 7.171 was judged to be statistically highly significant. This obviously implies that administering swallow therapy to individuals with cerebrovascular accident was shown to be useful in enhancing post-test swallowing skills. The swallowing therapy was found to be one of the most successful traditional therapies for improving the swallowing skills of people with swallowing difficulties, according to the findings.

32

Joo, S. H., T. S. Chung, J. H. Suh, D. I. Kim, I. S. Choi, and M. S. Lee. "Aspiration in cerebrovascular accident patients: videofluoroscopic findings." Journal of the Korean Radiological Society 26, no. 4 (1990): 672. http://dx.doi.org/10.3348/jkrs.1990.26.4.672.

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33

Shankar, SK, BN Nandeesh, A. Mahadevan, A. Tandon, and P. Satishchandra. "Aortoiliac occlusive disease masquerading as cerebrovascular accident." Annals of Indian Academy of Neurology 10, no. 3 (2007): 178. http://dx.doi.org/10.4103/0972-2327.34799.

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34

Dhiman, JitenderKumar, D. Prasanth, MahapatraArun Kumar, and SantoshKumar Bhatted. "Management of Cerebrovascular Accident (CVA) through Ayurveda." Journal of Ayurveda Case Reports 1, no. 2 (2018): 22. http://dx.doi.org/10.4103/2667-0593.350867.

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35

Rahman, Md Siddiqur, Md Monowarul Islam, Mohammad Asaduzzaman, Shahana Afroze, Is mot Ara Zannat, and Mohammad Mahbubul Haque. "Voiding Disorders in Patients with Cerebrovascular Accident." Bangladesh Journal of Urology 23, no. 2 (November 15, 2020): 136–41. http://dx.doi.org/10.3329/bju.v23i2.50304.

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Background: Cerebrovascular accident often happens in elderly group, frequently causing voiding dysfunction. The aim of the study was to determine the magnitudes of various types of voiding disorders among patients of cerebrovascular accidents attending in Dhaka Medical College Hospital. Methods: It was a cross-sectional study conducted at the department of Urology ofDhaka Medical College Hospital from July 2009 to June 2011. Hundred and eight patients were enrolled in the study. Results: More than 61% subjects were male (n=66).Leading number of patients (45.4%) had three to six months disease duration. In this group 67.3% had overactive bladder, 64.7% had underactive bladder and 24.5% had normal activity of bladder. In patients with irritative symptoms majority had overactive bladder (71.4%) while in patients with obstructive symptoms half of the subjects had overactive bladder (50.0%). Majority of the subjects with DM (71.4%) and HTN (82.6%) had overactive bladder. Seventy percent (n=) subjects with overactive bladder had arterial stroke while around 82% (n=) of the underactive bladder subjects had venous stroke. More than 85% subjects with underactive bladder and around 78% subjects with overactive bladder had infection. Conclusion: Voiding dysfunction is a common sequel of acute stroke and most of the patients with irritative symptoms and about half of the subjects with obstructive symptoms had overactive bladder. Diabetes mellitus, hypertension and arterial stroke were found to be associated with overactive bladder. Bangladesh Journal of Urology, Vol. 23, No. 2, July 2020 p.136-141

36

UEMATSU, Yuji, Toshikazu KUWATA, Ichiro KAMEI, Munehisa IWAMOTO, and Tsuyoshi KURIYAMA. "Cerebrovascular Accident associated with Toxemia of Pregnancy." Neurologia medico-chirurgica 28, no. 5 (1988): 477–80. http://dx.doi.org/10.2176/nmc.28.477.

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37

Karagoz, Bulent, Oguz Bilgi, Ilker Akyol, Alpaslan Ozgun, Orhan Turken, and Emin Gokhan Kandemir. "Cerebrovascular Accident After Chemotherapy for Testicular Cancer." Military Medicine 174, no. 3 (March 2009): 320–21. http://dx.doi.org/10.7205/milmed-d-01-3508.

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38

Lindsay, M. "Frequency of cerebrovascular accident after cardiac surgery." Critical Care Nurse 18, no. 5 (October 1, 1998): 19–25. http://dx.doi.org/10.4037/ccn1998.18.5.19.

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39

Del Prado, Martinez, Jose Andres Meana, and Jose Rafael Carrión. "Acute Cerebrovascular Accident after Treatment with Cisplatin." Acta Oncologica 31, no. 5 (January 1992): 593–95. http://dx.doi.org/10.3109/02841869209088314.

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40

Cooke, Mathew, Michael A. Cuddy, Brad Farr, and Paul A. Moore. "Cerebrovascular Accident Under Anesthesia During Dental Surgery." Anesthesia Progress 61, no. 2 (June 1, 2014): 73–77. http://dx.doi.org/10.2344/0003-3006-61.2.73.

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Abstract Stroke, or cerebrovascular accident (CVA), is a medical emergency that may lead to permanent neurological damage, complications, and death. The rapid loss of brain function due to disruption of the blood supply to the brain is caused by blockage (thrombosis, arterial embolism) or hemorrhage. The incidence of CVA during anesthesia for noncardiac nonvascular surgery is as high as 1% depending on risk factors. Comprehensive preoperative assessment and good perioperative management may prevent a CVA. However, should an ischemic event occur, appropriate and rapid management is necessary to minimize the deleterious effects caused to the patient. This case report describes a patient who had an ischemic CVA while under general anesthesia for dental alveolar surgery and discusses the anesthesia management.

41

Uhumwangho, OdarosaM, OsayemJ Olubor, and AfekhideE Omoti. "Retinal abnormalities in patients with cerebrovascular accident." African Journal of Medical and Health Sciences 14, no. 2 (2015): 105. http://dx.doi.org/10.4103/2384-5589.170172.

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42

Humphrey, Michael. "Sexual Consequences of Cerebrovascular Accident: Discussion Paper." Journal of the Royal Society of Medicine 78, no. 5 (May 1985): 388–90. http://dx.doi.org/10.1177/014107688507800508.

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43

ELIA, M., R. J. STRATTON, C. HOLDEN, N. MEADOWS, A. MICKLEWRIGHT, C. RUSSELL, D. SCOTT, et al. "Home enteral tube feeding following cerebrovascular accident." Clinical Nutrition 20, no. 1 (February 2001): 27–30. http://dx.doi.org/10.1054/clnu.2000.0146.

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44

Okafor, U. V., E. R. Efetie, I. Onwuekwe, and T. C. Uwaezeoke. "Cerebrovascular accident with quadriplegia following postpartum eclampsia." Acta Anaesthesiologica Scandinavica 50, no. 3 (March 2006): 382–84. http://dx.doi.org/10.1111/j.1399-6576.2006.00920.x.

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45

Rowe, Fiona J., David Wright, Darren Brand, Carole Jackson, Shirley Harrison, Tallat Maan, Claire Scott, et al. "Profile of Gaze Dysfunction following Cerebrovascular Accident." ISRN Ophthalmology 2013 (October 10, 2013): 1–8. http://dx.doi.org/10.1155/2013/264604.

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Aim. To evaluate the profile of ocular gaze abnormalities occurring following stroke. Methods. Prospective multicentre cohort trial. Standardised referral and investigation protocol including assessment of visual acuity, ocular alignment and motility, visual field, and visual perception. Results. 915 patients recruited: mean age 69.18 years (SD 14.19). 498 patients (54%) were diagnosed with ocular motility abnormalities. 207 patients had gaze abnormalities including impaired gaze holding (46), complete gaze palsy (23), horizontal gaze palsy (16), vertical gaze palsy (17), Parinaud’s syndrome (8), INO (20), one and half syndrome (3), saccadic palsy (28), and smooth pursuit palsy (46). These were isolated impairments in 50% of cases and in association with other ocular abnormalities in 50% including impaired convergence, nystagmus, and lid or pupil abnormalities. Areas of brain stroke were frequently the cerebellum, brainstem, and diencephalic areas. Strokes causing gaze dysfunction also involved cortical areas including occipital, parietal, and temporal lobes. Symptoms of diplopia and blurred vision were present in 35%. 37 patients were discharged, 29 referred, and 141 offered review appointments. 107 reviewed patients showed full recovery (4%), partial improvement (66%), and static gaze dysfunction (30%). Conclusions. Gaze dysfunction is common following stroke. Approximately one-third of patients complain of visual symptoms, two thirds show some improvement in ocular motility.

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Moussa, N. A., A. R. Osman, and T. M. Yahya. "Acute Hyponatremic Encephalopathy After a Cerebrovascular Accident." American Journal of the Medical Sciences 316, no. 1 (July 1998): 56–59. http://dx.doi.org/10.1016/s0002-9629(15)40372-6.

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Silva, M. T., and D. J. Spalton. "Patent foramen ovale in occipital cerebrovascular accident." Eye 9, no. 3 (May 1995): 384–85. http://dx.doi.org/10.1038/eye.1995.82.

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&NA;. "Cerebrovascular Accident with Quadriplegia following Postpartum Eclampsia." Obstetric Anesthesia Digest 26, no. 3 (September 2006): 162. http://dx.doi.org/10.1097/00132582-200609000-00068.

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Jafri, Rizwan, Yameen Rashid, and Joehar Hamdan. "Cerebellar Cerebrovascular Accident Secondary to Ulcerative Colitis." American Journal of Gastroenterology 108 (October 2013): S425. http://dx.doi.org/10.14309/00000434-201310001-01437.

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