Готові списки джерел за темами / Brain-derived neurotropic factor

Добірка наукової літератури з теми "Brain-derived neurotropic factor"

Автор: Grafiati
Опубліковано: 10 грудня 2022
Оновлено: 29 січня 2023

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Статті в журналах з теми "Brain-derived neurotropic factor"

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Ibrahim, Abdallah Mohammad, Lalita Chauhan, Aditi Bhardwaj, Anjali Sharma, Faizana Fayaz, Bhumika Kumar, Mohamed Alhashmi, Noora AlHajri, Md Sabir Alam, and Faheem Hyder Pottoo. "Brain-Derived Neurotropic Factor in Neurodegenerative Disorders." Biomedicines 10, no. 5 (May 16, 2022): 1143. http://dx.doi.org/10.3390/biomedicines10051143.

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Globally, neurodegenerative diseases cause a significant degree of disability and distress. Brain-derived neurotrophic factor (BDNF), primarily found in the brain, has a substantial role in the development and maintenance of various nerve roles and is associated with the family of neurotrophins, including neuronal growth factor (NGF), neurotrophin-3 (NT-3) and neurotrophin-4/5 (NT-4/5). BDNF has affinity with tropomyosin receptor kinase B (TrKB), which is found in the brain in large amounts and is expressed in several cells. Several studies have shown that decrease in BDNF causes an imbalance in neuronal functioning and survival. Moreover, BDNF has several important roles, such as improving synaptic plasticity and contributing to long-lasting memory formation. BDNF has been linked to the pathology of the most common neurodegenerative disorders, such as Alzheimer’s and Parkinson’s disease. This review aims to describe recent efforts to understand the connection between the level of BDNF and neurodegenerative diseases. Several studies have shown that a high level of BDNF is associated with a lower risk for developing a neurodegenerative disease.
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Pandit, Mahashweta, Tapan Behl, Monika Sachdeva, and Sandeep Arora. "Role of brain derived neurotropic factor in obesity." Obesity Medicine 17 (March 2020): 100189. http://dx.doi.org/10.1016/j.obmed.2020.100189.

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Buckley, Peter F., Anilkumar Pillai, Denise Evans, Edna Stirewalt, and Sahebarao Mahadik. "Brain derived neurotropic factor in first-episode psychosis." Schizophrenia Research 91, no. 1-3 (March 2007): 1–5. http://dx.doi.org/10.1016/j.schres.2006.12.026.

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Lee, Eugine, Yeon Ik Jeong, Seon Mi Park, Jong Yun Lee, Ji Hye Kim, Sun Woo Park, M. S. Hossein, et al. "Beneficial effects of brain-derived neurotropic factor on in vitro maturation of porcine oocytes." Reproduction 134, no. 3 (September 2007): 405–14. http://dx.doi.org/10.1530/rep-06-0288.

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In an effort to improve the quality ofin vitroproduced porcine embryos, we investigated the effect of brain-derived neurotropic factor (BDNF), a neurotropin family member, onin vitromaturation (IVM) of porcine oocytes. The expression of BDNF and truncated isoforms of its receptor, tyrosine kinase B (TrkB), and p75 common neurotropin receptor was detected in both follicular cells and metaphase-I stage oocytes by RT-PCR. However, mRNA of full-length TrkB was not found in oocytes although it was detected in follicular cells. The expression pattern of BDNF and TrkB was confirmed by immunohistochemistry. Supplementation with BDNF (30 ng/ml) during IVM significantly (P< 0.05) increased the first polar body extrusion and glutathione levels in oocytes, whereas the effect of BDNF on nuclear maturation was diminished when gonadotropin and epidermal growth factor (EGF) were added to the culture media. However, treatment with BDNF (30 ng/ml) along with EGF (10 ng/ml) in the presence of gonadotropin significantly (P< 0.05) increased the developmental competence of oocytes to the blastocyst stage after bothin vitrofertilization (IVF; 29.1% when compared with control, 15.6%) and somatic cell nuclear transfer (SCNT; 13.6% when compared with control, 3%). This appeared to reflect a stimulatory interaction between BDNF and EGF to enhance the cytoplasmic maturation of oocytes to support successful preimplantation development. In conclusion, BDNFenhanced nuclearand cytoplasmic maturation of oocytes by autocrine and/or paracrine signals. Also, when used together with EGF, BDNF increased the developmental potency of embryos after IVF and SCNT, demonstrating an improvedin vitroproduction protocol for porcine oocytes.
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Yang, Miyoung, Changjong Moon, Jinwook Kim, Sueun Lee, Sohi Kang, Sung-Ho Kim, and Jong-Choon Kim. "Brain-derived neurotropic factor and GABAergic transmission in neurodegeneration and neuroregeneration." Neural Regeneration Research 12, no. 10 (2017): 1733. http://dx.doi.org/10.4103/1673-5374.217353.

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Sedlacek, Carly, Ryan T. Wiet, Emily C. Tagesen, Eliott Arroyo, Ellen L. Glickman, and Adam R. Jajtner. "Brain Derived Neurotropic Factor Response To Aerobic Exercise In The Cold." Medicine & Science in Sports & Exercise 52, no. 7S (July 2020): 771. http://dx.doi.org/10.1249/01.mss.0000683604.90282.6d.

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Stranahan, Alexis M., Thiruma V. Arumugam, and Mark P. Mattson. "Lowering Corticosterone Levels Reinstates Hippocampal Brain-Derived Neurotropic Factor and Trkb Expression without Influencing Deficits in Hypothalamic Brain-Derived Neurotropic Factor Expression in Leptin Receptor-Deficient Mice." Neuroendocrinology 93, no. 1 (2011): 58–64. http://dx.doi.org/10.1159/000322808.

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Hidayat, Andri, Mansyur Arief, Andi Wijaya, and Suryani As'ad. "Vascular Endothelial Growth Factor and Brain-Derived Neurotropic Factor Levels in Ischemic Stroke Subject." Indonesian Biomedical Journal 8, no. 2 (August 1, 2016): 115. http://dx.doi.org/10.18585/inabj.v8i2.206.

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BACKGROUND: Vascular endothelial growth factor (VEGF) and brain-derived neurotropic factor (BDNF) present during early neuronal development and play important roles in the process of neurorepairing includes angiogenesis, neurogenesis and neuronal plasticity after ischemic stroke. In this study, we observed VEGF and BDNF levels of subjects with ischemic stroke in different onset time.METHODS: A cross sectional study was designed. Study subjects were 51 ischemic stroke subjects, aged 30-80 years old, recruited from Gatot Subroto Army Central Hospital, Jakarta, Indonesia. Ischemic stroke was diagnosed by neurologist, based on clinical examination and magnetic resonance imaging (MRI) result. Subjects were divided into 3 groups based on onset time of stroke: <7 days (group A), 7-30 days (group B) and >30 days (Group C). VEGF and BDNF levels from serum were measured using lumine Magpix. The data was analyzed for comparison and correlation.RESULTS: VEGF and BDNF levels of group B and C were significantly different with p=0.034 and p=0.007, respectively. Group B had the highest VEGF levels, whereas Group C had the highest BDNF level. VEGF and BDNF levels in each group were not significantly correlated.CONCLUSION: Each stage of time after ischemic stroke has different recovery activities like angiogenesis, neurogenesis and plasticity. Angiogenesis process was optimum in 7-30 days after onset. in more than 30 days onset, Low VEGF with high BDNF have important role in a long period of time after the onset of stroke in the regeneration and repair, such as maintaining neuronal survival and plasticity.KEYWORDS: ischemic stroke, VEGF, BDNF
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Mishchenko, T. A., M. V. Vedunova, E. V. Mitroshina, A. S. Pimashkin, and I. V. Mukhina. "Neurotropic Effect of Brain-Derived Neurotrophic Factor at Different Stages of Dissociated Hippocampal Cultures Development in vitro." Sovremennye tehnologii v medicine 7, no. 3 (September 2015): 47–54. http://dx.doi.org/10.17691/stm2015.7.3.06.

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Mohan, Nithyakalyani, and Anusha Sunder. "A comprehensive evaluation of brain derived neurotropic growth factor gene on Bharatnatyam dancers." Precision Medicine Research 4, no. 2 (2022): 6. http://dx.doi.org/10.53388/pmr20220006.

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Дисертації з теми "Brain-derived neurotropic factor"

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Khundakar, Ahmad Adam. "The effect of antidepressant treatment on brain-derived neurotropic factor expression in the rat hippocampus." Thesis, De Montfort University, 2004. http://hdl.handle.net/2086/13266.

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Agerman, Karin. "Specificity of neurotrophins in the nervous system : a genetic approach to determine receptor engagement by neurotrophins /." Stockholm, 2003. http://diss.kib.ki.se/2004/91-7349-730-4/.

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Linnarsson, Sten. "Neurotrophic factors and neuronal plasticity /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4618-3/.

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Gunther, Erik Christian. "Molecular mechanisms of brain derived neurotrophic factor secretion and action /." Thesis, Connect to this title online; UW restricted, 2000. http://hdl.handle.net/1773/5086.

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Martinez, Humberto Jose. "Nerve growth factor actions on the brain /." Access full-text from WCMC, 1989. http://proquest.umi.com/pqdweb?did=744572291&sid=1&Fmt=2&clientId=8424&RQT=309&VName=PQD.

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Wu, Linyan, and wu0071@flinders edu au. "BRAIN DERIVED NEUROTROPHIC FACTOR TRANSPORT AND PHYSIOLOGICAL SIGNIFICANCE." Flinders University. Medicine, 2007. http://catalogue.flinders.edu.au./local/adt/public/adt-SFU20071204.113001.

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Neurotrophins are important signaling molecules in neuronal survival and differentiation. The precursor forms of neurotrophins (proneurotrophins) are the dominant form of gene products in animals, which are cleaved to generate prodomain and mature neurotrophins, and are sorted to constitutive or regulated secretory pathway and released. Brain-derived neurotrophic factor (BDNF) plays a pivotal role in the brain development and in the pathogenesis of neurological diseases. In Huntington’s disease, the defective transport of BDNF in cortical and striatal neurons and the highly expressed polyQ mutant huntingtin (Htt) result in the degeneration of striatal neurons. The underlying mechanism of BDNF transport and release is remains to be investigated. Current studies were conducted to identify the mechanisms of how BDNF is transported in axons post Golgi trafficking. By using affinity purification and 2D-DIGE assay, we show Huntingtin-associated protein 1 (HAP1) interacts with the prodomain and mature BDNF. The GST pull-down assays have addressed that HAP1 directly binds to the prodomain but not to mature BDNF and this binding is decreased by PolyQ Htt. HAP1 immunoprecipitation shows that less proBDNF is associated with HAP1 in the brain homogenate of Huntington’s disease compared to the control. Co-transfections of HAP1 and BDNF plasmids in PC12 cells show HAP1 is colocalized with proBDNF and the prodomain, but not mature BDNF. ProBDNF was accumulated in the proximal and distal segments of crushed sciatic nerve in wild type mice but not in HAP1-/- mice. The activity-dependent release of the prodomain of BDNF is abolished in HAP1-/- mice. We conclude that HAP1 is the cargo-carrying molecule for proBDNF-containing vesicles and plays an essential role in the transport and release of BDNF in neuronal cells. 20-30% of people have a valine to methionine mutation at codon 66 (Val66Met) in the prodomain BDNF, which results in the retardation of transport and release of BDNF, but the mechanism is not known. Here, GST-pull down assays demonstrate that HAP1 binds Val66Met prodomain with less efficiency than the wild type and PolyQ Htt further reduced the binding, but the PC12 cells colocalization rate is almost the same between wt prodomain/HAP1 and Val66Met prodomain/HAP1, suggesting that the mutation in the prodomain may reduce the release by impairing the cargo-carrying efficiency of HAP1, but the mutation does not disrupt the sorting process. Recent studies have shown that proneurotrophins bind p75NTR and sortilin with high affinity, and trigger apoptosis of neurons in vitro. Here, we show that proBDNF plays a role in the death of axotomized sensory neurons. ProBDNF, p75NTR and sortilin are highly expressed in DRG neurons. The recombinant proBDNF induces the dose-dependent death of PC12 cells and the death activity is completely abolished in the presence of antibodies against the prodomain of BDNF. The exogenous proBDNF enhances the death of axotomized sensory neurons and the antibodies to the prodomain or exogenous sortilin-extracellular domain-Fc fusion molecule reduces the death of axotomized sensory neurons. We conclude that proBDNF induces the death of sensory neurons in neonatal rats and the suppression of endogenous proBDNF rescued the death of axotomized sensory neurons.
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Palm, Kaia. "Regulation of neuronal gene expression /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19980612palm.

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Androutsellis-Theotokis, Andreas. "The release and distribution of brain derived neurotrophic factor in brain." Thesis, Imperial College London, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266203.

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Kawamoto, Yasuhiro. "Immunohistochemical localization of brain-derived neurotrophic factor in adult rat brain." Kyoto University, 1997. http://hdl.handle.net/2433/202181.

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Roeding, Ross L., Marla K. Perna, Elizabeth D. Cummins, Daniel J. Peterson, Matthew I. Palmatier, and Russell W. Brown. "Sex Differences in Adolescent Methylphenidate Sensitization: Effects on Glial Cell-Derived Neurotrophic Factor and Brain-Derived Neurotrophic Factor." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/etsu-works/952.

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This study analyzed sex differences in methylphenidate (MPH) sensitization and corresponding changes in glial cell-derived neurotrophic factor (GDNF) and brain-derived neurotprhic factor protein (BDNF) in adolescent male and female rats. After habituation to a locomotor arena, animals were sensitized to MPH (5mg/kg) or saline from postnatal day (P) 33–49, tested every second day. On P50, one group of animals were injected with saline and behavior assessed for conditioned hyperactivity. Brain tissue was harvested on P51 and analyzed for GDNF protein. A second group of animals was also sensitized to MPH from P33 to 49, and expression of behavioral sensitization was analyzed on a challenge given at P60, and BDNF protein analyzed at P61. Females demonstrated more robust sensitization to MPH than males, but only females given MPH during sensitization demonstrated conditioned hyperactivity. Interestingly, MPH resulted in a significant increase in striatal and accumbal GDNF with no sex differences revealed. Results of the challenge revealed that females sensitized and challenged with MPH demonstrated increased activity compared to all other groups. Regarding BDNF, only males given MPH demonstrated an increase in dorsal striatum, whereas MPH increased accumbal BDNF with no sex differences revealed. A hierarchical regression analysis revealed that behavioral sensitization and the conditioned hyperactivity test were reliable predictors of striatal and accumbal GDNF, whereas sensitization and activity on the challenge were reliable predictors of accumbal BDNF, but had no relationship to striatal BDNF. These data have implications for the role of MPH in addiction and dopamine system plasticity.
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Книги з теми "Brain-derived neurotropic factor"

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Duarte, Carlos B., and Enrico Tongiorgi, eds. Brain-Derived Neurotrophic Factor (BDNF). New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-8970-6.

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2

Khundakar, Ahmad Adam. The effect of antidepressant treatment on brain-derived neurotrophic factor expression in the rat hippocampus. Leicester: De Montfort University, 2004.

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3

Katayama, Yusuke. Prolonged release of brain-derived neurotrophic factor from poly(lactide-co-glycolide) microspheres dispersed within a polyethylene glycol hydrogel. Ottawa: National Library of Canada, 2003.

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Duarte, Carlos B., and Enrico Tongiorgi. Brain-Derived Neurotrophic Factor (BDNF). Springer New York, 2019.

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5

Duman, Ronald S. Neurotrophic Mechanisms of Depression. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0027.

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Early theories of depression and treatment response were centered on the monoamine neurotransmitters, but more recent work has focused on functional and structural synaptic plasticity and the role of neurotrophic factors, particularly brain derived neurotrophic factor (BDNF). Neurotrophic factors regulate all aspects of neuronal function, including adaptive plasticity, synapse formation, and neuronal survival. Chronic stress and depression cause reductions in levels of BDNF and other key factors, including vascular endothelial growth factor (VEGF) and fibroblast growth factor 2 (FGF2), in cortical regions that contribute to atrophy and loss of neurons observed in depressed patients and rodent stress models. In contrast, these neurotrophic factors are upregulated by chronic administration of typical antidepressants and are required for antidepressant responses. Moreover, fast acting, highly efficacious antidepressant agents such as ketamine rapidly increase BDNF release and synapse formation, paving the way for a new generation of medications for the treatment of depression.
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Strauss, John. Molecular genetic investigation of brain-derived neurotrophic factor in childhood-onset mood disorder. 2005.

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Strauss, John. Molecular genetic investigation of brain-derived neurotrophic factor in childhood-onset mood disorder. 2005.

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8

Brain-Derived Neurotrophic Factor: Therapeutic Approaches, Role in Neuronal Development and Effects on Cognitive Health. Nova Science Publishers, Incorporated, 2015.

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Poretti, Andrea, and Michael V. Johnston. Genetic Disorders and Stroke. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0110.

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A variety of monogenic and polygenic genetic disorders have been linked to stroke, making it important for the clinician to keep up with the new discoveries and the potential to provide new gene-based therapies. Hematologic disorders such as sickle cell disease and thrombophilia due to mutations in prothrombin, factor V Leiden, and homocysteine metabolism are fairly well known, but mutations in mitochondrial metabolism and matrix metalloproteinases are less recognized. In addition, results of genome-wide association studies (GWAS) in stroke populations are revealing mutations that could predispose to stroke in specific ethnic populations. These studies are also revealing some crossover in mutations between stroke and familial hemiplegic migraine as well as mutations in growth factors such as brain derived neurotrophic factor (BDNF) that appear to influence the recovery from stroke by altering cortical plasticity.
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Razzoli, Maria, Alessandro Bartolomucci, and Valeria Carola. Gene-by-Environment Mouse Models for Mood Disorders. Edited by Turhan Canli. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.013.

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Much of the impact of genes on mood disorders likely depends on interactions between genes and the environment. Recent studies demonstrating an interaction between specific genes and life stressful events (early and/or adult) in the modulation of several mood disorders (e.g., serotonin transporter and brain-derived neurotrophic factor genes) have compelled researchers to incorporate information about adverse environmental experiences into the study of genetic risk factors; these same gene-by-environment (G×E) interactions have been identified in mouse models. Notably, G×E not yet described in humans (e.g., serotonin 1A receptor gene) have been uncovered, providing helpful indications to discover similar interactions in humans. Accurate knowledge of the modality of expression of gene-by-stress interaction may help design prevention protocols aimed at identifying susceptibility to mood disorders on the basis of genetic predisposition and exposure to environmental stressful conditions, thus providing patients with appropriate pharmacological and psychological support.
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Частини книг з теми "Brain-derived neurotropic factor"

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Katz, D. M. "Brain-Derived Neurotrophic Factor and Rett Syndrome." In Neurotrophic Factors, 481–95. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-45106-5_18.

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Tyrer, Peter J., Mark Slifstein, Joris C. Verster, Kim Fromme, Amee B. Patel, Britta Hahn, Christer Allgulander, et al. "Brain-Derived Neurotrophic Factor." In Encyclopedia of Psychopharmacology, 247–50. Berlin, Heidelberg: Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_388.

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Middlemas, David S., and David B. Bylund. "Brain-Derived Neurotrophic Factor." In Encyclopedia of Psychopharmacology, 310–13. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-36172-2_388.

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Middlemas, David S., and David B. Bylund. "Brain-Derived Neurotrophic Factor." In Encyclopedia of Psychopharmacology, 1–5. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27772-6_388-2.

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Vatanashevanopakorn, Chinnavuth, Amit Grover, Arup R. Nath, Kevin Clark, Paul Sopp, Claus Nerlov, and Liliana Minichiello. "Studying BDNF/TrkB Signaling: Transcriptome Analysis from a Limited Number of Purified Adult or Aged Murine Brain Neurons." In Brain-Derived Neurotrophic Factor (BDNF), 55–76. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/7657_2017_3.

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Nath, Arup R., Roy Drissen, Fei Guo, Claus Nerlov, and Liliana Minichiello. "Studying BDNF/TrkB Signaling: High-Throughput Microfluidic Gene Expression Analysis from Rare or Limited Samples of Adult and Aged Central Neurons." In Brain-Derived Neurotrophic Factor (BDNF), 77–86. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/7657_2017_4.

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Salio, Chiara, and Adalberto Merighi. "Ultrastructural Localization of BDNF and trkB Receptors." In Brain-Derived Neurotrophic Factor (BDNF), 133–48. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/7657_2017_5.

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Gomes, João R., Andrea Lobo, Carlos B. Duarte, and Mário Grãos. "BDNF-Induced Intracellular Signaling." In Brain-Derived Neurotrophic Factor (BDNF), 161–83. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/7657_2017_6.

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Panja, Debabrata, and Clive R. Bramham. "BDNF Function in Long-Term Synaptic Plasticity in the Dentate Gyrus In Vivo: Methods for Local Drug Delivery and Biochemical Analysis of Translation." In Brain-Derived Neurotrophic Factor (BDNF), 241–56. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/7657_2017_7.

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Jaanson, Kaur, Angela Pärn, and Tõnis Timmusk. "Usage of Bacterial Artificial Chromosomes for Studying BDNF Gene Regulation in Primary Cultures of Cortical Neurons and Astrocytes." In Brain-Derived Neurotrophic Factor (BDNF), 13–25. New York, NY: Springer New York, 2018. http://dx.doi.org/10.1007/7657_2018_10.

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Тези доповідей конференцій з теми "Brain-derived neurotropic factor"

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Siahaan, Andre Marolop Pangihutan, Iskandar Japardi, and Wismaji Sadewo. "Turmeric Extract Administration Increases the Expression of Brain Derived Neurotropic Factor Following Repetitive Traumatic Brain Injuries." In The 2nd International Conference on Tropical Medicine and Infectious Disease. SCITEPRESS - Science and Technology Publications, 2019. http://dx.doi.org/10.5220/0009841200150018.

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Plinta, Klaudia, Krzysztof Pawlicki, Michał Morek, Edyta Bogunia, Andrzej Plewka, and Monika Rudzińska-Bar. "D11 Plasma brain-derived neurotrophic factor level as huntington disease severity biomarker." In EHDN 2018 Plenary Meeting, Vienna, Austria, Programme and Abstracts. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/jnnp-2018-ehdn.93.

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Hartman, William R., Farah Yusuf, Lucas W. Meuchel, Christina M. Pabelick, and Y. S. Prakash. "Brain Derived Neurotrophic Factor In Proliferation Of Human Pulmonary Artery Smooth Muscle." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a3449.

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4

Xia, Zongxin, Hong Yang, Fei Li, Shulai Zhu, and Ying Xing. "The research of targeted liposome embedding brain-derived neurotrophic factor through the blood–brain barrier." In International Conference on Modern Engineering Soultions for the Industry. Southampton, UK: WIT Press, 2014. http://dx.doi.org/10.2495/mesi140972.

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5

Britt, R. D., M. Thompson, S. A. Wicher, B. Yang, S. Sasse, A. N. Gerber, C. M. Pabelick, and Y. S. Prakash. "Dexamethasone Reduces Effects of Brain-Derived Neurotrophic Factor on Human Airway Smooth Muscle." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a4286.

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6

Lam, CT, ZF Yang, ST Fan, and RTP Poon. "Abstract 1305: The proangiogenic role of brain-derived neurotrophic factor in tumor development." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-1305.

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7

Hartman, William R., Lucas W. Meuchel, Dan F. Smelter, Michael A. Thompson, Christina M. Pabelick, and Y. S. Prakash. "Brain Derived Neurotrophic Factor Prevents Apoptosis In Human Pulmonary Artery Smooth Muscle Cells." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a3400.

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Abcejo, Amard J., Sathish Venkatachalem, Bharathi Aravamudan, Lucas Meuchel, Michael A. Thompson, Christina Pabelick, and Y. S. Prakash. "TrkB Mediated Brain-Derived Neurotrophic Factor (BDNF) Effects On Human Airway Smooth Muscle." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4142.

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Cardenas, Silvia, Kathryn Moffett, Maple Landvoigt, Usha Phillips, Linda Baer, Lennie J. Samsell, and Giovanni Piedimonte. "Serum Nerve Growth Factor And Brain-Derived Neurotrophic Factor As Biomarkers Of Cystic Fibrosis Pulmonary Exacerbation." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a5258.

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Popović, Nataša, Vesna Stojiljković, Snežana Pejić, Ana Todorović, Ivan Pavlović, and Snežana B. Pajović and Ljubica Gavrilović. "INTERRELATIONSHIP OF PREFRONTAL BRAIN-DERIVED NEUROTROPHIC FACTOR AND NEUROENDOCRINE SYSTEM DURING CHRONIC RESTRAINT STRESS." In RAP Conference. Sievert Association, 2020. http://dx.doi.org/10.37392/rapproc.2019.44.

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Звіти організацій з теми "Brain-derived neurotropic factor"

1

Hicks, Ramona R. Brain-Derived Neurotrophic Factor (BDNF) and Traumatic Brain Injury (Head and Spinal). Fort Belvoir, VA: Defense Technical Information Center, January 2000. http://dx.doi.org/10.21236/ada375796.

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2

Zhou, Bojun, Zhisheng Wang, Timon Chengyi Liu, Yuan Wei, and Bing Li. Effects of Different Physical Activity on Brain-Derived Neurotrophic Factor: A Systematic Review and Bayesian Network Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, May 2022. http://dx.doi.org/10.37766/inplasy2022.5.0164.

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