Статті в журналах з теми "Body core temperature"

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1

Heikens, Marc J., Alexander M. Gorbach, Henry S. Eden, David M. Savastano, Kong Y. Chen, Monica C. Skarulis, and Jack A. Yanovski. "Core body temperature in obesity." American Journal of Clinical Nutrition 93, no. 5 (March 2, 2011): 963–67. http://dx.doi.org/10.3945/ajcn.110.006270.

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2

Kurosaka, Chie, Takashi Maruyama, Shimpei Yamada, Yuriko Hachiya, Yoichi Ueta, and Toshiaki Higashi. "Estimating core body temperature using electrocardiogram signals." PLOS ONE 17, no. 6 (June 28, 2022): e0270626. http://dx.doi.org/10.1371/journal.pone.0270626.

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Suppressing the elevation in core body temperature is an important factor in preventing heatstroke. However, there is still no non-invasive method to sense core body temperature. This study proposed an algorithm that estimates core body temperature based on electrocardiogram signals. A total of 12 healthy men (mean age ± SD = 39.6 ± 13.4) performed an ergometric exercise load test under two conditions of exercise load in an environmental chamber adjusted to a temperature of 35°C and humidity of 50%. Vital sensing data such as electrocardiograms, core body temperatures, and body surface temperatures were continuously measured, and physical data such as body weight were obtained from participants pre- and post-experiment. According to basic physiological knowledge, heart rate and body temperature are closely related. We analyzed the relationship between core body temperature and several indexes obtained from electrocardiograms and found that the amount of change in core body temperature had a strong relationship with analyzed data from electrocardiograms. Based on these findings, we developed the amount of change in core body temperature estimation model using multiple regression analysis including the Poincaré plot index of the ECG R-R interval. The estimation model showed an average estimation error of -0.007°C (average error rate = -0.02%) and an error range of 0.457–0.445°C. It is suggested that continuous core body temperature change can be estimated using electrocardiogram signals regardless of individual characteristics such as age and physique. Based on this applicable estimation model, we plan to enhance estimation accuracy and further verify efficacy by considering clothing and environmental conditions.
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3

Green, Angela R., Richard S. Gates, and Laurie M. Lawrence. "Measurement of horse core body temperature." Journal of Thermal Biology 30, no. 5 (July 2005): 370–77. http://dx.doi.org/10.1016/j.jtherbio.2005.03.003.

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4

Lenhardt, Rainer, and Daniel I. Sessler. "Estimation of Mean Body Temperature from Mean Skin and Core Temperature." Anesthesiology 105, no. 6 (December 1, 2006): 1117–21. http://dx.doi.org/10.1097/00000542-200612000-00011.

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Background Mean body temperature (MBT) is the mass-weighted average temperature of body tissues. Core temperature is easy to measure, but direct measurement of peripheral tissue temperature is painful and risky and requires complex calculations. Alternatively MBT can be estimated from core and mean skin temperatures with a formula proposed by Burton in 1935: MBT = 0.64 x TCore + 0.36 x TSkin. This formula remains widely used, but has not been validated in the perioperative period and seems unlikely to remain accurate in dynamic perioperative conditions such as cardiopulmonary bypass. Therefore, the authors tested the hypothesis that MBT, as estimated with Burton's formula, poorly estimates measured MBT at a temperature range between 18 degrees and 36.5 degrees C. Methods The authors reevaluated four of their previously published studies in which core and mass-weighted mean peripheral tissue temperatures were measured in patients undergoing substantial thermal perturbations. Peripheral compartment temperatures were estimated using fourth-order regression and integration over volume from 18 intramuscular needle thermocouples, 9 skin temperatures, and "deep" hand and foot temperature. MBT was determined from mass-weighted average of core and peripheral tissue temperatures and estimated from core temperature and mean skin temperature (15 area-weighted sites) using Burton's formula. Results Nine hundred thirteen data pairs from 44 study subjects were included in the analysis. Measured MBT ranged from 18 degrees to 36.5 degrees C. There was a remarkably good relation between measured and estimated MBT: MBTmeasured = 0.94 x MBTestimated + 2.15, r = 0.98. Differences between the estimated and measured values averaged -0.09 degrees +/- 0.42 degrees C. Conclusions The authors concluded that estimation of MBT from mean skin and core temperatures is generally accurate and precise.
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5

Salerian, Alen J., and Nansen G. Saleri. "Cooling Core Body Temperature May Slow Down Neurodegeneration." CNS Spectrums 13, no. 3 (March 2008): 227–29. http://dx.doi.org/10.1017/s1092852900028479.

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ABSTRACTReduction of core body temperature has been proposed to contribute to the increased lifespan and the anti-aging effects conferred by caloric restriction in mice and higher primates. Cooler biologically compatible core body temperatures have also been hypothesized to combat neurodegenerative disorders. Yet, validation of these hypotheses has been difficult until recently, when it demonstrated that transgenic mice engineered to have chronic low core body temperature have longer lifespan independent of alteration in diet or caloric restriction. This article reviews the literature and highlights the potential influence of core body temperature's governing role on aging and in the pathophysiology of neurodegenerative disorders in humans. What makes recent findings more significant for humans is the existence of several methods to lower and maintain low core body temperatures in human subjects. The therapeutic potential of “cooler people” may also raise the possibility that this could reverse the adverse-health consequences of elevations in core body temperature.
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6

Chen, Anming, Jia Zhu, Qunxiong Lin, and Weiqiang Liu. "A Comparative Study of Forehead Temperature and Core Body Temperature under Varying Ambient Temperature Conditions." International Journal of Environmental Research and Public Health 19, no. 23 (November 29, 2022): 15883. http://dx.doi.org/10.3390/ijerph192315883.

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When the ambient temperature, in which a person is situated, fluctuates, the body’s surface temperature will alter proportionally. However, the body’s core temperature will remain relatively steady. Consequently, using body surface temperature to characterize the core body temperature of the human body in varied situations is still highly inaccurate. This research aims to investigate and establish the link between human body surface temperature and core body temperature in a variety of ambient conditions, as well as the associated conversion curves. Methods: Plan an experiment to measure temperature over a thousand times in order to get the corresponding data for human forehead, axillary, and oral temperatures at varying ambient temperatures (14–32 °C). Utilize the axillary and oral temperatures as the core body temperature standards or the control group to investigate the new approach’s accuracy, sensitivity, and specificity for detecting fever/non-fever conditions and the forehead temperature as the experimental group. Analyze the statistical connection, data correlation, and agreement between the forehead temperature and the core body temperature. Results: A total of 1080 tests measuring body temperature were conducted on healthy adults. The average axillary temperature was (36.7 ± 0.41) °C, the average oral temperature was (36.7 ± 0.33) °C, and the average forehead temperature was (36.2 ± 0.30) °C as a result of the shift in ambient temperature. The forehead temperature was 0.5 °C lower than the average of the axillary and oral temperatures. The Pearson correlation coefficient between axillary and oral temperatures was 0.41 (95% CI, 0.28–0.52), between axillary and forehead temperatures was 0.07 (95% CI, −0.07–0.22), and between oral and forehead temperatures was 0.26 (95% CI, 0.11–0.39). The mean differences between the axillary temperature and the oral temperature, the oral temperature and the forehead temperature, and the axillary temperature and the forehead temperature were −0.08 °C, 0.49 °C, and 0.42 °C, respectively, according to a Bland-Altman analysis. Finally, the regression analysis revealed that there was a linear association between the axillary temperature and the forehead temperature, as well as the oral temperature and the forehead temperature due to the change in ambient temperature. Conclusion: The changes in ambient temperature have a substantial impact on the temperature of the forehead. There are significant differences between the forehead and axillary temperatures, as well as the forehead and oral temperatures, when the ambient temperature is low. As the ambient temperature rises, the forehead temperature tends to progressively converge with the axillary and oral temperatures. In clinical or daily applications, it is not advised to utilize the forehead temperature derived from an uncorrected infrared thermometer as the foundation for a body temperature screening in public venues such as hospital outpatient clinics, shopping malls, airports, and train stations.
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7

Srirangapatanam, Sudarshan, Scott Wiener, and Marshall L. Stoller. "Role of core body temperature in nephrolithiasis." BJU International 126, no. 5 (August 26, 2020): 620–24. http://dx.doi.org/10.1111/bju.15185.

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8

Kumar, Neeraj, Prakash K. Dubey, Amarjeet Kumar, and Veena Singh. "Core body temperature monitoring using Baska airway." Trends in Anaesthesia and Critical Care 25 (April 2019): 36–37. http://dx.doi.org/10.1016/j.tacc.2019.01.003.

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9

Thompson, Daniel P., Perry S. Barboza, John A. Crouse, Thomas J. McDonough, Oriana H. Badajos, and Andrew M. Herberg. "Body temperature patterns vary with day, season, and body condition of moose (Alces alces)." Journal of Mammalogy 100, no. 5 (July 26, 2019): 1466–78. http://dx.doi.org/10.1093/jmammal/gyz119.

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Abstract Variation in core body temperature of mammals is a result of endogenous regulation of heat from metabolism and the environment, which is affected by body size and life history. We studied moose (Alces alces) in Alaska to examine the effects of endogenous and exogenous factors on core body temperature at seasonal and daily time scales. We used a modified vaginal implant transmitter to record core body temperature in adult female moose at 5-min intervals for up to 1 year. Core body temperature in moose showed a seasonal fluctuation, with a greater daily mean core body temperature during the summer (38.2°C, 95% CI = 38.1–38.3°C) than during the winter (37.7°C, 95% CI = 37.6–37.8°C). Daily change in core body temperature was greater in summer (0.92°C, 95% CI = 0.87–0.97°C) than in winter (0.58°C, 95% CI = 0.53–0.63°C). During winter, core body temperature was lower and more variable as body fat decreased among female moose. Ambient temperature and vapor pressure accounted for a large amount of the residual variation (0.06–0.09°C) in core body temperature after accounting for variation attributed to season and individual. Ambient temperature and solar radiation had the greatest effect on the residual variation (0.17–0.20°C) of daily change in core body temperature. Our study suggests that body temperature of adult female moose is influenced by body reserves within seasons and by environmental conditions within days. When studying northern cervids, the influence of season and body condition on daily patterns of body temperature should be considered when evaluating thermal stress.
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10

Słomko, Joanna, Mariusz Kozakiewicz, Jacek J. Klawe, Małgorzata Tafil-Klawe, Piotr Siermontowsk, and Paweł Zalewski. "Circadian Rhythm of Core Body Temperature (Part II): Hyperbaric Environment Influence on Circadian Rhythm of Core Body Temperature." Polish Hyperbaric Research 57, no. 4 (December 1, 2016): 19–25. http://dx.doi.org/10.1515/phr-2016-0022.

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Abstract The aim of this study was to analyse dynamic fluctuations in the circadian rhythm of the core body temperature in healthy adults exposed to conditions in a hyperbaric chamber, using fully objective-telemetric measurement methods. The study group consisted of 13 healthy males (age 32±6.4 years, height 1.85±0.1 m, body weight 84.00±6.3 kg; BMI 24.7±1.2 kg/m2). The core body temperature (CBT) was measured with the Vital Sense telemetry system. The volunteers were placed in a hyperbaric chamber, exposed to compression of 400 kPa, with the exposure plateau of approx. 30 minutes, followed by gradual decompression. The mean core temperature was 36.71°C when registered within 10 minutes before the exposure, 37.20°C during the exposure, 37.27°C one hour after the exposure, 37.36°C 2 hours after the exposure, and 37.42°C three hours after the exposure. The conducted observations show that one-hour stay in a hyperbaric chamber at a depth of 30 m results in an increase in the body temperature, particularly significant after the exposure ends, and maintained for at least 3 hours after the exposure.
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11

Göbel, S., D. Cysarz, and F. Edelhaeuser. "Water temperature affects heart rate and core body temperature during whole body immersion." European Journal of Integrative Medicine 1, no. 4 (December 2009): 256–57. http://dx.doi.org/10.1016/j.eujim.2009.08.066.

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12

Souza, Gladis Aparecida Galindo Reisemberger de, Marcos Leal Brioschi, José Viriato Coelho Vargas, Keli Cristiane Correia Morais, Carlos Dalmaso Neto, and Eduardo Borba Neves. "Reference breast temperature: proposal of an equation." Einstein (São Paulo) 13, no. 4 (December 2015): 518–24. http://dx.doi.org/10.1590/s1679-45082015ao3392.

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ABSTRACT Objective To develop an equation to estimate the breast reference temperature according to the variation of room and core body temperatures. Methods Four asymptomatic women were evaluated for three consecutive menstrual cycles. Using thermography, the temperature of breasts and eyes was measured as indirect reference of core body and room temperatures. To analyze the thermal behavior of the breasts during the cycle, the core body and room temperatures were normalized by means of a mathematical equation. Results We performed 180 observations and the core temperature had the highest correlation with the breast temperature, followed by room temperature. The proposed prediction model could explain 45.3% of the breast temperature variation, with variable room temperature variable; it can be accepted as a way to estimate the reference breast temperature at different room temperatures. Conclusion The average breast temperature in healthy women had a direct relation with the core and room temperature and can be estimated mathematically. It is suggested that an equation could be used in clinical practice to estimate the normal breast reference temperature in young women, regardless of the day of the cycle, therefore assisting in evaluation of anatomical studies.
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13

Jacob, R. H., V. S. M. Surridge, D. T. Beatty, G. E. Gardner, and R. D. Warner. "Grain feeding increases core body temperature of beef cattle." Animal Production Science 54, no. 4 (2014): 444. http://dx.doi.org/10.1071/an13463.

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The core body temperature and post slaughter loin temperatures of steers fed on grass pasture was compared with those of steers fed a grain-based feedlot diet. The feeding treatments were grass for 300 days (Grass), grass for 150 days then feedlot for 150 days (Short Feedlot) and feedlot for 300 days (Long Feedlot). Temperature telemeters were inserted under the peritoneum of the steers and temperature measured at intervals of 1 h for the 300 days, and then at intervals of 1 min for the 48-h period before slaughter. The pH and temperature decline post mortem was also measured. The carcasses of the feedlot steers were heavier and fatter than those from the Grass-fed steers. The core body temperature of the steers from the feedlot treatments was 0.3–0.4°C higher than for the Grass treatment at the time of slaughter. The loin temperature was higher in the feedlot treatments than the Grass treatment at all times measured post mortem as was the temperature at pH 6. Feedlotting can increase the likelihood of ‘high rigor temperature’ conditions of high temperature and low pH occurring in beef carcasses, due to an increase in core body temperature before slaughter, a decrease in the rate of cooling and an increase in the rate of pH decline post mortem. These effects are possibly due to a combination of a direct effect of feed type on body temperature as well as indirect effects on bodyweight and condition score.
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14

Bongers, Coen C. W. G., Thijs M. H. Eijsvogels, Ilse J. W. van Nes, Maria T. E. Hopman, and Dick H. J. Thijssen. "Effects of Cooling During Exercise on Thermoregulatory Responses of Men With Paraplegia." Physical Therapy 96, no. 5 (May 1, 2016): 650–58. http://dx.doi.org/10.2522/ptj.20150266.

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Background People with spinal cord injury (SCI) have an altered afferent input to the thermoregulatory center, resulting in a reduced efferent response (vasomotor control and sweating capacity) below the level of the lesion. Consequently, core body temperature rises more rapidly during exercise in individuals with SCI compared with people who are able-bodied. Cooling strategies may reduce the thermophysiological strain in SCI. Objective The aim of this study was to examine the effects of a cooling vest on the core body temperature response of people with a thoracic SCI during submaximal exercise. Methods Ten men (mean age=44 years, SD=11) with a thoracic lesion (T4–T5 or below) participated in this randomized crossover study. Participants performed two 45-minute exercise bouts at 50% maximal workload (ambient temperature 25°C), with participants randomized to a group wearing a cooling vest or a group wearing no vest (separate days). Core body temperature and skin temperature were continuously measured, and thermal sensation was assessed every 3 minutes. Results Exercise resulted in an increased core body temperature, skin temperature, and thermal sensation, whereas cooling did not affect core body temperature. The cooling vest effectively decreased skin temperature, increased the core-to-trunk skin temperature gradient, and tended to lower thermal sensation compared with the control condition. Limitations The lack of differences in core body temperature among conditions may be a result of the relative moderate ambient temperature in which the exercise was performed. Conclusions Despite effectively lowering skin temperature and increasing the core-to-trunk skin temperature gradient, there was no impact of the cooling vest on the exercise-induced increase in core body temperature in men with low thoracic SCI.
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15

Holland, R. L., J. A. Sayers, W. R. Keatinge, H. M. Davis, and R. Peswani. "Effects of raised body temperature on reasoning, memory, and mood." Journal of Applied Physiology 59, no. 6 (December 1, 1985): 1823–27. http://dx.doi.org/10.1152/jappl.1985.59.6.1823.

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Volunteers' body core temperatures were raised to 38.80–39.05 degrees C within a few minutes by immersion in water at 41 degrees C. Tests were then made with the subjects insulated and cooling slowly. Control immersions were made in water at 37 degrees C when core temperatures remained at 36.60–37.40 degrees C. Neither memory registration nor recall of memories registered an hour earlier, nor immediate ability to recall digit spans forward or backward was affected by the increase in core temperature. The increase in temperature did not have any significant effect on accuracy of performance of verbal logic problems or of two-digit subtractions. However, the increase in core temperature was associated with a significant increase in the speed of performance of the tests, by 11 and 10%, respectively. The warm immersions also induced a significant decrease in alertness and an increase in irritability as assessed subjectively by the volunteers; control immersions had no such effects.
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16

Høiseth, Lars Øivind, Jørgen Melau, Martin Bonnevie-Svendsen, Christoffer Nyborg, Thijs M. H. Eijsvogels, and Jonny Hisdal. "Core Temperature during Cold-Water Triathlon Swimming." Sports 9, no. 6 (June 20, 2021): 87. http://dx.doi.org/10.3390/sports9060087.

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Triathlon and other endurance races have grown in popularity. Although participants are generally fit and presumably healthy, there is measurable morbidity and mortality associated with participation. In triathlon, most deaths occur during the swim leg, and more insight into risk factors, such as hypothermia, is warranted. In this study, we measured the core temperature of 51 participants who ingested temperature sensor capsules before the swim leg of a full-distance triathlon. The water temperature was 14.4–16.4 °C, and the subjects wore wetsuits. One subject with a low body mass index and a long swim time experienced hypothermia (<35 °C). Among the remaining subjects, we found no association between core temperature and swim time, body mass index, or sex. To conclude, the present study indicates that during the swim leg of a full-distance triathlon in water temperatures ≈ 15–16 °C, subjects with a low body mass index and long swim times may be at risk of hypothermia even when wearing wetsuits.
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17

Sookram, Sunil M., Samantha Barker, Karen D. Kelly, William Patton, Terry Sosnowski, Kevin Neilson, and Brian H. Rowe. "Can body temperature be maintained during aeromedical transport?" CJEM 4, no. 03 (May 2002): 172–77. http://dx.doi.org/10.1017/s1481803500006345.

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ABSTRACTBackground:Aeromedical transport in northern areas may be associated with hypothermia. The objective of this study was to determine whether significant hypothermia (core temperature &lt;35ºC) occurs in severely injured or ill intubated patients during transport by rotary wing aircraft.Methods:In this prospective cohort study, all intubated patients over 16 years of age who were transported by rotary wing aircraft from rural hospitals or trauma scenes in northern Alberta to regional hospitals in Edmonton were eligible for study. Esophageal thermometers were used to measure core temperature at 10-minute intervals during transport.Results:Of 133 potentially eligible patients, 116 were enrolled; 69 (59%) had esophageal thermometers inserted, and 47 (41%) had other temperature measurements. Severe hypothermia occurred in only 1% to 2% of cases, but 28% to 39% of patients met criteria for mild hypothermia prior to transport. Core temperatures did not fall during transport, despite the fact that warming techniques were documented in only 38% of cases.Conclusions:During brief (&lt;225 km) rotary wing aeromedical transport of severely injured or ill patients, significant hypothermia is uncommon and body temperature is generally well maintained with the use of simple passive measures. These findings do not justify recommendations for more aggressive core temperature monitoring during this type of aeromedical transport.
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18

Audet, Doris, and Donald W. Thomas. "Evaluation of the accuracy of body temperature measurement using external radio transmitters." Canadian Journal of Zoology 74, no. 9 (September 1, 1996): 1778–81. http://dx.doi.org/10.1139/z96-196.

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The facultative depression of body temperature represents an important energy strategy for small homeotherms. However, measuring body temperature under field conditions by means other than externally attached temperature-sensitive radio transmitters is problematical. We show that skin temperatures measured by external radio transmitters can accurately reflect core temperature for the bat Carollia perspicillata. We compared body and skin temperatures at three ambient temperatures (Ta; 21, 26, and 31 °C). The difference between skin and body temperature (ΔT) was linearly correlated with Ta and can be predicted by ΔT = 4.396 − 0.118Ta. We argue that external temperature-sensitive radio transmitters can provide a reliable index of core temperature and so permit the study of torpor or facultative hypothermia under field conditions.
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19

Yan, QiuYi, Tongyao Shen, Shuilian Yan, Yinglu Kuang, Dingxiu Fan, Wenwen lao, and Xiaqi Lan. "Accurate Monitoring Technology of Core Body Temperature of "yunwentie" Human Body Based on TRIZ Theory." MATEC Web of Conferences 359 (2022): 01003. http://dx.doi.org/10.1051/matecconf/202235901003.

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Novel coronavirus pneumonia is a new type of core body temperature measurement product. Methods using TRIZ theory, the problems of existing temperature measurement methods in China were analyzed. Using the corresponding invention principle, solve the relevant problems of thermometer in clinical use, and put forward innovative solutions. Results according to the innovative design idea of TRIZ theory, it was applied to the new human core temperature measurement product. Novel coronavirus pneumonia is difficult to achieve the current core temperature monitoring of new crown pneumonia epidemic. Conclusion the application of TRIZ theory to the research and development of new human core thermometer products defines the improvement direction of thermometer, and puts forward a feasible scheme for the design and development of new human core thermometer products.
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20

Kim, Keonil, Jisoo Ahn, Kwangyong Yoon, Minjung Ko, Jiyoung Ahn, Hyesung Kim, Jihyeon Park, Chulhyun Lee, Dongwoo Chang, and Sukhoon Oh. "In Vivo Magnetic Resonance Thermometry for Brain and Body Temperature Variations in Canines under General Anesthesia." Sensors 22, no. 11 (May 26, 2022): 4034. http://dx.doi.org/10.3390/s22114034.

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The core body temperature tends to decrease under general anesthesia. Consequently, monitoring the core body temperature during procedures involving general anesthesia is essential to ensure patient safety. In veterinary medicine, rectal temperature is used as an indicator of the core body temperature, owing to the accuracy and convenience of this approach. Some previous studies involving craniotomy reported differences between the brain and core temperatures under general anesthesia. However, noninvasive imaging techniques are required to ascertain this because invasive brain temperature measurements can cause unintended temperature changes by inserting the temperature sensors into the brain or by performing the surgical operations. In this study, we employed in vivo magnetic resonance thermometry to observe the brain temperatures of patients under general anesthesia using the proton resonance frequency shift method. The rectal temperature was also recorded using a fiber optic thermometer during the MR thermometry to compare with the brain temperature changes. When the rectal temperature decreased by 1.4 ± 0.5 °C (mean ± standard deviation), the brain temperature (white matter) decreased by 4.8 ± 0.5 °C. Furthermore, a difference in the temperature reduction of the different types of brain tissue was observed; the reduction in the temperature of white matter exceeded that of gray matter mainly due to the distribution of blood vessels in the gray matter. We also analyzed and interpreted the core temperature changes with the body conditioning scores of subjects to see how the body weight affected the temperature changes.
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21

Yamaoka, Ippei. "Modulation of Core Body Temperature by Amino Acids." Nippon Eiyo Shokuryo Gakkaishi 64, no. 2 (2011): 83–89. http://dx.doi.org/10.4327/jsnfs.64.83.

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22

Vassilieff, Nicolas, Nadia Rosencher, Daniel I. Sessler, Christian Conseiller, and André Lienhart. "Nifedipine and Intraoperative Core Body Temperature in Humans." Anesthesiology 80, no. 1 (January 1, 1994): 123–28. http://dx.doi.org/10.1097/00000542-199401000-00019.

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23

Grahn, D., C. J. Coombs, and H. C. Heller. "BODY CORE TEMPERATURE REDUCTION FOLLOWING EXERCISE-INDUCED HYPERTHERMIA." Medicine & Science in Sports & Exercise 35, Supplement 1 (May 2003): S29. http://dx.doi.org/10.1097/00005768-200305001-00152.

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24

Richmond, Victoria L., Sarah Davey, Katy Griggs, and George Havenith. "Prediction of Core Body Temperature from Multiple Variables." Annals of Occupational Hygiene 59, no. 9 (August 12, 2015): 1168–78. http://dx.doi.org/10.1093/annhyg/mev054.

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25

GÖGENUR, Ismail, Andreas EVERSBUSCH, Michael ACHIAM, Pernille SØLVING, and Jacob ROSENBERG. "Disturbed core body temperature rhythm after major surgery." Sleep and Biological Rhythms 2, no. 3 (October 2004): 226–28. http://dx.doi.org/10.1111/j.1479-8425.2004.00141.x.

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SEVERINSEN and MUNCH. "Body core temperature during food restriction in rats." Acta Physiologica Scandinavica 165, no. 3 (March 1999): 299–305. http://dx.doi.org/10.1046/j.1365-201x.1999.00488.x.

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27

Zalewski, Pawel, Anna Bitner, Joanna Słomko, Justyna Szrajda, Jacek J. Klawe, Malgorzata Tafil-Klawe, and Julia L. Newton. "Whole-body cryostimulation increases parasympathetic outflow and decreases core body temperature." Journal of Thermal Biology 45 (October 2014): 75–80. http://dx.doi.org/10.1016/j.jtherbio.2014.08.001.

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28

Henry, Belinda A., Dominique Blache, Alexandra Rao, Iain J. Clarke, and Shane K. Maloney. "Disparate effects of feeding on core body and adipose tissue temperatures in animals selectively bred for Nervous or Calm temperament." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 299, no. 3 (September 2010): R907—R917. http://dx.doi.org/10.1152/ajpregu.00809.2009.

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In addition to homeostatic regulation of body mass, nonhomeostatic factors impact on energy balance. Herein we describe effects of temperament on adipose and core body temperatures in sheep. Animals were genetically selected for Nervous or Calm traits. We characterized the effects of 1) high- and low-energy intake and maintenance feeding, 2) meal anticipation, and 3) adrenocorticotropin challenge on core body and adipose temperatures. Temperature measurements (5 min) were made using a thermistor inserted into the carotid artery (core body) and a probe in the retroperitoneal fat. An imposed feeding window was used to establish postprandial elevations in temperature. Fat tissue was taken from retroperitoneal and subcutaneous regions for real-time PCR analyses. We demonstrate that innate differences in temperament impact on adipose and core body temperatures in response to various dietary and evocative stimuli. In response to homeostatic cues (low-energy intake and maintenance feeding) core body temperature tended to be higher in Calm compared with Nervous animals. In contrast, in response to nonhomeostatic cues, Nervous animals had higher anticipatory thermogenic responses than Calm animals. Expression of uncoupling protein (UCP)-1 and -2 mRNA were higher in retroperitoneal tissue than in subcutaneous tissue, but UCP3 and leptin mRNA levels were similar at both sites; expression of these genes was similar in Nervous and Calm animals. There were no differences in stress responsiveness. We conclude that temperament differentially influences adipose thermogenesis and the regulation of core body temperature in responses to both homeostatic and nonhomeostatic stimuli.
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29

Mazerolle, Stephanie M., Matthew S. Ganio, Douglas J. Casa, Jakob Vingren, and Jennifer Klau. "Is Oral Temperature an Accurate Measurement of Deep Body Temperature? A Systematic Review." Journal of Athletic Training 46, no. 5 (September 1, 2011): 566–73. http://dx.doi.org/10.4085/1062-6050-46.5.566.

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Context: Oral temperature might not be a valid method to assess core body temperature. However, many clinicians, including athletic trainers, use it rather than criterion standard methods, such as rectal thermometry. Objective: To critically evaluate original research addressing the validity of using oral temperature as a measurement of core body temperature during periods of rest and changing core temperature. Data Sources: In July 2010, we searched the electronic databases PubMed, Scopus, Cumulative Index to Nursing and Allied Health Literature (CINAHL), SPORTDiscus, Academic Search Premier, and the Cochrane Library for the following concepts: core body temperature, oral, and thermometers. Controlled vocabulary was used, when available, as well as key words and variations of those key words. The search was limited to articles focusing on temperature readings and studies involving human participants. Data Synthesis: Original research was reviewed using the Physiotherapy Evidence Database (PEDro). Sixteen studies met the inclusion criteria and subsequently were evaluated by 2 independent reviewers. All 16 were included in the review because they met the minimal PEDro score of 4 points (of 10 possible points), with all but 2 scoring 5 points. A critical review of these studies indicated a disparity between oral and criterion standard temperature methods (eg, rectal and esophageal) specifically as the temperature increased. The difference was −0.50°C ± 0.31°C at rest and −0.58°C ± 0.75°C during a nonsteady state. Conclusions: Evidence suggests that, regardless of whether the assessment is recorded at rest or during periods of changing core temperature, oral temperature is an unsuitable diagnostic tool for determining body temperature because many measures demonstrated differences greater than the predetermined validity threshold of 0.27°C (0.5°F). In addition, the differences were greatest at the highest rectal temperatures. Oral temperature cannot accurately reflect core body temperature, probably because it is influenced by factors such as ambient air temperature, probe placement, and ingestion of fluids. Any reliance on oral temperature in an emergency, such as exertional heat stroke, might grossly underestimate temperature and delay proper diagnosis and treatment.
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30

Zhang, Yanyan, Guiying Liu, and Ling Tang. "Research progress in core body temperature measurement during target temperature management." Journal of Integrative Nursing 4, no. 1 (2022): 36. http://dx.doi.org/10.4103/jin.jin_40_21.

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31

Vongraven, Dag, Morten Ekker, Arild R. Espelien, and Frode J. Aarvik. "Postmortem body temperatures in the minke whale, Balaenoptera acutorostrata." Canadian Journal of Zoology 68, no. 1 (January 1, 1990): 140–43. http://dx.doi.org/10.1139/z90-020.

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Postmortem temperature regimes were measured in 11 minke whales, Balaenoptera acutorostrata, at tissue depths varying from 1.5 (in the blubber) to 30 cm, at two different sites on the whales' sides, one in the flipper region (site A) and one in the dorsal fin region (site B). Body temperatures of instantaneously killed whales were assumed to represent those of living animals. Core body temperatures were 35.0 °C at site A and 35.6 °C at site B. Core body temperature and the size of the thermal core were affected by blubber thickness and the time between harpoon strike and death, but were not influenced by duration of pursuit prior to harpooning. Both intra- and inter-specific comparisons reveal that the thickness of the blubber layer is important for the maintenance of thermal gradients and, thereby, for heat conservation.
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32

Limbaugh, Jayme D., Gregory S. Wimer, Lynn H. Long, and William H. Baird. "Body Fatness, Body Core Temperature, and Heat Loss During Moderate-Intensity Exercise." Aviation, Space, and Environmental Medicine 84, no. 11 (November 1, 2013): 1153–58. http://dx.doi.org/10.3357/asem.3627.2013.

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33

Kang, Hyungsuk, Rebeka R. Zsoldos, Solomon M. Woldeyohannes, John B. Gaughan, and Albert Sole Guitart. "The Use of Percutaneous Thermal Sensing Microchips for Body Temperature Measurements in Horses Prior to, during and after Treadmill Exercise." Animals 10, no. 12 (December 2, 2020): 2274. http://dx.doi.org/10.3390/ani10122274.

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Accurately measuring body temperature in horses will improve the management of horses suffering from or being at risk of developing postrace exertional heat illness. PTSM has the potential for measuring body temperature accurately, safely, rapidly, and noninvasively. This study was undertaken to investigate the relation between the core body temperature and PTSM temperatures prior to, during, and immediately after exercise. The microchips were implanted into the nuchal ligament, the right splenius, gluteal, and pectoral muscles, and these locations were then compared with the central venous temperature, which is considered to be the “gold standard” for assessing core body temperature. The changes in temperature of each implant in the horses were evaluated in each phase (prior to, during, and immediately postexercise) and combining all phases. There were strong positive correlations ranging from 0.82 to 0.94 (p < 0.001) of all the muscle sites with the central venous temperature when combining all the phases. Additionally, during the whole period, PTSM had narrow limits of agreement (LOA) with central venous temperature, which inferred that PTSM is essentially equivalent in measuring horse body temperature. Overall, the pectoral PTSM provided a valid estimation of the core body temperature.
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34

Schuster, C. J., and D. S. J. Pang. "Forced-air pre-warming prevents peri-anaesthetic hypothermia and shortens recovery in adult rats." Laboratory Animals 52, no. 2 (June 9, 2017): 142–51. http://dx.doi.org/10.1177/0023677217712539.

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General anaesthesia disrupts thermoregulation in mammals, which can cause hypothermia. Decreases in core body temperature of 1℃ cause significant postoperative complications in humans, and peri-anaesthetic hypothermia in mice increases data variability, which can potentially increase animal use. In rats, the impact of different temperature management strategies on the incidence and severity of hypothermia, and the accuracy of different temperature measurement methods, is unknown. Eighteen adult male and female SD rats were block-randomized to one of three treatment groups: no-warming (NW), limited-warming (LW, heat pad during anaesthesia), and pre-warming (PW, warm air exposure before anaesthesia, followed by heat pad). Anaesthesia (isoflurane) duration was for 40 min. Core body temperature (intra-abdominal telemetric temperature capsule) was recorded during anaesthesia and recovery. During anaesthesia, rectal, skin, and tail temperatures were also recorded. In the PW group, core temperature was maintained during anaesthesia and recovery. By contrast, the NW group was hypothermic (11% temperature decrease) during anaesthesia. The LW group showed a decrease in temperature during recovery. Recovery to sternal recumbency was significantly faster in the PW (125 [70–186] s, P = 0.0003) and the LW (188 [169–420] s, P = 0.04) groups than in the NW group (525 [229–652] s). Rectal temperature underestimated core temperature (bias −0.90℃, 95% limits of agreement −0.1 to 1.9℃). Skin and tail temperatures showed wide 95% limits of agreement, spanning 6 to 15℃, respectively. The novel strategy of PW was effective at maintaining core temperature during and after anaesthesia. Rectal temperature provided an acceptable proxy for core body temperature.
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35

Rajek, Angela, Robert Greif, Daniel I. Sessler, James Baumgardner, Sonja Laciny, and Hiva Bastanmehr. "Core Cooling by Central Venous Infusion of Ice-cold (4°C and 20°C) Fluid." Anesthesiology 93, no. 3 (September 1, 2000): 629–37. http://dx.doi.org/10.1097/00000542-200009000-00010.

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Background Central venous infusion of cold fluid may be a useful method of inducing therapeutic hypothermia. The aim of this study was to quantify systemic heat balance and regional distribution of body heat during and after central infusion of cold fluid. Methods The authors studied nine volunteers, each on two separate days. Anesthesia was maintained with use of isoflurane, and on each day 40 ml/kg saline was infused centrally over 30 min. On one day, the fluid was 20 degrees C and on the other it was 4 degrees C. By use of a tympanic membrane probe core (trunk and head) temperature and heat content were evaluated. Peripheral compartment (arm and leg) temperature and heat content were estimated with use of fourth-order regressions and integration over volume from 18 intramuscular thermocouples, nine skin temperatures, and "deep" hand and foot temperature. Oxygen consumption and cutaneous heat flux estimated systemic heat balance. Results After 30-min infusion of 4 degrees C or 20 degrees C fluid, core temperature decreased 2.5 +/- 0.4 degrees C and 1.4 +/- 0.2 degrees C, respectively. This reduction in core temperature was 0.8 degrees C and 0.4 degrees C more than would be expected if the change in body heat content were distributed in proportion to body mass. Reduced core temperature resulted from three factors: (1) 10-20% because cutaneous heat loss exceeded metabolic heat production; (2) 50-55% from the systemic effects of the cold fluid per se; and (3) approximately 30% because the reduction in core heat content remained partially constrained to core tissues. The postinfusion period was associated with a rapid and spontaneous recovery of core temperature. This increase in core temperature was not associated with a peripheral-to-core redistribution of body heat because core temperature remained warmer than peripheral tissues even at the end of the infusion. Instead, it resulted from constraint of metabolic heat to the core thermal compartment. Conclusions Central venous infusion of cold fluid decreases core temperature more than would be expected were the reduction in body heat content proportionately distributed. It thus appears to be an effective method of rapidly inducing therapeutic hypothermia. When the infusion is complete, there is a spontaneous partial recovery in core temperature that facilitates rewarming to normothermia.
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36

Matilda, Ek, Emma Westergaard-Nielsen, and Maria Henricson. "Preoperative peripheral and core temperature: an observational study at a day-surgery unit." British Journal of Nursing 29, no. 3 (February 13, 2020): 160–64. http://dx.doi.org/10.12968/bjon.2020.29.3.160.

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Background: Hypothermia is a common problem in the surgical context and can lead to serious consequences for the patient and increased costs for society. Aims: To study day-surgery patients' peripheral and core temperatures during the preoperative phase. Methods: In total, 50 day-surgery patients participated in the study. Two sets of measurements of temperatures were made: core temperature and peripheral temperatures (two measuring points on the upper body and lower extremities respectively) were measured on arrival at the day-surgery unit, as well as on arrival at the operating theatre. The data were normally distributed and a paired t-test was used for statistical analysis. Findings: Peripheral temperatures had significant changes, with measuring points on the upper body decreasing and measuring points on the lower extremities increasing in temperature. The results show no significant change in core temperature. Conclusion: The measurements show that 28% of the patients were below recommended preoperative temperature on arrival at the operating theatre. Further research is needed to study the development of body temperature perioperatively as well as at what point reheating interventions should be introduced.
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37

Tanaka, Y., D. Matsunaga, T. Tajima, and M. Seyama. "Robust Skin Attachable Sensor for Core Body Temperature Monitoring." IEEE Sensors Journal 21, no. 14 (July 15, 2021): 16118–23. http://dx.doi.org/10.1109/jsen.2021.3075864.

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38

Odabasi, Ersin, and Mustafa Turan. "The importance of body core temperature evaluation in balneotherapy." International Journal of Biometeorology 66, no. 1 (October 8, 2021): 25–33. http://dx.doi.org/10.1007/s00484-021-02201-1.

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39

Catalina, Fernando, Leon Milewich, William Frawley, Vinay Kumar, and Michael Bennett. "Decrease of Core Body Temperature in Mice by Dehydroepiandrosterone." Experimental Biology and Medicine 227, no. 6 (June 2002): 382–88. http://dx.doi.org/10.1177/153537020222700603.

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Dietary dehydroepiandrosterone (DHEA) reduces food intake in mice, and this response is under genetic control. Moreover, both food restriction and DHEA can prevent or ameliorate certain diseases and mediate other biological effects. Mice fed DHEA (0.45% w/w of food) and mice pair-fed to these mice (food restricted) for 8 weeks were tested for changes in body temperature. DHEA was more efficient than food restriction alone in causing hypothermia. DHEA injected intraperitoneally also induced hypothermia that reached a nadir at 1 to 2 hr, and slowly recovered by 20 to 24 hr. This effect was dose dependent (0.5–50 mg). Each mouse strain tested (four) was susceptible to this effect, suggesting that the genetics differ for induction of hypophagia and induction of hypothermia. Because serotonin and dopamine can regulate (decrease) body temperature, we treated mice with haloperidol (dopamine receptor antagonist), 5,7-dihydroxytryptamine (serotonin production inhibitor), or ritanserin (serotonin receptor antagonist) prior to injection of DHEA. All of these agents increased rather than decreased the hypothermic effects of DHEA. DHEA metabolites that are proximate (5-androstene-3β, 17β-diol and androstenedione) or further downstream (estradiol-17β) were much less effective than DHEA in inducing hypothermia. However, the DHEA analog, 16α-chloroepiandrosterone, was as active as DHEA. Thus, DHEA administered parentally seems to act directly on temperature-regulating sites in the body. These results suggest that DHEA induces hypothermia independent of its ability to cause food restriction, to affect serotonin or dopamine functions, or to act via its downstream steroid metabolites.
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40

Gatti, Silvia, Jennifer Beck, Giamila Fantuzzi, Tamas Bartfai, and Charles A. Dinarello. "Effect of interleukin-18 on mouse core body temperature." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 282, no. 3 (March 1, 2002): R702—R709. http://dx.doi.org/10.1152/ajpregu.00393.2001.

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We have studied, using a telemetry system, the pyrogenic properties of recombinant murine interleukin-18 (rmIL-18) injected into the peritoneum of C57BL/6 mice. The effect of IL-18 was compared with the febrile response induced by human IL-1β, lipopolysaccharide (LPS), and recombinant murine interferon-γ (rmIFN-γ). Both IL-1β and LPS induced a febrile response within the first hour after the intraperitoneal injection, whereas rmIL-18 (10–200 μg/kg) and rmIFN-γ (10–150 μg/kg) did not cause significant changes in the core body temperature of mice. Surprisingly, increasing doses of IL-18, injected intraperitoneally 30 min before IL-1β, significantly reduced the IL-1β-induced fever response. In contrast, the same pretreatment with IL-18 did not modify the febrile response induced by LPS. IFN-γ does not seem to play a role in the IL-18-mediated attenuation of IL-1β-induced fever. In fact, there was no elevation of IFN-γ in the serum of mice treated with IL-18, and a pretreatment with IFN-γ did not modify the fever response induced by IL-1β. We conclude that IL-18 is not pyrogenic when injected intraperitoneally in C57BL/6 mice. Furthermore, a pretreatment with IL-18, 30 min before IL-1β, attenuates the febrile response induced by IL-1β.
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41

Liu, Kai, Qingjun Jiang, Li Li, Bo Li, Zhen Yang, Shaowen Qian, Min Li, and Gang Sun. "Impact of Elevated Core Body Temperature on Attention Networks." Cognitive And Behavioral Neurology 28, no. 4 (December 2015): 198–206. http://dx.doi.org/10.1097/wnn.0000000000000078.

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42

Cintron-Colon, Rigo, Kokila Shankar, Manuel Sanchez-Alavez, and Bruno Conti. "Gonadal hormones influence core body temperature during calorie restriction." Temperature 6, no. 2 (April 3, 2019): 158–68. http://dx.doi.org/10.1080/23328940.2019.1607653.

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43

Dollberg, Shaul, Ayelet Rimon, Harry D. Atherton, and Steve B. Hoath. "CONTINUOUS MEASUREMENT OF CORE BODY TEMPERATURE IN PRETERM INFANTS." American Journal of Perinatology Volume 17, Number 05 (2000): 257–64. http://dx.doi.org/10.1055/s-2000-10008.

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44

Uth, Marc-Florian, Jochim Koch, and Frank Sattler. "Body Core Temperature Sensing: Challenges and New Sensor Technologies." Procedia Engineering 168 (2016): 89–92. http://dx.doi.org/10.1016/j.proeng.2016.11.154.

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45

Hamilos, Daniel L., David Nutter, Josh Gershtenson, Daniel P. Redmond, Jeannie D. Di Clementi, Karen B. Schmaling, Barry J. Make, and James F. Jones. "Core body temperature is normal in chronic fatigue syndrome." Biological Psychiatry 43, no. 4 (February 1998): 293–302. http://dx.doi.org/10.1016/s0006-3223(97)83214-3.

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46

Bollani, L., C. Dolci, A. Montaruli, G. Rondini, and F. Carandente. "Temporal Structure of Body Core Temperature in Twin Newborns." Biological Rhythm Research 28, no. 1 (February 1997): 29–35. http://dx.doi.org/10.1076/brhm.28.1.29.12976.

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47

Dunleavy, Kathleen J. "Which core body temperature measurement method is most accurate?" Nursing 40, no. 12 (December 2010): 18–19. http://dx.doi.org/10.1097/01.nurse.0000390678.95642.7f.

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48

Giuffre, Maureen, Tanya Heidenreich, Patricia Carney-Gersten, James A. Dorsch, and Eric Heidenreich. "The relationship between axillary and core body temperature measurements." Applied Nursing Research 3, no. 2 (May 1990): 52–55. http://dx.doi.org/10.1016/s0897-1897(05)80158-2.

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49

Hudson-Davies, R., V. Pocock, R. White, M. Parker, and S. R. Milligan. "Disturbances in core body temperature in RIP140-null mice." Journal of Thermal Biology 34, no. 2 (February 2009): 100–108. http://dx.doi.org/10.1016/j.jtherbio.2008.11.003.

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50

Mazgaoker, Savyon, Itay Ketko, Ran Yanovich, Yuval Heled, and Yoram Epstein. "Measuring core body temperature with a non-invasive sensor." Journal of Thermal Biology 66 (May 2017): 17–20. http://dx.doi.org/10.1016/j.jtherbio.2017.03.007.

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