Статті в журналах: "Blood air"

1

Chute, John P. "Tet2 helps blood cells balance in air." Blood 140, no. 11 (September 15, 2022): 1186–87. http://dx.doi.org/10.1182/blood.2022017532.

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2

Wong, John. "Blood substitutes are gasping for air." Nature Medicine 3, no. 1 (January 1997): 10. http://dx.doi.org/10.1038/nm0197-10.

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3

Seaton, A., A. Soutar, V. Crawford, R. Elton, S. McNerlan, J. Cherrie, M. Watt, R. Agius, and R. Stout. "Particulate air pollution and the blood." Thorax 54, no. 11 (November 1, 1999): 1027–32. http://dx.doi.org/10.1136/thx.54.11.1027.

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4

Manley, Kate, Aman Coonar, Frank Wells, and Marco Scarci. "Blood patch for persistent air leak." Current Opinion in Pulmonary Medicine 18, no. 4 (July 2012): 333–38. http://dx.doi.org/10.1097/mcp.0b013e32835358ca.

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5

Kiessling, Arndt-H., Mahmud Khalil, Ohmed Assaf, Frank Isgro, Kai-U. Kretz, and Werner Saggau. "Blood-Air Interface during Cardiopulmonary Bypass." Asian Cardiovascular and Thoracic Annals 12, no. 3 (September 2004): 198–201. http://dx.doi.org/10.1177/021849230401200304.

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6

Kumawat, Vijay, and Anil Aribandi. "Air Bubbles Produced During Rapid Blood Warming with Inline Blood Warmer Leading to Panic of Air Embolism." Indian Journal of Hematology and Blood Transfusion 33, no. 2 (November 3, 2016): 281–82. http://dx.doi.org/10.1007/s12288-016-0741-4.

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7

Harrison, Greg J. "Infiltration of Blood Vessels into Air Sacs." AAV Today 2, no. 2 (1988): 99. http://dx.doi.org/10.2307/30134423.

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8

Linn, William S., and Henry Gong. "AIR POLLUTION, WEATHER STRESS, AND BLOOD PRESSURE." American Journal of Public Health 91, no. 9 (September 2001): 1345—b—1346. http://dx.doi.org/10.2105/ajph.91.9.1345-b.

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9

Athanassiadi, K., E. Bagaev, and A. Haverich. "Autologous Blood Pleurodesis for Persistent Air Leak." Thoracic and Cardiovascular Surgeon 57, no. 08 (December 2009): 476–79. http://dx.doi.org/10.1055/s-0029-1185913.

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10

Baumgartner, Jill, Yuanxun Zhang, James J. Schauer, Majid Ezzati, Jonathan A. Patz та Leonelo E. Bautista. "Household Air Pollution and Childrenʼs Blood Pressure". Epidemiology 23, № 4 (липень 2012): 641–42. http://dx.doi.org/10.1097/ede.0b013e3182593fa9.

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11

Dubowsky, Sara D., Diane R. Gold, Joel Schwartz, Brent Coull, and Helen Suh. "AIR POLLUTION AND INFLAMMATORY MARKERS IN BLOOD." Epidemiology 15, no. 4 (July 2004): S23. http://dx.doi.org/10.1097/00001648-200407000-00045.

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12

Cheng-Yen Chen, Hsin-Wen Fan, S. P. Kuo, Jenghwa Chang, T. Pedersen, T. J. Mills, and Cheng-Chiu Huang. "Blood Clotting by Low-Temperature Air Plasma." IEEE Transactions on Plasma Science 37, no. 6 (June 2009): 993–99. http://dx.doi.org/10.1109/tps.2009.2016344.

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13

Sega, R. "Ambulatory Blood Pressure in Air Traffic Controllers." American Journal of Hypertension 11, no. 2 (February 1998): 208–12. http://dx.doi.org/10.1016/s0895-7061(97)00321-x.

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14

Brook, Robert D., and Sanjay Rajagopalan. "Particulate matter, air pollution, and blood pressure." Journal of the American Society of Hypertension 3, no. 5 (September 2009): 332–50. http://dx.doi.org/10.1016/j.jash.2009.08.005.

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15

Simionescu, Maya. "Cellular components of the air-blood barrier." Journal of Cellular and Molecular Medicine 5, no. 3 (July 2001): 320–21. http://dx.doi.org/10.1111/j.1582-4934.2001.tb00167.x.

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16

Breen, Peter H., and Aaron Hong. "Beware of Air in the Blood Pump." Anesthesia & Analgesia 91, no. 4 (October 2000): 1038. http://dx.doi.org/10.1097/00000539-200010000-00053.

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17

Brugnone, F., L. Perbellini, L. Romeo, M. Bianchin, A. Tonello, G. Pianalto, D. Zambon, and G. Zanon. "Benzene in environmental air and human blood." International Archives of Occupational and Environmental Health 71, no. 8 (November 16, 1998): 554–59. http://dx.doi.org/10.1007/s004200050323.

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18

Fu, Anchen, Mingyang Chang, Haiyan Zhu, Hongrui Liu, Danhong Wu, and Hulie Zeng. "Air-blood barrier (ABB) on a chip." TrAC Trends in Analytical Chemistry 159 (February 2023): 116919. http://dx.doi.org/10.1016/j.trac.2023.116919.

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19

Rahman, Md Masudur, Md Abdul Wahab, Md Zakir Hossain, Md Muaz Yasin, and Nur Kawser Binte Golam Quddus. "Risk of High Systolic Blood Pressure among the Air Crew in Bangladesh Air Force." Journal of Armed Forces Medical College, Bangladesh 17, no. 1 (February 22, 2022): 35–38. http://dx.doi.org/10.3329/jafmc.v17i1.56719.

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Introduction: Military flight circumstances have been shown to affect Blood Pressure levels. Hypertension is the most severe threat that relates to myocardial infarction, stroke and kidney failure. Exposure to regular flight environment has been shown to Individuals Blood Pressure levels. Objective: To find out the factors associate to presentation of hypertension in air crew in Bangladesh Air force. Materials and Methods: It was a cross-sectional observational study was conducted among 100 Air Force’ pilots of Bangladesh. Data were sort out from medical record files. Participants filled in a survey about demographics, lifestyle factors and dietary habits. Arterial Blood Pressure (BP) was also measured. Multivariate linear regression was implemented. Results: Two patients had history of hypertension among them 2(100.0%) in high systolic blood pressure. Which was statistically significant (p<0.05) between normal and high BP. Patients having age (≥40 years) 2.6 (95% CI 1.1% to 6.1%) times the risk for systolic blood pressure. patients having age (≥30 years) 2.5 (95% CI 0.3% to 22.4%) times the risk for systolic blood pressure. Patients having history of hypertension 1.7 (95% CI 0.01% to 46.3%) times the risk for systolic blood pressure. Patients having Pulse (>80 beats per minute) 3.5 (95% CI 0.2% to 52.8%) times the risk for systolic blood pressure. In multivariate analysis, age, history of hypertension and pulse were not statistically significant. Conclusion: BP value of most of the air crew in Bangladesh Air Force’ were within normal range. Few of BP standards have been reported in the study history of hypertension they have moreover family history of HTN. Contrastingly, there were some overweight and fast food eating while few reported not exercising. JAFMC Bangladesh. Vol 17, No 1 (June) 2021: 35-38

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20

Javaudin, Olivier, A. Baillon, N. Varin, C. Martinaud, T. Pouget, C. Civadier, B. Clavier, and A. Sailliol. "Air-drop blood supply in the French Army." Journal of the Royal Army Medical Corps 164, no. 4 (February 12, 2018): 240–44. http://dx.doi.org/10.1136/jramc-2017-000886.

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BackgroundHaemorrhagic shock remains the leading cause of preventable death in overseas and austere settings. Transfusion of blood components is critical in the management of this kind of injury. For French naval and ground military units, this supply often takes too long considering the short shelf-life of red blood cell concentrates (RBCs) and the limited duration of transport in cooling containers (five to six days). Air-drop supply could be an alternative to overcome these difficulties on the condition that air-drop does not cause damage to blood units.MethodsAfter a period of study and technical development of packaging, four air-drops at medium and high altitudes were performed with an aircraft of the French Air Force. After this, one air-drop was carried out at medium altitude with 10 RBCs and 10 French lyophilised plasma (FLYP). A second air-drop was performed with a soldier carrying one FLYP unit at 12 000 feet. For these air-drops real blood products were used, and quality control testing and temperature monitoring were performed.ResultsThe temperatures inside the containers were within the normal ranges. Visual inspection indicated that transfusion packaging and dumped products did not undergo deterioration. The quality control data on RBCs and FLYP, including haemostasis, suggested no difference before and after air-drop.DiscussionThe operational implementation of the air-drop of blood products seems to be one of the solutions for the supply of blood products in military austere settings or far forward on battlefield, allowing safe and early transfusion.

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21

Baile, E. M., S. Osborne, and P. D. Pare. "Effect of autonomic blockade on tracheobronchial blood flow." Journal of Applied Physiology 62, no. 2 (February 1, 1987): 520–25. http://dx.doi.org/10.1152/jappl.1987.62.2.520.

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Tracheobronchial blood flow increases two to five times in response to cold and warm dry air hyperventilation in anesthetized tracheostomized dogs. In this series of experiments we have attempted to attenuate this increase by blockade of the autonomic nervous system. Four groups of anesthetized, tracheostomized, open-chest dogs were studied. Group 1 (n = 5) were hyperventilated for 30 min with 1) warm humid [approximately 26 degrees C, 100% relative humidity, (rh)] air followed by bilateral vagotomy, 2) warm humid air, 3) cold (-22 degrees C, 0% rh) dry air, and 4) warm humid air. Groups 2, 3, and 4 (n = 3/group) were hyperventilated for 30 min with 1) warm humid (approximately 41 degrees C, 100% rh) air, 2) warm dry (approximately 41 degrees C) air, 3) warm humid air, and 4) warm dry air. Group 2 were controls. Group 3 were given phentolamine, 0.6 mg/kg intravenously, as an alpha-blockade, and group 4 were given propranolol, 1 mg/kg, as a beta-blockade after warm dry air hyperventilation (period 2). Five minutes before the end of each 30-min period of hyperventilation, measurements of vascular pressures, cardiac output, arterial blood gases, and inspired, body, and tracheal temperatures were measured, and differently labeled radioactive microspheres were injected into the left atrium to make separate measurements of airway blood flow. After the last measurements had been made animals were killed and their lungs were excised. Blood flow to the airways and lung parenchyma was calculated.(ABSTRACT TRUNCATED AT 250 WORDS)

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22

Pieter, Marsiline, and B. M. Wara Kushartanti. "Pengaruh air mineral, air berglukosa mineral, susu coklat terhadap hidrasi dan kadar glukosa darah." Jurnal Pedagogi Olahraga dan Kesehatan 3, no. 1 (May 16, 2022): 25–38. http://dx.doi.org/10.21831/jpok.v3i1.18006.

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Penelitian untuk mengungkapkan: (1) pengaruh minuman terhadap status hidrasi dan kadar glukosa darah; (2) pengaruh air mineral, air berglukosa mineral, dan susu cokelat terhadap status hidrasi dan kadar glukosa darah, dan (3) perbedaan pengaruh ketiga minuman terhadap status hidrasi dan kadar glukosa darah. Metode pra eksperimen. Populasi Atlet karate 21 orang. Instrumen BJ urine clinitek analyzer dan kadar glukosa darah gluko DrTm. Teknik analisis data ANOVA satu jalur α = 0,05. Hasil menunjukkan: (1) Ada perbedaan berat badan setelah minum air mineral, air berglukosa mineral, dan susu coklat; (2) Pemberian air mineral setelah minum belum bisa mengembalikan kadar glukosa darah; (3) Ada perbedaan air mineral, air berglukosa mineral, dan susu cokelat dalam mengembalikan status hidrasi dan kadar glukosa darah, yaitu. (a) Air putih belum dapat mengembalikan kadar glukosa atlet; (b) Tidak ada perbedaan antara air berglukosa mineral dan susu cokelat. The impact of mineral water, glucose mineral water, and chocolate milk on hydration and blood glucose levels Abstract: Research to reveal: (1) the effect of drinking on hydration status and blood glucose levels; (2) the effect of mineral water, mineral glucose water, and chocolate milk on hydration status and blood glucose levels, and (3) different effects of the three drinks on hydration status and blood glucose levels. Pre-experimental method. The population of karate athletes is 21 people. BJ instrument clinical urine analyzer and blood glucose levels glucose DrTm. One-way ANOVA data analysis technique = 0.05. The results showed: (1) There was a difference in body weight after drinking mineral water, mineral glucose water, and chocolate milk; (2) Giving mineral water after drinking has not been able to restore blood glucose levels; (3) There are differences in mineral water, mineral glucose water, and chocolate milk in restoring hydration status and blood glucose levels, namely. (a) Water has not been able to restore the athlete's glucose level; (b) There is no difference between mineral glucose water and chocolate milk.

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23

Akçıl, Ali Murat, Merve Hatipoğlu, Levent Cansever, Deniz Sansar, Celal Buğra Sezen, and Mehmet Ali Bedirhan. "Blood pleurodesis for air leak after pulmonary resection." Current Thoracic Surgery 4, no. 2 (2019): 63. http://dx.doi.org/10.26663/cts.2019.00012.

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24

Parsanathan, Rajesh, and Rajaguru Palanichamy. "Air pollution impairs endothelial function and blood pressure." Hypertension Research 45, no. 2 (December 2, 2021): 380–81. http://dx.doi.org/10.1038/s41440-021-00807-x.

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25

Schwartz, Joel. "Air Pollution and Blood Markers of Cardiovascular Risk." Environmental Health Perspectives 109 (June 2001): 405. http://dx.doi.org/10.2307/3434788.

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26

van Rossem, Lenie, Sheryl L. Rifas-Shiman, Steven J. Melly, Itai Kloog, Heike Luttmann-Gibson, Antonella Zanobetti, Brent A. Coull, et al. "Prenatal Air Pollution Exposure and Newborn Blood Pressure." Environmental Health Perspectives 123, no. 4 (April 2015): 353–59. http://dx.doi.org/10.1289/ehp.1307419.

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27

Linden, Jeanne V., Harold S. Kaplan, and Mark T. Murphy. "Fatal Air Embolism Due to Perioperative Blood Recovery." Anesthesia & Analgesia 84, no. 2 (February 1997): 422–26. http://dx.doi.org/10.1097/00000539-199702000-00034.

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28

Schwartz, J. "Air pollution and blood markers of cardiovascular risk." Environmental Health Perspectives 109, suppl 3 (June 2001): 405–9. http://dx.doi.org/10.1289/ehp.01109s3405.

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29

SORENSEN, J., S. TROELSEN, and J. KAALUND. "Changes in Blood Gases During Venous Air Embolism." Survey of Anesthesiology XXXV, no. 2 (April 1991): 81. http://dx.doi.org/10.1097/00132586-199104000-00015.

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30

Linden, Jeanne V., Harold S. Kaplan, and Mark T. Murphy. "Fatal Air Embolism Due to Perioperative Blood Recovery." Anesthesia & Analgesia 84, no. 2 (February 1997): 422–26. http://dx.doi.org/10.1213/00000539-199702000-00034.

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31

Cascio, Wayne E., M. Ian Gilmour, and David B. Peden. "Ambient Air Pollution and Increases in Blood Pressure." Hypertension 66, no. 3 (September 2015): 469–71. http://dx.doi.org/10.1161/hypertensionaha.115.05563.

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32

Gaskins, Joe E., and John W. Goldkrand. "Air contamination in umbilical cord blood gas sampling." American Journal of Obstetrics and Gynecology 171, no. 6 (December 1994): 1546–49. http://dx.doi.org/10.1016/0002-9378(94)90399-9.

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33

Hong, Y. C., J. H. Kim, S. S. Hwang, Y. J. Kim, H. J. Lee, J. S. Ha, H. Kim, J. H. Choi, S. G. Park, and Y. K. Kim. "EFFECTS OF AMBIENT AIR POLLUTION ON BLOOD PRESSURE." Epidemiology 16, no. 5 (September 2005): S77. http://dx.doi.org/10.1097/00001648-200509000-00189.

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34

Elwood, P. C., J. E. J. Gallacher, and C. Toothill. "Air lead, blood lead and travel by car." Environmental Geochemistry and Health 8, no. 3 (September 1986): 68–70. http://dx.doi.org/10.1007/bf02311024.

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35

Ishimatsu, A., N. M. Aguilar, K. Ogawa, Y. Hishida, T. Takeda, S. Oikawa, T. Kanda, and K. K. Huat. "Arterial blood gas levels and cardiovascular function during varying environmental conditions in a mudskipper, periophthalmodon schlosseri." Journal of Experimental Biology 202, no. 13 (July 1, 1999): 1753–62. http://dx.doi.org/10.1242/jeb.202.13.1753.

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Changes in blood gas levels, blood pressure and heart rate were studied in chronically cannulated mudskippers, Periophthalmodon schlosseri, subjected to air exposure (6 h), aquatic hypoxia with access to air (water PO2 <0.9 kPa, 6 h) and forced submersion in normoxic water (12 h) at 30 degrees C. Air exposure did not affect either blood O2 and had little effect on blood CO2 levels, but blood pH increased slightly, but significantly. Blood ammonia concentration was elevated sixfold during air exposure. Aquatic hypoxia caused no significant changes in blood gas levels. When the fish was forcibly submerged, blood O2 saturation decreased rapidly to approximately 30 %. Blood PCO2 and total CO2 also decreased, but blood pH was unaffected by forcible submersion. Air exposure did not affect blood pressure or heart rate. Aquatic hypoxia did not affect blood pressure but transiently increased heart rate. In contrast, forced submersion significantly depressed heart rate throughout the period of submersion, while blood pressure decreased only transiently. Upon emersion, the heart rate immediately increased to above the control level when the fish took its first air breath.

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36

Chuang, Kai-Jen, Yuan-Horng Yan, and Tsun-Jen Cheng. "Effect of Air Pollution on Blood Pressure, Blood Lipids, and Blood Sugar: A Population-Based Approach." Journal of Occupational and Environmental Medicine 52, no. 3 (March 2010): 258–62. http://dx.doi.org/10.1097/jom.0b013e3181ceff7a.

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37

Kang, Yang Jun. "Experimental Investigation of Air Compliance Effect on Measurement of Mechanical Properties of Blood Sample Flowing in Microfluidic Channels." Micromachines 11, no. 5 (April 28, 2020): 460. http://dx.doi.org/10.3390/mi11050460.

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Air compliance has been used effectively to stabilize fluidic instability resulting from a syringe pump. It has also been employed to measure blood viscosity under constant shearing flows. However, due to a longer time delay, it is difficult to quantify the aggregation of red blood cells (RBCs) or blood viscoelasticity. To quantify the mechanical properties of blood samples (blood viscosity, RBC aggregation, and viscoelasticity) effectively, it is necessary to quantify contributions of air compliance to dynamic blood flows in microfluidic channels. In this study, the effect of air compliance on measurement of blood mechanical properties was experimentally quantified with respect to the air cavity in two driving syringes. Under periodic on–off blood flows, three mechanical properties of blood samples were sequentially obtained by quantifying microscopic image intensity (<I>) and interface (α) in a co-flowing channel. Based on a differential equation derived with a fluid circuit model, the time constant was obtained by analyzing the temporal variations of β = 1/(1–α). According to experimental results, the time constant significantly decreased by securing the air cavity in a reference fluid syringe (~0.1 mL). However, the time constant increased substantially by securing the air cavity in a blood sample syringe (~0.1 mL). Given that the air cavity in the blood sample syringe significantly contributed to delaying transient behaviors of blood flows, it hindered the quantification of RBC aggregation and blood viscoelasticity. In addition, it was impossible to obtain the viscosity and time constant when the blood flow rate was not available. Thus, to measure the three aforementioned mechanical properties of blood samples effectively, the air cavity in the blood sample syringe must be minimized (Vair, R = 0). Concerning the air cavity in the reference fluid syringe, it must be sufficiently secured about Vair, R = 0.1 mL for regulating fluidic instability because it does not affect dynamic blood flows.

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38

Mutlu, Gökhan M., Paul J. Bryce, and G. R. Scott Budinger. "Linking air pollution exposure with thrombosis." Blood 118, no. 9 (September 1, 2011): 2636–37. http://dx.doi.org/10.1182/blood-2011-07-367144.

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39

Kuleshov, A. P., A. S. Buchnev, A. A. Drobyshev, O. Yu Esipova, and G. P. Itkin. "Development of a cannula device for gas fraction removal in surgical drains." Russian Journal of Transplantology and Artificial Organs 24, no. 4 (September 1, 2022): 46–53. http://dx.doi.org/10.15825/1995-1191-2022-4-46-53.

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The development of low-traumatic surgical drains aimed at maximum possible separation of blood and air, is an important trend in modern medicine. The objective of this work is to create an inexpensive, user-friendly and low-traumatic dynamic blood aspiration system (DBAS). The system allows effective separation of blood and air when drawing blood from a wound under vacuum conditions required for blood aspiration. The operating principle of the system is to separate liquid and gas fractions of the blood-air mixture by modifying the blood intake cannula. The effect is achieved by applying the principles of centrifugal forces of a rotating blood-air flow combined with Archimedes lift forces.

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40

McKinney-Freeman, Shannon L. "A breath of fresh air for umbilical cord blood." Blood 128, no. 25 (December 22, 2016): 2878–80. http://dx.doi.org/10.1182/blood-2016-11-748202.

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41

Ulenbelt, Paul, Mieke E. G. L. Lumens, Henri M. A. G�ron, and Robert F. M. Herber. "An inverse lead air to lead blood relation: the impact of air-stream helmets." International Archives of Occupational and Environmental Health 63, no. 2 (June 1991): 89–95. http://dx.doi.org/10.1007/bf00379070.

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42

Kupchak, Brian. "Exercise and Air-Travel–Induced Alterations in Blood Hemostasis." Seminars in Thrombosis and Hemostasis 44, no. 08 (October 3, 2018): 756–64. http://dx.doi.org/10.1055/s-0038-1670640.

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AbstractHemostasis is the ability of the body to control blood loss following vascular injury. The process is composed of a complex array of pathways made up of the coagulation and fibrinolytic systems that allow the fluid blood to clot after injury and then the subsequent breakdown of the clot, permitting repair of the injured tissue. Studies to date have shown exercise to be a stimulating factor in both the coagulation and fibrinolytic pathways. Additionally, air travel has been shown to be a risk factor for thrombosis. However, few studies have examined the combination of exercise and air travel on hemostasis, despite documented evidence of venous thrombotic episodes in the sports and endurance (marathon/triathlon) communities. This review summarizes and analyzes the literature with regard to (1) acute and chronic exercise, (2) air travel, and (3) exercise and air travel. In addition, the review examines confounding variables that may contribute to coagulation and strategies to prevent blood clot formation after exercise and during air travel.

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43

Schadt, James C., and Eileen M. Hasser. "Defense reaction alters the response to blood loss in the conscious rabbit." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 280, no. 4 (April 1, 2001): R985—R993. http://dx.doi.org/10.1152/ajpregu.2001.280.4.r985.

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The interaction of sensory stressors with the cardiovascular response to blood loss has not been studied. The cardiovascular response to a stressor (i.e., the defense reaction) includes increased skeletal muscle blood flow and perhaps a reduction in arterial baroreflex function. Arterial pressure maintenance during blood loss requires baroreflex-mediated skeletal muscle vasoconstriction. Therefore, we hypothesized that the defense reaction would limit arterial pressure maintenance during blood loss. Male, New Zealand White rabbits were chronically prepared with arterial and venous catheters and Doppler flow probes. We removed venous blood in conscious rabbits until mean arterial pressure decreased to <40 mmHg. We repeated the experiment with (air) and without (sham) simultaneous exposure to an air jet stressor. Air resulted in a defense reaction (e.g., mean arterial pressure = 94 ± 1 and 67 ± 1 mmHg for air and sham, respectively). Contrary to our hypothesis, air increased the blood loss necessary to produce hypotension (19.3 ± 0.2 vs. 16.9 ± 0.2 ml/kg for sham). Air did not reduce skeletal muscle vasoconstriction during normotensive hemorrhage. However, air did enhance renal vasoconstriction (97 ± 3 and 59 ± 3% of baseline for sham and air, respectively) during the normotensive phase. Thus the defense reaction did not limit but rather extended defense of arterial pressure during hemorrhage.

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44

Brown, Leslie, Daniel P. Davis, Kaitlyn Price, and Laura Smith. "Air Medical Administration of Whole Blood Versus Packed Red Blood Cells for Trauma Patients." Air Medical Journal 41, no. 1 (January 2022): 33. http://dx.doi.org/10.1016/j.amj.2021.11.012.

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Kaneko, Takashi, Pei-Yu Wang, and Akio Sato. "Relationship between blood/air partition coefficients of lipophilic organic solvents and blood triglyceride levels." Toxicology 143, no. 2 (February 2000): 203–8. http://dx.doi.org/10.1016/s0300-483x(99)00170-5.

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Sandermann, Jes, Jan Aagaard, and Preben Lovgreen Nielsen. "Thermocoagulation with Hot Air in Thoracic Surgery." Asian Cardiovascular and Thoracic Annals 3, no. 1 (March 1995): 39–40. http://dx.doi.org/10.1177/021849239500300111.

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Thermocoagulation with hot air is a new hemostatic procedure. During pulmonary surgery oozing blood and air leaking can be controlled by sweeping a high velocity air stream of about 420°C Cover a pulmonary surface stripped of visceral pleura. The hot air thermocoagulator can also control oozing of blood from the epicardium.

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Hasan, Irsa S., Mark S. Allen, Stephen D. Cassivi, William S. Harmsen, Nandita Mahajan, Francis C. Nichols, Janani Reisenauer, Robert K. Shen, Dennis A. Wigle, and Shanda H. Blackmon. "Autologous blood patch pleurodesis for prolonged postoperative air leaks." Journal of Thoracic Disease 13, no. 6 (June 2021): 3347–58. http://dx.doi.org/10.21037/jtd-20-1761.

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Khadzhieva, M. B., A. S. Gracheva, A. V. Ershov, Yu V. Chursinova, V. A. Stepanov, L. S. Avdeikina, O. A. Grebenchikov, et al. "Biomarkers of Air-Blood Barrier Damage In COVID-19." General Reanimatology 17, no. 3 (July 3, 2021): 16–31. http://dx.doi.org/10.15360/1813-9779-2021-3-2-0.

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The search for sensitive and specific markers enabling timely identification of patients with a life-threatening novel coronavirus infection (COVID-19) is important for a successful treatment.The aim of the study was to examine the association of molecular biomarkers of air-blood barrier damage, surfactant proteins SP-A and SP-D and Club cell protein CC16, with the outcome of patients with COVID-19.Materials and methods. A cohort of 109 patients diagnosed with COVID-19 was retrospectively divided into two groups. Group 1 comprised survivor patients discharged from the ICU (w=90). Group 2 included the patients who did not survive (w=19). Association of disease outcome and SP-A, SP-D, and CC16 levels in blood serum, clinical, and laboratory data were examined taking into account the day of illness at the time of biomaterial collection.Results. The non-survivors had higher SP-A (from days 1 to 10 of symptoms onset) and lower CC16 (from days 11 to 20 of symptoms onset) levels vs survivors discharged from ICU. No significant differences in SP-D levels between the groups were found.Conclusion. According to the study results, the surfactant protein SP-A and Club cell protein CC16 are associated with increased COVID-19 mortality.

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BACCARELLI, A., A. ZANOBETTI, I. MARTINELLI, P. GRILLO, L. HOU, S. GIACOMINI, M. BONZINI, et al. "Effects of exposure to air pollution on blood coagulation." Journal of Thrombosis and Haemostasis 5, no. 2 (February 2007): 252–60. http://dx.doi.org/10.1111/j.1538-7836.2007.02300.x.

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van Rossem, Lenie, Steven Melley, Sheryl Rifas-Shiman, Antonella Zanobetti, Brent A. Coull, Joel D. Schwartz, Murray A. Mittleman, et al. "Prenatal Exposure to Air Pollution and Newborn Blood Pressure." ISEE Conference Abstracts 2013, no. 1 (September 19, 2013): 4062. http://dx.doi.org/10.1289/isee.2013.p-2-05-26.

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