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Книги з теми "Bladder disease"

Щоб переглянути інші типи публікацій з цієї теми, перейдіть за посиланням: Bladder disease.

Автор: Grafiati

Опубліковано: 10 грудня 2022

Оновлено: 28 січня 2023

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1

David, Staskin, ed. Atlas of bladder disease. Philadelphia: Current Medicine Group, 2009.

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2

Atala, Anthony, and Debra Slade, eds. Bladder Disease, Part A. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4419-8889-8.

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3

1958-, Atala Anthony, and Slade Debra, eds. Bladder disease: Research concepts and clinical applications. New York: Kluwer Academic/Plenum Publishers, 2003.

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4

Schulze, Richard. Healing kidney disease naturally. Marina del Rey, Calif: Natural Healing Publications, 2003.

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5

Leckey, Joan Lesley. Urinary and tumour markers of disease recurrence and prognosis in transitional cell carcinoma of the bladder. [s.l: The Author], 1997.

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6

New low fat recipes: Nutritionaltherapy for heart disease, weight control, digestive, gall bladder and pancreas disorders. London: Foulsham, 1986.

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7

J, Manyak Michael, and Kahn Leonard B, eds. Bladder biopsy interpretation. New York: Raven Press, 1992.

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8

Wein, Alan J. Overactive bladder in clinical practice. London: Springer, 2007.

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9

Antonio, Lopez-Beltran, and Bostwick David G, eds. Bladder pathology. Hoboken, N.J: Wiley-Blackwell, 2012.

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10

1930-, Mackay Bruce, ed. Atlas of bladder pathology. New York: Igaku-Shoin, 1991.

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11

K, Newman Diane, and Wein Alan J, eds. Fast facts: Bladder Disorders. 2nd ed. Abingdon: HEALTH Press Limited, 2011.

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12

Epstein, Jonathan I. Bladder biopsy interpretation. Philadelphia, PA: Lippincott Williams & Wilkins, 2005.

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13

B, Amin Mahul, and Reuter Victor E, eds. Bladder biopsy interpretation. Philadelphia: Lippincott Williams & Wilkins, 2004.

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14

D, Oliver R. T., and Coptcoat Malcolm J, eds. Bladder cancer. Plainview, N.Y: Cold Spring Harbor Laboratory Press, 1998.

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15

Hunt, Jennifer M. Irritable bladder & incontinence: A natural approach. Berkeley, CA: Ulysses Press, 1998.

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16

Treatment and management of bladder cancer. London: Informa Healthcare, 2008.

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17

1952-, Whitmore Kristene E., ed. Overcoming bladder disorders: Compassionate, authoritative medical and self-help solutions for incontinence, cystitis, interstitial cystitis, prostate problems, and bladder cancer. New York: HarperPerennial, 1991.

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18

Chalker, Rebecca. Overcoming bladder disorders: Compassionate, authoritative medical and self-help solutions for incontinence, cystitis, interstitial cystitis, prostate problems, and bladder cancer. New York: Harper & Row, 1990.

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19

Keshav, Satish, and Alexandra Kent. Gall bladder disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0200.

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The gall bladder is a sac which lies underneath the liver and stores and concentrates bile produced by the liver. As food enters the duodenum, it stimulates the release of cholecystokinin, which in turn stimulates the release of bile, which passes via the cystic duct to the common bile duct, which connects to the duodenum at the sphincter of Oddi. Bile is required in digestion, especially for the emulsification and absorption of fat. Biliary disease can take several forms. Cholelithiasis refers to the presence of gallstones in the gall bladder, whereas choledocholithiasis refers to gallstones in the biliary tree. Cholecystitis is inflammation and infection of the gall bladder. Cholangitis is inflammation and infection of the biliary tree. Sphincter of Oddi dysfunction (SOD) is characterized by symptoms of biliary obstruction, with no structural cause. Other forms of biliary disease are gall bladder polyps, primary biliary cholangitis, and primary sclerosing cholangitis.

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20

Staskin, David. Atlas of Bladder Disease. Current Medicine Group LLC, 2017.

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21

Bladder disease: Research concepts and clinical applications. New York: Kluwer Academic/Plenum Publishers, 2004.

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22

1958-, Atala Anthony, and Slade Debra, eds. Bladder disease: Research concepts and clinical applications. New York: Kluwer Academic/Plenum Publishers, 2004.

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23

Healing Kidney and Bladder Disease Naturally. Natural Healing Publications, 2003.

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24

Atala, Anthony, and Debra Slade. Bladder Disease: Research Concepts and Clinical Applications. Springer, 2013.

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25

Atala, Anthony, and Debra Slade. Bladder Disease: Research Concepts and Clinical Applications. Springer London, Limited, 2012.

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26

(Editor), Anthony Atala, and Debra Slade (Editor), eds. Bladder Disease: Research Concepts and Clinical Applications (Advances in Experimental Medicine and Biology Volume 539 Parts A & B) TWO VOLUME SET. Springer, 2003.

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27

Mayr, Roman, and Maximilian Burger. Squamous cell bladder cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0080.

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In the developed countries, over 90% of the bladder cancer cases are transitional cell carcinoma (TCC), with squamous cell carcinoma (SCC), adenocarcinomas, and rare types of bladder cancer comprising the remaining 10% of bladder cancer cases. In Western regions, pure SCC of the bladder constitutes 1.2–4.5% of all bladder tumours. SCC can occur in both non-bilharzial and bilharzial bladders; the two subtypes differ in epidemiology, pathogenesis, and clinical outcome. Squamous cell carcinoma in the bilharzial bladder is an endemic disease in many regions of the Middle East, Africa, Southeast Asia, and South America. The knowledge of SCC of the bladder is nevertheless important due to different aetiology, clinical pathways, and clinical outcome.

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28

Nesterova, D. V. Treatment of Liver Disease, Kidney, Bladder, Biliary and Urinary Tracts. Book on Demand Ltd., 2018.

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29

Zehnder, Pascal, and George N. Thalmann. Muscle-invasive bladder cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0078.

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In the United Kingdom, >4,000 people die of bladder cancer every year. This reflects around one-third of affected patients and occurs in those with primary metastatic disease, with invasion at presentation, and in persons whose tumour progresses to invasion from non-invasive disease. The outcome from invasive cancers has not dramatically altered over the last 30 years, due to a lack of screening programmes, a lack of advances in treatment, and the fact that many patients present with tumours at an advanced stage. Around 50% of patients with invasive disease die from bladder cancer despite radical treatment, suggesting the disease is metastatic at presentation. Cure is rarely possible in patients with locally advanced tumours and lymph node metastases. Therapeutic options include systemic chemotherapy and salvage radical treatment for responders or palliation. Following radical cystectomy for cancer, patients require lifelong follow-up for both oncologic and functional reasons.

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30

Mano, Roy, and Ofer Yossepowitch. Adenocarcinoma of the bladder. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0081.

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Adenocarcinoma of the bladder accounts for 0.5–2 of bladder tumours. Risk factors include bladder exstrophy, bladder augmentation, schistosomiasis, and endometriosis. Bladder adenocarcinoma is classified as primary, arising from the bladder or urachal remnant, and secondary (metastatic). Most patients present with haematuria and irritative voiding symptoms. On imaging, a typical lesion is commonly located at the bladder dome. Compared to urothelial carcinoma (UC), most adenocarcinomas are diagnosed at high grade and advanced stage. Surgical treatment of localized disease entails partial cystectomy for urachal tumours and radical cystectomy for non-urachal or large urachal adenocarcinoma. The optimal treatment for metastatic disease has yet to be defined. Overall survival rates are 20–70% at 5 years, similar to those for UC, when adjusted for stage and grade. Secondary adenocarcinomas commonly arise from a genitourinary or gastrointestinal origin. Differentiation from primary tumours may be complex. Treatment depends on the prognosis of the primary cancer.

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31

Brinkman, Maree, and Maurice Zeegars. Screening for bladder cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0073.

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Bladder cancer (BC) is one of the most common urological malignancies and ranks ninth among all cancers worldwide. While screening has the potential to detect early cases of BC and reduce disease specific mortality, to date there are no routine screening programmes of asymptomatic individuals conducted anywhere in the world. There are however, a range of tests and procedures available for the detection and subsequent diagnosis of BC for select individuals presenting with urological symptoms and who are at increased risk of the disease.This chapter provides an overview of the traditional screening tools used for the detection of BC, such as urinalysis for haematuria and urinary cytology, as well as a brief description of follow-up procedures including cystoscopy, imaging, and treatment modalities.

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32

Said, Neveen, and Dan Theodorescu. Molecular biology of bladder cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0071.

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Bladder cancer is the most common malignancy involving the urinary system, caused primarily by tobacco use and exposure to industrial chemicals with an estimated 73,510 patients affected and 14,880 deaths in 2012. This chapter will summarize what is known about the most common molecular derangements in human bladder cancer. It will focus on the function and biological/clinical relevance of these genes in models of urothelial cancer and in patients with this disease. It is not meant as a comprehensive review of all the functions of the aforementioned genes in normal physiology or other cancer types. Furthermore, the selection of what genes/pathways are described is by necessity empirical and so we apologize to any author whose work was not described or quoted.

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33

Calabrò, Fabio, and Cora N. Sternberg. Treatment of metastatic bladder cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0079.

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Although bladder cancer is considered a chemosensitive malignancy, the prognosis of patients with metastatic disease is poor, with a median survival of approximately 12–14 months in good prognosis patients and with cure in only a minority. The addition of new drugs to the standard cisplatin-based regimens has not improved these outcomes. In this chapter, we highlight the role of chemotherapy and the impact of the new targeted agents in the treatment of metastatic bladder carcinoma. A better understanding of the underlying biology and the molecular patterns of urothelial bladder cancer has led to clinical investigation of several therapeutic targets. To date, these agents have yet to demonstrate an improvement in overall survival. Urothelial cancer is extremely sensitive to checkpoint inhibition with both anti PD-1 and anti PDL1 antibodies. The future seems brighter with the advent of these new therapies.

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34

FRED, Benard. Vegetarian Diet for Bladder Cancer: The Complete Necessary Guide You Need to Know about Vegetarian Diet Benefit and Uses for Bladder Disease. Independently Published, 2020.

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35

Sandler, Gaye Grissom, Jill Heidi Osborne, and Andrew Sandler. IC 101 - It's Not Just a Bladder Disease: A Complete Guide to Interstitial Cystitis, Bladder Pain Syndrome, Chronic Pelvic Pain & Chronic Overlapping Pain Conditions. Interstitial Cystitis Network, 2021.

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36

Sandler, Gaye Grissom, Jill Heidi Osborne, and Andrew Sandler. IC 101 - It's Not Just a Bladder Disease: A Complete Guide to Interstitial Cystitis, Bladder Pain Syndrome, Chronic Pelvic Pain & Chronic Overlapping Pain Conditions. Interstitial Cystitis Network, 2021.

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37

Bagrodia, Aditya, and Yair Lotan. Low and intermediate risk non-muscle-invasive bladder cancer. Edited by James W. F. Catto. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0076.

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Bladder cancer is a common disease that affects more males than females. Most bladder tumours are histologically typed as urothelial cell carcinoma, and these are best divided into cancers invading the muscularis propria and non-invasive malignancies confined to the bladder. The latter are the majority of cancers and include low risk, indolent cancers that may recur within the bladder but not progress to invasion or metastases, and a proportion that subsequently progress to muscle invasion. The risk of intravesical recurrence or progression to invasion from a non-invasive bladder cancer can be stratified as low, intermediate, and high using various pathological factors (such as tumour grade, stage, size, multiplicity, and the presence of carcinoma in situ). In this chapter, we will give an overview of bladder cancer and focus upon tumours at low or intermediate risk of developing future progression to invasion.

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38

1957-, Chancellor Michael B., and Blaivas Jerry G, eds. Practical neuro-urology: Genitourinary complications in neurologic disease. Boston: Butterworth-Heinemann, 1995.

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39

Watson, Max, Caroline Lucas, Andrew Hoy, and Jo Wells. Genitourinary problems. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199234356.003.0018.

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This chapter focuses on the anatomy and physiology of the bladder and micturition. It covers bladder wall, sphincter active urethra, nerve supply, bladder pain and its treatment, blood supply of the bladder, urinary tract infection (UTI), renal pain, ureteric colic, pelvic pain, urinary retention, ureteric obstruction, urinary incontinence, haematuria, catheterization, genitourinary fistulae, vesicoenteric fistulae, vesicovaginal fistulae, and sexual health in advanced disease.

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40

(Editor), Linda M. Ross, and Peter Dresser (Editor), eds. Kidney and Urinary Tract Diseases and Disorders Sourcebook: Basic Information About Kidney Stones, Urinary Incontinence, Bladder Disease, End Stage Renal ... Statistical and (Health Reference Series). Omnigraphics, 1997.

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41

Panicker, Jalesh N., and Clare J. Fowler. Non-traumatic neurourology. Edited by Christopher R. Chapple. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0046.

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This chapter reviews bladder disturbances in non-traumatic neurological conditions and provides an approach to its evaluation and management. The pattern of bladder dysfunction depends upon the level of neurological localisation and accordingly, lesions can be suprapontine, infrapontine/suprasacral (spinal), or infrasacral. The importance of the frontal lobes for bladder control has been confirmed and vascular disease or tumour can result in incontinence. There is better understanding about the very different urological profile of the two sometimes confused conditions, multiple system atrophy and Parkinson’s disease. Guidelines for the management of lower urinary tract dysfunction in multiple sclerosis are reviewed. Lower urinary tract (LUT) dysfunction is common in neurological disease and its importance to patient health and quality of life is now widely recognized.

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42

Reynard, John, Simon Brewster, and Suzanne Biers. Oxford Handbook of Urology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199696130.001.0001.

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The Oxford Handbook of Urology, Third Edition, covers a wide spectrum of diseases and their treatment in urology, as well as surgical aspects of kidney, bladder, prostate and scrotal disorders, and aims to give a brief overview of many different urological subjects, including urological emergencies, cancers, infections, children's disorders and kidney stone disease

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43

M, Ross Linda, ed. Kidney and urinary tract diseases and disorders sourcebook: Basic information about kidney stones, urinary incontinence, bladder disease, end stage renal disease, dialysis, and more, along with statistical and demographic data and reports on current research initiatives. Detroit, MI: Omnigraphics, 1997.

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44

Bowker, Lesley K., James D. Price, Ku Shah, and Sarah C. Smith. Gastroenterology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738381.003.0012.

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This chapter provides information on the ageing gastrointestinal system, the elderly mouth, nutrition, enteral feeding, the ethics of clinically assisted feeding, oesophageal disease, dysphagia, peptic ulcer disease, the liver and gall bladder, constipation, diverticular disease, inflammatory bowel disease, diarrhoea in older patients, other colonic conditions, the ‘acute surgical abdomen’, obstructed bowel in older patients, and obesity in older people.

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45

Herrington, William G., Aron Chakera, and Christopher A. O’Callaghan. Urinary tract obstruction. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0165.

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The urinary tract can become obstructed by various disease processes, including tumours. Obstruction at any level of the urinary tract can impair the free flow of urine and may be partial or complete, and unilateral or bilateral. Bilateral obstruction usually occurs at the level of the bladder or lower. Retroperitoneal fibrosis and extrinsic compression of both ureters by a malignancy are exceptions. Children are affected by congenital vesicoureteric junction obstruction or pelvi-ureteric junction obstruction. Young adults suffer stone disease. The elderly are prone to urothelial cancers, and older men to bladder outflow obstruction. Retroperitoneal fibrosis is an inflammatory condition that typically affects men over 50 years of age. Diagnosis should be confirmed by biopsy to exclude a lymphoma or malignancy.

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46

Yaqoob, Muhammad M., Katherine Bennett-Richards, and Islam Junaid. The patient with urinary tract obstruction. Edited by Adrian Woolf. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0356.

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Several terms usually describe obstruction of the urinary tract and its consequences such as hydronephrosis, obstructive uropathy, and obstructive nephropathy. Obstruction can be due to anatomical or functional abnormalities of the urethra, bladder, ureter, or renal pelvis. These abnormalities can be congenital or acquired. Obstructive uropathy also can occur during the course of diseases extrinsic to the urinary tract. This is a reversible cause of renal impairment and calls for urgent diagnosis and treatment to prevent chronic irreversible renal scarring and consequent progressive chronic renal disease.

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47

Schreuder, Michiel F. Posterior urethral valves. Edited by Adrian Woolf. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0354.

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Posterior urethral valves is the most common congenital cause of lower urinary tract obstruction in males, and a common cause (15–17%) for end-stage renal disease in childhood. Most commonly, posterior urethral valves is suspected on basis of a screening antenatal ultrasound. Ultrasound will not detect posterior urethral valves itself, but recognizes the consequences of lower urinary tract obstruction with a dilated thick-walled bladder and dilation of the prostatic portion of the urethra. After birth, urine drainage has to be secured by placement of a bladder catheter, and imaging is needed to confirm the presence of the urethral valves and estimate the degree of damage to the kidney. Consequences of posterior urethral valves depend on the degree of renal dysplasia and bladder dysfunction. Prevention or minimization of such consequences by intrauterine urine drainage has not definitively shown a benefit of early vesico-amniotic shunting.

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48

M, Fitzpatrick John, and Krane Robert J. 1943-, eds. The bladder. Edinburgh: Churchill Livingstone, 1995.

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49

Fast Facts Bladder Disorders: Bladder Disorders (Fast Facts). 2nd ed. Not Avail, 2008.

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50

Ku, Ja Hyeon. Bladder Cancer. Elsevier Science & Technology Books, 2017.

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