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Статті в журналах з теми "B cells"

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Автор: Grafiati

Опубліковано: 4 червня 2021

Оновлено: 15 червня 2024

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1

Tangye, Stuart G., and Kim L. Good. "Human IgM+CD27+B Cells: Memory B Cells or “Memory” B Cells?" Journal of Immunology 179, no. 1 (June 19, 2007): 13–19. http://dx.doi.org/10.4049/jimmunol.179.1.13.

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2

MacConmara, Malcolm, and James A. Lederer. "B cells." Critical Care Medicine 33, Suppl (December 2005): S514—S516. http://dx.doi.org/10.1097/01.ccm.0000190616.15952.4b.

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3

Dörner, Thomas, and Peter E. Lipsky. "B cells." Current Opinion in Rheumatology 26, no. 2 (March 2014): 228–36. http://dx.doi.org/10.1097/bor.0000000000000000.

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4

Ollila, Juha, and Mauno Vihinen. "B cells." International Journal of Biochemistry & Cell Biology 37, no. 3 (March 2005): 518–23. http://dx.doi.org/10.1016/j.biocel.2004.09.007.

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5

Haas, Karen M. "Noncanonical B Cells: Characteristics of Uncharacteristic B Cells." Journal of Immunology 211, no. 9 (November 1, 2023): 1257–65. http://dx.doi.org/10.4049/jimmunol.2200944.

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Abstract B lymphocytes were originally described as a cell type uniquely capable of secreting Abs. The importance of T cell help in Ab production was revealed soon afterward. Following these seminal findings, investigators made great strides in delineating steps in the conventional pathway that B cells follow to produce high-affinity Abs. These studies revealed generalized, or canonical, features of B cells that include their developmental origin and paths to maturation, activation, and differentiation into Ab-producing and memory cells. However, along the way, examples of nonconventional B cell populations with unique origins, age-dependent development, tissue localization, and effector functions have been revealed. In this brief review, features of B-1a, B-1b, marginal zone, regulatory, killer, NK-like, age-associated, and atypical B cells are discussed. Emerging work on these noncanonical B cells and functions, along with the study of their significance for human health and disease, represents an exciting frontier in B cell biology.

6

Hananeh, W., R. Al Rukibat, and M. Daradka. "Primary splenic diffuse large B-cell lymphoma with multinucleated giant cells in a horse." Veterinární Medicína 66, No. 2 (February 2, 2021): 76–79. http://dx.doi.org/10.17221/61/2020-vetmed.

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A diagnosis of a diffuse splenic large B-cell lymphoma with multinucleated giant cells in a 5-year-old mare was made based upon the clinical, pathological, and immunohistochemical findings. The enormous primary splenic mass weighed 51.75 kg. To the best of our knowledge, this is the biggest reported splenic mass and the first case of an equine diffuse large B-cell lymphoma with multinucleated giant cells.

7

ANDREW, ANN. "DEVELOPMENTAL RELATIONSHIPS OF NEUROENDOCRINE CELLS ." Biomedical Research 6, no. 4 (1985): 191–96. http://dx.doi.org/10.2220/biomedres.6.191.

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8

Yeo, Seung Geun, Joong Saeng Cho, Dong Choon Park, and Thomas L. Rothstein. "B-1 Cells Differ from Conventional B (B-2) Cells: Difference in Proliferation." Immune Network 4, no. 3 (2004): 155. http://dx.doi.org/10.4110/in.2004.4.3.155.

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9

YANABA, Koichi. "Regulatory B cells." Japanese Journal of Clinical Immunology 32, no. 3 (2009): 135–41. http://dx.doi.org/10.2177/jsci.32.135.

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10

Cory, Suzanne. "Masterminding B Cells." Journal of Immunology 195, no. 3 (July 17, 2015): 763–65. http://dx.doi.org/10.4049/jimmunol.1501277.

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11

Desiderio, Stephen. "Becoming B cells." Nature 361, no. 6409 (January 1993): 202–3. http://dx.doi.org/10.1038/361202a0.

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12

Mollejo, Manuela, Javier Menárguez, Eva Cristóbal, Patrocinio Algara, Esther Sánchez-Díaz, Máximo Fraga, and Miguel A. Piris. "Monocytoid B Cells." American Journal of Surgical Pathology 18, no. 11 (November 1994): 1131–39. http://dx.doi.org/10.1097/00000478-199411000-00007.

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13

Dart, Anna. "Bad B cells." Nature Reviews Cancer 18, no. 2 (February 2018): 66. http://dx.doi.org/10.1038/nrc.2018.7.

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14

Fend, Falko, David Nachbaur, and Heinz Huber. "Thymic B Cells." American Journal of Clinical Pathology 96, no. 1 (July 1, 1991): 148–49. http://dx.doi.org/10.1093/ajcp/96.1.148.

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15

CHEIKH, MARCIA CURY, MIREILLE-HONTEBEYRIE JOSKOWICZ, ANTONIO COUTINHO, and PAOLA MINOPRIO. "CD5 B Cells." Annals of the New York Academy of Sciences 651, no. 1 (May 1992): 557–63. http://dx.doi.org/10.1111/j.1749-6632.1992.tb24662.x.

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16

Tierens, Anne, Jan Delabie, and Chris De Wolf-Peeters. "Monocytoid B cells." Blood 96, no. 4 (August 15, 2000): 1612–14. http://dx.doi.org/10.1182/blood.v96.4.1612.

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17

Tierens, Anne, Jan Delabie, and Chris De Wolf-Peeters. "Monocytoid B cells." Blood 96, no. 4 (August 15, 2000): 1612–14. http://dx.doi.org/10.1182/blood.v96.4.1612.h8001608c_1612_1614.

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18

Dempsey, Laurie A. "Clipping B cells." Nature Immunology 14, no. 3 (February 15, 2013): 204. http://dx.doi.org/10.1038/ni.2562.

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19

Fehervari, Zoltan. "Thymic B cells." Nature Immunology 14, no. 12 (November 15, 2013): 1211. http://dx.doi.org/10.1038/ni.2777.

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20

Papatriantafyllou, Maria. "ChATty B cells." Nature Reviews Immunology 13, no. 2 (January 25, 2013): 70. http://dx.doi.org/10.1038/nri3396.

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21

Kurosaki, Tomohiro, Kohei Kometani, and Wataru Ise. "Memory B cells." Nature Reviews Immunology 15, no. 3 (February 13, 2015): 149–59. http://dx.doi.org/10.1038/nri3802.

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22

Sinkorova, Z., J. Sinkora, L. Zarybnicka, Z. Vilasova, and J. Pejchal. "Radiosensitivity of peripheral blood B cells in pigs." Veterinární Medicína 54, No. 5 (June 1, 2009): 223–35. http://dx.doi.org/10.17221/59/2009-vetmed.

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Анотація:

: Swine are here introduced to biodosimetry in an attempt to develop a large animal model allowing for comparison of <I>in vitro</I> experiments with the <I>in vivo</I> processes occurring after exposure to gamma radiation. This work investigates the radiosensitivity of the B cell compartment in peripheral blood. Four-week-old piglets were irradiated using the whole body protocol or full blood samples were irradiated <I>in vitro</I> in the dose range of 0–10 Gy. Relative radioresistance of B cell subpopulations and subsets was determined by measuring their relative numbers in leukocyte preparations at selected time intervals after irradiation using two color immunophenotyping and flow cytometry. Porcine B cells represent the most radiosensitive lymphocyte population in peripheral blood. Among B cell subpopulations and subsets investigated, the CD21+SWC7+ and CD21+CD1+ cells are highly radiosensitive and possess biodosimetric potential, at least in the range of low doses. Differences between cultures irradiated <I>in vitro</I> and lymphocyte dynamics in peripheral blood of irradiated animals clearly document the limits of <I>in vitro</I> data extrapolation in biodosimetry. We have shown that pigs can successfully be used in radiobiology and experimental biodosimetry due mainly to their availability, size and a relatively broad spectrum of available immunoreagents for lymphocyte classification.

23

Quách, Tâm D., Thomas J. Hopkins, Nichol E. Holodick, Raja Vuyyuru, Tim Manser, Ruthee-Lu Bayer, and Thomas L. Rothstein. "Human B-1 and B-2 B Cells Develop from Lin−CD34+CD38loStem Cells." Journal of Immunology 197, no. 10 (October 7, 2016): 3950–58. http://dx.doi.org/10.4049/jimmunol.1600630.

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24

DeFranco, Anthony L. "Between B cells and T cells." Nature 351, no. 6328 (June 1991): 603–4. http://dx.doi.org/10.1038/351603a0.

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25

Rodríguez-Pinto, Daniel. "B cells as antigen presenting cells." Cellular Immunology 238, no. 2 (December 2005): 67–75. http://dx.doi.org/10.1016/j.cellimm.2006.02.005.

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26

Bell, Elaine. "Turning B cells into T cells." Nature Reviews Immunology 7, no. 11 (November 2007): 838–39. http://dx.doi.org/10.1038/nri2196.

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27

Kurtin, Paul J. "Marginal Zone B Cells, Monocytoid B Cells, and the Follicular Microenvironment." American Journal of Clinical Pathology 114, no. 4 (October 1, 2000): 505–8. http://dx.doi.org/10.1309/l69g-f64h-4f3j-l2r5.

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28

Küppers, Ralf. "Human memory B cells: Memory B cells of a special kind." Immunology & Cell Biology 86, no. 8 (August 12, 2008): 635–36. http://dx.doi.org/10.1038/icb.2008.59.

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29

Kantor, Aaron B. "The development and repertoire of B-1 cells (CD5 B cells)." Immunology Today 12, no. 11 (November 1991): 389–91. http://dx.doi.org/10.1016/0167-5699(91)90136-h.

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30

Cook, Matthew C. "B cells: B cell back catalogue (remastered)." Immunology & Cell Biology 86, no. 2 (January 22, 2008): 109–10. http://dx.doi.org/10.1038/sj.icb.7100162.

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31

Lydyard, Peter M., Andrew P. Jewell, Christoph Jamin, and Pierre Y. Youinou. "CD5 B cells and B-cell malignancies." Current Opinion in Hematology 6, no. 1 (January 1999): 30. http://dx.doi.org/10.1097/00062752-199901000-00006.

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32

Scott, David W. "Transduced B cells: B is for ‘beneficial’!" European Journal of Immunology 41, no. 6 (May 26, 2011): 1528–30. http://dx.doi.org/10.1002/eji.201141649.

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33

KAWANO, Eisuke, Taku TORIUMI, Shinya IGUCHI, Daigo SUZUKI, Shuichi SATO, and Masaki HONDA. "Induction of neural crest cells from human dental pulp-derived induced pluripotent stem cells ." Biomedical Research 38, no. 2 (2017): 135–47. http://dx.doi.org/10.2220/biomedres.38.135.

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34

Krieger, J. I., S. F. Grammer, H. M. Grey, and R. W. Chesnut. "Antigen presentation by splenic B cells: resting B cells are ineffective, whereas activated B cells are effective accessory cells for T cell responses." Journal of Immunology 135, no. 5 (November 1, 1985): 2937–45. http://dx.doi.org/10.4049/jimmunol.135.5.2937.

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Abstract In this study, we have investigated the ability of splenic B cells to act as antigen-presenting cells. Previous data had established that lipopolysaccharide (LPS)-activated B cells were effective antigen-presenting cells; however, the relative capacity of resting B cells to carry out this function remains controversial. Splenic B cells from naive BALB/c mice were depleted of macrophages, dendritic cells, and T cells, and were fractionated on the basis of cell density by using Percoll gradient centrifugation. Fractions were collected from the 50/60, 60/65, and 65/72% interfaces and from greater than 72% (pellet). Cytofluorograph analysis of the fractionated B cells showed that the two lower density fractions (50/60 and 60/65) contained a number of cells which, by cell size determination, appeared to be activated B cells, whereas the two higher density fractions (65/72 and greater than 72) appeared to contain predominantly small resting B cells contaminated by many fewer activated B cells. Functionally, the capacity of fractionated B cells to act as accessory cells for a concanavalin A response or present the antigens chicken ovalbumin (OVA) or OVA-tryptic digest gave similar results, which indicated a striking hierarchy of accessory cell function in the different Percoll fractions. When normalized to the most active low-density fraction (50/60%), the activity of the other fractions were: 60/65 = 78%; 65/72 = 25%; and greater than 72 = 4%. The differences in the functional capacity between the various Percoll fractions did not appear to be due to differences in Ia expression. Although the expression of Ia varied approximately 12-fold within any one fraction, there was little difference in the mean amount of Ia on cells obtained from the various fractions. Kinetic studies showed that activation of B cells with LPS and dextran sulfate resulted in the expression of two stages of functional development. The first stage was an increased efficiency of accessory cell function that was abrogated by irradiation with 4000 rad followed by a second stage, which was characterized by the acquisition of resistance to treatment with 4000 rad. When nonfractionated B cells that had been stimulated with LPS and DexSO4 were sorted on the basis of cell size into a small B cell fraction and a large B cell fraction, only the large B cells were able to present antigen. Taken together, these data suggest that much of the accessory cell function associated with splenic B cells can be accounted for by the relatively small percentage of activated B cells present in the spleen.(ABSTRACT TRUNCATED AT 400 WORDS)

35

Pattarabanjird, Tanyaporn, Cynthia Li, and Coleen McNamara. "B Cells in Atherosclerosis." JACC: Basic to Translational Science 6, no. 6 (June 2021): 546–63. http://dx.doi.org/10.1016/j.jacbts.2021.01.006.

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36

Engelhard, Victor, Jose R. Conejo-Garcia, Rafi Ahmed, Brad H. Nelson, Karen Willard-Gallo, Tullia C. Bruno, and Wolf H. Fridman. "B cells and cancer." Cancer Cell 39, no. 10 (October 2021): 1293–96. http://dx.doi.org/10.1016/j.ccell.2021.09.007.

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37

Linnebacher, Michael, and Claudia Maletzki. "Tumor-infiltrating B cells." OncoImmunology 1, no. 7 (October 2012): 1186–88. http://dx.doi.org/10.4161/onci.20641.

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38

Gisselbrecht, Christian. "Large B cells lymphoma." Hématologie 20, no. 3 (May 2014): 183–88. http://dx.doi.org/10.1684/hma.2014.0947.

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39

Hamel, Keith M., Vladimir M. Liarski, and Marcus R. Clark. "Germinal Center B-cells." Autoimmunity 45, no. 5 (April 2, 2012): 333–47. http://dx.doi.org/10.3109/08916934.2012.665524.

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40

Bashyam, Hema. "Gut-friendly B cells?" Journal of Experimental Medicine 205, no. 6 (June 2, 2008): 1246. http://dx.doi.org/10.1084/jem.2056iti3.

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41

Pujanandez, Lindsey. "Teaching baby B cells." Science 363, no. 6430 (February 28, 2019): 941.6–942. http://dx.doi.org/10.1126/science.363.6430.941-f.

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42

Scanlon, Seth Thomas. "IgE B cells unmasked." Science 362, no. 6420 (December 13, 2018): 1259.6–1260. http://dx.doi.org/10.1126/science.362.6420.1259-f.

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43

Dörner, Thomas, Annett M. Jacobi, and Peter E. Lipsky. "B cells in autoimmunity." Arthritis Research & Therapy 11, no. 5 (2009): 247. http://dx.doi.org/10.1186/ar2780.

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44

Seton-Rogers, Sarah. "Spotlight on B cells." Nature Reviews Cancer 16, no. 2 (January 29, 2016): 67. http://dx.doi.org/10.1038/nrc.2016.7.

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45

Bernard, Nicholas J. "Double-negative B cells." Nature Reviews Rheumatology 14, no. 12 (October 26, 2018): 684. http://dx.doi.org/10.1038/s41584-018-0113-6.

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46

Zachary, Andrea A., Dessislava Kopchaliiska, Robert A. Montgomery, and Mary S. Leffell. "HLA-Specific B Cells." Transplantation 83, no. 7 (April 2007): 982–88. http://dx.doi.org/10.1097/01.tp.0000259017.32857.99.

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47

Zachary, Andrea A., Dessislava Kopchaliiska, Robert A. Montgomery, Joseph K. Melancon, and Mary S. Leffell. "HLA-Specific B Cells." Transplantation 83, no. 7 (April 2007): 989–94. http://dx.doi.org/10.1097/01.tp.0000259019.68244.d7.

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48

Allie, S. Rameeza, and Troy D. Randall. "Resident Memory B Cells." Viral Immunology 33, no. 4 (May 1, 2020): 282–93. http://dx.doi.org/10.1089/vim.2019.0141.

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49

Gough, N. R. "Suppressed by B Cells." Science Signaling 7, no. 318 (March 25, 2014): ec79-ec79. http://dx.doi.org/10.1126/scisignal.2005296.

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50

Cancro, Michael P. "Age-Associated B Cells." Annual Review of Immunology 38, no. 1 (April 26, 2020): 315–40. http://dx.doi.org/10.1146/annurev-immunol-092419-031130.

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Анотація:

The age-associated B cell subset has been the focus of increasing interest over the last decade. These cells have a unique cell surface phenotype and transcriptional signature, and they rely on TLR7 or TLR9 signals in the context of Th1 cytokines for their formation and activation. Most are antigen-experienced memory B cells that arise during responses to microbial infections and are key to pathogen clearance and control. Their increasing prevalence with age contributes to several well-established features of immunosenescence, including reduced B cell genesis and damped immune responses. In addition, they are elevated in autoimmune and autoinflammatory diseases, and in these settings they are enriched for characteristic autoantibody specificities. Together, these features identify age-associated B cells as a subset with pivotal roles in immunological health, disease, and aging. Accordingly, a detailed understanding of their origins, functions, and physiology should make them tractable translational targets in each of these settings.