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Статті в журналах з теми "Arl15"
Brito, Cheila, Bruno Costa-Silva, Duarte C. Barral, and Marta Pojo. "Unraveling the Relevance of ARL GTPases in Cutaneous Melanoma Prognosis through Integrated Bioinformatics Analysis." International Journal of Molecular Sciences 22, no. 17 (August 26, 2021): 9260. http://dx.doi.org/10.3390/ijms22179260.
Повний текст джерелаCorre, Tanguy, Francisco J. Arjona, Caroline Hayward, Sonia Youhanna, Jeroen H. F. de Baaij, Hendrica Belge, Nadine Nägele, et al. "Genome-Wide Meta-Analysis Unravels Interactions between Magnesium Homeostasis and Metabolic Phenotypes." Journal of the American Society of Nephrology 29, no. 1 (November 1, 2017): 335–48. http://dx.doi.org/10.1681/asn.2017030267.
Повний текст джерелаZolotarov, Yevgen, Chao Ma, Irene González-Recio, Serge Hardy, Gijs A. C. Franken, Noriko Uetani, Femke Latta, et al. "ARL15 modulates magnesium homeostasis through N-glycosylation of CNNMs." Cellular and Molecular Life Sciences 78, no. 13 (June 5, 2021): 5427–45. http://dx.doi.org/10.1007/s00018-021-03832-8.
Повний текст джерелаLi, Yiping, Ying Yang, Yueting Yao, Xianli Li, Li Shi, Ying Zhang, Yuxin Xiong, Man Yan, Yufeng Yao, and Chunjie Xiao. "Association Study of ARL15 and CDH13 with T2DM in a Han Chinese Population." International Journal of Medical Sciences 11, no. 5 (2014): 522–27. http://dx.doi.org/10.7150/ijms.8206.
Повний текст джерелаde Baaij, Jeroen H. F., Yevgen Zolotarov, Chao Ma, Gijs Franken, Michel L. Tremblay, and Joost Hoenderop. "ARL15 Regulates CNNM2‐dependent Mg 2+ Transport by Modulating its N‐linked Glycosylation." FASEB Journal 34, S1 (April 2020): 1. http://dx.doi.org/10.1096/fasebj.2020.34.s1.06380.
Повний текст джерелаRichards, J. Brent, Dawn Waterworth, Stephen O'Rahilly, Marie-France Hivert, Ruth J. F. Loos, John R. B. Perry, Toshiko Tanaka, et al. "A Genome-Wide Association Study Reveals Variants in ARL15 that Influence Adiponectin Levels." PLoS Genetics 5, no. 12 (December 11, 2009): e1000768. http://dx.doi.org/10.1371/journal.pgen.1000768.
Повний текст джерелаYang, Yong-Kang, Hong Qu, Dong Gao, Wei Di, Hai-Wei Chen, Xin Guo, Zhong-He Zhai, and Dan-Ying Chen. "ARF-like Protein 16 (ARL16) Inhibits RIG-I by Binding with Its C-terminal Domain in a GTP-dependent Manner." Journal of Biological Chemistry 286, no. 12 (January 13, 2011): 10568–80. http://dx.doi.org/10.1074/jbc.m110.206896.
Повний текст джерелаBenabdelkamel, Hicham, Afshan Masood, Meshail Okla, Mohammed Y. Al-Naami, and Assim A. Alfadda. "A Proteomics-Based Approach Reveals Differential Regulation of Urine Proteins between Metabolically Healthy and Unhealthy Obese Patients." International Journal of Molecular Sciences 20, no. 19 (October 3, 2019): 4905. http://dx.doi.org/10.3390/ijms20194905.
Повний текст джерелаShen, Jiayi, Miao Liu, Jing Xu, Bao Sun, Heng Xu, and Wei Zhang. "ARL15 overexpression attenuates high glucose-induced impairment of insulin signaling and oxidative stress in human umbilical vein endothelial cells." Life Sciences 220 (March 2019): 127–35. http://dx.doi.org/10.1016/j.lfs.2019.01.030.
Повний текст джерелаWicky, Sidonie, Heinz Schwarz, and Birgit Singer-Krüger. "Molecular Interactions of Yeast Neo1p, an Essential Member of the Drs2 Family of Aminophospholipid Translocases, and Its Role in Membrane Trafficking within the Endomembrane System." Molecular and Cellular Biology 24, no. 17 (September 1, 2004): 7402–18. http://dx.doi.org/10.1128/mcb.24.17.7402-7418.2004.
Повний текст джерелаДисертації з теми "Arl15"
Panić, Bojana. "The small GTPases Arl1p/Arl1 and Arl3p/ARFRP1 act in a pathway for targeting proteins to the Golgi apparatus." Thesis, University of Cambridge, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.616124.
Повний текст джерелаLasić, Maja. "The yeast endosomal/TGN-localized Ysl2p-Arl1p-Neo1p network: search for novel interaction partners." [S.l. : s.n.], 2008. http://nbn-resolving.de/urn:nbn:de:bsz:93-opus-34910.
Повний текст джерелаFerreira, Cláudia Susana da Rosa. "Organelle-specific roles for the Arf-like G proteins Arl5 and Arl8." Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.609430.
Повний текст джерелаMan, Zhiqiu. "Localization and function of Arfaptins: Arl1-dependent trans-Golgi localization and induction of membrane deformation." 京都大学 (Kyoto University), 2012. http://hdl.handle.net/2433/157901.
Повний текст джерелаDing, Jian. "Functional analysis of the extended N-terminus for the Drosophila Raf protein and initial characterization of the Arl1 gene." [Ames, Iowa : Iowa State University], 2010. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3403792.
Повний текст джерелаSharma, Prerna. "Unravelling the functional role of Arf-like GTPases 14 and 15 in mammalian cells." Thesis, 2022. https://etd.iisc.ac.in/handle/2005/5831.
Повний текст джерелаHsu, Hsin-Chia, and 許俽嘉. "Characterization of an Arl1p Guanine-Nucleotide Exchange Factor, Syt1p." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/04867349717273321965.
Повний текст джерела臺灣大學
分子醫學研究所
98
ARF-like (ARL) proteins belong to ADP-ribosylation factor (ARF) GTPase family, which are involved in protein trafficking and cytoskeleton organization. Those small G proteins require guanine-nucleotide exchange factors (GEFs) to switch from GDP-bound to GTP-bound form and become active. Previous reports suggested that Syt1p is the GEF of Arf2p and is involved in vesicle trafficking. Recently, our studies showed that Syt1p also acted as a GEF for Arl1p. In this study, Syt1p was further characterized. Firstly, it has been shown that Syt1p belongs to BFA-resistant GEFs. Secondly, Syt1p can use multiple regions to interact with Arl1p. The N-terminus, Sec7 domain, and C-terminus of Syt1p can all interact with Arl1d17N form, whose N-terminal first 17 amino acids are deleted. The interactions between all of the three regions and Arl1d17N are stronger than the interaction between full-length Syt1p and Arl1pd17N. Therefore, it might hint that Syt1p is autoregulated as other GEFs of Small GTPases. Surprisingly, we next found that Syt1p has an intramolecular interaction between the C-terminal region and Sec7 domain and an intermolecular interaction between C-terminal regions, indicating that Syt1p could form dimers or oligomers and might be autoregulated. Syt1p dimerization or oligomerization is also supported by in vivo pull down results, which proved that Syt1p can interact with itself. Moreover, the N-terminus is important for the formation of dimers or oligomers. Yeast two-hybrid screen was also performed to search for putative regulators of Syt1p. However, those candidates remain to be elucidated. Besides, whether dimerization or oligomerization plays an important role in Syt1p activation or membrane tethering and whether autoinhibition truly exists in Syt1p in vivo require further investigation.
LIn, Ching-Yi, and 林靜宜. "Functional Characterization of ADP-Ribosylation Factor-Like Proteins:ARL4 and ARL5." Thesis, 2001. http://ndltd.ncl.edu.tw/handle/37795439008511565193.
Повний текст джерела國立臺灣大學
分子醫學研究所
89
ADP-ribosylation factors (ARFs) Ras-related small GTP-binding proteins (~20 kDa), were originally identified as protein cofactors for the cholera toxin-catalyzed ADP-ribosylation of GS, the stimulatory subunit for adenylate cyclase. Their cofactor activity depends on GTP-binding- regulated by interactions with ARF-specific guanine nucleotide exchange factors (GEFs) or ARF-specific GTPase activating proteins (GAPs). Today, at least six mammalian ARF members have been identified and grouped into three classes. ARFs have been found in different subcellular localizations, and regulate several different vesicle transport steps in the exocytic and endocytic pathways- including budding from the endoplasmic reticulum and fusion to the Golgi stacks, endosomes, and nuclear vesicles. ARF-like (ARL) proteins, which belong to another subfamily of ARFs, have been cloned recently from Drosophila, rat, human and yeast. Although ARLs and ARFs have very similar amino acid sequences, ARLs lack cofactor activity for the cholera toxin-catalyzed ADP-ribosylation. dARL1 is essential in Drosophila, while disruption of yeast ARL1 gene is not lethal. ARL1 from yeast and human were reported to localize at the Golgi complex. In this thesis, we report the biochemical characterization and subcellular localizations of two ARLs- ARL4 and ARL5. We first cloned mouse and human ARL4 (mARL4 and hARL4) cDNA, respectively. Mice ARL4 is abundant in testis and has developmental stages-specific expression pattern. The appearance of mARL4 mRNA during embryonic development coincides with the sequential formation of somites and the establishment of brain compartmentation. Using ARL4-specific antibody in immuno-fluorescence microscopy, we found that endogenous mARL4 in cultured Sertoli and neuroblastoma cells was mainly concentrated in nuclei. When ectopically expressed in COS7 cells, ARL4-T34N mutant (predicted to be the GDP-bound form) was concentrated in nucleoli. Yeast two-hybrid screen and in vitro protein-interaction assays showed that hARL4 interacts with importin-a-required its C-terminal NLS region, and the interaction is nucleotide-independent. Like ARL2 and ARL3, recombinant hARL4 does not enhance cholera toxin-catalyzed auto-ADP-ribosylation. Its binding with GTPγS could be affected by phospholipid, and the N-terminus of hARL4, like that of ARF, is myristoylated. Our findings suggest that ARL4- with its distinctive nuclear/nucleolar localization and developmentally regulated expression pattern-may play a unique role(s) in neurogenesis and somitogenesis during embryonic development, and in the early stages of spermatogenesis in adults, respectively. Mouse and human ARL5 (mARL5 and hARL5) were also cloned, and the hARL5 mRNA is ubiquitously expressed in many tissues and considerably abundant in liver. mARL5 mRNA is mainly present at day 7 mouse embryos, then disappears at day 11. Our findings suggest that ARL5 may have an important role(s) in the early events of embryonic development. Immunostaining of COS7 cells transfected with Myc-tagged hARL5-WT, hARL5-T35N, hARL5-Q80L, and hARL5-dC constructs provide direct evidence for its subcellular localization in the cell. Interestingly, hARL5-T35N mutant, the predicted GDP-bound form, was most concentrated in nucleoli, but sometimes present in cytosol. By yeast two-hybrid screen, hARL5 interacts with NLS-receptor, importin-a, through the putative bipartite NLS in its C-terminal region. When NLS is removed from hARL5, hARL5-dC is colocalized with mitochondria; hARL5-T35N has the same pattern when it appears in the cytosol. In addition, yeast two-hybrid screen and in vitro protein-interaction assays showed that hARL5-WT, hARL5-Q80L can interact with HP1a, and the interaction is nucleotide-dependent. The interaction was further confirmed by in vivo protein-interaction assays. hARL5 contains MIR homology sequence- VPVLVL (residue 128~133)- that is crucial for hARL5 binding to HP1a. As ARL2, 3, and 4, recombinant hARL5 did not enhance the cholera toxin-catalyzed auto-ADP-ribosylation. Its intrinsic GTPgS-binding activity was low. The N-terminus of hARL5 is myristoylated as are ARFs. Based on these results, we have demonstrated that hARL5, like hARL4, with its specific interaction proteins and distinctive nuclear/nucleolar localizations may have important biological functions.
Diamantino, João Marques da Cunha dos Santos. "The role of Arl17 in healthy and influenza A virus infected cells." Master's thesis, 2019. http://hdl.handle.net/10362/89281.
Повний текст джерелаWu, Tsung-Shung. "Functional characterization of Arf-like protein, ARL5 and its interacting protein EB1." 2005. http://www.cetd.com.tw/ec/thesisdetail.aspx?etdun=U0001-0908200521130700.
Повний текст джерелаКниги з теми "Arl15"
Build Your Own AR15 Rifle. Createspace, 2010.
Знайти повний текст джерелаZediker, Glen D. America's Gun: The Practical AR15. Primedia eLaunch LLC, 2018.
Знайти повний текст джерелаGun Digest Shooters Guide To The Ar15. F+W Media Inc, 2014.
Знайти повний текст джерелаThe Competitive AR15; The Mouse That Roared. Zediker Publishing, 1998.
Знайти повний текст джерелаBartocci, Christopher. Armorer's Manual AR15/M16/M4 Family of Weapons. Independently published, 2017.
Знайти повний текст джерелаGun Digest Book of the AR15 Volume 4. Gun Digest Books, 2012.
Знайти повний текст джерелаThe M16/AR15 Rifle (A Shooter's and Collector's Guide). 3rd ed. North Cape Publications Inc, 2007.
Знайти повний текст джерелаLuggage, Handbags, Saddlery & Harness (UK Markets: Annual Reports 1993: AR15). The Stationery Office Books (Agencies), 1994.
Знайти повний текст джерелаLuggage, Handbags, Saddlery and Harness (UK Markets: Annual Reports 1994: AR15). The Stationery Office Books (Agencies), 1995.
Знайти повний текст джерелаLINDA. 2nd Amendment for Men 556 2A AR15 Accessories Pro Gun Rights Meme. Independently Published, 2022.
Знайти повний текст джерелаЧастини книг з теми "Arl15"
Schrapper, Christian. "Sozialpädagogische Diagnosen und sozialpädagogisches Fallverstehen." In Handbuch Allgemeiner Sozialer Dienst (ASD), 3. aktual. u. erw. Auflage. München: Ernst Reinhardt Verlag, 2019. http://dx.doi.org/10.2378/asda3.art15.
Повний текст джерелаCross, Eric, and Helen Pickering. "La contribution de l'enseignement supérieur au développement culturel régional dans le Nord-Est de l'Angleterre." In Politiques et gestion de l'enseignement supérieur, Volume 20 Numéro 2, 1–15. OECD, 2008. http://dx.doi.org/10.1787/hemp-v20-art15-fr.
Повний текст джерелаPacheco-Rodriguez, Gustavo, Joel Moss, and Martha Vaughan. "[44] Preparation and assay of recombinant ADP-ribosylation factor-like protein-1 (ARL1)." In Methods in Enzymology, 424–28. Elsevier, 2001. http://dx.doi.org/10.1016/s0076-6879(01)29103-4.
Повний текст джерелаTai, Guihua, Lei Lu, Ludger Johannes, and Wanjin Hong. "Functional Analysis of Arl1 and Golgin‐97 in Endosome‐to‐TGN Transport Using Recombinant Shiga Toxin B Fragment." In Methods in Enzymology, 442–53. Elsevier, 2005. http://dx.doi.org/10.1016/s0076-6879(05)04039-5.
Повний текст джерелаLu, Lei, Guihua Tai, and Wanjin Hong. "Interaction of Arl1 GTPase with the GRIP Domain of Golgin‐245 as Assessed by GST (Glutathione‐S‐transferase) Pull‐Down Experiments." In Methods in Enzymology, 432–41. Elsevier, 2005. http://dx.doi.org/10.1016/s0076-6879(05)04038-3.
Повний текст джерелаТези доповідей конференцій з теми "Arl15"
Silva, Daniel Campos, Leonardo Paes Rangel, Marcone Lima Sobreira, Natan Padoin, and Cíntia Soares. "COMPUTATIONAL SIMULATION OF ABDOMINAL AORTIC ANEURISM AND ITS ASSESSMENT TO MEDICAL TREATMENT." In Simpósio Paranaense de Modelagem, Simulação e Controle de Processos. Departamento de Engenharia Química UFPR, 2020. http://dx.doi.org/10.5380/simproc4.2019.art15.
Повний текст джерелаLepre, Priscilla. "DO TANGÍVEL AO INTANGÍVEL NO DESIGN DE S.PSS - REFLEXÕES SOBRE A POSSE E O VALOR." In Simpósio de Design Sustentável. Departamento de Design da UFPR, 2021. http://dx.doi.org/10.5380/8sds2021.art15.
Повний текст джерелаSchennach, S., A. Müller, A. Frank, J. Haselbauer, W. Spies, O. Uwira, M. Wagner та ін. "Dielectronic recombination of Ar15+ ions involving Δn=0 and Δn=1 transitions". У 6th International conference on the physics of highly charged ions. AIP, 1993. http://dx.doi.org/10.1063/1.43658.
Повний текст джерелаLee, Sooyong, Sangkyou Lee, Jolanta Bondaruk, Hua Wang, Shizhen Zhang, Menashe Bar-Eli, Peter Black, et al. "Abstract 3065: Loss of ARL11 function promotes growth ofin situneoplasia by activating the ras pathway." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3065.
Повний текст джерелаMoazzen-Ahmadi, Nasser, A. R. W. McKellar, and A. Barclay. "COMPLETION OF THE FIRST SOLVATION SHELL OF CARBON DIOXIDE IN ARGON: ROTATIONALLY RESOLVED INFRARED SPECTRA OF CO2-AR15 AND CO2-AR17." In 2022 International Symposium on Molecular Spectroscopy. Urbana, Illinois: University of Illinois at Urbana-Champaign, 2022. http://dx.doi.org/10.15278/isms.2022.ri09.
Повний текст джерела"ПРАКТИКА ОКАЗАНИЯ ПСИХОЛОГО-ПЕДАГОГИЧЕСКОЙ ПОМОЩИ И СОПРОВОЖДЕНИЯ ДЕТЕЙ С РАССТРОЙСТВАМИ АУТИСТИЧЕСКОГО СПЕКТРА В Г. МАГАДАНЕ". У НА ПЕРЕКРЕСТКЕ СЕВЕРА И ВОСТОКА (МЕТОДОЛОГИИ И ПРАКТИКИ РЕГИОНАЛЬНОГО РАЗВИТИЯ). Science and Innovation Center Publishing House, 2023. http://dx.doi.org/10.12731/978-5-907608-10-8-art15.
Повний текст джерелаЗвіти організацій з теми "Arl15"
Sweeney, Steven, L. D. Stephenson, Ralph Eichlin, and Robert Weber. Demonstration and validation of stainless steel materials for critical above-grade piping in highly corrosive locations : final report on Project F07-AR15. Construction Engineering Research Laboratory (U.S.), May 2017. http://dx.doi.org/10.21079/11681/22548.
Повний текст джерела