Добірка наукової літератури з теми "Arl14"
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Статті в журналах з теми "Arl14"
Brito, Cheila, Bruno Costa-Silva, Duarte C. Barral, and Marta Pojo. "Unraveling the Relevance of ARL GTPases in Cutaneous Melanoma Prognosis through Integrated Bioinformatics Analysis." International Journal of Molecular Sciences 22, no. 17 (August 26, 2021): 9260. http://dx.doi.org/10.3390/ijms22179260.
Повний текст джерелаYang, Feng, Tiantian Li, Ziqing Peng, Yang Liu, and Yusong Guo. "The amphipathic helices of Arfrp1 and Arl14 are sufficient to determine subcellular localizations." Journal of Biological Chemistry 295, no. 49 (September 24, 2020): 16643–54. http://dx.doi.org/10.1074/jbc.ra120.014999.
Повний текст джерелаZhang, Binbin, Aiqun Xu, Dong Wu, Wanli Xia, Pulin Li, Enze Wang, Rui Han, Peng Sun, Sijing Zhou, and Ran Wang. "ARL14 as a Prognostic Biomarker in Non-Small Cell Lung Cancer." Journal of Inflammation Research Volume 14 (December 2021): 6557–74. http://dx.doi.org/10.2147/jir.s340119.
Повний текст джерелаYang, Yong-Kang, Hong Qu, Dong Gao, Wei Di, Hai-Wei Chen, Xin Guo, Zhong-He Zhai, and Dan-Ying Chen. "ARF-like Protein 16 (ARL16) Inhibits RIG-I by Binding with Its C-terminal Domain in a GTP-dependent Manner." Journal of Biological Chemistry 286, no. 12 (January 13, 2011): 10568–80. http://dx.doi.org/10.1074/jbc.m110.206896.
Повний текст джерелаAcosta-Herrera, Marialbert, Martin Kerick, David González-Serna, Cisca Wijmenga, Andre Franke, Peter K. Gregersen, Leonid Padyukov, et al. "Genome-wide meta-analysis reveals shared new loci in systemic seropositive rheumatic diseases." Annals of the Rheumatic Diseases 78, no. 3 (December 20, 2018): 311–19. http://dx.doi.org/10.1136/annrheumdis-2018-214127.
Повний текст джерелаWicky, Sidonie, Heinz Schwarz, and Birgit Singer-Krüger. "Molecular Interactions of Yeast Neo1p, an Essential Member of the Drs2 Family of Aminophospholipid Translocases, and Its Role in Membrane Trafficking within the Endomembrane System." Molecular and Cellular Biology 24, no. 17 (September 1, 2004): 7402–18. http://dx.doi.org/10.1128/mcb.24.17.7402-7418.2004.
Повний текст джерелаZolotarov, Yevgen, Chao Ma, Irene González-Recio, Serge Hardy, Gijs A. C. Franken, Noriko Uetani, Femke Latta, et al. "ARL15 modulates magnesium homeostasis through N-glycosylation of CNNMs." Cellular and Molecular Life Sciences 78, no. 13 (June 5, 2021): 5427–45. http://dx.doi.org/10.1007/s00018-021-03832-8.
Повний текст джерелаHsu, Jia-Wei, Pei-Hua Tang, I.-Hao Wang, Chia-Lun Liu, Wen-Hui Chen, Pei-Chin Tsai, Kuan-Yu Chen, Kuan-Jung Chen, Chia-Jung Yu, and Fang-Jen S. Lee. "Unfolded protein response regulates yeast small GTPase Arl1p activation at late Golgi via phosphorylation of Arf GEF Syt1p." Proceedings of the National Academy of Sciences 113, no. 12 (March 10, 2016): E1683—E1690. http://dx.doi.org/10.1073/pnas.1518260113.
Повний текст джерелаLiu, Ya-Wen, Chun-Fang Huang, Kai-Bin Huang, and Fang-Jen S. Lee. "Role for Gcs1p in Regulation of Arl1p atTrans-Golgi Compartments." Molecular Biology of the Cell 16, no. 9 (September 2005): 4024–33. http://dx.doi.org/10.1091/mbc.e05-01-0023.
Повний текст джерелаXie, Ning, Qiuai Shu, Ziwei Wang, Xindi Huang, Yalan Wang, Bin Qin, Yan Chen, et al. "ARL11 correlates with the immunosuppression and poor prognosis in breast cancer: A comprehensive bioinformatics analysis of ARL family members." PLOS ONE 17, no. 11 (November 11, 2022): e0274757. http://dx.doi.org/10.1371/journal.pone.0274757.
Повний текст джерелаДисертації з теми "Arl14"
Panić, Bojana. "The small GTPases Arl1p/Arl1 and Arl3p/ARFRP1 act in a pathway for targeting proteins to the Golgi apparatus." Thesis, University of Cambridge, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.616124.
Повний текст джерелаHofmann, Irmgard Maria Rita. "Characterisation of the human Arl4 and Arl8 families of small GTPases." Thesis, University of Cambridge, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604140.
Повний текст джерелаLasić, Maja. "The yeast endosomal/TGN-localized Ysl2p-Arl1p-Neo1p network: search for novel interaction partners." [S.l. : s.n.], 2008. http://nbn-resolving.de/urn:nbn:de:bsz:93-opus-34910.
Повний текст джерелаMan, Zhiqiu. "Localization and function of Arfaptins: Arl1-dependent trans-Golgi localization and induction of membrane deformation." 京都大学 (Kyoto University), 2012. http://hdl.handle.net/2433/157901.
Повний текст джерелаDing, Jian. "Functional analysis of the extended N-terminus for the Drosophila Raf protein and initial characterization of the Arl1 gene." [Ames, Iowa : Iowa State University], 2010. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3403792.
Повний текст джерелаMan, Hon-wai, and 文漢威. "A study of zebrafish hematopoiesis based on chemical screening and gene knock-down by morpholino with particular reference to ADP-ribosylation factor like 4 (ARL4)." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47770521.
Повний текст джерелаpublished_or_final_version
Medicine
Master
Master of Medical Sciences
Hsu, Hsin-Chia, and 許俽嘉. "Characterization of an Arl1p Guanine-Nucleotide Exchange Factor, Syt1p." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/04867349717273321965.
Повний текст джерела臺灣大學
分子醫學研究所
98
ARF-like (ARL) proteins belong to ADP-ribosylation factor (ARF) GTPase family, which are involved in protein trafficking and cytoskeleton organization. Those small G proteins require guanine-nucleotide exchange factors (GEFs) to switch from GDP-bound to GTP-bound form and become active. Previous reports suggested that Syt1p is the GEF of Arf2p and is involved in vesicle trafficking. Recently, our studies showed that Syt1p also acted as a GEF for Arl1p. In this study, Syt1p was further characterized. Firstly, it has been shown that Syt1p belongs to BFA-resistant GEFs. Secondly, Syt1p can use multiple regions to interact with Arl1p. The N-terminus, Sec7 domain, and C-terminus of Syt1p can all interact with Arl1d17N form, whose N-terminal first 17 amino acids are deleted. The interactions between all of the three regions and Arl1d17N are stronger than the interaction between full-length Syt1p and Arl1pd17N. Therefore, it might hint that Syt1p is autoregulated as other GEFs of Small GTPases. Surprisingly, we next found that Syt1p has an intramolecular interaction between the C-terminal region and Sec7 domain and an intermolecular interaction between C-terminal regions, indicating that Syt1p could form dimers or oligomers and might be autoregulated. Syt1p dimerization or oligomerization is also supported by in vivo pull down results, which proved that Syt1p can interact with itself. Moreover, the N-terminus is important for the formation of dimers or oligomers. Yeast two-hybrid screen was also performed to search for putative regulators of Syt1p. However, those candidates remain to be elucidated. Besides, whether dimerization or oligomerization plays an important role in Syt1p activation or membrane tethering and whether autoinhibition truly exists in Syt1p in vivo require further investigation.
Diamantino, João Marques da Cunha dos Santos. "The role of Arl17 in healthy and influenza A virus infected cells." Master's thesis, 2019. http://hdl.handle.net/10362/89281.
Повний текст джерелаChen, Yan-Ting, and 陳彥廷. "Functional Characterization of small GTPase Arl1 and Golgin Protein Imh1 in Saccharomyces cerevisiae." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/5s8s72.
Повний текст джерела國立臺灣大學
分子醫學研究所
106
Arf-like proteins (Arls) are important regulators involved in a diversity of biological events, especially vesicle trafficking. In Saccharomyces cerevisiae, Arl1 acts to recruit a golgin protein Imh1 to the trans-Golgi network (TGN). However, besides the role of Imh1 as a high-copy suppressor of Ypt6, an important Rab protein mediating endosome-to-Golgi trafficking, less is known about it physiological significance. In previous studies (Hsu et al., 2016), they demonstrated that the Unfolded-Protein Response (UPR) augmented the activity of Arl1 and the subsequent Imh1 recruitment, but it remained to be elucidated on its specific functions under ER stress. Here, we showed that the UPR-activated Arl1 and Imh1 act to specifically maintain the endosome-to-Golgi trafficking when cell encounters ER stress. Moreover, this regulation does not simply depend on GARP complex recruitment, an important tethering complex, to affect retrograde trafficking. We found that the first five amino acids at Imh1 N-terminus is required for its function, as it contributes to the recruitment of Sft2, a tetra-spanning membrane protein involved in vesicle fusion. The N-terminus of Sft2 is responsible for facilitating the transport of Snc1 and Tlg1 and promoting retrograde trafficking. Together, our study is one of the first to demonstrate the physiological function of golgin Imh1 and elucidate the importance of the trafficking machinery in response to ER stress.
Huang, Hsiao-Chuan. "Functional characterization of human ADP-ribosylation factor like-1(ARL1) and its interacting proteins." 2006. http://www.cetd.com.tw/ec/thesisdetail.aspx?etdun=U0001-2807200600120100.
Повний текст джерелаКниги з теми "Arl14"
Tanning & Dressing of Leather (UK Markets: Annual Reports 1994: AR14). The Stationery Office Books (Agencies), 1996.
Знайти повний текст джерелаTanning & Dressing of Leather (UK Markets: Annual Reports 1993: AR14). The Stationery Office Books (Agencies), 1994.
Знайти повний текст джерелаЧастини книг з теми "Arl14"
Merchel, Joachim. "Hilfeplanung." In Handbuch Allgemeiner Sozialer Dienst (ASD), 3. aktual. u. erw. Auflage. München: Ernst Reinhardt Verlag, 2019. http://dx.doi.org/10.2378/asda3.art14.
Повний текст джерелаKonu, Ari, and Eero Pekkarinen. "Université provinciale de Laponie." In Politiques et gestion de l'enseignement supérieur, Volume 20 Numéro 2, 1–11. OECD, 2008. http://dx.doi.org/10.1787/hemp-v20-art14-fr.
Повний текст джерелаPacheco-Rodriguez, Gustavo, Joel Moss, and Martha Vaughan. "[44] Preparation and assay of recombinant ADP-ribosylation factor-like protein-1 (ARL1)." In Methods in Enzymology, 424–28. Elsevier, 2001. http://dx.doi.org/10.1016/s0076-6879(01)29103-4.
Повний текст джерелаTai, Guihua, Lei Lu, Ludger Johannes, and Wanjin Hong. "Functional Analysis of Arl1 and Golgin‐97 in Endosome‐to‐TGN Transport Using Recombinant Shiga Toxin B Fragment." In Methods in Enzymology, 442–53. Elsevier, 2005. http://dx.doi.org/10.1016/s0076-6879(05)04039-5.
Повний текст джерелаLu, Lei, Guihua Tai, and Wanjin Hong. "Interaction of Arl1 GTPase with the GRIP Domain of Golgin‐245 as Assessed by GST (Glutathione‐S‐transferase) Pull‐Down Experiments." In Methods in Enzymology, 432–41. Elsevier, 2005. http://dx.doi.org/10.1016/s0076-6879(05)04038-3.
Повний текст джерелаТези доповідей конференцій з теми "Arl14"
Mueller, Eduarda, Julia Volkmann, Jonathan Utzig, and Henry França Meier. "ANÁLISE DOS FORMATOS GEOMÉTRICOS DE ANÉIS DEFLETORES INTERNOS EM ESCOAMENTO GÁS-SÓLIDO EM RISERS." In Simpósio Paranaense de Modelagem, Simulação e Controle de Processos. Departamento de Engenharia Química UFPR, 2020. http://dx.doi.org/10.5380/simproc4.2019.art14.
Повний текст джерелаRamirez, Mariano. "DESIGN FOR OTHER SPECIES: AUSTRALIAN EXAMPLES." In Simpósio de Design Sustentável. Departamento de Design da UFPR, 2021. http://dx.doi.org/10.5380/8sds2021.art14.
Повний текст джерелаLee, Sooyong, Sangkyou Lee, Jolanta Bondaruk, Hua Wang, Shizhen Zhang, Menashe Bar-Eli, Peter Black, et al. "Abstract 3065: Loss of ARL11 function promotes growth ofin situneoplasia by activating the ras pathway." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3065.
Повний текст джерела"СЕМЕЙНЫЕ АРХИВЫ. «БОЛЬШАЯ» ИСТОРИЯ В ЧАСТНОМ ПРЕЛОМЛЕНИИ". У НА ПЕРЕКРЕСТКЕ СЕВЕРА И ВОСТОКА (МЕТОДОЛОГИИ И ПРАКТИКИ РЕГИОНАЛЬНОГО РАЗВИТИЯ). Science and Innovation Center Publishing House, 2023. http://dx.doi.org/10.12731/978-5-907608-10-8-art14.
Повний текст джерелаЗвіти організацій з теми "Arl14"
Divan, Brooke, Richard Lampo, and Lawrence Clark. Demonstration of a standard steel coating system modified with a vapor-phase corrosion inhibitor : final report on Project F12-AR14. Engineer Research and Development Center (U.S.), September 2018. http://dx.doi.org/10.21079/11681/29516.
Повний текст джерела