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Книги з теми "Animal models"

Щоб переглянути інші типи публікацій з цієї теми, перейдіть за посиланням: Animal models.

Автор: Grafiati

Опубліковано: 4 червня 2021

Оновлено: 28 липня 2024

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1

National Institute on Alcohol Abuse and Alcoholism (U.S.), ed. Animal models. [Rockville, Md.]: Public Health Service, National Institutes of Health, 2000.

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2

Joost, Ruitenberg E., and Peters P. W. J, eds. Laboratory animals: Laboratory animal models for domestic animal production. Amsterdam: Elsevier Science Publishers, 1986.

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3

Davies, Jamie, ed. Replacing Animal Models. Chichester, UK: John Wiley & Sons, Ltd, 2012. http://dx.doi.org/10.1002/9781119940685.

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4

Technology Information Forecasting and Assessment Council (India), ed. Transgenic animal models. New Delhi: Technology Information, Forecasting, and Assessment Council, 2003.

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5

1948-, Gad Shayne C., and Chengelis Christopher P. 1949-, eds. Animal models in toxicology. New York: M. Dekker, 1992.

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6

King, Aileen J. F., ed. Animal Models of Diabetes. New York, NY: Springer US, 2020. http://dx.doi.org/10.1007/978-1-0716-0385-7.

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7

Risling, Mårten, and Johan Davidsson, eds. Animal Models of Neurotrauma. New York, NY: Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9711-4.

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8

Sharpe-Timms, Kathy L., ed. Animal Models for Endometriosis. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-51856-1.

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9

Ma, Chao, and Jun-Ming Zhang, eds. Animal Models of Pain. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60761-880-5.

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10

De Deyn, Peter Paul, and Debby Van Dam, eds. Animal Models of Dementia. Totowa, NJ: Humana Press, 2011. http://dx.doi.org/10.1007/978-1-60761-898-0.

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11

Olivier, B., J. Mos, and J. L. Slangen, eds. Animal Models in Psychopharmacology. Basel: Birkhäuser Basel, 1991. http://dx.doi.org/10.1007/978-3-0348-6419-0.

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12

Koob, George F., Cindy L. Ehlers, and David J. Kupfer, eds. Animal Models of Depression. Boston, MA: Birkhäuser Boston, 1989. http://dx.doi.org/10.1007/978-1-4684-6762-8.

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13

Baraban, Scott C., ed. Animal Models of Epilepsy. Totowa, NJ: Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-263-6.

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14

1947-, Baker Glen B., Boulton A. A, and Martin-Iverson Mathew Thomas 1956-, eds. Animal models in psychiatry. Clifton, N.J: Humana Press, 1991.

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15

Osteoporosis research: Animal models. London: Springer, 2011.

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16

Vijayakumar Sreelatha, Harikrishnan, Satish Patel, and Perumal Nagarajan, eds. Animal Models in Research. Singapore: Springer Nature Singapore, 2024. http://dx.doi.org/10.1007/978-981-97-0048-6.

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17

1948-, Gad Shayne C., ed. Animal models in toxicology. 2nd ed. Boca Raton: Taylor & Francis, 2006.

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18

F, Koob George, Ehlers Cindy L, Kupfers David J. 1941-, and MacArthur Foundation Research Network on the Psychobiology of Depression., eds. Animal models of depression. Boston: Birhäuser, 1989.

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19

Huub, Schellekens, Horzinek Marian C, and Nederlandse Organisatie voor Toegepast-Natuurwetenschappelijk Onderzoek., eds. Animal models in AIDS. Amsterdam: Elsevier, 1990.

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20

Berend, Olivier, Mos J, Slangen Jef L, and International Congress of Pharmacology (11th : 1990 : Amsterdam, Netherlands), eds. Animal models in psychopharmacology. Basel: Birkhäuser, 1991.

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21

Debby, Van Dam, ed. Animal Models of Dementia. Totowa, NJ: Humana Press, 2011.

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22

F, Sima Anders A., and Shafrir Eleazar, eds. Animal models of diabetes: A primer. Amsterdam: Harwood Academic, 2001.

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23

Animal models in cardiovascular research. Boston: Nijhoff, 1985.

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24

1937-, Miyaji Makoto, ed. Animal models in medical mycology. Boca Raton, Fla: CRC Press, 1987.

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25

Gross, David R. Animal models in cardiovascular research. 3rd ed. Dordrecht: Springer, 2009.

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26

Avena, Nicole M. Animal models of eating disorders. Totowa [N.J.]: Humana Press, 2013.

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27

Boulton, Alan A., Glen B. Baker, and Mathew T. Martin-Iverson. Animal Models in Psychiatry, II. New Jersey: Humana Press, 1991. http://dx.doi.org/10.1385/0896031772.

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28

Boulton, Alan A., Glen B. Baker, and Mathew T. Martin-Iverson. Animal Models in Psychiatry, I. New Jersey: Humana Press, 1991. http://dx.doi.org/10.1385/0896031985.

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29

Boulton, Alan A., Glen B. Baker, and Peter H. Wu. Animal Models of Drug Addiction. New Jersey: Humana Press, 1992. http://dx.doi.org/10.1385/0896032175.

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30

Alper, Joe, Lida Anestidou, and Jenna Ogilvie, eds. Animal Models for Microbiome Research. Washington, D.C.: National Academies Press, 2018. http://dx.doi.org/10.17226/24858.

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31

Gross, David. Animal Models in Cardiovascular Research. New York, NY: Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-95962-7.

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32

Martínez Murillo, Ricardo, and Alfredo Martínez, eds. Animal Models of Brain Tumors. Totowa, NJ: Humana Press, 2013. http://dx.doi.org/10.1007/978-1-62703-209-4.

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33

Gross, David R. Animal Models in Cardiovascular Research. Dordrecht: Springer Netherlands, 1985. http://dx.doi.org/10.1007/978-94-009-5006-1.

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34

Pittenger, Christopher, Stephanie Dulawa, and Summer L. Thompson. Animal Models of OCD. Edited by Christopher Pittenger. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228163.003.0029.

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Анотація:

Obsessive-compulsive disorder and related conditions are characterized by demonstrable alterations in brain function, and aspects of these may, in principle, be recapitulated and studied in animals. However, the relationship between animal models and the clinical syndrome is complex. Many clinical aspects of OCD, especially those that can only be evaluated by subjective report, cannot be assessed in an animal. As a result, some discount the utility of animal modeling of OCD altogether. However, conservation of both genes and brain anatomy across mammalian species supports the opposite perspective, that key aspects of the pathophysiology of OCD and related disorders can be recapitulated in animals, and thus fruitfully studied in model systems. This introductory chapter addresses these issues, seeking to identify both the strengths and the limitations of animal studies as contributors to our understanding of OCD. This discussion provides a framework for the more specific material about particular animal models presented in this section.

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35

Animal models. [Rockville, Md.]: Public Health Service, National Institutes of Health, 2000.

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36

Torres-Ruiz, Raul, and Sandra Rodriguez-Perales. CRISPR in Animals and Animal Models. Elsevier Science & Technology Books, 2017.

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37

Torres-Ruiz, Raul, and Sandra Rodriguez-Perales. CRISPR in Animals and Animal Models. Elsevier Science & Technology Books, 2017.

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38

Crabbe, John C., and Tamara J. Phillips. Genetic Animal Models. Edited by Kenneth J. Sher. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199381678.013.003.

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Genetic animal models have a long history of contribution to the understanding of alcohol dependence. Studies have provided insight into genetic contributions to risk and are beginning to be used to suggest better targeted pharmacotherapies. This review discusses how different species and genetic methods have been used to target specific aspects of alcohol dependence. It identifies areas of future promise and those in which more information is needed. It considers how genetic and environmental sources interact to affect risk and treatment response. The review concentrates on alcohol because the majority of data have come from alcohol studies, but it also gives examples of generalization of the alcohol work to other drugs of abuse and identifies some areas where drug-specific factors are important.

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39

Baglietto-Vargas, David, Rahasson R. Ager, Rodrigo Medeiros, and Frank M. LaFerla. Animal Models of Neurodegenerative Diseases. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190233563.003.0014.

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The incidence and prevalence of neurodegenerative disorders (e.g., Alzheimer’s disease (AD), Parkinson’s disease (PD), and Huntington’s disease (HD), etc.) are growing rapidly due to increasing life expectancy. Researchers over the past two decades have focused their efforts on the development of animal models to dissect the molecular mechanisms underlying neurodegenerative disorders. Existing models, however, do not fully replicate the symptomatic and pathological features of human diseases. This chapter focuses on animal models of AD, as this disorder is the most prevalent of the brain degenerative conditions afflicting society. In particular, it briefly discusses the current leading animal models, the translational relevance of the preclinical studies using such models, and the limitations and shortcomings of using animals to model human disease. It concludes with a discussion of potential means to improve future models to better recapitulate human conditions.

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40

Perkins, Elizabeth C., Shaun P. Brothers, and Charles B. Nemeroff. Animal Models for Post-Traumatic Stress Disorder. Edited by Charles B. Nemeroff and Charles R. Marmar. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190259440.003.0024.

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Animal models of post-traumatic stress disorder (PTSD) provide a wellspring of biological information about this complex condition by providing the opportunity to manipulate trauma exposure and measure biological outcomes in a systematic manner that is not possible in clinical studies. Symptoms of PTSD may be induced in animals by physical (immobilization, foot shock, underwater stress) and psychological stressors (exposure to predator, social defeat, early life trauma) or a combination of both. In addition, genetic, epigenetic and transgenic models have been created by breeding animals with a behavioral propensity for maladaptive stress response or by directly manipulating genes that have been implicated in PTSD. The effect of stressors in animals is measured by a variety of means, including observation of behavior, measurement of structural alterations in the brain and of physiological markers such as HPA axis activity and altered gene expression of central nervous system neurotransmitter system components including receptors. By comparing changes observed in stress exposed animals to humans with PTSD and by comparing animal response to treatments that are effective in humans, we can determine the validity of PTSD animal models. The identification of a reliable physiological marker of maladaptive stress response in animals as well as standard use of behavioral cutoff criteria are critical to the development of a valid animal model of PTSD.

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41

Dodman, Nicholas H., and Louis Shuster. Spontaneously Occurring Animal Models of OCD. Edited by Christopher Pittenger. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228163.003.0032.

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This chapter summarizes what we know about compulsive behavioral disorders in several animal species. Animals can develop repetitive behaviors in a range of circumstances, generally associated with anxiety or stress. It is increasingly apparent that these behaviors recapitulate core features of obsessive-compulsive disorder. They are clearly partially genetic; for example, specific breeds of dog are susceptible to specific compulsive behavioral disorders. Understanding such OCD-like behaviors provides a potentially fruitful avenue towards understanding OCD in humans. This chapter reviews this literature, emphasizing the points of parallelism between repetitive behavior syndromes in animals and human disease. Recent advances in our understanding of the biology of these spontaneously occurring animal models, especially in dogs, have great potential to elucidate the pathophysiology of OCD.

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42

Gad, Shayne C. Animal Models in Toxicology. 2nd ed. CRC, 2006.

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43

Animal models in toxicology. 2nd ed. Boca Raton, FL: CRC Press/Taylor & Francis, 2006.

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44

Keehn, J. D. Animal Models for Psychiatry. Taylor & Francis Group, 2018.

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45

Lu, Bingwei. Animal Models of Neurodegeneration. Wiley & Sons, Incorporated, John, 2016.

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46

Gad, Shayne C., ed. Animal Models in Toxicology. CRC Press, 2006. http://dx.doi.org/10.1201/9781420014204.

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47

Christopher, Evans J., Brigitte L. Kieffer, David Jentsch, and Rafael J. Maldonado. Animal Models of Addiction. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0043.

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Drug addiction, now officially diagnosed as substance use disorder (SUD), is a chronic brain syndrome characterized by the compulsive use of drugs, loss of control over drug taking in spite of its adverse consequences, and relapse even after long periods of drug abstinence. Animal models have played a critical role in our understanding of the molecules, circuits, and behaviors associated with substance use disorders. This chapter reviews animal models that have been widely used to assess all stages of the addiction cycle: from drug initiation, through drug seeking, to withdrawal and relapse. We discuss the power of genetics, especially in generating rodent models for the discovery of essential proteins and pathways regulating behaviors exhibited during the different stages of the addiction cycle. Preclinical research in animal models will undoubtedly continue to reveal therapeutic strategies for substance use disorders.

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48

Gad, Shayne C., and Shayne C. Gad, eds. Animal Models in Toxicology. CRC Press, 2016. http://dx.doi.org/10.1201/b18705.

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49

Animal models in psychiatry. Clifton, NJ: Humana, 1991.

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50

Keehn, J. D. Animal Models for Psychiatry. Routledge, 2018. http://dx.doi.org/10.4324/9780203712436.

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