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Статті в журналах з теми "Alzheimers’s disease"
Zhou, Can-Can, Zhen-Yan Gao, Ju Wang, Mei-Qin Wu, Shuang Hu, Fei Chen, Jun-Xia Liu, Hui Pan, and Chong-Huai Yan. "Lead exposure induces Alzheimers’s disease (AD)-like pathology and disturbes cholesterol metabolism in the young rat brain." Toxicology Letters 296 (October 2018): 173–83. http://dx.doi.org/10.1016/j.toxlet.2018.06.1065.
Повний текст джерелаSun, Binggang, Kay Chow, Amy Zhao, Jason Lehmann, Weiping Jiang, and Shaoquan Ji. "Novel Flow-Based Multiplex Assay Panel for Quantifying Human Neuroinflammation Biomarkers." Journal of Immunology 202, no. 1_Supplement (May 1, 2019): 130.25. http://dx.doi.org/10.4049/jimmunol.202.supp.130.25.
Повний текст джерелаZarrouk, Amira, Meryam Debbabi, Maryem Bezine, El Mostafa Karym, Asmaa Badreddine, Olivier Rouaud, Thibault Moreau, et al. "Lipid Biomarkers in Alzheimer's Disease." Current Alzheimer Research 15, no. 4 (February 22, 2018): 303–12. http://dx.doi.org/10.2174/1567205014666170505101426.
Повний текст джерелаNesteruk, Marta, Tomasz Nesteruk, Maria Styczyńska, and Maria Barcikowska. "Wpływ obciążenia chorobami naczyniowymi na progresję łagodnych zaburzeń poznawczych do choroby Alzheimera." Aktualności Neurologiczne 15, no. 1 (April 30, 2015): 18–21. http://dx.doi.org/10.15557/an.2015.0003.
Повний текст джерелаHemangkorn, Nicha, Pamonphon Phummai, and Patranan Punyacharoen. "Effects of Essential Oils and Aromatic Plants on Alzheimer’s Disease and Dementia." International Journal of Science and Healthcare Research 6, no. 3 (September 3, 2021): 350–58. http://dx.doi.org/10.52403/ijshr.20210760.
Повний текст джерелаKyselova, A. A., E. S. Kravtsova, D. O. Mishchenko, and E. R. Chernishova. "The Modern View on Alzheimer’s Disease." Ukraïnsʹkij žurnal medicini, bìologìï ta sportu 3, no. 4 (May 18, 2018): 169–73. http://dx.doi.org/10.26693/jmbs03.04.169.
Повний текст джерелаMonisha, M., K. M. Harshitha, N. H. Dhanalakshmi, Kokatam Sai Prakash Reddy, C. R. Nagarathna, and M. Kusuma. "Early detection of Alzheimer’s: Modalities and Methods." March 2022 4, no. 1 (May 4, 2022): 69–79. http://dx.doi.org/10.36548/jaicn.2022.1.005.
Повний текст джерелаBombón-Albán, Paulina E., and Pablo E. Fierro-Altamirano. "Criterios de diagnóstico de la enfermedad de Alzheimer: Aplicaciones prácticas." Revista Ecuatoriana de Neurologia 31, no. 1 (May 10, 2022): 12–14. http://dx.doi.org/10.46997/revecuatneurol31100012.
Повний текст джерелаShahzadi, Maria, Bareera Saeed, Muhammad Azzam Khan, Amna Rashid, Muhammad Bilal, Roma Imtiaz, and Tallat Anwar Faridi. "A Review on the Techniques for Early Diagnosis of Alzheimer’s Disease." Lahore Garrison University Journal of Life Sciences 6, no. 03 (September 15, 2022): 268–81. http://dx.doi.org/10.54692/lgujls.2022.0603228.
Повний текст джерелаFiore, Vincenzo, Antonia De Rosa, Paolo Falasca, Massimo Marci, Edoardo Guastamacchia, Brunella Licchelli, Vito Angelo Giagulli, Giovanni De Pergola, Antonella Poggi, and Vincenzo Triggiani. "Focus on the Correlations between Alzheimer’s Disease and Type 2 Diabetes." Endocrine, Metabolic & Immune Disorders - Drug Targets 19, no. 5 (June 3, 2019): 571–79. http://dx.doi.org/10.2174/1871530319666190311141855.
Повний текст джерелаДисертації з теми "Alzheimers’s disease"
Holt, Jim. "Alzheimer’s Disease." Digital Commons @ East Tennessee State University, 2009. https://dc.etsu.edu/etsu-works/6482.
Повний текст джерела梁欣珮 and Yan-pui Irene Leung. "Potential impact of alzheimer's disease on retina." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B42905059.
Повний текст джерелаHolt, Jim, J. Guduru, and S. Pathi. "Alzheimer’s Disease, 2nd Revision." Digital Commons @ East Tennessee State University, 2015. https://dc.etsu.edu/etsu-works/6478.
Повний текст джерелаHolt, Jim, J. Guduru, M. Medipally, and S. Pathi. "Alzheimer’s Disease, 1st Revision." Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/etsu-works/6480.
Повний текст джерелаRickle, Annika. "PTEN and Akt signalling in Alzheimer's disease /." Stockholm : Karolinska institutet, 2005. http://diss.kib.ki.se/2005/91-7140-514-3/.
Повний текст джерелаHolt, Jim, Christopher T. Bridges, and Christian B. Potter. "Dementia (Alzheimer’s Disease), 3rd Revision." Digital Commons @ East Tennessee State University, 2019. https://dc.etsu.edu/etsu-works/6472.
Повний текст джерелаDomingues, Catarina de Barros Pinto Salvador. "Chemokines impact in Alzheimer’s disease." Master's thesis, Universidade de Aveiro, 2015. http://hdl.handle.net/10773/16081.
Повний текст джерелаAlzheimer’s Disease (AD) is a neurodegenerative disorder neuropathologically characterized by the presence of extracellular senile plaques, intracellular neurofibrillary tangles and synaptic loss. Neuroinflammation has been associated with some neurodegenerative diseases, such as AD. In AD, increased Aβ production and aggregation, have a fundamental role in the activation of the inflammatory process. In turn, this could be fundamental in the early stages of this pathology, regarding the Aβ clearance and brain protection. However, chronic inflammation leads to an increase of the inflammatory mediators, such as cytokines, released by activated microglia, astrocytes, and neurons. The excessive production of these inflammatory components promotes alterations in both amyloid precursor protein (APP) expression and processing, stimulating the increase of Aβ accumulation and abnormal tau phosphorylation. This results in neurotoxic effects, irreversible damage and neuronal loss. Chronic inflammation is a feature of AD however, little is known about the effects of some chemokines on its pathogenesis. Thus, the main aim of this thesis was to study the impact of the interleukin-8 (IL-8) and monocyte chemoattractant protein-1 (MCP-1) on apoptosis, APP and tau. The both studied chemokines resulted in small alterations regarding the cytotoxicity on SH-SY5Y differentiated cells, being a significant increase in apoptosis observed only for the MCP-1 at the highest concentration. For the APP processing no significant differences were obtained, although a tendency to increase at different concentrations and periods was registered for both IL-8 and MCP-1. With respect to tau and other cytoskeleton-associated proteins, it was possible to observe a tendency to increase in the phosphorylated residue (Ser396) at the higher concentrations, as well as alterations on actin and tubulin with an increase on acetylated-α tubulin. This effect can be translated by neuronal architectural and survival alterations. Therefore additional studies could contribute to a better understanding of the way that these chemokines act on AD pathogenesis.
A Doença de Alzheimer (DA) é uma doença neurodegenerativa, caracterizada pela presença de placas senis extracelulares, tranças neurofibrilares intracelulares e perda sináptica. A neuroinflamação tem sido associada com algumas doenças neurodegenerativas, tal como a DA. Na DA, a produção e agregação aumentada do péptido Aβ, tem um papel fundamental na activação do processo inflamatório, que pode ser importante nas fases iniciais da doença, devido à remoção de Aβ e à proteção do cérebro. No entanto, uma inflamação crónica leva a um aumento de mediadores inflamatórios como são as citocinas, libertadas por microglia activada, astrócitos e neurónios. A produção excessiva de componentes inflamatórios promove alterações tanto na expressão como no processamento da proteína percursora amilóide (APP), levando a uma maior acumulação de Aβ e fosforilação anormal da proteína tau. Isto resulta em efeitos neurotóxicos, dano irreversível e perda neuronal. A inflamação crónica é uma característica da DA, no entanto pouco se sabe sobre os efeitos de algumas quimiocinas na sua patogénese. Assim, o principal objectivo desta tese foi o estudo do impacto da IL-8 e da MCP-1 na apoptose, APP e tau. Ambas as quimiocinas em estudo resultaram em pequenas alterações ao nível da citotoxicidade de células SH-SY5Y diferenciadas, tendo sido apenas observado um aumento significativo da apoptose para MCP-1 à concentração mais elevada. Relativamente ao processamento de APP, não foram observadas alterações significativas, no entanto alguma tendência para aumentar a diferentes concentrações e períodos foi obtida tanto para a IL-8 como para a MCP-1. Ao nível da tau e outras proteínas associadas ao citoesqueleto, foi possível observar uma tendência de aumento do resíduo fosforilado Ser396 às concentrações mais elevadas assim como alterações na actina e tubulina, com um aumento da αtubulina acetilada. Este efeito pode ser traduzido em alterações na arquitetura e sobrevivência neuronal. Assim sendo, estudos adicionais podem contribuir para uma melhor compreensão do modo de ação destas quimiocinas na patogénese da DA.
Barra, Cátia Isabel de Almeida. "Inflammatory biomarkers in Alzheimer’s disease." Master's thesis, Universidade de Aveiro, 2014. http://hdl.handle.net/10773/13277.
Повний текст джерелаAlzheimer’s disease (AD) is the most common form of dementia. Histopathologically it is characterized by the presence of two major hallmarks, the intracellular neurofibrillary tangles (NFT) and the extracellular senile plaques (SP), which are surrounded by activated astrocytes and microglia. Neuroinflammation has been associated with some neurodegenerative diseases. In AD the inflammatory process, prompted by increased Aβ production and aggregation, was reported to have a fundamental role in disease pathogenesis. In early stages the inflammation could have a beneficial role in the pathology, since it has been proposed that the microglia and astrocytes activated could be involved in (amyloid β) Aβ clearance. Nevertheless, the chronic activation of the microglia leads to excessive production of the inflammatory components, including cytokines. It promotes alterations in amyloid precursor protein (APP) expression and processing, stimulating the increase of Aβ accumulation, abnormal Tau phosphorylation and, consequently, neurotoxic effects, irreversible damage and loss of neurons. Since chronic neuroinflammation is a feature of AD, inflammatory proteins may constitute potential biomarkers candidates to assist clinical diagnosis of this dementia. Thus, the main aim of this study was to identify putative inflammatory biomarkers for AD by flow citometry analysis. For plasma samples of individuals examined by clinical dementia rating (CDR) and mini mental (MM) diagnostic tests were used. Subjects were subdivided in 3 distinct groups, a control group (CDR-/MM-) and two patient groups, CDR+/MM- and CRD+/MM+, the former may include mild cognitive impairment (MCI) patients and the latest group included 5 patients clinical diagnosed as AD. Data analysis revealed differences in the inflammatory proteins levels of both patients groups (CDR+/MM- and CDR+/MM+) in comparison to healthy individuals (CDR-/MM-). Interleukin-8 (IL-8) plasma levels were statistically different (P<0,05) from control group. Significant correlation between IL-8 concentrations and the CDR stages was also identified. Additionally, correlations of monocyte chemoattractant protein-1 (MCP-1) with both IL-8 and IL-6 were observed. Taken together these findings suggested that IL-8 could be a potential biomarker not only for AD but also for diagnosis of initial stages of dementia.
A doença de Alzheimer (DA) é o tipo de demência mais comum. Histopatologicamente é caracterizada pela presença de tranças neurofibrilares intracelulares (TNF) e de placas senis extracelulares (PS), as quais estão rodeadas pela microglia e por astrócitos. A neuroinflamação tem sido associada com várias doenças neurodegenerativas. Na DA o processo inflamatório, desencadeado pelo aumento da produção e agregação do péptido Aβ, desempenha um papel fundamental na patogénese da doença. Nas fases inicias, a inflamação possui um papel benéfico na patologia, uma vez que tem sido proposto que a microglia e os astrócitos quando ativados estão envolvidos na remoção de β-amilóide (Aβ). No entanto, a ativação crónica da microglia conduz à produção excessiva de componentes inflamatórios, incluindo citocinas. Isto provoca alterações na expressão e processamento da proteína percursora de amilóide (PPA), estimulando o aumento da produção e acumulação de Aβ, fosforilação anormal da proteína Tau e, consequentemente, efeitos neurotóxicos e perda de neurónios. Uma vez que a neuroinflamação crónica é uma característica da DA, proteínas inflamatórias poderão constituir potenciais candidatos a biomarcadores que auxiliem no diagnóstico clínico desta doença. Desta forma, o principal objectivo deste trabalho foi identificar biomarcadores inflamatórios para a DA através da técnica de citometria de fluxo. Para tal, foram analisadas amostras de plasma de doentes que foram, previamente, examinados por testes de avaliação cognitiva, clinical dementia rating (CDR) e mini mental (MM). Os sujeitos foram divididos em três grupos distintos, o grupo controlo (CDR-/MM-) e dois grupos de pacientes, CDR+/MM- e CDR+/MM+. O primeiro grupo de pacientes pode conter indivíduos com ligeiras alterações cognitivas (MCI) e o segundo inclui 5 pacientes clinicamente diagnosticados para DA. A análise dos dados revelou diferenças nos níveis de proteínas inflamatórias de ambos os grupos de doentes (CDR+/MM- e CDR+/MM+) em comparação com os indivíduos saudáveis (CDR-/MM-). Os níveis plasmáticos de interleucina-8 (IL-8) foram estatisticamente deferentes (p<0,05) do grupo controlo. Correlação significativa entre as concentrações de IL-8 e os estados de CDR foi identificada. Adicionalmente, foram observadas correlações entre MCP-1 e IL-8 e a IL-6. Em conjunto, estes resultados sugerem que a IL-8 poderá ser um potencial biomarcador não só para a DA mas também para o diagnóstico precoce de demência.
Navaratnam, Dasakumar Selveraj. "Cholinesterases in Alzheimer's disease." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.306734.
Повний текст джерелаNewman, Tracey Anne. "Ageing and Alzheimer's disease." Thesis, University of Southampton, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.246220.
Повний текст джерелаКниги з теми "Alzheimers’s disease"
Altman, Harvey J., ed. Alzheimer’s Disease. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4615-6414-0.
Повний текст джерелаJha, Arun, and Kaushik Mukhopadhaya. Alzheimer’s Disease. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-56739-2.
Повний текст джерелаSisodia, Sangram S., and Rudolph E. Tanzi, eds. Alzheimer’s Disease. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-35135-3.
Повний текст джерелаHarris, J. Robin, and Falk Fahrenholz, eds. Alzheimer’s Disease. Boston, MA: Springer US, 2005. http://dx.doi.org/10.1007/b100942.
Повний текст джерелаTurner, J. D., K. Beyreuther, and F. Theuring, eds. Alzheimer’s Disease. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-662-03248-0.
Повний текст джерелаChun, Jerold, ed. Alzheimer’s Disease. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2655-9.
Повний текст джерелаParks, Peggy J. Alzheimer's disease. San Diego: ReferencePoint Press, 2009.
Знайти повний текст джерелаSammons, Vivian O. Alzheimer's disease. Washington, D.C: Science Reference Section, Science and Technology Division, Library of Congress, 1987.
Знайти повний текст джерелаDempsey, Denise P. Alzheimer's disease. Washington, D.C: Science Reference Section, Science and Technology Division, Library of Congress, 1997.
Знайти повний текст джерелаParliament, Canada Library of. Alzheimer's Disease. Ottawa: Libray of Parliament, 1993.
Знайти повний текст джерелаЧастини книг з теми "Alzheimers’s disease"
Denham, Nathan C., James A. R. Nicoll, and Delphine Boche. "Synapses and Alzheimers’s Disease: Effect of Immunotherapy?" In Folding for the Synapse, 269–87. Boston, MA: Springer US, 2010. http://dx.doi.org/10.1007/978-1-4419-7061-9_14.
Повний текст джерелаGregori, Maria, and Francesca Re. "Neurodegenerative Diseases - Alzheimer's Disease." In Pharmaceutical Nanotechnology: Innovation and Production, 649–60. Weinheim, Germany: Wiley-VCH Verlag GmbH & Co. KGaA, 2016. http://dx.doi.org/10.1002/9783527800681.ch27.
Повний текст джерелаFuller, Stephanie J., Hamid R. Sohrabi, Kathryn G. Goozee, Anoop Sankaranarayanan, and Ralph N. Martins. "Alzheimer's Disease and Other Neurodegenerative Diseases." In Neurodegeneration and Alzheimer's Disease, 9–42. Chichester, UK: John Wiley & Sons, Ltd, 2019. http://dx.doi.org/10.1002/9781119356752.ch2.
Повний текст джерелаWakabayashi, Tomoko, Takeshi Iwatsubo, and Bart De Strooper. "The Biology of the Presenilin Complexes." In Alzheimer’s Disease, 35–58. Boston, MA: Springer US, 2007. http://dx.doi.org/10.1007/978-0-387-35135-3_3.
Повний текст джерелаJha, Arun, and Kaushik Mukhopadhaya. "Memory, Cognitive Impairment and Dementia." In Alzheimer’s Disease, 1–20. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-56739-2_1.
Повний текст джерелаJha, Arun, and Kaushik Mukhopadhaya. "Dementia Due to Alzheimer’s Disease (AD)." In Alzheimer’s Disease, 21–30. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-56739-2_2.
Повний текст джерелаJha, Arun, and Kaushik Mukhopadhaya. "Dementia Assessment." In Alzheimer’s Disease, 31–47. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-56739-2_3.
Повний текст джерелаJha, Arun, and Kaushik Mukhopadhaya. "RUDAS Cognitive Scale." In Alzheimer’s Disease, 49–56. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-56739-2_4.
Повний текст джерелаJha, Arun, and Kaushik Mukhopadhaya. "Diagnosis." In Alzheimer’s Disease, 57–64. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-56739-2_5.
Повний текст джерелаJha, Arun, and Kaushik Mukhopadhaya. "Supporting Diagnosis and Treatment." In Alzheimer’s Disease, 65–85. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-56739-2_6.
Повний текст джерелаТези доповідей конференцій з теми "Alzheimers’s disease"
Collar, Giovanna Carello, Marco Antônio De Bastiani, and Eduardo R. Zimmer. "HUNTINGTON’S DISEASE AND EARLYONSET ALZHEIMER’S DISEASE SHARE A TRANSCRIPTOMIC SIGNATURE." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda082.
Повний текст джерелаAntonijević, Marko, Žiko Milanović, Edina Avdović, Dušica Simijonović, and Zoran Marković. "ANOTHER LOOK AT THE BIOLOGICAL ROLES OF A PLANT ALKALOID-BERBERINE." In XXVII savetovanje o biotehnologiji. University of Kragujevac, Faculty of Agronomy, 2022. http://dx.doi.org/10.46793/sbt27.455a.
Повний текст джерелаXavier, Lais de Paiva, Gabriella de Andrade Moreira, Luana Maria Amaral Cherain, Lucas Ferreira Flávio Souza, Tânia Maria da Silva Novaretti, and Valdeci de Oliveira Santos Rigolin. "Preliminary study on the number of deaths from neurodegenerative diseases in former Brazilian soccer players." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.212.
Повний текст джерелаOliveira, Leonardo Santana Ramos, Lucca Ribeiro Alves, and Rodrigo Bartolomeu Sobral Neves. "Mortality of chronic-degenerative and infectious neurological disorders from 2009 to 2019: follow-up of epidemiological transition." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.527.
Повний текст джерелаChan, C., and S. Baek. "Model Based Surface Design to Incorporate the Effect of Soluble Cues." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53439.
Повний текст джерелаFaria, Gustavo Hugo de Souza. "The impact of epigenetics on the development of neurodegenerative diseases." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.654.
Повний текст джерелаRumao, Priti, and Mamta Padole. "Alzheimer's Disease." In DSMLAI '21': International Conference on Data Science, Machine Learning and Artificial Intelligence. New York, NY, USA: ACM, 2021. http://dx.doi.org/10.1145/3484824.3484886.
Повний текст джерелаRizzi, Liara, and Marcio Balthazar. "THE SUSPECTED NON-ALZHEIMER’S DISEASE PATHOPHYSIOLOGY." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda070.
Повний текст джерела"Hybrid CNN-SVM for Alzheimer's Disease Classification from Structural MRI and the Alzheimer’s Disease Neuroimaging Initiative (ADNI)." In 2018 International Conference on Biomedical Engineering, Machinery and Earth Science. Francis Academic Press, 2018. http://dx.doi.org/10.25236/bemes.2018.041.
Повний текст джерелаElcin, Huseyn. "EARLY IDENTIFICATION OF THE NEUROLOGICAL COMPLICATIONS OF DIABETES MELLITUS." In International Trends in Science and Technology. RS Global Sp. z O.O., 2021. http://dx.doi.org/10.31435/rsglobal_conf/30032021/7474.
Повний текст джерелаЗвіти організацій з теми "Alzheimers’s disease"
Lattaf, Sara, Lamiaa Abdallaoui, and Amal Bouziane. Effect of periodontal disease on Alzheimer’s disease: protocol of a systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2020. http://dx.doi.org/10.37766/inplasy2020.8.0033.
Повний текст джерелаMehegan, Laura. Alzheimer's Disease and Dementia Awareness Poll 2018. AARP Research, June 2018. http://dx.doi.org/10.26419/res.00232.001.
Повний текст джерелаChandra, Amitabh, Courtney Coile, and Corina Mommaerts. What Can Economics Say About Alzheimer's Disease? Cambridge, MA: National Bureau of Economic Research, August 2020. http://dx.doi.org/10.3386/w27760.
Повний текст джерелаKamaruzzaman, Mohd Amir, Muhammad Hibatullah Romli, Razif Abas, Sharmili Vidyadaran, Mohamad Taufik Hidayat Baharuldin, Muhammad Luqman Nasaruddin, Vishnnumukkala Thirupathirao, et al. Impact of Endocannabinoid Mediated Glial Cells on Cognitive Function in Alzheimer’s Disease: A Systematic Review and Meta-Analysis of Animal Studies. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, August 2022. http://dx.doi.org/10.37766/inplasy2022.8.0094.
Повний текст джерелаMintzer, Jacobo E., D. L. Bachman, M. Stuckey, M. Ebeling, M. T. Wagner, W. J. Evans, V. Hirth, A. Walker, R. Joglekar, and W. Faison. A State-Wide Research Network for Alzheimer's Disease. Office of Scientific and Technical Information (OSTI), March 2014. http://dx.doi.org/10.2172/1123171.
Повний текст джерелаMehegan, Laura. Alzheimer's Disease and Dementia Awareness Poll 2018: Infographic. AARP Research, June 2018. http://dx.doi.org/10.26419/res.00232.002.
Повний текст джерелаPericak-Vance, Margaret A. Whole Exome Analysis of Early Onset Alzheimer's Disease. Fort Belvoir, VA: Defense Technical Information Center, April 2013. http://dx.doi.org/10.21236/ada602412.
Повний текст джерелаPericak-Vance, Margaret A. Whole Exome Analysis of Early Onset Alzheimer's Disease. Fort Belvoir, VA: Defense Technical Information Center, April 2014. http://dx.doi.org/10.21236/ada603027.
Повний текст джерелаMoran, Marcel Moran. Retrospective: Alzheimer’s Disease Research: An Overview of 1998-2015 Grantmaking. San Francisco, CA United States: S. D. Bechtel, Jr. Foundation, November 2017. http://dx.doi.org/10.15868/socialsector.37561.
Повний текст джерелаHan, Nana, Yang Fang, Guozhen Zhao, and Bo Ji. The comparative efficacy and safety of acupuncture for mild and moderate Alzheimer's disease: A systematic review and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2021. http://dx.doi.org/10.37766/inplasy2021.11.0014.
Повний текст джерела