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Книги з теми "ALS Amyotrophic"

Щоб переглянути інші типи публікацій з цієї теми, перейдіть за посиланням: ALS Amyotrophic.

Автор: Grafiati

Опубліковано: 10 грудня 2022

Оновлено: 28 січня 2023

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Ознайомтеся з топ-50 книг для дослідження на тему "ALS Amyotrophic".

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1

Respiratory management of ALS: Amyotrophic lateral sclerosis. Sudbury, Mass: Jones and Bartlett, 2009.

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2

Guion, Lee. Respiratory management of ALS: Amyotrophic lateral sclerosis. Sudbury, Mass: Jones and Bartlett, 2010.

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3

Behind those eyes: Our journey with ALS. Renfrew, Ont: General Store Publishing House, 2013.

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4

ALS: Lou Gehrig's disease: Patient advocate. Riverside, CA: Health Scouter - Equity Press, 2009.

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5

author, Silverman Jennifer, and Holt Kim illustrator, eds. My dad has ALS? Place of publication not identified]: [Bookbaby], 2015.

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6

Stinton, William M. I choose to live: A journey through life with ALS. Gurnee, IL: Banbury Pub., 2003.

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7

Stinton, William M. I choose to live: A journey through life with ALS. Gurnee, IL: Banbury Pub., 2003.

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8

I choose to live: A journey through life with ALS. Gurnee, IL: Banbury Pub., 2003.

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9

Kaye, Dennis. Laugh, I thought I'd die: My life with ALS. Toronto: Penguin, 1993.

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10

ALS Registry Act: Report (to accompany H.R. 2295) (including cost estimate of the Congressional Budget Office). [Washington, D.C: U.S. G.P.O., 2007.

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11

Massachusetts. Bureau of Environmental Health. Environmental Epidemiology Program. The Amyotrophic Lateral Sclerosis (ALS) Disease Registry: Frequently asked questions for patients and families. Boston, Mass.]: Massachusetts Department of Public Health, Bureau of Environmental Health, Environmental Epidemiology Program, 2009.

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12

The butcher's daughter: The story of an army nurse with ALS. College Station, TX: Virtualbookworm.com Pub., 2009.

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13

DeGraff, Rosemary. Twilight of a dancer: The story of Katherine Collingwood and her heroic battle with ALS. Winnipeg: Peguis Publishers, 1987.

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14

Not in Kansas anymore: A memoir of the farm, New York City, and life with ALS. Winnipeg, Canada: Gemma B. Publishing, 2008.

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15

Lou Gehrig Disease, ALS or Amyotrophic Lateral Sclerosis explained: ALS symptoms, signs, stages, types, diagnosis, treatment, caregiver tips, aids and what to expect is all covered. [Ireland]: IMB Publishing, 2013.

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16

Williams, Brian J. (Brian Jeremy), 1976- and American Academy of Orthopaedic Surgeons, eds. Principles of ALS care. Sudbury, Mass: Jones and Bartlett Publishers, 2011.

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17

Robert), Ball Bob (Ball, ed. Elephants in the tea: A journey through ALS. Portland, OR: ACTS Publishers, 2010.

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18

Gajdusek, D. Carleton. Calcium deficiency induced secondary hyperparathyroidism and resultant CNS deposition of calcium and other metallic cations as the cause of ALS and PD in high incidence among the Auyu and Jakai people in West New Guinea. Bethesda, Md: [National Institute of Neurological and Communicative Disorders and Stroke], 1985.

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19

Sparrow: A journey of grace and miracles while battling ALS. New York: Morehouse Publishing, 2016.

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20

Wakefield, Darcy. I remember running: The year I got everything I ever wanted and ALS. New York: Marlowe & Co., 2005.

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21

United States. Congress. Senate. A bill to amend the Social Security Act to waive the 24-month waiting period for Medicare coverage of individuals disabled with amyotrophic lateral sclerosis (ALS), and to provide Medicare coverage of drugs and biologicals used for the treatment of ALS or for the alleviation of symptoms relating to ALS. Washington, D.C: U.S. G.P.O., 1999.

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22

Where there is faith He is with you: One woman's journey of faith and her husband's battle with ALS. Decatur, Ill: House of Prayer Ministries, 2011.

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23

United States. Congress. House. A bill to amend the Social Security Act to waive the 24-month waiting period for Medicare coverage of individuals disabled with amyotrophic lateral sclerosis (ALS), and to provide Medicare coverage of drugs used for treatment of ALS. Washington, D.C: U.S. G.P.O., 1999.

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24

author, Sands Michael 1962, ed. Hold on, let go: Facing ALS with courage and hope. [North Vancouver, British Columbia]: Influence Publishing, 2015.

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25

United, States Congress Senate Committee on Appropriations Subcommittee on Departments of Labor Health and Human Services Education and Related Agencies. Amyotrophic lateral sclerosis (ALS): Hearing before a subcommittee of the Committee on Appropriations, United States Senate, One Hundred Ninth Congress, first session, special hearing, May 11, 2005, Washington, DC. Washington: U.S. G.P.O., 2006.

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26

Living with ALS. Calabasas Hills, CA: ALS Association, 2002.

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27

Limmer, Daniel. ALS Vital Signs. Prentice Hall, 2007.

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28

Limmer, Daniel. ALS Vital Signs. Prentice Hall, 2007.

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29

Limmer, Daniel. ALS Vital Signs: Network Version. Prentice Hall, 2007.

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30

Amyotrophic Lateral Sclerosis Society of Canada., ed. Resources for ALS healthcare providers. Toronto: The Society, 1994.

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31

Limmer, Daniel. ALS Vital Signs: Network Version. Prentice Hall, 2007.

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32

Harris, Brent T., Galam A. Khan, and Saed Sadeghi. Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0029.

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Анотація:

Although the basic gross and microscopic pathological changes in amyotrophic lateral sclerosis (ALS) have been known for more than 100 years, emerging technology and research into the cellular and molecular changes found in this disease are challenging our understanding about the pathogenesis and pathophysiology. All cell types of the CNS/PNS as well as circulating immune cells have been implicated in the pathology of ALS. Numerous genes, their proteins, and environmental factors have also been associated. However, we still do not understand the specific gene-environmental interactions that bring about and drive this devastating disease in most cases. This short chapter does not address the causal factors and molecular pathogeneses that have been hypothesized and actively researched in the pathology of ALS-as these are discussed in other sections of this text. Here, it shows and discusses the basic pathological changes at the tissue and cellular levels that help to establish the pathological diagnosis of ALS at autopsy.

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33

Ice Bucket Challenge: Pete Frates and the Fight Against ALS. University Press of New England, 2017.

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34

Casey, Sherman, and Dave Wedge. Ice Bucket Challenge: Pete Frates and the Fight Against ALS. University Press of New England, 2017.

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35

Jun, Kimura, and Kaji Ryuji, eds. Physiology of ALS and related diseases. Amsterdam: Elsevier, 1997.

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36

Wainger, Brian J. Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0028.

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Mouse and cellular models of ALS including stem cells have revealed tremendous insight into the molecular processes that lead to ALS. Models of ALS and other neurodegenerative diseases have led to emergent molecular themes that span several diseases. Future models must account for neuronal subtype specificity of different neurodegenerative diseases, particularly between tightly related diseases such as FTD and ALS. Human iPSC-derived motor neurons offer promise both with regard to the use of human cells and in particular the ability to model sporadic disease, which is critically important given the overwhelming abundance of sporadic disease in ALS and other neurodegenerative diseases.

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37

Sherman, Casey, and Dave Wedge. The Ice Bucket Challenge: Pete Frates and the Fight Against ALS. Blackstone Audio, Inc., 2017.

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38

Pagnini, Francesco, and Zachary Simmons, eds. Amyotrophic Lateral Sclerosis. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198757726.001.0001.

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Amyotrophic lateral sclerosis: Understanding and optimizing quality of life and psychological well-being presents a comprehensive and up-to-date review of the enhancement of the lives of people with amyotrophic lateral sclerosis (ALS) and their caregivers. ALS is a progressive, fatal neurodegenerative disorder. No current medical therapy can reverse or stop its progression, and the promotion of quality of life and psychological well-being is a central component of ALS care. Health care professionals who work in this field should incorporate attention to psychological, emotional, and relational aspects of the disease into their approach to care. This book provides some of the knowledge and direction necessary for optimizing the quality of care for individuals with ALS and their caregivers. Topics discussed include an ALS-centred view of quality of life, depressive features, anxiety, resilience, cognitive impairment, complementary and alternative medicines, and psychological research. Specific elements of ALS, such as end-of-life concerns and bulbar dysfunction, are described through the lens of their psychological impact. There is extensive discussion of the development of new psychological treatments, as well as the impact and incorporation of new technologies, with the goal of fostering optimal quality of life and psychological well-being as key parts of a holistic approach to care for the patients and for those who are close to such individuals.

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39

Ferraiuolo, Laura, and Stephen J. Kolb. Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0026.

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An overriding mystery of ALS pathogenesis orbits around the molecular basis of selective motor neuron vulnerability and clouds our view. There are likely mechanisms involved in the initiation of motor neuron loss and mechanisms involved in the progression of motor neuron loss once initiated. Motor neuron vulnerability is likely related to the unique biological characteristics of these cells. This chapter introduces central molecular pathways that appear to be involved in the pathogenesis of ALS, and highlights why dysregulation of these mechanisms could lead to motor neuron death. Indeed, there are likely mechanisms involved in the initiation of motor neuron loss and mechanisms involved in the progression of motor neuron loss once initiated. Our task is to determine those mechanisms that are relevant to ALS pathogenesis that may be targeted therapeutically to prevent onset and/or halt progression.

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40

Harms, Matthew B., and Timothy M. Miller. Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0027.

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Recent advances in sequencing technologies have dramatically expanded the number of genes associated with amyotrophic lateral sclerosis, including rare but highly penetrant causative mutations as well as common risk alleles. This chapter discusses these gene discoveries and how they have implicated a diverse array of biological pathways essential for motor neuron health and have begun to inform our understanding of ALS pathogenesis as a heterogeneous and multistep process. Insights from these discoveries are leading to a new generation of targeted therapies directed at specific genes and are poised to inform how patients with amyotrophic lateral sclerosis are evaluated and treated in the clinic.

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41

Als-Lou Gehrig's Disease (Diseases and People). Enslow Publishers, 2001.

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42

Als Skills Review. Jones & Bartlett Publishers, 2008.

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43

Paine, John R., and Seth Haines. Luckiest Man: How a Seventeen-Year Battle with ALS Led Me to Intimacy with God. Nelson Incorporated, Thomas, 2018.

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44

O’Reilly, Éilis J. Epidemiology of Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0025.

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Анотація:

The epidemiology of ALS is an emerging field and, like the epidemiology of cardiovascular disease and cancer in the mid last century, requires time for convergence of findings. There appears to be a genetic influence, and one study of twins found that heritability of sporadic ALS is 60%. At present it is thought that 60$ to 70% of genetic mutations responsible for fALS in populations of European ancestry are known. SOD1 mutations were the earliest discoveries in fALS. Subsequently, mutations were identified in TARDBP, which encodes TDP-43 protein found in neuronal inclusions in fALS and frontotemporal dementia (FTD). High rates of ALS have also been reported in military personnel and in certain groups of elite athletes such as soccer players.

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45

Miller, Mary E. Nerve Disease ALS and Gradual Loss of Muscle Function: Amyotrophic Lateral Sclerosis. Momentum Press, 2016.

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46

The Luckiest Man: How a Seventeen-Year Battle with ALS Led Me to Intimacy with God. Thomas Nelson, 2018.

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47

Nicholson, Katharine A., and James D. Berry. Amyotrophic Lateral Sclerosis Clinical Trials. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0030.

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Анотація:

The current state of amyotrophic lateral sclerosis (ALS) trial design is best understood within the context of ALS research over the past quarter century. Before the early 1990s, trials in ALS were typically small and clinical trial methodology was less rigorous than it is today. With the discovery of the SOD1 gene mutation in the early 1990s, a new era of excitement and innovation for ALS research began. Since then, the number of ALS trials has steadily increased and trial design and methodology has become increasingly sophisticated.

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48

Hansen, Gregg. Cbd Oil for Amyotrophic Lateral Sclerosis: Effective and Efficient Remedy to Overcoming ALS. Independently Published, 2019.

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49

Hilton, Eric. CBD Oil for Amyotrophic Lateral Sclerosis: Effective and Efficient Remedy to Overcoming ALS. Independently Published, 2019.

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50

Gladman, Matthew, and Lorne Zinman. Symptomatic Management in Amyotrophic Lateral Sclerosis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0024.

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Анотація:

Although ALS remains an incurable disease, there are specific treatments that can significantly improve quality of life and some that can modify survival. Patients are best followed in multidisciplinary ALS clinics where a number of medical experts can address a wide range of symptoms. Additional studies are needed to develop evidenced-based best practice guidelines to further improve care for patients with ALS. Bulbar dysfunction eventually affects the vast majority of ALS patients, and progressive dysphagia results from weakness and spasticity of the muscles involved in swallowing. Dysphagia is associated with increased risk of aspiration and choking, as well as with long-term risk of malnutrition, weight loss, and dehydration. Appropriate measures to enhance caloric intake contribute to a greater sense of well-being. Augmentative communication devices and ventilator support can also be used to reduce suffering.

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