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Статті в журналах з теми "Activation barrier"
Stepanov, A. V. "Activation process model: Einstein coefficients for activation barrier." Journal of Molecular Structure: THEOCHEM 805, no. 1-3 (March 2007): 87–90. http://dx.doi.org/10.1016/j.theochem.2006.10.021.
Повний текст джерелаGessain, Grégoire, Yu-Huan Tsai, Laetitia Travier, Matteo Bonazzi, Solène Grayo, Pascale Cossart, Caroline Charlier, Olivier Disson, and Marc Lecuit. "PI3-kinase activation is critical for host barrier permissiveness to Listeria monocytogenes." Journal of Experimental Medicine 212, no. 2 (January 26, 2015): 165–83. http://dx.doi.org/10.1084/jem.20141406.
Повний текст джерелаBushuev, Mark B., Denis P. Pishchur, Elena B. Nikolaenkova, and Viktor P. Krivopalov. "Compensation effects and relation between the activation energy of spin transition and the hysteresis loop width for an iron(ii) complex." Physical Chemistry Chemical Physics 18, no. 25 (2016): 16690–99. http://dx.doi.org/10.1039/c6cp01892k.
Повний текст джерелаDoering, Charles R., and Jonathan C. Gadoua. "Resonant activation over a fluctuating barrier." Physical Review Letters 69, no. 16 (October 19, 1992): 2318–21. http://dx.doi.org/10.1103/physrevlett.69.2318.
Повний текст джерелаde Jong, G. Theodoor, and F. Matthias Bickelhaupt. "Bond activation by group-11 transition-metal cations." Canadian Journal of Chemistry 87, no. 7 (July 2009): 806–17. http://dx.doi.org/10.1139/v09-009.
Повний текст джерелаJiang, Heming, and Tian-Yu Sun. "The Activating Effect of Strong Acid for Pd-Catalyzed Directed C–H Activation by Concerted Metalation-Deprotonation Mechanism." Molecules 26, no. 13 (July 4, 2021): 4083. http://dx.doi.org/10.3390/molecules26134083.
Повний текст джерелаCheng, Chunyu, Yiming Zou, Jiahui Li, Amanda Jiamin Ong, Ronn Goei, Jingfeng Huang, Shuzhou Li, and Alfred Iing Yoong Tok. "Adsorption and Reaction Mechanisms of Direct Palladium Synthesis by ALD Using Pd(hfac)2 and Ozone on Si (100) Surface." Processes 9, no. 12 (December 13, 2021): 2246. http://dx.doi.org/10.3390/pr9122246.
Повний текст джерелаWinter, Michael C., Sandra S. Shasby, Dana R. Ries та D. Michael Shasby. "PAR2 activation interrupts E-cadherin adhesion and compromises the airway epithelial barrier: protective effect of β-agonists". American Journal of Physiology-Lung Cellular and Molecular Physiology 291, № 4 (жовтень 2006): L628—L635. http://dx.doi.org/10.1152/ajplung.00046.2006.
Повний текст джерелаStavenuiter, Fabian, and Laurent O. Mosnier. "Non-Canonical PAR3 Activation Induces Tie2-Dependent Endothelial Barrier Protective Effects." Blood 124, no. 21 (December 6, 2014): 2802. http://dx.doi.org/10.1182/blood.v124.21.2802.2802.
Повний текст джерелаCarpenter, Chris. "Using Shale as a Barrier Simplifies Well Abandonment." Journal of Petroleum Technology 73, no. 01 (January 1, 2021): 62–63. http://dx.doi.org/10.2118/0121-0062-jpt.
Повний текст джерелаДисертації з теми "Activation barrier"
Pesic, Marija. "Visualizing T cell activation around the blood-brain barrier Dissertation." Diss., Ludwig-Maximilians-Universität München, 2013. http://nbn-resolving.de/urn:nbn:de:bvb:19-159898.
Повний текст джерелаHussain, Imran [Verfasser]. "Insulin modulates the recovery of endothelial barrier function via Rac1 activation / Imran Hussain." Gießen : Universitätsbibliothek, 2016. http://d-nb.info/1112909990/34.
Повний текст джерелаSchwaiger, Christine S. "Voltage sensor activation and modulation in ion channels." Doctoral thesis, KTH, Beräkningsbiofysik, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-104742.
Повний текст джерелаQC 20121115
Pan, Yongping. "Characterization of Low Barrier Hydrogen Bonds in Enzyme Catalysis: an Ab Initio and DFT Investigation." Thesis, University of North Texas, 1999. https://digital.library.unt.edu/ark:/67531/metadc278586/.
Повний текст джерелаPesic, Marija [Verfasser], and Mark [Akademischer Betreuer] Hübener. "Visualizing T cell activation around the blood-brain barrier Dissertation / Marija Pesic. Betreuer: Mark Hübener." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2013. http://d-nb.info/1038152488/34.
Повний текст джерелаPriori, Davide <1976>. "Diet Effects on Activation and Maturation of Feed Control over the Gastrointestinal Defence Barrier in Piglets." Doctoral thesis, Alma Mater Studiorum - Università di Bologna, 2012. http://amsdottorato.unibo.it/4764/.
Повний текст джерелаRiaz, Muhammad Assad [Verfasser]. "Activation of AMP-activated kinase at reperfusion protects the endothelial barrier against reperfusion-induced failure / Muhammad Assad Riaz." Gießen : Universitätsbibliothek, 2012. http://d-nb.info/1069065315/34.
Повний текст джерелаMohd, Nasir Mohd Hamzah. "Activation of endothelial cells and its potential involvement in blood-brain barrier damage in cerebral malaria : an in vitro study." Thesis, Keele University, 2015. http://eprints.keele.ac.uk/3252/.
Повний текст джерелаChaves, Catarina Alexandra da Silva. "Mechanisms of regulation of P-glycoprotein and breast cancer resistance protein at the blood-brain barrier : focus on the role of morphine, and P-glycoprotein activation." Thesis, Sorbonne Paris Cité, 2015. http://www.theses.fr/2015USPCB162/document.
Повний текст джерелаThe blood-brain barrier (BBB) is the main interface of molecular exchange between the bloodstream and the central nervous system (CNS), where it plays an essential role on the control over the bi-directional passage of endogenous and exogenous compounds. At the BBB, P-glycoprotein (P-gp) and Breast Cancer Resistance Protein (BCRP) are the most important ABC drug efflux transporters preventing the entry into the brain of toxic compounds, drugs and xenobiotics circulating in the blood. There is increasing interest in understanding the molecular mechanisms underlying the modulation of P-gp and BCRP expression and function in order to control CNS accumulation of neurotoxicants and to overcome pharmacoresistance phenomena. Recent studies showed that morphine, itself a substrate of P-gp, is implicated in the up-regulation of P-gp expression, which may contribute to its poor brain penetration and tolerance. However, it was unknown the mechanism underlying P-gp induction by morphine and its role on BCRP expression. Rats were used as an animal model for the study of the amplitude and the kinetics of the modulation of P-gp and Bcrp expressions at the BBB following a subchronic morphine treatment, in an escalating morphine dose regimen. Freshly isolated rat brain microvessels were used as BBB model to study P-gp and Bcrp contents following the in vivo treatment, while the hCMEC/D3 cell line was occasionally used for complementary studies. Our results demonstrated that a 5-day subchronic morphine regimen up-regulated both P-gp and Bcrp 12 to 24h after the last dose of morphine, which was not registered at earlier time-points of animal sacrifice, nor with a single dose of morphine. The animal treatment with a glutamatergic NMDA receptor antagonist, or a COX-2 inhibitor abolished the subchronic morphine-induced P-gp and Bcrp protein up-regulation, 24h after the last dose of morphine, suggesting that both are implicated in the morphine-dependent P-gp and Bcrp up-regulation. Since the registered up-regulation only occurred from 12h after the last dose of morphine-onwards, we investigated whether it was a direct effect of continued exposure to morphine, or rather a consequence of the morphine withdrawal developed after discontinuation of treatment. Rats were treated either with a constant morphine infusion (5 days), or two chronic morphine regimens where withdrawal was precipitated by naloxone administration: an escalating dose (5 days) or a constant dose morphine regimen followed by a withdrawal period (2 days) and resume of the treatment for 3 additional days. Continuous i.v. morphine did not change P-gp and Bcrp levels in rat brain microvessels, it does not have a direct consequence on the cascade of regulation of these transporters at the BBB. Naloxone-precipitated withdrawal after escalating or chronic morphine dose regimen increased Mdr1a and Bcrp mRNA levels, but protein expression and activity remained unchanged after naloxone administration. This latter result discrepancy may be due to posttranslational regulation or naloxone action at non-opioid receptors hampering P-gp and Bcrp up-regulation. Subsequently, we did a large screening of the expression of several neurotransmitter receptors at the rat BBB, many of them implicated in the inflammatory cell-cell signaling, and which may have a role in the modulation of these ABC transporters. Also, we compared two different approaches of isolation of rat brain microvessels, mechanical dissection and enzymatic digestion, to assess which yield the purest microvessel fraction for the BBB study. The enzymatic digestion provided the highest enrichment of endothelial cells and pericytes, and the least contamination with astrocyte and neuron markers. (...)
A barreira hemato-encefálica (BHE) representa a principal interface entre a corrente sanguínea e o sistema nervoso central (SNC), desempenhando um papel essencial no controlo da passagem sangue-cérebro de diversos compostos endógenos e exógenos. A glicoproteina P (P-gp) e a proteína de resistência ao cancro da mama (BCRP) são os principais transportadores de efluxo da família ABC presentes ao nível da BHE, limitando a passagem cerebral de compostos tóxicos, fármacos e xenobióticos circulantes na corrente sanguínea. Actualmente, regista-se um crescente interesse na comunidade científica para a melhor compreensão dos mecanismos moleculares subjacentes à modulação quer da expressão quer da função da P-gp e BCRP, no sentido de desenvolver medidas mais eficazes quer para prevenção da acumulação de compostos neurotóxicos no SNC, quer para superar fenómenos de farmacorresistência associados à terapêutica. Estudos recentes evidenciam que a morfina, por si só um substrato da P-gp, está envolvida na indução da expressão da P-gp, o que poderá contribuir para a sua menor penetração cerebral, bem como para o desenvolvimento de tolerância. No entanto, não se conhece o mecanismo subjacente a tal indução da P-gp pela morfina, nem o seu eventual papel na expressão da BCRP. Com efeito, na condução da presente dissertação, realizamos um estudo da amplitude e a cinética da regulação da expressão da P-gp e BCRP ao nível da BHE na sequência de um tratamento subcrónico com morfina, em regime de doses crescentes, usando o rato como modelo animal. Para o efeito, foram isolados os capilares cerebrais dos animais sujeitos a tratamento, in vivo, enquanto que a linha celular hCMEC/D3 foi ocasionalmente utilizada para estudos complementares. Os nossos resultados demonstraram que um tratamento subcrónico com morfina (5 dias) foi capaz de induzir tanto a P-gp como a Bcrp 12 a 24 horas após a última dose de morfina administrada, mas não para tempos de sacrifício anteriores, bem como tal indução não foi registada quando a morfina foi administrada de forma aguda. O tratamento animal com um antagonista do receptor glutamatérgico NMDA, ou com um inibidor da COX-2 anulou este efeito de indução da P-gp e Bcrp pela administraçãosubcrónica de morfina, o que sugere o envolvimento destes dois componentes na indução da P-gp e Bcrp dependente da morfina. Uma vez que este aumento da expressão só surgiu a partir de 12h após a última dose de morfina, decidimos investigar se tal seria um efeito direto da exposição continuada à morfina, ou por outro lado, uma consequência do síndrome de abstinência à morfina, desenvolvido após a descontinuação do tratamento. Desta forma, os animais foram tratados por um lado com uma infusão contínua de morfina (5 dias), ou sujeitos a dois diferentes regimes de exposição crónica à morfina, após os quais o síndrome de abstinência foi provocado pela administração de naloxona. A administração de morfina em contínuo, via i.v., não alterou os níveis de P-gp e BCRP nos capilares cerebrais de rato, o que indica a ausência de uma consequência directa da morfina na cascata de regulação destes transportadores ao nível da BHE. O síndrome de abstinência opióide provocado pela naloxona aumentou os níveis de mRNA Mdr1a e Bcrp, mas tanto a expressão e atividade proteicas mantiveram-se inalteradas após a administração de naloxona. Esta discrepância de resultados pode-se dever ou a um regulamento pós-translacional, ou a uma acção inespecífica da naloxona em receptores não opiáceos, impedindo a indução da P-gp e Bcrp. Num outro estudo, foi feito um screening da expressão de vários receptores de neurotransmissores na BHE de rato, muitos deles envolvidos na sinalização célula-célula em processos inflamatórios, e que podem ter um papel na modulação destes transportadores ABC. (...)
Chung, Charlotte Yuk-Yan. "Tight Junctions - The Link Between HIV-Associated Intestinal Barrier Dysfunction and Loss of Immune Homeostasis." Case Western Reserve University School of Graduate Studies / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=case1417822947.
Повний текст джерелаКниги з теми "Activation barrier"
Cortese, R. A. Flame-powered trigger device for activating explosion suppression barrier. Washington, D.C: U.S. Dept. of the Interior, Bureau of Mines, 1991.
Знайти повний текст джерелаCortese, R. A. Flame powered trigger device for activating explosion suppression barrier. Washington, DC: U.S. Dept. of the Interior, Bureau of Mines, 1991.
Знайти повний текст джерелаMagalhaes, Eric, Angelo Polito, Andréa Polito, and Tarek Sharshar. Sepsis-Associated Encephalopathy. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0032.
Повний текст джерелаHenriksen, Niels Engholm, and Flemming Yssing Hansen. Microscopic Interpretation of Arrhenius Parameters. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198805014.003.0008.
Повний текст джерелаBazzan, Anthony J., and Daniel A. Monti. Diet, Gut, and Brain: A New Horizon. Edited by Anthony J. Bazzan and Daniel A. Monti. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190690557.003.0001.
Повний текст джерелаDangayach, Neha S., Charles L. Francoeur, Stephan A. Mayer, and Tarek Sharshar. Neuroprotection in Sepsis and Acute Respiratory Distress Syndrome. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0013.
Повний текст джерелаDe Souza, Jonathan. Sounding Actions. Oxford University Press, 2017. http://dx.doi.org/10.1093/acprof:oso/9780190271114.003.0003.
Повний текст джерелаЧастини книг з теми "Activation barrier"
Persidsky, Yuri, and Howard E. Gendelman. "HIV-1 Encephalitis is a Consequence of Viral Infection and Neuroimmune Activation." In Biology and Physiology of the Blood-Brain Barrier, 365–67. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_60.
Повний текст джерелаMarshall, G. R., M. L. Smythe, S. E. Huston, and R. D. Bindal. "The molten helix: low activation energy barrier for helix-helix transitions." In Peptides, 896–98. Dordrecht: Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-0683-2_301.
Повний текст джерелаDore-Duffy, Paula, Roumen Balabanov, and Ruth Washington. "Recovery from Acute Experimental Autoimmune Encephalomyelitis (EAE) Characterized by Endothelial Cell Unresponsiveness to Cytokines and Pericyte Activation." In Biology and Physiology of the Blood-Brain Barrier, 347–51. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9489-2_57.
Повний текст джерелаRapoport, S. I., and P. J. Robinson. "Long-Chain Fatty Acid Transport at the Blood-Brain Barrier and Incorporation into Brain Phospholipids: A New In Vivo Method for Examining Neuroplasticity, and Brain Second Messenger Systems Involving Phospholipase A2 Activation." In New Concepts of a Blood—Brain Barrier, 119–40. Boston, MA: Springer US, 1995. http://dx.doi.org/10.1007/978-1-4899-1054-7_13.
Повний текст джерелаSchatz, George C. "Perspective on “Exchange reactions with activation energy. I. Simple barrier potential for (H, H2)”." In Theoretical Chemistry Accounts, 270–72. Berlin, Heidelberg: Springer Berlin Heidelberg, 2000. http://dx.doi.org/10.1007/978-3-662-10421-7_31.
Повний текст джерелаKoenig, Harold, Jerome J. Trout, Alfred D. Goldstone, and Chung Y. Lu. "NMDA Receptors Mediate Activation of Polyamine Synthesis and Blood-Brain Barrier Breakdown after Cold Injury." In The Role of Neurotransmitters in Brain Injury, 279–83. Boston, MA: Springer US, 1992. http://dx.doi.org/10.1007/978-1-4615-3452-5_42.
Повний текст джерелаHuegler, Nathalie, and Natasha Kersh. "Social Inclusion, Participation and Citizenship in Contexts of Neoliberalism: Examples of Adult Education Policy and Practice with Young People in the UK, The Netherlands and Ireland." In Young Adults and Active Citizenship, 57–78. Cham: Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-65002-5_4.
Повний текст джерелаWang, Z., and A. R. Crofts. "The Mechanism of Quinol Oxidation: Activation Barriers in UQH2:CYT C2 Oxidoreductase." In Current Research in Photosynthesis, 2197–200. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0511-5_502.
Повний текст джерелаWhittington, Chris, John Latham, and Adam R. Offenbacher. "Tunneling through the Barriers: Resolving the Origins of the Activation of C-H Bonds Catalyzed by Enzymes." In ACS Symposium Series, 139–60. Washington, DC: American Chemical Society, 2020. http://dx.doi.org/10.1021/bk-2020-1357.ch007.
Повний текст джерелаBanks, William A. "Transport of Pituitary Adenylate Cyclase Activating Polypeptide Across the Blood–Brain Barrier: Consequences for Disease States and Therapeutic Effects." In Current Topics in Neurotoxicity, 423–32. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-35135-3_25.
Повний текст джерелаТези доповідей конференцій з теми "Activation barrier"
Iwaniszewski, J. "On the resonant activation and inhibition of activation in fluctuating barrier problems." In Stochastic and chaotic dynamics in the lakes. AIP, 2000. http://dx.doi.org/10.1063/1.1302399.
Повний текст джерелаLu, Q., G. Radin, J. Newton, and S. Rounds. "Pentostatin Enhances Endothelial Baseline Barrier Function through Rac-1 Activation." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a6117.
Повний текст джерелаWang, Shengzhao, Dayong Huang, and Jianliang Qiao. "The evolution of GaN photocathode surface barrier before and after activation." In Optoelectronic Materials and Devices for Sensing and Imaging, edited by Mingbo Pu, Xue Feng, Yadong Jiang, Xiong Li, Xiaoliang Ma, and Bernard Kippelen. SPIE, 2019. http://dx.doi.org/10.1117/12.2505545.
Повний текст джерелаWadgaonkar, Raj, Satish Gowda, and Fatima Anjum. "Essential Role Of Sphingomyelin Synthase Activation In Endotoxin Induced Barrier Dysfunction." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a1863.
Повний текст джерелаSoong, Grace, Jarin Chun, Francis Martin, Taylor S. Cohen, and Alice S. Prince. "Staphylococcal Activation Of EGFR Facilitates Invasion Across The Pulmonary Mucosal Barrier." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6777.
Повний текст джерелаKristiansen, Tron Golder, Laurent Delabroy, Guillermo Andres Obando Palacio, Tonje Winther, Nils Andre Aarseth, Andreas Bauer, Karstein Hagenes, Anders Lindal, and Pål Tyberø. "Implementing a Strategy for Shale as Well Barrier in New Wells." In SPE/IADC International Drilling Conference and Exhibition. SPE, 2021. http://dx.doi.org/10.2118/204075-ms.
Повний текст джерелаSoskin, S. M. "Drastic Reduction of the Activation Barrier by a Moderately Weak Periodic Driving." In UNSOLVED PROBLEMS OF NOISE AND FLUCTUATIONS: UPoN 2002: Third International Conference on Unsolved Problems of Noise and Fluctuations in Physics, Biology, and High Technology. AIP, 2003. http://dx.doi.org/10.1063/1.1584916.
Повний текст джерелаKoehler, Andrew D., Travis J. Anderson, Marko J. Tadjer, Boris N. Feigelson, Karl D. Hobart, Francis J. Kub, Anindya Nath, and David I. Shahin. "Vertical GaN junction barrier schottky diodes by Mg implantation and activation annealing." In 2016 IEEE 4th Workshop on Wide Bandgap Power Devices and Applications (WiPDA). IEEE, 2016. http://dx.doi.org/10.1109/wipda.2016.7799965.
Повний текст джерелаMuscarella, Loreta, Eline Hutter, Francesca Wittmann, Young Won Woo, Young-Kwang Jung, Lucie McGovern, Jan Versluis, Aron Walsh, Huib Bakker, and Bruno Ehrler. "Lattice compression increases the activation barrier for phase segregation in mixed-halide perovskites." In International Conference on Impedance Spectroscopy and Related Techniques in Metal Halide Perovskites. València: Fundació Scito, 2020. http://dx.doi.org/10.29363/nanoge.perimped.2020.005.
Повний текст джерелаPan, Mingqiang, Furong Yao, Jizhu Liu, Yangjun Wang, and Lining Sun. "Effect of Magnetic Field on Activation Performance of Silicon/Glass Dielectric Barrier Discharge." In 2018 19th International Conference on Electronic Packaging Technology (ICEPT). IEEE, 2018. http://dx.doi.org/10.1109/icept.2018.8480568.
Повний текст джерелаЗвіти організацій з теми "Activation barrier"
McGuinness, Seamus, Adele Whelan, Adele Bergin, and Judith Delaney. Profiling barriers to social inclusion in Ireland: the relative roles of individual characteristics and location. ESRI, July 2018. http://dx.doi.org/10.26504/rs71.
Повний текст джерелаEvans, Donald L., Avigdor Eldar, Liliana Jaso-Friedmann, and Herve Bercovier. Streptococcus Iniae Infection in Trout and Tilapia: Host-Pathogen Interactions, the Immune Response Towards the Pathogen and Vaccine Formulation. United States Department of Agriculture, February 2005. http://dx.doi.org/10.32747/2005.7586538.bard.
Повний текст джерела