Добірка наукової літератури з теми "Actin remodeling proteins"
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Статті в журналах з теми "Actin remodeling proteins"
Olave, Ivan A., Samara L. Reck-Peterson, and Gerald R. Crabtree. "Nuclear Actin and Actin-Related Proteins in Chromatin Remodeling." Annual Review of Biochemistry 71, no. 1 (June 2002): 755–81. http://dx.doi.org/10.1146/annurev.biochem.71.110601.135507.
Повний текст джерелаGalkin, V. E., A. Orlova, D. S. Kudryashov, A. Solodukhin, E. Reisler, G. F. Schroder, and E. H. Egelman. "Remodeling of actin filaments by ADF/cofilin proteins." Proceedings of the National Academy of Sciences 108, no. 51 (December 7, 2011): 20568–72. http://dx.doi.org/10.1073/pnas.1110109108.
Повний текст джерелаEitzen, Gary, Li Wang, Naomi Thorngren, and William Wickner. "Remodeling of organelle-bound actin is required for yeast vacuole fusion." Journal of Cell Biology 158, no. 4 (August 12, 2002): 669–79. http://dx.doi.org/10.1083/jcb.200204089.
Повний текст джерелаStolp, Bettina, Libin Abraham, Jochen M. Rudolph, and Oliver T. Fackler. "Lentiviral Nef Proteins Utilize PAK2-Mediated Deregulation of Cofilin as a General Strategy To Interfere with Actin Remodeling." Journal of Virology 84, no. 8 (February 10, 2010): 3935–48. http://dx.doi.org/10.1128/jvi.02467-09.
Повний текст джерелаHoffman, Laura M., Christopher C. Jensen, Aashi Chaturvedi, Masaaki Yoshigi, and Mary C. Beckerle. "Stretch-induced actin remodeling requires targeting of zyxin to stress fibers and recruitment of actin regulators." Molecular Biology of the Cell 23, no. 10 (May 15, 2012): 1846–59. http://dx.doi.org/10.1091/mbc.e11-12-1057.
Повний текст джерелаLiu, Jing, Xinhua Guo, Narla Mohandas, Joel A. Chasis, and Xiuli An. "Membrane remodeling during reticulocyte maturation." Blood 115, no. 10 (March 11, 2010): 2021–27. http://dx.doi.org/10.1182/blood-2009-08-241182.
Повний текст джерелаGoodson, Holly V., and William F. Hawse. "Molecular evolution of the actin family." Journal of Cell Science 115, no. 13 (July 1, 2002): 2619–22. http://dx.doi.org/10.1242/jcs.115.13.2619.
Повний текст джерелаFurman, Craig, Alisha L. Sieminski, Adam V. Kwiatkowski, Douglas A. Rubinson, Eliza Vasile, Roderick T. Bronson, Reinhard Fässler, and Frank B. Gertler. "Ena/VASP is required for endothelial barrier function in vivo." Journal of Cell Biology 179, no. 4 (November 12, 2007): 761–75. http://dx.doi.org/10.1083/jcb.200705002.
Повний текст джерелаRusso, Ashley J., Alyssa J. Mathiowetz, Steven Hong, Matthew D. Welch, and Kenneth G. Campellone. "Rab1 recruits WHAMM during membrane remodeling but limits actin nucleation." Molecular Biology of the Cell 27, no. 6 (March 15, 2016): 967–78. http://dx.doi.org/10.1091/mbc.e15-07-0508.
Повний текст джерелаOffenhäuser, Nina, Alessandro Borgonovo, Andrea Disanza, Pascale Romano, Isabella Ponzanelli, Gioacchin Iannolo, Pier Paolo Di Fiore, and Giorgio Scita. "The eps8 Family of Proteins Links Growth Factor Stimulation to Actin Reorganization Generating Functional Redundancy in the Ras/Rac Pathway." Molecular Biology of the Cell 15, no. 1 (January 2004): 91–98. http://dx.doi.org/10.1091/mbc.e03-06-0427.
Повний текст джерелаДисертації з теми "Actin remodeling proteins"
Richardson, Brian Edward. "The role of actin cytoskeleton remodeling : during embryonic myoblast fusion in Drosophila /." Access full-text from WCMC, 2008. http://proquest.umi.com/pqdweb?did=1642921951&sid=10&Fmt=2&clientId=8424&RQT=309&VName=PQD.
Повний текст джерелаJin, Janet Yikai. "Missing in metastasis, an I-BAR protein regulating actin remodeling and cell migration /." May be available electronically:, 2008. http://proquest.umi.com/login?COPT=REJTPTU1MTUmSU5UPTAmVkVSPTI=&clientId=12498.
Повний текст джерелаWillis, William L. "YB-1 Stress-Response Protein Conformation Implicated in Post-transcriptional Control of Myofibroblast Differentiation." The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1376593223.
Повний текст джерелаChan, Huater. "Roles of actin remodeling proteins, gelsolin and Flightless-I in epidermal wound healing." Thesis, 2011. http://hdl.handle.net/2440/72723.
Повний текст джерелаFelipe-López, Alfonso. "Remodelling of the F-actin Cytoskeleton of Polarized Epithelial Cells by the Type 3 Secretion System-1 Effector Proteins of Salmonella enterica sv. Typhimurium." Doctoral thesis, 2015. https://repositorium.ub.uni-osnabrueck.de/handle/urn:nbn:de:gbv:700-2015113013690.
Повний текст джерелаRatni, Sara. "Étude des voies de signalisation activées par la prostaglandine F2α dans les cellules de cancer colorectal". Thèse, 2016. http://hdl.handle.net/1866/16281.
Повний текст джерелаColorectal cancer is the third most common form of cancer in Canada. Despite recent developments, no curative treatment is available for patients diagnosed at an advanced stage of the disease. Several evidences support an important role of prostaglandins in this disease. Indeed, overexpression of EP-like and FP receptors is noticed. These and intracellular signaling molecules that are activated following receptor stimulation, represent new targets for treating cancer. We and others have demonstrated that monomeric G proteins are important molecular switches coordinating intracellular signaling cascades initiated by receptors in normal cells but that disruption thereof is associated with cancer. The main objective of this research project aims to identify the signaling pathways by which the FP receptor contributes to the invasive capacity of tumor cells of colorectal origin. Our hypothesis is that the proteins ARF6, one of the 6 ARF isoforms, and RhoA protein, act to coordinate the activation of signaling pathways associated with cell migration and invasion. Our results show that stimulation of HEK293 cells that stably express the FP receptor (HA-FP) and SW480 cells, a lineage of invasive colorectal cancer that endogenously express FP receptor, by PGF2α, increased the level of activation of ARF6, but also that of RhoA. In addition, other mediators and intermediates associated with cytoskeleton reorganization such as cofilin and the myosin light chain (MLC) were found highly phosphorylated following stimulation by prostaglandin PGF2α. These observations are associated with an increase of stress fibers in cells. Finally, we have examinated whether inhibition of ARF6 and RhoA, affected the ability of PGF2α to activate these effectors and some biological effects. Thus, our results will contribute to identify new potential therapeutic targets for the treatment of colorectal cancer.
Bergeron, Valérie. "Étude des voies de signalisation en aval du récepteur FFA1/GPR40 dans la cellule bêta pancréatique." Thèse, 2017. http://hdl.handle.net/1866/20252.
Повний текст джерелаBivona, Dario Antonio. "Modulation allostérique de la fonction des récepteurs FP et PAF." Thèse, 2009. http://hdl.handle.net/1866/3215.
Повний текст джерелаG protein coupled receptors (GPCRs) are involved in the first step of most signalling pathways that regulate a variety of physiological events. The classical view of GPCR activation suggests that ligand binding to the inactive receptor will trigger a conformational change leading to an active conformation of the receptor. The GPCRs activated regulate the activity of a target membrane protein which can then activate other signalling proteins such as enzymes and ionic channels. The interaction between the receptor and the target requires an intermediary, in this case an heterotrimeric protein named « G protein », which is activated in order to facilitate the exchange of GDP (guanosine diphospate) for a GTP (guanosine triphosphate) and allow the transduction of the signal from the receptor to the effector. The molecular mechanisms leading to the activation of signalling effectors via the activation of GPCRs by its heterotrimeric G protein have not yet been well characterized. We focused our study on two GPCRs, the FP and PAF receptors, their natural ligands, PGF2α and Carbamyl-PAF respectively, and their antagonist ligands. Agonists are ligands that bind to the target receptor and trigger the same effects as the natural ligand of the GPCR. In contrast with agonists, antagonist ligands are molecules that prevent the effects of the natural ligand by keeping the GPCR from changing to its active conformation and can be competitive or non-competitive. We have also studied orthosteric and allosteric ligands of the FP and PAF receptors. An orthosteric ligand binds the same site as the natural ligand of the receptor and can act as an agonist or an antagonist. In the contrary, an allosteric ligand will rather have a different binding site then the natural ligand (agonist) and can positively or negatively modulate the effects of the natural ligand.
Книги з теми "Actin remodeling proteins"
Skiba, Grzegorz. Fizjologiczne, żywieniowe i genetyczne uwarunkowania właściwości kości rosnących świń. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_gs_2020.
Повний текст джерелаЧастини книг з теми "Actin remodeling proteins"
Van Wagoner, David R. "Mechanisms of atrial remodelling." In ESC CardioMed, 2125–28. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0498.
Повний текст джерелаMajeed Shah, Ishteyaq, Mashooq Ahmad Dar, Kaiser Ahmad Bhat, Tashook Ahmad Dar, Fayaz Ahmad, and Syed Mudasir Ahmad. "Long Non-Coding RNAs: Biogenesis, Mechanism of Action and Role in Different Biological and Pathological Processes." In Recent Advances in Non-Coding RNAs [Working Title]. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.104861.
Повний текст джерелаKopecki, Zlatko, and Allison J. Cowin. "The Role of Actin Remodelling Proteins in Wound Healing and Tissue Regeneration." In Wound Healing - New insights into Ancient Challenges. InTech, 2016. http://dx.doi.org/10.5772/64673.
Повний текст джерелаТези доповідей конференцій з теми "Actin remodeling proteins"
Kaunas, Roland. "A Theoretical Model of Stretch-Induced Stress Fiber Remodeling." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-193241.
Повний текст джерелаRonan, William, Vikram S. Deshpande, Robert M. McMeeking, and J. Patrick McGarry. "Simulation of Stress Fiber Remodeling and Mixed-Mode Focal Adhesion Assembly During Cell Spreading and for Cells Adhered to Elastic Substrates." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53878.
Повний текст джерелаBryan, Andrea, Amy Sung, Ian Lian, and Jeffrey Omens. "The Role of Tropomodulin in Cardiac Function and Remodeling." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-61363.
Повний текст джерелаWasi, S., S. Juodvalkis, P. Alles, and J. E. Aubin. "STUDIES ON THE DIRECT PROTEOLYTIC ACTION OF HUMAN TISSUE PLASMINOGEN ACTIVATOR ON HUMAN FIBRONECTIN AND VITRONECTIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644376.
Повний текст джерелаTing, Lucas H., and Nathan J. Sniadecki. "Hemodynamic Shear Regulates Intercellular Forces and Permeability of Endothelial Cell Monolayers." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80789.
Повний текст джерела