Готові списки джерел за темами / Abnormal childhood

Добірка наукової літератури з теми "Abnormal childhood"

Автор: Grafiati
Опубліковано: 7 липня 2024

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Зміст

  1. Статті в журналах
  2. Дисертації
  3. Книги
  4. Частини книг
  5. Тези доповідей конференцій
  6. Звіти організацій

Статті в журналах з теми "Abnormal childhood":

1

Loder, Randall T., Gretchen Huffman, Eugene Toney, L. Daniel Wurtz, and Robert Fallon. "Abnormal Rib Number in Childhood Malignancy." Spine 32, no. 8 (April 2007): 904–10. http://dx.doi.org/10.1097/01.brs.0000259834.28893.97.

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2

Murashkin, Nikolay N., Eduard T. Ambarchian, Roman V. Epishev, Alexander I. Materikin, Leonid A. Opryatin, Roman A. Ivanov, and Daria S. Kukoleva. "Photodermatoses in Childhood." Current Pediatrics 20, no. 5 (November 7, 2021): 360–69. http://dx.doi.org/10.15690/vsp.v20i5.2308.

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Photodermatoses is a heterogeneous group of diseases resulting from abnormal skin hypersensitivity to sunlight and presented as local or generalized rashes. Specific sensitivity of children's skin to ultraviolet is often the first sign or clinical symptom of photodermatosis. Abnormal photosensitivity can be represented by diverse group of primary idiopathic conditions or photo-mediated aggravation of existing dermatosis. Number of genetic genodermatoses, metabolic disorders and connective tissue diseases is also widely known. These conditions can manifest with photosensitivity associated to other extracutaneous clinical and laboratory features. Timely diagnosis of photosensitivity in childhood allows to minimize long-term complications associated with insufficient photoprotection.
3

Millichap, J. Gordon. "Abnormal Cerebral Blood Flow in Childhood Autism." Pediatric Neurology Briefs 14, no. 9 (September 1, 2000): 72. http://dx.doi.org/10.15844/pedneurbriefs-14-9-14.

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4

De Rossi, G., D. Pasqualetti, G. De Sanctis, M. Lopez, M. L. Moleti, V. Bottari, C. Guglielmi, and F. Mandelli. "Childhood Abnormal Expansion of Large Granular Lymphocytes." Acta Haematologica 73, no. 4 (1985): 206–9. http://dx.doi.org/10.1159/000206329.

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5

Crawley, Jane, Michael English, Catherine Waruiru, Isiah Mwangi, and Kevin Marsh. "Abnormal respiratory patterns in childhood cerebral malaria." Transactions of the Royal Society of Tropical Medicine and Hygiene 92, no. 3 (May 1998): 305–8. http://dx.doi.org/10.1016/s0035-9203(98)91023-9.

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6

Kohn, Gary L., Cathy Walston, Julie Feldstein, Brad W. Warner, Paul Succop, and William D. Hardie. "Persistent Abnormal Lung Function After Childhood Empyema." American Journal of Respiratory Medicine 1, no. 6 (December 2002): 441–45. http://dx.doi.org/10.1007/bf03257171.

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7

Pais, Ray C., Ellen Vanous, Bettye Hollins, Bahjat A. Faraj, Rogena Davis, Vernon M. Camp, and Abdelsalam H. Ragab. "Abnormal vitamin B6 status in childhood leukemia." Cancer 66, no. 11 (December 1, 1990): 2421–28. http://dx.doi.org/10.1002/1097-0142(19901201)66:11<2421::aid-cncr2820661130>3.0.co;2-m.

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8

Thanasiu, Page L. "Childhood Sexuality: Discerning Healthy From Abnormal Sexual Behaviors." Journal of Mental Health Counseling 26, no. 4 (October 1, 2004): 309–19. http://dx.doi.org/10.17744/mehc.26.4.y0ndah5y1hkm18cf.

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Empirically determined characteristics that mental health counselors can use as a reference when assessing the normalcy of sexual behaviors in preadolescent children are summarized. Once sexual behaviors have been determined to be problematic, mental health counselors need to be aware of and address factors that will affect children's sexual attitudes and behaviors.
9

Crawford, Christina, Gregory Stores, and Anne Thomson. "304 CHILDHOOD ASTHMA: PSYCHOLOGICAL EFFECTS OF ABNORMAL SLLEP." Pediatric Research 36, no. 1 (July 1994): 53A. http://dx.doi.org/10.1203/00006450-199407000-00304.

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10

Ohnishi, T. "Abnormal regional cerebral blood flow in childhood autism." Brain 123, no. 9 (September 1, 2000): 1838–44. http://dx.doi.org/10.1093/brain/123.9.1838.

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Статті в журналах

Дисертації з теми "Abnormal childhood":

1

Qiu, Deqiang, and 邱德強. "Diffusion tensor imaging in evaluating normal and abnormal white matter development in childhood." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41508324.

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2

Giunta, Michele. "Exosomal protein deficiencies : how abnormal RNA metabolism results in childhood-onset neurological diseases." Thesis, University of Newcastle upon Tyne, 2017. http://hdl.handle.net/10443/3669.

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RNA metabolism is of critical importance for normal cellular functions and needs to be finely tuned in order to maintain stable conditions within the cell. The exosome complex is the most important RNA processing machinery, responsible for the correct processing of many different types of RNAs and interacting with different co-factors which bind and carry specific subtypes of RNA for degradation to the complex. Mutations in exosome complex subunits (EXOSC3, EXOSC8) were reported to cause severe childhood onset complex neurological disorders presenting with pontocerebellar hypoplasia type 1 (PCH1), spinal muscular atrophy (SMA) and central nervous system hypomyelination. We have recently identified a homozygous pathogenic mutation in RNA Binding Motif Protein 7 RBM7, a subunit of the nuclear exosome targeting (NEXT) complex in a single patient with SMA-like phenotype and proved that RBM7 is a novel human disease gene related to the exosome complex. In order to understand the disease mechanism in RBM7 deficiency and to explore the role of exosome complex in neurodevelopment, we performed gene expression studies (RT-PCR, RNA sequencing) in human cells of patients carrying mutations in EXOSC8 and RBM7. Furthermore we performed functional studies in zebrafish (D. rerio) by morpholino oligonucleotide mediated knock-down of rbm7, exosc8 and exosc3 and also by introducing pathogenic mutations in exosomal protein genes in zebrafish embryos by the CRISPR/Cas9 system. We showed that mutations in RBM7 and EXOSC8 mutant fibroblasts cause differential expression of several different transcripts, 62 of them being shared between the two cell lines. Altered gene expression of some AU-rich element containing genes may potentially contribute to the clinical presentation. Knock-down of rbm7, exosc8 and exosc3 caused impaired neurodevelopment in zebrafish, illustrated by abnormal growth of motor neuron axons and failure to differentiate cerebellar Purkinje cells. RT-PCR analysis in zebrafish showed a dramatic increase in expression of atxn1b (an AU-rich element containing homolog of the human ATXN1 gene) in rbm7, exosc8 and exosc3 downregulated fish, which may be responsible for the cerebellar defects. We have successfully introduced several germline mutations with CRISPR/Cas9 technology in rbm7. Phenotype of the F1 mutants is milder than what observed with the morpholino oligonucleotide injected fish. Mutants at a closer look do not show any morphological defect but further experiment may indicate similar characteristics to the morphants, although more iv subtle. Further studies on the CRISPR/Cas9 generated zebrafish models will extend our knowledge on the disease mechanisms caused by defective RNA metabolism.
3

Qiu, Deqiang. "Diffusion tensor imaging in evaluating normal and abnormal white matter development in childhood." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B41508324.

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4

Vora, Komal Ashokbhai. "Effect of Childhood Type 1 Diabetes on Bone Health: Is Abnormal Bone Development During Childhood Related to Increased Fracture Risk?" Thesis, The University of Sydney, 2021. https://hdl.handle.net/2123/26802.

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Type 1 diabetes (T1D) is associated with multiple micro and macrovascular complications, with guidelines available on early screening and management. There has been growing interest in diabetes-related bone disease and fracture risk in adults with T1D. However, there is a paucity of knowledge on the effect of T1D on bone health in growing children and adolescents. Also, pathogenesis underlying diabetes-related bone disease is yet to be fully understood. Chapter 1 summarises the hypotheses and aims of our study. Chapter 2 is a review of the literature and provides the background to the study. It briefly summarises the known microvascular complications of T1D and the potential mechanisms involved in diabetes-related bone disease. It also describes the current knowledge of bone health in T1D with a particular focus on children and adolescents. Chapter 3 presents a cross-sectional study describing bone mineral density and structure in adolescents with T1D. It also describes the fracture rate and physical activity of adolescents in the study. The study explores associations between the various diabetes-related characteristics, microvascular complications and bone parameters. Chapter 4 presents future directions and potential areas of research in the field.
5

Toutain, Julien. "ITARD, SEGUIN, BOURNEVILLE : Une étude de la sensation dans les pédagogies spécialisées auprès des enfants anormaux au XIXème siècle." Electronic Thesis or Diss., Normandie, 2024. http://www.theses.fr/2024NORMR009.

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La question des sens, de l’éveil sensoriel et de la sensorialité est très présente dans le travail au quotidien auprès des enfants présentant des troubles envahissants du développement. Les neurosciences, la psychanalyse, la psychologie cognitivo-comportementaliste accordent tous, à leur façon, une grande importance à la question de la sensation dans le développement des enfants ou encore dans le fonctionnement de son processus psychique. Dans leurs écrits, Claude Wacjman, Jacques Hochmann notent comment la philosophie du XVIIIème siècle, et notamment celle de Condillac, a contribué à forger les premières méthodes éducatives spécialisées qui font, encore aujourd’hui, sentir leur influence. Trois pédagogues ont en effet marqué, en France, la prise en charge des enfants présentant des difficultés majeures dans leur développement, enfants appelés alors « anormaux » : Itard, Seguin et Bourneville. Cette thèse est donc l’occasion de venir interroger la place de la sensorialité dans la pédagogie spécialisée en choisissant une approche historique et en questionnant, au regard de cette notion, le travail de ces trois pionniers. Une analyse des différents ouvrages laissés par ces trois auteurs, de la philosophie de Condillac et de ses successeurs, permet de dégager des éléments de réponse quant à la structuration de leur pédagogie au regard de cette question de la sensorialité. Apparaissent alors deux modèles pédagogiques qui perçoivent le rôle de la sensation d’une manière différente : le premier fait de la sensation la condition indispensable au bon développement de l’enfant et le second propose un modèle global, incluant la sensorialité dans un ensemble d’éléments interconnectés. Ces différents modèles obligent alors les acteurs de la pédagogie spécialisée du XIXème à faire des choix, à porter leur attention sur des éléments spécifiques du développement de l’enfant et à créer des outils pour améliorer la prise en charge de chacun. Ces différents éléments, comme une boucle, font alors écho à des pratiques actuelles dans le domaine de l’accompagnement des enfants présentant des troubles variés du développement
The question of the senses, sensory awakening and sensoriality is very present in the day-to-day work with children with Pervasive Developmental Disorder. Neuroscience, psychoanalysis and cognitive-behavioural psychology all attach great importance, in their own way, to the question of sensation in children's development and in the functioning of their psychological processes. In their writings, Claude Wacjman and Jacques Hochmann note how eighteenth-century philosophy, and Condillac's in particular, helped to forge the first specialised educational methods, whose influence is still felt today. In France, three educationalists made their mark in the care of children with major developmental difficulties, known at the time as 'abnormal' children: Itard, Seguin and Bourneville. This thesis is therefore an opportunity to examine the place of sensoriality in special education by taking a historical approach and examining the work of these three pioneers in the light of this concept. An analysis of the various works left by these three authors, of Condillac's philosophy and that of his successors, will provide some clues as to how their pedagogy was structured in relation to this question of sensoriality. Two pedagogical models emerge which perceive the role of sensation in a different way: the first makes sensation the essential condition for the child's proper development, while the second proposes a global model, including sensoriality in a set of interconnected elements. These different models meant that those involved in special education in the 19th century had to make choices, focus their attention on specific aspects of children's development and create tools to improve the care they received. These different elements, like a loop, then echo current practices in the field of support for children with various developmental disorders
6

Long, Zsofia Banhegyi. "Incidence and clinical relevance of abnormal complete blood counts in survivors of childhood cancer." 2005. http://edissertations.library.swmed.edu/pdf/LongZ032105/LongZsofia.pdf.

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7

Carter, Tamara-Lyn. "An exploration of the relationship between unintegrated primitive reflexes and symptoms of anxiety in children between 10-13 years in the Western Cape Province of South Africa." Diss., 2020. http://hdl.handle.net/10500/27005.

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Bibliography: leaves 149-178
Anxiety Disorder is one of the most common disorders experienced by children and, if not managed, can extend into adulthood. Research has established a link between unintegrated primitive reflexes (UPR) and Learning Disorders. Learning Disorders are often co-morbid with symptoms of anxiety, however, the relationship between symptoms of anxiety and UPR have not been studied. This study aims to explore the relationship between the UPR and symptoms of anxiety in children between 10 – 13 years of age. No correlation was found between the total primitive reflex score and total symptoms of anxiety score; however, a significant relationship was found between symptoms of anxiety and the Moro, Plantar and Spinal Galant reflex. These UPR play an important role in balance. Research on balance dysfunction indicates a relationship with symptoms of anxiety. Prenatal maternal stress, common childhood illness and comorbidity with ADHD were also found to be factors in symptoms of anxiety in children.
Psychology
M.A. (Psychology)

Книги з теми "Abnormal childhood":

1

Kurt, Simons, and National Research Council (U.S.). Committee on Vision., eds. Early visual development, normal and abnormal. New York: Oxford University Press, 1993.

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2

Ginsberg, Rachel E., Samantha Morrison, and Anthony Puliafico. Pediatric OCD. Edited by Christopher Pittenger. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228163.003.0003.

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This chapter outlines the clinical presentation and course of pediatric OCD, discusses its etiology and phenomenology, and describes principal assessment methods for evaluation of symptoms. Obsessive-compulsive disorder (OCD) often manifests during childhood and adolescence. Symptom presentation in children is similar to that in adults and is typically characterized by the presence of both obsessions and compulsions. Pediatric OCD is highly comorbid with other psychiatric disorders, and evidence suggests abnormal brain functioning in youth with OCD. The onset and progression of OCD in childhood have developmental implications, given the associated distress and interference with academic, social, and home functioning. Multiinformant and multidiagnostic evaluation, including administration of evidence-based semistructured clinical interviews and rating scales, is optimal.
3

NA. Behavr Disorder Childhood&abnorm Psych Pkg. Addison Wesley Longman, 2006.

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4

Frazier, Linda M., and Deborah Barkin Fromer. Reproductive and Developmental Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190662677.003.0027.

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This chapter describes reproductive disorders and developmental disorders, with a focus on their recognition and prevention. Certain hazardous exposures at sufficient doses during preconception among men and women have been shown to increase the risk for infertility, miscarriage (spontaneous abortion) and birth defects. Women’s exposures during pregnancy can cause fetal death, congenital anomalies, and neurodevelopmental disorders. Fathers’ occupational exposures can secondarily expose pregnant women through contamination of the home environment. Numerous chemical pollutants readily cross the placenta and are transmitted into breast milk. Prenatal and perinatal exposures have been linked to abnormal development of the immune system, childhood cancer, and learning disabilities. Specific examples, such as lead and dibromochloropropane, are provided in the chapter to illustrate general concepts. A final section addresses evaluation and control of risk.
5

Morava, Eva, and Mirian C. H. Janssen. Congenital Disorders of Glycosylation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0063.

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Congenital disorders of glycosylation (CDGs) are usually diagnosed during infancy or childhood with severe multisystem disorder and neurologic presentation. With the increasing number of surviving adult patients, recognition of the distinct adult phenotype and awareness of the diagnostic difficulties in adulthood is essential. Patients with O-glycosylation defects or with abnormal dolichol synthesis might present first in adulthood. The majority of cases with adult CDG have a neurologic disease with intellectual disability, ataxia, speech disorder, visual disturbance, and skeletal findings. Psychological abnormalities are also common. Thrombotic complications and endocrine dysfunction might persist to adulthood. MPI-CDG, the only treatable form of CDG, might progress to chronic liver failure. Genetic testing is recommended in suspected cases, since transferrin screening analysis can be normal in adults, even in N-linked glycosylation disorders.
6

Paris, Joel. Thinking Interactively. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190601010.003.0004.

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The human mind favors linear thinking, with single causes leading to single effects. Thinking interactively is much more difficult. Understanding mental disorders as due to chemical imbalances or abnormal neural connections is tempting. However, it is wrong to view the neural level as more “real” than measures of the mind. This kind of thinking pays lip service to psychosocial factors but loses sight of the important role that life events play in the etiology of mental disorders. In the past, psychotherapists were just as blindly linear in their thinking. They made broad generalizations, oversimplifying the role of life experiences, sometimes attributing all psychopathology to adverse events in childhood. In parallel with the reductionism of biological psychiatry, these models failed to consider the complexity of pathways from risk factors to outcomes. A more scientifically valid view is that mental disorders arise from complex interactions between genetic vulnerability and psychosocial adversity.
7

Case, Dr John B., and John B. Case. An Abnormally Normal Childhood: A free range childhood during the Great Depression,WW11, the postwar period and commencement of training as a ... Alberta, Canada. Life as a rural surgeon.). BAC/LAC, 2021.

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8

Hall, Andrew, and Shamima Rahman. Mitochondrial diseases and the kidney. Edited by Neil Turner. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0340.

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Mitochondrial disease can affect any organ in the body including the kidney. As increasing numbers of patients with mitochondrial disease are either surviving beyond childhood or being diagnosed in adulthood, it is important for all nephrologists to have some understanding of the common renal complications that can occur in these individuals. Mitochondrial proteins are encoded by either mitochondrial or nuclear DNA (mtDNA and nDNA, respectively); therefore, disease causing mutations may be inherited maternally (mtDNA) or autosomally (nDNA), or can arise spontaneously. The commonest renal phenotype in mitochondrial disease is proximal tubulopathy (Fanconi syndrome in the severest cases); however, as all regions of the nephron can be affected, from the glomerulus to the collecting duct, patients may also present with proteinuria, decreased glomerular filtration rate, nephrotic syndrome, water and electrolyte disorders, and renal tubular acidosis. Understanding of the relationship between underlying genotype and clinical phenotype remains incomplete in mitochondrial disease. Proximal tubulopathy typically occurs in children with severe multisystem disease due to mtDNA deletion or mutations in nDNA affecting mitochondrial function. In contrast, glomerular disease (focal segmental glomerulosclerosis) has been reported more commonly in adults, mainly in association with the m.3243A<G point mutation. Co-enzyme Q10 (CoQ10) deficiency has been particularly associated with podocyte dysfunction and nephrotic syndrome in children. Underlying mitochondrial disease should be considered as a potential cause of unexplained renal dysfunction; clinical clues include lack of response to conventional therapy, abnormal mitochondrial morphology on kidney biopsy, involvement of other organs (e.g. diabetes, cardiomyopathy, and deafness) and a maternal family history, although none of these features are specific. The diagnostic approach involves acquiring tissue (typically skeletal muscle) for histological analysis, mtDNA screening and oxidative phosphorylation (OXPHOS) complex function tests. A number of nDNA mutations causing mitochondrial disease have now been identified and can also be screened for if clinically indicated. Management of mitochondrial disease requires a multidisciplinary approach, and treatment is largely supportive as there are currently very few evidence-based interventions. Electrolyte deficiencies should be corrected in patients with urinary wasting due to tubulopathy, and CoQ10 supplementation may be of benefit in individuals with CoQ10 deficiency. Nephrotic syndrome in mitochondrial disease is not typically responsive to steroid therapy. Transplantation has been performed in patients with end-stage kidney disease; however, immunosuppressive agents such as steroids and tacrolimus should be used with care given the high incidence of diabetes in mitochondrial disease.
9

Meyers, Marc, and Derf Backderf. My Friend Dahmer (Movie Tie-In Edition). Abrams, Inc., 2017.

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10

My Friend Dahmer: A Graphic Novel. Abrams Comicarts, 2012.

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Частини книг з теми "Abnormal childhood":

1

Sauerbrun-Cutler, May-Tal, Christine Mullin, and Avner Hershlag. "Survivors of Childhood Cancer and Cancer Treatments." In Abnormal Female Puberty, 241–59. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-27225-2_12.

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2

Sergi, Consolato M. "Placenta, Abnormal Conception, and Prematurity." In Pathology of Childhood and Adolescence, 1409–569. Berlin, Heidelberg: Springer Berlin Heidelberg, 2020. http://dx.doi.org/10.1007/978-3-662-59169-7_18.

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3

Miller, Ronald B. "Psychological suffering in childhood." In Not so abnormal psychology: A pragmatic view of mental illness., 95–119. Washington: American Psychological Association, 2015. http://dx.doi.org/10.1037/14693-004.

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4

Gupta, Nidhi, Kashish Goel, and Anoop Misra. "Childhood Obesity in Developing Countries: Facets of Abnormal Growth." In Handbook of Growth and Growth Monitoring in Health and Disease, 1491–524. New York, NY: Springer New York, 2011. http://dx.doi.org/10.1007/978-1-4419-1795-9_89.

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5

Lee, Kyunghwa. "Who is Normal? Who is Abnormal? Rethinking Child Development from a Cultural Psychological Perspective." In Developmentalism in Early Childhood and Middle Grades Education, 35–58. New York: Palgrave Macmillan US, 2010. http://dx.doi.org/10.1057/9780230107854_3.

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6

Tabak, Jana. "“Children Without Childhood”: Representations of the Child-Soldier as an International Emergency." In The Politics of Children’s Rights and Representation, 161–80. Cham: Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-04480-9_7.

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AbstractConsidering the prevailing discourse about the child-soldier, whose most iconic figure is a poor, vulnerable, prepubescent, male African who carries a gun bigger than he is, this chapter investigates how child-soldiers are invariably framed as an essentially deviant and pathological child—and as such a threat to world security—in need of solution. Regardless of many historical examples of children’s participation in war, the child-soldier is assumed to be a new international emergency, an exception to the norm of the child, owing primarily to the outbreak of “new wars” in the post–Cold War era. The focus of this chapter turns to two main discourses that articulate and authorize the limits that (re)produce the child-soldier as an international problem, setting boundaries within which only certain subjects, narratives and responses are admitted: (1) the discourse of the law, that is, international legal standards that articulate children’s participation in war as something that is wrong and must be banned under international law; and (2) what I call the “discourse of the norm,” which is analyzed through the three contrasting images of the child-soldier as dangerous and disorderly, the hapless victim, and the redeemed hero, as identified by Myriam Denov (Child Soldiers: Sierra Leone’s Revolutionary United Front. Cambridge, UK: Cambridge University Press, 2010). The discourse of the norm, in particular, makes visible child-soldiers as a pathology, excluding their aspects of disorder, dysfunction, and risk from the accepted boundaries of what is to be a child and its childhood. In this case, it is not only that children’s participation in wars is wrong, but it is absolutely abnormal once their childhood has been lost together with any semblance of the “civilized world.” At the end of the day, the logic of opposite extremes—to be a child-soldier is to be an innocent victim or to be a feared monster—operates to (re)produce children as targets of international intervention (or protection) with no chance of autonomous decision-making; child-soldiers are either the objects of exploitation or the objects of salvation.
7

"Childhood behaviour disorders." In Abnormal Psychology, 1–36. Psychology Press, 2003. http://dx.doi.org/10.4324/9780203496480-1.

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"Psychophysiological Research on Childhood Psychopathology." In Advanced Abnormal Child Psychology, 71–97. Routledge, 2000. http://dx.doi.org/10.4324/9781410605320-10.

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Scher, Mark S. "Neonatal Electroencephalography: Abnormal Features." In Clinical Neurophysiology of Infancy, Childhood, and Adolescence, 273–301. Elsevier, 2006. http://dx.doi.org/10.1016/b978-0-7506-7251-1.50017-4.

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"III: Problems With Onset in Late Childhood or Adolescence: An Introduction." In Abnormal Child Psychology, 440–518. Routledge, 2008. http://dx.doi.org/10.4324/9780203893258-17.

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Тези доповідей конференцій з теми "Abnormal childhood":

1

Amrulloh, Yusuf A., and Lalu M. H. Maulidin. "Spectral Analysis of Abnormal Breath Sounds in Childhood Pneumonia." In 2018 International Symposium on Electronics and Smart Devices (ISESD). IEEE, 2018. http://dx.doi.org/10.1109/isesd.2018.8605486.

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2

Abshire, T., L. Fink, J. Christian, J. O'Connell, and W. Hathaway. "THE DYSFIBRINOGEN OF CHILDHOOD NEPHROSIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643334.

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An abnormal fibrinogen (Fib) related to increased sialic acid (SA) has been described in adults with liver disease. This dysfibrinogen (Dysfib) seems most like fetal Fib. A review of 11 patients with nephrosis revealed an unexplained prolonged thrombin time (TT) and otherwise normal coagulation studies. Based on these observations, we sought to answer whether the prolonged TT defined a Dysfib and if this abnormal Fib was similar to fetal Fib. Pooled adult, fetal plasma, and the plasma of 3 patients with nephrosis were studied with TT and reptilase times (RT). Fib was measured by functional (Fib-act) and immunologic (Fib-ag) assays. An enzyme linked immunosorbent assay (ELISA) was established using antifibrinogen as the first antibody and either peroxidase conjugated Fib or a lectin (Limulus Polyphemus) specific for SA as the second antibody. The optical density was recorded per μgm Fib for both conjugated antifibrinogen or lectin and the ratio compared in order to estimate SA reactivity. Patient 3 was also studied by: 1) crossed immunoelectrophoresis (CIE) employing lectin in the first dimension and 2) polyacrylamide gel electrophoresis (PAGE) with transfer to nitrocellulose paper using Western Blot technique.Results of the CIE showed patient 3 and fetal plasma were similar in electrophoretic pattern and different from adult plasma. The PAGE with Western Blot revealed a similar pattern of Fib for patient 3, fetal and adult plasma. We conclude that the prolonged TT and RT, the greater amount of Fib-ag when compared to Fib-act in patients 1-3 and fetal plasma and the absence of evidence for Fib degradation products, support the diagnosis of Dysfib. The similarity of the CIE for patient 3 and fetal plasma and the difference between ELISA lectin/Fib ratio of patients 1-3 and fetal compared with adult plasma suggest that the Dysfib of nephrosis may be similar to fetal Fib.
3

Ryland, Kimberly, Carl A. Nelson, and Thomas Hejkal. "Design and Development of a Novel Infant Surgical Table for 4-DOF Patient Manipulation." In ASME 2007 International Design Engineering Technical Conferences and Computers and Information in Engineering Conference. ASMEDC, 2007. http://dx.doi.org/10.1115/detc2007-34830.

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Retinopathy of Prematurity, caused by abnormal blood vessel development in the retina of premature infants, is a leading cause of childhood blindness. It is treated using laser photocoagulation. Current methods require the surgeon to assume awkward standing positions, which can result in injury to the surgeon if repeated often. To assist surgeons in providing quality care and prevent occupational injury, a new infant surgical table was designed. The engineered solution is an attachment to a standard surgical table, saving cost and space. This takes advantage of the adjustable height and tilt provided by the standard table, while 360° rotation designed into the attachment allows the surgeon to sit during surgery. The critical cords and tubes are routed through the attachment to avoid pulling and kinking. A four-bar locking mechanism allows easy attachment to standard medical railing. Finally, a straight line mechanism provides positive locking of the rotation, allowing precise positioning of the infant.
4

Sadikova, Daniela. "ASSESSMENT OF BODY POSTURE OF 6-7 YEARS OLD CHILDREN IN RELATION TO THE BMI." In INTERNATIONAL SCIENTIFIC CONGRESS “APPLIED SPORTS SCIENCES”. Scientific Publishing House NSA Press, 2022. http://dx.doi.org/10.37393/icass2022/155.

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ABSTRACT Introduction: Increased weight in childhood is a determining factor in developing several diseases in adulthood. The poor posture of the child might be a result of the rapidly growing musculoskeletal system, the increased BMI, or a combination of both. This study aims to investigate the relation between BMI and poor posture in 7-6 years old children. Material and Methodology: The study included 2 kindergartens in Sofia and was held in September 2018. The New York Posture Rating Chart was used to evaluate the posture. The anthropometric data were assessed and the BMI was calculated by BMI-for-age (5-19) years. The screening covered 112 children with a mean age of 6.14 years. Results: Poor posture was identified in 32 (28.5%) of the subjects. Abnormal BMI was amounted to 22 (19.6%) of the children: 4 (3.5%) underweight, 16 (14.3%) overweight and 2 (1.7 %) obese. The increase in BMI correlated with a higher chance of poor posture was identified by almost 11.6 % of the pupils.
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Scott, FRS, JW Walker, and PR Ramesh. "G252(P) How relevant is an incidental finding of abnormal cardiac silhouette on chest x-ray?" In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 13–15 March 2018, SEC, Glasgow, Children First – Ethics, Morality and Advocacy in Childhood, The Journal of the Royal College of Paediatrics and Child Health. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/archdischild-2018-rcpch.245.

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6

Neamah Almkhelif, Shaymaa. "A Psychoanalytic View of Louisa's Character in Charles Dickens' Novel Hard Times." In VIII. International Congress of Humanities and Educational Research. Rimar Academy, 2023. http://dx.doi.org/10.47832/ijhercongress8-11.

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The current study considers Sigmund Freud's theory of Psychoanalysis in Charles Dickens' novel Hard Times. It aims to analyze the character of Louisa Gradgrind from a psychoanalytic perspective. Theory of Psychoanalysis performs this task as it deals with the analysis of the human mind. Sigmund Freud is the most prominent psychologist who developed the theory of Psychoanalysis. The field is established as Freud starts treating a young woman, who suffers massive hysterical attacks in 1881. Later, Freud investigates the unconscious under normal and abnormal conditions. Furthermore, Freud develops the theory as soon as he introduces The Interpretation of Dreams to the field of Psychology. This book, which is written in 1900, marks a turning point in the field because it tackles the role of dreams, as a part of the unconscious, in revealing the mechanisms of fears, delusions and fixed ideas that are rooted in childhood experiences. Hence, the study takes the character of Louisa as an example through which Dickens psychoanalytically manifests the way in which the mentioned mechanisms work in the human mind and their impact on the other aspects of life
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Hoskote, A., M. Lakhanpaul, V. Banks, J. Wray, and K. Brown. "G240 Using the delphi method to develop consensus on a referral pathway for potentially abnormal neurodevelopment in children with heart disease." In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 13–15 March 2018, SEC, Glasgow, Children First – Ethics, Morality and Advocacy in Childhood, The Journal of the Royal College of Paediatrics and Child Health. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/archdischild-2018-rcpch.233.

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8

Banks, V., A. Hoskote, S. Kakat, D. Rideout, M. Lakhanpaul, C. Pagel, R. Franklin, and T. Witter. "G245 Risk factors for abnormal development and cognitive function in children with congenital heart disease and implications for service provision in the uk." In Royal College of Paediatrics and Child Health, Abstracts of the Annual Conference, 13–15 March 2018, SEC, Glasgow, Children First – Ethics, Morality and Advocacy in Childhood, The Journal of the Royal College of Paediatrics and Child Health. BMJ Publishing Group Ltd and Royal College of Paediatrics and Child Health, 2018. http://dx.doi.org/10.1136/archdischild-2018-rcpch.238.

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9

Santiago, Mayhara Rosany da Silva, Renata Amaral Andrade, Ana Caroline Paiva Simeão, and Heloisy Maria Nunes Galvão. "Congenital myasthenic syndrome due rapsyn mutation presenting with predominant ocular symptoms and good therapeutic response with salbutamol." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.622.

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Introduction: Rapsyn mutations are found in Congenital Myasthenic Syndromes (CMS), usually described with early onset, hypotonia and respiratory insufficiency. Late onset phenotypes had already been described, with mild limb weakness and semiptosis. Episodic respiratory symptons and lack of ophthalmoparesis are considered hallmarks of rapsyn – CMS. The aim is to describe an unusual phenotype of a commom mutation of Rapsyn CMS and therapeutic response with salbutamol. Case report: A 29-year-old female patient was referred complaining of diplopia and fatigable assymmetrical semiptosis during the last six years. She pratices physical exercise, with no fatigue or weakness in limbs. Her past medical history was unremarkable except for drop neck episodes triggered by viral infectons during childhood, with first episode taking place around two years of age. Her parents were consaguinous, howeve no similar clinical picture was reported in family members. In clinical examination besides assymetrical semiptosis and ophthalmoparesis, was noted winged scapula, with normal muscle trofism and strength in four limbs. Laboratory investigation showed negative acetylcholine receptor antibodies (anti-AChR), and negative muscle-specific kinase antibodies (anti-MuSK). Single fiber electromyography revealed abnormal jitter. Genetic panel found a pathogenic homozygous mutation in the RAPSN gene (chr11:47.448.079G>T; p.Asn88Lys). She was treated with pyridostigmine and showed poor response. We opted to start salbutamol with marked clinical improvement. Conclusion: Rapsyn-CMS may present as late episodic diplopia, with semiptosis and ophthalmoparesis, and no limb or respiratory muscle weakness. That diagnosis possibility should be also considered in patients with late onset ocular symptons, incomplete response to pyridostigmine and negative assays for auto antibodies.
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Teixeira, Elaine Calumby, Camila Rodrigues Nepomuceno, Maria Sheila Guimarães Rocha, and Eduardo de Paula Estephan. "Charcot-Marie-Tooth type 1F with bi-allelic NEFL mutation." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.628.

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Introduction: Charcot-Marie-Tooth disease (CMT) is the most common hereditary neuropathy, with a diverse phenotypic and genotypic spectrum. The main clinical features are onset during infancy, slowly progressing symptoms and foot deformities, especially if there is a positive family history, although the lack of family awareness can be present. Over 70 distinct genes have been associated, however, their genetic diagnosis can be challenging, especially if we consider the fact that the same gene can transmit disease either dominantly or recessively. The aim to describe a case of CMT1F as a rare case of recessive demyelinating hereditary neuropathy. Clinical case: A 25-year-old woman, born of consanguineous parents, had history of distal weakness and burning sensation in lower limbs, with onset in infancy. Her childhood was marked for abnormal gait and falls, and evolved with foot deformities, requiring surgical corrections. The symptoms progressed slowly and reached upper limbs in few years. On physical evaluation was noted: muscle weakness of upper and lower limbs, predominantly distal, associated with atrophy, foot drop and absent reflexes. Electroneuromyography demonstrated signs of chronic demyelinating polyneuropathy. An initial sequencing analysis of the PMP22 gene was indicated, with normal results. A panel for neuropathies was performed, showing a homozygous frameshift mutation in NEFL (p.Lys362Glufs*2; c.1084_1085delAA), classified as probably pathogenic variant. Conclusion: CMT due to bi-allelic NEFL mutations is a rare condition that should be considered in hereditary demyelinating neuropathy, especially when recessive inheritance is suspected. Our study illustrates this condition and brings attention to the importance of the disponibility of high throughput genetic tests.

Звіти організацій з теми "Abnormal childhood":

1

Wu, Bin, Lixia Guo, Kaikai Zhen, and Chao Sun. Diagnostic and prognostic value of miRNAs in hepatoblastoma: A systematic review with meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, November 2021. http://dx.doi.org/10.37766/inplasy2021.11.0045.

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Review question / Objective: Background and aim: Increasing evidence has revealed the valuable diagnostic and prognostic applications of dysregulated microRNAs (miRNAs) in hepatoblastoma (HB), the most common hepatic malignancy during childhood. However, these results are inconsistent and remain to be elucidated. In the present study, we aimed to systematically compile up-to-date information regarding the clinical value of miRNAs in HB. Methods: Articles concerning the diagnostic and prognostic value of single miRNAs for HB were searched from databases. The sensitivity (SEN), specificity (SPE), positive and negative likelihood ratios (PLR and NLR), diagnostic odds ratio (DOR), area under the curve (AUC), and hazard ratios (HRs) were separately pooled to explore the diagnostic and prognostic performance of miRNA. Subgroup and meta-regression analyses were further carried out only in the event of heterogeneity. Results: In all, 20 studies, involving 264 HB patients and 206 healthy individuals, met the inclusion criteria in the six included literature articles. For the diagnostic analysis of miRNAs in HB, the pooled SEN and SPE were 0.76 (95% CI: 0.72–0.80) and 0.75 (95% CI: 0.70–0.80), respectively. Moreover, the pooled PLR was 2.79 (95% CI: 2.12–3.66), NLR was 0.34 (95% CI: 0.26–0.45), DOR was 10.24 (95% CI: 6.55–16.00), and AUC was 0.83, indicating that miRNAs had moderate diagnostic value in HB. For the prognostic analysis of miRNAs in HB, the abnormal expressions of miR-21, miR-34a, miR-34b, miR-34c, miR-492, miR-193, miR-222, and miR-224 in patients were confirmed to be associated with a worse prognosis. The pooled HR was 1.74 (95% CI: 1.20–2.29) for overall survival (OS) and 1.74 (95% CI: 1.31–2.18) for event-free survival (EFS), suggesting its potential as a prognostic indicator for HB. Conclusion: To the best of our knowledge, this is the first comprehensive systematic review and meta-analysis that examines the diagnostic and prognostic role of dysregulated miRNAs in HB patients. The combined meta-analysis results supported the previous individual finds that miRNAs might provide a new, noninvasive method for the diagnostic and prognostic analyses ofHB.
2

Accelerated cortical thinning correlates with early signs of depression. ACAMH, August 2018. http://dx.doi.org/10.13056/acamh.10568.

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The brain undergoes structural changes as it develops over childhood, but whether abnormal structural changes are associated with emerging depressive symptoms in adolescence is unknown. Now, a longitudinal study that enrolled 205 participants aged 8-25 years without signs of depression has used magnetic resonance imaging (MRI) to monitor these brain changes over adolescence.

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