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Bibliografias temáticas / Vitamin D Metabolism

Literatura científica selecionada sobre o tema "Vitamin D Metabolism"

Autor: Grafiati

Publicado: 4 de junho de 2021

Última modificação: 31 de janeiro de 2023

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Índice

Artigos de revistas
Teses / dissertações
Livros
Capítulos de livros
Trabalhos de conferências
Relatórios de organizações

Artigos de revistas sobre o assunto "Vitamin D Metabolism":

1

Goodman, William G. "Vitamin D metabolism". Current Opinion in Orthopaedics 5, n.º 5 (outubro de 1994): 60–65. http://dx.doi.org/10.1097/00001433-199410000-00010.

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2

Swaminathan, R. "Vitamin D Metabolism". Annals of Clinical Biochemistry: An international journal of biochemistry and laboratory medicine 32, n.º 1 (1 de janeiro de 1995): 98–100. http://dx.doi.org/10.1177/000456329503200114.

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3

Christakos, Sylvia, Dare V. Ajibade, Puneet Dhawan, Adam J. Fechner e Leila J. Mady. "Vitamin D: Metabolism". Rheumatic Disease Clinics of North America 38, n.º 1 (fevereiro de 2012): 1–11. http://dx.doi.org/10.1016/j.rdc.2012.03.003.

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4

Christakos, Sylvia, Dare V. Ajibade, Puneet Dhawan, Adam J. Fechner e Leila J. Mady. "Vitamin D: Metabolism". Endocrinology and Metabolism Clinics of North America 39, n.º 2 (junho de 2010): 243–53. http://dx.doi.org/10.1016/j.ecl.2010.02.002.

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5

Lehmann, Bodo, e Michael Meurer. "Vitamin D metabolism". Dermatologic Therapy 23, n.º 1 (janeiro de 2010): 2–12. http://dx.doi.org/10.1111/j.1529-8019.2009.01286.x.

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6

Salle, B. L., F. H. Glorieux e N. Bishop. "Perinatal vitamin D metabolism". Seminars in Neonatology 3, n.º 2 (maio de 1998): 143–47. http://dx.doi.org/10.1016/s1084-2756(98)80032-8.

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7

Salle, B. L., F. H. Glorieux e E. E. Delvin. "Perinatal Vitamin D Metabolism". Neonatology 54, n.º 4 (1988): 181–87. http://dx.doi.org/10.1159/000242850.

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8

Ramasamy, Indra. "Vitamin D Metabolism and Guidelines for Vitamin D Supplementation". Clinical Biochemist Reviews 41, n.º 3 (8 de dezembro de 2020): 103–26. http://dx.doi.org/10.33176/aacb-20-00006.

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Vitamin D is essential for bone health and is known to be involved in immunomodulation and cell proliferation. Vitamin D status remains a significant health issue worldwide. However, there has been no clear consensus on vitamin D deficiency and its measurement in serum, and clinical practice of vitamin D deficiency treatment remains inconsistent. The major circulating metabolite of vitamin D, 25-hydroxyvitamin D (25(OH)D), is widely used as a biomarker of vitamin D status. Other metabolic pathways are recognised as important to vitamin D function and measurement of other metabolites may become important in the future. The utility of free 25(OH)D rather than total 25(OH)D needs further assessment. Data used to estimate the vitamin D intake required to achieve a serum 25(OH)D concentration were drawn from individual studies which reported dose-response data. The studies differ in their choice of subjects, dose of vitamin D, frequency of dosing regimen and methods used for the measurement of 25(OH)D concentration. Baseline 25(OH)D, body mass index, ethnicity, type of vitamin D (D2 or D3) and genetics affect the response of serum 25(OH)D to vitamin D supplementation. The diversity of opinions that exist on this topic are reflected in the guidelines. Government and scientific societies have published their recommendations for vitamin D intake which vary from 400–1000 IU/d (10–25 µg/d) for an average adult. It was not possible to establish a range of serum 25(OH)D concentrations associated with selected non-musculoskeletal health outcomes. To recommend treatment targets, future studies need to be on infants, children, pregnant and lactating women.

9

Thomas, Melissa K., e Marie B. Demay. "VITAMIN D DEFICIENCY AND DISORDERS OF VITAMIN D METABOLISM". Endocrinology and Metabolism Clinics of North America 29, n.º 3 (setembro de 2000): 611–27. http://dx.doi.org/10.1016/s0889-8529(05)70153-5.

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10

Jovičić, Snežana, Svetlana Ignjatović e Nada Majkić-Singh. "Biochemistry and metabolism of vitamin D / Biohemija i metabolizam vitamina D". Journal of Medical Biochemistry 31, n.º 4 (1 de outubro de 2012): 309–15. http://dx.doi.org/10.2478/v10011-012-0028-8.

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Summary Vitamin D is not technically a vitamin, since it is not an essential dietary factor. It is rather a prohormone produced photochemically in the skin from 7-dehydrocholesterol. Vitamin D and its metabolites may be categorized as either cholecalciferols or ergocalciferols. Cholecalciferol (vi - tamin D3) is the parent compound of the naturally occurring family and is produced in the skin from 7-dehydrocholesterol on exposure to the ultraviolet B portion of sunlight. Vitamin D2 (ergocalciferol), the parent compound of the other family, is manufactured by irradiation of ergosterol produced by yeasts and its potency is less than one-third of vitamin D3’s potency. The steps in the vitamin D endocrine system include the following: 1) the photoconversion of 7- dehydrocholesterol to vitamin D3 in the skin or dietary intake of vitamin D3; 2) metabolism of vitamin D3 by the liver to 25-hydroxyvitamin-D3 [25(OH)D3 ], the major form of vitamin D circulating in the blood compartment; 3) conversion of 25(OH)D3 by the kidney (functioning as an endocrine gland) to the hormone 1,25-dihydroxyvitamin D3 [1,25(OH)2D3 ]; 4) systemic transport of the dihydroxylated metabolite 1,25(OH)2D3 to distal target organs; and 5) binding of 1,25(OH)2D3 to a nuclear receptor (VDR) at target organs, followed by generation of appropriate biological responses. The activation of vitamin D to its hormonal form is mediated by cytochrome P450 enzymes. Six cytochrome P450 (CYP) isoforms have been shown to hydroxylate vitamin D. Four of these, CYP27A1, CYP2R1, CYP3A4 and CYP2J3, are candidates for the enzyme vitamin D 25-hy - droxylase that is involved in the first step of activation. The highly regulated, renal enzyme 25-hydroxyvitamin D-1a-hy - dro xylase contains the component CYP27B1, which completes the activation pathway to the hormonal form 1,25(OH)2D3. A five-step inactivation pathway from 1,25(OH)2D3 to calcitroic acid is attributed to a single multifunctional CYP, CYP24A1, which is transcriptionally in du - ced in vitamin D target cells by the action of 1,25(OH)2D3. An additional key component in the operation of the vitamin D endocrine system is the plasma vitamin D binding protein (DBP), which carries vitamin D3 and its metabolites to their metabolism and target organs. DBP is a specific, high-affinity transport protein. It is synthesized by the liver and circulates in great excess, with fewer than 5% of the binding sites normally occupied. 1,25(OH)2D3, acts as a ligand for a nuclear transcription factor, vitamin D receptor - VDR, which like all other nuclear receptors, regulates gene transcription and cell function. The widespread presence of VDR, and the key activating (1a-hydroxylase, CYP27B1) and inactivating (24-hydroxylase, CYP24A1) en - zy mes in most mammalian cells means that the cells in these tissues have the potential to produce biological res pon ses, depending on the availability of appropriate amounts of vi - tamin D3. Thanks to this widespread presence of elements of vitamin D endocrine system, its biological features are being recognized outside bone tissue, i.e. calcium and pho - sphate metabolism.

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Teses / dissertações sobre o assunto "Vitamin D Metabolism":

1

Hall, Judith. "Physiology and pathophysiology of vitamin D metabolism". Thesis, University of Newcastle upon Tyne, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.377459.

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2

Bitler, Chad. "Vitamin D and Markers of Glucose Metabolism". University of Cincinnati / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1416231511.

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3

Asano, Lisa. "Vitamin D metabolite, 25-Hydroxyvitamin D, regulates lipid metabolism by inducing degradation of SREBP/SCAP". 京都大学 (Kyoto University), 2017. http://hdl.handle.net/2433/225512.

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4

Turner, Andrew. "Vitamin D metabolism in an In Vitro mineralisation model /". Title page and abstract only, 1999. http://web4.library.adelaide.edu.au/theses/09SB/09sbt9438.pdf.

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5

Laird, Eamon John. "Vitamin D status and metabolism : implications for bone health". Thesis, Ulster University, 2012. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.674922.

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In addition to its established role in bone health, vitamin D (2S(OH)D) may also have a role in modulating immune function and early life development. Despite recent advances, there is a lack of consensus with regards to the optimal vitamin D cut-offs for multiple health outcomes and this uncertainty is further compounded by the wide measurement variability for the vitamin. Consequently, the work described in this thesis aimed to explore these areas of controversy. Using data from ongoing studies at the University of Ulster, a comparison study (n 131), of vitamin D status in the two most widely used methods (liquid chromatography mass spectrometry (LC-MS/MS) and enzyme immunoassay (ELISA)) of measurement was undertaken. Significant variation in definition of status was observed, with overestimation of vitamin D concentrations by ELISA >2S% compared to LC-MS/MS. In a second study, using LC-MS IMS, the vitamin D status and markers of bone health of a sample of older Irish adults (n 1936) form the Trinity, Ulster Department of Agriculture (TUDA) study was assessed. A total of 16% were vitamin D deficient «2Snmolll) and 42% were deemed to be insufficient (2S-S0nmolll). These levels of nonoptimal vitamin D concentrations were coupled with high rates of impaired bone health (31 % classified as osteopenic and 18% osteoporotic from BMD measures). A higher prevalence of impaired bone health and vitamin D inadequacy was observed in females compared to males while individuals who were vitamin D deficient or insufficient were significantly more likely to be osteoporotic than those who were sufficient (>SO nmol/l). These data provide additional evidence to support the recent 10M recommendation of a 2S(OH)D concentration of 50 nmol/l for optimal bone health. In a third study, the association between vitamin D status, immune markers of inflammation and the ratio of pro: anti-cytokines was investigated in a sub-sample of TUDA participants (n 998). Vitamin D was significantly correlated with pro-inflammatory markers and a 25(OH)D concentration >75nmol was associated with an improved inflammatory (profile as determined by the pro:anti cytokine ratio) compared to individuals with a 25(OH)D status <25 or 25-75nmol/l. In a fourth study, vitamin D status was assessed within a sample (n 260) of pregnant women from a sunny equatorial country (5degS) (Seychelles). Maternal vitamin D status was observed to be >75nmol/l through all sample periods of pregnancy and was significantly associated with higher birth weight and length with no apparent upper limit of effect. These results demonstrate the importance of optimal vitamin D status during pregnancy and the need for adequate dietary recommendations in order to achieve this level within far latitude populations that are exposed to low UVB sun light. In conclusion, the results within the current thesis suggest concentrations of vitamin D greater than recently recommended cut-offs for bone health (50nmol/l) are associated with extra-skeletal health benefits. Furthermore, consideration needs to be given to the current vitamin D dietary recommendations within the UK and Ireland in order to address the high level of deficiency observed in the older adult population and to achieve the optimal vitamin D concentration in terms of benefits for bone health, immune function and neonatal health outcomes for the whole population

6

KELLY, MICHAEL ALAN. "CHARACTERIZATION OF RECEPTORS AND BINDING PROTEINS FOR THE ACTIVE METABOLITES OF VITAMINS A AND D IN NORMAL AND RESISTANT CELLS (PRIMATE RESEARCH)". Diss., The University of Arizona, 1986. http://hdl.handle.net/10150/183919.

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Involvement of Cellular Retinoic Acid (CRABP) or Retinol (CRBP) Binding Proteins and 1,25-dihydroxyvitamin D₃ (1,25(OH)₂D₃) receptors in the response of cultured cells to retinoic acid and 1,25(OH)₂D₃ was examined. A new method for saturation analysis of CRABP and CRBP was applied to human tumors, human neuroblastoma cells, which retinoic acid causes to differentiate, and a bioselected subline resistant to retinoic acid. These data suggest that CRABP may not mediate cell differentiation by retinoic acid. In other studies, 1,25(OH)₂D₃ receptors and bioresponses were characterized in cultured primate cells. Rhesus monkey kidney cells (LLC-MK₂) were resistant to 1,25(OH)₂D₃-dependent induction of 25(OH)D-24-hydroxylase enzyme. The ED₅₀ in LLC-MK₂ cells was 10-100 fold higher than in other cultured cells. This resistance resulted from a low affinity receptor. Since the LLC-MK₂ variant receptor did not differ in size from the wild type rhesus 1,25(OH)₂D₃ receptor, (Mᵣ = 52 kDa) a subtle alteration in the receptor likely caused the decreased ligand affinity. Also of interest was the possible cellular resistance to 1,25(OH)₂D₃, in the owl monkey (Aotus trivurgatus), which generally occurs in new world primates. Owl monkey kidney (OMK) cells had the same content of receptors for 1,25(OH)₂D₃ and sensitivity to this hormone as cells from the rhesus monkey (old world primate). The ED₅₀ for induction of 24hydroxylase was 2-3 nM in both the OMK cells and the rhesus monkey fibroblasts. Both cells contained 2300 high affinity receptor molecules per cell, which bound DNA and were characterized by immunoblot as 52 kDa proteins. 1,25(OH)₂D₃ treatment increased the content of 1,25(OH)₂D₃ receptors in OMK cells, by increasing the synthesis of receptor mRNA. These data indicate the owl monkey is not resistant to 1,25(OH)₂D₃, unlike other new world primates. This finding was confirmed independently by demonstration that the owl monkey maintained mean serum 1,25(OH)₂D₃ levels (29 pg/ml) in the range of old world primates (33 pg/ml) and humans, in contrast to the elevated 1,25(OH)₂D₃ in other new world primates (97-129 pg/ml). This result suggests the alteration of 1,25(OH)₂D₃-endocrine dynamics in new world primates occurred subsequent to the evolutionary divergence of the owl monkey.

7

Osman, O. M. "Clinical and experimental studies on vitamin D and PTH metabolism". Thesis, University of Newcastle Upon Tyne, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.382572.

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8

Anderson, Paul Hamill. "The regulation of Vitamin D metabolism in the kidney and bone". Title page, contents and abstract only, 2002. http://web4.library.adelaide.edu.au/theses/09PH/09pha5486.pdf.

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Includes bibliographical references (leaves 226-273.) Investigates the regulation of the expression of CYP27B1, CYP24 and vitamin D receptor (VDR) mRNA, both in the bone and in the kidney, with the aim to determine whether the regulation of the vitamin D metabolism in the bone is independent from that in the kidney. The effects of age, dietary calcium and vitamin D status on the expression of these genes in both the kidney and the bone, as well as on a number of biochemical factors known to regulate the renal metabolism of 1,25D, such as PTH, calcium and 1,25D itself, were examined. CYP27B1 mRNA expression was also studied in histological sections of rat femoral bone.

9

Bashir, S. E. O. "Studies of vitamin-D and mineral metabolism in health and disease". Thesis, University of Newcastle Upon Tyne, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.375130.

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10

Kusudo, Tatsuya. "Structure-function analysis of cytochromes P450 involved in vitamin D metabolism". Kyoto University, 2004. http://hdl.handle.net/2433/147749.

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Kyoto University (京都大学)
0048
新制・課程博士
博士(農学)
甲第10900号
農博第1406号
新制||農||890(附属図書館)
学位論文||H16||N3911(農学部図書室)
UT51-2004-G747
京都大学大学院農学研究科食品生物科学専攻
(主査)教授井上國世, 教授大東肇, 教授伏木亨
学位規則第4条第1項該当

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Livros sobre o assunto "Vitamin D Metabolism":

1

Robertson, R. Paul, ed. Translational Endocrinology & Metabolism: Vitamin D Update. 8401 Connecticut Avenue, Suite 900, Chevy Chase, Maryland 20815: The Endocrine Society, 2011. http://dx.doi.org/10.1210/team.9781879225848.

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2

Dambacher, M. A. Osteoporosis and active vitamin D metabolites: The shape of things to come. Basel: EULAR Publishers, 1996.

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3

Workshop on Vitamin D (12th 2003 Maastricht, Netherlands). Vitamin D: Proceedings of the 12th Workshop on Vitamin D : July 6-10th, 2003, Maastricht, The Netherlands. Amsterdam: Elsevier, 2004.

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4

Bauman, V. K. Biokhimii͡a︡ i fiziologii͡a︡ vitamina D. Riga: "Zinatne", 1989.

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5

Workshop on Vitamin D (6th 1985 Merano, Italy). Vitamin D: Chemical, biochemical, and clinical update : proceedings of the Sixth Workshop on Vitamin D, Merano, Italy, March 1985. Editado por Norman A. W. 1938-. Berlin: De Gruyter, 1985.

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6

1947-, Kragballe Knud, ed. Vitamin D in dermatology. New York: Marcel Dekker, 2000.

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7

Haas, Jochen. Vigantol: Adolf Windaus und die Geschichte des Vitamin D. Stuttgart: WVG, Wissenschaftliche Verlagsgesellschaft, 2007.

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8

Razzaque, Mohammed S. Phosphate and vitamin D in chronic kidney disease. Basel: Karger, 2013.

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9

Cannell, John. The athlete's edge: Quicker, stronger, faster with Vitamin D. [San Dimas, Calif.]: Here & Now Books, 2011.

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10

Wagner, Carol L. New insights into vitamin D during pregnancy, lactation, & early infancy. Amarillo, TX: Hale Pub., 2010.

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Capítulos de livros sobre o assunto "Vitamin D Metabolism":

1

Demay, Marie B. "Inherited Defects of Vitamin D Metabolism". In Vitamin D, 679–89. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_36.

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2

Eisman, J. A. "Vitamin D Metabolism". In Physiology and Pharmacology of Bone, 333–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-77991-6_10.

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3

Plum, Lori A., e Hector F. DeLuca. "The Functional Metabolism and Molecular Biology of Vitamin D Action". In Vitamin D, 61–97. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_3.

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4

Jones, Glenville. "Metabolism and Catabolism of Vitamin D, Its Metabolites and Clinically Relevant Analogs". In Vitamin D, 99–134. Totowa, NJ: Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60327-303-9_4.

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5

Dawson-Hughes, Bess. "Calcium and Vitamin D". In Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 403–7. Ames, USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118453926.ch47.

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6

Lips, Paul, Natasja M. van Schoor e Nathalie Bravenboer. "Vitamin D-Related Disorders". In Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 613–23. Ames, USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118453926.ch75.

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7

Zerwekh, Joseph E. "Vitamin D Metabolism and Stones". In Urinary Tract Stone Disease, 169–79. London: Springer London, 2010. http://dx.doi.org/10.1007/978-1-84800-362-0_13.

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8

Cheung, Florence S. G., e Juergen K. V. Reichardt. "Molecular Biology of Vitamin D Metabolism and Skin Cancer". In Vitamin D and Cancer, 191–219. New York, NY: Springer New York, 2010. http://dx.doi.org/10.1007/978-1-4419-7188-3_9.

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9

Gallagher, J. Christopher. "Vitamin D Insufficiency and Deficiency". In Primer on the Metabolic Bone Diseases and Disorders of Mineral Metabolism, 624–31. Ames, USA: John Wiley & Sons, Inc., 2013. http://dx.doi.org/10.1002/9781118453926.ch76.

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10

Fanconi, G. "Variations in Sensitivity to Vitamin D: From Vitamin D Resistant Rickets, Vitamin D Avitaminotic Rickets and Hypervitaminosis D to Idiopathic Hypercalcaemia". In Ciba Foundation Symposium - Bone Structure and Metabolism, 187–205. Chichester, UK: John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470715222.ch15.

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Trabalhos de conferências sobre o assunto "Vitamin D Metabolism":

1

Hansdottir, Sif, Martha M. Monick, Nina Lovan, Linda S. Powers e Gary W. Hunninghake. "Smoking Disrupts Vitamin D Metabolism In The Lungs". In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1425.

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2

Yarshevich, A. V., e P. M. Marozik. "ANALYSIS OF ASSOCIATION OF VDR GENE VARIANTS WITH SERUM VITAMIN D LEVEL IN PATIENTS WITH BONE-MUSCULAR DISEASE". In SAKHAROV READINGS 2021: ENVIRONMENTAL PROBLEMS OF THE XXI CENTURY. International Sakharov Environmental Institute of Belarusian State University, 2021. http://dx.doi.org/10.46646/sakh-2021-2-146-149.

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Currently, the pathology of the musculoskeletal system is considered in several multifactorial diseases, the pathogenesis of which is complex and is due to the interaction of environmental and endogenous factors. An important role in the progression of pathology is played by disorders in metabolism and a decrease in sensitivity to vitamin D. Studies of the past two decades have shown that the various biological actions of the active metabolite of vitamin D - 1,25-dihydroxy vitamin D (calcitriol) - are carried out by modulating the expression of genes that are mediated by interaction with the intracellular vitamin D receptor (VDR). VDR is a product of the corresponding gene - VDR, which determines its structure and functional activity. In this gene, a certain number of polymorphic variants have been identified that can affect gene expression.

3

Ahearn, Thomas, Marjorie L. McCullough, W. Dana Flanders, Qi Long, Jessica Fung, Anita Ofori-Addo, Shuva Dawadi e Roberd M. Bostick. "Abstract 2890: Effects of calcium and vitamin D on markers of vitamin D metabolism in normal colon mucosa: A randomized clinical trial". In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-2890.

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4

Schrumpf, Jasmijn A., Dennis K. Ninaber, Anne M. Van Der Does e Pieter S. Hiemstra. "TGF-ß1 affects epithelial vitamin D metabolism and expression of host defence peptides". In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4251.

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5

Shulaev, G. M., e R. K. Milushev. "IMPROVMENT THE BIOLOGICAL VALUE OF PORK". In INNOVATIVE TECHNOLOGIES IN SCIENCE AND EDUCATION. DSTU-Print, 2020. http://dx.doi.org/10.23947/itno.2020.467-471.

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The purpose - improve the quality of pig farming production through the use of functional feed additive at the final stage of feeding. The additive ingredients (in %): vitamin E - 0,32; vitamin D₃ - 0,30; vitamin C - 0,30; vitamin B₄ - 1,56; vitamin B₁₂ - 0,004; betaine - 10,00; %; bentonite - 77,166; Omek J – 0,15%; selen - 0,2 %; soya flour (filling substance) - 10,00. Components activity: vitamin E-50,0 of %; D₃ - 0,15,0 thousand ME in 1g; B₄ - 60,0 %; B₁₂ - 1,0%; Omek J - 2,0%; seleno-KI - 0,2 %. The functional feed additive is approved on fattened pigs of large white breed. Animals from control group received the mixed fodder constantly used in a factory, skilled - same as well as control, but the containing 1 % of the additive within 10 days before slaughter. Growth, some indicators of a metabolism, quality of meat are studied.

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Pillai, Dinesh K., Sabah F. Iqbal, Angela S. Benton, Jenifer Lerner, Andrew Wiles, Matthew Foerster, Tugba Ozedirne, Henry Holbrook, Stephen J. Teach e Robert J. Freishtat. "Variants In Genes Responsible For Vitamin D Metabolism Influence Asthma Phenotypes In African American Children". In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1308.

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Kraus, A., M. Penna-Martinez, G. Meyer e K. Badenhoop. "Impaired Vitamin D metabolism with low IL-6 and CCL-2 responsiveness to in-vitro Vitamin D treatment in autoimmune polyglandular syndrome type 2 (APS-2)". In Diabetes Kongress 2018 – 53. Jahrestagung der DDG. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1641941.

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Tavera, Luz E., Thomas Westerling e Myles Brown. "Abstract 3562: Genome-wide analysis of the vitamin D receptor (VDR) binding sites reveals vitamin D role modulating autophagy and metabolism in luminal-like breast cancer cells." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-3562.

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Suetani, Rachel J., Kristen Ho, Shalini Jindal, Paul M. Neilsen, Grantley Gill, Howard A. Morris e David F. Callen. "Abstract 5159: Investigation of local vitamin D metabolism in breast cancer using anex vivotissue explant system". In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-5159.

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Dai, Qi, Xiangzhu Zhu, JoAnn E. Manson, Yiqing Song, Xingnan Li, Adrian Franke, Rebecca B. Costello et al. "Abstract CT093: Bimodal relationship between magnesium supplementation and vitamin D status and metabolism: Results from a randomized trial". In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-ct093.

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Relatórios de organizações sobre o assunto "Vitamin D Metabolism":

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Hassanein, Mohammed, Hasniza Huri e Abduelmula R. Abduelkarem. Determinants of serum vitamin D and its metabolites and the reflection on vitamin D status in postmenopausal women: A systematic review. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, junho de 2022. http://dx.doi.org/10.37766/inplasy2022.6.0116.

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Review question / Objective: What are the factors that affect vitamin D metabolism and status in post-menopausal women? Condition being studied: Menopause: Menopause is defined as permanent cessation of menstruation resulting from loss of ovarian follicular activity. The occurrence of the last menstruation can only be diagnosed retrospectively and is usually taken as being final if it is followed by a 12-month bleed-free interval; such women are defined as being post-menopausal. Information sources: MEDLINE (by PubMed), Embase (by OvidSP), Cochrane Central Register of Controlled Trials (CENTRAL), Google Scholar, Web of Science Core Collection, ClinicalTrials.gov, ISRCTN registry, EU Clinical Trials Register.

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Chen, HongPeng, e YuQian Zhao. Effects of vitamin D supplementation on body composition, glucose metabolism, and inflammation in obese or overweight patients: a systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, março de 2022. http://dx.doi.org/10.37766/inplasy2022.3.0152.

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