Literatura científica selecionada sobre o tema "Skin dysbiosis"
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Artigos de revistas sobre o assunto "Skin dysbiosis"
Scharschmidt, Tiffany C. "Skin Dysbiosis Goes “Off-Leish”". Cell Host & Microbe 22, n.º 1 (julho de 2017): 1–3. http://dx.doi.org/10.1016/j.chom.2017.06.017.
Texto completo da fonteTeng, Vannia C., e Prima K. Esti. "Skin microbiome dysbiosis in leprosy cases". International Journal of Research in Dermatology 7, n.º 5 (23 de agosto de 2021): 741. http://dx.doi.org/10.18203/issn.2455-4529.intjresdermatol20213355.
Texto completo da fonteMarson, Justin, Stefano Berto, Paul Mouser e Hilary Baldwin. "Association between Rosacea, Environmental Factors, and Facial Cutaneous Dysbiosis". SKIN The Journal of Cutaneous Medicine 5, n.º 5 (13 de setembro de 2021): 487–95. http://dx.doi.org/10.25251/skin.5.5.6.
Texto completo da fonteMuharram, Luthfia Hastiani, Fauzia Ningrum Syaputri, Wulan Pertiwi e Rizki Fika Saputri. "Aktivitas Antibakteri Ekstrak Bawang Hitam Variasi Waktu Aging Terhadap Pencegahan Dysbiosis Kulit Penyebab Jerawat". Jurnal Sains dan Kesehatan 4, n.º 2 (30 de abril de 2022): 181–88. http://dx.doi.org/10.25026/jsk.v4i2.1035.
Texto completo da fontePessôa, Rodrigo, Patricia Bianca Clissa e Sabri Saeed Sanabani. "The Interaction between the Host Genome, Epigenome, and the Gut–Skin Axis Microbiome in Atopic Dermatitis". International Journal of Molecular Sciences 24, n.º 18 (20 de setembro de 2023): 14322. http://dx.doi.org/10.3390/ijms241814322.
Texto completo da fonteDe Pessemier, Britta, Lynda Grine, Melanie Debaere, Aglaya Maes, Bernhard Paetzold e Chris Callewaert. "Gut–Skin Axis: Current Knowledge of the Interrelationship between Microbial Dysbiosis and Skin Conditions". Microorganisms 9, n.º 2 (11 de fevereiro de 2021): 353. http://dx.doi.org/10.3390/microorganisms9020353.
Texto completo da fonteTao, Rong, Ruoyu Li e Ruojun Wang. "Dysbiosis of skin mycobiome in atopic dermatitis". Mycoses 65, n.º 3 (3 de dezembro de 2021): 285–93. http://dx.doi.org/10.1111/myc.13402.
Texto completo da fonteIto, T., R. Aoyama, S. Nakagawa, Y. Yamazaki, N. Inohara, Y. Ichikawa, N. Shimojo, Y. Matsuoka-Nakamura e M. Fujimoto. "955 Skin care improves newborn skin dysbiosis associated with atopic dermatitis". Journal of Investigative Dermatology 143, n.º 5 (maio de 2023): S164. http://dx.doi.org/10.1016/j.jid.2023.03.966.
Texto completo da fonteMagnifico, Irene, Angelica Perna, Marco Alfio Cutuli, Alessando Medoro, Laura Pietrangelo, Antonio Guarnieri, Emanuele Foderà et al. "A Wall Fragment of Cutibacterium acnes Preserves Junctional Integrity Altered by Staphylococcus aureus in an Ex Vivo Porcine Skin Model". Pharmaceutics 15, n.º 4 (12 de abril de 2023): 1224. http://dx.doi.org/10.3390/pharmaceutics15041224.
Texto completo da fonteBlicharz, Leszek, Lidia Rudnicka, Joanna Czuwara, Anna Waśkiel-Burnat, Mohamad Goldust, Małgorzata Olszewska e Zbigniew Samochocki. "The Influence of Microbiome Dysbiosis and Bacterial Biofilms on Epidermal Barrier Function in Atopic Dermatitis—An Update". International Journal of Molecular Sciences 22, n.º 16 (5 de agosto de 2021): 8403. http://dx.doi.org/10.3390/ijms22168403.
Texto completo da fonteTeses / dissertações sobre o assunto "Skin dysbiosis"
Lamiaux, Marie. "ImoHS : Etude de l'immunomodulation de la réponse immunitaire dans l'Hidradénite suppurée et Evaluation in vitro et ex vivo d'une nouvelle stratégie thérapeutique". Electronic Thesis or Diss., Université de Lille (2022-....), 2023. http://www.theses.fr/2023ULILS116.
Texto completo da fonteHidradenitis suppurativa (HS) is a chronic, debilitating and suppurative dermatosis affecting the folds. Its pathophysiology remains poorly understood, and only surgery is curative, although it can cause significant tissue damage. Studies are limited by the absence of animal models of the disease, its heterogeneity and multifactorial nature. Several studies have demonstrated the role of cutaneous dysbiosis in the chronic inflammation of the disease, which may be caused by altered secretion of antimicrobial peptides (AMPs) linked to keratinocyte dysfunction. Photodynamic therapy (PDT) has been shown to be a rational treatment for inflammatory dermatoses. However, the low penetrance of the photosensitizer (PS) limits its therapeutic action and therefore its relevance in HS due to deep fistulous tracts.The first part of the results allowed us to characterize our in vitro model of the immortalized human keratinocyte line HaCaT, mutated on the Nicastrin gene (HS model), where we observe disturbances in morphological and protein keratinocyte differentiation compared with the wild-type line. Secondly, we assessed the secretome of our culture media, where we observed the presence of pro-inflammatory cytokines of the TH1 and TH17 pathways (IL-1β, IL-17a, IL-23 and IFN-γ) as well as a defect in IL-10 secretion in our KO control model compared with the wild-type line, which could be involved in the imbalance of the inflammatory response. Furthermore, we show a change in the expression profile of keratinocyte differentiation markers, in our in vitro HS model, exposed to induced inflammation (TNF-α + IL-6 + LPS), which resembles a basal layer keratinocyte profile, characterized by high proliferative capacity and prolonged cell survival. These properties could be the cause of the epidermal acanthosis described in HS, which is responsible for the follicular occlusion and hypertrophic scarring characteristic of the disease. Based on wounding tests, we have observed that our HaCaT KO line has a better wound healing capacity than the WT line, and that induction of differentiation enhances this capacity. The characteristics displayed by our HaCaT KO line are like a state of keratinocyte activation described during the healing process, where keratinocytes follow an alternative epithelial differentiation pathway enabling them to acquire migration and proliferation capacities.Secondly, we tested the effect of PDT on our in vitro model. We observed greater efficacy and selectivity of methylene blue PDT on our HaCaT KO line, compared with 5-ALA PDT. At the same time, we obtained the authorization of the French Committee for the Protection of Individuals to conduct the ImoHS clinical study, to evaluate the local and systemic immune response, using blood samples and skin explants from HS patients. In this context, we have developed a new PDT illumination device for the treatment of human skin explants (ex vivo model). We also developed and validated our analysis strategy for studying innate lymphoid cell subgroups by flow cytometry from healthy donor blood.The thesis project enabled us to better characterize the in vitro HS model and the impact of differentiation and inflammation on HaCaT KOs. We also demonstrated the efficacy of methylene blue PDT on our in vitro HS model. The authorization obtained to set up the ImoHS clinical trial will enable us, through its translational aspect, to validate our results obtained on an ex vivo model
Janvier, Xavier. "Etude de l'effet d'un polluant atmosphérique (NO2) sur le microbiote cutané Dialog between skin and its microbiota : Emergence of "Cutaneous bacterial endocrinology" Deleterious effects of an air pollutant on a selection of commensal skin bacterial strains, potential contributor to dysbiosis Response of a commensal skin bacterium to nitrogen oxides (NOx), air pollutants : potential tools for testing anti-pollution active cosmetic ingredient effectiveness Draft genome sequence of the commensal strain Corynebacterium tuberculostearicum CIP 102622 isolated from human skin Draft genome sequences of four commensal strains of Staphylococcus and Pseudomonas isolated from healthy human skin". Thesis, Normandie, 2021. http://www.theses.fr/2021NORMR007.
Texto completo da fonteNitrogen dioxide (NO2), as the second most deadly air pollutant in Europe, is one of the most of concern for human health according to the European Environment Agency. It is notably known to be responsible for cardiovascular and respiratory diseases and also contributes to skin aging and atopic dermatitis. Host endogenous factors such as the cutaneous microbiota are also involved in this pathology, which is common in urban and suburban areas. Indeed, many skin pathologies are correlated to an imbalance (dysbiosis) of the bacterial microbiota, an essential player in the preservation of skin homeostasis. However, it is strongly presumed that the effect of pollutants on the skin involves direct mechanisms of action but also an indirect mechanism linked to the alteration of the cutaneous microbiota by the pollutant. Consequently, it is relevant to address the effect of gaseous NO2 (gNO2) on the cutaneous microbiota. This thesis aims to assess the physiological, morphological and molecular impact of gNO2 on commensal bacterial strains of representative species of the cutaneous microbiota (Staphylococcus aureus, Staphylococcus epidermidis, Staphylococcus capitis, Pseudomonas fluorescens, Corynebacterium tuberculostearicum). Depending on the species, different responses to gNO2-generated nitrosative stress were thus highlighted as well as a higher tolerance to gNO2 for some of them. This work therefore suggests that gNO2 could contribute to the formation of a dysbiotic state of the cutaneous microbiota and participate in the pollutant indirect action on the skin
Capítulos de livros sobre o assunto "Skin dysbiosis"
Amyes, Sebastian G. B. "3. Microbiota and microbiome in humans". In Bacteria: A Very Short Introduction, 14–21. Oxford University Press, 2022. http://dx.doi.org/10.1093/actrade/9780192895240.003.0003.
Texto completo da fonteTungland, Bryan. "The Gut-Brain-Skin Axis and Role of Intestinal Dysbiosis in Acne Vulgaris: Therapeutic Effects of Pro- and Prebiotic Use". In Human Microbiota in Health and Disease, 595–604. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-12-814649-1.00013-2.
Texto completo da fonte