Literatura científica selecionada sobre o tema "Promotor <Genetik>"
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Artigos de revistas sobre o assunto "Promotor <Genetik>"
Oktariana, Desi, Irsan Saleh, Zen Hafy e Iche Andriyani Liberty. "Peran Polimorfisme Promotor Gen Interleukin-10 pada Penyakit Kusta: Tinjauan Sistematik". Journal Of The Indonesian Medical Association 73, n.º 1 (5 de maio de 2023): 15–24. http://dx.doi.org/10.47830/jinma-vol.73.1-2023-816.
Texto completo da fonteWurlina, Wurlina, Rimayanti Rimayanti, Mas'ud Hariadi e Dewa Ketut Meles. "PEMBIBITAN DAN PENGGEMUKAN KAMBING “LOKETAWA” PENGHASIL DAGING DAN SUSU RAKITAN TEKNOBREEDING DAN TEKNOFATTENING PAKAN TANPA HIJAUAN (COMPLETE FEED)". Jurnal Layanan Masyarakat (Journal of Public Services) 1, n.º 1 (1 de junho de 2017): 46. http://dx.doi.org/10.20473/jlm.v1i1.2017.46-50.
Texto completo da fonteLi, Jinyang, Sheng Tong, Farrukh Raza Amin, Habiba Khalid, Kai Chen, Xiaoguang Zhao, Jinling Cai e Demao Li. "Enhancing the Activity of a Self-Inducible Promoter in Escherichia coli through Saturation Mutation and High-Throughput Screening". Fermentation 9, n.º 5 (13 de maio de 2023): 468. http://dx.doi.org/10.3390/fermentation9050468.
Texto completo da fonteShujaat, Muhammad, Abdul Wahab, Hilal Tayara e Kil To Chong. "pcPromoter-CNN: A CNN-Based Prediction and Classification of Promoters". Genes 11, n.º 12 (21 de dezembro de 2020): 1529. http://dx.doi.org/10.3390/genes11121529.
Texto completo da fonteMahoney, Michael E., e Daniel L. Wulff. "Mutations that Improve the pRE Promoter of Coliphage Lambda". Genetics 115, n.º 4 (1 de abril de 1987): 591–95. http://dx.doi.org/10.1093/genetics/115.4.591.
Texto completo da fonteGeorge, Janet A., e Mary-Lou Pardue. "The Promoter of the Heterochromatic Drosophila Telomeric Retrotransposon, HeT-A, Is Active When Moved Into Euchromatic Locations". Genetics 163, n.º 2 (1 de fevereiro de 2003): 625–35. http://dx.doi.org/10.1093/genetics/163.2.625.
Texto completo da fonteVoelkel-Meiman, K., e G. S. Roeder. "Gene conversion tracts stimulated by HOT1-promoted transcription are long and continuous." Genetics 126, n.º 4 (1 de dezembro de 1990): 851–67. http://dx.doi.org/10.1093/genetics/126.4.851.
Texto completo da fonteDavey, James A., e Corey J. Wilson. "Engineered signal-coupled inducible promoters: measuring the apparent RNA-polymerase resource budget". Nucleic Acids Research 48, n.º 17 (5 de setembro de 2020): 9995–10012. http://dx.doi.org/10.1093/nar/gkaa734.
Texto completo da fonteFandl, J. P., L. K. Thorner e S. W. Artz. "Mutations that affect transcription and cyclic AMP-CRP regulation of the adenylate cyclase gene (cya) of Salmonella typhimurium." Genetics 125, n.º 4 (1 de agosto de 1990): 719–27. http://dx.doi.org/10.1093/genetics/125.4.719.
Texto completo da fonteGreener, A., S. M. Lehman e D. R. Helinski. "Promoters of the broad host range plasmid RK2: analysis of transcription (initiation) in five species of gram-negative bacteria." Genetics 130, n.º 1 (1 de janeiro de 1992): 27–36. http://dx.doi.org/10.1093/genetics/130.1.27.
Texto completo da fonteTeses / dissertações sobre o assunto "Promotor <Genetik>"
Quehl, Eike. "Etablierung transgener Zelllinien zur Visualisierung der Aktivität des Doublecortin-Promotors als Modell der Neurogenese in vitro". kostenfrei, 2008. http://www.opus-bayern.de/uni-regensburg/volltexte/2009/1149/.
Texto completo da fonteHohenstatt, Antonia. "Hypoxia inducible factor 1 (HIF 1) alpha- und beta-vermittelte Induktion der ABCA1-Promotor-Aktivität". kostenfrei, 2009. http://www.opus-bayern.de/uni-regensburg/volltexte/2009/1306/.
Texto completo da fonteSommer, Heide. "Der Übergang von der Latenz zur lytischen Replikation des Epstein-Barr-Virus vergleichende Analysen zur Bedeutung regulatorischer HI-Motive im Promotor des viralen Gens BZLF-1 /". [S.l.] : [s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=962765007.
Texto completo da fonteSigvardsson, Mikael. "Regulation of immunoglobulin transcription during B-cell differentiation". Lund : Lund University, 1995. http://books.google.com/books?id=TJNqAAAAMAAJ.
Texto completo da fonteEzpeleta, Jessica. "The characterization of the ABF-1 promoter". Scholarly Commons, 2001. https://scholarlycommons.pacific.edu/uop_etds/559.
Texto completo da fonteConradie, E. C. (Elizabeth Cornelia). "Promotor engineering in Saccharomyces cerevisiae for transcriptional control under different physiological conditions". Thesis, Stellenbosch : University of Stellenbosch, 2011. http://hdl.handle.net/10019.1/16512.
Texto completo da fonteENGLISH ABSTRACT: To manipulate recombinant microorganisms for industrial processes, controllable genetic systems are needed that can coordinate expression of recombinant metabolic pathways. All components are sensitive to change and thus putative targets for modification and genetic elements and regulatory systems need to be understood and determined. Central in gene regulation is the transcription activators that mediate gene transcription mechanisms by binding to promoters in response to environmental signals. Promoter engineering entails the modification of transcription factors and their target promoters. In this study, a metabolic control system in Saccharomyces cerevisiae was constructed that would allow induction in response to physiological environment, specifically hypoxia and low temperature conditions. Two approaches were undertaken to find such a system. Firstly, a bi-directional reporter gene cloning vector was designed to search for novel hypoxiainducible promoters. Secondly, a transcription regulatory circuit was built, consisting of an inducible transcription regulator and promoter with a reporter gene through which it mediates transcription. Advantage was taken of the modular nature of proteins and functional domains originating from different transcriptional proteins were combined. A search for promoter elements sensitive to hypoxia from a S. cerevisiae genomic DNA (gDNA) library, using a bi-directional cloning vector, did not yield highly inducible promoters. It was concluded that a multitude of signals overlap, rendering genetic induction difficult to control. A synthetic regulatory system would minimize the impact of these multiple interactions. Such a genetic circuit was constructed, consisting of a chimeric transcription activator and a target fusion promoter. The chimeric transcription activator consisted of the GAL4 DNA binding domain, ADR1 TADIII transactivation domain and three domains of the MGA2 regulatory protein. The functional domains of Mga2p responsible for unregulated expression (at high basal levels) under both aerobic and hypoxia conditions were located, as well as a further upregulation under low temperature, and were mapped to the Nterminal and mid-Mga2p regions. A target fusion promoter consisting of a partial GAL10/1 promoter sequence and a Trichoderma reesei core xyn2 promoter were constructed as target for this chimeric transactivator. This synthetic promoter was fused to the T. reesei xyn2 open reading frame encoding for a readily assayable β-xylanase activity. Both the chimeric transactivator and fusion promoter-reporter gene cassettes were expressed from the same episomal plasmid, named pAR. Transformed into S. cerevisiae Y294, this regulatory system induced transcription under aerobic and hypoxia conditions. Furthermore, the reporter gene expression was upregulated by the chimeric transactivator at low temperatures. The chimeric transactivator mediated a seven-fold induction of the reporter gene under aerobic conditions in S. cerevisiae Y294 when transformed with plasmid AR. A two- to three-fold induction at 23ºC was reported under anaerobic conditions, relative to a reference strain expressing a transcription activator without the Mga2p domains. At 30ºC, a two- to three-fold induction under aerobic conditions and similar induction under oxygen-limited conditions were observed. Replacing the reporter gene with your favorite gene (for example a recombinant enzyme) and incorporating such a pAR system into a recombinant yeast should induce expression of the chosen gene under low temperatures, both aerobic and anaerobically (thus creating a controllable system). The system also has wider application in identifying other transcription factors’ signal-sensitive domains. The design of this system provides the ability to add a linker to a transactivator and to either create specific signal sensitivity or relieve the regulator of its signal dependence. It creates an easy system for assessing other transactivators and their domains with unknown functions and thus provides a ”workhorse and prospector in one”.
AFRIKAANSE OPSOMMING: Vir die manipulering van rekombinante mikroörganismes vir industriële prosesse word beheerbare genetiese stelsels benodig om gekoördineerde uitdrukking van rekombinante metaboliese weë teweeg te bring. Alle komponente van sulke stelsels is sensitief vir verandering en genetiese elemente en reguleerbare sisteme moet dus deeglik verstaan of bepaal word. Sentraal tot geenregulering is die transkripsie-aktiveerders wat geentranskripsie beheer deur aan promoters te bind in reaksie op eksterne omgewingsfaktore. Promotoringenieurswese behels wysigings van transkripsiefaktore en hul teikenpromotors. In hierdie studie is 'n genetiese beheerstelsel vir Saccaromyces cerevisiae ontwikkel wat induksie in reaksie tot spesifieke fisiologiese omgewingreaksies, naamlik hipoksie- en lae temperatuur, toelaat. Twee benaderings is gevolg: eerstens is ‘n tweerigting verklikker-geen vektor ontwikkel en gebruik om vir unieke induseerbare hipoksie-promoters te soek. Tweedens is ‘n transkripsie reguleringstelsel gebou wat uit ‘n induseerbare transkripsiereguleerder and promotor met ‘n verklikkergeen bestaan, waardeur transkripsie bemiddel kan word. Hierdie benadering benut die modulêre onderbou van proteïene en funksionele domeine afkomstig vanaf verskillende transkripsiefaktore is gekombineer. 'n Soektog na hipoksie-sensitiewe promotors vanuit 'n Saccharomyces cerevisiae-genoom- DNA (gDNA), deur van ‘n tweerigting verklikker-vektor gebruik te maak, het ongelukkig nie hoogs-induseerbare promotors opgelewer nie. Die gevolgtrekking was dat ‘n veelvoud van seine met mekaar oorvleuel en die beheer van genetiese induksie dus bemoeilik. Die ontwikkeling van ‘n sintetiese regulering-sisteem kan die impak van die veelvuldige interaksies verminder. Vir dié doel is ‘n sintetiese reguleringstelsel ontwerp, bestaande uit ‘n chimeriese transkripsie-aktiveerder met ‘n teiken fusie-promotor. Die chimeriese transaktiveerder bestaan uit die GAL4 DNA bindingsdomein, die ADR1 TAD III transaktiveringsdomein en drie domeine van die Mga2 reguleringsproteïen. In die studie is die funksionele domeins van Mga2p betrokke by lae temperatuur-respons en ongereguleerde uitdrukking (teen hoë basale vlakke) onder beide aërobiese en anaërobiese toestande aangedui en is tot die N-terminaal en middel-Mga2p areas gekarteer. ‘n Teiken-fusie-promoter, bestaande uit 'n gedeeltelike GAL1/10 DNA promotoropeenvolging en ‘n Trichoderma reesei kern xyn2-promoter, is as teiken vir hierdie chimeriese transaktiveerder saamgestel. Hierdie sintetiese promotor is aan die T. reesei xyn2 oopleesraam, wat vir ‘n maklik meetbare β-xylanase aktiwiteit kodeer, gekoppel. Beide die chimeriese transaktiveerder and fusie-promoter-verklikker-geenkaset word vanaf dieselfde episomale plasmied, bekend as pAR, uitgedruk. Hierdie reguleringsisteem induseer transkripsie onder aërobiese en hipoksie toestande in S. cerevisiae Y294. Verder word die verklikkergeen se uitdrukking deur die chimeriese transaktiveerder by lae temperature verhoog. Die chimeriese transaktiveerder induseer ‘n sewe-voudige induksie van die verklikkergeen onder aërobiese toestande by 23ºC vanaf die pAR-stelsel in S. cerevisiae Y294. ‘n Twee- tot drie-voudige induksie teen 23ºC is onder hipoksie toestande gevind, relatief tot induksievlakke van ‘n verwysingstam met ‘n transaktiveerder sonder die Mga2 domeine. By 30ºC is ‘n twee- tot drie-voudige induksie onder aërobiese en lae suurstofvlakke waargeneem. Deur die verklikker geen met ‘n jou-gunsteling-geen te vervang (bv. ‘n rekombinante ensiem) en so 'n pAR-sisteem in ‘n rekombinante gis te inkorporeer, word uitdrukking onder lae temperature onder beide aërobiese- en anaërobiese toestande geïnduseer (en sodoende word ‘n reguleerbare sisteem geskep). Die sisteem het wyer toepassing om sein-sensitiewe domeine van ander transkripsiefaktore te identifiseer. Die ontwerp van die stelsel maak dit moontlik om 'n skakel tot die transaktiveerder by te voeg wat óf sensitiwiteit tot 'n spesifieke sein skep, óf die reguleerder vanaf seinafhanklikheid verlos. So word ‘n bruikbare stelsel vir die bestudering van ander transaktivators en hul domeine met onbekende funksie geskep – ‘n “werksesel en prospekteerder in een”.
Grade, Carla Vermeulen Carvalho 1983. "Caracterização funcional do promotor gênico da miostatina". [s.n.], 2013. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317671.
Texto completo da fonteTese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia
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Resumo: A Miostatina é um regulador negativo da deposição de musculatura esquelética e mutações no gene que codifica esta proteína têm sido associadas a um aumento marcante na massa muscular de organismos vertebrados, resultado de hiperplasia e hipertrofia das fibras musculares. Nosso grupo identificou previamente o promotor basal do gene da Miostatina e análises de bioinformática revelaram a presença de sítios evolutivamente conservados para a ligação de CREB, Meis, FXR e NFY, além de um sítio TATA. No presente trabalho nós utilizamos mutagênese sítio-dirigida para gerar diversas construções delecionais que possuem um ou mais sítios mutados, e testamos sua atividade in vitro usando mioblastos C2C12 de camundongo sob condições de proliferação e diferenciação, para analisar o papel destes sítios de ligação sobre a atividade do promotor. Os resultados mostraram que FXR aparentemente não confere efeito na atividade transcricional do promotor da Miostatina em ambos os momentos analisados, indicando que o papel regulador desta proteína pode estar relacionado ao controle da expressão da Miostatina em outro tipo celular, que não o mioblasto. O NFY apresentou um papel de ativador transcricional, enquanto CREB e Meis atuaram inicialmente como repressores durante a proliferação, passando a relaxar esta repressão durante a diferenciação dos mioblastos, permitindo que a atividade do promotor aumentasse significativamente. Trabalhando juntos, estes fatores de transcrição são capazes de manter a atividade do promotor em níveis mais baixos durante a proliferação dos mioblastos e, com o início da diferenciação, a repressão é liberada, e os níveis de atividade podem aumentar. Este padrão está de acordo com o padrão de expressão dinâmico observado para a proteína da Miostatina durante o desenvolvimento da musculatura esquelética em vertebrados
Abstract: Myostatin is a negative regulator of skeletal muscle deposition and mutations in the gene that encodes this protein have been associated to a remarkable increase in skeletal muscle mass, attributable to both hyperplasia and hypertrophy. We have previously identified Myostatin's basal promoter and bioinformatic analyses revealed the presence of evolutionarily conserved binding sites for CREB, Meis, FXR and NFY, besides a TATA box. In the present study we used site-directed mutagenesis to generate several expression constructs possessing one or more mutated sites, and tested their activity in vitro using mouse C2C12 myoblasts in proliferation and differentiation conditions, to analyze the role of these sites on the activity of the promoter. The results show that FXR appears not to confer any effect on the transcriptional activity of the promoter in both conditions, indicating that the regulatory role of this protein might be involved in the control of Myostatin expression in another cell type. NFY presents a role as transcriptional activator, while CREB and Meis act initially as repressors during proliferation, releasing this repression upon differentiation, which allows the activity of the promoter to significantly increase. Working together, these transcription factors are capable of maintaining the promoter activity at lower levels during the proliferation of myoblasts and, upon differentiation, the repression is released, and activity levels can be increased. This pattern is in agreement with the dynamic expression pattern observed for Myostatin during the skeletal muscle development in vertebrates
Doutorado
Histologia
Doutor em Biologia Celular e Estrutural
Zhang, Xiao-Qun. "Functional Studies on the PDGFR α gene promoter and effects of autocrine PDGF-A stimulation in vivo". Doctoral thesis, Uppsala universitet, Institutionen för genetik och patologi, 2001. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-1455.
Texto completo da fonteWarshamana, Gnana Sakuntala. "Interactions of T7 RNA polymerase with its promoters : Part I: T7 promoter contacts essential for promoter activity in vivo ; Part II: Isolation and characterization of a mutant T7 RNA polymerase with altered promoter specificity". Diss., Georgia Institute of Technology, 1992. http://hdl.handle.net/1853/26303.
Texto completo da fonteLorson, Christian. "An analysis of transcriptional regulation of the MVM capsid gene promoter". free to MU campus, to others for purchase, 1997. http://wwwlib.umi.com/cr/mo/fullcit?p9841319.
Texto completo da fonteLivros sobre o assunto "Promotor <Genetik>"
Lutz, Nover, ed. Plant promoters and transcription factors. Berlin: Springer-Verlag, 1994.
Encontre o texto completo da fonteLawrence, Privalsky Martin, ed. Transcriptional corepressors: Mediators of eukaryotic gene repression. Berlin: Springer, 2001.
Encontre o texto completo da fonteInternational Congress and Exhibition on Nutrition, Fitness, and Health (2006 Shanghai, China). Nutrition and fitness: Cultural, genetic, and metabolic aspects. Editado por Simopoulos Artemis P. 1933-. Basel: Karger, 2008.
Encontre o texto completo da fontePonnambalam, Sreenivasan. Transcription initiation at the 'Escherichia Coli' galactose Operon promoter region: Genetic and biochemicalstudies. Birmingham: University of Birmingham, 1988.
Encontre o texto completo da fonteYeomans, Gary M. Generic milk promotion in the United States and Canada. Uckfield: Nuffield Farming Scholarships Trust, 1998.
Encontre o texto completo da fonteEPA Workshop on the Development of Risk Assessment Methodologies for Tumor Promoters (1987 Bethesda, Md.). Report of the EPA Workshop on the development of risk assessment methodologies for tumor promoters. Washington, DC: Office of Health and Environmental Assessment and Office of Regulatory Support and Scientific Analysis, Office of Research and Development, U.S. Environmental Protection Agency, 1987.
Encontre o texto completo da fonteKamp, Philip Vande. Commodity promotion programs in the United States. Ithaca, N.Y: Dept. of Agricultural, Resource, and Managerial Economics, College of Agriculture and Life Sciences, Cornell University, 1999.
Encontre o texto completo da fonteKaiser, Harry Mason. An analysis of generic dairy promotion in the United States. Ithaca, N.Y: Dept. of Agricultural, Resource, and Managerial Economics, College of Agriculture and Life Sciences, Cornell University, 1995.
Encontre o texto completo da fonteBlisard, William Noel. Generic promotion of agricultural products: Balancing producers' and consumers' needs. [Washington, D.C.?]: U.S. Dept. of Agriculture, Economic Research Service, 1989.
Encontre o texto completo da fonteBlisard, William Noel. Generic promotion of agricultural products: Balancing producers' and consumers' needs. [Washington, D.C.?]: U.S. Dept. of Agriculture, Economic Research Service, 1989.
Encontre o texto completo da fonteCapítulos de livros sobre o assunto "Promotor <Genetik>"
Komarnytsky, Slavko, e Nikolai Borisjuk. "Functional Analysis of Promoter Elements in Plants". In Genetic Engineering, 113–41. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0073-5_6.
Texto completo da fonteGuilfoyle, Tom J. "The Structure of Plant Gene Promoters". In Genetic Engineering, 15–47. Boston, MA: Springer US, 1997. http://dx.doi.org/10.1007/978-1-4615-5925-2_2.
Texto completo da fontePelham, Hugh. "Properties and Uses of Heat Shock Promoters". In Genetic Engineering, 27–44. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4684-5377-5_2.
Texto completo da fonteSantos, Efrén, Ricardo Pacheco, Liliana Villao, Luis Galarza, Daniel Ochoa, Carlos Jordán e José Flores. "Promoter Analysis in Banana". In Banana: Genomics and Transgenic Approaches for Genetic Improvement, 157–79. Singapore: Springer Singapore, 2016. http://dx.doi.org/10.1007/978-981-10-1585-4_11.
Texto completo da fonteReuter, Ingmar, Thomas Werner e Edgar Wingender. "Computer-Assisted Methods for the Identification and Characterization of Polymerase II Promoters". In Genetic Engineering, 25–40. Boston, MA: Springer US, 1998. http://dx.doi.org/10.1007/978-1-4899-1739-3_2.
Texto completo da fonteNowell, Peter C. "Genetic Instability and Tumor Development". In Boundaries between Promotion and Progression during Carcinogenesis, 221–31. Boston, MA: Springer US, 1991. http://dx.doi.org/10.1007/978-1-4684-5994-4_19.
Texto completo da fonteAngel, J. M., e J. DiGiovanni. "Genetics of Skin Tumor Promotion". In Animal Models of Cancer Predisposition Syndromes, 143–57. Basel: KARGER, 1999. http://dx.doi.org/10.1159/000062010.
Texto completo da fonteBarry, Margaret M. "A Generic Template for Implementing Mental Health Promotion". In Implementing Mental Health Promotion, 131–59. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-23455-3_5.
Texto completo da fonteHoward, Daniel, e Karl Benson. "Evolutionary Computation Method for Promoter Site Prediction in DNA". In Genetic and Evolutionary Computation — GECCO 2003, 1690–701. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/3-540-45110-2_62.
Texto completo da fonteNordgren, Anders. "The Human Genome Project: Justification, Promotion, and Access to Results". In Responsible Genetics, 91–126. Dordrecht: Springer Netherlands, 2001. http://dx.doi.org/10.1007/978-94-015-9741-8_3.
Texto completo da fonteTrabalhos de conferências sobre o assunto "Promotor <Genetik>"
Frénoy, Antonine, François Taddei e Dusan Misevic. "Constrained Genetic Architecture Promotes Cooperation". In Artificial Life 14: International Conference on the Synthesis and Simulation of Living Systems. The MIT Press, 2014. http://dx.doi.org/10.7551/978-0-262-32621-6-ch004.
Texto completo da fonteFrénoy, Antonine, François Taddei e Dusan Misevic. "Constrained Genetic Architecture Promotes Cooperation". In Artificial Life 14: International Conference on the Synthesis and Simulation of Living Systems. The MIT Press, 2014. http://dx.doi.org/10.1162/978-0-262-32621-6-ch004.
Texto completo da fonteMedvet, Eric, Alberto Bartoli e Giovanni Squillero. "An effective diversity promotion mechanism in grammatical evolution". In GECCO '17: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3067695.3076057.
Texto completo da fonteTalamini, Jacopo, Giovanni Scaini, Eric Medvet e Alberto Bartoli. "Selfish vs. global behavior promotion in car controller evolution". In GECCO '18: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2018. http://dx.doi.org/10.1145/3205651.3208254.
Texto completo da fonteSingh, Abhyudai, Cesar A. Vargas e Rajesh Karmakar. "Stochastic analysis of genetic promoter architectures with memory". In 2013 IEEE 52nd Annual Conference on Decision and Control (CDC). IEEE, 2013. http://dx.doi.org/10.1109/cdc.2013.6761034.
Texto completo da fonteByeon, B., e K. Rasheed. "Bayesian Networks and Genetic Algorithms for Promoter Recognition". In IASTED Technology Conferences 2010. Calgary,AB,Canada: ACTAPRESS, 2010. http://dx.doi.org/10.2316/p.2010.728-030.
Texto completo da fonteLarson, Ari, Anton Bernatskiy, Collin Cappelle, Ken Livingston, Nicholas Livingston, John Long, Jodi Schwarz, Marc Smith e Josh Bongard. "Recombination Hotspots Promote the Evolvability of Modular Systems". In GECCO '16: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2016. http://dx.doi.org/10.1145/2908961.2908990.
Texto completo da fonteCarvalho, Jonata Tyska, e Stefano Nolfi. "Exploiting environmental differentiation to promote evolvability in artificial evolution". In GECCO '17: Genetic and Evolutionary Computation Conference. New York, NY, USA: ACM, 2017. http://dx.doi.org/10.1145/3067695.3075994.
Texto completo da fonteGuzman-Ruiz, Omar, Manuel Mejia-Lavalle, Alicia Martinez e Yasmin Hernandez. "Machine learning Algorithms applied to Genetic Promoter Sequences Recognition". In 2020 International Conference on Mechatronics, Electronics and Automotive Engineering (ICMEAE). IEEE, 2020. http://dx.doi.org/10.1109/icmeae51770.2020.00016.
Texto completo da fonte"Analysis of the activity of the DR5 promoter in tuber-forming plants". In Plant Genetics, Genomics, Bioinformatics, and Biotechnology. Novosibirsk ICG SB RAS 2021, 2021. http://dx.doi.org/10.18699/plantgen2021-111.
Texto completo da fonteRelatórios de organizações sobre o assunto "Promotor <Genetik>"
Brinckerhoff, Constance E. Genetic Analysis of a Single Nucleotide Polymorphism in the Matrix Metalloproteinase 1 Promoter in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, julho de 2002. http://dx.doi.org/10.21236/ada407580.
Texto completo da fonteBrinckerhoff, Constqance B. Genetic Analysis of a Single Nucleotide Polymorphism in the Matrix Metalloproteinase 1 Promoter in Breast Cancer. Fort Belvoir, VA: Defense Technical Information Center, julho de 2003. http://dx.doi.org/10.21236/ada419338.
Texto completo da fonteStrauss, S. H., V. Busov, K. Kosola, J. Kennedy, J. Morrell, C. Ma, A. Elias e E. Etherington. Genetic modification of gibberellic acid signaling to promote carbon sequestration in tree roots and stems. Office of Scientific and Technical Information (OSTI), maio de 2009. http://dx.doi.org/10.2172/952484.
Texto completo da fonteBusov, Victor. GENETIC MODIFICATION OF GIBBERELLIC ACID SIGNALING TO PROMOTE CARBON SEQUESTRATION IN TREE ROOTS AND STEMS. Office of Scientific and Technical Information (OSTI), março de 2013. http://dx.doi.org/10.2172/1067341.
Texto completo da fonteNorelli, John L., Moshe Flaishman, Herb Aldwinckle e David Gidoni. Regulated expression of site-specific DNA recombination for precision genetic engineering of apple. United States Department of Agriculture, março de 2005. http://dx.doi.org/10.32747/2005.7587214.bard.
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Texto completo da fonteSolvin, Thomas, e Inger Sundheim Fløistad. Statistics: Forest Seeds and Plants in the Nordic Region – Version 2023. The Nordic Genetic Resource Center (NordGen), agosto de 2023. http://dx.doi.org/10.53780/qoub7866.
Texto completo da fonteDawson, William O., e Moshe Bar-Joseph. Creating an Ally from an Adversary: Genetic Manipulation of Citrus Tristeza. United States Department of Agriculture, janeiro de 2004. http://dx.doi.org/10.32747/2004.7586540.bard.
Texto completo da fonteFridman, Eyal, e Eran Pichersky. Tomato Natural Insecticides: Elucidation of the Complex Pathway of Methylketone Biosynthesis. United States Department of Agriculture, dezembro de 2009. http://dx.doi.org/10.32747/2009.7696543.bard.
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