Literatura científica selecionada sobre o tema "NPY/AgRP neuron"
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Artigos de revistas sobre o assunto "NPY/AgRP neuron"
Oh, Youjin, Eun-Seon Yoo, Sang Hyeon Ju, Eunha Kim, Seulgi Lee, Seyun Kim, Kevin Wickman e Jong-Woo Sohn. "GIRK2 potassium channels expressed by the AgRP neurons decrease adiposity and body weight in mice". PLOS Biology 21, n.º 8 (18 de agosto de 2023): e3002252. http://dx.doi.org/10.1371/journal.pbio.3002252.
Texto completo da fontevan de Wall, Esther, Rebecca Leshan, Allison W. Xu, Nina Balthasar, Roberto Coppari, Shun Mei Liu, Young Hwan Jo et al. "Collective and Individual Functions of Leptin Receptor Modulated Neurons Controlling Metabolism and Ingestion". Endocrinology 149, n.º 4 (27 de dezembro de 2007): 1773–85. http://dx.doi.org/10.1210/en.2007-1132.
Texto completo da fonteCoutinho, Eulalia A., Melanie Prescott, Sabine Hessler, Christopher J. Marshall, Allan E. Herbison e Rebecca E. Campbell. "Activation of a Classic Hunger Circuit Slows Luteinizing Hormone Pulsatility". Neuroendocrinology 110, n.º 7-8 (21 de outubro de 2019): 671–87. http://dx.doi.org/10.1159/000504225.
Texto completo da fonteJones, Edward S., Nicolas Nunn, Adam P. Chambers, Søren Østergaard, Birgitte S. Wulff e Simon M. Luckman. "Modified Peptide YY Molecule Attenuates the Activity of NPY/AgRP Neurons and Reduces Food Intake in Male Mice". Endocrinology 160, n.º 11 (10 de maio de 2019): 2737–47. http://dx.doi.org/10.1210/en.2019-00100.
Texto completo da fonteMorton, GJ, e MW Schwartz. "The NPY/AgRP neuron and energy homeostasis". International Journal of Obesity 25, S5 (dezembro de 2001): S56—S62. http://dx.doi.org/10.1038/sj.ijo.0801915.
Texto completo da fonteLandry, Taylor, Daniel Shookster, Alec Chaves, Katrina Free, Tony Nguyen e Hu Huang. "Exercise increases NPY/AgRP and TH neuron activity in the hypothalamus of female mice". Journal of Endocrinology 252, n.º 3 (1 de março de 2022): 167–77. http://dx.doi.org/10.1530/joe-21-0250.
Texto completo da fonteSmith, A. W., M. A. Bosch, E. J. Wagner, O. K. Rønnekleiv e M. J. Kelly. "The membrane estrogen receptor ligand STX rapidly enhances GABAergic signaling in NPY/AgRP neurons: role in mediating the anorexigenic effects of 17β-estradiol". American Journal of Physiology-Endocrinology and Metabolism 305, n.º 5 (1 de setembro de 2013): E632—E640. http://dx.doi.org/10.1152/ajpendo.00281.2013.
Texto completo da fonteTeaney, Nicole A., e Nicole E. Cyr. "Sirtuin 1 Regulates Synapsin 1 in POMC-Producing N43-5 Neurons via FOXO1". Journal of the Endocrine Society 5, Supplement_1 (1 de maio de 2021): A56—A57. http://dx.doi.org/10.1210/jendso/bvab048.114.
Texto completo da fonteJohnson, Miranda D., Sebastien G. Bouret, Ambrose A. Dunn-Meynell, Christina N. Boyle, Thomas A. Lutz e Barry E. Levin. "Early postnatal amylin treatment enhances hypothalamic leptin signaling and neural development in the selectively bred diet-induced obese rat". American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 311, n.º 6 (1 de dezembro de 2016): R1032—R1044. http://dx.doi.org/10.1152/ajpregu.00326.2016.
Texto completo da fonteKrashes, Michael J., Bhavik P. Shah, Shuichi Koda e Bradford B. Lowell. "Rapid versus Delayed Stimulation of Feeding by the Endogenously Released AgRP Neuron Mediators GABA, NPY, and AgRP". Cell Metabolism 18, n.º 4 (outubro de 2013): 588–95. http://dx.doi.org/10.1016/j.cmet.2013.09.009.
Texto completo da fonteTeses / dissertações sobre o assunto "NPY/AgRP neuron"
Qu, Mengdi. "Molecular mechanism underlying CaMK1D-dependent function in AgRP neurons". Electronic Thesis or Diss., Strasbourg, 2024. http://www.theses.fr/2024STRAJ029.
Texto completo da fonteDisruption of stress response mechanisms in organisms can lead to cellular dysfunction and diseases like metabolic syndrome. Energy balance is mainly regulated by the central nervous system (CNS), which integrates hormonal, neuronal, and dietary signals from various tissues. Dysfunction in this system is linked to obesity and metabolic syndrome, both precursors to type 2 diabetes (T2D). Our laboratory discovered that calcium/calmodulin-dependent protein kinase ID (CaMK1D), a gene associated with T2D, promotes ghrelin-mediated food intake in mice. However, CaMK1D signaling in NPY/AgRP neurons still remains questions. In this work, we proformed RNA sequencing using the GT1-7 hypothalamic cell line. To this end, we found that CalHM6 is a downstream target of CaMK1D signaling. CalHM6 mRNA levels were significantly upregulated in CaMK1D-/- cells and downregulated when CaMK1D was re-expressed. This was confirmed in vivo in the hypothalamus of CaMK1D-/- mice. CalHM6, likely a voltage-gated calcium channel, showed increased intracellular Ca2+ levels in response to ghrelin in CaMK1D-/- cells compared to CaMK1D+/+ cells using jGCamps method. Altogether, our work showed CalHM6 is a novel target of CaMK1D. High CaMK1D, leading to low CalHM6 expression, may enhance food intake and obesity by modulating calcium-dependent signaling in NPY/AgRP neuron
Ramírez, Flores Sara. "Hypothalamic Ceramide Levels regulated by CPT1C mediate the Orexigenic effect of Ghrelin". Doctoral thesis, Universitat Internacional de Catalunya, 2014. http://hdl.handle.net/10803/276184.
Texto completo da fonteLivros sobre o assunto "NPY/AgRP neuron"
Wójcik-Gładysz, Anna. Ghrelin – hormone with many faces. Central regulation and therapy. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_awg_2020.
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