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Teses / dissertações sobre o tema "Neurotrophic peptide"

Autor: Grafiati

Publicado: 7 de julho de 2024

Última modificação: 31 de julho de 2025

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1

Littrell, Ofelia Meagan. "NIGROSTRIATAL DOPAMINE-NEURON FUNCTION FROM NEUROTROPHIC-LIKE PEPTIDE TREATMENT AND NEUROTROPHIC FACTOR DEPLETION." UKnowledge, 2011. http://uknowledge.uky.edu/neurobio_etds/1.

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Trophic factors have shown great promise in their potential to treat neurological disease. In particular, glial cell line-derived neurotrophic factor (GDNF) has been identified as a potent neurotrophic factor for midbrain dopamine (DA) neurons in the substantia nigra (SN), which lose function in Parkinson’s disease (PD). GDNF progressed to phase II clinical trials, which did not meet proposed endpoints. The large size and binding characteristics of GDNF have been suspected to contribute to some of the shortcomings of GDNF related to delivery to target brain regions. Smaller peptides derived fr

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2

Kaska, Jennifer Lynn. "Ependymin Mechanism of Action: Full Length EPN VS Peptide CMX-8933." Link to electronic thesis, 2003. http://www.wpi.edu/Pubs/ETD/Available/etd-0528103-102730/.

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3

Parikh, Suchi Vipin. "Ependymin peptide mimetics that assuage ischemic damage increase gene expression of the anti-oxidative enzyme SOD." Link to electronic thesis, 2003. http://www.wpi.edu/Pubs/ETD/Available/etd-0429103-132144.

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4

Wu, Yu. "Neuroprotective liquid crystalline cubosome and hexosome nanoparticle formulations by self-assembly of plasmalogen lipids and a neurotrophic peptide." Electronic Thesis or Diss., université Paris-Saclay, 2024. http://www.theses.fr/2024UPASQ003.

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L'objectif principal de cette thèse est d'étudier l'effet neuroprotecteur des plasmalogènes (Pls) et d'explorer le potentiel des nanoparticules lipidiques contre les maladies neurodégénératives. Notre stratégie vise à créer un système auto-assemblé, augmentant l'efficacité des plasmalogènes et d'un neuropeptide, le polypeptide activateur de l'adénylate cyclase hypophysaire (PACAP), pour la neuroprotection. Pls, un groupe distinctif de glycérophospholipides membranaires, contiennent généralement une chaîne d'acyle gras polyinsaturé en position sn-2 et une chaîne alkyle liée par une liaison éthe

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5

Grimsholm, Ola. "Neuropeptides and neurotrophins in arthritis : studies on the human and mouse knee joint." Doctoral thesis, Umeå universitet, Integrativ medicinsk biologi, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1863.

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Neuropeptides, such as substance P (SP) and bombesin/gastrin-releasing peptide (BN/GRP), and neurotrophins are involved in neuro-immunomodulatory processes and have marked trophic, growth-promoting and inflammation-modulating properties. The impact of these modulators in rheumatoid arthritis (RA) is, however, unclear. An involvement of the innervation, including the peptidergic innervation, is frequently proposed as an important factor for arthritic disease. Many patients with RA, but not all, benefit from treatment with anti-TNF medications. The studies presented here aimed to investigate the

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6

Lim, Robyn Renata. "Vasoactive intestinal polypeptide (VIP) and pituitary adenylate cyclase activating polypeptide (PACAP) : peptide neurotrophic actions in comparison with those of nerve growth factor (NGF) on rat adrenal pheochromocytoma PC12 cells." Thesis, University of Cambridge, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627532.

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7

Zussy, Charleine. "Caractérisation des effets de l'injection intracérébroventriculaire du peptide β-amyloïde [25-35] chez le rat mâle adulte : impact sur un système de neuroprotection endogène : le BDNF (Brain-derived neurotrophic factor) et ses récepteurs". Montpellier 2, 2009. http://www.theses.fr/2009MON20204.

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La maladie d'Alzheimer (MA) est une pathologie neurodégénérative caractérisée par la présence de plaques séniles majoritairement composées par la protéine β-amyloïde (Aβ). Afin de caractériser les effets de la toxicité amyloïde, nous avons évalué l'impact au cours du temps d'une injection intracérébroventriculaire (icv) du peptide Aβ25-35 agrégé sur des paramètres comportementaux, physiologiques et biochimiques chez le rat et sur un système neuroprotecteur endogène, le BDNF. Nous avons caractérisé 1, 2, 3 et 6 sem après l'injection icv d'Aβ25-35, les effets sur la mémoire à court- et long-term

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8

Farias, Caroline Brunetto de. "BDNF/TrkB em câncer colorretal : interações funcionais com GRPR e EGFR." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2012. http://hdl.handle.net/10183/72306.

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BDNF/TrkB são descritos em diversas neoplasias onde iniciam sinais mitogênicos, facilitam o crescimento tumoral, previnem apoptose e regulam angiogênese e metástase. Outros fatores de crescimento também são importantes para tumorigênese, como GRP/GRPR e EGF/EGFR. O objetivo geral deste trabalho foi investigar o papel de BDNF/TrkB em câncer colorretal avaliando possíveis interações com GRPR e EGFR. Verificamos que BDNF e seu receptor, TrkB, estão presentes em amostras de pacientes com câncer colorretal esporádico, e os níveis de BDNF encontram-se mais elevados no tecido neoplásico que no tecido

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9

Dyer, Jason Kim. "Presence of melanocortin receptors in Schwann cells in culture & functional relevance to the neurotrophic response : with an appendix on the establishment & characterisation of a new rat Schwann cell line." Thesis, University of Bristol, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.238825.

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10

Kritz, Angelika. "Peptides from phage display libraries for targeted gene delivery via the p75 neurotrophic receptor." Thesis, University College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.408712.

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11

Basille, Magali. "Contribution à l'étude des récepteurs du pituitary adenylate cyclase-activating polypeptide (PACAP) au cours de l'ontogénèse du cervelet de rat. Recherche d'une activité trophique potentielle du PACAP sur les neuroblastes cérébelleux." Rouen, 1998. http://www.theses.fr/1998ROUES010.

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Le pituitary adenylate cyclase-activating polypeptide (PACAP) est présent en quantités non négligeables dans le cervelet de rat au cours des trois semaines qui suivent la naissance c'est à dire pendant la période de multiplication, migration et différenciation des cellules granulaires, suggérant un rôle neurotrophique du PACAP dans le cervelet des mammifères au cours du développement. L'ontogénèse des sites de liaison du PACAP par autoradiographie quantitative dans le cervelet immature du rat a démontré une expression transitoire des sites de liaison au niveau de la couche granulaire externe (

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12

Arca, Turkan. "Attempts to clone the Limulus ependymin gene, and the effects of a human ependymin peptide on human SHSY neuroblastoma cells." Link to electronic thesis, 2005. http://www.wpi.edu/Pubs/ETD/Available/etd-050405-180333/.

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13

Rezende, Alexandre Cesar Santos de. "Investigação da ação neuroprotetora do fator neurotrofico ciliar (CNTF) conjugado com peptideo contendo dominio de translocação de proteina (PTD)." [s.n.], 2009. http://repositorio.unicamp.br/jspui/handle/REPOSIP/317520.

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Orientador: Francesco Langone<br>Tese (doutorado) - Universidade Estadual de Campinas, Instituto de Biologia<br>Made available in DSpace on 2018-08-14T01:41:43Z (GMT). No. of bitstreams: 1 Rezende_AlexandreCesarSantosde_D.pdf: 5210413 bytes, checksum: 60e511530e7c408001bf455f9968219f (MD5) Previous issue date: 2009<br>Resumo: O fator neurotrófico ciliar (CNTF) despertou grande interesse com a descoberta do seu efeito neuroprotetor sobre motoneurônios após secção de nervos periféricos em ratos neonatos e camundongos adultos. Contudo, seus efeitos colaterais de perda de peso e caquexia impedem

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14

Petit-Dop, Florence. "Effets de deux neurotrophines, BDNF et NT3, et d'un nouveau peptide, le "Melanin concentrating hormone Gene Overprinted Polypeptide" (MGOP) sur le développement de neurones hypophysiotropes de Rat in vitro." Paris 11, 2001. http://www.theses.fr/2001PA11T031.

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Le développement neuronal est contrôlé par des facteurs épigénétiques de l'environnement cellulaire. Parmi les facteurs diffusibles, les neurotrophines agissent dans plusieurs régions centrales sur la différenciation morphologique et fonctionnelle des neurones post-mitotiques. Dans ce travail, nous nous sommes intéressés à l'effet du BDNF et de la NT3 sur la différenciation de sous populations neuronales hypothalamiques, et particulièrement celles impliquées dans les régulation neuroendocrin iennes. Nous avons caractérisé la différenciation phénotypique des neurones hypophysiotropes au cours d

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15

Jerónimo-Santos, André. "Brain-derived neurotrophic factor and adenosine signalling on amyloid-β peptide induced toxicity : impact on hippocampal function". Doctoral thesis, 2014. http://hdl.handle.net/10451/15639.

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Tese de doutoramento, Ciências Biomédicas (Neurociências), Universidade de Lisboa, Faculdade de Medicina, 2014<br>Brain-derived neurotrophic factor (BDNF) and its high-affinity full-length receptor, TrkB-FL, play a central role in the nervous system by providing trophic support to neurons and by regulating synaptic transmission and plasticity. BDNF signalling is impaired in Alzheimer’s disease (AD), a neurodegenerative disorder characterized, among other features, by the accumulation of the amyloid-β (Aβ) peptide. Although the mechanisms implicated in the reduction of BDNF signalling in AD wer

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16

Ng, Tiffany. "The Effect of Teneurin C-terminal Associated Peptide-1 (TCAP-1): Protection Against Hypoxic-stress and Regulation of Brain-derived Neurotrophic Factor (BDNF) in Immortalized Hypothalamic N38 Cells." Thesis, 2010. http://hdl.handle.net/1807/25875.

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Teneurin C-terminal associated peptide-1 (TCAP-1) is a recently characterized peptide that may act as one potential neuroprotective agent as it has been shown to regulate several stress-associated behaviours in rodents and possesses a number of protective actions on cells, however the mechanism remains unknown. Brain-derived neurotrophic factor (BDNF) is a neurotrophin recognized for mediating survival, differentiation, and proliferation. TCAP-1 may act, in part, via BDNF to provide neuroprotection via modulation of BDNF expression. The aim of this research was to further investigate the mecha

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17

Nnanabu, Ernest. "C10 semi-peptoid beta-turn peptidomimetics: syntheses, characterization and biological studies." 2006. http://hdl.handle.net/1969.1/ETD-TAMU-1710.

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Over the years, the Burgess group has been focusing on the preparation and testing of small molecules that mimic protein secondary structures for protein-protein interactions. The most successful compounds made are C10 peptide macrocycles that effectively mimic Î²-turns and have given promising results from biological testing. These peptide macrocycles have also been dimerized to give even more effective ligands for protein-protein interaction. The successes of the peptide macrocycles have enabled us to look into increasing the chemical diversity of our libraries. This we believe will not only

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