Literatura científica selecionada sobre o tema "Maladie artérielle périphérique – chirurgie"
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Artigos de revistas sobre o assunto "Maladie artérielle périphérique – chirurgie"
Qi, Arthur A., e Christina S. Korownyk. "L’exercice contre la maladie artérielle périphérique". Canadian Family Physician 68, n.º 4 (abril de 2022): 278–79. http://dx.doi.org/10.46747/cfp.6804278.
Texto completo da fonteElouakili, I., M. Bouayad, Y. Tijani, L. Benjilali, B. Lekehal, Y. Sefiani, A. El Mesnaoui, F. Ammar e Y. Bensaid. "Maladie de Horton et atteinte artérielle périphérique". Journal des Maladies Vasculaires 35, n.º 5 (setembro de 2010): 313. http://dx.doi.org/10.1016/j.jmv.2010.07.061.
Texto completo da fonteChaparala, Ramakrishna P. C., Nicolas M. Orsi, Nigel J. Lindsey, Raman S. Girn e Shervanthi Homer-Vanniasinkam. "Profil Inflammatoire de la Maladie Artérielle Périphérique". Annales de Chirurgie Vasculaire 23, n.º 2 (março de 2009): 186–93. http://dx.doi.org/10.1016/j.acvfr.2009.05.012.
Texto completo da fonteKeller, Sanjiv, Luca Calanca, Enrica Porceddu, Marco Fresa, Céline Deslarzes e Lucia Mazzolai. "Maladie artérielle périphérique : du diagnostic au traitement". Revue Médicale Suisse 19, n.º 853 (2023): 2304–9. http://dx.doi.org/10.53738/revmed.2023.19.853.2304.
Texto completo da fonteLengani, A., A. Ouédraogo, G. Sanou, J. I. Nitiéma, Y. J. Bonzi, S. H. Traoré e G. Coulibaly. "Maladie artérielle périphérique chez les patients atteints de maladie rénale chronique non dialysés". Néphrologie & Thérapeutique 16, n.º 5 (setembro de 2020): 308. http://dx.doi.org/10.1016/j.nephro.2020.07.160.
Texto completo da fonteBulaïd, Y., C. Klein, R. Gouron, O. Jardé e F. Deroussen. "Résultats du traitement des pieds creux médiaux d’enfants atteints de la maladie de Charcot-Marie-Tooth par des orthèses de détorsion nocturnes innovantes". Médecine et Chirurgie du Pied 37, n.º 1 (março de 2021): 1–9. http://dx.doi.org/10.3166/mcp-2021-0065.
Texto completo da fonteAuboire, L., M. Becquet e C. Le Hello. "Atteinte artérielle périphérique dans la maladie de Horton : comment différencier athérome et inflammation ?" Journal des Maladies Vasculaires 36, n.º 4 (julho de 2011): 280–84. http://dx.doi.org/10.1016/j.jmv.2011.06.003.
Texto completo da fonteKeller, Sanjiv, Rosaria Del Giorno, Giacomo Buso, Céline Deslarzes, Luca Calanca, Stefano Lanzi e Lucia Mazzolai. "Passeport vasculaire : un outil pour la prévention secondaire des patients avec maladie artérielle périphérique". Revue Médicale Suisse 17, n.º 762 (2021): 2128–31. http://dx.doi.org/10.53738/revmed.2021.17.762.2128.
Texto completo da fonteBlomstrand, David, Tilo Kölbel, Bengt Lindblad e Anders Gottsäter. "Le complexe Protéine C activée - inhibiteur de protéine C dans la maladie artérielle périphérique". Annales de Chirurgie Vasculaire 24, n.º 5 (julho de 2010): 642–50. http://dx.doi.org/10.1016/j.acvfr.2010.12.044.
Texto completo da fonteBoned, A., R. Granger, M. Bousquet, L. Richard, V. Gremeaux, M. Labrunée e T. Guiraud. "Effets du TENS sur la distance de marche chez un patient atteint d’une maladie artérielle périphérique". Annals of Physical and Rehabilitation Medicine 55 (outubro de 2012): e317. http://dx.doi.org/10.1016/j.rehab.2012.07.799.
Texto completo da fonteTeses / dissertações sobre o assunto "Maladie artérielle périphérique – chirurgie"
Mougin, Justine. "Mise au point et optimisation d'un modèle lagomorphe hypercholestérolémique de resténose intra-stent et application pour le développement d'un stent obtenu par électrofilage". Electronic Thesis or Diss., Université de Lille (2022-....), 2023. http://www.theses.fr/2023ULILS019.
Texto completo da fonteObjective: This study aimed to evaluate the use of a double injured atherosclerotic iliac rabbit model for myointimal hyperplasia evaluation. Secondarily, this animal model was use for pre clincial evaluation of a new anti-in stent restenosis simvastatin electrospun covered stent. Methods: Twenty four New Zealand White (NZW) rabbits were included in this study. In order to enhance and accelerate atherogenesis, atherogenic diet (0.3% cholesterol and 4.9% coconut oil) and mechanical endothelial injury of iliac artery were used. Twelve rabbits (24 iliac arteries) were used to evaluate the benefit of intimal balloon injury compared to the diet alone on myointimal hyperplasia. On day 7, rabbits beneficiated balloon iliac injury on the left side only. Eight weeks they were scarified and iliac arteries were harvested to histologic examination and comparaison. Twelve rabbits were included in the second evaluation to compare anti instent restenosis effect of a new drug eluting chrome-cobalt stent (DES) coated with polycyclodextrin-chitosan-simvastatin polymer after electrospinning technique. Balloon injury were performed on both iliacs on day 7, then stenting of BMS on right iliac and DES on left iliac and animals were sacrified 4 weeks later after angiography for histologic examination. Results: Balloon iliac injuries (BI) revealed a significant higher Schwartz injury score (0.599±0.368 control vs 1.150±0.306 BI, p=0.013), percentage of stenosis (25.7±19.1 control vs 49.9±21.9 BI, p=0.012) and ratio collagen (0.252 ± 0.017 control vs 0.365 ± 0.011 BI, p=0.0001) than atherosclerotic diet only. Five rabbits died before the end of the protocol. After appaired comparison of BMS and DES, results were significantly better for BMS and pre-euthanasia angiography revealed that 3/7 DES were thrombosed versus 0/7 for BMS. Conclusions : Association of atherogenic diet and balloon injuries allow to obtain important neointimal hyperplasia and wall remodeling in rabbit iliac arteries that confirm its interest as in stent restenosis model for new DES
Gignac, Catherine. "Prévalence de la maladie artérielle périphérique dépistée lors d'une épreuve d'effort dans un centre de cardiologie tertiaire". Thesis, Université Laval, 2007. http://www.theses.ulaval.ca/2007/24201/24201.pdf.
Texto completo da fonteLétourneau-Montminy, Myriam. "Facteurs influençant la prescription de la médication cardioprotectrice recommandée chez les patients avec une maladie artérielle périphérique". Master's thesis, Université Laval, 2015. http://hdl.handle.net/20.500.11794/27130.
Texto completo da fonteIntroduction: Guidelines recommend that patients with peripheral arterial disease (PAD) should be medically treated to reduce the occurrence of serious cardiovascular events. Objective: To identified factors associated with the prescription of preventive therapies in patients with PAD. Methods: Consecutive patients with PAD (n=362) who underwent peripheral percutaneous transluminal angioplasty between 2008 and 2010 in one tertiary care center (CHU de Quebec, Canada) were considered for the study. Data were collected from the medical charts. The main outcome was the combined prescription of three therapies. Results: A total of 52% of the patients received the three combined therapies. Having at least three cardiovascular risk factors (Odds Ratio (OR)=4.51; 95% CI: 2.76-7.37) was the factor most strongly associated with the prescription of the combined therapies. Conclusion: Canadian patients with PAD are still not optimally managed. We still need to better understand the barriers and facilitators to the application of the guidelines in PAD patients.
Droupy, Stéphane. "Approches anatomique et fonctionnelle, expérimentale et clinique de la vascularisation artérielle du pénis". Paris 11, 2000. http://www.theses.fr/2000PA11T044.
Texto completo da fonteMarlinge, Marion. "Profil du récepteur de l’adénosine A2A dans les pathologies cardio-vasculaires". Thesis, Aix-Marseille, 2019. http://www.theses.fr/2019AIXM0670.
Texto completo da fonteAtherosclerosis is responsible for the decrease in the diameter of the vessels by formation of a "plaque" consisting in particular of lipids limiting the blood circulation (ischemia) and tissue oxygenation. Adenosine is able to regulate cardiovascular function, particularly through its A2A receptor, which induces vasodilation to increase blood intake. A low presence of A2AR seems to mark ischemia (without specificity of territory) while the presence of reserve receptors (maximum biological response despite a small number of occupied sites) sign a severe disturbance in the blood flow coronary (inducible ischemia). These analyzes can be done on a classic blood sample. Blood adenosine could predict the risk of death from cardiogenic shock complicating the initial disease, where peripheral organs are hypoperfused (cardiac pump dysfunction) that the body attempts to compensate for by vasoconstriction (less A2AR). This work suggests the possibility of using the adenosinergic system both at the level of diagnosis (absence of biological marker of ischemia and coronary artery disease reliable to date), prognosis, than at the therapeutic level
Pottecher, Julien. "Muscle squelettique et ischémie-reperfusion expérimentale des membres : mécanismes impliqués dans la protection ou les effets délétères de la cyclosporine et facteurs limitant les conditionnements pharmacologique et ischémique". Phd thesis, Université de Strasbourg, 2012. http://tel.archives-ouvertes.fr/tel-00871846.
Texto completo da fonte"Prévalence de la maladie artérielle périphérique dépistée lors d'une épreuve d'effort dans un centre de cardiologie tertiaire". Thesis, Université Laval, 2007. http://www.theses.ulaval.ca/2007/24201/24201.pdf.
Texto completo da fonteCapítulos de livros sobre o assunto "Maladie artérielle périphérique – chirurgie"
Stouffer, George A., Leslie P. Wong e Marschall S. Runge. "Maladie artérielle périphérique". In Médecine interne de Netter, 234–42. Elsevier, 2011. http://dx.doi.org/10.1016/b978-2-294-70951-7.00031-1.
Texto completo da fonte