Livros: "Indice de stress"

1

C, Parsons K., Fox J. G, and Metz B, eds. Heat stress indices. Taylor & Francis, 1995.

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2

Bootsma, A. Stress indices for spring wheat on the Canadian prairies. Research Branch, Agriculture Canada, 1992.

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3

Magnólia Sousa Bandeira de Melo. Indice toponímico do centro histórico de São Luís. Universidade Federal do Maranhão, Departamento de Biblioteconomia, 1990.

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4

Comics, Evil Twin. Awesome: The indie spinner rack anthology. Evil Twin Comics, 2007.

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5

W, May C., University of Washington. Environmental Engineering and Science Program., and Washington (State). Dept. of Ecology., eds. Quality indices for urbanization effects in Puget Sound Lowland streams. [Environmental Engineering and Science, Dept. of Civil Engineering, University of Washington, 1997.

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6

Hypothermia and cold stress. Aspen Systems Corp., 1986.

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7

United Service Institution of India, ed. Stress, suicides, and fratricides in the army: Crisis within. Vij Books India Pvt Ltd, 2011.

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8

Stress and coping: The Indian experience. 2nd ed. Sage, 1998.

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9

Stress and coping: The Indian experience. Sage Publications, 1992.

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10

1947-, Cohen Sheldon, ed. Behavior, health, and environmental stress. Plenum Press, 1986.

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11

Boler, Frances M. Seismicity and stress changes subsequent to destress blasting at the Galena mine and implications for stress control strategies. U.S. Dept. of the Interior, Bureau of Mines, 1993.

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12

Italy) International Conference on Environmental Stressors in Biology and Medicine (2nd 2011 Siena. Environmental stressors in biology and medicine. Edited by Valacchi Giuseppe. Published by Blackwell Pub. on behalf of the New York Academy of Sciences, 2012.

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13

Ṭhakkara, Sādhanā. Indiyana strīta phūda. Gūrjara Grantharatna Kāryālaya, 2010.

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14

Claude, Lévi-Strauss, ed. Lévi-Strauss, le passage du nord-ouest: Précédé d'un texte de Claude Lévi-Strauss, Indian cosmetics. Herne, 2008.

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15

MD, Stacey B. Day. Biopower And Induced Stress Or Four Oaths Of Hagakure - Which Culture Will Define Life In The 21st Century: Four oaths and Induced Stress. Hagakure Society, Saga, Japan, 2010.

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16

Pai, Anant, Luis M. Fernandes, and Ram Waeerkar. Manduka: The accidental astrologer. Amar Chitra Katha Pvt Ltd, 2011.

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17

Jēkkab, Ṭensi. Taṭṭukaṭa speṣyals. D.C. Life, 2014.

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18

The matchless wits. Amar Chitra Katha Pvt Ltd., 2010.

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19

M, Guéra R., Loughridge Lee, and Balsman Phil, eds. Indian country. [Titan], 2007.

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20

Indian painting: The lesser-known traditions. Niyogi Books, 2011.

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21

Great Indian emperors. Amar Chitra Katha Pvt Ltd., 2009.

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22

Tales from the Panchatantra. Amar Chitra Katha Pvt Ltd., 2009.

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23

Singh, Sandipa. Monthly maps of sea surface height in the North Atlantic and zonal indices for the Gulf Stream using TOPEX/Poseidon altimeter data. Woods Hole Oceanographic Institution, 1997.

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24

A, Kelly Kathryn, and United States. National Aeronautics and Space Administration., eds. Monthly maps of sea surface height in the North Atlantic and zonal indices for the Gulf Stream using TOPEX/Poseidon altimeter data. Woods Hole Oceanographic Institution, 1997.

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25

A, Kelly Kathryn, and United States. National Aeronautics and Space Administration., eds. Monthly maps of sea surface height in the North Atlantic and zonal indices for the Gulf Stream using TOPEX/Poseidon altimeter data. Woods Hole Oceanographic Institution, 1997.

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26

Hain, Richard D. W., and Satbir Singh Jassal. Coping skills. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198745457.003.0024.

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Working within health care induces different levels of stress in professionals. When dealing with life-limited children, these stresses can be immense and, if not managed correctly, can lead to burnout. This chapter addresses specific issues that health-care professionals may face, with specific attention paid to how to effectively manage these issues.

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27

Zilliox, Lindsay, and James W. Russell. Diabetic and Prediabetic Neuropathy. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0115.

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Impaired glucose regulation (IGR) constitutes a spectrum of impaired glucose and metabolic regulation that can result in neuropathy. Several different pathways of injury in the diabetic peripheral nervous system that include metabolic dysregulation induced by metabolic syndrome induce oxidative stress, failure of nitric oxide regulation, and dysfunction of certain key signaling pathways. Oxidative stress can directly injure both dorsal route ganglion neurons and axons. Modulation of the nitric oxide system may have detrimental effects on endothelial function and neuronal survival. Reactive oxidative species can alter mitochondrial function, protein and DNA structure, interfere with signaling pathways, and deplete antioxidant defenses. Advanced glycelation end (AGE) products and formation of ROS are activated by and in turn regulate key signal transduction pathways.

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28

Quijije, Nadia. Trauma in the Medical-Surgical Patient. Edited by Frederick J. Stoddard, David M. Benedek, Mohammed R. Milad, and Robert J. Ursano. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190457136.003.0018.

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This chapter reviews psychiatric consultation for trauma and stress in medical-surgical patients. Hospitalization can induce psychologic or psychiatric disturbance and worsen the clinical condition of patients who are suffering from medical and surgical comorbidities. Some medical conditions can be related to stress related disorders indirectly, while others, such as critical illness/intensive care unit treatment or direct physical injury, are themselves traumatic stressors that can promote trauma and stressor-related disorders (TSRDs). Given the negative impact of stress-related disorders on quality of life, mental health clinicians should diagnose TSRDs to ensure patients receive appropriate care. Treatment and management can be provided in multiple forms of psychological therapies and psychopharmacology, and within a multidisciplinary team, particularly for the medical surgical patient. Psychiatrists, psychologists, and social workers must assist patients with terminal illnesses by optimizing end-of-life care, supporting patients and their families, and encouraging approaches to allow the transformative process of dying to be meaningful.

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29

Kay, William K., and Stephen J. Hunt. Pentecostal Churches and Homosexuality. Edited by Adrian Thatcher. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199664153.013.39.

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Historically, the majority of Pentecostal churches stem from holiness and revivalistic streams of Christianity, while neo-Pentecostal churches are often indigenous plantings that broke away from congregations established by earlier Protestant mission. Given their stress on religious experience and their belief in the indwelling power of the Holy Spirit, Pentecostal churches have always stressed individual holiness, and this holiness is understood in terms of abstinence from drugs, alcohol, gambling, immodest dress, and sexual immorality as traditionally defined. This chapter describes adjustments and initiatives that indicate how new norms may emerge. The issue is essentially concerned with the interpretation of Scripture and variations in church government. Where these interpretations align with an LBGT-friendly hermeneutic, LBGT-friendly Pentecostal churches will and have emerged. Such changes tend to occur in new or split-off groups rather than in traditional Pentecostal denominations, especially when denominations are governed by large ministerial conferences where decisions are by secret ballot.

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30

Heer, Jeet. This Is Serious: Canadian Indie Comics. Conundrum Press, 2019.

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31

Alcantara, Lyonna F., Eric M. Parise, and Carlos A. Bolaños-Guzmán. Animal Models of Mood Disorders. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0026.

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Animal modeling has advanced our understanding of the underlying pathophysiology of human neuropsychiatric disorders and facilitated development of safer, more efficient medications. Similar to humans with depression, rodents exposed to various stress paradigms exhibit aberrant responses to rewarding stimuli, along with hormonal and immunological dysregulation. Development of more complex models, such as social defeat, has led to a firmer grasp of the mechanisms mediating resilience and susceptibility to stress; and adapted versions of social defeat have yielded insights into how emotional stress influences development of mood disorders. This chapter focuses on stress-induced models of mood disorders and outlines how a depression-like phenotype is induced and tested in rodents.

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32

Terry, Ph D. Lyles. Good Stress: Living Younger Longer. Healthful Communications, 2007.

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33

Makan, Chetna. Chai, chaat & chutney: A street food journey through India. 2017.

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34

Ghori, Ambreen, and Aarti Gupta. Anxiety Disorders. Edited by Rajiv Radhakrishnan and Lily Arora. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190265557.003.0019.

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This chapter reviews topics on anxiety disorders including panic disorder, specific phobia, social anxiety disorder, obsessive-compulsive disorder, posttraumatic stress disorder, acute stress disorder, generalized anxiety disorder, anxiety disorder due to a general medical condition, Substance/medication-induced anxiety disorder and body dysmorphic disorder

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35

Arditti, Joyce A. Parental Incarceration and Family Inequality in the United States. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198810087.003.0003.

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This chapter argues that mass incarceration is an insidious mechanism to limit equal opportunity to freely and optimally ‘do family’. Indeed, research documents a host of negative family outcomes associated with parental incarceration and children seem to be particularly vulnerable. This chapter introduces a ‘Family Inequality Framework’ (FIF), which builds on research and theory that conceptualizes parental incarceration as an ongoing family stressor that influences critical parenting processes and indices of family functioning. Based on family stress theory and ecological frameworks, the FIF points to material hardship as the main conduit through which parental incarceration contributes to and reproduces family inequality. Moreover, an FIF represents a shift in emphasis from how mass imprisonment contributes to inequality among incarcerated adults, to how parental incarceration contributes to inequality among children.

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36

Thomas, Gregory S., L. Samuel Wann, and Myrvin H. Ellestad, eds. Ellestad's Stress Testing. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190225483.001.0001.

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The 6th edition of the textbook Ellestad’s Stress Testing: Principles and Practice was written for the new and veteran clinician alike performing stress testing. Thoroughly updated, referenced and interspersed with case examples, the book reviews how to get the most out exercise testing, without and with ancillary imaging. In addition to evaluation of ST segment depression, other powerful tools to detect ischemia and forecast the future are reviewed to increase the diagnostic accuracy and prognostic ability of exercise testing. The recognition and significance of exercise induced arrhythmias and conduction defects are examined. When to convert to pharmacologic stress or add ancillary imaging, including myocardial perfusion imaging, echocardiography, coronary calcium scoring, and magnetic reference imaging are reviewed. The use of stress testing in the management of obstructive and non-obstructive coronary artery disease (CAD), heart failure, cardiac rehabilitation, peripheral vascular disease, congenital heart and other cardiovascular diseases (CVD) is examined. Options to optimize the diagnostic capabilities of exercise and other diagnostic testing for women are highlighted. Strategic use of exercise testing in the face of a decreasing burden of CAD in the developed world, as well as the opportunity to rely on exercise testing as the first test to evaluate CVD in the developing world, are reviewed. The fundamentals of exercise physiology and myocardial ischemia that serve as the foundation for exercise testing in health and disease are explained.

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37

FUN, A. G. E. Relax with Mandala: 120 Mandalas Coloring Book to Reduce the Accumulated Stress and to Induce Relaxation. Independently Published, 2020.

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38

Adam, Sheila, Sue Osborne, and John Welch. The patient within the critical care environment. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199696260.003.0003.

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Both critical illness and treatment in the critical care unit are extremely stressful, presenting great physical and psychological challenges for patients and their families. There are a range of compensatory responses to stress which may be adaptive, but severe or prolonged stress can induce a destructive spiral of decompensation. The importance of a holistic approach to care cannot be overemphasized; this chapter sets out the priorities of care for critically ill patients, and the common needs and problems for both patients and their families. The issues discussed include the mechanisms of stress in critical illness, the promotion of sleep, use of analgesia and sedation, management of delirium, complications of immobility, mouth, eye, and skin care, infection control, requirements for safe transfer, and care of the dying patient.

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39

MacNair, Rachel M. Perpetration-Induced Traumatic Stress. Praeger, 2002. http://dx.doi.org/10.5040/9798400696183.

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This volume introduces the concept of Perpetration-Induced Traumatic Stress (PITS), a form of PTSD symptoms caused not by traditionally expected roles, such as being a victim or rescuer in trauma, but by being an active participant in causing trauma. Sufferers of PITS may be in the roles of soldiers, executioners, or police officers, where it is socially acceptable or even expected for them to cause trauma, including death. Scattered evidence of PITS is consolidated, its implications are explored, and exciting potentials for future research are suggested. Compared to the more widely understood PTSD, there appears to be greater severity and different symptom patterns for those affected by PITS. Obvious differences to be explored for those who kill include questions of context, guilt, meaning, content of dreams, and sociological questions, leading to special implications for therapy, research into the causality of PTSD, and violence prevention efforts. Disciplines including sociology, public policy, history, philosophy, and theology will also find applications for this groundbreaking material.

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40

Wiles, Pauline. Indie With Ease: Practical ways to conquer stress, boost productivity, and love your self-publishing career. Pauline Wiles, 2018.

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41

Lieres, Sophia von. Tsunami in Kerala, India: Long-Term Psychological Distress, Sense of Coherence, Social Support, and Coping in a Non-Industrialized Setting. Lang GmbH, Internationaler Verlag der Wissenschaften, Peter, 2013.

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42

Lieres, Sophia von. Tsunami in Kerala, India: Long-Term Psychological Distress, Sense of Coherence, Social Support, and Coping in a Non-Industrialized Setting. Lang GmbH, Internationaler Verlag der Wissenschaften, Peter, 2013.

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43

Madl, Ulrike. Pathophysiology of glucose control. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0258.

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Hyperglycaemia is a frequent phenomenon in critically-ill patients, associated with increased morbidity and mortality. Hyperglycaemia results in cellular glucose overload and toxic adverse effects of glycolysis and oxidative phosphorylation, especially in tissues with insulin-independent glucose uptake, and acute hyperglycaemia can exert a variety of negative effects. It is the main side effect of intensive insulin therapy. Both severe and moderate hypoglycaemia are independent risk factors of mortality in critically-ill patients. Prolonged hypoglycaemia induces neuronal damage, but may also have adverse cardiovascular effects. Several risk factors predispose critically-ill patients to hypoglycaemic events. Rapid glucose fluctuations may induce oxidative stress and lead to vascular damage. Glucose complexity is a marker of endogenous glucose regulation. Association between hyperglycaemia and outcome is weaker in diabetic critically-ill patients than in non-diabetic patients. Pre-admission glucose control in diabetic critically-ill patients plays a role in the response to glucose control and mortality.

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44

Pestonjee, D. M. Stress and Coping: The Indian Experience. Sage Publications Pvt. Ltd, 1999.

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45

Martin, Jeffrey J. Negative Affect. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780190638054.003.0020.

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Experiencing negative affect during training and during competition is common. Much of the disability sport psychology research on negative affect has not been embedded in common sport theories of anxiety. This chapter presents an overview of the most common concepts and theories regarding anxiety and stress in sport. It also discusses the body of literature in disability sport that addresses negative affect. Most of the research in disability sport on negative affect has been on anxiety and stress, and other negative emotions such as shame, disappointment, frustration, grief, or disgust have not been examined. For athletes with disabilities there are many potential sources of stress, such as a person’s impairment, impairment effects, general sport, disability sport, and non-sport-related considerations. All of these factors can be stressful and tax an athlete’s ability to manage challenges that influence the ability to train and compete optimally. The chapter concludes with a discussion of how the classification process and the wheelchair in various wheelchair sports can also induce anxiety.

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46

Baum, Nehami. Therapist Self-Care to Mitigate Secondary Traumatization. Edited by Sara Maltzman. Oxford University Press, 2016. http://dx.doi.org/10.1093/oxfordhb/9780199739134.013.13.

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Secondary traumatic stress (STS) is among various stress responses to which therapists who work with traumatized clients are susceptible. In the broad sense of the term, it refers to posttraumatic stress disorder (PTSD) and other symptoms of stress stemming from exposure to traumatized survivors. The chapter begins with a discussion of why therapists who work with traumatized clients are susceptible to STS and the main mechanisms by which it is transmitted. It then goes on to indicate the repercussions of STS on therapists’ effectiveness. The remainder of the chapter is devoted to the self-help methods that therapists can use to build up their resilience and manage STS. It highlights the need to normalize and identify the symptoms, then goes on to discuss what needs to be done at work, what can be done in the therapist’s personal life, and the importance of making meaning.

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47

Facemire, Charles F. Comparison of the sensitivity of electrophoresis and ecological indices for the detection of environmental stress in aquatic ecosystems. 1989.

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48

Sommer, Claudia. Endogenous opioids mediate stress-induced analgesia. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0031.

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This chapter reviews the landmark paper published in 1990 by Stein et al. and entitled ‘Opioids from immunocytes interact with receptors on sensory nerves to inhibit nociception in inflammation’. Opioids, besides acting centrally as analgesics, may act peripherally upon opioid receptors located on axons and on immune cells. In the publication by Stein et al., it was shown for the first time that endogenous opioid peptides released from immune cells mediate stress-induced analgesia, potentially through opioid receptors on peripheral nerve endings. This finding has led to numerous follow-up studies on endogenous analgesia, including work showing that cannabinoid analgesia is mediated via the peripheral release of opioids, and to the concept of topical opioid analgesia, which may be a good way of using the potent analgesia that opioids can convey, without their CNS-associated side effects.

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49

Naninck, E. F. G., P. J. Lucassen, and Aniko Korosi. Consequences of Early-Life Experiences on Cognition and Emotion. Edited by Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.003.

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Perinatal experiences during a critical developmental period program brain structure and function “for life,” thereby determining vulnerability to psychopathology and cognition in adulthood. Although these functional consequences are associated with alterations in HPA-axis activity and hippocampal structure and function, the underlying mechanisms remain unclear. The parent-offspring relationship (i.e., sensory and nutritional inputs by the mother) is key in mediating these lasting effects. This chapter discusses how early-life events, for example, the amount of maternal care, stress, and nutrition, can affect emotional and cognitive functions later in life. Interestingly, effects of perinatal malnutrition resemble the perinatal stress-induced long-term deficits. Because stress and nutrition are closely interrelated, it proposes that altered stress hormones and changes in specific key nutrients during critical developmental periods act synergistically to program brain structure and function, possibly via epigenetic mechanisms. Understanding how the adult brain is shaped by early experiences is essential to develop behavioural and nutritional preventive therapy.

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50

Falk, Bareket, and Raffy Dotan. Temperature regulation. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199232482.003.0023.

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This chapter outlines the physical and physiological changes that occur during growth and maturation and the possible effects these changes can have on the nature and effectiveness of thermoregulation. The physiological responses to heat stress are discussed in terms of metabolic, circulatory, hormonal, and sweating responses, changes in body temperature, and in terms of heat tolerance. Also discussed is hydration status, which can affect thermoregulatory effectiveness in the heat. The physiological response to cold stress is considered in terms of the metabolic and circulatory responses and their possible influence on the effectiveness of thermoregulation. The discussion does not outline the thermoregulatory response per se, but rather emphasizes the differences in that response between children and adults. Finally, child–adult differences in the acclimatization- and training-induced adaptations to thermal stress are discussed.

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