Livros: "Implantable cardiac stimulation devices"

1

Korpas, David. Implantable Cardiac Devices Technology. Boston, MA: Springer US, 2013. http://dx.doi.org/10.1007/978-1-4614-6907-0.

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2

Korpas, David. Implantable Cardiac Devices Technology. Boston, MA: Springer US, 2013.

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3

Diemberger, Igor, e Giuseppe Boriani, eds. Infections of Cardiac Implantable Devices. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-46255-0.

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4

Proietti, Riccardo, Gian Mauro Manzoni, Giada Pietrabissa e Gianluca Castelnuovo, eds. Psychological, Emotional, Social and Cognitive Aspects of Implantable Cardiac Devices. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-55721-2.

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5

Sanjeev, Saksena, e Goldschlager Nora, eds. Electrical therapy for cardiac arrhythmias: Pacing, antitachycardia devices, catheter ablation. Philadelphia: Saunders, 1990.

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6

Korpas, David. Implantable Cardiac Devices Technology. Springer, 2015.

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7

Korpas, David. Implantable Cardiac Devices Technology. Springer, 2013.

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8

Radiographic Atlas of Cardiac Implantable Electronic Devices. Elsevier, 2022. http://dx.doi.org/10.1016/c2020-0-03768-0.

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9

Ryan, James D., David L. Hayes, Siva K. Mulpuru, Nora E. Olson e Tracy L. Webster. Workbook of Diagnostics for Cardiac Implantable Devices. Cardiotext Publishing, 2020.

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10

Ryan, James D., David L. Hayes, Siva K. Mulpuru, Nora E. Olson e Tracy L. Webster. Workbook of Diagnostics for Cardiac Implantable Devices. Cardiotext Publishing, 2020.

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11

Haghjoo, Majid, Farzad Kamali e Amirfarjam Fazelifar. Radiographic Atlas of Cardiac Implantable Electronic Devices. Elsevier, 2021.

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12

Diemberger, Igor, e Giuseppe Boriani. Infections of Cardiac Implantable Devices: A Comprehensive Guide. Springer International Publishing AG, 2021.

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13

Diemberger, Igor, e Giuseppe Boriani. Infections of Cardiac Implantable Devices: A Comprehensive Guide. Springer, 2020.

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14

Morley-Smith, Andrew C., André R. Simon e John Pepper. Implanted cardiac support devices. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199687039.003.0031.

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Mechanical circulatory support forms a crucial and expanding element of advanced heart failure management. Short-term assistance is delivered in emergency situations or where the underlying condition is likely to quickly worsen, and these approaches are discussed in Chapter 30. This chapter focuses on implantable devices intended for the medium and long term. The first half of the chapter is aimed directly at practical clinical management, whilst the second half considers the evidence base for contemporary practice. The chapter concludes by considering new paradigms for implantable cardiac support. Most frequently, these devices support left ventricular function (left ventricular assist devices), and this comprises the majority of our discussion.

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15

Morley-Smith, Andrew C., André R. Simon e John Pepper. Implanted cardiac support devices. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199687039.003.0031_update_001.

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Mechanical circulatory support forms a crucial and expanding element of advanced heart failure management. Short-term assistance is delivered in emergency situations or where the underlying condition is likely to quickly worsen, and these approaches are discussed in Chapter 30. This chapter focuses on implantable devices intended for the medium and long term. The first half of the chapter is aimed directly at practical clinical management, whilst the second half considers the evidence base for contemporary practice. The chapter concludes by considering new paradigms for implantable cardiac support. Most frequently, these devices support left ventricular function (left ventricular assist devices), and this comprises the majority of our discussion.

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16

ANSI/AAMI/ISO 14117:2019; Active implantable medical devices—Electromagnetic compatibility—EMC test protocols for implantable cardiac pacemakers, implantable cardioverter defibrillators and cardiac resynchronization devices. AAMI, 2019. http://dx.doi.org/10.2345/9781570207280.

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17

Castelnuovo, Gianluca, Riccardo Proietti, Gian Mauro Manzoni e Giada Pietrabissa. Psychological, Emotional, Social and Cognitive Aspects of Implantable Cardiac Devices. Springer, 2018.

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18

Castelnuovo, Gianluca, Riccardo Proietti, Gian Mauro Manzoni e Giada Pietrabissa. Psychological, Emotional, Social and Cognitive Aspects of Implantable Cardiac Devices. Springer, 2017.

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19

Barold, S. Serge, e Philippe Ritter. Devices for Cardiac Resynchronization : : Technologic and Clinical Aspects. Springer London, Limited, 2007.

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20

(Editor), S. Serge Barold, e Philippe Ritter (Editor), eds. Devices for Cardiac Resynchronization: Technologic and Clinical Aspects. Springer, 2007.

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21

Implantable Devices: Design, Manufacturing, and Malfunction, an Issue of Cardiac Electrophysiology Clinics. Elsevier, 2014.

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22

Contemporary Issues in Patients with Implantable Devices, an Issue of Cardiac Electrophysiology Clinics. Elsevier - Health Sciences Division, 2018.

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23

Cardiac Pacing for the Clinician. Lippincott Williams & Wilkins, 2001.

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24

Rajappan, Kim. Cardiac device therapy. Editado por Patrick Davey e David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0121.

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The term ‘device therapy’ is used in cardiology to refer to three different types of implantable cardiac-rhythm-management devices: pacemakers, implantable cardioverter defibrillators (also known as ICDs), and cardiac resynchronization therapy devices (also known as CRT devices). There has been a steady increase in the number of patients receiving these cardiac devices; in relation to CRT devices, the increase has been almost exponential.

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25

Goldschlager, Nora, e Sanjeev Saksena. Electrical Therapy for Cardiac Arrhythmias: Pacing, Antitachycardia Devices, Catheter Ablation. Saunders, 1989.

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26

Friedman, Paul A., e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Incorporated, John, 2009.

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27

Friedman, Paul A., Samuel J. Asirvatham e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Limited, John, 2020.

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28

Friedman, Paul A., Samuel J. Asirvatham e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Incorporated, John, 2012.

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29

Friedman, Paul A., e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Incorporated, John, 2011.

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30

Friedman, Paul A., Samuel J. Asirvatham e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Incorporated, John, 2020.

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31

Friedman, Paul A., Samuel J. Asirvatham e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Incorporated, John, 2020.

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32

Friedman, Paul A., Samuel J. Asirvatham e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Incorporated, John, 2012.

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33

Friedman, Paul A., e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Incorporated, John, 2011.

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34

Friedman, Paul A., Samuel J. Asirvatham e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Incorporated, John, 2012.

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35

Friedman, Paul A., Samuel J. Asirvatham e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Limited, John, 2012.

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36

Friedman, Paul A., Samuel J. Asirvatham e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Incorporated, John, 2021.

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37

Friedman, Paul A., e David L. Hayes. Cardiac Pacing, Defibrillation and Resynchronization: A Clinical Approach. Wiley & Sons, Limited, John, 2009.

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38

Brusch, John L. Infective Endocarditis: Management in the Era of Intravascular Devices. Taylor & Francis Group, 2007.

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39

Infective Endocarditis: Management in the Era of Intravascular Devices (Infectious Disease and Therapy). Informa Healthcare, 2007.

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40

Brusch, John L. Infective Endocarditis: Management in the Era of Intravascular Devices. Taylor & Francis Group, 2007.

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41

Device therapy in heart failure. New York, NY: Humana Press, 2010.

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42

Katritsis, Demosthenes G., Bernard J. Gersh e A. John Camm. Technical issues. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.1571_update_004.

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Implantable devices, such as permanent pacemakers, implantable cardioverter defibrillators, and biventricular pacemakers, are discussed. This chapter presents the indications for selection of pacemaker mode and instructions for troubleshooting. Indications for permanent pacing or ICD therapy are covered in other relevant chapters, such as those on bradyarrhythmias, cardiac failure, and tachyarrhythmias.

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43

Thorne, Sara, e Sarah Bowater. Device therapy in ACHD. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198759959.003.0020.

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This chapter discusses device therapy in ACHD. Insertion of devices in these patients requires special consideration. Knowledge of underlying anatomy, previous surgical procedures, and vascular access routes; an understanding of problems likely to be encountered with lead placement in complex hearts; and obtaining a stable position are all crucial. This chapter discusses bradycardia pacing, implantable cardiac defibrillators (ICD), cardiac resynchronization therapy (CRT), and technical considerations for device therapy in ACHD.

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44

Katritsis, Demosthenes G., Bernard J. Gersh e A. John Camm. Chronic heart failure. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.0754_update_004.

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The diagnosis and management of chronic heart failure are discussed. Medical therapy and indications for cardiac resynchronization therapy (CRT), implantable cardioverter-defibrillators (ICD), left ventricular assist devices (LVAD), and transplantation are presented. Recommendations by the ACC/AHA and ESC on the management of patients with heart failure have been summarized and tabulated.

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45

Fogelman, Patricia Maani, e Janine A. Gerringer. Withdrawal of Cardiology Technology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190204709.003.0011.

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The care of the cardiac patient requires exquisite assessment including history, physical examinations, and diagnostic data in order to make differential diagnoses and formulate individualized treatment plans. Interventions include education about lifestyle modifications, the introduction and titration of cardiac medications, and referral for more advanced treatments such as vasoactive or inotropic medications, cardiovascular implantable electronic devices, and ventricular assist devices. Often, patients decide to discontinue these therapies. Standardized protocols for withdrawal of life-sustaining respiratory therapies provide structured guidance, reduce variation in practice, and improve satisfaction of families and healthcare providers. This chapter reviews such therapies and the process for cessation while simultaneously attending to symptom management.

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46

Drake, Sarah, e Jonathan Sandoe. Fungal cardiovascular infections. Editado por Christopher C. Kibbler, Richard Barton, Neil A. R. Gow, Susan Howell, Donna M. MacCallum e Rohini J. Manuel. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198755388.003.0021.

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Fungal cardiovascular disease can broadly be divided into four groups: infective endocarditis (including implantable cardiac electronic devices), mycotic aneurysms, vascular graft infections, and intravascular catheter-related infections. These conditions are rare but are associated with significant morbidity and mortality, which may be in excess of 80% in certain groups of patients. Candida spp. and Aspergillus spp. account for the majority of these infections, but rare fungi may also be involved, particularly in infective endocarditis, where they are responsible for approximately 25% of cases. This chapter will cover the epidemiology, causative fungi, clinical features, diagnosis, management, and prevention of these four fungal cardiovascular conditions.

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47

Burri, Haran, Carsten Israel e Jean-Claude Deharo, eds. The EHRA Book of Pacemaker, ICD, and CRT Troubleshooting. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780198727774.001.0001.

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The EHRA Book of Pacemaker, ICD and CRT Troubleshooting assists device specialists in tackling both common and unusual situations that that they may encounter during daily practice. Taking a case-based approach, it examines pacemakers, implantable cardioverter defibrillators and cardiac resynchronisation therapy. Much more than just a technical manual of device algorithms, the cases help readers to consolidate their technical knowledge, and improve their reasoning and observation skills so they are able to tackle device troubleshooting with confidence. The 70 cases are arranged in three sections by increasing levels of difficulty to walk readers through all the skills and knowledge they need in an easy to use and structured format. Each case contains a short clinical description and a device tracing followed by a multiple choice question. Answers are supplied with detailed annotations of the tracing and an in-depth discussion of the case, highlighting practical hints and tips as well as providing an overview of the technical function of devices. A useful summary of principal device features and functions is also included.

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48

Selim, Bernardo, e Kannan Ramar. Beyond positive airway pressure therapy: experimental and non-conventional treatments in sleep apnoea. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0259.

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With decreased adherence to positive airway pressure therapy to treat sleep apnoeas, non-conventional treatments based on new therapeutic targets are emerging. In central sleep apnoea syndrome associated with heart failure, phrenic nerve stimulation and non-conventional pharmacological treatments such as carbonic anhydrase inhibitors, gas therapies, and cardiac devices are novel alternative therapies. In obstructive sleep apnoea, a better understanding of predominant pathophysiological pathways is characterizing diverse clinical phenotypes. For patients with low arousal threshold, sedatives or hypnotics might be effective, whereas for those with unstable ventilatory control, carbonic anhydrase inhibitors or oxygen might improve obstructive sleep apnoea. For patients with upper airway muscle dysfunction, an increase in pharyngeal tone might be beneficial. This chapter describes ‘experimental’ therapies and novel technologies to treat these disorders.

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49

Zoccali, Carmine, Davide Bolignano e Francesca Mallamaci. Left ventricular hypertrophy in chronic kidney disease. Editado por David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0107_update_001.

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Alterations in left ventricular (LV) mass and geometry and LV dysfunction increase in prevalence from stage 2 to stage 5 in CKD. Nuclear magnetic resonance is the most accurate and precise technique for measuring LV mass and function in patients with heart disease. Quantitative echocardiography is still the most frequently used means of evaluating abnormalities in LV mass and function in CKD. Anatomically, myocardial hypertrophy can be classified as concentric or eccentric. In concentric hypertrophy, the muscular component of the LV (LV wall) predominates over the cavity component (LV volume). Due to the higher thickness and myocardial fibrosis in patients with concentric LVH, ventricular compliance is reduced and the end-diastolic volume is small and insufficient to maintain cardiac output under varying physiological demands (diastolic dysfunction). In those with eccentric hypertrophy, tensile stress elongates myocardiocytes and increases LV end-diastolic volume. The LV walls are relatively thinner and with reduced ability to contract (systolic dysfunction). LVH prevalence increases stepwisely as renal function deteriorates and 70–80% of patients with kidney failure present with established LVH which is of the concentric type in the majority. Volume overload and severe anaemia are, on the other hand, the major drivers of eccentric LVH. Even though LVH may regress after renal transplantation, the prevalence of LVH after transplantation remains close to that found in dialysis patients and a functioning renal graft should not be seen as a guarantee of LVH regression. The vast majority of studies on cardiomyopathy in CKD are observational in nature and the number of controlled clinical trials in these patients is very small. Beta-blockers (carvedilol) and angiotensin receptors blockers improve LV performance and reduce mortality in kidney failure patients with LV dysfunction. Although current guidelines recommend implantable cardioverter-defibrillators in patients with ejection fraction less than 30%, mild to moderate symptoms of heart failure, and a life expectancy of more than 1 year, these devices are rarely offered to eligible CKD patients. Conversion to nocturnal dialysis and to frequent dialysis schedules produces a marked improvement in LVH in patients on dialysis. More frequent and/or longer dialysis are recommended in dialysis patients with asymptomatic or symptomatic LV disorders if the organizational and financial resources are available.

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