Bibliografias temáticas / Idenitification

Literatura científica selecionada sobre o tema "Idenitification"

Autor: Grafiati
Publicado: 25 de julho de 2024
Última modificação: 26 de julho de 2024

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Artigos de revistas sobre o assunto "Idenitification"

1

Young, P. C., W. Tych e A. Chotai. "Idenitification, estimation and control of glasshouse systems". IFAC Proceedings Volumes 24, n.º 11 (setembro de 1991): 307–15. http://dx.doi.org/10.1016/b978-0-08-041273-3.50059-8.

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Tischler, Mark B., Joseph G. M. Leung e Daniel C. Dugan. "Frequency‐Domain Idenitification of XV‐15 Tilt‐Rotor Aircrart Dynamics in Hovering Flight". Journal of the American Helicopter Society 30, n.º 2 (1 de abril de 1985): 38–48. http://dx.doi.org/10.4050/jahs.30.38.

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SUEHIRO, Tadashi, Kenzo YOSHIDA, Toshinao YAMANO e Fumitoshi OHNO. "Idenitification and characterization of a new variant of apolipoprotein E (apo E-Kochi)." Japanese Journal of Medicine 29, n.º 6 (1990): 587–94. http://dx.doi.org/10.2169/internalmedicine1962.29.587.

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4

Florian, Michael K., Michael D. Gladders, Nan Li e Keren Sharon. "THE GINI COEFFICIENT AS A TOOL FOR IMAGE FAMILY IDENITIFICATION IN STRONG LENSING SYSTEMS WITH MULTIPLE IMAGES". Astrophysical Journal 816, n.º 2 (12 de janeiro de 2016): L23. http://dx.doi.org/10.3847/2041-8205/816/2/l23.

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Kouzy, Ramez, Daniel Lin, Sonal Suresh Noticewala, Lauren Elizabeth Colbert e Cullen M. Taniguchi. "Enhanced microbial diversity and chemoradiation response in HPV+ anal cancer." Journal of Clinical Oncology 38, n.º 4_suppl (1 de fevereiro de 2020): 4. http://dx.doi.org/10.1200/jco.2020.38.4_suppl.4.

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4 Background: Anal cancer is a rare, but treatable, malignancy caused by the human papilloma virus (HPV). Because anal cancer is uncommon and carries a significant social stigma, this disease has rarely been studied in a prospective fashion, creating a large knowledge gap of which biological factors can improve treatment responses, particularly the microbiome which regulates many facets of oncobiology. Methods: We collected microbiome samples before, during, and after definitive chemoradiation for localized anal cancer as part of an IRB-approved institutional clinical trial (2017-0606). Samples were collected by a non-invasive swab biopsy that enabled collection of tumor cells and microbiome during standard-of-care treatment visits. Bacterial genomic DNA is extracted using MO BIO PowerSoil DNA Isolation Kit (MO BIO Laboratories) and the 16S rDNA V4 region is amplified by PCR and sequenced on the MiSeq platform (Illumina) using the 2x250 bp paired-end protocol, yielding pair-end reads that overlapped almost completely. Sequence read pairs are demultiplexed using unique molecular barcodes, and reads are merged using USEARCH version 7.0.1090 and grouped by organizational taxonomy units (OTU’s) representative of unique bacterial species. Responders are were those patients who had more than 60% tumor shrinkage by MRI at the midpoint of treatment, and all others were considered non-responders. Results: As of June 2019 we enrolled 17 patients and analyzed 14. The majority of patients (10/14, 71.4%) were responders but unfortunately, 4 patients developed in-field recurrences of their anal cancer within a year after their treatment ended and were all T3 or T4 tumors. We found that alpha-diversity of the microbiome did not change during chemoradiation, but non-responders exhibited a lower alpha diversity compared to responders at baseline. Furthermore, specific taxa were correlated with treatment response, which imply a basis for future therapy. Conclusions: The alpha diversity of the microbiome is correlated with treatment response in HPV+ anal cancer. The idenitification of specific bacterial species between responders and non-responder might pave the way for more effective therapeis in the future.
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Franco, Theresa, Susan Schreiner, Kerry J. Rodabaugh e Susan Stensland. "Establishing a medical-legal partnership program for cancer patients." Journal of Clinical Oncology 31, n.º 31_suppl (1 de novembro de 2013): 281. http://dx.doi.org/10.1200/jco.2013.31.31_suppl.281.

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281 Background: Treating cancer patients requires a comprehensive approach extending beyond their medical needs. Patients may have issues with legal implications, which, if not addressed, hinder ones’ ability to focus on their care. The objective in establishing a medical legal partnership program is to create a mechanism for identifying such issues and developing plans for resolution. Brought to the organization’s attention by a newly recruited gynecological oncologist, a collaborative, interdisciplinary program was developed that provides resources necessary to address legal problems such as custody, employment rights, and disability. This reduces the burden on patients and families and improves the quality of life during and after cancer treatment. Methods: The Medical Legal Partnership Program was established under the guidance of a gynecologic oncologist cancer service line administrator, social work department and representatives from legal Aid of Nebraska and Iowa Legal Aid. A memorandum of understanding was signed and the team crafted a process to provide support. This included determining issues in which legal counsel is critical, appropriate patient idenitification, communication strategies, and follow up support. Results: Results include: since the inception of the program in FY 2010 to FY 2013 (to date), 271 patients have been served; a variety of legal issues have been resolved including custody designation, receipt of appropriate benefits, development of wills, and identification of end of life issues; at program inception, one attorney worked one day/ week with a part-time paralegal; in FY 2013, assistance has grown to 20 hours/ week of attorney coverage and a full-time paralegal; and over $250,000 health care dollars have been recovered. Conclusions: Development of a medical legal partnership program has resulted in addressing legal needs that arise during a cancer patient’s course of treatment, reducing their burden to find assistance. It provides an opportunity to recover health care dollars from denied/ unpaid sources and enhances the health care team’s understanding of the important role legal support plays in cancer care.
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Teses / dissertações sobre o assunto "Idenitification"

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Drezet, Pierre M. L. "Kernel methods and their application to systems idenitification and signal processing". Thesis, University of Sheffield, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.247172.

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Shy, Ru-Shan, e 施如珊. "The mutantion studies of ClpY I domain and idenitification of its function for specific substrates recognition". Thesis, 2004. http://ndltd.ncl.edu.tw/handle/81367549827993963978.

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碩士
國立臺灣大學
農業化學研究所
92
Recognition of the appropriate molecular targets is critical for most biological activity in the cells. For irreversible processes such as protein degradation, recognizing of the correct substrates are important because cleavage of the wrong targets might be damaging to the cells or even lethal. It is required for cells to degrade proper targets like misfolded or abnormal proteins and to control the levels of critical short-lived regulatory proteins. The ATP-dependent proteases play a role in phenomena described above. In bacteria and other organisms, many of intracellular proteases have to hydrolyze ATP to degrade complex substrates；including peptide enzymes, such as Lon, and two-component protease, such as ClpXP, ClpQY, ClpAP. In ClpQY, the small subunit ClpQ (19kDa) is a peptidase, and the larger subunit ClpY (49kDa) exhibits both ATPase and chaperone activities. ClpY can recognize, unfold, and translocate the specific substrates. It is unclear about the mechanisms of how the ClpY recognizes, binds and translocates the specific substrates to ClpQ and the ClpQY degrades the substrates. In this study, the yeast two-hybrid system was used to screen the ClpY mutants either interact or not with the specific substrates. To test the abilities of interaction of ClpY mutants with substrates, lacZ and leu2 expressions was used in yeast system；cps-lacZ expression was used to detect the RcsA degraded by the ClpY mutants in the presence of ClpQ, MMS test was used to detect the SulA degradation.In addition, the ClpYmutants were tested of their influences on cell growth while overproducted.We show that the loop of I domain(175-209) is necessary for substrate recognition and the altered specific amino acid residues on the loop have an influence on ClpY cellular activities.
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Livros sobre o assunto "Idenitification"

1

Mikolajski, Andrew. Orchids. Paris: Marabout, 2006.

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Loss Prevention: Hazard Idenitification, Assessment and Control. 2a ed. Butterworth-Heinemann, 1996.

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3

Lees, Frank. Loss Prevention: Hazard Idenitification, Assessment and Control. Elsevier Science & Technology Books, 1996.

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4

Mikolajski, Andrew. Orchids: An Illustrated Guide to Varieties, Cultivation and Care. Anness Publishing, 2015.

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