Literatura científica selecionada sobre o tema "Hypercholesteremia"
Crie uma referência precisa em APA, MLA, Chicago, Harvard, e outros estilos
Consulte a lista de atuais artigos, livros, teses, anais de congressos e outras fontes científicas relevantes para o tema "Hypercholesteremia".
Ao lado de cada fonte na lista de referências, há um botão "Adicionar à bibliografia". Clique e geraremos automaticamente a citação bibliográfica do trabalho escolhido no estilo de citação de que você precisa: APA, MLA, Harvard, Chicago, Vancouver, etc.
Você também pode baixar o texto completo da publicação científica em formato .pdf e ler o resumo do trabalho online se estiver presente nos metadados.
Artigos de revistas sobre o assunto "Hypercholesteremia"
Visavadiya, Nishant P., e A. V. R. L. Narasimhacharya. "AsparagusRoot Regulates Cholesterol Metabolism and Improves Antioxidant Status in Hypercholesteremic Rats". Evidence-Based Complementary and Alternative Medicine 6, n.º 2 (2009): 219–26. http://dx.doi.org/10.1093/ecam/nem091.
Texto completo da fonteKhan, Safi U., e Khurram Nasir. "Hypercholesteremia in Indigenous Population". JACC: Advances 2, n.º 3 (maio de 2023): 100352. http://dx.doi.org/10.1016/j.jacadv.2023.100352.
Texto completo da fonteNordin, Jenizan, e Ahmad Badruddin Ghazali. "Osteonecrosis of the jaw in patients taking Atorvastatin: Case series". IIUM Journal of Orofacial and Health Sciences 3, n.º 2 (30 de julho de 2022): 219–22. http://dx.doi.org/10.31436/ijohs.v3i2.73.
Texto completo da fonteKamalova, Aelita A. "Diet Therapy in Children with Hypercholesteremia". Current Pediatrics 19, n.º 4 (19 de setembro de 2020): 309–15. http://dx.doi.org/10.15690/vsp.v19i4.2140.
Texto completo da fonteDietrich, Hans H. "Apolipoprotein E in Hypercholesteremia and Beyond". Stroke 38, n.º 7 (julho de 2007): 2036. http://dx.doi.org/10.1161/strokeaha.107.489856.
Texto completo da fonteLEWIS, S. "P514 Hypercholesteremia induces baroreceptro reflex dysfunction". European Heart Journal 24, n.º 5 (março de 2003): 81. http://dx.doi.org/10.1016/s0195-668x(03)93952-2.
Texto completo da fonteNAKAI, TSUGUHIKO. "Familial hypercholesteremia complicated by nephrotic syndrome." Nihon Naika Gakkai Zasshi 79, n.º 8 (1990): 1079–80. http://dx.doi.org/10.2169/naika.79.1079.
Texto completo da fonteHURLEY, D. "Hypercholesteremia induces enhanced circulating inflammatory activity". American Journal of Hypertension 17, n.º 5 (maio de 2004): S242. http://dx.doi.org/10.1016/j.amjhyper.2004.03.648.
Texto completo da fonteLi, Nan F., Hong M. Wang, Jin Yang, Ling Zhou, Xiao G. Yao e Jing Hong. "Serum uric acid is associated with metabolic risk factors for cardiovascular disease in the Uygur population". Applied Physiology, Nutrition, and Metabolism 34, n.º 6 (dezembro de 2009): 1032–39. http://dx.doi.org/10.1139/h09-101.
Texto completo da fonteKawsar, Dr Syeda Masuma, e Dr Amirul Islam Bhuyan. "Outcome of Heart Rate and Blood Pressure of a Patient after Successful Coronary Angioplasty and Stenting". Scholars Journal of Applied Medical Sciences 11, n.º 03 (30 de março de 2022): 668–71. http://dx.doi.org/10.36347/sjams.2023.v11i03.031.
Texto completo da fonteTeses / dissertações sobre o assunto "Hypercholesteremia"
Staples, Heidi. "The effect of lipid-lowering pharmacotherapy on concurrent diet and exercise behaviors /". Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=31543.
Texto completo da fonteIt was therefore hypothesized that pharmacologically-treated patients with untreated hypercholesterolemia started on a program of lifestyle modification would achieve relatively less reduction in dietary fat intake and body weight, and participate less often in physical activity, if a pharmacologic agent was simultaneously prescribed. This was tested by a protocol in which these and related variables were assessed in participants who thought they were taking a lipid-lowering medication at diagnosis, compared to conventional initial treatment of diet and exercise alone. (Abstract shortened by UMI.)
Collins, Melissa. "Effects of plant sterols and exercise training on apolipoprotein A and B, adiponectin, growth hormone and ghrelin in hypercholesterolemic sedentary adults". Thesis, McGill University, 2006. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=99331.
Texto completo da fonteVergotine, Joseph Vincent. "The MED-PED project : presymptomatic diagnosis in families with disease- related LDL receptor gene mutations". Thesis, Stellenbosch : Stellenbosch University, 2000. http://hdl.handle.net/10019.1/51997.
Texto completo da fonteENGLISH ABSTRACT: Familial hypercholesterolaemia (FH) contributes significantly to the high death rate from cardiovascular disease worldwide. FH is a common autosomal co-dominant disease characterised by raised cholesterol levels and premature coronary heart disease (CHD). Whilst these features usually are very prominent in homozygotes the clinical diagnosis of heterozygotes is complicated by variable phenotypic expression. Specific founder genes in the low-density lipoprotein receptor (LDLR) gene have increased the prevalence of FH in South African Afrikaners, Indians, Jews and Coloureds, and screening for these known mutations allows unequivocal diagnosis of FH-affected individuals. The systematic molecular analysis of FH resulted in the identification of at least ten founder-type LDLR gene mutations among the 56 different gene defects described to date in the diverse South African population. DNA screening of 792 at-risk family members for the FH-related mutations identified in 379 index cases, allowed accurate disease diagnosis in an additional 340 relatives and exclusion of the relevant mutation in 452 individuals. This effort forms part of the MED PED FH initiative, a collaborative project to "Make Early Diagnosis and Prevent Early Deaths in MEDical PEDigrees with FH". Evaluation of clinical criteria versus DNA diagnosis of three founder-related mutations (D154N, D206E and V408M) in the South African population demonstrated that the sensitivity and specificity of diagnoses, based on total cholesterol values measured in family members of index cases recruited for this study, were 88% and 77%, respectively. A population-directed DNA diagnosis of FH is therefore justified in South Africa on a routine basis, since expression of the defective gene measured in biochemical tests does not allow accurate diagnosis of FH in all cases. The application of mutation detection was illustrated by prenatal diagnosis of FH performed for a couple who are both heterozygous for the most common Afrikaner mutation, D206E. The mutation was absent in the foetus and a normocholesterolaemic infant was born. Prenatal diagnosis of FH, aimed at the detection of homozygous cases, is particularly applicable in populations and families with molecularly defined LDLR gene mutations. The MED-PED approach resulted in accurate diagnosis and subsequent treatment of FH in more patients, and referral to lipid clinics where they could receive the intensive care their condition justifies. Molecularly diagnosed FH patients will be the first to benefit from future treatment approaches based on mutation type.
AFRIKAANSE OPSOMMING: Familiële hiprcholesterolemie dra grootliks by tot die wêreldwye hoë sterftesyfer van kardiovaskulêre siekte. FH is 'n algemene outosomale ko-dominante siekte wat gekenmerk word deur verhoogde cholesterolvlakke en vroeë koronêre hartsiekte. Terwyl hierdie kenmerke prominent is in homosigote, word die kliniese diagnose van heterosigote bemoeilik deur variasie in fenotipiese uitdrukking. Spesifieke stigtergene in die lae-digtheids lipoproteien reseptor (LDLR) geen het die voorkomssyfer van FH verhoog in Suid Afrikaanse Afrikaners, Indiërs, Jode en Kleurlinge. Sifting vir hierdie bekende mutasies maak akkurate diagnose van FH geaffekteerde individue moontlik. Die sistematiese molekulêre analise van FH het aangetoon dat ten minste tien van die 56 verskillende geen defekte wat tot dusver beskryf is in die Suid-Afrikaanse populasie stigtertipe LDLR geen mutasies is. DNA sifting van 792 familielede vir die FH-verwante mutasie in 379 indeksgevalle geïdentifiseer is, het akkurate diagnose moontlik gemaak in 340 addisionele familielede, en uitsluiting daarvan in 452 individue. Hierdie poging vorm deel van die MED-PED FH ("Make Early Diagnosis and Prevent Early Deaths in MEDical PEDigrees with FH) inisiatief. Evaluering van kliniese kriteria teenoor DNA diagnose van drie stigter verwante mutasies (D154N, D206E en V408M) in die Suid Afrikaanse populasie het getoon dat die sensitiwiteit en spesifisiteit van die diagnose, wat gebasseer is op totale cholesterol waardes in familielede van indeksgevalle, onderskeidelik 88% en 77% was. 'n Populasie gerigte DNA diagnose van FH is dus geregverdig in Suid-Afrika op "n roetine basis, omdat die defektiewe geen nie altyd in biochemiese toetse uitgedruk word nie. Die waarde van mutasie opsporing is geillustreer deur 'n voorgeboortelike diagnose van FH wat aangevra is vir ouers wat beide heterosigoties is vir die mees algemene Afrikaner mutasie, D206E. Die mutasie was afwesig in die fetus en 'n normocholesterolemiese baba is gebore. Voorgeboortelike diagnose van FH, wat gemik is op die opsporing van homosigotiese gevalle, is veral van toepassing in populasies en families met bekende LDLR geen mutasies. Die MED-PED benadering het gelei tot akkurate diagnose en daaropvolgende behandeling van FH in meer pasiënte, en verwysings na lipiedklinieke waar hulle intensiewe aandag kan geniet. Molekulêre gediagnoseerde FH pasiënte sal die eerste wees om baat te vind by toekomstige behandeling wat moontlik gebasseer sal word op mutasie status.
Wong, Kwok-kit Sunny. "A study of DNA mutations in LDL receptor gene of Chinese patients with familial hypercholesterolaemia /". Hong Kong : University of Hong Kong, 1997. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19109878.
Texto completo da fonteSt-Onge, Marie-Pierre. "Effect of kefir supplementation on blood lipid parameters in free-living hypercholesterolemic men". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape9/PQDD_0024/MQ50885.pdf.
Texto completo da fonteMiller, Stephanie M. "The Relationship of Waist Size to Blood Pressure and Cholesterol Among College Students". Lynchburg, Va. : Liberty University, 2007. http://digitalcommons.liberty.edu.
Texto completo da fonteYang, Tat-chi Teddy. "Effect of Chinese green tea on diet-induced hypercholesterolemia and arteriosclerosis in rats /". Hong Kong : University of Hong Kong, 1998. http://sunzi.lib.hku.hk/hkuto/record.jsp?B19657560.
Texto completo da fonteNeubauer, Tamara E. "Cholesterol reduction in men : an experimental investigation of intensive treatment with frequent feedback versus a simple educational treatment /". Thesis, This resource online, 1990. http://scholar.lib.vt.edu/theses/available/etd-03122009-040807/.
Texto completo da fonteMatthan, Nirupa Rachel. "Impact of hydrogenated fat consumption on in vivo lipid metabolism in moderately hypercholesterolemic women". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape4/PQDD_0033/NQ64617.pdf.
Texto completo da fonteFourie, Anne Madeleine. "The expression and metabolism of low density lipoprotein receptors in familial hypercholesterolaemia". Doctoral thesis, University of Cape Town, 1989. http://hdl.handle.net/11427/27174.
Texto completo da fonteLivros sobre o assunto "Hypercholesteremia"
National Cholesterol Education Program (U.S.). Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults., ed. Report of the Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults. [Bethesda, Md.]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1989.
Encontre o texto completo da fonteHuang, Zhaosheng. Fei pang bing zhi fang gan yu gao zhi xue zheng Zhong Xi yi zhen liao yu tiao yang =: Obesity fat liver and high-lipidemia. Xianggang: Tian heng wen hua chu ban gong si, 2001.
Encontre o texto completo da fonteNational, Cholesterol Education Program (U S. ). Expert Panel on Detection Evaluation and Treatment of High Blood Cholesterol in Adults. Third report of the National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (adult treatment panel III): Final report. [Washington, D.C.]: The Program, 2002.
Encontre o texto completo da fonteMcCord, Holly. Win the cholesterol war: 100 real-life secrets to trimming points (and pounds). New York: Berkley Books, 2003.
Encontre o texto completo da fonteWin the cholesterol war: 100 real-life secrets to trimming points (and pounds). New York: Berkley Books, 2003.
Encontre o texto completo da fonteNational Cholesterol Education Program (U.S.). Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults. Second report of the Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (adult treatment panel II). [Bethesda, Md.?]: National Cholesterol Education Program, National Institute of Health, National Heart, Lung, and Blood Institute, 1993.
Encontre o texto completo da fonteNational Cholesterol Education Program (U.S.), ed. Third report of the National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (adult treatment panel III): Executive summary. [Bethesda, Md.]: National Cholesterol Education Program, National Heart, Lung, and Blood Institute, National Institutes of Health, 2001.
Encontre o texto completo da fonteMcCord, Holly. Win the cholesterol war: 100 real-life secrets to trimming points (and pounds). New York: Berkley Books, 2003.
Encontre o texto completo da fonteWetherill, Douglas. High cholesterol. Cincinnati, OH: Betterway Books, 2000.
Encontre o texto completo da fonteR, Gordon Bruce, e Gotto Antonio M, eds. The Treatment of severe hypercholesterolemia-- can we impact disease course?: Proceedings of a symposium, November 9, 1991, Anaheim, California. Princeton, NJ: Excerpta Medica, 1992.
Encontre o texto completo da fonteCapítulos de livros sobre o assunto "Hypercholesteremia"
Loria, Roger M. "Effect of Dietary Hypercholesteremia on Host Immune Response". In Nutrients, Stress, and Medical Disorders, 267–78. Totowa, NJ: Humana Press, 2006. http://dx.doi.org/10.1385/1-59259-952-4:267.
Texto completo da fonteHunninghake, D. B., D. M. Hibbard, W. C. Duane, M. L. Freeman, W. F. Prigge, K. J. Graham e R. L. Gebhard. "Metabolic Studies with Lovastatin in Patients with Primary Hypercholesteremia". In Drugs Affecting Lipid Metabolism, 150–54. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71702-4_27.
Texto completo da fonteWisnu Murti, Tridjoko. "Fermentation of Bovine, Non-Bovine and Vegetable Milk". In Fermentation - Processes, Benefits and Risks. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96699.
Texto completo da fonte