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Livros sobre o tema "High-Risk for psychosis"

Autor: Grafiati

Publicado: 29 de março de 2025

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1

Addington, Jean, Shona M. Francey e Anthony P. Morrison, eds. Working with People at High Risk of Developing Psychosis. Chichester, UK: John Wiley & Sons, Ltd, 2005. http://dx.doi.org/10.1002/9780470712979.

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2

Jean, Addington, Francey Shona M e Morrison Anthony P. 1969-, eds. Working with people at high risk of developing psychosis: A treatment handbook. Chichester, England: Wiley, 2006.

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3

French, Paul, e Anthony P. Morrison, eds. Early Detection and Cognitive Therapy for People at High Risk of Developing Psychosis. West Sussex, London: John Wiley & Sons, Ltd, 2004. http://dx.doi.org/10.1002/9780470713259.

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4

Morrison, Anthony P., Jean Addington e Shona Francey. Working with People at High Risk of Developing Psychosis. Wiley & Sons, Incorporated, John, 2006.

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5

Working with People at High Risk of Developing Psychosis. New York: John Wiley & Sons, Ltd., 2006.

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6

Morrison, Anthony P., Jean Addington e Shona Francey. Working with People at High Risk of Developing Psychosis: A Treatment Handbook. Wiley & Sons, Incorporated, John, 2010.

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7

Morrison, Anthony P., Shona Francey e J. Addington. Working with People at High Risk of Developing Psychosis: A Treatment Handbook. Wiley & Sons, Limited, John, 2006.

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8

(Editor), Jean Addington, Shona Francey (Editor) e Anthony P. Morrison (Editor), eds. Working with People at High Risk of Developing Psychosis: A Treatment Handbook. Wiley, 2006.

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9

(Editor), Jean Addington, Shona Francey (Editor) e Anthony P. Morrison (Editor), eds. Working with People at High Risk of Developing Psychosis: A Treatment Handbook. Wiley, 2007.

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10

French, Paul, e Anthony P. Morrison. Early Detection and Cognitive Therapy for People at High Risk of Developing Psychosis. Wiley & Sons, Incorporated, John, 2004.

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11

Zhang, Tianhong, Jijun Wang e Kristen Woodberry, eds. Identifying Individuals at Clinical High Risk of Psychosis in Different Cultures and Countries. Frontiers Media SA, 2020. http://dx.doi.org/10.3389/978-2-88963-652-5.

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12

Muth, Anne-Katrin. Neural Activation During Emotion Regulation in People with Ultra-High Risk for Psychosis. GRIN Verlag GmbH, 2019.

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13

National Academies of Sciences, Engineering, and Medicine. Early Interventions for Psychosis : First Episodes and High-Risk Populations: Proceedings of a Workshop. National Academies Press, 2023.

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14

National Academies of Sciences, Engineering, and Medicine. Early Interventions for Psychosis : First Episodes and High-Risk Populations: Proceedings of a Workshop. National Academies Press, 2023.

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15

National Academies of Sciences, Engineering, and Medicine. Early Interventions for Psychosis : First Episodes and High Risk Populations: Proceedings of a Workshop. National Academies Press, 2023.

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16

Shahzad, Sidra. Propagation of Hippocampal Activity in Ultra-High Risk Schizophrenia Psychosis Subjects Using Independent Component Analysis. GRIN Verlag GmbH, 2018.

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17

French, Paul, e Anthony P. Morrison. Early Detection and Cognitive Therapy for People at High Risk of Developing Psychosis: A Treatment Approach. Wiley & Sons, Incorporated, John, 2007.

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18

French, Paul, e Anthony P. Morrison. Early Detection and Cognitive Therapy for People at High Risk of Developing Psychosis: A Treatment Approach. Wiley & Sons, Incorporated, John, 2004.

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19

French, Paul, e Anthony P. Morrison. Early Detection and Cognitive Therapy for People at High Risk of Developing Psychosis: A Treatment Approach. Wiley & Sons, Limited, John, 2008.

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20

French, Paul, e Anthony P. Morrison. Early Detection and Cognitive Therapy for People at High Risk of Developing Psychosis: A Treatment Approach. Wiley, 2004.

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21

French, Paul, e Anthony P. Morrison. Early Detection and Cognitive Therapy for People at High Risk of Developing Psychosis: A Treatment Approach. Wiley, 2004.

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22

Gupta, Nikhil, e Vinod H. Srihari. North American Prodrome Longitudinal Study. Editado por Ish P. Bhalla, Rajesh R. Tampi, Vinod H. Srihari e Michael E. Hochman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190625085.003.0045.

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This chapter provides a summary of a landmark study on schizophrenia. The question studied was “In patients identified clinically to be at high risk for psychosis, which variables (or their combinations) best predict conversion to schizophrenia or another psychotic disorder?” Starting with that question, it describes the basics of the study, including funding, study location, who was studied, how many patients, study design, study intervention, follow-up, endpoints, results, and criticism and limitations. This study demonstrates that presence of some characteristics can better prognosticate conversion of a prodromal state to a psychotic disorder. Finally, the chapter briefly reviews other relevant studies and information, discusses implications, and concludes with a relevant clinical case.

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23

Bjørk, Marte Helene, e Malin Eberhard-Gran. Perinatal Depression in Neurological Disease and Disability. Editado por Emma Ciafaloni, Cheryl Bushnell e Loralei L. Thornburg. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190667351.003.0034.

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Women and men with neurological disease more often suffer from depression in relation to pregnancy and delivery than other parents. Perinatal depression may harm the parent-child relationship as well as the health of the child. Postnatal psychosis, suicide, and infanticide are rare but severe consequences of the disorder. Symptoms of perinatal depression may overlap with symptoms of neurological disease. Both disorders may aggravate each other. Side effects from neurological treatment could mimic symptoms of depression, and antidepressive drugs could worsen neurological symptoms and interact with other treatment. Neurological patients should be evaluated for risk factors for perinatal depression before delivery. These include previous psychiatric disease, sexual or psychical abuse, sleep problems, high neurological disease activity, and low social support. Pregnant women with previous psychotic episodes or bipolar disease should be referred for psychiatric evaluation before delivery. All patients should be screened for depressive symptoms during follow-up using a 3-step method.

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24

Cavanna, Andrea E. Vigabatrin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198791577.003.0015.

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Vigabatrin is a second-generation antiepileptic drug characterized by a few antiepileptic indications, with a very good interaction profile in polytherapy. Clinically relevant adverse effects (especially visual field defects) have reduced its use as antiepileptic drug in routine clinical practice considerably. Vigabatrin has an acceptable behavioural tolerability profile, although patients with epilepsy treated with this medication can report depression, psychosis, and irritability. Risk factors for developing psychiatric adverse effects include high starting and maintenance doses, past psychiatric history, and epilepsy severity. Vigabatrin has no approved indications or clinical uses in psychiatry, despite weak evidence for its usefulness in the treatment of anxiety disorders and addictions.

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25

Scott, Jan. Psychological interventions for early stage bipolar disorder. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198748625.003.0011.

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Clinical staging and early intervention models used in psychosis and depression have only recently been applied to individuals ‘at risk’ of bipolar disorder (BD), or experiencing a first episode of BD. This chapter briefly discusses the concept of staging and then reviews ongoing research into the adaptation and use of psychological interventions in ‘at risk’ and ‘first BD episode’ populations. Evidence indicates that the current interventions may not sufficiently target specific developmentally normal changes in cognitive–emotional and sleep–circadian regulation systems that may act as triggers for mood episodes. So the chapter discusses how to tackle these ‘dysregulations’ and how to ensure any ‘early stage’ therapy is sufficiently flexible to tackle the range of problems experienced, including mood symptoms, harmful alcohol or substance use, and/or co-morbid physical ill-health and that the therapy models must take into account that not all individuals in high-risk populations actually develop BD.

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26

Tzilos, Golfo, Kristina Davis e Caron Zlotnick. Prevention of Postpartum Psychopathology. Editado por Amy Wenzel. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199778072.013.29.

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Approximately 26% of postpartum women meet diagnostic criteria for a psychiatric disorder. Untreated psychopathology in the postpartum period is associated with a range of adverse outcomes for both infant and mother. Fortunately, the perinatal period provides an opportune time to intervene with and prevent postpartum psychopathology. Women have increased contact with health care providers during this time, providing an avenue through which access to prevention can be improved. Furthermore, with increased knowledge to identify high-risk women, preventive interventions can be delivered to assist both the woman and infant. Preventive efforts for postpartum psychopathology are aimed at modifying risk factors or protective factors to prevent the psychiatric disorder and primarily use three distinct approaches: universal, selective, and indicated. This chapter provides a review of the empirical research in the prevention of postpartum psychopathologies including postpartum depression, anxiety, bipolar disorder, psychosis, and addictions (tobacco, alcohol, and illicit drugs).

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27

Burdick, Katherine E., Luz H. Ospina, Stephen J. Haggarty e Roy H. Perlis. The Neurobiology and Treatment of Bipolar Disorder. Editado por Dennis S. Charney, Eric J. Nestler, Pamela Sklar e Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0020.

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Bipolar disorder (BPD) is a severe mood disorder that often has psychotic features. Its most severe forms are more common and significantly more likely to cause disability than originally thought. Studies of high-risk children have found them to be at increased risk for a variety of symptoms and neurobiological abnormalities. In contrast to schizophrenia, there is no formal prodromal syndrome that has been identified, and cognitive abnormalities do not precede the onset of the disorder. Abnormal sleep and circadian rhythms are prominent and have led to intriguing biological models. Neurobiological experiments have primarily focused on candidate pathways and include circadian abnormalities, epigenetic processes including histone modification, WNT/GSK3 signaling, other modulators of neuroplasticity, and mitochondrial dysfunction. Recent data suggest that BPD is a highly polygenic disease and that integration of prior modeling and data with the wide variety of new genetic risk loci will be productive in the future.

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28

Cavanna, Andrea E. Topiramate. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198791577.003.0013.

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Topiramate is a second-generation antiepileptic drug characterized by a good range of antiepileptic indications, with an acceptable interaction profile in polytherapy. Topiramate has an acceptable behavioural tolerability profile, although it has been associated with a number of negative behavioural effects in patients with epilepsy (in particular depression, irritability, and psychotic symptoms). Identified risk factors for the development of behavioural adverse effects include high starting doses and rapid titration schedules, as well as personal or family history of psychiatric disorders. Topiramate has a restricted range of psychiatric uses , although its clinically significant effect in promoting weight loss in patients with behavioural problems can increase its potential usefulness in psychiatric populations.

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29

Ganança, Licínia, David A. Kahn e Maria A. Oquendo. Mood Disorders. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199326075.003.0003.

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This chapter discusses the mood disorders. Major depressive disorder is characterized by neurovegetative changes, anhedonia, and suicidal ideation. Persistent depressive disorder is a milder form of depression, lasting for at least 2 years, with little or no remission during that time... Psychotic features can occur in both depressive and manic episodes. Premenstrual dysphoric disorder is diagnosed through use of a prospective daily symptom ratings log showing a cyclical pattern over at least 2 consecutive months. Patients with mood episodes with mixed features have a high risk of suicide. Some patients with bipolar disorder and major depressive disorder may develop catatonic features characterized by marked psychomotor disturbance. Selective serotonin reuptake inhibitors (SSRIs) are the usual first-line medication treatment for patients with major depressive disorder. For patients with bipolar disorder the mainstays of somatic therapy are lithium and the anticonvulsants valproate and carbamazepine.

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30

Steinberg, Martin. Treatment of Depression. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199959549.003.0006.

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Most depression in the elderly can be effectively treated in the primary care setting. Psychiatric referral should be considered in the setting of severe depression, suicidal ideation, prior suicide attempts, multiple risk factors, psychotic symptoms, bipolar disorder, poor response to prior treatment, or high medical comorbidity. Combining pharmacological and psychosocial interventions is most likely to be effective. Available antidepressants include serotonin-specific reuptake inhibitors, serotonin norepinephrine reuptake inhibitors, novel mechanism agents, tricyclic antidepressants, and monoamine oxidase inhibitors. Antidepressant selection should take into account adverse effects, medical comorbidities, potential medication interactions, and patient preferences. Additional strategies (e.g. augmentation) are available for treatment resistant depression. Available psychotherapies include supportive, cognitive-behavioral, interpersonal, and problem solving. Lifestyle interventions (e.g. exercise) may be helpful adjuncts. Given limited evidence for antidepressant treatment in cognitive impairment, for those with mild to moderate depression severity, non-pharmacological interventions should be attempted first.

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