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Bibliografias temáticas / Graft rejection / Teses / dissertações

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Teses / dissertações sobre o tema "Graft rejection"

Autor: Grafiati

Publicado: 4 de junho de 2021

Última modificação: 25 de julho de 2025

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1

Dooldeniya, Mohanta Deevan. "The role of graft expressed Fasligand in graft rejection." Thesis, Imperial College London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.419907.

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2

Waters, Cheryl Denise. "The cellular requirements for graft rejection." Thesis, The University of Sydney, 1985. https://hdl.handle.net/2123/26737.

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The studies described in this thesis were designed to develop a model in which the capacity of various lymphoid cells subpopulations to cause graft rejection could be tested and correlated with their capacity to effect in vitro lysis of appropriate target cells.

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3

Wu, Guosheng. "Experimental studies on xenograft rejection /." Stockholm, 2001. http://diss.kib.ki.se/2001/91-628-4805-4/.

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4

Patrick, Guy M. "Studies of cytokines in alloimmune responses /." Title page, table of contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09php314.pdf.

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5

Kumar, Rajesh. "Does graft-expressed TRAIL modify the rejection process?" Thesis, Imperial College London, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.544289.

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6

Duguid, I. G. M. "Prevention of corneal graft rejection with monoclonal antibodies." Thesis, University of Aberdeen, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.387460.

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This thesis aims to place corneal allograft rejection in the context of general transplantation immunology, examine the role of lymphocyte subsets in the rejection process and consider the potential application of monoclonal antibody therapy in clinical corneal graft rejection. The literature relating to the current clinical practice of corneal grafting, with particular reference to corneal allograft rejection, is reviewed in chapter 1 to present the extent of the problem. Chapter 2 then reviews the mechanisms of allograft rejection from the literature of transplantation immunology, much of wh

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7

Chain, Robert Whatley. "THE ROLE OF DENDRITIC CELLS IN GRAFT REJECTION." Master's thesis, Temple University Libraries, 2012. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/194726.

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Microbiology and Immunology<br>M.S.<br>Induction of acquired immunological tolerance is the ultimate goal in transplantation. So far the acceptance of a mismatched graft is achieved through immunosuppression that requires long-term treatment and a variety of methods have been explored to prevent rejection and achieve transplant tolerance in mouse models. There are several factors that contribute to acquired tolerance. Recent studies have focused on the inhibition of costimulatory molecules and TLRs in Dendritic Cells (DCs), as a key to the mechanisms underlying the barrier to tolerance inducti

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8

Choi, Chi-wai, and 蔡志維. "Detection of class I-related polypeptide-related sequence A (MICA) and angiotensin II type 1 receptor (AT1R) antibodies in antibody mediated rejection in Hong Kong." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/206596.

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Background: Rejection is considered as a major barrier to achieve successful transplantation. Non self-human leucocyte antigen (HLA) is a well-known antigenic target for antibodies binding that can result in antibody-mediated rejection (AMR). To reduce risk of rejection in kidney transplant, preventive measures are undertaken, which include HLA-matching between donor and recipient, and in-vitro pre-transplant crossmatch with potential donor cells and recipient sera, furthermore, periodic HLA antibodies monitoring for donor-specific antibodies (DSA) is carried out before and after transplant. N

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9

Toogood, Giles John. "Cytokines in small bowel transplantation : expression during graft rejection." Thesis, University of Oxford, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.297078.

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10

Figueiredo, Francisco Carlos D'Amorim de. "Immunopathology of corneal graft rejection in a rat model." Thesis, University of Bristol, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.296671.

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11

Endo, Kosuke. "Pretransplant replacement of donor liver grafts with recipient Kupffer cells attenuates liver graft rejection in rats." Kyoto University, 2015. http://hdl.handle.net/2433/199205.

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12

Larsen, Christian Peter. "The migration and function of dendritic leukocytes after transplantation." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.256294.

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13

Antoniou, Antony Nicodemus. "Stimulation of immune responses by mutated transgenic self-products." Thesis, Open University, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.262551.

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14

Saxton, Nina Elizabeth. "Anti-TNF-#alpha# treatment in the rat heterotropic cardiac allograft model." Thesis, Open University, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.321566.

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15

Wang, Wen-Hua 1965. "Cytokine gene expression and gene therapy in experimental corneal graft rejection." Thesis, McGill University, 2001. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=38529.

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It has been proposed that CD4+ T cells and cell-mediated immunity play a central role in corneal allograft rejection. Two subsets of CD4+ T cells, Th1 and Th2 cells, are known to cross-regulate each other through their cytokine pattern and the immune response might be directed predominantly in one or the other direction. As such, it has been hypothesized here that predominant Th1 type immune response could lead to corneal allograft rejection, and that previous inflamed corneal beds (high-risk eyes) might augment the Th1 response and thus accelerate the graft rejection.<br>Reverse transcription

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16

Loh, Yik Wen. "Analysis of CD4 T cell-dependent skin and islet graft rejection." Thesis, The University of Sydney, 2014. http://hdl.handle.net/2123/13085.

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Studies described in this thesis made use of the CD4+ 5C.C7 TCR transgenic model to analyse CD4+ T cell responses in two models of fully MHC-mismatched non-vascularised grafting: skin and islet grafts. The crossreactive specificities of the 5C.C7 TCR model allow the investigation of direct allorecognition and the role of heterologously primed memory cells in allogeneic graft rejection or acceptance. Data presented is consistent with a model in which memory, not naïve CD4+ T cells, primed by environmental exposure before the time of grafting and expressing allo-crossreactive specificities, are

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17

Hunt, James Barrie. "Endomyocardial biopsy diagnosis of acute cardiac allograft rejection." Master's thesis, University of Cape Town, 1991. http://hdl.handle.net/11427/25718.

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The aims of the present investigation are fourfold: (i) to review the range of non-invasive methods that may be used to diagnose acute cardiac allograft rejection; (ii) to review the use of the bioptome in sampling the donor heart endomyocardium; (iii) to review the light microscopic and histological grading of acute cardiac rejection; (iv) to characterise the mononuclear populations in endomyocardial biopsy samples and correlate the findings with the light microscopic appearances of the same biopsy specimens.

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18

Otasevic-Wieschalla, Ljiljana [Verfasser]. "New drugs in prevention of experimental corneal graft rejection / Ljiljana Otasevic-Wieschalla." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2014. http://d-nb.info/1057870056/34.

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19

Robinson, Rebecca Hartzell. "Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection." Diss., Temple University Libraries, 2014. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/246094.

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Microbiology and Immunology<br>Ph.D.<br>Cannabinoids are known to have anti-inflammatory and immunomodulatory properties. Cannabinoid receptor 2 (CB2) is expressed mainly on leukocytes and is the receptor implicated in mediating many of the effects of cannabinoids on immune processes. The capacity of delta-9-tetrahydrocannabinol (delta-9-THC) and of two CB2-selective agonists to inhibit the murine Mixed Lymphocyte Reaction (MLR), an in vitro correlate of graft rejection following skin and organ transplantation was tested. Both CB2-selective agonists and delta-9-THC significantly suppressed the

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20

Ballow, Amany A. "The implications of different IgG subclasses on graft rejection in sensitized individuals." Thesis, Imperial College London, 2011. http://hdl.handle.net/10044/1/6182.

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The presence of memory B cells may contribute to graft damage. In fact, pre-transplant B cell sensitization in the absence of circulating antibody represents a risk factor, since grafts transplanted into recipients with historical positive but current negative direct crossmatch do less well than patients with no evidence of B cell sensitisation. This definition of ‘negative’ is based on conventional complement-dependent cytotoxicity (CDC) assays and, more recently, on flow cytometry. In this study, I have analysed the significance of antibodies undetectable by these conventional techniques but

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21

Wennberg, Lars. "Islet xenograft rejection : studies in the pig-to-rodents and pig-to-primate models /." Stockholm, 1998. http://diss.kib.ki.se/1998/91-628-2835-5.

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22

Niimi, Masanori. "An investigation to determine the ability of allogeneic resting B cells to induce specific unresponsiveness in vivo." Thesis, University of Oxford, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244813.

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23

Lovegrove, Emma. "Indirect T cell allorecognition of the RT1.A'a MHC class I molecule." Thesis, University of Glasgow, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.340253.

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24

Coxon, Fraser P. "Properties of cyclophilins and their ligands in bone." Thesis, University of Sheffield, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263761.

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25

Sharma, Ankit. "The impact of eplet mismatches and de novo donor specific antibodies in kidney and simultaneous pancreas-kidney transplantation." Thesis, University of Sydney, 2020. https://hdl.handle.net/2123/24228.

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Improving kidney transplant graft survival is of critical importance to patients, caregivers and health professionals. Advances in immunosuppression regimens have mitigated the risk and injurious effects of T-cell mediated rejection resulting in increased graft survival rates, particularly in the first year after transplantation. The leading cause of graft attrition is now antibody mediated rejection (AMR), implicated in up to two thirds of graft loss after one year. Donor specific antibodies (DSA) are central to the pathogenesis of AMR, with the presence of pre-transplant DSA (or sensitisatio

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26

Wang, Chuanmin. "Studies of allograft tolerance in rodents." Thesis, The University of Sydney, 2000. https://hdl.handle.net/2123/27722.

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Allograft rejection is still the main obstacle to successful treatment of patients with end—stage organ failure by organ transplantation. Both tolerance and rejection of allografts are complicated responses of the host immune system to foreign antigens. It is almost 50 years since Billingham, Brent, and Medawar first described the phenomenon of neonatal tolerance to skin allografts in a murine model. Tolerance to vascularised organ allografts was subsequently described in pig, rodent, and primate models. Knowledge regarding the mechanisms by which tolerance is induced and maintained has

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27

Cai, Qi, and 蔡綺. "The possible mechanisms of peroxisome proliferator-activatedreceptor (PPAR) agonists in controlling graft rejection." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B36396199.

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28

Wilson, Nicole K. "Borderline Lesions Exhibit Clinical and Graft Survival Characteristics Common to Acute Cellular Rejection." University of Cincinnati / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1627665576477761.

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29

Wong, Jeffrey K. W. "Chemokines and chemokine receptors in islet xenograft rejection." Thesis, The University of Sydney, 2006. https://hdl.handle.net/2123/28055.

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This project investigates the role of chemokine and chemokine receptors in a model of CD4 T cell dependent cellular xenograft rejection, specifically the transplantation of fetal pig pancreas tissue to the renal subcapsular space of mice. Chemokines and chemokine receptor gene expression was assessed by cDNA arrays, and confirmed by multi-probe ribonuclease protection assay. Immunostaining for a selected chemokine, RANTES was performed to demonstrate upregulation at the protein level. These methods were applied to several different models to dissect the role Chemokines and their re

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30

Chinaelli, Marco. "99mTc labelling of interleukin-2 for in-vivo detection of lymphocytic infiltration." Thesis, Queen Mary, University of London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.243365.

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31

Reel, Michael Stephen. "The Role of Ectopic Lymphoid Tissue in Allograft Rejection." Yale University, 2006. http://ymtdl.med.yale.edu/theses/available/etd-06282006-140255/.

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The location of the immunologic response to an allograft is not known with certainty. However, organized collections of T cells, B cells and antigen presenting cells have been found in peripheral tissue, in close proximity to organs undergoing rejection. It is hypothesized that this tertiary lymphoid tissue may be a location in which activation of lymphocytes can occur, leading to rejection of an allograft. We report here that in a splenectomized aly/aly mouse, which is devoid of secondary lymphoid organs and will normally fail to reject an allograft, the presence of tertiary lymphoid organs i

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32

McLean, Adam George. "Patterns of graft infiltration and cytokine gene expression during the first ten days of kidney transplantation." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.390513.

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33

Cai, Qi. "The possible mechanisms of peroxisome proliferator-activated receptor (PPAR) agonists in controlling graft rejection." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B36396199.

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34

Ge, Xupeng. "Mechanisms of liver allograft rejections /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-330-2/.

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35

Scully, Ralph. "Mechanisms in transplantation tolerance." Thesis, University of Cambridge, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.321084.

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36

Li, Daxu, and 李大旭. "Role of adiponectin in preventing chronic rejection and the underlyingmolecular immunoregulatory signaling pathway." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47155966.

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Chronic rejection is a major obstacle to long-term survival of organ transplants. PPAR-γ agonist rosiglitazone has been shown to reduce graft rejection but the underlying mechanisms remain unclear. Combined treatment of rosiglitazone and anti-IL-5 antibody prevented MHC class II histoincompatiblecardiac graft rejection with a reduction of cellular infiltration, vasculopathy and interstitial fibrosis in a heterotopic heart transplantation model. In particularly, rosiglitazone decreased CD8 T cells infiltration and luminal occlusion, while anti-IL-5 antibody reduced eosinophil infiltration and

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37

Khosravi, Maharlooei Mohsen. "Deploying the tolerogenic effects of IDO enzyme and skin fibroblasts in prevention of graft rejection." Thesis, University of British Columbia, 2017. http://hdl.handle.net/2429/62706.

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Indoleamine 2,3-dioxygenase (IDO) is an immunosuppressive enzyme with tolerogenic effects on different immune cells. Our group has previously shown that co-transplantation of IDO-expressing fibroblasts with donor tissues can delay immune rejection by inducing local immunosuppression. We first asked a question whether we can improve this effect by delivering the IDO-fibroblasts through a systemic intraperitoneal approach, instead of local co-transplantation, and secondly whether this effect is only delivered by the immunosuppressive effects of IDO or the fibroblast cells have additional immunos

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38

Stone, John. "Assessing the impact of ex vivo perfusion on graft immunogenicity." Thesis, University of Manchester, 2017. https://www.research.manchester.ac.uk/portal/en/theses/assessing-the-impact-of-ex-vivo-perfusion-on-graft-immunogenicity(a8ad264a-8925-44ee-94c0-465d3ddd7e14).html.

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Whilst the major caveat to the success of organ transplantation remains the severe lack of donor organs, rejection is still a primary confounding factor to transplant outcomes. This is an allospecific response that occurs when the recipient immune system recognises conserved proteins on donor-derived cells as 'non-self'. Currently, all immunosuppressive regimes target the recipient immune response, ignoring the large donor immune repertoire despite these cells playing a central role in acute rejection. This is likely as a result of a lack of understanding of the temporal migration of the donor

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39

Ehrnfelt, Cecilia. "In vitro models of xenograft rejection : studies on leukocyte-endothelial cell interactions /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-807-6/.

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40

Kassis, Elias Noah. "Nanoparticle use in the modulation of transplant rejection in a murine model." Yale University, 2010. http://ymtdl.med.yale.edu/theses/available/etd-03052010-124710/.

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Solid organ transplant has emerged over the last half century as an important treatment for solid organ failure. Management has matured dramatically over the past two decades with improvements in acute rejection, but long-term graft survival has improved very little and current treatment is limited by the side-effects and toxicities of immunosuppressive medications. Nanoparticle delivery of therapeutics, improving transport characteristics and decreasing systemic and local toxicity has emerged as a dynamic treatment modality, but little work has been done using nanoparticles in transplantation

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41

Adeegbe, Dennis O. "Allogeneic CD4+CD25+Foxp3+ T Regulatory Cells in Autoimmunity and Transplantation Tolerance: Therapeutic Potential and TCR Repertoire Requirement." Scholarly Repository, 2008. http://scholarlyrepository.miami.edu/oa_dissertations/43.

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CD4+CD25+Foxp3+ T regulatory (Treg) cells are critical in maintaining self tolerance and promoting the acceptance of allogeneic tissue/organ grafts. To be widely applied in clinical settings, there needs to be a readily available source of Treg cells, a requirement that is better met if non-histocompatible donor cells could be utilized in adoptive therapy. Therefore, to investigate the therapeutic potential of fully allogeneic Treg cells to control autoimmune disease or allograft rejection, we utilized IL-2R beta-deficient mice that exhibit rapid lethal autoimmunity due to low production of a

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42

Lui, Sing-leung, and 雷聲亮. "The in vivo mechanism of actions of mycophenolate mofetil: insights from murine models of allograft rejection,endotoxemia, ischemia reperfusion injury and lupus nephritis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B26625374.

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43

Lai, Sum Wing Christina. "An Analysis of Strategies Targeting Early Clinical and Immunological Events to Improve Kidney Transplant Outcomes." Thesis, The University of Sydney, 2022. https://hdl.handle.net/2123/29651.

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Background: Kidney transplantation is an essential treatment for patients with end-stage renal failure. The long-term fate of the transplant often depends on early events, including the development of delayed graft function (DGF) and rejection. However, there are barriers to targeting these risk factors to improve outcomes. First, DGF is associated with an increased incidence of acute rejection, but there is a lack of consensus around how to diagnose DGF. Second, despite growing recognition of the role of inflammatory monocytes (MΦ) in rejection, no clinically available treatment specifically

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44

Sleater, Michelle Leigh. "Cellular and molecular effector mechanisms of islet allograft rejection /." Connect to full text via ProQuest. IP filtered, 2006.

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Thesis (Ph.D. in Immunology) -- University of Colorado at Denver and Health Sciences Center, 2006.<br>Typescript. Includes bibliographical references (leaves 151-168). Free to UCDHSC affiliates. Online version available via ProQuest Digital Dissertations;

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45

Reichenspurner, Hermann. "An assessment of a new immunosuppressive agent 15-deoxyspergualin (15-DS) following cardiac and renal allotransplantation and cardiac xenotransplantation in primates / does 15-deoxyspergualin induce graft nonreactivity." Doctoral thesis, University of Cape Town, 1992. http://hdl.handle.net/11427/26253.

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46

Cena, Tiziana. "Post-kidney transplant malignancies affect graft survival: results from a time-dependent analysis." Doctoral thesis, Università del Piemonte Orientale, 2018. http://hdl.handle.net/11579/105206.

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Aim of this study was to evaluate the association between cancer occurrence and risk of graft failure in kidney transplant recipients. From 1998 to 2013, 672 adults receiving their first kidney transplant from a deceased donor, with at least six months of follow-up, were included in the study. To illustrate the effect of tumors incidence on graft failure risk, a modified Kaplan-Meier method was used. To quantify the tumor effect as hazard ratio, multivariable adjusted Cox models were fitted considering the diagnosis of non-cutaneous malignancies (NCM) and non-melanoma skin cancer (NMSC) as a t

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47

Zhao, Xiangli [Verfasser]. "Investigation on the role of CD26 in Th1 and Th17 cell differentiation and allogeneic graft rejection / Xiangli Zhao." Berlin : Freie Universität Berlin, 2018. http://d-nb.info/1158597665/34.

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48

Forman, Daron. "Viral Abrogation of Stem Cell Transplantation Tolerance Causes Graft Rejection and Host Death by Different Mechanisms: A Dissertation." eScholarship@UMMS, 2002. https://escholarship.umassmed.edu/gsbs_diss/72.

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Tolerance-based stem cell transplantation using sub-lethal conditioning is being considered for the treatment of human disease, but safety and efficacy remain to be established. In order to study these two issues, we first established that mouse bone marrow recipients treated with sub-lethal irradiation plus transient blockade of the CD40-CD154 costimulatory pathway develop permanent hematopoietic chimerism across allogeneic barriers. Our conditioning regimen of 6 Gy irradiation, a short course of anti-CD154 mAb and 25 million fully allogeneic BALB/c bone marrow cells consistently produced lon

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49

Goldsmith, Paul Joseph. "1H NMR spectroscopic identification of non-invasive biomarkers of acute rejection and delayed graft function in renal transplantation." Thesis, University of Leeds, 2013. http://etheses.whiterose.ac.uk/5901/.

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Delayed graft function (DGF) and acute rejection (AR) are complications after renal transplantation. It is impossible to differentiate between these clinically. Renal biopsy is the gold standard for diagnosis but is invasive and associated with complications. We aimed to identify early biomarkers, of DGF and AR in renal transplantation, which could lead to a diagnostic test that has no morbidity or mortality associated with its use. In total 163 from twenty-four patients using blood samples over several different pre and post-operative time-points were analysed. Plasma was extracted and analys

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50

Li, Xiaosong. "The mechanism study of novel approaches to control chronic allograft rejection in rat orthotopic small bowel transplantation." Click to view the E-thesis via HKUTO, 2006. http://sunzi.lib.hku.hk/hkuto/record/B36395778.

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