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Bibliografias temáticas / Glomerulonephritis Immunological aspects

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Literatura científica selecionada sobre o tema "Glomerulonephritis Immunological aspects"

Autor: Grafiati

Publicado: 4 de junho de 2021

Última modificação: 28 de janeiro de 2023

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Artigos de revistas sobre o assunto "Glomerulonephritis Immunological aspects"

1

Sethi, D., and E. R. Maher. "Immunological Aspects of Glomerulonephritis." Journal of the Royal Society of Medicine 80, no. 3 (March 1987): 189–91. http://dx.doi.org/10.1177/014107688708000322.

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2

Bondareva, L., and L. Vykhristenko. "Pathogenetic and morphological features in certain types of primary glomerulonephritis." Immunopathology, Allergology, Infectology 2021, no. 2 (April 1, 2021): 69–79. http://dx.doi.org/10.14427/jipai.2021.2.69.

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The kidney, as an organ, quite often appears as the target of immune system dysregulation in the context of a primary or systemic disease. Glomerulonephritis (GL) is the most common clinical and pathological manifestation of this process. Currently, there is no unified view on the classification of GN. However, the study and recognition of the pathophysiological mechanisms of GN made it possible to determine the immunological features, biomarkers and genetic aspects of the disease. In the review, we update modern ideas about the genetic factors, etiology, immunopathogenesis of primary GN, and

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3

Schena, F. P., L. Gesualdo, and V. Montinaro. "Immunopathological aspects of immunoglobulin A nephropathy and other mesangial proliferative glomerulonephritides." Journal of the American Society of Nephrology 2, no. 10 (April 1992): S167. http://dx.doi.org/10.1681/asn.v210s167.

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Immunoglobulin A nephropathy (IgAN) is an immune complex (IC) glomerulonephritis (GN) that represents one of the most common forms of primary glomerular disease. Proliferation of mesangial cells and the increase of mesangial matrix are histological hallmarks of mesangioproliferative GN. Increased serum levels of IgA, polymeric IgA, IgA rheumatoid factor, IgA-IC, and spontaneous or pokeweed mitogen-induced production of IgA by peripheral blood mononuclear cells are major humoral immune alterations reported in IgAN. Recently, we focused on the role of cytokines and growth factors in the mediatio

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4

Maixnerova, Dita, Delphine El Mehdi, Dana V. Rizk, Hong Zhang, and Vladimir Tesar. "New Treatment Strategies for IgA Nephropathy: Targeting Plasma Cells as the Main Source of Pathogenic Antibodies." Journal of Clinical Medicine 11, no. 10 (May 16, 2022): 2810. http://dx.doi.org/10.3390/jcm11102810.

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Immunoglobulin A nephropathy (IgAN) is a rare autoimmune disorder and the leading cause of biopsy-reported glomerulonephritis (GN) worldwide. Disease progression is driven by the formation and deposition of immune complexes composed of galactose-deficient IgA1 (Gd-IgA1) and Gd-IgA1 autoantibodies (anti-Gd-IgA1 antibodies) in the glomeruli, where they trigger complement-mediated inflammation that can result in loss of kidney function and end-stage kidney disease (ESKD). With the risk of progression and limited treatment options, there is an unmet need for therapies that address the formation of

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Teses / dissertações sobre o assunto "Glomerulonephritis Immunological aspects"

1

Wootton, Andrew. "The glomerular basement membrane and nephritis /." Title page, contents and abstract only, 1985. http://web4.library.adelaide.edu.au/theses/09PH/09phw918.pdf.

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2

"Prognostic and immunogenetic factors of IgA nephropathy." 2003. http://library.cuhk.edu.hk/record=b6073641.

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Li Kam-tao, Philip.<br>"January 2003."<br>Thesis (M.D.)--Chinese University of Hong Kong, 2003.<br>Includes bibliographical references (p. 252-281).<br>Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Mode of access: World Wide Web.

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3

Wootton, Andrew. "The glomerular basement membrane and nephritis." 1986. http://web4.library.adelaide.edu.au/theses/09PH/09phw918.pdf.

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4

"The association of various HLA-A, -B and -DR loci with membranous glomerulonephritis, IgA nephropathy, and focal segmental glomerulosclerosis in KwaZulu-Natal renal patients." Thesis, 2007. http://hdl.handle.net/10413/1789.

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This KwaZulu-Natal (KZN) based study investigates hypertension, glomerulonephritides and the rarity of IgA Nephropathy (IgAN) in Africans in association with the Human Leukocyte Antigen (HLA). A retrospective hypertensive study found a positive association with HLA-B40 (P c<0.05) and HLA-B15 (Pc<0.02) in Indians and Africans respectively. No association was found in Whites. A prospective study showed glomerulonephritides to be positively associated with HLA-A33 in Indians (Pc 0.049). No associations were found with glomerulonephritides in Africans and Whites. Combined Race groups show no HLA a

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Livros sobre o assunto "Glomerulonephritis Immunological aspects"

1

A, Rakiti͡anskai͡a I., and Shutko A. N, eds. Pochki i sistema immuniteta. Leningrad: "Nauka," Leningradskoe otd-nie, 1989.

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2

D, Pusey C., ed. The treatment of glomerulonephritis. Dordrecht: Kluwer Academic, 1999.

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3

Claudio, Ponticelli, Minetti L, and D'Amico G. 1929-, eds. Antiglobulins, cryoglobulins, and glomerulonephritis: Second International Milano Meeting of Nephrology, 30 September-1 October 1985. Dordrecht: M. Nijhoff, 1986.

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4

Glomerulopathies: Cell biology and immunology. Australia: Harwood Academic Press, 1996.

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5

Pusey, C. D. The Treatment of Glomerulonephritis. Springer, 2014.

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6

Pusey, C. D. The Treatment of Glomerulonephritis. Springer, 1999.

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7

Pusey, Charles. Treatment of Glomerulonephritis. Springer London, Limited, 2007.

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8

1947-, Ballardie Francis W., ed. Autoimmunity in nephritis. Chur: Harwood Academic Publishers, 1992.

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9

B, Wilson Curtis, ed. Immunopathology of renal disease. New York: Churchill Livingstone, 1988.

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10

Berden, Jo H. M., and Jack F. M. Wetzels. Immunological investigation of the patient with renal disease. Edited by Christopher G. Winearls. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0017.

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Laboratory techniques (electrophoresis, indirect immunofluorescence, ELISA, and immunoblotting) required for immunological investigation of the patient with renal disease are described. Renal disease-related aspects of immunoglobulins (immunoglobulin A, paraproteins, cryoglobulins), complement, antinuclear antibodies, anti-C1q antibodies, antineutrophil cytoplasmic antibodies, anti-glomerular basement membrane antibodies, antipodocyte antibodies, antiphospholipid antibodies, and antimicrobial responses (streptococci, hepatitis C, hepatitis B) are reviewed. Laboratory assays which evaluate the

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