Literatura científica selecionada sobre o tema "Gal4/gal80"
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Artigos de revistas sobre o assunto "Gal4/gal80"
Jiang, Fenglei, Benjamin R. Frey, Margery L. Evans, Jordan C. Friel e James E. Hopper. "Gene Activation by Dissociation of an Inhibitor from a Transcriptional Activation Domain". Molecular and Cellular Biology 29, n.º 20 (3 de agosto de 2009): 5604–10. http://dx.doi.org/10.1128/mcb.00632-09.
Texto completo da fonteLue, N. F., D. I. Chasman, A. R. Buchman e R. D. Kornberg. "Interaction of GAL4 and GAL80 gene regulatory proteins in vitro". Molecular and Cellular Biology 7, n.º 10 (outubro de 1987): 3446–51. http://dx.doi.org/10.1128/mcb.7.10.3446-3451.1987.
Texto completo da fonteLue, N. F., D. I. Chasman, A. R. Buchman e R. D. Kornberg. "Interaction of GAL4 and GAL80 gene regulatory proteins in vitro." Molecular and Cellular Biology 7, n.º 10 (outubro de 1987): 3446–51. http://dx.doi.org/10.1128/mcb.7.10.3446.
Texto completo da fonteSalmeron, J. M., S. D. Langdon e S. A. Johnston. "Interaction between transcriptional activator protein LAC9 and negative regulatory protein GAL80". Molecular and Cellular Biology 9, n.º 7 (julho de 1989): 2950–56. http://dx.doi.org/10.1128/mcb.9.7.2950-2956.1989.
Texto completo da fonteSalmeron, J. M., S. D. Langdon e S. A. Johnston. "Interaction between transcriptional activator protein LAC9 and negative regulatory protein GAL80." Molecular and Cellular Biology 9, n.º 7 (julho de 1989): 2950–56. http://dx.doi.org/10.1128/mcb.9.7.2950.
Texto completo da fonteBhat, P. J., e J. E. Hopper. "Overproduction of the GAL1 or GAL3 protein causes galactose-independent activation of the GAL4 protein: evidence for a new model of induction for the yeast GAL/MEL regulon". Molecular and Cellular Biology 12, n.º 6 (junho de 1992): 2701–7. http://dx.doi.org/10.1128/mcb.12.6.2701-2707.1992.
Texto completo da fonteBhat, P. J., e J. E. Hopper. "Overproduction of the GAL1 or GAL3 protein causes galactose-independent activation of the GAL4 protein: evidence for a new model of induction for the yeast GAL/MEL regulon." Molecular and Cellular Biology 12, n.º 6 (junho de 1992): 2701–7. http://dx.doi.org/10.1128/mcb.12.6.2701.
Texto completo da fonteParthun, M. R., e J. A. Jaehning. "A transcriptionally active form of GAL4 is phosphorylated and associated with GAL80". Molecular and Cellular Biology 12, n.º 11 (novembro de 1992): 4981–87. http://dx.doi.org/10.1128/mcb.12.11.4981-4987.1992.
Texto completo da fonteParthun, M. R., e J. A. Jaehning. "A transcriptionally active form of GAL4 is phosphorylated and associated with GAL80." Molecular and Cellular Biology 12, n.º 11 (novembro de 1992): 4981–87. http://dx.doi.org/10.1128/mcb.12.11.4981.
Texto completo da fonteSalmeron, J. M., K. K. Leuther e S. A. Johnston. "GAL4 mutations that separate the transcriptional activation and GAL80-interactive functions of the yeast GAL4 protein." Genetics 125, n.º 1 (1 de maio de 1990): 21–27. http://dx.doi.org/10.1093/genetics/125.1.21.
Texto completo da fonteTeses / dissertações sobre o assunto "Gal4/gal80"
Goupil, Alix. "Genome instability : from genome content variations to gene expression plasticity". Electronic Thesis or Diss., Université Paris sciences et lettres, 2021. http://www.theses.fr/2021UPSLS053.
Texto completo da fonteMost animal cells are diploid, containing two copies of each chromosome. Establishment of proper bipolar mitotic spindle containing two centrosomes, one at each pole contributes to accurate chromosome segregation. This is essential for the maintenance of genome stability, tissue and organism homeostasis. However, numerical deviations to the diploid set are observed in healthy tissues. Polyploidy is the doubling of the whole chromosome set and aneuploidy concerns the gain or loss of whole chromosomes. Importantly, whole genome duplications and aneuploidy have also been associated to pathological conditions. For example, variations to genome content are associated with chromosome instability and cancer development, however their exact contribution to cancer genome remains poorly understood.In the first part of my PhD project, I investigated the consequences of polyploidy during cell division. I found that the presence of extra DNA and extra centrosomes generated invariably multipolar spindles. Then I identified contributors to the multipolar status using in vivo approaches in Drosophila neural stem cells and in vitro culture of cancer cells. Further I combined DNA and spindle perturbations with computer modelling and found that in polyploid cells, the presence of excessive DNA acts as a physical barrier blocking spindle pole coalescence and bipolarity. Indeed, laser ablation to disrupt and increase in microtubule stability and length to bypass the DNA-barrier could rescue bipolar spindle formation. This discovery challenges the current view that suggested extra-centrosomes as only contributor to spindle multipolarity and provides a rational to understand chromosome instability typical of polyploid cells.The aim of the second part of my PhD project was to generate a novel tool to quantitively probe chromosome loss in vivo in Drosophila tissues. Aneuploidy has been observed in various physiological tissues, however the frequency of this error remained highly debatable. In addition, tools developed so far to assess aneuploidy lack a temporal dimension. To circumvent this, I used the expression of a GFP report gene driven by the GAL4/UAS system and its inhibition by GAL80. In principle, the random loss of the chromosome carrying the GAL80 sequence leads to GFP appearance in aneuploid cells that can therefore be followed in live tissues. I found that chromosome loss was extremely infrequent in most tissues of the wild type fly. This tool combined with fluorescent marker and/or tested in various genetic background, might help understanding mechanisms behind aneuploidy genesis and outcome in vivo.While developing this tool, I discovered that in the larval brain, GFP cells where not a by-product of chromosome loss but rather an unexpected mis-regulation in the expression of the GAL80 gene. These results have strong implications for the Drosophila community as it can result in false positive in clonal experiments. Further, I discovered a mosaicism and plasticity of the Drosophila brain in neural stem cells for gene expression which differs from other organs and that is influenced by environmental stimuli. This possibly reflects a certain level of plasticity in the brain necessary for neuronal diversity, adaptation and survival
Egriboz, Onur. "THE MOLECULAR MECHANISMS GOVERNING THE GAL GENE SWITCH OF SACCHAROMYCES CEREVISIAE". The Ohio State University, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=osu1338319985.
Texto completo da fonteGoswami, Sudip. "Investigation Of The Behavior Of The Gal4 Inhibitor Gal80 Of The GAL Genetic Switch In The Yeast Saccharomyces Cerevisiae". The Ohio State University, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=osu1408542557.
Texto completo da fonteZugowski, Constance [Verfasser], K. [Akademischer Betreuer] Breunig, H. [Akademischer Betreuer] Lilie e K. [Akademischer Betreuer] Melcher. "Molecular genetic and biochemical interaction studies of the transcriptional activator Gal4 and its repressor Gal80 in Saccharomyces cerevisiae and Kluyveromyces lactis / Constance Zugowski. Betreuer: K. Breunig ; H. Lilie ; K. Melcher". Halle, Saale : Universitäts- und Landesbibliothek Sachsen-Anhalt, 2013. http://d-nb.info/1033306649/34.
Texto completo da fonteAnders, Alexander [Verfasser]. "Molekularer Mechanismus eines Transkriptionsschalters : experimentelle Analyse und mathematische Modellierung des Gal4-Gal80-Gal1-Regulationsmoduls aus Kluyveromyces lactis / von Alexander Anders". 2006. http://d-nb.info/985265698/34.
Texto completo da fonte