Literatura científica selecionada sobre o tema "Fear circuit"
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Artigos de revistas sobre o assunto "Fear circuit"
Sah, Pankaj, e R. Frederick Westbrook. "The circuit of fear". Nature 454, n.º 7204 (julho de 2008): 589–90. http://dx.doi.org/10.1038/454589a.
Texto completo da fonteSekiguchi, Masayuki. "Fear Circuit and Anxiety Disorders". Anxiety Disorder Research 10, n.º 1 (31 de outubro de 2018): 2–9. http://dx.doi.org/10.14389/jsad.10.1_2.
Texto completo da fonteShultz, Brianna, Abigail Farkash, Bailey Collins, Negin Mohammadmirzaei e Dayan Knox. "Fear learning-induced changes in AMPAR and NMDAR expression in the fear circuit". Learning & Memory 29, n.º 3 (15 de fevereiro de 2022): 83–92. http://dx.doi.org/10.1101/lm.053525.121.
Texto completo da fonteMcNally, Gavan P., Joshua P. Johansen e Hugh T. Blair. "Placing prediction into the fear circuit". Trends in Neurosciences 34, n.º 6 (junho de 2011): 283–92. http://dx.doi.org/10.1016/j.tins.2011.03.005.
Texto completo da fonteSelemon, Lynn D., Keith A. Young, Dianne A. Cruz e Douglas E. Williamson. "Frontal Lobe Circuitry in Posttraumatic Stress Disorder". Chronic Stress 3 (janeiro de 2019): 247054701985016. http://dx.doi.org/10.1177/2470547019850166.
Texto completo da fonteWang, Qin, Qi Wang, Xing-Lei Song, Qin Jiang, Yan-Jiao Wu, Ying Li, Ti-Fei Yuan et al. "Fear extinction requires ASIC1a-dependent regulation of hippocampal-prefrontal correlates". Science Advances 4, n.º 10 (outubro de 2018): eaau3075. http://dx.doi.org/10.1126/sciadv.aau3075.
Texto completo da fonteBirnie, Matthew T., e Tallie Z. Baram. "Principles of emotional brain circuit maturation". Science 376, n.º 6597 (3 de junho de 2022): 1055–56. http://dx.doi.org/10.1126/science.abn4016.
Texto completo da fonteAmano, T., S. Duvarci, D. Popa e D. Pare. "The Fear Circuit Revisited: Contributions of the Basal Amygdala Nuclei to Conditioned Fear". Journal of Neuroscience 31, n.º 43 (26 de outubro de 2011): 15481–89. http://dx.doi.org/10.1523/jneurosci.3410-11.2011.
Texto completo da fonteCoryell, Matthew W., Adam E. Ziemann, Patricia J. Westmoreland, Jill M. Haenfler, Zlatan Kurjakovic, Xiang-ming Zha, Margaret Price, Mikael K. Schnizler e John A. Wemmie. "Targeting ASIC1a Reduces Innate Fear and Alters Neuronal Activity in the Fear Circuit". Biological Psychiatry 62, n.º 10 (novembro de 2007): 1140–48. http://dx.doi.org/10.1016/j.biopsych.2007.05.008.
Texto completo da fonteBukalo, Olena, Courtney R. Pinard, Shana Silverstein, Christina Brehm, Nolan D. Hartley, Nigel Whittle, Giovanni Colacicco et al. "Prefrontal inputs to the amygdala instruct fear extinction memory formation". Science Advances 1, n.º 6 (julho de 2015): e1500251. http://dx.doi.org/10.1126/sciadv.1500251.
Texto completo da fonteTeses / dissertações sobre o assunto "Fear circuit"
Wright, Kristina M. "Revising the Role of the Ventrolateral Periaqueductal Gray in the Fear Circuit:". Thesis, Boston College, 2021. http://hdl.handle.net/2345/bc-ir:109159.
Texto completo da fonteThesis advisor: John P. Christianson
The ability to accurately evaluate and respond to threats is vital to survival. Disruptions in neural circuits of fear give rise to maladaptive threat responding, and have clinical implications in fear and anxiety disorders. To better inform therapeutic interventions, it is imperative that roles for regions classically associated with fear continue to be refined, and that novel nodes are incorporated into what is most certainly a larger fear circuit. In the canonical view, threat estimates are generated at the level of the amygdala and sent to the ventrolateral periaqueductal gray (vlPAG), which organizes an appropriate behavioral response, most notably freezing. Despite a multitude of studies successfully linking the vlPAG and Pavlovian fear behavior, evidence of a direct neural correlate for fear expression in the vlPAG is lacking. By contrast, a role for the caudal substantia nigra (cSN) in fear, stands apart from its canonical associations with movement and reward processes. Although there is new interest in examining a role for the nigra in fear modulation, this is essentially an uncharted area of discovery. The goals of this dissertation are three-fold. First, to propose a role for vlPAG activity in threat estimation, a function previously restricted to the upstream amygdala. Second, to scrutinize vlPAG neural activity using a novel multi-cue Pavlovian procedure and identify the long-anticipated, direct neural correlate for fear expression. Third, to present causal evidence supporting the cSN as a potential node in a circuit that most certainly extends beyond regions canonically associated with fear
Thesis (PhD) — Boston College, 2021
Submitted to: Boston College. Graduate School of Arts and Sciences
Discipline: Psychology
Yuzhe, Li. "Computational Investigations on Uncertainty-Dependent Extinction of Fear Memory". 京都大学 (Kyoto University), 2017. http://hdl.handle.net/2433/225756.
Texto completo da fontePagani, Jerome H. "The medial hypothalamic defensive circuit and predator odor-induced fear a comparison of electrolytic and neurotoxic lesions /". Access to citation, abstract and download form provided by ProQuest Information and Learning Company; downloadable PDF file, 127 p, 2007. http://proquest.umi.com/pqdweb?did=1397903941&sid=8&Fmt=2&clientId=8331&RQT=309&VName=PQD.
Texto completo da fonteAime, Mattia. "Circuit mechanisms for encoding discriminative learning in the dorsal prefrontal cortex of behaving mice". Thesis, Bordeaux, 2017. http://www.theses.fr/2017BORD0805/document.
Texto completo da fonteThe ability of an organism to predict forthcoming events is crucial for survival, and depends on the repeated contingency and contiguity between sensory cues and the events (i.e. danger) they must predict. The resulting learned association provides an accurate representation of the environment by increasing discriminative skills between threat and safety signals, most likely as a result of the interaction between the prefrontal cortex (PFC) and the basolateral amygdala (BLA). Although it suggests that local neuronal networks in the PFC might encode opposing memories that are preferentially selected during recall by recruiting specific cortical or subcortical structures, whether such a discriminative representation is wired within discrete prefrontal circuits during learning and by which synaptic mechanisms remain unclear. Here, the work at issue demonstrates that discrimination learning of both safe and fear-conditioned stimuli depends on full activity of the frontal association cortex, and is associated with the formation of cue-specific neuronal assemblies therein. During learning, prefrontal pyramidal neurons were potentiated through sensory-driven dendritic non-linearities supported by the activation of long-range inputs from the basolateral amygdala (BLA). Taken together, these data provide evidence for a new synaptic level mechanism that coincidently link (or meta-associate) during learning features of perceived experience with BLA mediated emotional state into prefrontal memory assemblies
Lima, Miguel Antonio Xavier de. "Investigação da circuitaria cortical envolvida no processamento do medo contextual à ameça predatória". Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/42/42131/tde-17022016-135344/.
Texto completo da fonteNeurochemical lesions placed into ventral part of anteromedial thalamic nucleus (AMv) disrupt contextual, but not innate, fear responses to predatory threats. In the present investigation, we determined whether the AMv is involved in the acquisition and/or retrieval of the conditioned responses, and if its cortical targets are involved in the fear memory processing. In the first assay, we found that AMv has a critical role in the acquisition of conditioned responses. The cortical areas prelimbic (PL), anterior cingulate area (ACA), anteromedial visual area (VISam) and the ventral part of retrosplenial area (RSPv), receive projections from AMv and are recruited during predator exposure. The integrity of these cortical areas is required for the processing of the mnemonic processes here addressed. Our data corroborate current ideas on functional cortical modules, and help to elucidate how they are involved in the acquisition of fear memories related to life threatening situations.
Bouarab, Chloe. "Modifications post-traductionnelles des histones au sein du circuit hippocampo-amygdalien déterminant le passage d'une mémoire de peur normale à une mémoire traumatique". Thesis, Bordeaux, 2016. http://www.theses.fr/2016BORD0261/document.
Texto completo da fonteMemory alterations associated with post-traumatic stress disorder (PTSD) are a fundamental feature of this pathology. PTSD is characterized both by hypermnesia for simple salient trauma-related stimuli and amnesia for peri-traumatic contextual cues. In humans, this disorder is associated with hippocampal hypofunction and amygdalar hyperfunction, which may underlie such paradoxical memory pattern. However, neurobiological bases of PTSD, particularly at the molecular level, remain largely unknown. A behavioral model based on aversive conditioning was developed in mice by our team. This model allows the comparison between a normal, i.e. “contextualized” and adaptive, fear memory, and a PTSD-like pathological fear memory, i.e. “decontextualized” and focused on a salient cue of the trauma. Since specific epigenetic alterations have been involved in the development of contextual fear memory, our aim was the identification of the alterations in post-translational histone modifications underlying the development of traumatic memory instead of normal fear memory. Our results first reveal that normal and PTSD-like fear memory are associated with distinct acetylation/methylation profiles of histone H3 in the hippocampal-amygdalar network. Specifically, we show that, compared to normal fear memory, PTSD-like memory is associated with a switch from H3K9 hyperacetylation (marker of transcriptional activation) to H3K9 hypermethylation (marker of transcriptional repression) in hippocampal CA1, as well as a significant reduction of H3K27 trimethylation, which results in an increased transcription, in the lateral amygdala. Second, we show that the pharmacological manipulation of the acetylation/methylation balance of H3K9 in the hippocampus can prevent or promote the development of PTSD-like memory. Finally, a last series of experiments shows that (i) prenatal stress is a risk factor for the development of PTSD-like memory, (ii) which is associated with specific epigenetic alterations and (iii) that such vulnerability to stress can be transmitted to subsequent generations
SILVA, BIANCA AMBROGINA. "INDEPENDENT HYPOTHALAMIC CIRCUITS FOR SOCIAL AND PREDATOR FEAR". Doctoral thesis, Università degli Studi di Milano, 2014. http://hdl.handle.net/2434/229915.
Texto completo da fonteCarey, Guillaume. "Imaging anxiety in Parkinson's disease". Electronic Thesis or Diss., Université de Lille (2022-....), 2024. https://pepite-depot.univ-lille.fr/ToutIDP/EDBSL/2024/2024ULILS023.pdf.
Texto completo da fonteAnxiety in Parkinson's disease (PD) is a frequent and disabilitating non-motor symptom. It is difficult to manage, partly due to a poor knowledge of the underlying mechanisms. The objective of this thesis was to identify the underlying mechanisms of PD-related anxiety, using multimodal brain MRI.A systematic review of the literature on imaging data in PD-related anxiety was first carried out, allowing the generation of initial hypotheses. Then, several studies including structural and functional brain MRI analyses were carried out in PD patients with or without clinically significant anxiety. Our analyses focused on the fear circuit, known to be involved in anxiety disorders and fear processing, and the limbic cortico-striato-thalamo-cortical circuit, known for its involvement in the neuropsychiatric symptoms of PD.Our results suggest that PD-related anxiety is the consequence of a functional and structural imbalance between these two circuits. Certain overlapping structures, such as the thalamus, the striatum or the brainstem nuclei, could be key areas whose alteration could explain the high prevalence of these disorders in PD. Further works based in particular on technological advances in imaging and new concepts concerning the pathophysiology of PD will be necessary to answer the remaining questions
Joly, Fanny. "Impacts d’une perturbation de la voie TSC2/mTOR dans l’amygdale dès l’adolescence sur le comportement de peur et la fonctionnalité du cortex préfrontal chez le rat adulte Disruption of Amygdala Tsc2 in Adolescence Leads to Changed Prelimbic Cellular Activity and Generalized Fear Responses at Adulthood in Rats". Thesis, université Paris-Saclay, 2021. http://www.theses.fr/2021UPASL016.
Texto completo da fonteAdolescence is a highly sensitive developmental period characterized by massive structural and functional changes in networks regulating emotional and cognitive behaviors, with maturational processes influenced by environmental and genetical factors. Post-traumatic stress disorder (PTSD) is a psychiatric disorder characterized by an exaggerated fear, overgeneralization, and deficits in fear extinction. Nowadays, genetical and/or environmental predisposal factors for PTSD are not fully understood, but we know that an intense stress or a trauma endured during adolescence promotes the appearance of PTSD at adulthood following a novel trauma exposure.In this thesis, we particularly studied two structures that belong to the fear-network, the amygdala and prefrontal cortex, which follow an asynchronous maturation. While the amygdala is functionally mature at a juvenile age, its activity could impact the late maturation of the medial prefrontal cortex (mPFC). We aimed to study the impact of a disruption of Tsc2/mTOR pathway in the excitatory cells of the basolateral nucleus of the amygdala (BLA) in rats at young adolescence (post-natal day 25, PN25) or at the end of adolescence (PN50). When animals had reached adulthood, we assessed emotional behavior through a Pavlovian fear conditioning protocol, and the basal mPFC activity through the measure of expression of immediate early gene c-FOS. We show that only animals altered during young adolescence presented at the adult age typical symptoms of PTSD (fear extinction deficits, overgeneralization of fear), associated with an increase of mPFC basal activity, especially in cortical layers known to be involved in the maintenance of fear memory and expression. Thus, we suggest that a developmental dysfunction of the amygdala early in adolescence could be a predisposal factor to PTSD appearance at adulthood
Hancock, Kate. "Women's perceptions of safety : CCTV in an inner city setting". Thesis, Edith Cowan University, Research Online, Perth, Western Australia, 2004. https://ro.ecu.edu.au/theses/801.
Texto completo da fonteLivros sobre o assunto "Fear circuit"
Stujenske, Joseph Matthew. Prefrontal-Amygdala Circuits Regulating Fear and Safety. [New York, N.Y.?]: [publisher not identified], 2016.
Encontre o texto completo da fonteill, Wiley Nancy 1964, ed. The blammo--surprise! book: A story to help children overcome fears. New York: Magination Press, 1988.
Encontre o texto completo da fonteBourguignon, Laurence. A friend for Tiger. Mahwah, NJ: BridgeWater Books, 1994.
Encontre o texto completo da fonteGavin, Andrews, ed. Stress-induced and fear circuitry disorders: Advancing the research agenda for DSM-V. Washington, DC: American Psychiatric Pub., 2009.
Encontre o texto completo da fontePedrozo, Sebastián. Abejas y flores marchitas. Montevideo, Uruguay: Alfaguara, 2007.
Encontre o texto completo da fonteMcCully, Emily Arnold. Juliette et Bellini. [France]: Kaléidoscope, 1994.
Encontre o texto completo da fonteMichael, Zulli, e Cooper Alice, eds. The compleat Alice Cooper: Incorporating the three acts of Alice Cooper - the last temptation. New York: Marvel Comics, 1994.
Encontre o texto completo da fonteWhen Panic Happens: Short-Circuit Anxiety and Fear in the Moment Using Neuroscience and Polyvagal Theory. New Harbinger Publications, 2024.
Encontre o texto completo da fonteSargin, Derya, Chen Yan e Sheena Josselyn. Genetic Tools in the Erasure of Emotional Memory. Editado por Turhan Canli. Oxford University Press, 2013. http://dx.doi.org/10.1093/oxfordhb/9780199753888.013.004.
Texto completo da fonteCuthbert, Bruce N. The Nimh Research Domain Criteria Project. Editado por Dennis S. Charney, Eric J. Nestler, Pamela Sklar e Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0071.
Texto completo da fonteCapítulos de livros sobre o assunto "Fear circuit"
Ren, Chaoran, e Qian Tao. "Neural Circuits Underlying Innate Fear". In Advances in Experimental Medicine and Biology, 1–7. Singapore: Springer Singapore, 2020. http://dx.doi.org/10.1007/978-981-15-7086-5_1.
Texto completo da fonteKawai, Nobuyuki. "The Underlying Neuronal Circuits of Fear Learning and the Snake Detection Theory (SDT)". In The Fear of Snakes, 33–58. Singapore: Springer Singapore, 2019. http://dx.doi.org/10.1007/978-981-13-7530-9_3.
Texto completo da fonteMarek, Roger, e Pankaj Sah. "Neural Circuits Mediating Fear Learning and Extinction". In Advances in Neurobiology, 35–48. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-94593-4_2.
Texto completo da fonteTait, Peta. "Technologies of Risk, Fear and Fun: Human and Nonhuman Circus Performance". In Circus, Science and Technology, 101–20. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-43298-0_6.
Texto completo da fonteLal, Pradeep, Hideyuki Tanabe e Koichi Kawakami. "Genetic Identification of Neural Circuits Essential for Active Avoidance Fear Conditioning in Adult Zebrafish". In Methods in Molecular Biology, 169–81. New York, NY: Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3401-1_11.
Texto completo da fonteMoulédous, L., P. Roullet e Bruno P. Guiard. "Brain Circuits Regulated by the 5-HT2A Receptor: Behavioural Consequences on Anxiety and Fear Memory". In 5-HT2A Receptors in the Central Nervous System, 231–58. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-70474-6_10.
Texto completo da fonteFrank, Guido K. W. "Interactions Between Fear-Driven Food Avoidance and the Ventral Striatal-Hypothalamic Circuitry Reinforce Eating Disorder Behaviors". In Handbook of the Biology and Pathology of Mental Disorders, 1–20. Cham: Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-32035-4_49-1.
Texto completo da fonteRosenbaum, Blake L., Kara K. Cover, Huijin Song e Mohammed R. Milad. "Neural Circuit Mechanisms of Fear Extinction". In Anxiety Disorders, 343–54. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199395125.003.0024.
Texto completo da fontePanksepp, Jaak. "The Sources of Fear and Anxiety in the Brain". In Affective Neuroscience, 206–22. Oxford University PressNew York, NY, 1998. http://dx.doi.org/10.1093/oso/9780195096736.003.0011.
Texto completo da fonteIqbal, Asifa. "Inclusive, Safe and Resilient Public Spaces: Gateway to Sustainable Cities?" In Urban Transition - Perspectives on Urban Systems and Environments [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.97353.
Texto completo da fonteTrabalhos de conferências sobre o assunto "Fear circuit"
Carbonieri, Matteo, Daniel Bachinski Pinhal, Jonathan Godbehere e Mircea Popescu. "FEA Augmented Equivalent Circuit for Accurate Performance Prediction of Induction Machines". In 2024 International Conference on Electrical Machines (ICEM), 1–7. IEEE, 2024. http://dx.doi.org/10.1109/icem60801.2024.10700286.
Texto completo da fonteLi, Guoshi, Gregory J. Quirk e Satish S. Nair. "Regulation of Fear by Amygdala Intercalated Cells in a Network Model of Fear Acquisition and Extinction". In ASME 2008 Dynamic Systems and Control Conference. ASMEDC, 2008. http://dx.doi.org/10.1115/dscc2008-2403.
Texto completo da fonteRam, Rashmirekha, Hemanta Kumar Palo e Mihir Narayan Mohanty. "Recognition of fear from speech using adaptive algorithm with MLP classifier". In 2016 International Conference on Circuit, Power and Computing Technologies (ICCPCT). IEEE, 2016. http://dx.doi.org/10.1109/iccpct.2016.7530149.
Texto completo da fonteLi, Guoshi, Stacy Cheng, Frank Ko, Scott L. Raunch, Gregory Quirk e Satish S. Nair. "Computational Modeling of Lateral Amygdala Neurons During Acquisition and Extinction of Conditioned Fear, Using Hebbian Learning". In ASME 2006 International Mechanical Engineering Congress and Exposition. ASMEDC, 2006. http://dx.doi.org/10.1115/imece2006-15078.
Texto completo da fonteMiranda, Jose A., Manuel F. Canabal, Jose M. Lanza-Gutierrez, Marta Portela Garcia e Celia Lopez-Ongil. "Toward Fear Detection using Affect Recognition". In 2019 XXXIV Conference on Design of Circuits and Integrated Systems (DCIS). IEEE, 2019. http://dx.doi.org/10.1109/dcis201949030.2019.8959852.
Texto completo da fonteYang, Ruoting, K. Sriram e Francis J. Doyle. "Control circuitry for fear conditioning associated with Post-Traumatic Stress Disorder (PTSD)". In 2010 49th IEEE Conference on Decision and Control (CDC). IEEE, 2010. http://dx.doi.org/10.1109/cdc.2010.5717136.
Texto completo da fontePendyam, Sandeep, Dongbeom Kim, Gregory J. Quirk e Satish S. Nair. "Acquisition of Fear and Extinction in Lateral Amygdala: A Modeling Study". In ASME 2010 Dynamic Systems and Control Conference. ASMEDC, 2010. http://dx.doi.org/10.1115/dscc2010-4218.
Texto completo da fonteSharmili, S. Shahana, e R. Kanagaraj. "Live Beyond Fear: A Virtual Reality Serious Game Platform to Overcome Phobias". In 2020 5th International Conference on Devices, Circuits and Systems (ICDCS). IEEE, 2020. http://dx.doi.org/10.1109/icdcs48716.2020.243592.
Texto completo da fonte"Accurate Modeling and Simulation for the Circuit Behavior of Si-Tip FEA". In 10th International Conference on Vacuum Microelectronics. IEEE, 1997. http://dx.doi.org/10.1109/ivmc.1997.627653.
Texto completo da fonteSu, Zou, Li Chuanri, Xu Fei e Qiao Liang. "Correlation Factor Analysis of FEA Model Simplification Methods of Printed Circuit Board". In 2011 Seventh International Conference on Computational Intelligence and Security (CIS). IEEE, 2011. http://dx.doi.org/10.1109/cis.2011.326.
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