Literatura científica selecionada sobre o tema "Exercise bible"
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Artigos de revistas sobre o assunto "Exercise bible"
Towner, W. Sibley. "The Future of Nature". Interpretation: A Journal of Bible and Theology 50, n.º 1 (janeiro de 1996): 27–35. http://dx.doi.org/10.1177/002096439605000104.
Texto completo da fonteKuwornu-Adjaottor, Jonathan Edward Tetteh. "critical study of the translation of Mark 1:12 in the Dangme translation of the Bible". Oguaa Journal of Religion and Human Values 5, n.º 2 (1 de dezembro de 2019): 97–109. http://dx.doi.org/10.47963/ojorhv.v5i2.1168.
Texto completo da fonteLee, Hyun Soo. "Studies on the Regimen Thought of Holy Bible Focused on Physical Exercise". Journal of Sport and Leisure Studies 26 (31 de maio de 2006): 81–96. http://dx.doi.org/10.51979/kssls.2006.05.26.81.
Texto completo da fonteMusopole, A. C. "Witchcraft Terminology, the Bible, and African Christian Theology: An Exercise in Hermeneutics". Journal of Religion in Africa 23, n.º 4 (novembro de 1993): 347. http://dx.doi.org/10.2307/1580990.
Texto completo da fonteMusopole, A. C. "Witchcraft Terminology, the Bible, and African Christian Theology: an Exercise in Hermeneutics". Journal of Religion in Africa 23, n.º 1-4 (1993): 347–54. http://dx.doi.org/10.1163/157006693x00193.
Texto completo da fonteMaust, Drew. "The Parable of the Peanut Butter Sandwich: An Exercise in Artificial Intelligence and (Pseudo-) Bible Translation". Journal of Translation 19, n.º 1 (2023): 127–48. http://dx.doi.org/10.54395/jot-pbmir.
Texto completo da fonteChancey, Mark A. "The Bible, the First Amendment, and the Public Schools in Odessa, Texas". Religion and American Culture: A Journal of Interpretation 19, n.º 2 (2009): 169–205. http://dx.doi.org/10.1525/rac.2009.19.2.169.
Texto completo da fonteNemes, Steven. "On Reading the Bible as Scripture, Encountering the Church". Perichoresis 18, n.º 5 (1 de dezembro de 2020): 67–86. http://dx.doi.org/10.2478/perc-2020-0029.
Texto completo da fonteVan der Merwe, D. G. "The glory-motif in John 17:1-5: An exercise in Biblical semantics". Verbum et Ecclesia 23, n.º 1 (6 de setembro de 2002): 226–49. http://dx.doi.org/10.4102/ve.v23i1.1250.
Texto completo da fonteKim-Cragg, HyeRan. "Preaching Addressing Environmental Crises through the Use of Scripture: An Exploration of a Practical Theological Methodology". Religions 13, n.º 3 (7 de março de 2022): 226. http://dx.doi.org/10.3390/rel13030226.
Texto completo da fonteTeses / dissertações sobre o assunto "Exercise bible"
Grondin, Christian. "« CE NE SONT PAS LA CHAIR ET LE SANG... » Les conditions bibliques de l'élaboration de l'élection dans la pratique des Exercices spirituels d'Ignace de Loyola". Thesis, Université Laval, 2013. http://www.theses.ulaval.ca/2013/29720/29720.pdf.
Texto completo da fonteChase, Mitchell. "Resurrection Hope in Daniel 12:2: An Exercise in Biblical Theology". Diss., 2013. http://hdl.handle.net/10392/4532.
Texto completo da fonteFarahnak, Zahra. "The effect of exercise training on cholesterol and bile acid metabolism in ovariectomized rats". Thèse, 2016. http://hdl.handle.net/1866/18529.
Texto completo da fonteThere has been accumulating evidence in recent years that the estrogen deficient state in ovariectomized (Ovx) animals and in postmenopausal women results in substantial liver triglyceride (TG) accumulation. However, evidence of disturbances in cholesterol metabolism in link with estrogen deficiency is limited to observations of higher plasma total cholesterol levels found in human as well as in animals. In fact, the impact of estrogen withdrawal on liver cholesterol metabolism has received little attention and shows some controversies. Therefore, the three studies presented in this thesis have been conducted in Ovx rats, as an animal model of post-menopausal women, to investigate the effects of estrogen withdrawal on key molecular markers of cholesterol and bile acid metabolism in liver and in transintestinal cholesterol excretion (TICE), and also to determine the potential role of exercise training as a positive alternative intervention. It has been shown that exercise training can improve plasma cholesterol levels. An enhanced transport of peripheral cholesterol toward the liver for subsequent secretion into bile and excretion from the body has been suggested; however, the underlying mechanism for this action is not fully understood. In the first study, we showed that estrogen withdrawal was associated with higher (P < 0.05) liver total cholesterol under the standard diet and the standard diet + cholesterol diet, while liver triglyceride (TG) content was higher in Ovx than in Sham rats in all three dietary conditions which are the standard diet, the standard diet + cholesterol and the high fat diet + cholesterol. Surprisingly, the standard diet + cholesterol was associated with lower (P < 0.001) plasma total cholesterol and TG levels in Ovx than in Sham rats, suggesting a decrease in very low-density lipoprotein (VLDL) secretion. Accordingly, several transcripts of key markers of VLDL synthesis including microsomal triglyceride transfer protein (MTP) and apoB-100 were decreased (P < 0.05) in Ovx compared to Sham rats under the three dietary conditions and even more so for MTP and apoB-100 when rats were fed the standard diet + cholesterol. To go one step further, in the second study we determined the effects of exercise training on hepatic key markers of farnesoid X receptor (FXR)-small heterodimer partner (SHP)-cholesterol 7 alpha-hydroxylase (CYP7A1) (FXR-SHP-CYP7A1) pathway, involved in cholesterol conversion into bile acid and excretion from the body, in Ovx cholesterol fed rats. Our main experimental group was Ovx rats fed a high cholesterol diet (Ovx-Chol) that was compared, on one hand, to a group of Ovx rats fed a standard diet (Ovx-SD) to observe the effects of the diet and, on the other hand, compared to a group of Sham operated rats fed the cholesterol diet (Sham-Chol) to observe the effect of estrogen withdrawal. Results of this study showed that plasma and liver total cholesterol levels were not affected by exercise training in any of the experimental conditions. Cholesterol feeding in both Sham and Ovx rats resulted in significantly (P<0.001) higher hepatic cholesterol accumulation than in Ovx-SD rats. A main effect of training (P< 0.05) was, however, found for transcripts of SHP and CYP7A1. The SHP and CYP7A1 transcripts were increased by training. These results suggest that exercise training through up-regulation of genes involved in bile acid formation may modulate cholesterol metabolism in Ovx animals. Finally, a recent growing body of evidence suggests that reverse cholesterol transport (RCT) can also proceed through a non-biliary pathway known as transintestinal cholesterol excretion (TICE). Indeed, both liver and intestine are involved in excretion of the excess cholesterol from the body. Based on this concept, we expanded our research to determine whether exercise training has an effect on intestinal membrane cholesterol receptors involved in TICE pathway in intact and Ovx rats fed a normal and a high cholesterol diet. Results of the third study showed that exercise training increased (P< 0.01) transcripts of intestinal LDL-R and PCSK9, which are involved in intestinal cholesterol uptake from circulation, and their nuclear transcription factor, intestinal sterol regulatory element-binding protein 2 (SREBP2) (P< 0.05) in both Sham and Ovx rats compared to rats remaining sedentary (Sed). On the other hand, hepatic LDL-R and PCSK9 gene expression was suppressed (P< 0.01) by cholesterol feeding but not affected by exercise training. Flavin monooxygenase 3 (FMO3) gene expression, as a cholesterol balance regulator in liver, was significantly decreased (P<0.01) by cholesterol feeding in both Sham and Ovx rats compared to rats were fed the SD diet but unchanged following exercise training and estrogen withdrawal. An up-regulation of intestinal gene expression of LDL-R and PCSK9 following voluntary wheel running in intact and Ovx rats suggests that exercise training may contribute to increased cholesterol elimination through the TICE pathway. Overall, our results indicate that a high cholesterol diet and ovariectomy combine to decrease the gene expression of key markers of VLDL synthesis suggesting a reduction in cholesterol excretion from the liver. Alternatively, it seems that reduced hepatic LDL-R transcript found in Ovx animals might be due to hepatic cholesterol accumulation. Moreover, our findings introduced exercise training as an appropriate non-pharmacological intervention to stimulate the excretion of the excess cholesterol from the body through upregulation of genes involved in bile acid biosynthesis in liver and intestinal basolateral cholesterol transporters in TICE.
Ngo, Sock Emilienne Tudor. "Carence en œstrogènes et bases moléculaires du métabolisme des triglycérides et du cholestérol dans le foie et l'intestin : effet de l'exercice physique". Thèse, 2013. http://hdl.handle.net/1866/10523.
Texto completo da fonteHepatic steatosis and plasma lipid profile deterioration are metabolic diseases favored by post-menopausal estrogen deficiency. However, mechanisms underlying these diseases have not been systematically adressed. The aim of this thesis was to investigate molecular mechanisms causing hypercholesterolemia and lipids (triglycerides: TG and cholesterol) accumulation in the liver using animal model of menopause, the ovariectomized (Ovx) Sprague Dawley rat. We also examined whether lifestyle modifications such as physical activity can prevent or correct changes induced by Ovx. Finally, rosuvastatin (statine) was used as a pharmacological therapy of hypercholesterolemia in order to understand its effect at the molecular level in Ovx rats. The first study was designed to determine how the Ovx may affect levels of TG and cholesterol in the liver of rats fed a high-fat diet (HF: 42% fat). Rats were submitted to a HF or a normal diet for 6 weeks prior to Ovx or being sham operated, and then kept on the same diets for another 6 weeks. The Ovx increased liver TG content, but not the HF diet alone. However, the combination of Ovx and HF diet resulted in a greater liver TG accumulation than that observed in Ovx submitted to normal diet. The mRNA levels of CPT-1, PGC1 and PPARα involved in liver lipid oxidation significantly increased in rats fed the HF diet (p ˂ 0.001; p ˂ 0.01; p ˂ 0.05 respectively); but this increase was substantially less if HF fed rats were Ovx (p ˂ 0.05; p ˂ 0.05; p ˂ 0.07 respectively), thus favouring TG accumulation in the liver. The combination of HF diet and Ovx also induced hypercholesterolemia and an increase in liver total cholesterol content, in spite of the reduction of liver HMGCoA-r gene expression, the key enzyme for cholesterol synthesis. This was also associated with a decrease of liver CYP7a1 gene expression, suggesting a reduction in bile acids synthesis. Having found in the first study that the Ovx increases liver and plasma cholesterol levels, we aimed in the second study at determining the effects of Ovx on gene expression of hepatic and intestinal transporters and enzymes involved in cholesterol and bile acids metabolism; and to verify whether treadmill exercise could prevent or correct changes induced by Ovx. The Ovx resulted in hypercholesterolemia associated with a reduction in gene expression of hepatic low-density lipoprotein receptor (LDL-R), lipoprotein remnants receptor (LRP1), SREBP-2 and PCSK9, suggesting a failure in the clearance of plasma lipoproteins particles. The Ovx also inhibited the expression of MTP and stimulated that of SR-B1 in the liver, but no change was observed with CYP7a1. These molecular changes might, therefore, favor cholesterol accumulation in the liver. Exercise training did not correct the deleterious effects caused by Ovx on gene expression of these molecular markers in the liver with the exception of SREBP-2. However, in the intestine (ileum) treadmill exercise reduced gene expression of molecular markers involved in the absorption of bile acids (OSTα/β, FXR, RXRα, Fgf15) and cholesterol (LXRα, NCP1L1) in Sham trained rats compared to sedentary rats. This could prevent the development of cholestasis and hypercholesterolemia protecting partially against the onset of atherosclerosis. In view of the deleterious effects (hypercholesterolemia and decreased in gene expression of LDL-R, PCSK9, LRP1, SREBP-2 and HMGCoA-r in the liver) caused by Ovx on cholesterol metabolism observed in the second study, the 3rd study was designed to test the effect of rosuvastatin (Ros) on gene expression of these molecular markers in Ovx sedentary rats or in Ovx rats submitted to voluntary training. Ros was injected to Ovx rats subcutaneously at dose of 5mg/kg/day during 21 days from the ninth week after ovariectomy. Ros failed to decrease plasma LDL-C and TC in Ovx rats. In contrast, Ros increased (P ˂ 0.05) PCSK9, SREBP-2, LRP1, HMGCoA-r and ACAT2 but not significantly (P ˂ 0.3) LDL-R mRNA in the Ovx sedentary and trained rat liver. Ros failed to decrease plasma LDL-C in Ovx rats probably because of a stronger induction of PCSK9 than LDL-R gene expression. However by increasing LRP1 expression, Ros could decrease circulating lipoprotein remnants and, therefore, protects partially against the onset of cardiovascular diseases. In conclusion, the studies of this thesis indicate that the decrease of ovarian estrogen levels causes radical changes in hepatic TG and cholesterol metabolism caused by alterations in the expression of key genes associated with metabolic pathways.
Leblond, François. "Régulation de la fonction vasculaire pendant le vieillissement : rôles de l’environnement post-natal et du gène suppresseur de tumeurs p53". Thèse, 2013. http://hdl.handle.net/1866/11065.
Texto completo da fonteEndothelial dysfunction is an early marker of atherosclerosis and cardiovascular diseases. Cardiovascular risk factors generate a chronic stress, challenging endogenous defense mechanisms that are critical to maintain endothelial function, such as antioxidant enzymes. The vascular environment impacts the integrity and long-term function of endothelial cells. Thus, a healthy lifestyle is beneficial for cardiovascular health: regular physical training and/or a Mediterranean diet are associated with the maintenance of endothelial function and a lower incidence of cardiovascular diseases. On the other hand, some key genes such as tumor suppressor gene p53, are known to regulate numerous cellular functions that are necessary to maintain endothelial cells integrity. We hypothesized that the post-natal vascular environment impacts the development of endogenous cellular defense mechanisms such as antioxidant enzymes, in order to protect against vascular stress that will occur later in life. Our major goal was to evaluate the impact of post-natal interventions and endogenous reduction of p53 expression on vascular endothelial function and its capacity to resist against a metabolic stress. In our first study, healthy C57Bl/6 mice were exposed from weaning to the age of 9 months to physical voluntary training (running wheel) or to the antioxidant catechin, and were compared to physically inactive mice that did not receive catechin. Then, exercise and catechin were stopped and mice were subjected to a regular or a high fat diet for 3 months; mice were sacrificed either at the age of 9 or 12 months. In trained mice, we observed that exercise prevented endothelial dysfunction and inflammation induced by the high fat diet. In catechin-treated mice, aortic endothelial function was maintained despite exposure to the high fat diet, but an inflammatory profile was observed. In physically inactive mice, the high fat diet induced endothelial dysfunction without inflammation. In our second study, mice partially deficient in p53 (p53+/-) were exposed to the same high fat diet from 3 to 6 months of age. Our rationale was based on the discovery that in vitro, p53 regulates antioxidant enzymes gene expression. In p53+/- mice, endothelial cells were protected from the stress of hypercholesterolemia induced by the high fat diet. However, this endothelial protection could be linked with an unexpected enhanced bile acid metabolism in p53+/- mice: low endogenous expression of p53 prevents the rise in plasma total cholesterol when fed a high fat diet, indirectly protecting the endothelium. In summary, we were able to demonstrate the importance of the vascular environment on endothelial function. The pro-atherosclerotic environment induced by the high fat diet stimulated vascular defense mechanisms, as observed by the activation of the compensatory EDHF pathway and superoxide dismutase activity, to maintain an adequate endothelial function. We observed that exercise and catechin had a different impact on the endothelium despite their antioxidant properties. These studies demonstrate the sensitivity of the endothelium to changes within the vascular environment. As the population is aging and the incidence of cardiovascular diseases is increasing, endothelial dysfunction will be more frequent. Therefore, a better comprehension of mechanisms or interventions that can protect endothelial function can only be beneficial.
Livros sobre o assunto "Exercise bible"
Villepigue, James C. The body sculpting bible for chest and arms: Men's edition. New York: Healthy Living Books, 2004.
Encontre o texto completo da fonteVillepigue, James C. The body sculpting bible express for men with the sculpt21 diet plan. New York: Hatherleigh Press, 2008.
Encontre o texto completo da fonteVillepigue, James C. The body sculpting bible swimsuit workout. Long Island City, NY: Hatherleigh Press, 2004.
Encontre o texto completo da fonteSutcliffe, Jenny. The back bible. White Plains, NY: Reader's Digest Association, 2012.
Encontre o texto completo da fonteJasper, David. Rhetoric, power and community: An exercise in reserve. Basingstoke: Macmillan, 1993.
Encontre o texto completo da fonteHall, Joanna. The exercise bible: The definitive guide to total body health and wellbeing. London: Kyle Cathie, 2002.
Encontre o texto completo da fonteVillepigue, James C. The body sculpting bible express for women with the sculpt21 diet plan. Long Island City, NY: Hatherleigh Press, 2008.
Encontre o texto completo da fonteVillepigue, James C. Body sculpting bible for men. 3a ed. Hobart, NY: Hatherleigh Press, 2011.
Encontre o texto completo da fonte1919-, Lewis Jack Pearl, ed. Interpreting 2 Corinthians 5:14-21: An exercise in hermeneutics. Lewiston, NY, USA: E. Mellen Press, 1989.
Encontre o texto completo da fonteGrantham, Nick. The strength & conditioning bible: How to train like an athlete. London: Bloomsbury, 2015.
Encontre o texto completo da fonteCapítulos de livros sobre o assunto "Exercise bible"
Leonhard, Clemens. "Marcion’s Gospel and the Pascha: An Exercise in Interdisciplinarity". In Liturgische Bibelrezeption/Liturgical Reception of the Bible, 57–68. Göttingen: Vandenhoeck & Ruprecht, 2022. http://dx.doi.org/10.13109/9783666567292.57.
Texto completo da fonteChristensen, Line Søgaard. "Homo Repetitivus and Anthropotechnics: Exercise Systems, Elite Practitioners, and Teaching Missions in the Hebrew Bible". In »What is Human?«, 45–64. Göttingen: Vandenhoeck & Ruprecht, 2016. http://dx.doi.org/10.13109/9783666531194.45.
Texto completo da fonte"Introduction: Preliminary exercise: Proverbs 15". In Major Poems of the Hebrew Bible, 11–17. BRILL, 2003. http://dx.doi.org/10.1163/9789004358799_003.
Texto completo da fonteChapman, Nathan S., e Michael W. McConnell. "Prayer, Bible Reading, and Coercion". In Agreeing to Disagree, 144–56. Oxford University PressNew York, 2023. http://dx.doi.org/10.1093/oso/9780195304664.003.0009.
Texto completo da fonteSchneidau, Herbert N. "Biblical Narrative and Modern Consciousness". In The Bible and the Narrative Tradition, 132–50. Oxford University PressNew York, NY, 1991. http://dx.doi.org/10.1093/oso/9780195070026.003.0007.
Texto completo da fonteRiches, John. "11. Conclusion". In The Bible: A Very Short Introduction, 130–40. Oxford University Press, 2021. http://dx.doi.org/10.1093/actrade/9780198863335.003.0011.
Texto completo da fonteDoak, Brian R. "Conclusion". In Ancient Israel's Neighbors, 194–98. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190690595.003.0009.
Texto completo da fonteCohen, Naomi W. "Scripture in the Schools". In Jews in Christian America, 187–213. Oxford University PressNew York, NY, 1992. http://dx.doi.org/10.1093/oso/9780195065374.003.0009.
Texto completo da fonte"An Exercise in the Critical Use of Models: The “Goodness of Fit” of Wilson’s Sect Model". In Social Scientific Models for Interpreting the Bible, 21–46. BRILL, 2000. http://dx.doi.org/10.1163/9789004496972_004.
Texto completo da fonte"The Bible and the Crocodile: An Exercise in Balancing Translation Technique and Text-Critical Data". In Authoritative Texts and Reception History, 11–19. BRILL, 2017. http://dx.doi.org/10.1163/9789004334960_003.
Texto completo da fonteTrabalhos de conferências sobre o assunto "Exercise bible"
Tamura, Hiroto, Hiroshi Hagiwara, Koji Kashihara e Hiroyuki Shinoda. "Psychophysiological Effects of Comfortable Walking Exercise on a Working Memory Task". In 2019 IEEE 19th International Conference on Bioinformatics and Bioengineering (BIBE). IEEE, 2019. http://dx.doi.org/10.1109/bibe.2019.00074.
Texto completo da fonteYahaya, S. Z., Z. Hussain e R. Boudville. "Design and simulation of wheel-chaired elliptical stepping exercise for stroke rehabilitation". In 2013 IEEE 13th International Conference on Bioinformatics and Bioengineering (BIBE). IEEE, 2013. http://dx.doi.org/10.1109/bibe.2013.6701668.
Texto completo da fonteNarciso, David, Miguel Melo, Susana Rodrigues, Joao Paulo Silva Cunha e Maximino Bessa. "Impact of Different Stimuli on User Stress During a Virtual Firefighting Training Exercise". In 2020 IEEE 20th International Conference on Bioinformatics and Bioengineering (BIBE). IEEE, 2020. http://dx.doi.org/10.1109/bibe50027.2020.00138.
Texto completo da fonteRuntti, Hilkka, Dimitris Filos, Mark van Gils, Ioanna Chouvarda, Anita Honka e Juha Parkka. "Guided physical exercise of cardiac patients during rehabilitation: Adherence and changes in physiological variables". In 2013 IEEE 13th International Conference on Bioinformatics and Bioengineering (BIBE). IEEE, 2013. http://dx.doi.org/10.1109/bibe.2013.6701533.
Texto completo da fonte